Heterozygosity for a hypomorphic Polβ mutation reduces the expansion frequency in a mouse model of the Fragile X-related disorders.
Autorzy:
Lokanga RA; Section on Gene Structure and Disease, Laboratory of Cell and molecular Biology, National Institute of Diabetes, Digestive and Kidney Diseases, National Institutes of Health, Bethesda, Maryland, United States of America; Department of Biochemistry, University of Cape Town Medical School, Cape Town, South Africa. SenejaniAG; Departments of Therapeutic Radiology and Human Genetics, Yale University, School of Medicine, New Haven, Connecticut, United States of America. Sweasy JB; Departments of Therapeutic Radiology and Human Genetics, Yale University, School of Medicine, New Haven, Connecticut, United States of America. Usdin K; Section on Gene Structure and Disease, Laboratory of Cell and molecular Biology, National Institute of Diabetes, Digestive and Kidney Diseases, National Institutes of Health, Bethesda, Maryland, United States of America.
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Źródło:
PLoS genetics [PLoS Genet] 2015 Apr 17; Vol. 11 (4), pp. e1005181. Date of Electronic Publication: 2015 Apr 17 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural
PPARγ negatively regulates T cell activation to prevent follicular helper T cells and germinal center formation.
Autorzy:
Park HJ; Department of Life Science, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea; Hanyang Biomedical Research Institute, Seoul, Korea. Kim DH; Department of Life Science, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea; Hanyang Biomedical Research Institute, Seoul, Korea. Choi JY; Department of Internal Medicine (Rheumatology), Yale University School of Medicine, New Haven, Connecticut, United States of America. Kim WJ; Department of Life Science, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea; Hanyang Biomedical Research Institute, Seoul, Korea. Kim JY; Department of Life Science, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea; Hanyang Biomedical Research Institute, Seoul, Korea. SenejaniAG; Department of Therapeutic Radiology and Genetics, Yale University School of Medicine, New Haven, Connecticut, United States of America. Hwang SS; Department of Life Science, Sogang University, Seoul, Korea. Kim LK; Department of Immunobiology, Yale University School of Medicine, New Haven, Connecticut, United States of America. Tobiasova Z; Department of Immunobiology, Yale University School of Medicine, New Haven, Connecticut, United States of America. Lee GR; Department of Life Science, Sogang University, Seoul, Korea. Craft J; Department of Internal Medicine (Rheumatology), Yale University School of Medicine, New Haven, Connecticut, United States of America; Department of Immunobiology, Yale University School of Medicine, New Haven, Connecticut, United States of America. Bothwell AL; Department of Immunobiology, Yale University School of Medicine, New Haven, Connecticut, United States of America. Choi JM; Department of Life Science, Research Institute for Natural Sciences, Hanyang University, Seoul, Korea; Hanyang Biomedical Research Institute, Seoul, Korea.
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Źródło:
PloS one [PLoS One] 2014 Jun 12; Vol. 9 (6), pp. e99127. Date of Electronic Publication: 2014 Jun 12 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Conservation of intron and intein insertion sites: implications for life histories of parasitic genetic elements.
Autorzy:
Swithers KS; Department of Molecular and Cell Biology, University of Connecticut, 91 North Eagleville Road, Storrs, CT 06269-3125, USA. />SenejaniAG Fournier GP Gogarten JP
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Źródło:
BMC evolutionary biology [BMC Evol Biol] 2009 Dec 31; Vol. 9, pp. 303. Date of Electronic Publication: 2009 Dec 31.
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