Temat: "Senescence" - OpacWWW

Skocz do pozycji: 1.

Tytuł:: Senescent cells at the crossroads of aging, disease, and tissue homeostasis.
Autorzy:: Kuehnemann C; Jean Mayer USDA Human Nutrition Research Center on Aging at Tufts University, Boston, Massachusetts, USA.
Wiley CD; Jean Mayer USDA Human Nutrition Research Center on Aging at Tufts University, Boston, Massachusetts, USA.; Pokaż więcej
Źródło:: Aging cell [Aging Cell] 2024 Jan; Vol. 23 (1), pp. e13988. Date of Electronic Publication: 2023 Sep 20.
Typ publikacji:: Journal Article; Review
MeSH Terms:: Aging*/physiology
Cellular Senescence*/physiology
Humans ; Animals ; Mice ; Disease Models, Animal ; Senescence-Associated Secretory Phenotype ; Homeostasis

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Skocz do pozycji: 2.

Tytuł:: Senescent Cells: Dual Implications on the Retinal Vascular System.
Autorzy:: Habibi-Kavashkohie MR; Department of Biological Sciences, Université du Québec à Montréal (UQAM), Montréal, QC H2L 2C4, Canada.; The Center of Excellence in Research on Orphan Diseases, Courtois Foundation (CERMO-FC), Montreal, QC H3G 1E8, Canada.
Scorza T; Department of Biological Sciences, Université du Québec à Montréal (UQAM), Montréal, QC H2L 2C4, Canada.; The Center of Excellence in Research on Orphan Diseases, Courtois Foundation (CERMO-FC), Montreal, QC H3G 1E8, Canada.
Oubaha M; Department of Biological Sciences, Université du Québec à Montréal (UQAM), Montréal, QC H2L 2C4, Canada.; The Center of Excellence in Research on Orphan Diseases, Courtois Foundation (CERMO-FC), Montreal, QC H3G 1E8, Canada.; Pokaż więcej
Źródło:: Cells [Cells] 2023 Sep 23; Vol. 12 (19). Date of Electronic Publication: 2023 Sep 23.
Typ publikacji:: Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:: Aging*/pathology
Cellular Senescence*
Humans ; Retina ; Retinal Vessels ; Senescence-Associated Secretory Phenotype

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Skocz do pozycji: 3.

Tytuł:: NR2F2 alleviates pulmonary fibrosis by inhibition of epithelial cell senescence.
Autorzy:: Wan R; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China.
Long S; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China.
Ma S; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China.
Yan P; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China.
Li Z; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China.
Xu K; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China.
Lian H; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China.
Li W; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China.
Duan Y; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China.
Zhu M; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China.
Wang L; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China. .; College of Life Science, Henan Normal University, Xinxiang, Henan, China. .
Yu G; State Key Laboratory Cell Differentiation and Regulation, Henan International Joint Laboratory of Pulmonary Fibrosis, Henan center for outstanding overseas scientists of pulmonary fibrosis, College of Life Science, Institute of Biomedical Science, Pingyuan Laboratory, Henan Normal University, Xinxiang, 453007, Henan, China. .; College of Life Science, Henan Normal University, Xinxiang, Henan, China. .; Pokaż więcej
Źródło:: Respiratory research [Respir Res] 2024 Apr 02; Vol. 25 (1), pp. 154. Date of Electronic Publication: 2024 Apr 02.
Typ publikacji:: Journal Article
MeSH Terms:: Cellular Senescence*
Idiopathic Pulmonary Fibrosis*/drug therapy
Animals ; Mice ; Bleomycin/adverse effects ; Epithelial Cells/metabolism ; Lung/metabolism

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Skocz do pozycji: 4.

Tytuł:: The Molecular Mechanisms in Senescent Cells Induced by Natural Aging and Ionizing Radiation.
Autorzy:: Ibragimova M; Department of General Biology and Genomics, Institute of Cell Biology and Biotechnology, L.N. Gumilyov Eurasian National University, Astana 010008, Kazakhstan.
Kussainova A; Department of General Biology and Genomics, Institute of Cell Biology and Biotechnology, L.N. Gumilyov Eurasian National University, Astana 010008, Kazakhstan.; Department of Health Sciences, University of Genova, Via Pastore 1, 16132 Genoa, Italy.
Aripova A; Department of General Biology and Genomics, Institute of Cell Biology and Biotechnology, L.N. Gumilyov Eurasian National University, Astana 010008, Kazakhstan.
Bersimbaev R; Department of General Biology and Genomics, Institute of Cell Biology and Biotechnology, L.N. Gumilyov Eurasian National University, Astana 010008, Kazakhstan.
Bulgakova O; Department of General Biology and Genomics, Institute of Cell Biology and Biotechnology, L.N. Gumilyov Eurasian National University, Astana 010008, Kazakhstan.; Pokaż więcej
Źródło:: Cells [Cells] 2024 Mar 21; Vol. 13 (6). Date of Electronic Publication: 2024 Mar 21.
Typ publikacji:: Journal Article; Review
MeSH Terms:: Aging*
Cellular Senescence*/genetics
Humans ; Cells, Cultured ; Radiation, Ionizing

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Skocz do pozycji: 5.

Tytuł:: Repurposing Drugs for Senotherapeutic Effect: Potential Senomorphic Effects of Female Synthetic Hormones.
Autorzy:: Bramwell LR; RNA-Mediated Mechanisms of Disease Group, Department of Clinical and Biomedical Sciences (Medical School), Faculty of Health and Life Sciences, University of Exeter, Exeter EX2 5DW, UK.
Frankum R; RNA-Mediated Mechanisms of Disease Group, Department of Clinical and Biomedical Sciences (Medical School), Faculty of Health and Life Sciences, University of Exeter, Exeter EX2 5DW, UK.
Harries LW; RNA-Mediated Mechanisms of Disease Group, Department of Clinical and Biomedical Sciences (Medical School), Faculty of Health and Life Sciences, University of Exeter, Exeter EX2 5DW, UK.; Pokaż więcej
Źródło:: Cells [Cells] 2024 Mar 15; Vol. 13 (6). Date of Electronic Publication: 2024 Mar 15.
Typ publikacji:: Journal Article
MeSH Terms:: Cellular Senescence*
Senotherapeutics*
Male ; Humans ; Female ; Drug Repositioning ; Hormones/pharmacology

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Skocz do pozycji: 6.

Tytuł:: High Glucose Promotes and Aggravates the Senescence and Dysfunction of Vascular Endothelial Cells in Women with Hyperglycemia in Pregnancy.
Autorzy:: Zheng L; Department of Gynecology and Obstetrics, Shanghai Sixth People's Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai 200233, China.
Li M; Laboratory for Reproductive Immunology, Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Obstetrics and Gynecology Hospital of Fudan University, Shanghai 200080, China.
Li H; Department of Gynecology and Obstetrics, Shanghai Sixth People's Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai 200233, China.; Department of Gynecology and Obstetrics, Jinshan Branch of Shanghai Sixth People's Hospital, Shanghai 201599, China.; Pokaż więcej
Źródło:: Biomolecules [Biomolecules] 2024 Mar 10; Vol. 14 (3). Date of Electronic Publication: 2024 Mar 10.
Typ publikacji:: Journal Article
MeSH Terms:: Cellular Senescence*
Hyperglycemia*
Humans ; Female ; Pregnancy ; Human Umbilical Vein Endothelial Cells ; Aging ; Glucose/pharmacology

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Skocz do pozycji: 7.

Tytuł:: Intermittent supplementation with fisetin improves arterial function in old mice by decreasing cellular senescence.
Autorzy:: Mahoney SA; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
Venkatasubramanian R; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
Darrah MA; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
Ludwig KR; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
VanDongen NS; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
Greenberg NT; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
Longtine AG; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
Hutton DA; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
Brunt VE; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
Campisi J; Buck Institute for Research on Aging, Novato, California, USA.; Lawrence Berkeley National Laboratory, Berkeley, California, USA.
Melov S; Buck Institute for Research on Aging, Novato, California, USA.
Seals DR; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
Rossman MJ; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.
Clayton ZS; Department of Integrative Physiology, University of Colorado Boulder, Boulder, Colorado, USA.; Pokaż więcej
Źródło:: Aging cell [Aging Cell] 2024 Mar; Vol. 23 (3), pp. e14060. Date of Electronic Publication: 2023 Dec 07.
Typ publikacji:: Journal Article
MeSH Terms:: Cellular Senescence*/genetics
Arteries*
Flavonols*
Mice ; Humans ; Animals ; Dietary Supplements ; Inflammation

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Skocz do pozycji: 8.

Tytuł:: IGFBP5 is released by senescent cells and is internalized by healthy cells, promoting their senescence through interaction with retinoic receptors.
Autorzy:: Alessio N; Department of Experimental Medicine, Luigi Vanvitelli Campania University, via Luigi De Crecchio 7, Naples, 80138, Italy.
Aprile D; Department of Experimental Medicine, Luigi Vanvitelli Campania University, via Luigi De Crecchio 7, Naples, 80138, Italy.
Peluso G; International Medical University UNICAMILLUS, Rome, Italy.
Mazzone V; Department of Experimental Medicine, Luigi Vanvitelli Campania University, via Luigi De Crecchio 7, Naples, 80138, Italy.
Patrone D; Department of Experimental Medicine, Luigi Vanvitelli Campania University, via Luigi De Crecchio 7, Naples, 80138, Italy.
Di Bernardo G; Department of Experimental Medicine, Luigi Vanvitelli Campania University, via Luigi De Crecchio 7, Naples, 80138, Italy. .
Galderisi U; Department of Experimental Medicine, Luigi Vanvitelli Campania University, via Luigi De Crecchio 7, Naples, 80138, Italy. .; Genome and Stem Cell Center (GENKÖK), Erciyes University, Kayseri, Turkey. .; Center for Biotechnology, Sbarro Institute for Cancer Research and Molecular Medicine Temple University, PA, Philadelphia, USA. .; Pokaż więcej
Źródło:: Cell communication and signaling : CCS [Cell Commun Signal] 2024 Feb 13; Vol. 22 (1), pp. 122. Date of Electronic Publication: 2024 Feb 13.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Cellular Senescence*/genetics
Neoplasms*/metabolism
Humans ; Aging ; Cells, Cultured ; Signal Transduction/genetics

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Skocz do pozycji: 9.

Tytuł:: Aging in the dermis: Fibroblast senescence and its significance.
Autorzy:: Zhang J; Immunology Department, Key Laboratory of Immune Microenvironment and Disease (Ministry of Education), Tianjin Medical University, Tianjin, China.
Yu H; Immunology Department, Key Laboratory of Immune Microenvironment and Disease (Ministry of Education), Tianjin Medical University, Tianjin, China.
Man MQ; Dermatology Hospital, Southern Medical University, Guangdong, China.; Department of Dermatology, University of California San Francisco and Veterans Affairs Medical Center, San Francisco, California, USA.
Hu L; Immunology Department, Key Laboratory of Immune Microenvironment and Disease (Ministry of Education), Tianjin Medical University, Tianjin, China.; Pokaż więcej
Źródło:: Aging cell [Aging Cell] 2024 Feb; Vol. 23 (2), pp. e14054. Date of Electronic Publication: 2023 Dec 01.
Typ publikacji:: Journal Article; Review
MeSH Terms:: Cellular Senescence*/physiology
Skin Aging*
Humans ; Fibroblasts/metabolism ; Dermis ; Inflammation/metabolism

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Skocz do pozycji: 10.

Tytuł:: Electric currents of 448 kHz upregulate anti-senescence pathways in human dermal fibroblasts.
Autorzy:: Toledano-Macías E; Bioelectromagnetic Laboratory, Instituto Ramón y Cajal de Investigación Sanitaria (IRYCIS), Madrid, Spain.
Martínez-Pascual MA; Bioelectromagnetic Laboratory, Instituto Ramón y Cajal de Investigación Sanitaria (IRYCIS), Madrid, Spain.
Hernández-Bule ML; Bioelectromagnetic Laboratory, Instituto Ramón y Cajal de Investigación Sanitaria (IRYCIS), Madrid, Spain.; Pokaż więcej
Źródło:: Journal of cosmetic dermatology [J Cosmet Dermatol] 2024 Feb; Vol. 23 (2), pp. 687-700. Date of Electronic Publication: 2023 Nov 09.
Typ publikacji:: Journal Article
MeSH Terms:: Skin*
Cellular Senescence*
Adult ; Infant, Newborn ; Humans ; Cell Proliferation ; Keratinocytes ; Fibroblasts ; Cells, Cultured

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Skocz do pozycji: 11.

Tytuł:: Non-Intrinsic, Systemic Mechanisms of Cellular Senescence.
Autorzy:: Schwartz RE; Department of Bioengineering, University of California Berkeley, Berkeley, CA 94720, USA.
Conboy IM; Department of Bioengineering, University of California Berkeley, Berkeley, CA 94720, USA.; Pokaż więcej
Źródło:: Cells [Cells] 2023 Dec 05; Vol. 12 (24). Date of Electronic Publication: 2023 Dec 05.
Typ publikacji:: Journal Article; Review; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
MeSH Terms:: Cellular Senescence*
Aging*
Animals ; Mice ; Reactive Oxygen Species/metabolism ; Inflammation

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Skocz do pozycji: 12.

Tytuł:: DNA damage response(DDR): a link between cellular senescence and human cytomegalovirus.
Autorzy:: Wu X; Affiliated Zhejiang Hospital, Zhejiang University School of Medicine, Hangzhou, 310030, China.
Zhou X; College of Life Science, Zhejiang Chinese Medical University, Hangzhou, 310053, China.
Wang S; Zhejiang Provincial Key Lab of Geriatrics & Geriatrics Institute of Zhejiang Province, Department of Geriatrics, Zhejiang Hospital, Hangzhou, 310030, China. .
Mao G; Affiliated Zhejiang Hospital, Zhejiang University School of Medicine, Hangzhou, 310030, China. .; Zhejiang Provincial Key Lab of Geriatrics & Geriatrics Institute of Zhejiang Province, Department of Geriatrics, Zhejiang Hospital, Hangzhou, 310030, China. .; Pokaż więcej
Źródło:: Virology journal [Virol J] 2023 Nov 01; Vol. 20 (1), pp. 250. Date of Electronic Publication: 2023 Nov 01.
Typ publikacji:: Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:: Cytomegalovirus*/genetics
Cellular Senescence*/genetics
Humans ; Signal Transduction ; Cell Cycle Checkpoints ; DNA Damage

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Skocz do pozycji: 13.

Tytuł:: Lipophilic Statins Eliminate Senescent Endothelial Cells by inducing Anoikis-Related Cell Death.
Autorzy:: Belakova B; Institute of Vascular Biology and Thrombosis Research, Center for Physiology and Pharmacology, Medical University of Vienna, 1090 Vienna, Austria.
Wedige NK; Institute of Vascular Biology and Thrombosis Research, Center for Physiology and Pharmacology, Medical University of Vienna, 1090 Vienna, Austria.
Awad EM; Institute of Vascular Biology and Thrombosis Research, Center for Physiology and Pharmacology, Medical University of Vienna, 1090 Vienna, Austria.; Institute of Specific Prophylaxis and Tropical Medicine, Center for Pathophysiology, Infectiology and Immunology, Medical University of Vienna, 1090 Vienna, Austria.
Hess S; Institute of Vascular Biology and Thrombosis Research, Center for Physiology and Pharmacology, Medical University of Vienna, 1090 Vienna, Austria.
Oszwald A; Institute of Vascular Biology and Thrombosis Research, Center for Physiology and Pharmacology, Medical University of Vienna, 1090 Vienna, Austria.; Department of Pathology, Medical University of Vienna, 1090 Vienna, Austria.
Fellner M; Institute of Vascular Biology and Thrombosis Research, Center for Physiology and Pharmacology, Medical University of Vienna, 1090 Vienna, Austria.
Khan SY; Institute of Vascular Biology and Thrombosis Research, Center for Physiology and Pharmacology, Medical University of Vienna, 1090 Vienna, Austria.; Department of Zoology, Government College University Lahore, Lahore 54000, Pakistan.
Resch U; Institute of Vascular Biology and Thrombosis Research, Center for Physiology and Pharmacology, Medical University of Vienna, 1090 Vienna, Austria.
Lipovac M; Karl Landsteiner Institute for Cell-Based Therapy in Gynecology, 2100 Korneuburg, Austria.
Šmejkal K; Department of Natural Drugs, Faculty of Pharmacy, Masaryk University, 612 00 Brno, Czech Republic.
Uhrin P; Institute of Vascular Biology and Thrombosis Research, Center for Physiology and Pharmacology, Medical University of Vienna, 1090 Vienna, Austria.
Breuss JM; Institute of Vascular Biology and Thrombosis Research, Center for Physiology and Pharmacology, Medical University of Vienna, 1090 Vienna, Austria.; Pokaż więcej
Źródło:: Cells [Cells] 2023 Dec 14; Vol. 12 (24). Date of Electronic Publication: 2023 Dec 14.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Cellular Senescence*
Hydroxymethylglutaryl-CoA Reductase Inhibitors*/pharmacology
Animals ; Humans ; Aged ; Endothelial Cells ; Anoikis ; Senotherapeutics

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Skocz do pozycji: 14.

Tytuł:: LEF1 isoforms regulate cellular senescence and aging.
Autorzy:: Jia M; Department of Computational and Systems Biology, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.; Joint Carnegie Mellon University-University of Pittsburgh Ph.D. Program in Computational Biology, Pittsburgh, Pennsylvania, USA.
Sayed K; Department of Computational and Systems Biology, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.; Department of Electrical & Computer Engineering and Computer Science, University of New Haven, West Haven, Connecticut, USA.
Kapetanaki MG; Department of Computational and Systems Biology, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.; Department of Epidemiology, University of Florida, Gainesville, Florida, USA.
Dion W; Aging Institute of UPMC, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.
Rosas L; Division of Pulmonary, Critical Care and Sleep Medicine, Department of Internal Medicine, The Ohio State University, Columbus, Ohio, USA.
Irfan S; Aging Institute of UPMC, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.
Valenzi E; Department of Rheumatology, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.
Mora AL; Division of Pulmonary, Critical Care and Sleep Medicine, Department of Internal Medicine, The Ohio State University, Columbus, Ohio, USA.
Lafyatis RA; Department of Rheumatology, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.
Rojas M; Division of Pulmonary, Critical Care and Sleep Medicine, Department of Internal Medicine, The Ohio State University, Columbus, Ohio, USA.
Zhu B; Aging Institute of UPMC, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.; Department of Medicine, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.; Pittsburgh Liver Research Center, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.
Benos PV; Department of Computational and Systems Biology, University of Pittsburgh, Pittsburgh, Pennsylvania, USA.; Joint Carnegie Mellon University-University of Pittsburgh Ph.D. Program in Computational Biology, Pittsburgh, Pennsylvania, USA.; Department of Epidemiology, University of Florida, Gainesville, Florida, USA.; Pokaż więcej
Źródło:: Aging cell [Aging Cell] 2023 Dec; Vol. 22 (12), pp. e14024. Date of Electronic Publication: 2023 Nov 13.
Typ publikacji:: Journal Article
MeSH Terms:: Aging*/genetics
Cellular Senescence*/genetics
Aged ; Animals ; Humans ; Mice ; Lung/pathology ; Lymphoid Enhancer-Binding Factor 1/genetics ; Lymphoid Enhancer-Binding Factor 1/metabolism ; Protein Isoforms/genetics

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Skocz do pozycji: 15.

Tytuł:: Biomarkers of cellular senescence and risk of death in humans.
Autorzy:: St Sauver JL; Department of Quantitative Health Sciences, Mayo Clinic, Rochester, Minnesota, USA.
Weston SA; Department of Quantitative Health Sciences, Mayo Clinic, Rochester, Minnesota, USA.
Atkinson EJ; Department of Quantitative Health Sciences, Mayo Clinic, Rochester, Minnesota, USA.
Mc Gree ME; Department of Quantitative Health Sciences, Mayo Clinic, Rochester, Minnesota, USA.
Mielke MM; Department of Epidemiology and Prevention, Wake Forest University School of Medicine, Winston-Salem, North Carolina, USA.
White TA; Robert and Arlene Kogod Center on Aging, Mayo Clinic, Rochester, Minnesota, USA.
Heeren AA; Robert and Arlene Kogod Center on Aging, Mayo Clinic, Rochester, Minnesota, USA.
Olson JE; Department of Quantitative Health Sciences, Mayo Clinic, Rochester, Minnesota, USA.
Rocca WA; Department of Quantitative Health Sciences, Mayo Clinic, Rochester, Minnesota, USA.; Department of Neurology, Mayo Clinic, Rochester, Minnesota, USA.; Women's Health Research Center, Mayo Clinic, Rochester, Minnesota, USA.
Palmer AK; Robert and Arlene Kogod Center on Aging, Mayo Clinic, Rochester, Minnesota, USA.; Division of Hospital Internal Medicine, Mayo Clinic, Rochester, Minnesota, USA.
Cummings SR; Departments of Medicine, Epidemiology and Biostatistics, University of California San Francisco, San Francisco, California, USA.; Research Institute, California Pacific Medical Center, San Francisco, California, USA.
Fielding RA; Nutrition, Exercise Physiology and Sarcopenia Laboratory, Jean Mayer USDA Human Nutrition Research Center on Aging, Tufts University, Boston, Massachusetts, USA.
Bielinski SJ; Department of Quantitative Health Sciences, Mayo Clinic, Rochester, Minnesota, USA.
LeBrasseur NK; Robert and Arlene Kogod Center on Aging, Mayo Clinic, Rochester, Minnesota, USA.; Paul F. Glenn Center for the Biology of Aging Research, Mayo Clinic, Rochester, Minnesota, USA.; Department of Physical Medicine and Rehabilitation, Mayo Clinic, Rochester, Minnesota, USA.; Pokaż więcej
Źródło:: Aging cell [Aging Cell] 2023 Dec; Vol. 22 (12), pp. e14006. Date of Electronic Publication: 2023 Oct 06.
Typ publikacji:: Journal Article
MeSH Terms:: Cellular Senescence*/genetics
Cytokines*/metabolism
Humans ; Aged ; Biomarkers ; Phenotype ; Chronic Disease

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Skocz do pozycji: 16.

Tytuł:: Form follows function: Nuclear morphology as a quantifiable predictor of cellular senescence.
Autorzy:: Belhadj J; Center for Pathobiochemistry & Genetics, Institute of Medical Genetics, Medical University of Vienna, Vienna, Austria.; Center for Pathobiochemistry & Genetics, Institute of Medical Chemistry and Pathobiochemistry, Medical University of Vienna, Vienna, Austria.
Surina S; Center for Pathobiochemistry & Genetics, Institute of Medical Genetics, Medical University of Vienna, Vienna, Austria.; Center for Pathobiochemistry & Genetics, Institute of Medical Chemistry and Pathobiochemistry, Medical University of Vienna, Vienna, Austria.; School of Medical Sciences, University of Campania Luigi Vanvitelli, Napoli, Italy.
Hengstschläger M; Center for Pathobiochemistry & Genetics, Institute of Medical Genetics, Medical University of Vienna, Vienna, Austria.
Lomakin AJ; Center for Pathobiochemistry & Genetics, Institute of Medical Genetics, Medical University of Vienna, Vienna, Austria.; Center for Pathobiochemistry & Genetics, Institute of Medical Chemistry and Pathobiochemistry, Medical University of Vienna, Vienna, Austria.; Pokaż więcej
Źródło:: Aging cell [Aging Cell] 2023 Dec; Vol. 22 (12), pp. e14012. Date of Electronic Publication: 2023 Oct 16.
Typ publikacji:: Journal Article
MeSH Terms:: Cellular Senescence*/physiology
Phenotype ; Biomarkers/metabolism

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Skocz do pozycji: 17.

Tytuł:: Targeting the redox system for cardiovascular regeneration in aging.
Autorzy:: Allemann MS; Center for Molecular Cardiology, University of Zurich, Schlieren, Switzerland.; Department of Internal Medicine, Cantonal Hospital Baden, Baden, Switzerland.
Lee P; Center for Molecular Cardiology, University of Zurich, Schlieren, Switzerland.; Department of Internal Medicine, Cantonal Hospital Baden, Baden, Switzerland.
Beer JH; Center for Molecular Cardiology, University of Zurich, Schlieren, Switzerland.; Department of Internal Medicine, Cantonal Hospital Baden, Baden, Switzerland.
Saeedi Saravi SS; Center for Translational and Experimental Cardiology, Department of Cardiology, University Hospital Zurich, University of Zurich, Schlieren, Switzerland.; Pokaż więcej
Źródło:: Aging cell [Aging Cell] 2023 Dec; Vol. 22 (12), pp. e14020. Date of Electronic Publication: 2023 Nov 13.
Typ publikacji:: Journal Article; Review
MeSH Terms:: Cellular Senescence*
Cardiovascular System*
Cell Differentiation ; Oxidation-Reduction

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Skocz do pozycji: 18.

Tytuł:: Carnitine acetyltransferase deficiency mediates mitochondrial dysfunction-induced cellular senescence in dermal fibroblasts.
Autorzy:: Song MJ; Department of Dermatology, Seoul National University College of Medicine, Seoul, Republic of Korea.; Department of Biomedical Sciences, Seoul National University Graduate School, Seoul, Republic of Korea.; Institute of Human-Environment Interface Biology, Medical Research Center, Seoul National University, Seoul, Republic of Korea.
Park CH; Department of Dermatology, Seoul National University College of Medicine, Seoul, Republic of Korea.; Institute of Human-Environment Interface Biology, Medical Research Center, Seoul National University, Seoul, Republic of Korea.
Kim H; Department of Dermatology, Seoul National University College of Medicine, Seoul, Republic of Korea.; Department of Biomedical Sciences, Seoul National University Graduate School, Seoul, Republic of Korea.; Institute of Human-Environment Interface Biology, Medical Research Center, Seoul National University, Seoul, Republic of Korea.
Han S; Department of Biomedical Sciences, Seoul National University Graduate School, Seoul, Republic of Korea.; Institute of Human-Environment Interface Biology, Medical Research Center, Seoul National University, Seoul, Republic of Korea.
Lee SH; Department of Dermatology, Seoul National University College of Medicine, Seoul, Republic of Korea.; Institute of Human-Environment Interface Biology, Medical Research Center, Seoul National University, Seoul, Republic of Korea.
Lee DH; Department of Dermatology, Seoul National University College of Medicine, Seoul, Republic of Korea.; Institute of Human-Environment Interface Biology, Medical Research Center, Seoul National University, Seoul, Republic of Korea.
Chung JH; Department of Dermatology, Seoul National University College of Medicine, Seoul, Republic of Korea.; Department of Biomedical Sciences, Seoul National University Graduate School, Seoul, Republic of Korea.; Institute of Human-Environment Interface Biology, Medical Research Center, Seoul National University, Seoul, Republic of Korea.; Institute on Aging, Seoul National University, Seoul, Republic of Korea.; Pokaż więcej
Źródło:: Aging cell [Aging Cell] 2023 Nov; Vol. 22 (11), pp. e14000. Date of Electronic Publication: 2023 Oct 13.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Carnitine O-Acetyltransferase*/metabolism
Cellular Senescence*/physiology
Animals ; Mice ; Humans ; Aged ; Mitochondria/metabolism ; NF-kappa B/metabolism ; Inflammation/metabolism ; Fibroblasts/metabolism
SCR Disease Name:: Carnitine Acetyltransferase Deficiency

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Skocz do pozycji: 19.

Tytuł:: Iron overload induces cerebral endothelial senescence in aged mice and in primary culture in a sex-dependent manner.
Autorzy:: Noh B; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.
Blasco-Conesa MP; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.
Rahman SM; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.
Monga S; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.
Ritzel R; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.
Guzman G; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.
Lai YJ; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.; Solomont School of Nursing, Zuckerberg College of Health Sciences, University of Massachusetts Lowell, Lowell, Massachusetts, USA.
Ganesh BP; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.
Urayama A; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.
McCullough LD; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.
Moruno-Manchon JF; Department of Neurology, McGovern Medical School at the University of Texas Health Science Center at Houston, Houston, Texas, USA.; Pokaż więcej
Źródło:: Aging cell [Aging Cell] 2023 Nov; Vol. 22 (11), pp. e13977. Date of Electronic Publication: 2023 Sep 07.
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:: Cellular Senescence*/physiology
Iron Overload*
Mice ; Humans ; Animals ; Male ; Female ; Aged ; Endothelial Cells ; Aging ; Iron ; Receptors, Cell Surface

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Skocz do pozycji: 20.

Tytuł:: Nectin-4 regulates cellular senescence-associated enlargement of cell size.
Autorzy:: Katasho R; Department of Biology, Graduate School of Science, Kobe University, 1-1 Rokkodai-Cho, Nada-Ku, Kobe, 657-8501, Japan.
Nagano T; Biosignal Research Center, Kobe University, 1-1 Rokkodai-Cho, Nada-Ku, Kobe, 657-8501, Japan.
Iwasaki T; Department of Biology, Graduate School of Science, Kobe University, 1-1 Rokkodai-Cho, Nada-Ku, Kobe, 657-8501, Japan.; Biosignal Research Center, Kobe University, 1-1 Rokkodai-Cho, Nada-Ku, Kobe, 657-8501, Japan.
Kamada S; Department of Biology, Graduate School of Science, Kobe University, 1-1 Rokkodai-Cho, Nada-Ku, Kobe, 657-8501, Japan. .; Biosignal Research Center, Kobe University, 1-1 Rokkodai-Cho, Nada-Ku, Kobe, 657-8501, Japan. .; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2023 Dec 07; Vol. 13 (1), pp. 21602. Date of Electronic Publication: 2023 Dec 07.
Typ publikacji:: Journal Article
MeSH Terms:: Cell Size*
Cellular Senescence*
Nectins/metabolism ; Oxidative Stress ; Phosphatidylinositol 3-Kinases/metabolism ; Humans

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