SevofluranePostconditioning Ameliorates Neuronal Migration Disorder Through Reelin/Dab1 and Improves Long-term Cognition in Neonatal Rats After Hypoxic-Ischemic Injury
Diabetes impairs the protective effects of sevofluranepostconditioning in the myocardium subjected to ischemia/ reperfusion injury in rats: important role of Drp1
Diabetes impairs the protective effects of sevofluranepostconditioning in the myocardium subjected to ischemia/ reperfusion injury in rats: important role of Drp1
SevofluranePostconditioning Inhibits Autophagy Through Activation of the Extracellular Signal-Regulated Kinase Cascade, Alleviating Hypoxic-Ischemic Brain Injury in Neonatal Rats
Sevofluranepostconditioning alleviates hypoxia-reoxygenation injury of cardiomyocytes by promoting mitochondrial autophagy through the HIF-1/BNIP3 signaling pathway
Remote Ischaemic Preconditioning and SevofluranePostconditioning Synergistically Protect Rats from Myocardial Injury Induced by Ischemia and Reperfusion Partly via Inhibition TLR4/MyD88/NF-κB Signaling Pathway
Downregulation of Na+/Ca2+ Exchanger Isoform 1 Protects Isolated Hearts by SevofluranePostconditioning but Not by Delayed Remote Ischemic Preconditioning in Rats
SevofluranePostconditioning-Induced Anti-Inflammation via Inhibition of the Toll-Like Receptor-4/Nuclear Factor Kappa B Pathway Contributes to Neuroprotection against Transient Global Cerebral Ischemia in Rats
Activation of Akt and cardioprotection against reperfusion injury are maximal with only five minutes of sevofluranepostconditioning in isolated rat hearts
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