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Wyświetlanie 1-10 z 10
Tytuł :
GSK3β and MCL-1 mediate cardiomyocyte apoptosis in response to high glucose.
Autorzy :
Su D; Department of Genetics, Center for Genetics, Health Department, National Research Institute for Family Planning, 12, Dahuisi Road, Haidian, Beijing, 100081, China.; Graduate School, Peking Union Medical College, Beijing, China.
Zhao J; Department of Cardiology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, 450052, China.
Hu S; Hongqi Hospital of Mudanjiang Medical College, Mudanjiang, Heilongjiang, China.
Guan L; Department of Genetics, Center for Genetics, Health Department, National Research Institute for Family Planning, 12, Dahuisi Road, Haidian, Beijing, 100081, China.
Li Q; Department of Genetics, Center for Genetics, Health Department, National Research Institute for Family Planning, 12, Dahuisi Road, Haidian, Beijing, 100081, China.
Shi C; Department of Genetics, Center for Genetics, Health Department, National Research Institute for Family Planning, 12, Dahuisi Road, Haidian, Beijing, 100081, China.
Ma X; Department of Genetics, Center for Genetics, Health Department, National Research Institute for Family Planning, 12, Dahuisi Road, Haidian, Beijing, 100081, China. .; Graduate School, Peking Union Medical College, Beijing, China. .
Gou J; Department of Cardiology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, 450052, China. .
Zhou Y; Hongqi Hospital of Mudanjiang Medical College, Mudanjiang, Heilongjiang, China. .
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Źródło :
Histochemistry and cell biology [Histochem Cell Biol] 2019 Sep; Vol. 152 (3), pp. 217-225. Date of Electronic Publication: 2019 Jun 14.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Blood Glucose/*metabolism
Diabetes, Gestational/*metabolism
Glycogen Synthase Kinase 3 beta/*metabolism
Heart Defects, Congenital/*metabolism
Myeloid Cell Leukemia Sequence 1 Protein/*metabolism
Myocytes, Cardiac/*metabolism
Animals ; Cells, Cultured ; Diabetes Mellitus, Experimental/metabolism ; Diabetes Mellitus, Experimental/pathology ; Diabetes, Gestational/pathology ; Female ; Heart Defects, Congenital/pathology ; Male ; Myocytes, Cardiac/pathology ; Phosphorylation ; Pregnancy ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł :
Role of GAB1/PI3K/AKT signaling high glucose-induced cardiomyocyte apoptosis.
Autorzy :
Su D; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Zhou Y; Hongqi Hospital of Mudanjiang Medical College, Mudanjiang, Heilongjiang Province, China.
Hu S; Hongqi Hospital of Mudanjiang Medical College, Mudanjiang, Heilongjiang Province, China.
Guan L; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Shi C; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Wang Q; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Chen Y; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Lu C; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Li Q; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Ma X; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China. Electronic address: .
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Źródło :
Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie [Biomed Pharmacother] 2017 Sep; Vol. 93, pp. 1197-1204.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*physiology
Glucose/*adverse effects
Myocytes, Cardiac/*metabolism
Phosphatidylinositol 3-Kinases/*metabolism
Phosphoproteins/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Signal Transduction/*physiology
Animals ; Apoptosis/drug effects ; Cell Line ; Chromones/pharmacology ; Diabetes Mellitus, Experimental/metabolism ; Diabetes, Gestational/metabolism ; Female ; Morpholines/pharmacology ; Phosphorylation/drug effects ; Phosphorylation/physiology ; Pregnancy ; Rats ; Rats, Sprague-Dawley ; Signal Transduction/drug effects
Czasopismo naukowe
Tytuł :
ROCK1/p53/NOXA signaling mediates cardiomyocyte apoptosis in response to high glucose in vitro and vivo.
Autorzy :
Su D; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Guan L; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Gao Q; Linyi Academy of Technology Cooperation and Application, Linyi, Shandong Province, China.
Li Q; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Shi C; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Liu Y; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Sun L; Fang Cheng Gang Senior Middle School, Guangxi Zhuang Autonomous, China.
Lu C; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China.
Ma X; Department of Genetics, National Research Institute for Family Planning, Health Department, Beijing, China. Electronic address: .
Zhao J; Department of Cardiology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou 450007, China. Electronic address: .
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Źródło :
Biochimica et biophysica acta. Molecular basis of disease [Biochim Biophys Acta Mol Basis Dis] 2017 Apr; Vol. 1863 (4), pp. 936-946. Date of Electronic Publication: 2017 Jan 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Glucose/*pharmacology
Myocytes, Cardiac/*metabolism
Proto-Oncogene Proteins c-bcl-2/*metabolism
Signal Transduction/*drug effects
Tumor Suppressor Protein p53/*biosynthesis
Up-Regulation/*drug effects
rho-Associated Kinases/*biosynthesis
Animals ; Female ; Glucose/metabolism ; Heart Defects, Congenital/metabolism ; Heart Defects, Congenital/pathology ; Heart Ventricles/metabolism ; Heart Ventricles/pathology ; Male ; Myocytes, Cardiac/pathology ; Pregnancy ; Pregnancy in Diabetics/metabolism ; Pregnancy in Diabetics/pathology ; Rats
Czasopismo naukowe
Tytuł :
Down-regulation of GJA3 is associated with lens epithelial cell apoptosis and age-related cataract.
Autorzy :
Su D; Department of Genetics, National Research Institute for Family Planning, Beijing, China.
Hu S; Department of Ophthalmology, Hongqi Hospital of Mudanjiang Medical College, Mudanjiang, Heilongjiang, China.
Guan L; Department of Genetics, National Research Institute for Family Planning, Beijing, China.
Wu X; Department of Genetics, National Research Institute for Family Planning, Beijing, China.
Shi C; Department of Genetics, National Research Institute for Family Planning, Beijing, China. Electronic address: .
Yang X; Department of Ophthalmology, Hongqi Hospital of Mudanjiang Medical College, Mudanjiang, Heilongjiang, China. Electronic address: .
Ma X; Department of Genetics, National Research Institute for Family Planning, Beijing, China. Electronic address: .
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Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2017 Feb 26; Vol. 484 (1), pp. 159-164. Date of Electronic Publication: 2017 Jan 12.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis*
Down-Regulation*
Aging/*pathology
Cataract/*metabolism
Connexins/*metabolism
Lens, Crystalline/*pathology
Animals ; Epithelial Cells/metabolism ; Epithelial Cells/pathology ; Humans ; Hydrogen Peroxide/metabolism ; Lens, Crystalline/metabolism ; Male ; Mice ; Mice, Inbred BALB C
Czasopismo naukowe
    Wyświetlanie 1-10 z 10

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