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Wyszukujesz frazę ""Shimozato, O"" wg kryterium: Autor


Tytuł:
Transcriptomic analysis reveals high ITGB1 expression as a predictor for poor prognosis of pancreatic cancer.
Autorzy:
Iwatate Y; Division of Hepato-Biliary-Pancreatic Surgery, Chiba Cancer Center, Chiba, Japan.
Yokota H; Department of Diagnostic Radiology and Radiation Oncology, Graduate School of Medicine, Chiba University, Chiba, Japan.
Hoshino I; Division of Gastroenterological Surgery, Chiba Cancer Center, Chiba, Japan.
Ishige F; Division of Hepato-Biliary-Pancreatic Surgery, Chiba Cancer Center, Chiba, Japan.
Kuwayama N; Division of Gastroenterological Surgery, Chiba Cancer Center, Chiba, Japan.
Itami M; Division of Clinical Pathology, Chiba Cancer Center, Chiba, Japan.
Mori Y; Graduate School of Engineering, Faculty of Engineering, Chiba University, Chiba, Japan.
Chiba S; Division of Hepato-Biliary-Pancreatic Surgery, Chiba Cancer Center, Chiba, Japan.
Arimitsu H; Division of Hepato-Biliary-Pancreatic Surgery, Chiba Cancer Center, Chiba, Japan.
Yanagibashi H; Division of Hepato-Biliary-Pancreatic Surgery, Chiba Cancer Center, Chiba, Japan.
Takayama W; Division of Hepato-Biliary-Pancreatic Surgery, Chiba Cancer Center, Chiba, Japan.
Uno T; Department of Diagnostic Radiology and Radiation Oncology, Graduate School of Medicine, Chiba University, Chiba, Japan.
Lin J; Laboratory of Cancer Genetics, Chiba Cancer Center Research Institute, Chiba Cancer Center, Chiba, Japan.
Nakamura Y; Laboratory of Cancer Genetics, Chiba Cancer Center Research Institute, Chiba Cancer Center, Chiba, Japan.
Tatsumi Y; Laboratory of Cancer Genetics, Chiba Cancer Center Research Institute, Chiba Cancer Center, Chiba, Japan.
Shimozato O; Laboratory of Cancer Genetics, Chiba Cancer Center Research Institute, Chiba Cancer Center, Chiba, Japan.
Nagase H; Laboratory of Cancer Genetics, Chiba Cancer Center Research Institute, Chiba Cancer Center, Chiba, Japan.
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Źródło:
PloS one [PLoS One] 2022 Jun 01; Vol. 17 (6), pp. e0268630. Date of Electronic Publication: 2022 Jun 01 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Pancreatic Neoplasms*/diagnosis
Pancreatic Neoplasms*/genetics
Pancreatic Neoplasms*/metabolism
Transcriptome*
Gene Expression Profiling ; Humans ; Prognosis ; Pancreatic Neoplasms
Czasopismo naukowe
Tytuł:
Depletion of runt-related transcription factor 2 (RUNX2) enhances SAHA sensitivity of p53-mutated pancreatic cancer cells through the regulation of mutant p53 and TAp63.
Autorzy:
Ogata T; Laboratory of DNA Damage Signaling, Chiba Cancer Center Research Institute, Chiba, Japan.
Nakamura M; Laboratory of DNA Damage Signaling, Chiba Cancer Center Research Institute, Chiba, Japan.
Sang M; Laboratory of DNA Damage Signaling, Chiba Cancer Center Research Institute, Chiba, Japan.; Department of Regenerative Medicine, Graduate School of Medicine, University of Toyama, Toyama, Japan.
Yoda H; Laboratory of Cancer Genetics, Chiba Cancer Center Research Institute, Chiba, Japan.
Hiraoka K; Laboratory of Cancer Genetics, Chiba Cancer Center Research Institute, Chiba, Japan.
Yin D; Research Center, Fourth Hospital of Hebei Medical University, Shijiazhuang, Hebei, P.R. China.
Sang M; Research Center, Fourth Hospital of Hebei Medical University, Shijiazhuang, Hebei, P.R. China.
Shimozato O; Laboratory of DNA Damage Signaling, Chiba Cancer Center Research Institute, Chiba, Japan.
Ozaki T; Laboratory of DNA Damage Signaling, Chiba Cancer Center Research Institute, Chiba, Japan.
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Źródło:
PloS one [PLoS One] 2017 Jul 03; Vol. 12 (7), pp. e0179884. Date of Electronic Publication: 2017 Jul 03 (Print Publication: 2017).
Typ publikacji:
Journal Article
MeSH Terms:
Gene Expression Regulation, Neoplastic*
Genes, p53*
Mutation*
Core Binding Factor Alpha 1 Subunit/*physiology
Hydroxamic Acids/*pharmacology
Pancreatic Neoplasms/*pathology
Transcription Factors/*genetics
Tumor Suppressor Proteins/*genetics
Cell Line, Tumor ; Core Binding Factor Alpha 1 Subunit/genetics ; Down-Regulation ; Gene Knockdown Techniques ; Humans ; Up-Regulation ; Vorinostat
Czasopismo naukowe
Tytuł:
Administration of DNA Encoding the Interleukin-27 Gene Augments Antitumour Responses through Non-adaptive Immunity.
Autorzy:
Li Q; Division of Pathology and Cell Therapy, Chiba Cancer Center Research Institute, Chuo-ku, Chiba, Japan.; Department of Immunology, Hebei Medical University, Shijiazhuang, China.; Cell Therapy Center, The 1st Hospital of Hebei Medical University, Shijiazhuang, China.; Department of Molecular Biology and Oncology, Graduate School of Medicine, Chiba University, Chuo-ku, Chiba, Japan.
Sato A; Division of Pathology and Cell Therapy, Chiba Cancer Center Research Institute, Chuo-ku, Chiba, Japan.; Department of Molecular Biology and Oncology, Graduate School of Medicine, Chiba University, Chuo-ku, Chiba, Japan.; Department of Biochemistry and Genetics, Graduate School of Medicine, Chiba University, Chuo-ku, Chiba, Japan.
Shimozato O; Division of Pathology and Cell Therapy, Chiba Cancer Center Research Institute, Chuo-ku, Chiba, Japan.
Shingyoji M; Department of Thoracic Diseases, Chiba Cancer Center, Chuo-ku, Chiba, Japan.
Tada Y; Department of Respirology, Graduate School of Medicine, Chiba University, Chuo-ku, Chiba, Japan.
Tatsumi K; Department of Respirology, Graduate School of Medicine, Chiba University, Chuo-ku, Chiba, Japan.
Shimada H; Department of Surgery, School of Medicine, Toho University, Tokyo, Japan.
Hiroshima K; Department of Pathology, Tokyo Women's Medical University Yachiyo Medical Center, Owada-Shinden, Yachiyo, Japan.
Tagawa M; Division of Pathology and Cell Therapy, Chiba Cancer Center Research Institute, Chuo-ku, Chiba, Japan.; Department of Molecular Biology and Oncology, Graduate School of Medicine, Chiba University, Chuo-ku, Chiba, Japan.
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Źródło:
Scandinavian journal of immunology [Scand J Immunol] 2015 Oct; Vol. 82 (4), pp. 320-7.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antigens, Neoplasm/*immunology
Cancer Vaccines/*therapeutic use
DNA/*therapeutic use
Interleukin-27/*genetics
Neoplasms/*therapy
Vaccines, DNA/*therapeutic use
beta-Galactosidase/*immunology
Animals ; Antibodies/immunology ; Antigens, Neoplasm/genetics ; CD8-Positive T-Lymphocytes/immunology ; Cancer Vaccines/genetics ; Cardiotoxins/administration & dosage ; DNA/administration & dosage ; DNA/genetics ; Interleukin-12/genetics ; Killer Cells, Natural/immunology ; Mice ; Mice, Inbred BALB C ; Mice, Inbred DBA ; Perforin/biosynthesis ; Phosphorylation ; STAT3 Transcription Factor/metabolism ; Vaccines, DNA/administration & dosage ; Vaccines, DNA/immunology ; beta-Galactosidase/genetics
Czasopismo naukowe
Tytuł:
Receptor-type protein tyrosine phosphatase κ directly dephosphorylates CD133 and regulates downstream AKT activation.
Autorzy:
Shimozato O; Division of Biochemistry and Molecular Carcinogenesis, Chiba Cancer Center Research Institute, Chiba, Japan.
Waraya M; Division of Biochemistry and Molecular Carcinogenesis, Chiba Cancer Center Research Institute, Chiba, Japan.
Nakashima K; Division of Biochemistry and Molecular Carcinogenesis, Chiba Cancer Center Research Institute, Chiba, Japan.
Souda H; Department of Gastrointestinal Surgery, Chiba Cancer Center Hospital, Chiba, Japan.
Takiguchi N; Department of Gastrointestinal Surgery, Chiba Cancer Center Hospital, Chiba, Japan.
Yamamoto H; Department of Gastrointestinal Surgery, Chiba Cancer Center Hospital, Chiba, Japan.
Takenobu H; Division of Biochemistry and Molecular Carcinogenesis, Chiba Cancer Center Research Institute, Chiba, Japan.
Uehara H; Division of Biochemistry and Molecular Carcinogenesis, Chiba Cancer Center Research Institute, Chiba, Japan.
Ikeda E; Division of Biochemistry and Molecular Carcinogenesis, Chiba Cancer Center Research Institute, Chiba, Japan.
Matsushita S; Laboratory of DNA Damage Signaling, Chiba Cancer Center Research Institute, Chiba, Japan.
Kubo N; Laboratory of DNA Damage Signaling, Chiba Cancer Center Research Institute, Chiba, Japan.
Nakagawara A; Division of Biochemistry and Innovative Cancer Therapeutics, Chiba Cancer Center Research Institute, Chiba, Japan.
Ozaki T; Laboratory of DNA Damage Signaling, Chiba Cancer Center Research Institute, Chiba, Japan.
Kamijo T; 1] Division of Biochemistry and Molecular Carcinogenesis, Chiba Cancer Center Research Institute, Chiba, Japan [2] Research Institute for Clinical Oncology, Saitama Cancer Center, Saitama, Japan.
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Źródło:
Oncogene [Oncogene] 2015 Apr 09; Vol. 34 (15), pp. 1949-60. Date of Electronic Publication: 2014 Jun 02.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antigens, CD/*metabolism
Colonic Neoplasms/*metabolism
Glycoproteins/*metabolism
Peptides/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Receptor-Like Protein Tyrosine Phosphatases, Class 2/*metabolism
AC133 Antigen ; Animals ; Caco-2 Cells ; Cell Proliferation/physiology ; Colonic Neoplasms/enzymology ; Colonic Neoplasms/pathology ; HT29 Cells ; Heterografts ; Humans ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Neoplastic Stem Cells/metabolism ; Neoplastic Stem Cells/pathology ; Phosphorylation ; Signal Transduction ; beta Catenin/metabolism
Czasopismo naukowe
Tytuł:
Fas ligand DNA enhances a vaccination effect by coadministered DNA encoding a tumor antigen through augmenting production of antibody against the tumor antigen.
Autorzy:
Zhong B; Department of Hematology, Fourth Hospital of Hebei Medical University, Shijiazhuang 050035, China ; Division of Pathology and Cell Therapy, Chiba Cancer Center Research Institute, Chiba 260-8717, Japan.
Ma G; Department of Hematology, Fourth Hospital of Hebei Medical University, Shijiazhuang 050035, China.
Sato A; Division of Pathology and Cell Therapy, Chiba Cancer Center Research Institute, Chiba 260-8717, Japan ; Department of Molecular Biology and Oncology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan.
Shimozato O; Division of Pathology and Cell Therapy, Chiba Cancer Center Research Institute, Chiba 260-8717, Japan ; Laboratory of DNA Damage Signaling, Chiba Cancer Center Research Institute, Chiba 260-8717, Japan.
Liu H; Division of Pathology and Cell Therapy, Chiba Cancer Center Research Institute, Chiba 260-8717, Japan.
Li Q; Division of Pathology and Cell Therapy, Chiba Cancer Center Research Institute, Chiba 260-8717, Japan ; Department of Molecular Biology and Oncology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan ; Department of Immunology, Hebei Medical University, Shijiazhuang 050017, China ; Cell Therapy Center, 1st Hospital of Hebei Medical University, Shijiazhuang 050031, China.
Shingyoji M; Department of Thoracic Diseases, Chiba Cancer Center, Chiba 260-8717, Japan.
Tada Y; Department of Respirology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan.
Tatsumi K; Department of Respirology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan.
Shimada H; Department of Surgery, School of Medicine, Toho University, Tokyo 143-8541, Japan.
Hiroshima K; Department of Pathology, Tokyo Women's Medical University Yachiyo Medical Center, Yachiyo 276-8524, Japan.
Tagawa M; Division of Pathology and Cell Therapy, Chiba Cancer Center Research Institute, Chiba 260-8717, Japan ; Department of Molecular Biology and Oncology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan.
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Źródło:
Journal of immunology research [J Immunol Res] 2015; Vol. 2015, pp. 743828. Date of Electronic Publication: 2015 Feb 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antibodies/*immunology
Antibody Formation/*immunology
Antigens, Neoplasm/*genetics
Cancer Vaccines/*genetics
Fas Ligand Protein/*genetics
Vaccines, DNA/*genetics
Animals ; Antigens, Neoplasm/administration & dosage ; Antigens, Neoplasm/immunology ; Cancer Vaccines/immunology ; Cell Line, Tumor ; Disease Models, Animal ; Fas Ligand Protein/immunology ; Immunization ; Mice ; Neoplasms/immunology ; Neoplasms/therapy ; T-Cell Antigen Receptor Specificity/immunology ; T-Lymphocytes/immunology ; T-Lymphocytes/metabolism ; Transgenes ; Vaccines, DNA/administration & dosage ; Vaccines, DNA/immunology
Czasopismo naukowe
Tytuł:
CD133 suppresses neuroblastoma cell differentiation via signal pathway modification.
Autorzy:
Takenobu H; Division of Biochemistry and Molecular Carcinogenesis,Chiba Cancer Center Research Institute, Chiba, Japan.
Shimozato O
Nakamura T
Ochiai H
Yamaguchi Y
Ohira M
Nakagawara A
Kamijo T
Pokaż więcej
Źródło:
Oncogene [Oncogene] 2011 Jan 06; Vol. 30 (1), pp. 97-105. Date of Electronic Publication: 2010 Sep 06.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antigens, CD/*metabolism
Glycoproteins/*metabolism
Neuroblastoma/*metabolism
Neuroblastoma/*pathology
Peptides/*metabolism
AC133 Antigen ; Animals ; Cell Differentiation/physiology ; Cell Growth Processes/physiology ; HEK293 Cells ; Humans ; Mice ; Mice, Nude ; Phosphorylation ; Proto-Oncogene Proteins c-ret/metabolism ; Signal Transduction
Czasopismo naukowe

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