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    Wyświetlanie 1-2 z 2
    Tytuł:
    RB depletion is required for the continuous growth of tumors initiated by loss of RB.
    Autorzy:
    Doan A; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
    Arand J; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.; Center of Anatomy and Cell Biology, Department of Cell and Developmental Biology, Medical University of Vienna, Vienna, Austria.
    Gong D; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.; Department of Bioengineering, Stanford University, Stanford, California, United States of America.
    Drainas AP; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
    Shue YT; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
    Lee MC; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
    Zhang S; Department of Biology, Stanford University, Stanford, California, United States of America.
    Walter DM; Department of Cancer Biology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.; Abramson Family Cancer Research Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
    Chaikovsky AC; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
    Feldser DM; Department of Cancer Biology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.; Abramson Family Cancer Research Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania, United States of America.
    Vogel H; Department of Pathology, Stanford University, Stanford, California, United States of America.
    Dow LE; Sandra and Edward Meyer Cancer Center, Weill Cornell Medicine, New York, New York, United States of America.; Department of Biochemistry, Weill Cornell Medicine, New York, New York, United States of America.; Department of Medicine, Weill Cornell Medicine, New York, New York, United States of America.
    Skotheim JM; Department of Biology, Stanford University, Stanford, California, United States of America.
    Sage J; Department of Pediatrics, Stanford University, Stanford, California, United States of America.; Department of Genetics, Stanford University, Stanford, California, United States of America.
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    Źródło:
    PLoS genetics [PLoS Genet] 2021 Dec 08; Vol. 17 (12), pp. e1009941. Date of Electronic Publication: 2021 Dec 08 (Print Publication: 2021).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Pituitary Neoplasms/*genetics
    Retinoblastoma Binding Proteins/*genetics
    Thyroid Neoplasms/*genetics
    Animals ; Cell Cycle Checkpoints/genetics ; Cell Line, Tumor ; Disease Models, Animal ; E2F Transcription Factors/metabolism ; Female ; Gene Expression Regulation, Neoplastic ; Gene Knockdown Techniques ; Humans ; Male ; Mice ; Mice, Transgenic ; NIH 3T3 Cells ; Pituitary Neoplasms/pathology ; RNA, Small Interfering/metabolism ; Thyroid Neoplasms/pathology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Embryonic stem cell differentiation requires full length Chd1.
    Autorzy:
    Piatti P; Division of Molecular Biology, Biocenter, Medical University of Innsbruck, Austria.
    Lim CY; Epithelial Epigenetics and Development Laboratory, Institute of Medical Biology, A*Star, Singapore.
    Nat R; Institute for Neuroscience, Medical University of Innsbruck, Austria.
    Villunger A; Division of Developmental Immunology, Biocenter, Medical University of Innsbruck, Austria.
    Geley S; Division of Molecular Pathophysiology, Biocenter, Medical University of Innsbruck, Austria.
    Shue YT; Epithelial Epigenetics and Development Laboratory, Institute of Medical Biology, A*Star, Singapore.
    Soratroi C; Division of Developmental Immunology, Biocenter, Medical University of Innsbruck, Austria.
    Moser M; Department of Molecular Medicine, Max Planck Institute of Biochemistry, Martinsried, Germany.
    Lusser A; Division of Molecular Biology, Biocenter, Medical University of Innsbruck, Austria.
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    Źródło:
    Scientific reports [Sci Rep] 2015 Jan 26; Vol. 5, pp. 8007. Date of Electronic Publication: 2015 Jan 26.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Embryonic Development*
    Embryonic Stem Cells*
    Cell Differentiation/*genetics
    DNA-Binding Proteins/*genetics
    Animals ; Chromatin Assembly and Disassembly ; DNA-Binding Proteins/biosynthesis ; Gene Expression Regulation, Developmental ; Mice ; Serine/genetics
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-2 z 2

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