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Tytuł:
YES-associated protein-regulated Smad7 worsen epithelial barrier injury of chronic sinusitis with nasal polyps.
Autorzy:
Jiang X; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Shu L; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Liu Y; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Shen Y; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Ke X; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Liu J; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
Yang Y; Department of Otorhinolaryngology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People's Republic of China.
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Źródło:
Immunity, inflammation and disease [Immun Inflamm Dis] 2023 Jun; Vol. 11 (6), pp. e907.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Nasal Polyps*
Sinusitis*
YAP-Signaling Proteins*/genetics
Smad7 Protein*/genetics
Humans ; Cadherins ; Chronic Disease ; Transcription Factors/genetics ; Transforming Growth Factor beta1
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The translational potential of miR-26 in atherosclerosis and development of agents for its target genes ACC1/2, COL1A1, CPT1A, FBP1, DGAT2, and SMAD7.
Autorzy:
Chen W; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China.
Wu X; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China.
Hu J; Department of Endocrinology, The Affiliated Hospital of Qingdao University, Qingdao, 266000, Shandong, China.
Liu X; Department of Gastrointestinal Surgery, The Affiliated Hospital of Qingdao University, Qingdao, 266000, Shandong, China.
Guo Z; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China.
Wu J; Department of Cardiology, The Second Affiliated Hospital, Hengyang Medical School, University of South China, Key Laboratory of Heart Failure Prevention & Treatment of Hengyang, Clinical Medicine Research Center of Arteriosclerotic Disease of Hunan Province, Hengyang, 421001, Hunan, China.
Shao Y; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China.
Hao M; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China.
Zhang S; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China.
Hu W; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China.; Department of Endocrinology, Qilu Hospital (Qingdao), Cheeloo College of Medicine, Shandong University, Qingdao, 266000, Shandong, China.
Wang Y; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China.
Zhang M; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China.
Zhu M; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China.; Department of Neurosurgery, The Affiliated Hospital of Qingdao University, Qingdao, 266071, Shandong, China.
Wang C; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China. .
Wu Y; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China. .
Wang J; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China. .
Xing D; Cancer Institute, Department of Orthopaedics, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao Cancer Institute, Qingdao, 266071, Shandong, China. xdm_.; School of Life Sciences, Tsinghua University, Beijing, 100084, China. xdm_.
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Źródło:
Cardiovascular diabetology [Cardiovasc Diabetol] 2024 Jan 09; Vol. 23 (1), pp. 21. Date of Electronic Publication: 2024 Jan 09.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
MicroRNAs*/genetics
Atherosclerosis*/genetics
Humans ; ADP-Ribosylation Factors ; Carotid Intima-Media Thickness ; Diacylglycerol O-Acyltransferase ; Proprotein Convertase 9 ; Smad7 Protein
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Smad7 in the hippocampus contributes to memory impairment in aged mice after anesthesia and surgery.
Autorzy:
Liu C; Department of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China.; Laboratory of Anesthesia and Critical Care Medicine, National-Local Joint Engineering Research Center of Translational Medicine of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China.
Wu J; Department of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China.; Laboratory of Anesthesia and Critical Care Medicine, National-Local Joint Engineering Research Center of Translational Medicine of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China.
Li M; Department of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China.
Gao R; Department of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China.
Zhang X; Department of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China.
Ye-Lehmann S; Diseases and Hormones of the Nervous System, University of Paris-Scalay Bicêtre Hosptial Bât. Grégory Pincus, 80 Rue du Gal Leclerc, Le Kremlin Bicêtre, 94276, CEDEX, Paris, France.
Song J; Monash Biomedicine Discovery Institute and Monash Data Futures Institute, Monash University, VIC, Melbourne, Australia.
Zhu T; Department of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China. .; Laboratory of Anesthesia and Critical Care Medicine, National-Local Joint Engineering Research Center of Translational Medicine of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China. .
Chen C; Department of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China. .; Laboratory of Anesthesia and Critical Care Medicine, National-Local Joint Engineering Research Center of Translational Medicine of Anesthesiology, West China Hospital, Sichuan University, Chengdu, China. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2023 Jul 28; Vol. 20 (1), pp. 175. Date of Electronic Publication: 2023 Jul 28.
Typ publikacji:
Journal Article
MeSH Terms:
Anesthesia*/adverse effects
Cognitive Dysfunction*/etiology
Cognitive Dysfunction*/genetics
Cognitive Dysfunction*/metabolism
Hippocampus*/metabolism
Postoperative Cognitive Complications*/etiology
Postoperative Cognitive Complications*/genetics
Postoperative Cognitive Complications*/metabolism
Animals ; Mice ; Inflammation/metabolism ; Memory Disorders/metabolism ; Mice, Inbred C57BL ; Neuroinflammatory Diseases ; RNA, Messenger/metabolism ; Smad7 Protein/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Smad7 Deficiency in Myeloid Cells Does Not Affect Liver Injury, Inflammation or Fibrosis after Chronic CCl 4 Exposure in Mice.
Autorzy:
Unrau L; Institute of Experimental Immunology and Hepatology, University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.; Hamburg Center for Translational Immunology (HCTI), University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.; Department of Virology, Bernhard Nocht Institute for Tropical Medicine, Bernhard-Nocht-Str. 74, 20359 Hamburg, Germany.
Endig J; Institute of Experimental Immunology and Hepatology, University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.; Hamburg Center for Translational Immunology (HCTI), University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.
Goltz D; Institute for Hematopathology, Fangdieckstrasse 75a, 22547 Hamburg, Germany.
Sprezyna P; Institute of Experimental Immunology and Hepatology, University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.; Hamburg Center for Translational Immunology (HCTI), University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.
Ulrich H; Institute of Experimental Immunology and Hepatology, University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.; Hamburg Center for Translational Immunology (HCTI), University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.
Hagenstein J; Institute of Experimental Immunology and Hepatology, University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.; Hamburg Center for Translational Immunology (HCTI), University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.
Geers B; Institute of Experimental Immunology and Hepatology, University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.; Hamburg Center for Translational Immunology (HCTI), University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.
Kaftan K; Institute of Experimental Immunology and Hepatology, University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.; Hamburg Center for Translational Immunology (HCTI), University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.
Heukamp LC; Institute for Hematopathology, Fangdieckstrasse 75a, 22547 Hamburg, Germany.
Tiegs G; Institute of Experimental Immunology and Hepatology, University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.; Hamburg Center for Translational Immunology (HCTI), University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.
Diehl L; Institute of Experimental Immunology and Hepatology, University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.; Hamburg Center for Translational Immunology (HCTI), University Medical Center Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Oct 27; Vol. 22 (21). Date of Electronic Publication: 2021 Oct 27.
Typ publikacji:
Journal Article
MeSH Terms:
Carbon Tetrachloride/*pharmacology
Inflammation/*metabolism
Liver Cirrhosis/*chemically induced
Liver Cirrhosis/*metabolism
Liver Diseases/*metabolism
Myeloid Cells/*metabolism
Smad7 Protein/*deficiency
Alanine Transaminase/metabolism ; Animals ; Disease Models, Animal ; Hepatocytes/metabolism ; Liver/metabolism ; Male ; Mice ; Signal Transduction/physiology ; Smad7 Protein/metabolism ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Association between MeCP2 and Smad7 in the pathogenesis and development of pathological scars.
Autorzy:
Li D; Department of Plastic and Burn Surgery, Chongqing Medical University First Affiliated Hospital, Chongqing, China.
Yang E; Department of Plastic and Burn Surgery, Chongqing Medical University First Affiliated Hospital, Chongqing, China.
Zhao J; Department of Plastic and Burn Surgery, Chongqing Medical University First Affiliated Hospital, Chongqing, China.
Zhang H; Department of Plastic and Burn Surgery, Chongqing Medical University First Affiliated Hospital, Chongqing, China.
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Źródło:
Journal of plastic surgery and hand surgery [J Plast Surg Hand Surg] 2021 Oct; Vol. 55 (5), pp. 284-293. Date of Electronic Publication: 2021 Jan 21.
Typ publikacji:
Journal Article
MeSH Terms:
Cicatrix, Hypertrophic*
Keloid*/pathology
Smad7 Protein*/genetics
Methyl-CpG-Binding Protein 2/*genetics
Fibroblasts/pathology ; Humans ; RNA, Messenger
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Rebalancing TGF-β/Smad7 signaling via Compound kushen injection in hepatic stellate cells protects against liver fibrosis and hepatocarcinogenesis.
Autorzy:
Yang Y; State Key Laboratory of Oncogenes and Related Genes, Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; CAS Key Laboratory of Nutrition, Metabolism and Food Safety, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.
Sun M; State Key Laboratory of Oncogenes and Related Genes, Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; CAS Key Laboratory of Nutrition, Metabolism and Food Safety, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.
Li W; State Key Laboratory of Oncogenes and Related Genes, Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Liu C; State Key Laboratory of Oncogenes and Related Genes, Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Jiang Z; State Key Laboratory of Oncogenes and Related Genes, Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Gu P; State Key Laboratory of Oncogenes and Related Genes, Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Li J; State Key Laboratory of Oncogenes and Related Genes, Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Wang W; Beijing Zhendong Pharmaceutical Research Institute Co., Ltd., Beijing, China.
You R; Beijing Zhendong Pharmaceutical Research Institute Co., Ltd., Beijing, China.
Ba Q; State Key Laboratory of Oncogenes and Related Genes, Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Li X; State Key Laboratory of Oncogenes and Related Genes, Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Wang H; State Key Laboratory of Oncogenes and Related Genes, Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; CAS Key Laboratory of Nutrition, Metabolism and Food Safety, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.
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Źródło:
Clinical and translational medicine [Clin Transl Med] 2021 Jul; Vol. 11 (7), pp. e410.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Drugs, Chinese Herbal/*pharmacology
Signal Transduction/*drug effects
Smad7 Protein/*metabolism
Transforming Growth Factor beta/*metabolism
Animals ; Carbon Tetrachloride/toxicity ; Carcinoma, Hepatocellular/drug therapy ; Carcinoma, Hepatocellular/pathology ; Cell Line ; Drugs, Chinese Herbal/therapeutic use ; Hepatic Stellate Cells/cytology ; Hepatic Stellate Cells/metabolism ; Humans ; Liver/metabolism ; Liver/pathology ; Liver Cirrhosis/chemically induced ; Liver Cirrhosis/drug therapy ; Liver Neoplasms/drug therapy ; Liver Neoplasms/pathology ; Male ; Medicine, Chinese Traditional ; Meta-Analysis as Topic ; Mice ; Mice, Inbred C57BL ; RNA Interference ; RNA, Small Interfering/metabolism ; Smad7 Protein/antagonists & inhibitors ; Smad7 Protein/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Involvement of Smad7 in Inflammatory Diseases of the Gut and Colon Cancer.
Autorzy:
Troncone E; Department of Systems Medicine, University of Rome 'Tor Vergata', 00133 Rome, Italy.
Marafini I; Department of Systems Medicine, University of Rome 'Tor Vergata', 00133 Rome, Italy.
Stolfi C; Department of Systems Medicine, University of Rome 'Tor Vergata', 00133 Rome, Italy.
Monteleone G; Department of Systems Medicine, University of Rome 'Tor Vergata', 00133 Rome, Italy.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Apr 10; Vol. 22 (8). Date of Electronic Publication: 2021 Apr 10.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Colonic Neoplasms/*genetics
Inflammatory Bowel Diseases/*genetics
Smad7 Protein/*genetics
Transforming Growth Factor beta1/*genetics
Animals ; Colonic Neoplasms/immunology ; Colonic Neoplasms/pathology ; Colonic Neoplasms/therapy ; Disease Models, Animal ; Gastrointestinal Microbiome/genetics ; Gastrointestinal Microbiome/immunology ; Humans ; Inflammatory Bowel Diseases/immunology ; Inflammatory Bowel Diseases/pathology ; Intestinal Mucosa/immunology ; Intestinal Mucosa/pathology ; Mice ; Oligonucleotides, Antisense/therapeutic use ; Signal Transduction/immunology ; Smad7 Protein/antagonists & inhibitors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Current perspectives on inhibitory SMAD7 in health and disease.
Autorzy:
de Ceuninck van Capelle C; Oncode Institute and Department of Cell and Chemical Biology, Leiden University Medical Center, Leiden, the Netherlands.
Spit M; Oncode Institute and Department of Cell and Chemical Biology, Leiden University Medical Center, Leiden, the Netherlands.
Ten Dijke P; Oncode Institute and Department of Cell and Chemical Biology, Leiden University Medical Center, Leiden, the Netherlands.
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Źródło:
Critical reviews in biochemistry and molecular biology [Crit Rev Biochem Mol Biol] 2020 Dec; Vol. 55 (6), pp. 691-715. Date of Electronic Publication: 2020 Oct 20.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Smad7 Protein/*metabolism
Transforming Growth Factor beta/*metabolism
Animals ; Bone Morphogenetic Proteins/genetics ; Bone Morphogenetic Proteins/metabolism ; Humans ; Signal Transduction/genetics ; Signal Transduction/physiology ; Smad7 Protein/genetics ; Transforming Growth Factor beta/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MicroRNA-216a Promotes Endothelial Inflammation by Smad7/I κ B α Pathway in Atherosclerosis.
Autorzy:
Yang S; State Key Laboratory of Cardiovascular Disease, FuWai Hospital, National Center for Cardiovascular Diseases, Peking Union Medical College & Chinese Academy of Medical Sciences, Beijing, China.; Xiamen Cardiovascular Hospital, Xiamen University, Xiamen, China.
Chen Y; State Key Laboratory of Cardiovascular Disease, FuWai Hospital, National Center for Cardiovascular Diseases, Peking Union Medical College & Chinese Academy of Medical Sciences, Beijing, China.
Mi X; State Key Laboratory of Cardiovascular Disease, FuWai Hospital, National Center for Cardiovascular Diseases, Peking Union Medical College & Chinese Academy of Medical Sciences, Beijing, China.
Zhang S; State Key Laboratory of Cardiovascular Disease, FuWai Hospital, National Center for Cardiovascular Diseases, Peking Union Medical College & Chinese Academy of Medical Sciences, Beijing, China.
Yang Y; State Key Laboratory of Cardiovascular Disease, FuWai Hospital, National Center for Cardiovascular Diseases, Peking Union Medical College & Chinese Academy of Medical Sciences, Beijing, China.
Hui R; State Key Laboratory of Cardiovascular Disease, FuWai Hospital, National Center for Cardiovascular Diseases, Peking Union Medical College & Chinese Academy of Medical Sciences, Beijing, China.
Zhang W; State Key Laboratory of Cardiovascular Disease, FuWai Hospital, National Center for Cardiovascular Diseases, Peking Union Medical College & Chinese Academy of Medical Sciences, Beijing, China.
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Źródło:
Disease markers [Dis Markers] 2020 Nov 17; Vol. 2020, pp. 8864322. Date of Electronic Publication: 2020 Nov 17 (Print Publication: 2020).
Typ publikacji:
Journal Article
MeSH Terms:
Atherosclerosis/*physiopathology
MicroRNAs/*genetics
NF-KappaB Inhibitor alpha/*genetics
Smad7 Protein/*genetics
3' Untranslated Regions ; Aged ; Atherosclerosis/genetics ; Carotid Artery Diseases/genetics ; Cell Adhesion/genetics ; Endothelium, Vascular/physiopathology ; Female ; Human Umbilical Vein Endothelial Cells ; Humans ; Male ; Middle Aged ; Monocytes/cytology ; NF-KappaB Inhibitor alpha/metabolism ; Plaque, Atherosclerotic/genetics ; Smad7 Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Co-overexpression of VEGF and Smad7 improved the therapeutic effects of adipose-derived stem cells on neurogenic erectile dysfunction in the rat model.
Autorzy:
He L; Department of Urology, Affiliated Hospital of Nantong University, Nantong, China.; Medical College, Nantong University, Nantong, China.
Yu T; Department of Urology, Affiliated Hospital of Nantong University, Nantong, China.; Medical College, Nantong University, Nantong, China.
Xiao Y; Department of Urology, Affiliated Hospital of Nantong University, Nantong, China.; Medical College, Nantong University, Nantong, China.
Huang Y; Department of Urology, Affiliated Hospital of Nantong University, Nantong, China.
Guan Y; Department of Urology, Affiliated Hospital of Nantong University, Nantong, China.
Zhao F; Department of Urology, Affiliated Hospital of Nantong University, Nantong, China.
Ma L; Department of Urology, Affiliated Hospital of Nantong University, Nantong, China.
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Źródło:
Andrologia [Andrologia] 2022 Nov; Vol. 54 (10), pp. e14538. Date of Electronic Publication: 2022 Aug 01.
Typ publikacji:
Journal Article
MeSH Terms:
Erectile Dysfunction*/etiology
Erectile Dysfunction*/therapy
Adipose Tissue/metabolism ; Animals ; Collagen ; Disease Models, Animal ; Humans ; Male ; Nitric Oxide Synthase Type I/metabolism ; Penile Erection ; Penis ; Phosphates/metabolism ; Rats ; Rats, Sprague-Dawley ; Smad7 Protein/metabolism ; Stem Cell Transplantation/methods ; Vascular Endothelial Growth Factor A/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Smad7 Binds Differently to Individual and Tandem WW3 and WW4 Domains of WWP2 Ubiquitin Ligase Isoforms.
Autorzy:
Wahl LC; School of Biological Sciences, University of East Anglia, Norwich NR4 7TJ, UK. .
Watt JE; School of Biological Sciences, University of East Anglia, Norwich NR4 7TJ, UK. .
Yim HTT; School of Biological Sciences, University of East Anglia, Norwich NR4 7TJ, UK. .
De Bourcier D; School of Chemistry, University of East Anglia, Norwich NR4 7TJ, UK. .
Tolchard J; School of Chemistry, University of East Anglia, Norwich NR4 7TJ, UK. .
Soond SM; School of Biological Sciences, University of East Anglia, Norwich NR4 7TJ, UK. .
Blumenschein TMA; School of Chemistry, University of East Anglia, Norwich NR4 7TJ, UK. .
Chantry A; School of Biological Sciences, University of East Anglia, Norwich NR4 7TJ, UK. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2019 Sep 21; Vol. 20 (19). Date of Electronic Publication: 2019 Sep 21.
Typ publikacji:
Journal Article
MeSH Terms:
Smad7 Protein/*chemistry
Smad7 Protein/*metabolism
Ubiquitin-Protein Ligases/*chemistry
HEK293 Cells ; Humans ; Isoenzymes/chemistry ; Isoenzymes/genetics ; Isoenzymes/metabolism ; Magnetic Resonance Spectroscopy ; Models, Molecular ; Signal Transduction ; Smad7 Protein/genetics ; Transforming Growth Factor beta/metabolism ; Transforming Growth Factor beta/pharmacology ; Ubiquitin-Protein Ligases/genetics ; Ubiquitin-Protein Ligases/metabolism ; WW Domains/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Angiotensin‑converting enzyme‑2 improves diabetic nephropathy by targeting Smad7 for ubiquitin degradation.
Autorzy:
Chen Z; Department of Nephrology, Beijing Friendship Hospital, Capital Medical University, Beijing 100050, P.R. China.
Chen X; Department of Nephrology, Beijing Friendship Hospital, Capital Medical University, Beijing 100050, P.R. China.
Bai Y; Department of Nephrology, Beijing Friendship Hospital, Capital Medical University, Beijing 100050, P.R. China.
Diao Z; Department of Nephrology, Beijing Friendship Hospital, Capital Medical University, Beijing 100050, P.R. China.
Liu W; Department of Nephrology, Beijing Friendship Hospital, Capital Medical University, Beijing 100050, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2020 Oct; Vol. 22 (4), pp. 3008-3016. Date of Electronic Publication: 2020 Jul 28.
Typ publikacji:
Journal Article
MeSH Terms:
Up-Regulation*
Angiotensin-Converting Enzyme 2/*blood
Diabetic Nephropathies/*metabolism
Glucose/*adverse effects
Kidney Tubules, Distal/*cytology
Smad7 Protein/*metabolism
Aged ; Aged, 80 and over ; Angiotensin-Converting Enzyme 2/genetics ; Angiotensin-Converting Enzyme 2/pharmacology ; Animals ; Case-Control Studies ; Cell Line ; Diabetic Nephropathies/blood ; Diabetic Nephropathies/genetics ; Epithelial-Mesenchymal Transition/drug effects ; Humans ; Kidney/metabolism ; Kidney Tubules, Distal/drug effects ; Kidney Tubules, Distal/metabolism ; Male ; Middle Aged ; Smad7 Protein/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MiR-581/SMAD7 Axis Contributes to Colorectal Cancer Metastasis: A Bioinformatic and Experimental Validation-Based Study.
Autorzy:
Zhao X; Key Laboratory of Resource Biology and Biotechnology in Western China, Ministry of Education, School of Life Sciences, Northwest University, Xi'an 710069, China.; Institute of Preventive Genomic Medicine, School of Life Sciences, Northwest University, Xi'an 710069, China.
Liu S; Key Laboratory of Resource Biology and Biotechnology in Western China, Ministry of Education, School of Life Sciences, Northwest University, Xi'an 710069, China.; Institute of Preventive Genomic Medicine, School of Life Sciences, Northwest University, Xi'an 710069, China.
Yan B; Key Laboratory of Resource Biology and Biotechnology in Western China, Ministry of Education, School of Life Sciences, Northwest University, Xi'an 710069, China.; Institute of Preventive Genomic Medicine, School of Life Sciences, Northwest University, Xi'an 710069, China.
Yang J; Key Laboratory of Resource Biology and Biotechnology in Western China, Ministry of Education, School of Life Sciences, Northwest University, Xi'an 710069, China.; Institute of Preventive Genomic Medicine, School of Life Sciences, Northwest University, Xi'an 710069, China.
Chen E; Key Laboratory of Resource Biology and Biotechnology in Western China, Ministry of Education, School of Life Sciences, Northwest University, Xi'an 710069, China.; Institute of Preventive Genomic Medicine, School of Life Sciences, Northwest University, Xi'an 710069, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2020 Sep 05; Vol. 21 (18). Date of Electronic Publication: 2020 Sep 05.
Typ publikacji:
Journal Article
MeSH Terms:
Colorectal Neoplasms/*genetics
MicroRNAs/*genetics
Smad7 Protein/*genetics
Biological Specimen Banks ; Cell Line, Tumor ; Cell Movement/genetics ; Cell Proliferation/genetics ; Colonic Neoplasms/genetics ; Colorectal Neoplasms/pathology ; Computational Biology/methods ; Databases, Genetic ; Epithelial-Mesenchymal Transition/genetics ; Gene Expression Regulation, Neoplastic/genetics ; Humans ; MicroRNAs/metabolism ; Neoplasm Metastasis/genetics ; Smad7 Protein/metabolism
Czasopismo naukowe
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Tytuł:
Dasatinib ameliorates thioacetamide-induced liver fibrosis: modulation of miR-378 and miR-17 and their linked Wnt/β-catenin and TGF-β/smads pathways.
Autorzy:
Zaafan MA; Faculty of Pharmacy, Pharmacology and Toxicology Department, October University for Modern Sciences and Arts (MSA), Dokki, Egypt.
Abdelhamid AM; Faculty of Pharmacy, Biochemistry Department, October University for Modern Sciences and Arts (MSA), Dokki, Egypt.
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Źródło:
Journal of enzyme inhibition and medicinal chemistry [J Enzyme Inhib Med Chem] 2022 Dec; Vol. 37 (1), pp. 118-124.
Typ publikacji:
Journal Article
MeSH Terms:
Dasatinib/*pharmacology
Liver Cirrhosis/*drug therapy
MicroRNAs/*antagonists & inhibitors
Smad7 Protein/*metabolism
Thioacetamide/*antagonists & inhibitors
Animals ; Dasatinib/chemistry ; Dose-Response Relationship, Drug ; Liver Cirrhosis/chemically induced ; Liver Cirrhosis/metabolism ; Male ; Mice ; MicroRNAs/metabolism ; Molecular Structure ; Structure-Activity Relationship ; Transforming Growth Factor beta/antagonists & inhibitors ; Transforming Growth Factor beta/metabolism ; Wnt Signaling Pathway/drug effects ; beta Catenin/antagonists & inhibitors ; beta Catenin/metabolism
Czasopismo naukowe
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Tytuł:
Inhibition of USP4 attenuates pathological scarring by downregulation of the TGF‑β/Smad signaling pathway.
Autorzy:
Zhang J; Department of Oral and Maxillofacial Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Na S; Department of Oral and Maxillofacial Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Pan S; Department of Oral and Maxillofacial Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Long S; Department of Oral and Maxillofacial Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Xin Y; Department of Oral and Maxillofacial Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Jiang Q; Department of Oral and Maxillofacial Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Lai Z; Department of Oral and Maxillofacial Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Yan J; Department of Oral and Maxillofacial Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
Cao Z; Department of Oral and Maxillofacial Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2019 Aug; Vol. 20 (2), pp. 1429-1435. Date of Electronic Publication: 2019 Jun 06.
Typ publikacji:
Journal Article
MeSH Terms:
Cicatrix/*genetics
Keloid/*genetics
Receptor, Transforming Growth Factor-beta Type I/*genetics
Smad7 Protein/*genetics
Transforming Growth Factor beta/*genetics
Ubiquitin-Specific Proteases/*genetics
Animals ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Cicatrix/metabolism ; Cicatrix/pathology ; Cicatrix/prevention & control ; Fibroblasts/metabolism ; Fibroblasts/pathology ; Fibroblasts/transplantation ; Gene Expression Regulation ; Humans ; Keloid/metabolism ; Keloid/pathology ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Receptor, Transforming Growth Factor-beta Type I/metabolism ; Signal Transduction ; Smad7 Protein/metabolism ; Terphenyl Compounds/pharmacology ; Transforming Growth Factor beta/metabolism ; Transplantation, Heterologous ; Ubiquitin-Specific Proteases/antagonists & inhibitors ; Ubiquitin-Specific Proteases/metabolism
Czasopismo naukowe
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Tytuł:
Molecular mechanism of the TGF‑β/Smad7 signaling pathway in ulcerative colitis.
Autorzy:
Bai B; Department of Gastroenterology, First Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230022, P.R. China.
Li H; The Key Laboratory of Digestive Diseases of Anhui Province, First Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230022, P.R. China.
Han L; The Key Laboratory of Digestive Diseases of Anhui Province, First Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230022, P.R. China.
Mei Y; The Key Laboratory of Digestive Diseases of Anhui Province, First Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230022, P.R. China.
Hu C; Department of Gastroenterology, First Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230022, P.R. China.
Mei Q; Department of Gastroenterology, First Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230022, P.R. China.
Xu J; Department of Gastroenterology, First Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230022, P.R. China.
Liu X; Department of Gastroenterology, First Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230022, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2022 Apr; Vol. 25 (4). Date of Electronic Publication: 2022 Feb 09.
Typ publikacji:
Journal Article
MeSH Terms:
Colitis, Ulcerative/*metabolism
Smad7 Protein/*genetics
Smad7 Protein/*metabolism
Transforming Growth Factor beta/*genetics
Transforming Growth Factor beta/*metabolism
Animals ; Cadherins/metabolism ; Colitis, Ulcerative/chemically induced ; Colon/pathology ; Dextran Sulfate/toxicity ; Disease Models, Animal ; Female ; Intestinal Mucosa/drug effects ; Intestinal Mucosa/pathology ; Intestinal Mucosa/ultrastructure ; Male ; Mesalamine/administration & dosage ; Mice, Inbred C57BL ; Myosin-Light-Chain Kinase/metabolism ; Occludin/metabolism ; Peroxidase/drug effects ; Severity of Illness Index ; Signal Transduction/drug effects ; Tight Junctions/metabolism ; Tumor Necrosis Factor-alpha/antagonists & inhibitors ; Zonula Occludens-1 Protein/metabolism ; Mice
Czasopismo naukowe
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Tytuł:
The matricellular protein CCN5 induces apoptosis in myofibroblasts through SMAD7-mediated inhibition of NFκB.
Autorzy:
Nguyen MT; College of Life Sciences, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea.
Lee MA; College of Life Sciences, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea.
Kim YK; Department of Biochemistry, Chonnam National University Medical School, Hwasun, Jeollanam-do, Republic of Korea.
Kook H; Department of Pharmacology, Chonnam National University Medical School, Hwasun, Jeollanam-do, Republic of Korea.
Jeong D; Department of Molecular & Life Science, College of Science and Convergence Technology, Hanyang University-ERICA, Ansan, Gyeonggi-do, Republic of Korea.
Jang SP; BethphaGen, S3-203, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea.
Kwak TH; BethphaGen, S3-203, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea.
Park WJ; College of Life Sciences, Gwangju Institute of Science and Technology, Gwangju, Republic of Korea.
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Źródło:
PloS one [PLoS One] 2022 Aug 02; Vol. 17 (8), pp. e0269735. Date of Electronic Publication: 2022 Aug 02 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Myofibroblasts*
Tumor Suppressor Protein p53*
CCN Intercellular Signaling Proteins/*metabolism
Repressor Proteins/*metabolism
Apoptosis ; Fibrosis ; Humans ; NF-kappa B ; RNA, Small Interfering ; Smad7 Protein/genetics
Czasopismo naukowe
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Tytuł:
Intratunical injection of rat-derived bone marrow mesenchymal stem cells prevents fibrosis and is associated with increased Smad7 expression in a rat model of Peyronie's disease.
Autorzy:
Wang W; Central Laboratory, The Affiliated Yantai Yuhuangding Hospital of Qingdao University, Yantai, 26400, China.
Ding W; Department of Health Care, The Affiliated Yantai Yuhuangding Hospital of Qingdao University, Yantai, 26400, China.
Zhang X; Reproductive Medicine Center, The Affiliated Yantai Yuhuangding Hospital of Qingdao University, Yantai, 26400, China.
Wu S; Department of Urology, The Affiliated Yantai Yuhuangding Hospital of Qingdao University, Yantai, 26400, China.
Yu T; Department of Urology, The Affiliated Yantai Yuhuangding Hospital of Qingdao University, Yantai, 26400, China.; School of Clinical Medicine, Weifang Medical University, Weifang, 261000, China.
Cui X; Department of Urology, The Affiliated Yantai Yuhuangding Hospital of Qingdao University, Yantai, 26400, China.; School of Clinical Medicine, Weifang Medical University, Weifang, 261000, China.
Xie Y; Department of Urology, The Affiliated Yantai Yuhuangding Hospital of Qingdao University, Yantai, 26400, China.; Binzhou Medical University, Yantai, 264000, China.
Yang D; Department of Urology, The Affiliated Yantai Yuhuangding Hospital of Qingdao University, Yantai, 26400, China. .
Lin C; Department of Urology, The Affiliated Yantai Yuhuangding Hospital of Qingdao University, Yantai, 26400, China. .
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Źródło:
Stem cell research & therapy [Stem Cell Res Ther] 2022 Jul 30; Vol. 13 (1), pp. 390. Date of Electronic Publication: 2022 Jul 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Erectile Dysfunction*/therapy
Mesenchymal Stem Cells*/metabolism
Penile Induration*/pathology
Penile Induration*/therapy
Animals ; Bone Marrow Cells/metabolism ; Disease Models, Animal ; Fibrosis ; Humans ; Male ; Osteopontin/metabolism ; Pancreatic Elastase ; RNA, Small Interfering ; Rats ; Rats, Sprague-Dawley ; Smad7 Protein/metabolism ; Transforming Growth Factor beta1/genetics ; Transforming Growth Factor beta1/metabolism
Czasopismo naukowe
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Tytuł:
Association of SMAD7 genetic markers and haplotypes with colorectal cancer risk.
Autorzy:
Alidoust M; Department of Medical Genetics and Molecular Medicine, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.; Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Hamzehzadeh L; Department of Medical Genetics and Molecular Medicine, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.; Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Khorshid Shamshiri A; Department of Medical Genetics and Molecular Medicine, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.; Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Afzaljavan F; Department of Medical Genetics and Molecular Medicine, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.; Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Kerachian MA; Department of Medical Genetics and Molecular Medicine, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.; Medical Genetics Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.; Cancer Genetics Research Unit, Reza Radiotherapy and Oncology Center, Mashhad, Iran.
Fanipakdel A; Cancer Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.
Aledavood SA; Cancer Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.
Allahyari A; Hematology and Oncology Department, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Bari A; Hematology and Oncology Department, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Moosanen Mozaffari H; Department of Gastroenterology and Hepatology, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Goshayeshi L; Department of Gastroenterology and Hepatology, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran. .; Surgical Oncology Research Center, Mashhad University of Medical Sciences, Mashhad, Iran. .
Pasdar A; Department of Medical Genetics and Molecular Medicine, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran. .; Medical Genetics Research Center, Mashhad University of Medical Sciences, Mashhad, Iran. .; Division of Applied Medicine, Medical School, University of Aberdeen, Foresterhill, Aberdeen, AB25 2ZD, UK. .
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Źródło:
BMC medical genomics [BMC Med Genomics] 2022 Jan 11; Vol. 15 (1), pp. 8. Date of Electronic Publication: 2022 Jan 11.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Colorectal Neoplasms*/epidemiology
Colorectal Neoplasms*/genetics
Case-Control Studies ; Genetic Markers ; Genetic Predisposition to Disease ; Genotype ; Haplotypes ; Humans ; Polymorphism, Single Nucleotide ; Risk Factors ; Smad7 Protein/genetics ; Smad7 Protein/metabolism
Czasopismo naukowe
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Tytuł:
Gene profiling of SEC13, SMAD7, GHRL, long non-coding RNA GHRLOS, HIF-1α in gastric cancer patients.
Autorzy:
Hussein NA; Applied Medical Chemistry Department, Medical Research Institute, Alexandria University, Alexandria, Egypt. .
Rashad MM; Applied Medical Chemistry Department, Medical Research Institute, Alexandria University, Alexandria, Egypt.
Abdou AS; Human Physiology Department, Medical Research Institute, Alexandria University, Alexandria, Egypt.
Hussein AM; Cancer Management and Research Department, Medical Research Institute, Alexandria University, Alexandria, Egypt.
Mohamed HM; Applied Medical Chemistry Department, Medical Research Institute, Alexandria University, Alexandria, Egypt.
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Źródło:
Scientific reports [Sci Rep] 2022 Apr 21; Vol. 12 (1), pp. 6555. Date of Electronic Publication: 2022 Apr 21.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
RNA, Long Noncoding*/genetics
Stomach Neoplasms*/pathology
Carcinoembryonic Antigen/metabolism ; Ghrelin/metabolism ; Humans ; Hypoxia-Inducible Factor 1, alpha Subunit/genetics ; Hypoxia-Inducible Factor 1, alpha Subunit/metabolism ; Prognosis ; Smad7 Protein/genetics ; Smad7 Protein/metabolism
Czasopismo naukowe
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