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Wyszukujesz frazę ""Stallings, CL"" wg kryterium: Autor

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    Wyświetlanie 1-6 z 6
    Tytuł:
    Autophagy prevents early proinflammatory responses and neutrophil recruitment during Mycobacterium tuberculosis infection without affecting pathogen burden in macrophages.
    Autorzy:
    Kinsella RL; Department of Molecular Microbiology, Center for Women's Infectious Disease Research, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Kimmey JM; Department of Molecular Microbiology, Center for Women's Infectious Disease Research, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Smirnov A; Department of Molecular Microbiology, Center for Women's Infectious Disease Research, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Woodson R; Department of Molecular Microbiology, Center for Women's Infectious Disease Research, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Gaggioli MR; Department of Molecular Microbiology, Center for Women's Infectious Disease Research, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Chavez SM; Department of Molecular Microbiology, Center for Women's Infectious Disease Research, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Kreamalmeyer D; Department of Molecular Microbiology, Center for Women's Infectious Disease Research, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Stallings CL; Department of Molecular Microbiology, Center for Women's Infectious Disease Research, Washington University School of Medicine, St. Louis, Missouri, United States of America.
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    Źródło:
    PLoS biology [PLoS Biol] 2023 Jun 15; Vol. 21 (6), pp. e3002159. Date of Electronic Publication: 2023 Jun 15 (Print Publication: 2023).
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
    MeSH Terms:
    Tuberculosis*/microbiology
    Mycobacterium tuberculosis*/physiology
    Animals ; Mice ; Humans ; Neutrophil Infiltration ; Macrophages/physiology ; Autophagy ; Inflammation
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Mycobacterium tuberculosis Rv3160c is a TetR-like transcriptional repressor that regulates expression of the putative oxygenase Rv3161c.
    Autorzy:
    Tükenmez H; Department of Chemistry, Umeå University, 90187, Umeå, Sweden. .; Department of Molecular Biology, Umeå University, 90187, Umeå, Sweden. .; Umeå Centre for Microbial Research, Umeå University, 90187, Umeå, Sweden. .; Molecular Infection Medicine, Sweden (MIMS), Umeå University, 90187, Umeå, Sweden. .
    Sarkar S; Department of Chemistry, Umeå University, 90187, Umeå, Sweden.; Umeå Centre for Microbial Research, Umeå University, 90187, Umeå, Sweden.
    Anoosheh S; Department of Chemistry, Umeå University, 90187, Umeå, Sweden.; Umeå Centre for Microbial Research, Umeå University, 90187, Umeå, Sweden.
    Kruchanova A; Department of Molecular Biology, Umeå University, 90187, Umeå, Sweden.; Umeå Centre for Microbial Research, Umeå University, 90187, Umeå, Sweden.
    Edström I; Department of Molecular Biology, Umeå University, 90187, Umeå, Sweden.; Umeå Centre for Microbial Research, Umeå University, 90187, Umeå, Sweden.
    Harrison GA; Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, MO, 63110, USA.
    Stallings CL; Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, MO, 63110, USA.
    Almqvist F; Department of Chemistry, Umeå University, 90187, Umeå, Sweden.; Umeå Centre for Microbial Research, Umeå University, 90187, Umeå, Sweden.
    Larsson C; Department of Molecular Biology, Umeå University, 90187, Umeå, Sweden. .; Umeå Centre for Microbial Research, Umeå University, 90187, Umeå, Sweden. .; Holmsund, Sweden. .
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    Źródło:
    Scientific reports [Sci Rep] 2021 Jan 15; Vol. 11 (1), pp. 1523. Date of Electronic Publication: 2021 Jan 15.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Drug Resistance, Microbial/*genetics
    Mycobacterium tuberculosis/*genetics
    Mycobacterium tuberculosis/*metabolism
    Antitubercular Agents/pharmacology ; Bacterial Proteins/metabolism ; Gene Expression/genetics ; Gene Expression Regulation, Bacterial/genetics ; Isoniazid/pharmacology ; Oxygenases/metabolism ; Protein Binding ; Repressor Proteins/metabolism ; Tetracycline/pharmacology ; Transcription Factors/metabolism ; Tuberculosis/metabolism ; Tuberculosis/microbiology ; Tuberculosis, Multidrug-Resistant/metabolism ; Tuberculosis, Multidrug-Resistant/microbiology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Rv0004 is a new essential member of the mycobacterial DNA replication machinery.
    Autorzy:
    Mann KM; Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Huang DL; Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Hooppaw AJ; Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Logsdon MM; Department of Molecular Biology and Microbiology, Tufts University School of Medicine, Boston, Massachusetts, United States of America.
    Richardson K; Department of Molecular Biology and Microbiology, Tufts University School of Medicine, Boston, Massachusetts, United States of America.
    Lee HJ; Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Kimmey JM; Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, Missouri, United States of America.
    Aldridge BB; Department of Molecular Biology and Microbiology, Tufts University School of Medicine, Boston, Massachusetts, United States of America.; Department of Biomedical Engineering, Tufts University School of Engineering, Medford, Massachusetts, United States of America.
    Stallings CL; Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, Missouri, United States of America.
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    Źródło:
    PLoS genetics [PLoS Genet] 2017 Nov 27; Vol. 13 (11), pp. e1007115. Date of Electronic Publication: 2017 Nov 27 (Print Publication: 2017).
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Bacterial Proteins/*genetics
    DNA Replication/*genetics
    DNA, Bacterial/*genetics
    DNA-Binding Proteins/*genetics
    Mycobacterium tuberculosis/*genetics
    Amino Acid Sequence ; Bacterial Proteins/metabolism ; Cell Cycle/genetics ; DNA, Bacterial/metabolism ; DNA-Binding Proteins/metabolism ; DnaB Helicases/metabolism ; Microbial Viability/genetics ; Mycobacterium tuberculosis/metabolism ; Protein Binding ; Sequence Homology, Amino Acid
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Unexpected role for IL-17 in protective immunity against hypervirulent Mycobacterium tuberculosis HN878 infection.
    Autorzy:
    Gopal R; Department of Pediatrics, Division of Infectious Diseases, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, United States of America.
    Monin L; Department of Pediatrics, Division of Infectious Diseases, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, United States of America.
    Slight S; Department of Pediatrics, Division of Infectious Diseases, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, United States of America.
    Uche U; Department of Pediatrics, Division of Infectious Diseases, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, United States of America.
    Blanchard E; Department of Pediatrics, Division of Infectious Diseases, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, United States of America.
    Fallert Junecko BA; Department of Infectious Diseases and Microbiology, University of Pittsburgh, Pittsburgh, Pennsylvania, United States of America.
    Ramos-Payan R; Faculty of Biological and Chemical Sciences, Autonomous University of Sinaloa, Culiacan, Sinaloa, Mexico; Department of Medicine, Division of Allergy, Immunology and Rheumatology, University of Rochester Medical Center, Rochester, New York, United States of America.
    Stallings CL; Department of Molecular Microbiology, Washington University in St. Louis, St. Louis, Missouri, United States of America.
    Reinhart TA; Department of Infectious Diseases and Microbiology, University of Pittsburgh, Pittsburgh, Pennsylvania, United States of America.
    Kolls JK; Richard King Mellon Foundation Institute for Pediatric Research, Children's Hospital of Pittsburgh of University of Pittsburgh Medical Center, Pittsburgh, Pennsylvania, United States of America.
    Kaushal D; Tulane National Primate Center, New Orleans, Louisiana, United States of America.
    Nagarajan U; Department of Pediatrics, University of North Carolina, Chapel Hill, Chapel Hill, North Carolina, United States of America.
    Rangel-Moreno J; Department of Medicine, Division of Allergy, Immunology and Rheumatology, University of Rochester Medical Center, Rochester, New York, United States of America.
    Khader SA; Department of Pediatrics, Division of Infectious Diseases, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, United States of America; Department of Molecular Microbiology, Washington University in St. Louis, St. Louis, Missouri, United States of America.
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    Źródło:
    PLoS pathogens [PLoS Pathog] 2014 May 15; Vol. 10 (5), pp. e1004099. Date of Electronic Publication: 2014 May 15 (Print Publication: 2014).
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Immunity, Innate/*genetics
    Interleukin-17/*physiology
    Mycobacterium tuberculosis/*immunology
    Animals ; Cells, Cultured ; Communicable Diseases, Emerging/genetics ; Communicable Diseases, Emerging/immunology ; Cytoprotection/genetics ; Cytoprotection/immunology ; Female ; Interleukin-17/genetics ; Interleukin-1beta/physiology ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mycobacterium tuberculosis/pathogenicity ; Receptors, Interleukin-1 Type I/genetics ; Toll-Like Receptor 2/physiology ; Tuberculosis/genetics ; Tuberculosis/immunology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Compensatory dendritic cell development mediated by BATF-IRF interactions.
    Autorzy:
    Tussiwand R; Department of Pathology and Immunology, Washington University School of Medicine, 660 South Euclid Avenue, St Louis, Missouri 63110, USA.
    Lee WL
    Murphy TL
    Mashayekhi M
    KC W
    Albring JC
    Satpathy AT
    Rotondo JA
    Edelson BT
    Kretzer NM
    Wu X
    Weiss LA
    Glasmacher E
    Li P
    Liao W
    Behnke M
    Lam SS
    Aurthur CT
    Leonard WJ
    Singh H
    Stallings CL
    Sibley LD
    Schreiber RD
    Murphy KM
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    Źródło:
    Nature [Nature] 2012 Oct 25; Vol. 490 (7421), pp. 502-7. Date of Electronic Publication: 2012 Sep 19.
    Typ publikacji:
    Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
    MeSH Terms:
    Basic-Leucine Zipper Transcription Factors/*metabolism
    Dendritic Cells/*cytology
    Dendritic Cells/*metabolism
    Interferon Regulatory Factors/*metabolism
    Animals ; Antigen Presentation ; Antigens, CD/metabolism ; Basic-Leucine Zipper Transcription Factors/chemistry ; Basic-Leucine Zipper Transcription Factors/deficiency ; Basic-Leucine Zipper Transcription Factors/genetics ; CD4-Positive T-Lymphocytes/cytology ; CD4-Positive T-Lymphocytes/immunology ; CD8 Antigens/immunology ; CD8 Antigens/metabolism ; CTLA-4 Antigen/metabolism ; Cell Differentiation ; Cell Line, Tumor ; Cell Lineage ; Dendritic Cells/immunology ; Female ; Fibrosarcoma/immunology ; Fibrosarcoma/metabolism ; Fibrosarcoma/pathology ; Gene Expression Regulation ; Integrin alpha Chains/metabolism ; Interferon Regulatory Factors/deficiency ; Interferon Regulatory Factors/genetics ; Interleukin-10/metabolism ; Interleukin-12/immunology ; Interleukin-12/metabolism ; Leucine Zippers ; Male ; Mice ; Mice, Inbred C57BL ; Neoplasm Transplantation ; Oncogene Protein p65(gag-jun)/metabolism ; Protein Binding ; Protein Structure, Tertiary ; Repressor Proteins/deficiency ; Repressor Proteins/genetics ; T-Lymphocytes, Helper-Inducer/cytology ; T-Lymphocytes, Helper-Inducer/immunology ; T-Lymphocytes, Helper-Inducer/metabolism ; Toxoplasma/immunology
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
      Wyświetlanie 1-6 z 6

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