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Tytuł:
Do cathepsins play a role in abdominal aortic aneurysm pathogenesis?
Autorzy:
Sukhova GK; Division of Cardiovascular Medicine, Brigham and Women's Hospital and Harvard Medical School, 77 Avenue Louis Pasteur, NRB-7, Room 730J, Boston, MA 02115, USA. />Shi GP
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Źródło:
Annals of the New York Academy of Sciences [Ann N Y Acad Sci] 2006 Nov; Vol. 1085, pp. 161-9.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Aortic Aneurysm, Abdominal/*enzymology
Aortic Aneurysm, Abdominal/*etiology
Cathepsins/*metabolism
Animals ; Aortic Aneurysm, Abdominal/pathology ; Apoptosis ; Atherosclerosis/enzymology ; Atherosclerosis/pathology ; Collagen/metabolism ; Cysteine Endopeptidases/metabolism ; Humans ; Neovascularization, Pathologic/drug therapy ; Neovascularization, Pathologic/enzymology ; Protease Inhibitors/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Mast cells promote atherosclerosis by releasing proinflammatory cytokines.
Autorzy:
Sun J; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Cardiovascular Medicine NRB-7, 77 Avenue Louis Pasteur, Boston, Massachusetts 02115, USA.
Sukhova GK
Wolters PJ
Yang M
Kitamoto S
Libby P
MacFarlane LA
Mallen-St Clair J
Shi GP
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Źródło:
Nature medicine [Nat Med] 2007 Jun; Vol. 13 (6), pp. 719-24. Date of Electronic Publication: 2007 Jun 03.
Typ publikacji:
Comparative Study; Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Atherosclerosis/*immunology
Atherosclerosis/*pathology
Cytokines/*metabolism
Inflammation Mediators/*physiology
Mast Cells/*immunology
Mast Cells/*metabolism
Animals ; Atherosclerosis/metabolism ; Humans ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Transgenic
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Cathepsin L activity controls adipogenesis and glucose tolerance.
Autorzy:
Yang M; Cardiovascular Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts 02115, USA.
Zhang Y
Pan J
Sun J
Liu J
Libby P
Sukhova GK
Doria A
Katunuma N
Peroni OD
Guerre-Millo M
Kahn BB
Clement K
Shi GP
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Źródło:
Nature cell biology [Nat Cell Biol] 2007 Aug; Vol. 9 (8), pp. 970-7. Date of Electronic Publication: 2007 Jul 22.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Glucose Intolerance*
Adipogenesis/*physiology
Cathepsins/*metabolism
Cysteine Endopeptidases/*metabolism
Adipocytes/cytology ; Adipocytes/physiology ; Animals ; Body Weight ; CCAAT-Enhancer-Binding Protein-alpha/genetics ; CCAAT-Enhancer-Binding Protein-alpha/metabolism ; Cathepsin L ; Cathepsins/antagonists & inhibitors ; Cathepsins/genetics ; Cell Differentiation/physiology ; Cells, Cultured ; Cysteine Endopeptidases/genetics ; Epoxy Compounds/metabolism ; Fibronectins/metabolism ; Glucose/metabolism ; Humans ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Obese ; PPAR gamma/genetics ; PPAR gamma/metabolism ; Pyridines/metabolism ; Receptor, IGF Type 1/metabolism ; Receptor, Insulin/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Cathepsin L activity controls adipogenesis and glucose tolerance.
Autorzy:
Min Yang
Yaou Zhang
Jiehong Pan
Jiusong Sun
Jian Liu
Libby, Peter
Sukhova, Galina K.
Doria, Alessandro
Katunuma, Nobuhiko
Peroni, Odile D.
Guerre-Millo, Michèle
Kahn, Barbara B.
Clement, Karine
Guo-Ping Shi
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Temat:
GLUCOSE
CYSTEINE proteinases
FIBRONECTINS
INSULIN
BODY weight
METABOLIC disorders
Źródło:
Nature Cell Biology; Aug2007, Vol. 9 Issue 8, p978-981, 4p, 8 Black and White Photographs, 1 Diagram, 4 Graphs
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Genetic deficiency and pharmacological stabilization of mast cells reduce diet-induced obesity and diabetes in mice.
Autorzy:
Liu J; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
Divoux A
Sun J
Zhang J
Clément K
Glickman JN
Sukhova GK
Wolters PJ
Du J
Gorgun CZ
Doria A
Libby P
Blumberg RS
Kahn BB
Hotamisligil GS
Shi GP
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Źródło:
Nature medicine [Nat Med] 2009 Aug; Vol. 15 (8), pp. 940-5. Date of Electronic Publication: 2009 Jul 26.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Cromolyn Sodium/*therapeutic use
Diabetes Mellitus, Experimental/*etiology
Mast Cells/*drug effects
Obesity/*drug therapy
Obesity/*genetics
Proto-Oncogene Proteins c-kit/*genetics
Animals ; Diabetes Mellitus, Experimental/drug therapy ; Diabetes Mellitus, Experimental/genetics ; Diabetes Mellitus, Experimental/immunology ; Diabetes Mellitus, Type 2/complications ; Diabetes Mellitus, Type 2/drug therapy ; Diabetes Mellitus, Type 2/genetics ; Diabetes Mellitus, Type 2/immunology ; Diet, Atherogenic ; Drug Evaluation, Preclinical ; Female ; Humans ; Male ; Mast Cells/metabolism ; Mice ; Mice, Transgenic ; Obesity/complications ; Obesity/etiology ; Obesity/immunology ; Organ Specificity/genetics ; Proto-Oncogene Proteins c-kit/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Cystatin C deficiency promotes epidermal dysplasia in K14-HPV16 transgenic mice.
Autorzy:
Yu W; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, United States of America.
Liu J
Shi MA
Wang J
Xiang M
Kitamoto S
Wang B
Sukhova GK
Murphy GF
Orasanu G
Grubb A
Shi GP
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Źródło:
PloS one [PLoS One] 2010 Nov 15; Vol. 5 (11), pp. e13973. Date of Electronic Publication: 2010 Nov 15.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Cystatin C/*deficiency
Epidermis/*metabolism
Keratin-14/*genetics
Oncogene Proteins, Viral/*genetics
Animals ; Apoptosis ; Cathepsins/genetics ; Cathepsins/metabolism ; Cell Proliferation ; Cystatin C/genetics ; Cystatin C/metabolism ; Disease Progression ; Epidermis/pathology ; Female ; Human papillomavirus 16/genetics ; Humans ; Hyperplasia ; Immunoblotting ; Male ; Mice ; Mice, 129 Strain ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Transgenic ; Papillomavirus E7 Proteins/genetics ; Promoter Regions, Genetic/genetics ; Repressor Proteins/genetics ; Reverse Transcriptase Polymerase Chain Reaction ; Skin Neoplasms/genetics ; Skin Neoplasms/metabolism ; Skin Neoplasms/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Local proliferation dominates lesional macrophage accumulation in atherosclerosis.
Autorzy:
Robbins CS; 1] Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA. [2] Toronto General Research Institute, University Health Network, Toronto, Ontario, Canada. [3] Department of Laboratory Medicine and Pathobiology, University of Toronto, Toronto, Ontario, Canada. [4] Department of Immunology, University of Toronto, Toronto, Ontario, Canada. [5].
Hilgendorf I
Weber GF
Theurl I
Iwamoto Y
Figueiredo JL
Gorbatov R
Sukhova GK
Gerhardt LM
Smyth D
Zavitz CC
Shikatani EA
Parsons M
van Rooijen N
Lin HY
Husain M
Libby P
Nahrendorf M
Weissleder R
Swirski FK
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Źródło:
Nature medicine [Nat Med] 2013 Sep; Vol. 19 (9), pp. 1166-72. Date of Electronic Publication: 2013 Aug 11.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Proliferation*
Atherosclerosis/*immunology
Atherosclerosis/*pathology
Macrophages/*physiology
Animals ; Cells, Cultured ; Inflammation ; Macrophages/immunology ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Monocytes/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Interleukin 18 function in atherosclerosis is mediated by the interleukin 18 receptor and the Na-Cl co-transporter.
Autorzy:
Wang J; 1] Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA. [2] State Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences, Department of Pathophysiology, Peking Union Medical College, Tsinghua University, Beijing, China.
Sun C; 1] Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA. [2] Department of Biochemistry and Molecular Biology, Nanjing Medical University, Nanjing, China.
Gerdes N; 1] Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA. [2] Institute for Cardiovascular Prevention (IPEK), Ludwig-Maximilians University Munich, Munich, Germany.
Liu C; 1] Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA. [2] Institute of Clinical Medicine, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Liao M; 1] Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA. [2] Institute of Cardiology, Union Hospital, Tongji Medical College of Huazhong University of Science and Technology, Wuhan, China.
Liu J; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
Shi MA; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
He A; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
Zhou Y; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
Sukhova GK; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
Chen H; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
Cheng XW; Departments of Cardiology and Geriatrics, Graduate School of Medicine, Nagoya University, Nagoya, Japan.
Kuzuya M; Departments of Cardiology and Geriatrics, Graduate School of Medicine, Nagoya University, Nagoya, Japan.
Murohara T; Departments of Cardiology and Geriatrics, Graduate School of Medicine, Nagoya University, Nagoya, Japan.
Zhang J; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
Cheng X; 1] Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA. [2] Institute of Cardiology, Union Hospital, Tongji Medical College of Huazhong University of Science and Technology, Wuhan, China.
Jiang M; Department of Molecular Genetics, Biochemistry and Microbiology, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.
Shull GE; Department of Molecular Genetics, Biochemistry and Microbiology, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA.
Rogers S; Division of Nephrology and Hypertension, Oregon Health and Science University, Portland, Oregon, USA.
Yang CL; Division of Nephrology and Hypertension, Oregon Health and Science University, Portland, Oregon, USA.
Ke Q; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
Jelen S; Department of Physiology, Radboud University Nijmegen Medical Centre, Nijmegen, the Netherlands.
Bindels R; Department of Physiology, Radboud University Nijmegen Medical Centre, Nijmegen, the Netherlands.
Ellison DH; Division of Nephrology and Hypertension, Oregon Health and Science University, Portland, Oregon, USA.
Jarolim P; Department of Pathology, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
Libby P; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
Shi GP; Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA.
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Źródło:
Nature medicine [Nat Med] 2015 Jul; Vol. 21 (7), pp. 820-6. Date of Electronic Publication: 2015 Jun 22.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Atherosclerosis/*metabolism
Interleukin-18/*metabolism
Receptors, Interleukin-18/*metabolism
Animals ; Aorta/pathology ; Apolipoproteins E/deficiency ; Apolipoproteins E/metabolism ; Atherosclerosis/pathology ; COS Cells ; Chemokines/metabolism ; Chlorocebus aethiops ; Macrophages/metabolism ; Mice ; Protein Binding ; Receptors, Interleukin-18/deficiency ; Signal Transduction ; Solute Carrier Family 12, Member 3/metabolism ; Tunica Intima/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Wyświetlanie 1-20 z 72

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