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Wyszukujesz frazę ""Sun, Lei"" wg kryterium: Autor


Tytuł :
Effects of glucagon-like peptide-1 analog liraglutide on the systemic inflammation in high-fat-diet-induced mice.
Autorzy :
Sha S; Department of Endocrinology, Qilu Hospital of Shandong University, Jinan, Shandong, China.; Institute of Endocrine and Metabolic Diseases of Shandong University, Jinan, Shandong, China.; Key Laboratory of Endocrine and Metabolic Diseases, Shandong Province medicine& health, Jinan, Shandong, China.
Liu X; Department of Endocrinology, Qilu Hospital of Shandong University, Jinan, Shandong, China.; Institute of Endocrine and Metabolic Diseases of Shandong University, Jinan, Shandong, China.; Key Laboratory of Endocrine and Metabolic Diseases, Shandong Province medicine& health, Jinan, Shandong, China.
Zhao R; Department of Endocrinology, Qilu Hospital of Shandong University, Jinan, Shandong, China.; Institute of Endocrine and Metabolic Diseases of Shandong University, Jinan, Shandong, China.; Key Laboratory of Endocrine and Metabolic Diseases, Shandong Province medicine& health, Jinan, Shandong, China.
Qing L; Department of Endocrinology, Qilu Hospital of Shandong University, Jinan, Shandong, China.; Institute of Endocrine and Metabolic Diseases of Shandong University, Jinan, Shandong, China.; Key Laboratory of Endocrine and Metabolic Diseases, Shandong Province medicine& health, Jinan, Shandong, China.
He Q; Department of Endocrinology, Qilu Hospital of Shandong University, Jinan, Shandong, China.; Institute of Endocrine and Metabolic Diseases of Shandong University, Jinan, Shandong, China.; Key Laboratory of Endocrine and Metabolic Diseases, Shandong Province medicine& health, Jinan, Shandong, China.
Sun L; Department of Endocrinology, Qilu Hospital of Shandong University, Jinan, Shandong, China. .; Institute of Endocrine and Metabolic Diseases of Shandong University, Jinan, Shandong, China. .; Key Laboratory of Endocrine and Metabolic Diseases, Shandong Province medicine& health, Jinan, Shandong, China. .
Chen L; Department of Endocrinology, Qilu Hospital of Shandong University, Jinan, Shandong, China. .; Institute of Endocrine and Metabolic Diseases of Shandong University, Jinan, Shandong, China. .; Key Laboratory of Endocrine and Metabolic Diseases, Shandong Province medicine& health, Jinan, Shandong, China. .
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Źródło :
Endocrine [Endocrine] 2019 Dec; Vol. 66 (3), pp. 494-502. Date of Electronic Publication: 2019 Sep 21.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Hypoglycemic Agents/*therapeutic use
Inflammation/*drug therapy
Liraglutide/*therapeutic use
Metabolic Syndrome/*prevention & control
T-Lymphocyte Subsets/*drug effects
Animals ; Diet, High-Fat/adverse effects ; Drug Evaluation, Preclinical ; Male ; Metabolic Syndrome/etiology ; Mice, Inbred C57BL ; Random Allocation
Czasopismo naukowe
Tytuł :
Isoalantolactone suppresses LPS-induced inflammation by inhibiting TRAF6 ubiquitination and alleviates acute lung injury.
Autorzy :
Ding YH; Engineering Research Center of Cell and Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China.
Song YD; Department of Clinical Laboratory, Shanghai Pudong Hospital, Fudan University, Shanghai, 201399, China.
Wu YX; Engineering Research Center of Cell and Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China.
He HQ; Engineering Research Center of Cell and Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China.
Yu TH; Engineering Research Center of Cell and Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China.
Hu YD; Engineering Research Center of Cell and Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China.
Zhang DP; Engineering Research Center of Cell and Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China.
Jiang HC; Engineering Research Center of Cell and Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China.
Yu KK; Engineering Research Center of Cell and Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China.
Li XZ; Department of Clinical Laboratory, Shanghai Pudong Hospital, Fudan University, Shanghai, 201399, China.
Sun L; Engineering Research Center of Cell and Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China.
Qian F; Engineering Research Center of Cell and Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China. .; Research Center for Cancer Precision Medicine, Department of Medical Oncology, Bengbu Medical College, Bengbu, 233003, China. .; Jiangsu Center for the Collaboration and Innovation of Cancer Biotherapy, Cancer Institute, Xuzhou Medical University, Xuzhou, 221004, China. .
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Źródło :
Acta pharmacologica Sinica [Acta Pharmacol Sin] 2019 Jan; Vol. 40 (1), pp. 64-74. Date of Electronic Publication: 2018 Jul 16.
Typ publikacji :
Journal Article
MeSH Terms :
Acute Lung Injury/*drug therapy
Anti-Inflammatory Agents/*therapeutic use
Inflammation/*drug therapy
Sesquiterpenes/*therapeutic use
TNF Receptor-Associated Factor 6/*metabolism
Ubiquitination/*drug effects
Acute Lung Injury/chemically induced ; Acute Lung Injury/pathology ; Animals ; Cytokines/metabolism ; HEK293 Cells ; Humans ; Intracellular Signaling Peptides and Proteins ; Lipopolysaccharides ; Lung/pathology ; Male ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; NF-kappa B p50 Subunit/metabolism ; Signal Transduction/drug effects
Czasopismo naukowe
Tytuł :
Excessive endosomal TLR signaling causes inflammatory disease in mice with defective SMCR8-WDR41-C9ORF72 complex function.
Autorzy :
McAlpine W; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Sun L; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Wang KW; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Liu A; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Jain R; Division of Gastroenterology, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
San Miguel M; Division of Gastroenterology, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Wang J; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Zhang Z; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Hayse B; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
McAlpine SG; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Choi JH; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Zhong X; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Ludwig S; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Russell J; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Zhan X; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Choi M; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Li X; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Tang M; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Moresco EMY; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
Beutler B; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505; .
Turer E; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505; .; Division of Gastroenterology, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
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Źródło :
Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2018 Dec 04; Vol. 115 (49), pp. E11523-E11531. Date of Electronic Publication: 2018 Nov 15.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
C9orf72 Protein/*metabolism
Carrier Proteins/*metabolism
Inflammation/*metabolism
Intracellular Signaling Peptides and Proteins/*metabolism
Toll-Like Receptors/*metabolism
Animals ; Autoimmune Diseases/genetics ; Autoimmune Diseases/metabolism ; Autophagy-Related Proteins ; C9orf72 Protein/genetics ; Carrier Proteins/genetics ; Colitis/chemically induced ; Dextran Sulfate ; Gene Expression Regulation ; Hematopoiesis/genetics ; Intracellular Signaling Peptides and Proteins/genetics ; Mice ; Mice, Knockout ; Mutation ; Signal Transduction/immunology ; Toll-Like Receptors/genetics
Czasopismo naukowe
Tytuł :
Emodin alleviates alternatively activated macrophage and asthmatic airway inflammation in a murine asthma model.
Autorzy :
Song YD; Department of Clinical Laboratory, Shanghai Pudong Hospital, Fudan University, Shanghai, 201399, China.
Li XZ; Department of Clinical Laboratory, Shanghai Pudong Hospital, Fudan University, Shanghai, 201399, China.
Wu YX; Engineering Research Center of Cell & Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China.
Shen Y; Department of Respiratory Medicine, Shanghai Pudong Hospital, Fudan University, Shanghai, 201399, China.
Liu FF; Department of Clinical Laboratory, Shanghai Pudong Hospital, Fudan University, Shanghai, 201399, China.
Gao PP; Department of Pharmaceutical Preparation Section, Shanghai Pudong Hospital, Fudan University, Shanghai, 201399, China.
Sun L; Engineering Research Center of Cell & Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China. .
Qian F; Engineering Research Center of Cell & Therapeutic Antibody, Ministry of Education, School of Pharmacy, Shanghai Jiao Tong University, Shanghai, 200240, China. .; Research Center for Cancer Precision Medicine, Bengbu Medical College, Bengbu, 233003, China. .
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Źródło :
Acta pharmacologica Sinica [Acta Pharmacol Sin] 2018 Aug; Vol. 39 (8), pp. 1317-1325. Date of Electronic Publication: 2018 Feb 08.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Asthmatic Agents/*therapeutic use
Asthma/*drug therapy
Emodin/*therapeutic use
Inflammation/*drug therapy
Macrophage Activation/*drug effects
Animals ; Asthma/pathology ; Bronchoalveolar Lavage Fluid/cytology ; Immunoglobulin E/metabolism ; Inflammation/pathology ; Interleukin-4/metabolism ; Interleukin-5/metabolism ; Lung/pathology ; Macrophages/metabolism ; Male ; Mice, Inbred C57BL ; Pulmonary Eosinophilia/drug therapy ; Pulmonary Eosinophilia/pathology
Czasopismo naukowe
Tytuł :
Immune response of bovine sourced cross-linked collagen sponge for hemostasis.
Autorzy :
Zhang L; 1 Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, P. R. China.
Niu X; 1 Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, P. R. China.
Sun L; 1 Key Laboratory for Biomechanics and Mechanobiology of Ministry of Education, School of Biological Science and Medical Engineering, Beihang University, Beijing, P. R. China.
She Z; 2 Shenzhen Lando Biomaterials Co., Ltd, Shenzhen, P. R. China.
Tan R; 2 Shenzhen Lando Biomaterials Co., Ltd, Shenzhen, P. R. China.
Wang W; 3 Department of Immunology, School of Basic Medical Sciences, 33133 Peking University , Key Laboratory of Medical Immunology, Ministry of Health (Peking University), Beijing, P. R. China.
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Źródło :
Journal of biomaterials applications [J Biomater Appl] 2018 Feb; Vol. 32 (7), pp. 920-931. Date of Electronic Publication: 2017 Dec 04.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Biocompatible Materials/*adverse effects
Collagen/*adverse effects
Cross-Linking Reagents/*adverse effects
Inflammation/*etiology
Animals ; Apoptosis ; Biocompatible Materials/chemistry ; CD4-CD8 Ratio ; Cattle ; Cell Proliferation ; Collagen/chemistry ; Collagen/immunology ; Cross-Linking Reagents/chemistry ; Female ; Hemostasis ; Immunity ; Inflammation/immunology ; Lymphocytes/cytology ; Lymphocytes/immunology ; Mice, Inbred BALB C ; Prostheses and Implants/adverse effects ; Spleen/cytology ; Spleen/immunology
Czasopismo naukowe
Tytuł :
[Effect of extracellular cyclophilin A on inflammatory response and anti-inflammatory activity of antibody against cyclophilin A].
Autorzy :
Li W; Key Laboratory of Pathogenic Microbiology and Immunology, Institute of Microbiology, Chinese Academy of Sciences, Beijing 100101, China.; University of Chinese Academy of Sciences, Beijing 100049, China.
Liu W; Key Laboratory of Pathogenic Microbiology and Immunology, Institute of Microbiology, Chinese Academy of Sciences, Beijing 100101, China.; University of Chinese Academy of Sciences, Beijing 100049, China.
Chen C; Key Laboratory of Pathogenic Microbiology and Immunology, Institute of Microbiology, Chinese Academy of Sciences, Beijing 100101, China.; University of Chinese Academy of Sciences, Beijing 100049, China.
Fan W; Key Laboratory of Pathogenic Microbiology and Immunology, Institute of Microbiology, Chinese Academy of Sciences, Beijing 100101, China.
Zhang H; Key Laboratory of Pathogenic Microbiology and Immunology, Institute of Microbiology, Chinese Academy of Sciences, Beijing 100101, China.
Liu W; Key Laboratory of Pathogenic Microbiology and Immunology, Institute of Microbiology, Chinese Academy of Sciences, Beijing 100101, China.; University of Chinese Academy of Sciences, Beijing 100049, China.
Sun L; Key Laboratory of Pathogenic Microbiology and Immunology, Institute of Microbiology, Chinese Academy of Sciences, Beijing 100101, China.; University of Chinese Academy of Sciences, Beijing 100049, China.
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Źródło :
Sheng wu gong cheng xue bao = Chinese journal of biotechnology [Sheng Wu Gong Cheng Xue Bao] 2018 Jan 25; Vol. 34 (1), pp. 90-101.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Antibodies/*immunology
Cyclophilin A/*pharmacology
Inflammation/*drug therapy
Lung/*drug effects
Animals ; Cyclophilin A/immunology ; Cytokines/immunology ; Leukocytes/immunology ; Lung/immunology ; Mice ; Recombinant Proteins/immunology ; Recombinant Proteins/pharmacology
Czasopismo naukowe
Tytuł :
Protein phosphatase 2A activation attenuates inflammation in murine models of acute lung injury.
Autorzy :
McHugh WM; Division of Pediatric Critical Care Medicine, Department of Pediatrics and Communicable Diseases, University of Michigan School of Medicine, and C. S. Mott Children's Hospital, University of Michigan, Ann Arbor, Michigan.
Russell WW; Division of Pediatric Critical Care Medicine, Department of Pediatrics and Communicable Diseases, University of Michigan School of Medicine, and C. S. Mott Children's Hospital, University of Michigan, Ann Arbor, Michigan.
Fleszar AJ; Division of Pediatric Critical Care Medicine, Department of Pediatrics and Communicable Diseases, University of Michigan School of Medicine, and C. S. Mott Children's Hospital, University of Michigan, Ann Arbor, Michigan.
Rodenhouse PE; Division of Pediatric Critical Care Medicine, Department of Pediatrics and Communicable Diseases, University of Michigan School of Medicine, and C. S. Mott Children's Hospital, University of Michigan, Ann Arbor, Michigan.
Rietberg SP; Division of Pediatric Critical Care Medicine, Department of Pediatrics and Communicable Diseases, University of Michigan School of Medicine, and C. S. Mott Children's Hospital, University of Michigan, Ann Arbor, Michigan.
Sun L; Division of Pediatric Critical Care Medicine, Department of Pediatrics and Communicable Diseases, University of Michigan School of Medicine, and C. S. Mott Children's Hospital, University of Michigan, Ann Arbor, Michigan.
Shanley TP; Division of Pediatric Critical Care Medicine, Department of Pediatrics and Communicable Diseases, University of Michigan School of Medicine, and C. S. Mott Children's Hospital, University of Michigan, Ann Arbor, Michigan.
Cornell TT; Division of Pediatric Critical Care Medicine, Department of Pediatrics and Communicable Diseases, University of Michigan School of Medicine, and C. S. Mott Children's Hospital, University of Michigan, Ann Arbor, Michigan .
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Źródło :
American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2016 Nov 01; Vol. 311 (5), pp. L903-L912. Date of Electronic Publication: 2016 Sep 16.
Typ publikacji :
Journal Article
MeSH Terms :
Acute Lung Injury/*enzymology
Inflammation/*metabolism
Inflammation/*pathology
Protein Phosphatase 2/*metabolism
Acute Lung Injury/pathology ; Adenosine/analogs & derivatives ; Adenosine/pharmacology ; Animals ; Cell Line ; Chemokines/biosynthesis ; Disease Models, Animal ; Endotoxins ; Enzyme Activation/drug effects ; Epithelial Cells/drug effects ; Epithelial Cells/metabolism ; JNK Mitogen-Activated Protein Kinases/metabolism ; Macrophages, Alveolar/drug effects ; Macrophages, Alveolar/metabolism ; Mice, Inbred C57BL ; Phosphorylation/drug effects ; Pseudomonas aeruginosa/drug effects ; Respiratory Distress Syndrome/enzymology ; Respiratory Distress Syndrome/pathology
Czasopismo naukowe
Tytuł :
Role of G protein-coupled receptors in inflammation.
Autorzy :
Sun L; School of Pharmacy, Shanghai Jiao Tong University, China.
Ye RD
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Źródło :
Acta pharmacologica Sinica [Acta Pharmacol Sin] 2012 Mar; Vol. 33 (3), pp. 342-50. Date of Electronic Publication: 2012 Feb 27.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms :
Inflammation/*metabolism
Receptors, G-Protein-Coupled/*metabolism
Animals ; Humans ; Signal Transduction
Czasopismo naukowe
Tytuł :
Mitogen-activated protein kinase phosphatase 2 regulates the inflammatory response in sepsis.
Autorzy :
Cornell TT; Division of Pediatric Critical Care Medicine, Department of Pediatrics and Communicable Diseases, University of Michigan School of Medicine, and CS Mott Children's Hospital, Ann Arbor, Michigan 48109-0243, USA. />Rodenhouse P
Cai Q
Sun L
Shanley TP
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Źródło :
Infection and immunity [Infect Immun] 2010 Jun; Vol. 78 (6), pp. 2868-76. Date of Electronic Publication: 2010 Mar 29.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Inflammation/*immunology
Protein Tyrosine Phosphatases/*immunology
Sepsis/*immunology
Animals ; Bacterial Infections/immunology ; Cytokines/blood ; Dual Specificity Phosphatase 1/analysis ; Female ; Lipopolysaccharides/immunology ; Lipopolysaccharides/toxicity ; MAP Kinase Kinase 4/analysis ; Macrophages/chemistry ; Macrophages/immunology ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Peritonitis/immunology ; Protein Tyrosine Phosphatases/deficiency ; Survival Analysis ; p38 Mitogen-Activated Protein Kinases/analysis
Czasopismo naukowe
Tytuł :
Beneficial alterations in body composition, physical performance, oxidative stress, inflammatory markers, and adipocytokines induced by long-term high-intensity interval training in an aged rat model
Autorzy :
Li, Fang-Hui
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Źródło :
In Experimental Gerontology November 2018 113:150-162
Czasopismo naukowe

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