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Wyszukujesz frazę ""Tanihata, Jun"" wg kryterium: Autor

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Wyświetlanie 1-20 z 72
Tytuł:
Full-length human dystrophin on human artificial chromosome compensates for mouse dystrophin deficiency in a Duchenne muscular dystrophy mouse model
Autorzy:
Hiramuki, Yosuke
Abe, Satoshi
Uno, NarumiAff1, Aff3, Aff4
Kazuki, Kanako
Takata, Shuta
Miyamoto, HitomaruAff5, Aff6
Takayama, Haruka
Morimoto, Kayoko
Takehara, Shoko
Osaki, MitsuhikoAff1, Aff7
Tanihata, JunAff8, Aff9
Takeda, Shin’ichi
Tomizuka, Kazuma
Oshimura, Mitsuo
Kazuki, YasuhiroAff1, Aff3, Aff5, Aff6, Aff10, Aff11, IDs41598023314813_cor15
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Źródło:
Scientific Reports. 13(1)
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Urinary titin is not an early biomarker of skeletal muscle atrophy induced by muscle denervation in mice.
Autorzy:
Tanihata J; Department of Cell Physiology, The Jikei University School of Medicine, Nishishinbashi, Minato-ku, Tokyo, Japan.
Minamisawa S; Department of Cell Physiology, The Jikei University School of Medicine, Nishishinbashi, Minato-ku, Tokyo, Japan.
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Źródło:
PloS one [PLoS One] 2023 Aug 15; Vol. 18 (8), pp. e0289185. Date of Electronic Publication: 2023 Aug 15 (Print Publication: 2023).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Muscle, Skeletal*/metabolism
Muscle Denervation*
Mice ; Male ; Animals ; Connectin/metabolism ; Muscular Atrophy/pathology ; Biomarkers/metabolism ; Denervation/adverse effects ; Protein Kinases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Involvement of Parkin‐mediated mitophagy in the pathogenesis of chronic obstructive pulmonary disease‐related sarcopenia.
Autorzy:
Ito, Akihiko
Hashimoto, Mitsuo
Tanihata, Jun
Matsubayashi, Sachi
Sasaki, Ryoko
Fujimoto, Shota
Kawamoto, Hironori
Hosaka, Yusuke
Ichikawa, Akihiro
Kadota, Tsukasa
Fujita, Yu
Takekoshi, Daisuke
Ito, Sabro
Minagawa, Shunsuke
Numata, Takanori
Hara, Hiromichi
Matsuoka, Tatsuki
Udaka, Jun
Araya, Jun
Saito, Mitsuru
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Źródło:
Journal of Cachexia, Sarcopenia & Muscle; Jun2022, Vol. 13 Issue 3, p1864-1882, 19p
Czasopismo naukowe
Szczegóły
Tytuł:
Transcriptome analysis of gravitational effects on mouse skeletal muscles under microgravity and artificial 1 g onboard environment
Autorzy:
Okada, RisaAff1, Aff2
Fujita, Shin-ichiroAff3, Aff4
Suzuki, RikuAff5, Aff6
Hayashi, TakutoAff3, Aff5
Tsubouchi, Hirona
Kato, ChihiroAff5, Aff7
Sadaki, Shunya
Kanai, MahoAff5, Aff6
Fuseya, SayakaAff3, Aff5
Inoue, YuriAff3, Aff5
Jeon, Hyojung
Hamada, Michito
Kuno, AkihiroAff5, Aff6
Ishii, Akiko
Tamaoka, Akira
Tanihata, Jun
Ito, Naoki
Shiba, DaiAff1, Aff2
Shirakawa, MasakiAff1, Aff2
Muratani, MasafumiAff1, Aff4
Kudo, TakashiAff1, Aff5
Takahashi, SatoruAff1, Aff5
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Źródło:
Scientific Reports. 11(1)
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The nSMase2/Smpd3 gene modulates the severity of muscular dystrophy and the emotional stress response in mdx mice.
Autorzy:
Matsuzaka Y; Administrative Section of Radiation Protection, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.; Medical Molecular Informatics, Meiji Pharmaceutical University, Noshio, Kiyose, Tokyo, Japan.
Tanihata J; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
Ooshima Y; Administrative Section of Radiation Protection, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Yamada D; Department of Degenerative Neurological Diseases, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.; Laboratory of Pharmacology, Department of Pharmacy, Faculty of Pharmaceutical Sciences, Tokyo University of Science, Noda, Chiba, Japan.
Sekiguchi M; Department of Degenerative Neurological Diseases, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Miyatake S; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Aoki Y; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Terumitsu M; Administrative Section of Radiation Protection, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Yashiro R; Administrative Section of Radiation Protection, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Komaki H; Department of Child Neurology, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Ishiyama A; Department of Child Neurology, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Oya Y; Department of Neurology, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Inoue YU; Department of Biochemistry and Cellular Biology, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Inoue T; Department of Biochemistry and Cellular Biology, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Takeda S; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Hashido K; Administrative Section of Radiation Protection, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan. .
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Źródło:
BMC medicine [BMC Med] 2020 Nov 19; Vol. 18 (1), pp. 343. Date of Electronic Publication: 2020 Nov 19.
Typ publikacji:
Journal Article
MeSH Terms:
Muscular Dystrophy, Duchenne/*genetics
Sphingomyelin Phosphodiesterase/*metabolism
Animals ; Disease Models, Animal ; Dystrophin/genetics ; Dystrophin/metabolism ; Dystrophin/pharmacology ; Humans ; Male ; Mice ; Mice, Inbred mdx ; Mice, Knockout
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
iNOS is not responsible for RyR1 S-nitrosylation in mdx mice with truncated dystrophin.
Autorzy:
Nogami K; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Tokyo, Japan.; Department of Neurology, Neurological Institute, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.
Maruyama Y; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Tokyo, Japan.
Elhussieny A; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Tokyo, Japan.; Department of Neurology, Faculty of Medicine, Minia University, Minia, Egypt.
Sakai-Takemura F; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Tokyo, Japan.
Tanihata J; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Tokyo, Japan.; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
Kira JI; Department of Neurology, Neurological Institute, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.
Miyagoe-Suzuki Y; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Tokyo, Japan.
Takeda S; National Center of Neurology and Psychiatry, Tokyo, Japan. .
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Źródło:
BMC musculoskeletal disorders [BMC Musculoskelet Disord] 2020 Jul 21; Vol. 21 (1), pp. 479. Date of Electronic Publication: 2020 Jul 21.
Typ publikacji:
Journal Article
MeSH Terms:
Dystrophin*/genetics
Dystrophin*/metabolism
Muscular Dystrophy, Duchenne*/genetics
Nitric Oxide Synthase Type II*
Ryanodine Receptor Calcium Release Channel*/genetics
Animals ; Mice ; Mice, Inbred C57BL ; Mice, Inbred mdx ; Muscle, Skeletal/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Characterization of a novel microRNA, miR-188, elevated in serum of muscular dystrophy dog model.
Autorzy:
Shibasaki H; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.; Department of Gene Regulation, Faculty of Pharmaceutical Sciences, Tokyo University of Science, Noda, Chiba, Japan.
Imamura M; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Arima S; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Tanihata J; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.; Department of Cell Physiology, The Jikei University School of Medicine, Minato, Tokyo, Japan.
Kuraoka M; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.; Laboratory of Experimental Animal Science, Nippon Veterinary and Life Science University, Musashino, Tokyo, Japan.
Matsuzaka Y; Department of Medical Molecular Informatics, Meiji Pharmaceutical University, Kiyose, Tokyo, Japan.
Uchiumi F; Department of Gene Regulation, Faculty of Pharmaceutical Sciences, Tokyo University of Science, Noda, Chiba, Japan.
Tanuma SI; Department of Genomic Medicinal Science, Research Institute for Science and Technology, Organization for Research Advancement, Tokyo University of Science, Noda, Chiba, Japan.
Takeda S; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
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Źródło:
PloS one [PLoS One] 2019 Jan 30; Vol. 14 (1), pp. e0211597. Date of Electronic Publication: 2019 Jan 30 (Print Publication: 2019).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation*
Biomarkers/*blood
MicroRNAs/*genetics
Muscle, Skeletal/*metabolism
Muscular Dystrophy, Animal/*genetics
Muscular Dystrophy, Duchenne/*genetics
Animals ; Cell Differentiation ; Cells, Cultured ; Disease Models, Animal ; Dogs ; Mice ; Muscle, Skeletal/pathology ; Muscular Dystrophy, Animal/blood ; Muscular Dystrophy, Animal/pathology ; Muscular Dystrophy, Duchenne/blood ; Muscular Dystrophy, Duchenne/pathology ; Myoblasts/cytology ; Myoblasts/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Treatment with the anti-IL-6 receptor antibody attenuates muscular dystrophy via promoting skeletal muscle regeneration in dystrophin-/utrophin-deficient mice.
Autorzy:
Wada E; Department of Pathophysiology, Tokyo Medical University, 6-1-1 Shinjuku, Shinjuku, Tokyo, Japan. .; Department of Life Sciences, Graduate School of Arts and Sciences, The University of Tokyo, 3-8-1 Komaba, Meguro, Tokyo, Japan. .
Tanihata J; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, 4-1-1 Ogawa-Higashi, Kodaira, Tokyo, Japan.; Department of Cell Physiology, The Jikei University School of Medicine, 3-25-8, Nishi-Shimbashi, Minato-ku, Tokyo, Japan.
Iwamura A; Public Health, Department of Social Medicine, Osaka University Graduate School of Medicine, 2-2 Yamadaoka, Suita, Osaka, Japan.
Takeda S; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, 4-1-1 Ogawa-Higashi, Kodaira, Tokyo, Japan.
Hayashi YK; Department of Pathophysiology, Tokyo Medical University, 6-1-1 Shinjuku, Shinjuku, Tokyo, Japan.
Matsuda R; Department of Life Sciences, Graduate School of Arts and Sciences, The University of Tokyo, 3-8-1 Komaba, Meguro, Tokyo, Japan.
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Źródło:
Skeletal muscle [Skelet Muscle] 2017 Oct 27; Vol. 7 (1), pp. 23. Date of Electronic Publication: 2017 Oct 27.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antibodies/*administration & dosage
Muscle, Skeletal/*drug effects
Muscular Dystrophies/*drug therapy
Receptors, Interleukin-6/*immunology
Regeneration/*drug effects
Animals ; Creatine Kinase/blood ; Disease Models, Animal ; Dystrophin/genetics ; Fibrosis/complications ; Inflammation/complications ; Male ; Mice, Inbred C57BL ; Mice, Knockout ; Muscle Fibers, Skeletal/drug effects ; Muscle Fibers, Skeletal/pathology ; Muscle, Skeletal/pathology ; Muscle, Skeletal/physiopathology ; Muscular Dystrophies/complications ; Muscular Dystrophies/immunology ; Receptors, Interleukin-6/antagonists & inhibitors ; Satellite Cells, Skeletal Muscle/drug effects ; Signal Transduction/drug effects ; Utrophin/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Characterization and Functional Analysis of Extracellular Vesicles and Muscle-Abundant miRNAs (miR-1, miR-133a, and miR-206) in C2C12 Myocytes and mdx Mice.
Autorzy:
Matsuzaka Y; Administrative Section of Radiation Protection, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Tanihata J; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Komaki H; Department of Child Neurology, Hospital, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Ishiyama A; Department of Child Neurology, Hospital, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Oya Y; Department of Neurology, Hospital, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Rüegg U; Department of Pharmacology, Geneva-Lausanne School of Pharmaceutical Sciences, University of Geneva and University of Lausanne, Geneva, Switzerland.
Takeda SI; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Hashido K; Administrative Section of Radiation Protection, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
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Źródło:
PloS one [PLoS One] 2016 Dec 15; Vol. 11 (12), pp. e0167811. Date of Electronic Publication: 2016 Dec 15 (Print Publication: 2016).
Typ publikacji:
Journal Article
MeSH Terms:
Extracellular Vesicles/*metabolism
MicroRNAs/*metabolism
Muscular Dystrophy, Duchenne/*metabolism
Animals ; Cell Line ; Mice ; Mice, Inbred mdx ; Muscle, Skeletal/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Characterization and Functional Analysis of Extracellular Vesicles and Muscle-Abundant miRNAs (miR-1, miR-133a, and miR-206) in C2C12 Myocytes and mdx Mice.
Autorzy:
Matsuzaka, Yasunari
Tanihata, Jun
Komaki, Hirofumi
Ishiyama, Akihiko
Oya, Yasushi
Rüegg, Urs
Takeda, Shin-ichi
Hashido, Kazuo
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Temat:
TREATMENT of Duchenne muscular dystrophy
DUCHENNE muscular dystrophy
MICRORNA
EXTRACELLULAR matrix
GENE expression
CELL death
CERAMIDES
GENETICS
Źródło:
PLoS ONE; 12/15/2016, Vol. 11 Issue 12, p1-23, 23p
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Endogenous Multiple Exon Skipping and Back-Splicing at the DMD Mutation Hotspot.
Autorzy:
Suzuki H; School of Materials Science, Japan Advanced Institute of Science and Technology, Nomi, Ishikawa 923-1292, Japan. .
Aoki Y; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry (NCNP), Kodaira, Tokyo 187-8502, Japan. .
Kameyama T; Division of Gene Expression Mechanism, Institute for Comprehensive Medical Science, Fujita Health University, Toyoake, Aichi 470-1192, Japan. .
Saito T; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry (NCNP), Kodaira, Tokyo 187-8502, Japan. .
Masuda S; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry (NCNP), Kodaira, Tokyo 187-8502, Japan. .
Tanihata J; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry (NCNP), Kodaira, Tokyo 187-8502, Japan. tanihata@ncnp.go.jp.
Nagata T; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry (NCNP), Kodaira, Tokyo 187-8502, Japan. .; Graduate School of Medical and Dental Sciences, Tokyo Medical and Dental University, Bunkyoku, Tokyo 113-0034, Japan. .
Mayeda A; Division of Gene Expression Mechanism, Institute for Comprehensive Medical Science, Fujita Health University, Toyoake, Aichi 470-1192, Japan. .
Takeda S; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry (NCNP), Kodaira, Tokyo 187-8502, Japan. .
Tsukahara T; School of Materials Science, Japan Advanced Institute of Science and Technology, Nomi, Ishikawa 923-1292, Japan. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2016 Oct 13; Vol. 17 (10). Date of Electronic Publication: 2016 Oct 13.
Typ publikacji:
Journal Article
MeSH Terms:
Exons*
RNA Splicing*
Dystrophin/*genetics
Muscular Dystrophy, Duchenne/*genetics
RNA Precursors/*genetics
Gene Expression ; Humans ; Mutation ; RNA, Messenger/genetics ; Transcription, Genetic
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Generation of muscular dystrophy model rats with a CRISPR/Cas system.
Autorzy:
Nakamura K; Department of Veterinary Physiology, Graduate School of Agricultural and Life Sciences, The University of Tokyo, 1-1-1 Yayoi, Bunkyo-ku, Tokyo 113-8657, Japan.
Fujii W; Department of Animal Resource Sciences, Graduate School of Agricultural and Life Sciences, The University of Tokyo, 1-1-1 Yayoi, Bunkyo-ku, Tokyo 113-8657, Japan.
Tsuboi M; Department of Veterinary Pathology, Graduate School of Agricultural and Life Sciences, The University of Tokyo, 1-1-1 Yayoi, Bunkyo-ku, Tokyo 113-8657, Japan.
Tanihata J; Department of Molecular Therapy, National Institute of Neuroscience, National Center of Neurology and Psychiatry, 4-1-1 Ogawa-Higashi, Kodaira, Tokyo 187-8502, Japan.
Teramoto N; Department of Veterinary Physiology, Graduate School of Agricultural and Life Sciences, The University of Tokyo, 1-1-1 Yayoi, Bunkyo-ku, Tokyo 113-8657, Japan.
Takeuchi S; Department of Veterinary Physiology, Graduate School of Agricultural and Life Sciences, The University of Tokyo, 1-1-1 Yayoi, Bunkyo-ku, Tokyo 113-8657, Japan.
Naito K; Department of Animal Resource Sciences, Graduate School of Agricultural and Life Sciences, The University of Tokyo, 1-1-1 Yayoi, Bunkyo-ku, Tokyo 113-8657, Japan.
Yamanouchi K; Department of Veterinary Physiology, Graduate School of Agricultural and Life Sciences, The University of Tokyo, 1-1-1 Yayoi, Bunkyo-ku, Tokyo 113-8657, Japan.
Nishihara M; Department of Veterinary Physiology, Graduate School of Agricultural and Life Sciences, The University of Tokyo, 1-1-1 Yayoi, Bunkyo-ku, Tokyo 113-8657, Japan.
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Źródło:
Scientific reports [Sci Rep] 2014 Jul 09; Vol. 4, pp. 5635. Date of Electronic Publication: 2014 Jul 09.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
CRISPR-Cas Systems/*genetics
Clustered Regularly Interspaced Short Palindromic Repeats/*genetics
Muscular Dystrophy, Duchenne/*genetics
Animals ; Diaphragm/metabolism ; Disease Models, Animal ; Dystrophin/genetics ; Exons/genetics ; Heart/physiology ; Male ; Muscle, Skeletal/metabolism ; Muscular Dystrophy, Duchenne/metabolism ; Mutation/genetics ; Phenotype ; Rats ; Rats, Wistar
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Wyświetlanie 1-20 z 72

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