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Tytuł:
Bariatric Surgery Induces Alterations in the Immune Profile of Peripheral Blood T Cells.
Autorzy:
Barbosa P; University of Coimbra, Institute for Interdisciplinary Research, Doctoral Programme in Experimental Biology and Biomedicine (PDBEB), 3030-789 Coimbra, Portugal.; Center for Neuroscience and Cell Biology (CNC), University of Coimbra, 3004-504 Coimbra, Portugal.; Institute for Interdisciplinary Research (IIIUC), University of Coimbra, 3030-789 Coimbra, Portugal.; Center for Innovative Biomedicine and Biotechnology (CIBB), University of Coimbra, 3000-504 Coimbra, Portugal.
Pinho A; Center for Neuroscience and Cell Biology (CNC), University of Coimbra, 3004-504 Coimbra, Portugal.; Center for Innovative Biomedicine and Biotechnology (CIBB), University of Coimbra, 3000-504 Coimbra, Portugal.; Department of Life Science, University of Coimbra, 3000-456 Coimbra, Portugal.
Lázaro A; General Surgery Unit, Centro Hospitalar e Universitário de Coimbra, 3000-075 Coimbra, Portugal.; Clinical Academic Center of Coimbra (CACC), 3004-061 Coimbra, Portugal.; Coimbra Institute for Clinical and Biomedical Research (iCBR), Group of Environmental Genetics of Oncobiology (CIMAGO), Faculty of Medicine (FMUC), University of Coimbra, 3000-548 Coimbra, Portugal.
Paula D; General Surgery Unit, Centro Hospitalar e Universitário de Coimbra, 3000-075 Coimbra, Portugal.; Clinical Academic Center of Coimbra (CACC), 3004-061 Coimbra, Portugal.
Tralhão JG; General Surgery Unit, Centro Hospitalar e Universitário de Coimbra, 3000-075 Coimbra, Portugal.; Clinical Academic Center of Coimbra (CACC), 3004-061 Coimbra, Portugal.; Coimbra Institute for Clinical and Biomedical Research (iCBR), Group of Environmental Genetics of Oncobiology (CIMAGO), Faculty of Medicine (FMUC), University of Coimbra, 3000-548 Coimbra, Portugal.; Institute of Biophysics, Faculty of Medicine, University of Coimbra, 3000-548 Coimbra, Portugal.
Paiva A; Center for Innovative Biomedicine and Biotechnology (CIBB), University of Coimbra, 3000-504 Coimbra, Portugal.; Clinical Academic Center of Coimbra (CACC), 3004-061 Coimbra, Portugal.; Coimbra Institute for Clinical and Biomedical Research (iCBR), Group of Environmental Genetics of Oncobiology (CIMAGO), Faculty of Medicine (FMUC), University of Coimbra, 3000-548 Coimbra, Portugal.; Flow Cytometry Unit, Department of Clinical Pathology, Centro Hospitalar e Universitário de Coimbra, 3000-076 Coimbra, Portugal.; Instituto Politécnico de Coimbra, ESTESC-Coimbra Health School, Ciências Biomédicas Laboratoriais, 3046-854 Coimbra, Portugal.
Pereira MJ; Department of Medical Sciences, Clinical Diabetology and Metabolism, Uppsala University, SE-75185 Uppsala, Sweden.
Carvalho E; Center for Neuroscience and Cell Biology (CNC), University of Coimbra, 3004-504 Coimbra, Portugal.; Institute for Interdisciplinary Research (IIIUC), University of Coimbra, 3030-789 Coimbra, Portugal.; Center for Innovative Biomedicine and Biotechnology (CIBB), University of Coimbra, 3000-504 Coimbra, Portugal.
Laranjeira P; Center for Neuroscience and Cell Biology (CNC), University of Coimbra, 3004-504 Coimbra, Portugal.; Center for Innovative Biomedicine and Biotechnology (CIBB), University of Coimbra, 3000-504 Coimbra, Portugal.; Clinical Academic Center of Coimbra (CACC), 3004-061 Coimbra, Portugal.; Coimbra Institute for Clinical and Biomedical Research (iCBR), Group of Environmental Genetics of Oncobiology (CIMAGO), Faculty of Medicine (FMUC), University of Coimbra, 3000-548 Coimbra, Portugal.; Flow Cytometry Unit, Department of Clinical Pathology, Centro Hospitalar e Universitário de Coimbra, 3000-076 Coimbra, Portugal.
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Źródło:
Biomolecules [Biomolecules] 2024 Feb 12; Vol. 14 (2). Date of Electronic Publication: 2024 Feb 12.
Typ publikacji:
Journal Article
MeSH Terms:
Th1 Cells*
Bariatric Surgery*
Humans ; T-Lymphocytes, Regulatory ; T-Lymphocyte Subsets ; Obesity/surgery ; Obesity/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Elevated profiles of peripheral Th22, Th17, Th2 cells, and decreased percentage of Th1 cells in breast cancer patients.
Autorzy:
Peng Z; Department of Organ Transplantation, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, China.
Dong X; Department of Oncology, Qilu Hospital of Shandong University Dezhou Hospital, Dezhou, China.
He M; Department of Medical Oncology, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, China.
Zhao Y; Department of Hematology, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, China.
Liu Y; Department of Medical Oncology, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, China.
Li M; Department of Oncology, Weifang People's Hospital, Weifang, China.
Li G; Department of Hematology, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, China.
Wang X; Department of Medical Oncology, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, China.
Li L; Department of Medical Oncology, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, China.
Hu Y; Department of Medical Oncology, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, China.
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Źródło:
Thoracic cancer [Thorac Cancer] 2023 Nov; Vol. 14 (33), pp. 3282-3294. Date of Electronic Publication: 2023 Sep 21.
Typ publikacji:
Journal Article
MeSH Terms:
Th1 Cells*/metabolism
Breast Neoplasms*/metabolism
Humans ; Female ; Th2 Cells ; Ki-67 Antigen/metabolism ; Cytokines/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
An ocular Th1 immune response promotes corneal nerve damage independently of the development of corneal epitheliopathy.
Autorzy:
Vereertbrugghen A; Innate Immunity Laboratory, Institute of Experimental Medicine (CONICET/National Academy of Medicine of Buenos Aires), Buenos Aires, Argentina.
Pizzano M; Innate Immunity Laboratory, Institute of Experimental Medicine (CONICET/National Academy of Medicine of Buenos Aires), Buenos Aires, Argentina.
Sabbione F; Innate Immunity Laboratory, Institute of Experimental Medicine (CONICET/National Academy of Medicine of Buenos Aires), Buenos Aires, Argentina.
Keitelman IA; Innate Immunity Laboratory, Institute of Experimental Medicine (CONICET/National Academy of Medicine of Buenos Aires), Buenos Aires, Argentina.
Shiromizu CM; Innate Immunity Laboratory, Institute of Experimental Medicine (CONICET/National Academy of Medicine of Buenos Aires), Buenos Aires, Argentina.
Aguilar DV; Innate Immunity Laboratory, Institute of Experimental Medicine (CONICET/National Academy of Medicine of Buenos Aires), Buenos Aires, Argentina.
Fuentes F; Confocal Microscopy Unit, Institute of Experimental Medicine (CONICET/National Academy of Medicine of Buenos Aires), Buenos Aires, Argentina.
de Paiva CS; Department of Ophthalmology, Ocular Surface Center, Cullen Eye Institute, Baylor College of Medicine, Houston, TX, USA.
Giordano M; Innate Immunity Laboratory, Institute of Experimental Medicine (CONICET/National Academy of Medicine of Buenos Aires), Buenos Aires, Argentina.
Trevani A; Innate Immunity Laboratory, Institute of Experimental Medicine (CONICET/National Academy of Medicine of Buenos Aires), Buenos Aires, Argentina.
Galletti JG; Innate Immunity Laboratory, Institute of Experimental Medicine (CONICET/National Academy of Medicine of Buenos Aires), Buenos Aires, Argentina. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2023 May 22; Vol. 20 (1), pp. 120. Date of Electronic Publication: 2023 May 22.
Typ publikacji:
Journal Article
MeSH Terms:
Th1 Cells*
Th2 Cells*
Mice ; Animals ; Adjuvants, Immunologic ; Cornea ; Adaptive Immunity ; Inflammation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
PCSK9 promotes T helper 1 and T helper 17 cell differentiation by activating the nuclear factor-κB pathway in ankylosing spondylitis.
Autorzy:
Cai J; Department of Rheumatology and Immunology, Huadong Hospital Affiliated with Fudan University, Shanghai, China.
Jiang Y; Department of Traditional Chinese Medicine and Pharmacy, China Pharmaceutical University, Nanjing, China.
Chen F; Department of Rheumatology and Immunology, Shanghai Qiang-zhi Hospital, Shanghai, China.
Wu S; Department of Rheumatology and Immunology, Shanghai Qiang-zhi Hospital, Shanghai, China.
Ren H; Department of Rheumatology and Immunology, Shanghai Qiang-zhi Hospital, Shanghai, China.
Wang P; Department of Rheumatology and Immunology, Shanghai Qiang-zhi Hospital, Shanghai, China.
Wang J; Department of Rheumatology and Immunology, Shanghai Qiang-zhi Hospital, Shanghai, China.
Liu W; Department of Rheumatology and Immunology, Shanghai Qiang-zhi Hospital, Shanghai, China.
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Źródło:
Immunity, inflammation and disease [Immun Inflamm Dis] 2023 May; Vol. 11 (5), pp. e870.
Typ publikacji:
Journal Article
MeSH Terms:
NF-kappa B*/metabolism
Proprotein Convertase 9*/genetics
Proprotein Convertase 9*/metabolism
Spondylitis, Ankylosing*
Th1 Cells*
Th17 Cells*
Humans ; Cell Differentiation ; Interleukin-17 ; Interleukin-4 ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
P2X7 receptor antagonists modulate experimental autoimmune neuritis via regulation of NLRP3 inflammasome activation and Th17 and Th1 cell differentiation.
Autorzy:
Xie Y; Department of Neurology, Tianjin Nankai Hospital, Tianjin Medical University, Tianjin, 300052, China.; Department of Neurology, Tianjin Neurological Institute, Tianjin Medical University General Hospital, Tianjin, 300052, China.
Han R; Department of Neurology, Tianjin Neurological Institute, Tianjin Medical University General Hospital, Tianjin, 300052, China.
Li Y; Department of Neurology, Tianjin Neurological Institute, Tianjin Medical University General Hospital, Tianjin, 300052, China.
Li W; Department of Neurology, Tianjin Nankai Hospital, Tianjin Medical University, Tianjin, 300052, China.; Department of Neurology, Tianjin Neurological Institute, Tianjin Medical University General Hospital, Tianjin, 300052, China.
Zhang S; Department of Urology, Tianjin Institute of Urology, The Second Hospital of Tianjin Medical University, Tianjin, 300102, China.
Wu Y; Department of Neurology, Tianjin Nankai Hospital, Tianjin Medical University, Tianjin, 300052, China.
Zhao Y; Department of Neurology, Tianjin Neurological Institute, Tianjin Medical University General Hospital, Tianjin, 300052, China.
Liu R; Department of Neurology, Tianjin Neurological Institute, Tianjin Medical University General Hospital, Tianjin, 300052, China.
Wu J; Department of Neurology, Tianjin Neurological Institute, Tianjin Medical University General Hospital, Tianjin, 300052, China.
Jiang W; Department of Neurology, Tianjin Neurological Institute, Tianjin Medical University General Hospital, Tianjin, 300052, China. .; Department of Radiology, The Third Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China. .
Chen X; Department of Neurology, Tianjin Nankai Hospital, Tianjin Medical University, Tianjin, 300052, China. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Mar 25; Vol. 21 (1), pp. 73. Date of Electronic Publication: 2024 Mar 25.
Typ publikacji:
Journal Article
MeSH Terms:
Guillain-Barre Syndrome*/drug therapy
Neuritis, Autoimmune, Experimental*
Purinergic P2X Receptor Antagonists*/pharmacology
Purinergic P2X Receptor Antagonists*/therapeutic use
Animals ; Humans ; Rats ; CD8-Positive T-Lymphocytes ; Cell Differentiation/drug effects ; Inflammasomes/drug effects ; Inflammasomes/metabolism ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Sciatic Nerve/metabolism ; Th17 Cells/drug effects ; Th17 Cells/metabolism ; Th1 Cells/drug effects ; Th1 Cells/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Th1 cells contribute to retinal ganglion cell loss in glaucoma in a VCAM-1-dependent manner.
Autorzy:
He C; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Peng K; Health Management Center, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Zhu X; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.; Department of Prenatal Diagnosis, Chengdu Women's and Children's Central Hospital, School of Medicine, University of Electronic Science and Technology of China, Chengdu, China.
Wang Z; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.; Department of Clinical Laboratory, Sichuan Cancer Hospital and Institute, Sichuan Cancer Center, School of Medicine, University of Electronic Science and Technology of China, Chengdu, China.
Xiu W; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Zhang G; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Chen Y; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Sun C; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Xiao X; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Liu D; Health Management Center, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Li A; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Gao Y; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Wang J; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Shuai P; Health Management Center, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Chen Y; Department of Ophthalmology, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China.
Yu L; Department of Ophthalmology, Daping Hospital, Army Medical Center, Army Medical University, Chongqing, China.
Lu F; Clinical Immunology Translational Medicine Key Laboratory of Sichuan Province, Sichuan Provincial People's Hospital, University of Electronic Science and Technology of China, Chengdu, China. .; Yangtze Delta Region Institute (Quzhou), University of Electronic Science and Technology of China, Quzhou, China. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Feb 05; Vol. 21 (1), pp. 43. Date of Electronic Publication: 2024 Feb 05.
Typ publikacji:
Journal Article
MeSH Terms:
Retinal Ganglion Cells*/pathology
Glaucoma*/metabolism
Humans ; Mice ; Animals ; Vascular Cell Adhesion Molecule-1/metabolism ; Th1 Cells/pathology ; Retina/pathology ; Vision Disorders/pathology ; Disease Models, Animal
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Identification of the prognostic value of Th1/Th2 ratio and a novel prognostic signature in basal-like breast cancer.
Autorzy:
Xiao Y; Affiliated Tumor Hospital, Guangxi Medical University, Nanning, Guangxi, China.; Department of Thyroid and Breast Surgery, Shenzhen Second People's Hospital, Shenzhen, Guangdong, China.
Huang Y; Department of Research, Affiliated Tumor Hospital, Guangxi Medical University, Nanning, Guangxi, China.
Jiang J; Affiliated Tumor Hospital, Guangxi Medical University, Nanning, Guangxi, China.
Chen Y; Affiliated Tumor Hospital, Guangxi Medical University, Nanning, Guangxi, China.
Wei C; Affiliated Tumor Hospital, Guangxi Medical University, Nanning, Guangxi, China. .
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Źródło:
Hereditas [Hereditas] 2023 Jan 25; Vol. 160 (1), pp. 2. Date of Electronic Publication: 2023 Jan 25.
Typ publikacji:
Journal Article
MeSH Terms:
Breast Neoplasms*/genetics
Breast Neoplasms*/metabolism
Th1 Cells*/metabolism
Th2 Cells*/metabolism
Female ; Humans ; Prognosis ; Sterol O-Acyltransferase 2
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Transfer of mesenchymal stem cell mitochondria to CD4 T cells contributes to repress Th1 differentiation by downregulating T-bet expression.
Autorzy:
Akhter W; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France.
Nakhle J; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France.; IGF, CNRS, INSERM, Université de Montpellier, Montpellier, France.; IGMM, CNRS, Université de Montpellier, Montpellier, France.
Vaillant L; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France.
Garcin G; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France.
Le Saout C; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France.
Simon M; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France.
Crozet C; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France.; INM, INSERM, Université de Montpellier, Montpellier, France.
Djouad F; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France.
Jorgensen C; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France.; CHU Montpellier, Montpellier, France.
Vignais ML; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France.; IGF, CNRS, INSERM, Université de Montpellier, Montpellier, France.
Hernandez J; Institute for Regenerative Medicine and Biotherapy (IRMB), INSERM U1183, Université de Montpellier, 34295, Montpellier, France. .
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Źródło:
Stem cell research & therapy [Stem Cell Res Ther] 2023 Jan 24; Vol. 14 (1), pp. 12. Date of Electronic Publication: 2023 Jan 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
CD4-Positive T-Lymphocytes*/metabolism
CD4-Positive T-Lymphocytes*/physiology
Mesenchymal Stem Cells*/metabolism
Mitochondria*/genetics
Mitochondria*/metabolism
Mitochondria*/physiology
Th1 Cells*/metabolism
Animals ; Mice ; Cell Differentiation ; Cells, Cultured ; Cytokines/metabolism ; T-Lymphocytes, Regulatory ; Th17 Cells
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Differential Activation of Splenic cDC1 and cDC2 Cell Subsets following Poxvirus Infection of BALB/c and C57BL/6 Mice.
Autorzy:
Szulc-Dąbrowska, Lidia (AUTHOR)
Biernacka, Zuzanna (AUTHOR)
Koper, Michał (AUTHOR)
Struzik, Justyna (AUTHOR)
Gieryńska, Małgorzata (AUTHOR)
Schollenberger, Ada (AUTHOR)
Lasocka, Iwona (AUTHOR)
Toka, Felix N. (AUTHOR)
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Źródło:
Cells (2073-4409). Jan2024, Vol. 13 Issue 1, p13. 24p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Elimination of Chlamydia muridarum from the female reproductive tract is IL-12p40 dependent, but independent of Th1 and Th2 cells.
Autorzy:
Rixon, Jordan A. (AUTHOR)
Fong, Kevin D. (AUTHOR)
Morris, Claire (AUTHOR)
Nguyen, Alana T. (AUTHOR)
Depew, Claire E. (AUTHOR)
McSorley, Stephen J. (AUTHOR)
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Źródło:
PLoS Pathogens. 1/2/2024, Vol. 20 Issue 1, p1-22. 22p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A Peripheral Blood Signature of Increased Th1 and Myeloid Cells Combined with Serum Inflammatory Mediators Is Associated with Response to Abatacept in Rheumatoid Arthritis Patients.
Autorzy:
Goutakoli, Panagiota (AUTHOR)
Papadaki, Garyfalia (AUTHOR)
Repa, Argyro (AUTHOR)
Avgoustidis, Nestor (AUTHOR)
Kalogiannaki, Eleni (AUTHOR)
Flouri, Irini (AUTHOR)
Bertsias, Antonios (AUTHOR)
Zoidakis, Jerome (AUTHOR)
Samiotaki, Martina (AUTHOR)
Bertsias, George (AUTHOR)
Semitekolou, Maria (AUTHOR)
Verginis, Panayotis (AUTHOR)
Sidiropoulos, Prodromos (AUTHOR)
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Źródło:
Cells (2073-4409). Dec2023, Vol. 12 Issue 24, p2808. 17p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
TNF-α Affects Signature Cytokines of Th1 and Th17 T Cell Subsets through Differential Actions on TNFR1 and TNFR2.
Autorzy:
Pesce B; Immune Regulation and Tolerance Research Group (IRTGroup), Programa Disciplinario de Inmunología, Instituto de Ciencias Biomédicas (ICBM), Facultad de Medicina, Universidad de Chile, Santiago 8380453, Chile.; Laboratorio MED.UCHILE-FACS, Instituto de Ciencias Biomédicas (ICBM), Facultad de Medicina, Universidad de Chile, Santiago 8380453, Chile.
Ribeiro CH; Immune Regulation and Tolerance Research Group (IRTGroup), Programa Disciplinario de Inmunología, Instituto de Ciencias Biomédicas (ICBM), Facultad de Medicina, Universidad de Chile, Santiago 8380453, Chile.
Larrondo M; Hospital Clínico, Universidad de Chile, Santiago 8380453, Chile.
Ramos V; Immune Regulation and Tolerance Research Group (IRTGroup), Programa Disciplinario de Inmunología, Instituto de Ciencias Biomédicas (ICBM), Facultad de Medicina, Universidad de Chile, Santiago 8380453, Chile.; Hospital Clínico, Universidad de Chile, Santiago 8380453, Chile.
Soto L; Immune Regulation and Tolerance Research Group (IRTGroup), Programa Disciplinario de Inmunología, Instituto de Ciencias Biomédicas (ICBM), Facultad de Medicina, Universidad de Chile, Santiago 8380453, Chile.; Hospital Clínico, Universidad de Chile, Santiago 8380453, Chile.
Catalán D; Immune Regulation and Tolerance Research Group (IRTGroup), Programa Disciplinario de Inmunología, Instituto de Ciencias Biomédicas (ICBM), Facultad de Medicina, Universidad de Chile, Santiago 8380453, Chile.
Aguillón JC; Immune Regulation and Tolerance Research Group (IRTGroup), Programa Disciplinario de Inmunología, Instituto de Ciencias Biomédicas (ICBM), Facultad de Medicina, Universidad de Chile, Santiago 8380453, Chile.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Aug 18; Vol. 23 (16). Date of Electronic Publication: 2022 Aug 18.
Typ publikacji:
Journal Article
MeSH Terms:
Arthritis, Rheumatoid*/metabolism
Receptors, Tumor Necrosis Factor, Type I*/metabolism
Receptors, Tumor Necrosis Factor, Type II*/metabolism
Th1 Cells*
Th17 Cells*
Cytokines/metabolism ; Humans ; Interleukin-17 ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
BHLHE40 drives protective polyfunctional CD4 T cell differentiation in the female reproductive tract against Chlamydia.
Autorzy:
Mercado MAB; Department of Microbiology and Immunology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
Li Q; Department of Microbiology and Immunology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
Quick CM; Department of Pathology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
Kim Y; Department of Microbiology and Immunology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
Palmer R; Department of Microbiology and Immunology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
Huang L; Department of Microbiology and Immunology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
Li LX; Department of Microbiology and Immunology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2024 Jan 25; Vol. 20 (1), pp. e1011983. Date of Electronic Publication: 2024 Jan 25 (Print Publication: 2024).
Typ publikacji:
Journal Article
MeSH Terms:
CD4-Positive T-Lymphocytes*/cytology
CD4-Positive T-Lymphocytes*/immunology
Chlamydia Infections*/immunology
Chlamydia muridarum*/physiology
Basic Helix-Loop-Helix Transcription Factors*/metabolism
Homeodomain Proteins*/metabolism
Animals ; Female ; Mice ; Cell Differentiation ; Interleukin-10/metabolism ; Mice, Inbred C57BL ; Th1 Cells/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Increased regulatory activity of intestinal innate lymphoid cells type 3 (ILC3) prevents experimental autoimmune encephalomyelitis severity.
Autorzy:
Lazarević M; Department of Immunology, Institute for Biological Research 'Siniša Stanković' - National Institute of Republic of Serbia, University of Belgrade, Despota Stefana 142, 11000, Belgrade, Serbia.
Stegnjaić G; Department of Immunology, Institute for Biological Research 'Siniša Stanković' - National Institute of Republic of Serbia, University of Belgrade, Despota Stefana 142, 11000, Belgrade, Serbia.
Jevtić B; Department of Immunology, Institute for Biological Research 'Siniša Stanković' - National Institute of Republic of Serbia, University of Belgrade, Despota Stefana 142, 11000, Belgrade, Serbia.
Despotović S; Institute of Histology and Embryology, School of Medicine, University of Belgrade, Dr Subotića 9, 11000, Belgrade, Serbia.
Ignjatović Đ; Department of Biochemistry, Institute for Biological Research 'Siniša Stanković' - National Institute of Republic of Serbia, University of Belgrade, Despota Stefana 142, 11000, Belgrade, Serbia.
Stanisavljević S; Department of Immunology, Institute for Biological Research 'Siniša Stanković' - National Institute of Republic of Serbia, University of Belgrade, Despota Stefana 142, 11000, Belgrade, Serbia.
Nikolovski N; Department of Immunology, Institute for Biological Research 'Siniša Stanković' - National Institute of Republic of Serbia, University of Belgrade, Despota Stefana 142, 11000, Belgrade, Serbia.
Momčilović M; Department of Immunology, Institute for Biological Research 'Siniša Stanković' - National Institute of Republic of Serbia, University of Belgrade, Despota Stefana 142, 11000, Belgrade, Serbia.
Fraser GL; Epics Therapeutics S.A, 47 Rue Adrienne Bolland, 6041, Gosselies, Belgium.
Dimitrijević M; Department of Immunology, Institute for Biological Research 'Siniša Stanković' - National Institute of Republic of Serbia, University of Belgrade, Despota Stefana 142, 11000, Belgrade, Serbia.
Miljković Đ; Department of Immunology, Institute for Biological Research 'Siniša Stanković' - National Institute of Republic of Serbia, University of Belgrade, Despota Stefana 142, 11000, Belgrade, Serbia. georgije_.
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Jan 18; Vol. 21 (1), pp. 26. Date of Electronic Publication: 2024 Jan 18.
Typ publikacji:
Journal Article
MeSH Terms:
Encephalomyelitis, Autoimmune, Experimental*/chemically induced
Encephalomyelitis, Autoimmune, Experimental*/prevention & control
Rats ; Animals ; Mice ; Immunity, Innate ; Spinal Cord/pathology ; Microglia ; Th17 Cells ; Th1 Cells ; Mice, Inbred C57BL
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
sFgl2 gene-modified MSCs regulate the differentiation of CD4 T cells in the treatment of autoimmune hepatitis.
Autorzy:
Ji W; Department of General Surgery, Tianjin Medical University General Hospital, No. 154, Anshan Road, Heping District, Tianjin, 300052, China.
Wang W; Department of General Surgery, Tianjin Medical University Baodi Clinical College, Guangchuan Road, Baodi, Tianjin, 301800, China.
Li P; Department of General Surgery, Tianjin Medical University General Hospital, No. 154, Anshan Road, Heping District, Tianjin, 300052, China.
Liu Y; Department of General Surgery, Tianjin Union Medical Center, Tianjin, China.; Tianjin Key Laboratory of General Surgery in Construction, Tianjin Union Medical Center, Tianjin, 300121, China.
Zhang B; Department of General Surgery, Tianjin Medical University General Hospital, No. 154, Anshan Road, Heping District, Tianjin, 300052, China.
Qi F; Department of General Surgery, Tianjin Medical University General Hospital, No. 154, Anshan Road, Heping District, Tianjin, 300052, China. .
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Źródło:
Stem cell research & therapy [Stem Cell Res Ther] 2023 Nov 03; Vol. 14 (1), pp. 316. Date of Electronic Publication: 2023 Nov 03.
Typ publikacji:
Journal Article
MeSH Terms:
Fibrinogen*/metabolism
Hepatitis, Autoimmune*/genetics
Hepatitis, Autoimmune*/therapy
Mesenchymal Stem Cells*/metabolism
T-Lymphocytes, Regulatory*
CD8-Positive T-Lymphocytes/metabolism ; Cell Differentiation ; Th1 Cells ; Th17 Cells ; Animals ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
IL-2-mTORC1 signaling coordinates the STAT1/T-bet axis to ensure Th1 cell differentiation and anti-bacterial immune response in fish.
Autorzy:
Ai K; State Key Laboratory of Estuarine and Coastal Research, School of Life Sciences, East China Normal University, Shanghai, China.
Li K; State Key Laboratory of Estuarine and Coastal Research, School of Life Sciences, East China Normal University, Shanghai, China.
Jiao X; State Key Laboratory of Estuarine and Coastal Research, School of Life Sciences, East China Normal University, Shanghai, China.
Zhang Y; State Key Laboratory of Estuarine and Coastal Research, School of Life Sciences, East China Normal University, Shanghai, China.
Li J; State Key Laboratory of Estuarine and Coastal Research, School of Life Sciences, East China Normal University, Shanghai, China.
Zhang Q; State Key Laboratory of Estuarine and Coastal Research, School of Life Sciences, East China Normal University, Shanghai, China.
Wei X; State Key Laboratory of Estuarine and Coastal Research, School of Life Sciences, East China Normal University, Shanghai, China.
Yang J; State Key Laboratory of Estuarine and Coastal Research, School of Life Sciences, East China Normal University, Shanghai, China.; Laboratory for Marine Biology and Biotechnology, Qingdao National Laboratory for Marine Science and Technology, Qingdao, China.
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Źródło:
PLoS pathogens [PLoS Pathog] 2022 Oct 25; Vol. 18 (10), pp. e1010913. Date of Electronic Publication: 2022 Oct 25 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Th1 Cells*
Antimutagenic Agents*/metabolism
Animals ; STAT5 Transcription Factor/metabolism ; Interleukin-2/metabolism ; Mechanistic Target of Rapamycin Complex 1/metabolism ; Interleukin-12/metabolism ; Trans-Activators/metabolism ; T-Box Domain Proteins/genetics ; T-Box Domain Proteins/metabolism ; Cell Differentiation ; Lymphocyte Activation ; Androgen Antagonists/metabolism ; CD4-Positive T-Lymphocytes
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Adenosine receptor A2a blockade by caffeine increases IFN-gamma production in Th1 cells from patients with rheumatoid arthritis.
Autorzy:
Gloyer L; Laboratory of Molecular Immunology, Department I of Internal Medicine, University of Cologne, Cologne, Germany.
Golumba-Nagy V; Laboratory of Molecular Immunology, Department I of Internal Medicine, University of Cologne, Cologne, Germany.
Meyer A; Laboratory of Molecular Immunology, Department I of Internal Medicine, University of Cologne, Cologne, Germany.
Yan S; Laboratory of Molecular Immunology, Department I of Internal Medicine, University of Cologne, Cologne, Germany.
Schiller J; Division of Clinical Immunology and Rheumatology, Department I of Internal Medicine, University of Cologne, and Center for Integrated Oncology Aachen Bonn Cologne Duesseldorf, Cologne, Germany.
Breuninger M; Division of Clinical Immunology and Rheumatology, Department I of Internal Medicine, University of Cologne, and Center for Integrated Oncology Aachen Bonn Cologne Duesseldorf, Cologne, Germany.
Jochimsen D; Division of Clinical Immunology and Rheumatology, Department I of Internal Medicine, University of Cologne, and Center for Integrated Oncology Aachen Bonn Cologne Duesseldorf, Cologne, Germany.
Kofler DM; Laboratory of Molecular Immunology, Department I of Internal Medicine, University of Cologne, Cologne, Germany.; Division of Clinical Immunology and Rheumatology, Department I of Internal Medicine, University of Cologne, and Center for Integrated Oncology Aachen Bonn Cologne Duesseldorf, Cologne, Germany.
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Źródło:
Scandinavian journal of rheumatology [Scand J Rheumatol] 2022 Jul; Vol. 51 (4), pp. 279-283. Date of Electronic Publication: 2022 Jan 13.
Typ publikacji:
Journal Article
MeSH Terms:
Adenosine A2 Receptor Antagonists*/pharmacology
Arthritis, Rheumatoid*/immunology
Caffeine*/pharmacology
Interferon-gamma*/metabolism
Th1 Cells*/metabolism
Humans ; Leukocytes, Mononuclear/metabolism ; Th2 Cells/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
iNOS is essential to maintain a protective Th1/Th2 response and the production of cytokines/chemokines against Schistosoma japonicum infection in rats.
Autorzy:
Shen J; Department of Parasitology and Key Laboratory of Tropical Disease Control (SYSU), Ministry of Education, Zhongshan School of Medicine, Sun Yat-Sen University, Guangzhou, P.R. China.; Provincial Engineering Technology Research Center for Biological Vector Control, Guangzhou, Guangdong, P.R. China.
Yu SF; Clinical Research Institute, The First People's Hospital of Foshan, Foshan, P.R. China.
Peng M; Department of Parasitology and Key Laboratory of Tropical Disease Control (SYSU), Ministry of Education, Zhongshan School of Medicine, Sun Yat-Sen University, Guangzhou, P.R. China.; Provincial Engineering Technology Research Center for Biological Vector Control, Guangzhou, Guangdong, P.R. China.
Lai DH; State Key Laboratory of Biocontrol, School of Life Sciences, Sun Yat-Sen University, Guangzhou, P.R. China.
Hide G; Ecosystems and Environment Research Centre and Biomedical Research Centre, School of Science, Engineering and Environment, University of Salford, Salford, United Kingdom.
Wu ZD; Department of Parasitology and Key Laboratory of Tropical Disease Control (SYSU), Ministry of Education, Zhongshan School of Medicine, Sun Yat-Sen University, Guangzhou, P.R. China.; Provincial Engineering Technology Research Center for Biological Vector Control, Guangzhou, Guangdong, P.R. China.
Lun ZR; State Key Laboratory of Biocontrol, School of Life Sciences, Sun Yat-Sen University, Guangzhou, P.R. China.
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Źródło:
PLoS neglected tropical diseases [PLoS Negl Trop Dis] 2022 May 18; Vol. 16 (5), pp. e0010403. Date of Electronic Publication: 2022 May 18 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Nitric Oxide Synthase Type II*/genetics
Nitric Oxide Synthase Type II*/metabolism
Nitric Oxide Synthase Type II*/physiology
Schistosoma japonicum*
Schistosomiasis japonica*/enzymology
Schistosomiasis japonica*/immunology
Th1 Cells*/immunology
Th2 Cells*/immunology
Animals ; Chemokines/metabolism ; Cytokines/metabolism ; Nitric Oxide/metabolism ; Rats
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
GPR65 promotes intestinal mucosal Th1 and Th17 cell differentiation and gut inflammation through downregulating NUAK2.
Autorzy:
Lin R; Center for Inflammatory Bowel Disease Research, The Shanghai Tenth People's Hospital, Tongji University of School Medicine, Shanghai, China.
Wu W; Center for Inflammatory Bowel Disease Research, The Shanghai Tenth People's Hospital, Tongji University of School Medicine, Shanghai, China.
Chen H; Center for Inflammatory Bowel Disease Research, The Shanghai Tenth People's Hospital, Tongji University of School Medicine, Shanghai, China.
Gao H; Center for Inflammatory Bowel Disease Research, The Shanghai Tenth People's Hospital, Tongji University of School Medicine, Shanghai, China.
Wu X; Center for Inflammatory Bowel Disease Research, The Shanghai Tenth People's Hospital, Tongji University of School Medicine, Shanghai, China.
Li G; Center for Inflammatory Bowel Disease Research, The Shanghai Tenth People's Hospital, Tongji University of School Medicine, Shanghai, China.
He Q; Center for Inflammatory Bowel Disease Research, The Shanghai Tenth People's Hospital, Tongji University of School Medicine, Shanghai, China.
Lu H; Center for Inflammatory Bowel Disease Research, The Shanghai Tenth People's Hospital, Tongji University of School Medicine, Shanghai, China.
Sun M; Center for Inflammatory Bowel Disease Research, The Shanghai Tenth People's Hospital, Tongji University of School Medicine, Shanghai, China.
Liu Z; Center for Inflammatory Bowel Disease Research, The Shanghai Tenth People's Hospital, Tongji University of School Medicine, Shanghai, China.
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Źródło:
Clinical and translational medicine [Clin Transl Med] 2022 Mar; Vol. 12 (3), pp. e771.
Typ publikacji:
Journal Article
MeSH Terms:
Th1 Cells*/pathology
Th17 Cells*/pathology
Animals ; Cell Differentiation/genetics ; Humans ; Inflammation/metabolism ; Inflammation/pathology ; Intestinal Mucosa/metabolism ; Intestinal Mucosa/pathology ; Mice ; Protein Serine-Threonine Kinases ; Receptors, G-Protein-Coupled
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
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