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Wyszukujesz frazę ""Toll-Like Receptor 9"" wg kryterium: Temat


Tytuł :
TLR9 mediates the activation of NLRP3 inflammasome and oxidative stress in murine allergic airway inflammation.
Autorzy :
Zhao CC; Anhui Geriatric Institute, Department of Geriatric Respiratory and Critical Care, The First Affiliated Hospital of Anhui Medical University, Jixi Road 218, Hefei, Anhui 230022, PR China.
Xie QM; Anhui Geriatric Institute, Department of Geriatric Respiratory and Critical Care, The First Affiliated Hospital of Anhui Medical University, Jixi Road 218, Hefei, Anhui 230022, PR China.
Xu J; Anhui Geriatric Institute, Department of Geriatric Respiratory and Critical Care, The First Affiliated Hospital of Anhui Medical University, Jixi Road 218, Hefei, Anhui 230022, PR China.
Yan XB; Anhui Geriatric Institute, Department of Geriatric Respiratory and Critical Care, The First Affiliated Hospital of Anhui Medical University, Jixi Road 218, Hefei, Anhui 230022, PR China.
Fan XY; Anhui Geriatric Institute, Department of Geriatric Respiratory and Critical Care, The First Affiliated Hospital of Anhui Medical University, Jixi Road 218, Hefei, Anhui 230022, PR China.
Wu HM; Anhui Geriatric Institute, Department of Geriatric Respiratory and Critical Care, The First Affiliated Hospital of Anhui Medical University, Jixi Road 218, Hefei, Anhui 230022, PR China. Electronic address: .
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Źródło :
Molecular immunology [Mol Immunol] 2020 Sep; Vol. 125, pp. 24-31. Date of Electronic Publication: 2020 Jul 02.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Asthma/*immunology
Hypersensitivity/*immunology
Inflammasomes/*immunology
NLR Family, Pyrin Domain-Containing 3 Protein/*immunology
Oxidative Stress/*physiology
Toll-Like Receptor 9/*immunology
Animals ; Asthma/metabolism ; Hypersensitivity/metabolism ; Hypersensitivity/physiopathology ; Inflammasomes/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Pneumonia/immunology ; Pneumonia/metabolism ; Pneumonia/physiopathology ; RAW 264.7 Cells ; Toll-Like Receptor 9/metabolism
Czasopismo naukowe
Tytuł :
Toll-like receptor 9 and the inflammatory response to surgical trauma and cardiopulmonary bypass.
Autorzy :
Naase H; Department of Surgery and Cancer, Imperial College London, St Mary's Hospital, London, W2 1NY, UK. .
Harling L; Department of Surgery and Cancer, Imperial College London, St Mary's Hospital, London, W2 1NY, UK.
Kidher E; Department of Surgery and Cancer, Imperial College London, St Mary's Hospital, London, W2 1NY, UK.
Sepehripour A; Imperial College Healthcare NHS Trust, Hammersmith Hospital, London, UK.
Nguyen B; National Heart and Lung Institute, Imperial College London, London, UK.
Kapelouzou A; Biomedical Research Foundation, Academy of Athens, Athens, Greece.
Cokkinos D; Biomedical Research Foundation, Academy of Athens, Athens, Greece.
Stavridis G; Biomedical Research Foundation, Academy of Athens, Athens, Greece.
Angelini G; National Heart and Lung Institute, Imperial College London, London, UK.
Evans PC; Department of Cardiovascular Science, University of Sheffield, Sheffield, UK.
Athanasiou T; Department of Surgery and Cancer, Imperial College London, St Mary's Hospital, London, W2 1NY, UK.
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Źródło :
Journal of cardiothoracic surgery [J Cardiothorac Surg] 2020 Jun 11; Vol. 15 (1), pp. 137. Date of Electronic Publication: 2020 Jun 11.
Typ publikacji :
Journal Article
MeSH Terms :
Cardiopulmonary Bypass/*adverse effects
DNA, Mitochondrial/*blood
Interleukin-6/*blood
Sternotomy/*adverse effects
Toll-Like Receptor 9/*blood
Animals ; Cardiac Surgical Procedures ; Female ; Inflammation/blood ; Male ; Mice ; Mitochondria ; Postoperative Complications ; Rats ; Signal Transduction ; Swine ; Toll-Like Receptor 9/antagonists & inhibitors
Czasopismo naukowe
Tytuł :
Antibody Opsonization of a TLR9 Agonist-Containing Virus-like Particle Enhances In Situ Immunization.
Autorzy :
Lemke-Miltner CD; Holden Comprehensive Cancer Center, University of Iowa, Iowa City, IA 52242.
Blackwell SE; Holden Comprehensive Cancer Center, University of Iowa, Iowa City, IA 52242.
Yin C; Holden Comprehensive Cancer Center, University of Iowa, Iowa City, IA 52242.
Krug AE; Holden Comprehensive Cancer Center, University of Iowa, Iowa City, IA 52242.
Morris AJ; Checkmate Pharmaceuticals, Cambridge, MA 02142; and.
Krieg AM; Checkmate Pharmaceuticals, Cambridge, MA 02142; and.
Weiner GJ; Holden Comprehensive Cancer Center, University of Iowa, Iowa City, IA 52242; .; Department of Internal Medicine, University of Iowa, Iowa City, IA 52242.
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Źródło :
Journal of immunology (Baltimore, Md. : 1950) [J Immunol] 2020 Mar 01; Vol. 204 (5), pp. 1386-1394. Date of Electronic Publication: 2020 Jan 17.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Immunization*
Lymphoma*/immunology
Lymphoma*/pathology
Lymphoma*/therapy
Toll-Like Receptor 9*/agonists
Toll-Like Receptor 9*/immunology
Immunity, Cellular/*drug effects
Oligodeoxyribonucleotides/*pharmacology
Vaccines, Virus-Like Particle/*pharmacology
Animals ; Antibodies, Neoplasm/immunology ; Cell Line, Tumor ; Female ; Humans ; Mice ; Mice, Knockout
Czasopismo naukowe
Tytuł :
Dysregulation of TLR9 in neonates leads to fatal inflammatory disease driven by IFN-γ.
Autorzy :
Stanbery AG; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, CA 94720.
Newman ZR; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, CA 94720.
Barton GM; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, CA 94720 .
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Źródło :
Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2020 Feb 11; Vol. 117 (6), pp. 3074-3082. Date of Electronic Publication: 2020 Jan 24.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms :
Autoimmune Diseases/*metabolism
Inflammation/*metabolism
Interferon-gamma/*metabolism
Toll-Like Receptor 9/*metabolism
Animals ; Animals, Newborn ; Autoimmune Diseases/immunology ; Cells, Cultured ; Inflammation/immunology ; Interferon-gamma/immunology ; Macrophages/immunology ; Macrophages/metabolism ; Mice ; Mice, Transgenic ; Monocytes/immunology ; Monocytes/metabolism ; Signal Transduction/immunology ; Toll-Like Receptor 9/genetics ; Toll-Like Receptor 9/immunology
Czasopismo naukowe
Tytuł :
TLR9 and beclin 1 crosstalk regulates muscle AMPK activation in exercise.
Autorzy :
Liu Y; Center for Autophagy Research, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA. .; Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA. .
Nguyen PT; Center for Autophagy Research, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Wang X; Children's Medical Center Research Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Pediatrics, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Zhao Y; Center for Autophagy Research, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Meacham CE; Children's Medical Center Research Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Pediatrics, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Zou Z; Center for Autophagy Research, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Bordieanu B; Children's Medical Center Research Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Pediatrics, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Johanns M; De Duve Institute, Université Catholique de Louvain, Brussels, Belgium.
Vertommen D; De Duve Institute, Université Catholique de Louvain, Brussels, Belgium.
Wijshake T; Center for Autophagy Research, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.
May H; Division of Cardiology, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Xiao G; Department of Clinical Sciences, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Shoji-Kawata S; Center for Autophagy Research, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Rider MH; De Duve Institute, Université Catholique de Louvain, Brussels, Belgium.
Morrison SJ; Children's Medical Center Research Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Pediatrics, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Mishra P; Children's Medical Center Research Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA.; Department of Pediatrics, University of Texas Southwestern Medical Center, Dallas, TX, USA.
Levine B; Center for Autophagy Research, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA. .; Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA. .; Department of Microbiology, University of Texas Southwestern Medical Center, Dallas, TX, USA. .
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Źródło :
Nature [Nature] 2020 Feb; Vol. 578 (7796), pp. 605-609. Date of Electronic Publication: 2020 Feb 12.
Typ publikacji :
Journal Article
MeSH Terms :
AMP-Activated Protein Kinases/*metabolism
Beclin-1/*metabolism
Muscle, Skeletal/*metabolism
Physical Conditioning, Animal/*physiology
Toll-Like Receptor 9/*metabolism
Animals ; Autophagy ; Enzyme Activation ; Exercise ; Glucose/metabolism ; Humans ; Male ; Mice ; Models, Animal ; Muscle, Skeletal/enzymology ; Phosphatidylinositol 3-Kinase/metabolism ; Toll-Like Receptor 9/deficiency ; Toll-Like Receptor 9/genetics ; Tumor Suppressor Proteins/metabolism
Czasopismo naukowe
Tytuł :
Effect of high mobility group box 1 on Toll-like receptor 9 in B cells in myeloperoxidase-ANCA-associated vasculitis.
Autorzy :
Wang C; Department of Medicine, Renal Division, Peking University First Hospital, Peking University Institute of Nephrology, Beijing, China.; Key Laboratory of Renal Disease, Ministry of Health of China, Beijing, China.; Key Laboratory of Chronic Kidney Disease Prevention and Treatment (Peking University), Ministry of Education, Beijing, China.
Deng H; Department of Medicine, Renal Division, Peking University First Hospital, Peking University Institute of Nephrology, Beijing, China.; Key Laboratory of Renal Disease, Ministry of Health of China, Beijing, China.; Key Laboratory of Chronic Kidney Disease Prevention and Treatment (Peking University), Ministry of Education, Beijing, China.
Gong Y; Department of Clinical Laboratory, Peking University First Hospital, Beijing, China.
You R; Department of Clinical Laboratory, Peking University First Hospital, Beijing, China.
Chen M; Department of Medicine, Renal Division, Peking University First Hospital, Peking University Institute of Nephrology, Beijing, China.; Key Laboratory of Renal Disease, Ministry of Health of China, Beijing, China.; Key Laboratory of Chronic Kidney Disease Prevention and Treatment (Peking University), Ministry of Education, Beijing, China.
Zhao MH; Department of Medicine, Renal Division, Peking University First Hospital, Peking University Institute of Nephrology, Beijing, China.; Key Laboratory of Renal Disease, Ministry of Health of China, Beijing, China.; Key Laboratory of Chronic Kidney Disease Prevention and Treatment (Peking University), Ministry of Education, Beijing, China.; Peking-Tsinghua Center for Life Sciences, Beijing, China.
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Źródło :
Autoimmunity [Autoimmunity] 2020 Feb; Vol. 53 (1), pp. 28-34. Date of Electronic Publication: 2019 Dec 02.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/*etiology
Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/*metabolism
B-Lymphocytes/*immunology
B-Lymphocytes/*metabolism
HMGB1 Protein/*genetics
Peroxidase/*metabolism
Toll-Like Receptor 9/*metabolism
Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/diagnosis ; Autoimmune Diseases ; Autoimmunity ; B-Lymphocyte Subsets/immunology ; B-Lymphocyte Subsets/metabolism ; Biomarkers ; HMGB1 Protein/metabolism ; Humans ; Immunophenotyping ; Leukocytes, Mononuclear/immunology ; Leukocytes, Mononuclear/metabolism ; Lymphocyte Activation/immunology ; Toll-Like Receptor 9/genetics
Czasopismo naukowe
Tytuł :
First-in-human phase I clinical trial of the NY-ESO-1 protein cancer vaccine with NOD2 and TLR9 stimulants in patients with NY-ESO-1-expressing refractory solid tumors.
Autorzy :
Ishihara M; Department of Medical Oncology, Mie University Hospital, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan. .
Tono Y; Department of Medical Oncology, Mie University Hospital, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.
Miyahara Y; Department of Personalized Cancer Immunotherapy, Mie University Graduate School of Medicine, 1577 Kurimamachiya-cho, Tsu, Mie, 514-8507, Japan.
Muraoka D; Department of Oncology, Nagasaki University Graduate School of Biomedical Sciences, 1-12-4 Sakamoto, Nagasaki, Nagasaki, 852-8523, Japan.
Harada N; United Immunity, Co., Ltd., Room220, Mie University Campus Incubator, 1577 Kurimamachiya-cho, Tsu, Mie, 514-8507, Japan.
Kageyama S; Department of Immuno-Gene Therapy, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.
Sasaki T; Department of Nephro-Urologic Surgery and Andrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.
Hori Y; Kameyama Nephro-Urologic Clinic, 1488-215 Sakaemachi, Kameyama, Mie, 519-0111, Japan.
Soga N; Department of Urology, Aichi Cancer Center Hospital, 1-1 Kanokoden, Chikusa-ku, Nagoya, Aichi, 464-8681, Japan.
Uchida K; Department of Pathology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.
Shiraishi T; Department of Pathology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.
Sato E; Department of Pathology, Institute of Medical Science (Medical Research Center), Tokyo Medical University, 6-7-1 Nishishinjuku, Shinjuku-ku, Tokyo, 160-0023, Japan.
Kanda H; Department of Nephro-Urologic Surgery and Andrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.
Mizuno T; Department of Medical Oncology, Mie University Hospital, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.
Webster GA; Innate Immunotherapeutics, Melbourne, VIC, 3051, Australia.
Ikeda H; Department of Oncology, Nagasaki University Graduate School of Biomedical Sciences, 1-12-4 Sakamoto, Nagasaki, Nagasaki, 852-8523, Japan.
Katayama N; Department of Hematology and Oncology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.
Sugimura Y; Department of Nephro-Urologic Surgery and Andrology, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan.
Shiku H; Department of Personalized Cancer Immunotherapy, Mie University Graduate School of Medicine, 1577 Kurimamachiya-cho, Tsu, Mie, 514-8507, Japan. .; Department of Immuno-Gene Therapy, Mie University Graduate School of Medicine, 2-174 Edobashi, Tsu, Mie, 514-8507, Japan. .
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Źródło :
Cancer immunology, immunotherapy : CII [Cancer Immunol Immunother] 2020 Apr; Vol. 69 (4), pp. 663-675. Date of Electronic Publication: 2020 Jan 24.
Typ publikacji :
Clinical Trial, Phase I; Journal Article
MeSH Terms :
Antigens, Neoplasm/*immunology
Cancer Vaccines/*immunology
Membrane Proteins/*immunology
Neoplasms/*immunology
Nod2 Signaling Adaptor Protein/*immunology
Toll-Like Receptor 9/*immunology
Adult ; Aged ; Aged, 80 and over ; Animals ; Antibodies, Neoplasm/blood ; Antibodies, Neoplasm/immunology ; CD8-Positive T-Lymphocytes/drug effects ; CD8-Positive T-Lymphocytes/immunology ; CD8-Positive T-Lymphocytes/metabolism ; Cancer Vaccines/administration & dosage ; Cell Line, Tumor ; Female ; Humans ; Interferon-gamma/immunology ; Interferon-gamma/metabolism ; Male ; Mice, Inbred BALB C ; Middle Aged ; Neoplasms/pathology ; Neoplasms/therapy ; Nod2 Signaling Adaptor Protein/metabolism ; Toll-Like Receptor 9/metabolism ; Vaccination/methods
Czasopismo naukowe
Tytuł :
Anti-inflammatory effects of nobiletin on TLR4/TRIF/IRF3 and TLR9/IRF7 signaling pathways in prostate cancer cells.
Autorzy :
Deveci Ozkan A; Department of Medical Biology, Faculty of Medicine, Sakarya University, Sakarya, Turkey.
Kaleli S; Department of Medical Biology, Faculty of Medicine, Sakarya University, Sakarya, Turkey.
Onen HI; Department of Medical Biology, Faculty of Medicine, Gazi University, Ankara, Turkey.
Sarihan M; Department of Medical Biology, Faculty of Medicine, Kocaeli University, Kocaeli, Turkey.
Guney Eskiler G; Department of Medical Biology, Faculty of Medicine, Sakarya University, Sakarya, Turkey.
Kalayci Yigin A; Department of Medical Genetic, Cerrahpaşa Medical Faculty, İstanbul University, Istanbul, Turkey.
Akdogan M; Department of Medical Biochemistry, Faculty of Medicine, Sakarya University, Sakarya, Turkey.
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Źródło :
Immunopharmacology and immunotoxicology [Immunopharmacol Immunotoxicol] 2020 Apr; Vol. 42 (2), pp. 93-100. Date of Electronic Publication: 2020 Feb 12.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Flavones/*pharmacology
Prostatic Neoplasms/*metabolism
Toll-Like Receptor 4/*metabolism
Toll-Like Receptor 9/*metabolism
Adaptor Proteins, Vesicular Transport/genetics ; Adaptor Proteins, Vesicular Transport/metabolism ; Cell Culture Techniques ; Cell Proliferation/drug effects ; Gene Expression/drug effects ; Humans ; Interferon Regulatory Factor-3/genetics ; Interferon Regulatory Factor-3/metabolism ; Male ; PC-3 Cells ; Prostatic Neoplasms/immunology ; Prostatic Neoplasms/pathology ; Signal Transduction ; Toll-Like Receptor 4/genetics ; Toll-Like Receptor 9/genetics
Czasopismo naukowe
Tytuł :
Unmethylated CpG motif-containing genomic DNA fragment of Bacillus calmette-guerin promotes macrophage functions through TLR9-mediated activation of NF- κ B and MAPKs signaling pathways.
Autorzy :
Li J; Division of Tuberculosis Vaccines, National Institutes for Food and Drug Control (NIFDC), Beijing, P.R. China.; NHC Key Laboratory of Human Disease Comparative Medicine, Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences (CAMS) and Peking Union Medical College (PUMC), Beijing, P.R. China.; Beijing Key Laboratory for Animal Models of Emerging and Reemerging Infectious, Beijing, P.R. China.; Key Laboratory of Human Diseases Animal Model, State Administration of Traditional Chinese Medicine, Beijing, P.R. China.; Tuberculosis Center, Chinese Academy of Medical Sciences (CAMS), Beijing, P.R. China.
Fu L; Division of Tuberculosis Vaccines, National Institutes for Food and Drug Control (NIFDC), Beijing, P.R. China.
Wang G; Division of Tuberculosis Vaccines, National Institutes for Food and Drug Control (NIFDC), Beijing, P.R. China.
Subbian S; Public Health Research Institute (PHRI) center at New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, USA.
Qin C; NHC Key Laboratory of Human Disease Comparative Medicine, Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences (CAMS) and Peking Union Medical College (PUMC), Beijing, P.R. China.; Beijing Key Laboratory for Animal Models of Emerging and Reemerging Infectious, Beijing, P.R. China.; Key Laboratory of Human Diseases Animal Model, State Administration of Traditional Chinese Medicine, Beijing, P.R. China.; Tuberculosis Center, Chinese Academy of Medical Sciences (CAMS), Beijing, P.R. China.
Zhao A; Division of Tuberculosis Vaccines, National Institutes for Food and Drug Control (NIFDC), Beijing, P.R. China.
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Źródło :
Innate immunity [Innate Immun] 2020 Apr; Vol. 26 (3), pp. 183-203. Date of Electronic Publication: 2019 Oct 15.
Typ publikacji :
Journal Article
MeSH Terms :
Adjuvants, Immunologic/*pharmacology
BCG Vaccine/*pharmacology
CpG Islands/*genetics
DNA, Bacterial/*genetics
MAP Kinase Signaling System/*drug effects
Macrophages/*drug effects
Mycobacterium bovis/*genetics
NF-kappa B/*drug effects
Signal Transduction/*genetics
Toll-Like Receptor 9/*drug effects
Animals ; Cytokines/biosynthesis ; Macrophages, Peritoneal/drug effects ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Toll-Like Receptor 9/genetics
Czasopismo naukowe
Tytuł :
Mitochondrial DNA Stimulates TLR9-Dependent Neutrophil Extracellular Trap Formation in Primary Graft Dysfunction.
Autorzy :
Mallavia B; Department of Medicine.
Liu F; Department of Medicine.
Lefrançais E; Department of Medicine.
Cleary SJ; Department of Medicine.
Kwaan N; Department of Medicine.
Tian JJ; Department of Medicine.
Magnen M; Department of Medicine.
Sayah DM; Department of Medicine and.
Soong A; Department of Medicine.
Chen J; Department of Surgery, and.
Saggar R; Department of Medicine and.
Shino MY; Department of Medicine and.
Ross DJ; Department of Medicine and.
Derhovanessian A; Department of Medicine and.
Lynch JP 3rd; Department of Medicine and.
Ardehali A; Department of Surgery, University of California, Los Angeles, Los Angeles, California.
Weigt SS; Department of Medicine and.
Belperio JA; Department of Medicine and.
Hays SR; Department of Medicine.
Golden JA; Department of Medicine.
Leard LE; Department of Medicine.
Shah RJ; Department of Medicine.
Kleinhenz ME; Department of Medicine.
Venado A; Department of Medicine.
Kukreja J; Department of Surgery, and.
Singer JP; Department of Medicine.
Looney MR; Department of Medicine.; Department of Laboratory Medicine, University of California, San Francisco, San Francisco, California; and.
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Źródło :
American journal of respiratory cell and molecular biology [Am J Respir Cell Mol Biol] 2020 Mar; Vol. 62 (3), pp. 364-372.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Cold Ischemia/*adverse effects
DNA, Mitochondrial/*pharmacology
Extracellular Traps/*metabolism
Neutrophils/*drug effects
Primary Graft Dysfunction/*immunology
Toll-Like Receptor 9/*physiology
Acute Lung Injury/etiology ; Animals ; Bronchoalveolar Lavage Fluid/cytology ; Citrullination ; DNA, Mitochondrial/administration & dosage ; Deoxyribonuclease I/metabolism ; Humans ; Lung Transplantation ; Male ; Mice ; Mice, Inbred C57BL ; Neutrophils/immunology ; Primary Graft Dysfunction/metabolism ; Protein-Arginine Deiminase Type 4/deficiency ; Protein-Arginine Deiminase Type 4/physiology ; Reperfusion Injury/etiology ; Reperfusion Injury/metabolism ; Specific Pathogen-Free Organisms ; Toll-Like Receptor 9/deficiency ; Warm Ischemia/adverse effects
Czasopismo naukowe
Tytuł :
Astroglial TLR9 antagonism promotes chemotaxis and alternative activation of macrophages via modulation of astrocyte-derived signals: implications for spinal cord injury.
Autorzy :
Li L; Reynolds Family Spine Laboratory, Department of Neurosurgery, New Jersey Medical School, Rutgers, The State University of New Jersey, 205 South Orange Avenue, F-1204, Newark, NJ, 07103, USA.
Ni L; Reynolds Family Spine Laboratory, Department of Neurosurgery, New Jersey Medical School, Rutgers, The State University of New Jersey, 205 South Orange Avenue, F-1204, Newark, NJ, 07103, USA.
Heary RF; Reynolds Family Spine Laboratory, Department of Neurosurgery, New Jersey Medical School, Rutgers, The State University of New Jersey, 205 South Orange Avenue, F-1204, Newark, NJ, 07103, USA.
Elkabes S; Reynolds Family Spine Laboratory, Department of Neurosurgery, New Jersey Medical School, Rutgers, The State University of New Jersey, 205 South Orange Avenue, F-1204, Newark, NJ, 07103, USA. .
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Źródło :
Journal of neuroinflammation [J Neuroinflammation] 2020 Feb 25; Vol. 17 (1), pp. 73. Date of Electronic Publication: 2020 Feb 25.
Typ publikacji :
Journal Article
MeSH Terms :
Astrocytes/*metabolism
Chemotaxis, Leukocyte/*physiology
Macrophage Activation/*physiology
Spinal Cord Injuries/*physiopathology
Toll-Like Receptor 9/*antagonists & inhibitors
Animals ; Female ; Mice ; Mice, Inbred C57BL ; Spinal Cord Injuries/immunology ; Spinal Cord Injuries/metabolism ; Toll-Like Receptor 9/immunology
Czasopismo naukowe
Tytuł :
Triad3A attenuates pathological cardiac hypertrophy involving the augmentation of ubiquitination-mediated degradation of TLR4 and TLR9.
Autorzy :
Lu X; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China.
He Y; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China.
Tang C; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China.
Wang X; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China.
Que L; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China.
Zhu G; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China.
Liu L; Department of Geriatrics, The First Affiliated Hospital of Nanjing Medical University, Nanjing, 210029, China.
Ha T; Department of Surgery, East Tennessee State University, Campus Box 70575, Johnson City, TN, 37614-0575, USA.
Chen Q; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China.
Li C; Department of Surgery, East Tennessee State University, Campus Box 70575, Johnson City, TN, 37614-0575, USA.
Xu Y; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China.; Institute of Biomedical Research, Liaocheng University, Liaocheng, China.
Li J; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China. .
Li Y; Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center For Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, 211166, China. .
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Źródło :
Basic research in cardiology [Basic Res Cardiol] 2020 Feb 01; Vol. 115 (2), pp. 19. Date of Electronic Publication: 2020 Feb 01.
Typ publikacji :
Journal Article
MeSH Terms :
Ventricular Function, Left*
Ventricular Remodeling*
Hypertrophy, Left Ventricular/*prevention & control
Myocardium/*enzymology
Toll-Like Receptor 4/*metabolism
Toll-Like Receptor 9/*metabolism
Ubiquitin-Protein Ligases/*metabolism
Animals ; Cells, Cultured ; Disease Models, Animal ; Hypertrophy, Left Ventricular/enzymology ; Hypertrophy, Left Ventricular/pathology ; Hypertrophy, Left Ventricular/physiopathology ; Male ; Mice, Inbred C57BL ; Myocardium/pathology ; NF-kappa B/metabolism ; Proteolysis ; Proto-Oncogene Proteins c-akt ; Rats, Sprague-Dawley ; Signal Transduction ; Toll-Like Receptor 4/genetics ; Toll-Like Receptor 9/genetics ; Ubiquitin-Protein Ligases/genetics ; Ubiquitination
Czasopismo naukowe
Tytuł :
Prediction of novel mouse TLR9 agonists using a random forest approach.
Autorzy :
Khanna V; College of Medicine and Public Health, Flinders University, Adelaide, SA, 5042, Australia.; Vaxine Pty Ltd, 11 Walkley Avenue, Warradale, Adelaide, SA, 5042, Australia.
Li L; College of Medicine and Public Health, Flinders University, Adelaide, SA, 5042, Australia.; Vaxine Pty Ltd, 11 Walkley Avenue, Warradale, Adelaide, SA, 5042, Australia.
Fung J; Vaxine Pty Ltd, 11 Walkley Avenue, Warradale, Adelaide, SA, 5042, Australia.
Ranganathan S; Department of Molecular Sciences, Macquarie University, Sydney, NSW, 2109, Australia.
Petrovsky N; College of Medicine and Public Health, Flinders University, Adelaide, SA, 5042, Australia. .; Vaxine Pty Ltd, 11 Walkley Avenue, Warradale, Adelaide, SA, 5042, Australia. .
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Źródło :
BMC molecular and cell biology [BMC Mol Cell Biol] 2019 Dec 20; Vol. 20 (Suppl 2), pp. 56. Date of Electronic Publication: 2019 Dec 20.
Typ publikacji :
Journal Article
MeSH Terms :
Algorithms*
Adjuvants, Immunologic/*chemistry
Drug Discovery/*methods
Oligodeoxyribonucleotides/*chemistry
Toll-Like Receptor 9/*agonists
Adjuvants, Immunologic/pharmacology ; Amino Acid Motifs ; Animals ; Mice ; Neural Networks, Computer ; Oligodeoxyribonucleotides/pharmacology ; Reproducibility of Results ; Structure-Activity Relationship ; Toll-Like Receptor 9/chemistry ; Toll-Like Receptor 9/metabolism
Czasopismo naukowe
Tytuł :
The adjuvant effect of melanin is superior to incomplete Freund's adjuvant in subunit/peptide vaccines in mice.
Autorzy :
Cuzzubbo S; PARCC, INSERM, UMR-970, Université de Paris, 56 rue Leblanc, 75015, Paris, France. .
Banissi C; Laboratoire de Recherches Biochirurgicales (Fondation Carpentier), Assistance Publique-Hôpitaux de Paris (AP-HP), Hôpital Européen Georges Pompidou, 75015, Paris, France.
Rouchon MS; Laboratoire de Recherches Biochirurgicales (Fondation Carpentier), Assistance Publique-Hôpitaux de Paris (AP-HP), Hôpital Européen Georges Pompidou, 75015, Paris, France.
Tran T; PARCC, INSERM, UMR-970, Université de Paris, 56 rue Leblanc, 75015, Paris, France.
Tanchot C; PARCC, INSERM, UMR-970, Université de Paris, 56 rue Leblanc, 75015, Paris, France.
Tartour E; PARCC, INSERM, UMR-970, Université de Paris, 56 rue Leblanc, 75015, Paris, France.; Service d'Immunologie Biologique, Assistance Publique-Hôpitaux de Paris (AP-HP), Hôpital Européen Georges Pompidou, 75015, Paris, France.
Carpentier AF; PARCC, INSERM, UMR-970, Université de Paris, 56 rue Leblanc, 75015, Paris, France.; Laboratoire de Recherches Biochirurgicales (Fondation Carpentier), Assistance Publique-Hôpitaux de Paris (AP-HP), Hôpital Européen Georges Pompidou, 75015, Paris, France.; Service de Neurologie, Assistance Publique-Hôpitaux de Paris (AP-HP), Hôpital Saint-Louis, 75010, Paris, France.
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Źródło :
Cancer immunology, immunotherapy : CII [Cancer Immunol Immunother] 2020 Dec; Vol. 69 (12), pp. 2501-2512. Date of Electronic Publication: 2020 Jun 19.
Typ publikacji :
Journal Article
MeSH Terms :
Adjuvants, Immunologic/*administration & dosage
Cancer Vaccines/*immunology
Melanins/*immunology
Neoplasms/*therapy
Animals ; Antigens, Neoplasm/genetics ; Antigens, Neoplasm/immunology ; Cancer Vaccines/administration & dosage ; Cancer Vaccines/genetics ; Cell Line, Tumor/transplantation ; Disease Models, Animal ; Female ; Freund's Adjuvant/administration & dosage ; Freund's Adjuvant/immunology ; Humans ; Immunogenicity, Vaccine ; Lipids/administration & dosage ; Lipids/immunology ; Melanins/administration & dosage ; Mice ; Neoplasms/immunology ; Neoplasms/pathology ; Oligodeoxyribonucleotides/administration & dosage ; Oligodeoxyribonucleotides/immunology ; Ovalbumin/genetics ; Ovalbumin/immunology ; Peptide Fragments/administration & dosage ; Peptide Fragments/genetics ; Peptide Fragments/immunology ; T-Lymphocytes, Cytotoxic/immunology ; Toll-Like Receptor 9/agonists ; Toll-Like Receptor 9/immunology ; Vaccines, Subunit/administration & dosage ; Vaccines, Subunit/genetics ; Vaccines, Subunit/immunology
Czasopismo naukowe
Tytuł :
Enteral virus depletion modulates experimental acute pancreatitis via toll-like receptor 9 signaling.
Autorzy :
Li J; State Key Laboratory of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China; School of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China.
Pan X; State Key Laboratory of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China; School of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China.
Yang J; Affiliated Hospital of Jiangnan University, Wuxi, Jiangsu, PR China.
Jia L; Wuxi School of Medicine, Jiangnan University, Wuxi, Jiangsu, PR China.
Wu C; State Key Laboratory of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China; School of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China.
Liu H; State Key Laboratory of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China; School of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China.
Ren Z; State Key Laboratory of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China; School of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China.
Pan LL; Wuxi School of Medicine, Jiangnan University, Wuxi, Jiangsu, PR China. Electronic address: .
Sun J; State Key Laboratory of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China; School of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, PR China. Electronic address: .
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Źródło :
Biochemical pharmacology [Biochem Pharmacol] 2020 Jan; Vol. 171, pp. 113710. Date of Electronic Publication: 2019 Nov 11.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Antiviral Agents/*pharmacology
Intestinal Mucosa/*metabolism
Pancreatitis/*metabolism
Toll-Like Receptor 9/*metabolism
Viruses/*drug effects
Animals ; Ceruletide ; Chemokines/metabolism ; Female ; Interleukin-6/metabolism ; Intestinal Mucosa/virology ; Mice, Inbred BALB C ; Pancreatitis/chemically induced ; STAT3 Transcription Factor/metabolism ; Signal Transduction/drug effects ; Toll-Like Receptor 9/agonists ; Toll-Like Receptor 9/antagonists & inhibitors ; Virulence/drug effects ; Viruses/pathogenicity
Czasopismo naukowe
Tytuł :
Inhibition of Ctsk modulates periodontitis with arthritis via downregulation of TLR9 and autophagy.
Autorzy :
Wei W; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Ren J; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Yin W; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.; Stomatological Hospital, Southern Medical University, Guangzhou, China.
Ding H; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Lu Q; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Tan L; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Deng S; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Liu J; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Yang Q; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Wang J; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Wang M; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Yue Y; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
Hao L; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, China.
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Źródło :
Cell proliferation [Cell Prolif] 2020 Jan; Vol. 53 (1), pp. e12722. Date of Electronic Publication: 2019 Nov 18.
Typ publikacji :
Journal Article
MeSH Terms :
Arthritis, Rheumatoid/*drug therapy
Cathepsin K/*antagonists & inhibitors
Down-Regulation/*drug effects
Oligodeoxyribonucleotides/*pharmacology
Periodontitis/*drug therapy
Toll-Like Receptor 9/*immunology
Animals ; Arthritis, Rheumatoid/genetics ; Arthritis, Rheumatoid/immunology ; Arthritis, Rheumatoid/pathology ; Basic Helix-Loop-Helix Leucine Zipper Transcription Factors/genetics ; Basic Helix-Loop-Helix Leucine Zipper Transcription Factors/immunology ; Cathepsin K/genetics ; Cathepsin K/immunology ; Disease Models, Animal ; Male ; Mice ; Mice, Inbred DBA ; Microtubule-Associated Proteins/genetics ; Microtubule-Associated Proteins/immunology ; Oligodeoxyribonucleotides/genetics ; Periodontitis/genetics ; Periodontitis/immunology ; Periodontitis/pathology ; Toll-Like Receptor 9/genetics
Czasopismo naukowe
Tytuł :
IL-17A activated by Toll-like receptor 9 contributes to the development of septic acute kidney injury.
Autorzy :
Naito Y; First Department of Medicine, Hamamatsu University School of Medicine, Hamamatsu, Shizuoka, Japan.
Tsuji T; First Department of Medicine, Hamamatsu University School of Medicine, Hamamatsu, Shizuoka, Japan.
Nagata S; First Department of Medicine, Hamamatsu University School of Medicine, Hamamatsu, Shizuoka, Japan.
Tsuji N; First Department of Medicine, Hamamatsu University School of Medicine, Hamamatsu, Shizuoka, Japan.
Fujikura T; First Department of Medicine, Hamamatsu University School of Medicine, Hamamatsu, Shizuoka, Japan.
Ohashi N; First Department of Medicine, Hamamatsu University School of Medicine, Hamamatsu, Shizuoka, Japan.
Kato A; Division of Blood Purification, Hamamatsu University School of Medicine Hospital, Hamamatsu, Shizuoka, Japan.
Miyajima H; First Department of Medicine, Hamamatsu University School of Medicine, Hamamatsu, Shizuoka, Japan.
Yasuda H; First Department of Medicine, Hamamatsu University School of Medicine, Hamamatsu, Shizuoka, Japan.
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Źródło :
American journal of physiology. Renal physiology [Am J Physiol Renal Physiol] 2020 Jan 01; Vol. 318 (1), pp. F238-F247. Date of Electronic Publication: 2019 Nov 25.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Acute Kidney Injury/*metabolism
Interleukin-17/*metabolism
Sepsis/*metabolism
Toll-Like Receptor 9/*metabolism
Acute Kidney Injury/pathology ; Animals ; Apoptosis ; Cytokines/metabolism ; Dendritic Cells/metabolism ; Disease Models, Animal ; Interleukin-17/genetics ; Interleukin-17/pharmacology ; Kidney/drug effects ; Kidney/metabolism ; Kidney/pathology ; Mice ; Mice, Knockout ; Sepsis/pathology ; Spleen/metabolism ; Toll-Like Receptor 9/genetics
Czasopismo naukowe
Tytuł :
Fcγ receptors and toll-like receptor 9 synergize to drive immune complex-induced dendritic cell maturation.
Autorzy :
Nelson NLJ; Department of Immunology and Microbial Disease, Albany Medical College, 47 New Scotland Avenue, Albany, NY 12208, United States. Electronic address: .
Zajd CM; Department of Regenerative and Cancer Cell Biology, Albany Medical College, 47 New Scotland Avenue, Albany, NY 12208, United States.
Lennartz MR; Department of Regenerative and Cancer Cell Biology, Albany Medical College, 47 New Scotland Avenue, Albany, NY 12208, United States.
Gosselin EJ; Department of Immunology and Microbial Disease, Albany Medical College, 47 New Scotland Avenue, Albany, NY 12208, United States. Electronic address: .
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Źródło :
Cellular immunology [Cell Immunol] 2019 Nov; Vol. 345, pp. 103962. Date of Electronic Publication: 2019 Aug 03.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Cell Differentiation/*immunology
Dendritic Cells/*immunology
Myeloid Differentiation Factor 88/*immunology
Receptors, IgG/*immunology
Toll-Like Receptor 9/*immunology
Animals ; Antibodies, Monoclonal/immunology ; Bacterial Vaccines/immunology ; Cytokines/immunology ; Cytokines/metabolism ; Dendritic Cells/metabolism ; Francisella tularensis/immunology ; Humans ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Transgenic ; Myeloid Differentiation Factor 88/metabolism ; Receptors, IgG/genetics ; Toll-Like Receptor 2/genetics ; Toll-Like Receptor 2/immunology ; Toll-Like Receptor 2/metabolism ; Toll-Like Receptor 4/genetics ; Toll-Like Receptor 4/immunology ; Toll-Like Receptor 4/metabolism ; Toll-Like Receptor 9/genetics ; Toll-Like Receptor 9/metabolism ; Tularemia/immunology ; Tularemia/microbiology
Czasopismo naukowe
Tytuł :
Lactoferrin deficiency induces a pro-metastatic tumor microenvironment through recruiting myeloid-derived suppressor cells in mice.
Autorzy :
Wei L; Hunan Key Laboratory of Nonresolving Inflammation and Cancer, Third Xiangya Hospital, Central South University, Changsha, China.; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.; Key Laboratory of Carcinogenesis of Ministry of Health, Changsha, China.; Key Laboratory of Carcinogenesis and Cancer Invasion of Ministry of Education, Changsha, China.; Hunan Key Laboratory of Translational Radiation Oncology, Hunan Cancer Hospital and The Affiliated Cancer Hospital of Xiangya School of Medicine, Central South University, Changsha, China.
Zhang X; Hunan Key Laboratory of Nonresolving Inflammation and Cancer, Third Xiangya Hospital, Central South University, Changsha, China.; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.; Hunan Key Laboratory of Translational Radiation Oncology, Hunan Cancer Hospital and The Affiliated Cancer Hospital of Xiangya School of Medicine, Central South University, Changsha, China.
Wang J; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.
Ye Q; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.
Zheng X; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.
Peng Q; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.
Zheng Y; Center for Medical Research, Second Xiangya Hospital, Central South University, Changsha, China.
Liu P; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.
Zhang X; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.
Li Z; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.
Liu C; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.
Yan Q; Department of Clinical Laboratory, Xiangya Hospital, Central South University, Changsha, China.
Li G; Hunan Key Laboratory of Nonresolving Inflammation and Cancer, Third Xiangya Hospital, Central South University, Changsha, China.; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China.; Key Laboratory of Carcinogenesis of Ministry of Health, Changsha, China.; Key Laboratory of Carcinogenesis and Cancer Invasion of Ministry of Education, Changsha, China.
Ma J; Hunan Key Laboratory of Nonresolving Inflammation and Cancer, Third Xiangya Hospital, Central South University, Changsha, China. .; Cancer Research Institute, School of Basic Medical Science, Central South University, Changsha, China. .; Key Laboratory of Carcinogenesis of Ministry of Health, Changsha, China. .; Key Laboratory of Carcinogenesis and Cancer Invasion of Ministry of Education, Changsha, China. .; Hunan Key Laboratory of Translational Radiation Oncology, Hunan Cancer Hospital and The Affiliated Cancer Hospital of Xiangya School of Medicine, Central South University, Changsha, China. .
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Źródło :
Oncogene [Oncogene] 2020 Jan; Vol. 39 (1), pp. 122-135. Date of Electronic Publication: 2019 Aug 28.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Lactoferrin/*genetics
Melanoma, Experimental/*genetics
Myeloid-Derived Suppressor Cells/*metabolism
Toll-Like Receptor 9/*genetics
Animals ; Apoptosis/genetics ; Cell Differentiation/genetics ; Cell Line, Tumor ; Disease Models, Animal ; Gene Expression Regulation, Neoplastic/genetics ; Heterografts ; Humans ; Immunity, Innate/genetics ; Lactoferrin/deficiency ; Lactoferrin/pharmacology ; Lung/metabolism ; Lung/pathology ; Melanoma, Experimental/pathology ; Mice ; Mice, Knockout ; Myeloid-Derived Suppressor Cells/pathology ; Neoplasm Metastasis ; Signal Transduction/genetics ; Toll-Like Receptor 9/agonists ; Tumor Microenvironment/genetics
Czasopismo naukowe

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