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Wyświetlanie 1-11 z 11
Tytuł:
Characterization of the Roles of SGT1/RAR1, EDS1/NDR1, NPR1, and NRC/ADR1/NRG1 in Sw-5b -Mediated Resistance to Tomato Spotted Wilt Virus.
Autorzy:
Chen Z; Key Laboratory of Plant Immunity, Department of Plant Pathology, Nanjing Agricultural University, Nanjing 210095, China.
Wu Q; Key Laboratory of Plant Immunity, Department of Plant Pathology, Nanjing Agricultural University, Nanjing 210095, China.
Tong C; Key Laboratory of Plant Immunity, Department of Plant Pathology, Nanjing Agricultural University, Nanjing 210095, China.
Chen H; Key Laboratory of Plant Immunity, Department of Plant Pathology, Nanjing Agricultural University, Nanjing 210095, China.
Miao D; Key Laboratory of Plant Immunity, Department of Plant Pathology, Nanjing Agricultural University, Nanjing 210095, China.
Qian X; Key Laboratory of Plant Immunity, Department of Plant Pathology, Nanjing Agricultural University, Nanjing 210095, China.
Zhao X; Key Laboratory of Plant Immunity, Department of Plant Pathology, Nanjing Agricultural University, Nanjing 210095, China.
Jiang L; Key Laboratory of Plant Immunity, Department of Plant Pathology, Nanjing Agricultural University, Nanjing 210095, China.
Tao X; Key Laboratory of Plant Immunity, Department of Plant Pathology, Nanjing Agricultural University, Nanjing 210095, China.
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Źródło:
Viruses [Viruses] 2021 Jul 25; Vol. 13 (8). Date of Electronic Publication: 2021 Jul 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Disease Resistance/*genetics
Solanum lycopersicum/*virology
Plant Proteins/*genetics
Signal Transduction/*genetics
Tospovirus/*genetics
Gene Silencing ; Immunity, Innate ; Solanum lycopersicum/immunology ; Plant Diseases/virology ; Plant Immunity/genetics ; Plant Proteins/classification ; Plant Proteins/immunology ; Plant Proteins/metabolism ; Plants, Genetically Modified/virology ; Protein Domains ; Signal Transduction/immunology ; Tospovirus/metabolism
Czasopismo naukowe
Tytuł:
Hypoxia-induced Downregulation of SRC-3 Suppresses Trophoblastic Invasion and Migration Through Inhibition of the AKT/mTOR Pathway: Implications for the Pathogenesis of Preeclampsia.
Autorzy:
He C; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China.; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Shan N; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China.; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Xu P; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China.; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Ge H; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China.; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Yuan Y; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China.; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Liu Y; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China.; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Zhang P; College of Pharmacy, Chongqing Medical University, Chongqing, 400016, China.
Wen L; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China.; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Zhang F; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China.; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Xiong L; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China.; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Peng C; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Qi H; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China. .; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China. .; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China. .
Tong C; Department of Obstetrics, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China. .; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China. .; State Key Laboratory of Maternal and Fetal Medicine of Chongqing Municipality, The First Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China. .
Baker PN; International Collaborative Joint Laboratory of Reproduction and Development, Ministry of Education of China, Chongqing Medical University, Chongqing, 400016, China.; Liggins Institute, University of Auckland, Auckland, 1142, New Zealand.; College of Life Sciences, University of Leicester, Leicester, LE1 7RH, UK.
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Źródło:
Scientific reports [Sci Rep] 2019 Jul 17; Vol. 9 (1), pp. 10349. Date of Electronic Publication: 2019 Jul 17.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Hypoxia/*physiopathology
Nuclear Receptor Coactivator 3/*physiology
Placenta/*metabolism
Pre-Eclampsia/*etiology
Proto-Oncogene Proteins c-akt/*physiology
Signal Transduction/*physiology
TOR Serine-Threonine Kinases/*physiology
Trophoblasts/*physiology
Acetates/pharmacology ; Adult ; Benzopyrans/pharmacology ; Cell Line ; Cell Movement ; Down-Regulation ; Female ; Humans ; Hypoxia/genetics ; Hypoxia-Inducible Factor 1, alpha Subunit/physiology ; Nuclear Receptor Coactivator 3/biosynthesis ; Nuclear Receptor Coactivator 3/genetics ; Pre-Eclampsia/genetics ; Pre-Eclampsia/physiopathology ; Pregnancy ; RNA Interference ; RNA, Small Interfering/genetics ; Uterine Artery/physiopathology ; Vascular Remodeling
Czasopismo naukowe
Tytuł:
11β-Hydroxysteroid Dehydrogenase Type 1(11β-HSD1) mediates insulin resistance through JNK activation in adipocytes.
Autorzy:
Peng K; Chemical Biology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China.; Diabetes Center, Department of Endocrinology, the Second Affiliated Hospital, Wenzhou Medical University, Wenzhou, Zhejiang, China.
Pan Y; Chemical Biology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China.
Li J; Chemical Biology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China.
Khan Z; Department of Pathology and Laboratory Medicine, Western University, London, ON N6A5C1, Canada.
Fan M; Diabetes Center, Department of Endocrinology, the Second Affiliated Hospital, Wenzhou Medical University, Wenzhou, Zhejiang, China.
Yin H; Chemical Biology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China.; Diabetes Center, Department of Endocrinology, the Second Affiliated Hospital, Wenzhou Medical University, Wenzhou, Zhejiang, China.
Tong C; Chemical Biology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China.
Zhao Y; Chemical Biology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China.
Liang G; Chemical Biology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China.
Zheng C; Chemical Biology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China.; Diabetes Center, Department of Endocrinology, the Second Affiliated Hospital, Wenzhou Medical University, Wenzhou, Zhejiang, China.
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Źródło:
Scientific reports [Sci Rep] 2016 Nov 14; Vol. 6, pp. 37160. Date of Electronic Publication: 2016 Nov 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Insulin Resistance*
Signal Transduction*
11-beta-Hydroxysteroid Dehydrogenase Type 1/*metabolism
Adipocytes/*enzymology
Prednisone/*pharmacology
3T3-L1 Cells ; Adipocytes/pathology ; Animals ; Enzyme Activation/drug effects ; MAP Kinase Kinase 4/antagonists & inhibitors ; MAP Kinase Kinase 4/metabolism ; Male ; Mice
Czasopismo naukowe
Tytuł:
Regulation of Smoothened Phosphorylation and High-Level Hedgehog Signaling Activity by a Plasma Membrane Associated Kinase.
Autorzy:
Li S; Department of Molecular Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.; Department of Developmental Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.
Li S; Department of Molecular Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.; Department of Developmental Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.
Han Y; Department of Molecular Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.; Department of Developmental Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.
Tong C; Department of Developmental Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.
Wang B; Department of Molecular Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.; Department of Developmental Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.
Chen Y; Department of Developmental Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.; Kunming Institute of Zoology, Chinese Academy of Sciences, Kunming, Yunnan, China.
Jiang J; Department of Molecular Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.; Department of Developmental Biology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.; Department of Pharmacology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States of America.
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Źródło:
PLoS biology [PLoS Biol] 2016 Jun 09; Vol. 14 (6), pp. e1002481. Date of Electronic Publication: 2016 Jun 09 (Print Publication: 2016).
Typ publikacji:
Journal Article
MeSH Terms:
Signal Transduction*
Casein Kinase I/*metabolism
Cell Membrane/*metabolism
Drosophila Proteins/*metabolism
Hedgehog Proteins/*metabolism
Smoothened Receptor/*metabolism
Animals ; Animals, Genetically Modified ; Binding Sites/genetics ; Blotting, Western ; Casein Kinase I/genetics ; Cell Line ; Cilia/genetics ; Cilia/metabolism ; Cyclic AMP-Dependent Protein Kinases/genetics ; Cyclic AMP-Dependent Protein Kinases/metabolism ; Drosophila Proteins/genetics ; Drosophila melanogaster/cytology ; Drosophila melanogaster/genetics ; Drosophila melanogaster/metabolism ; Fluorescence Resonance Energy Transfer ; Hedgehog Proteins/genetics ; Luminescent Proteins/genetics ; Luminescent Proteins/metabolism ; Microscopy, Confocal ; Mutation ; Phosphorylation ; RNA Interference ; Smoothened Receptor/genetics ; Wings, Animal/metabolism
Czasopismo naukowe
Tytuł:
Gossypol suppresses mouse T lymphocytes via inhibition of NFκB, NFAT and AP-1 pathways.
Autorzy:
Song B; Key Laboratory of Zoonosis, Ministry of Education, College of Veterinary Medicine, Jilin University , Changchun, Jilin , People's Republic of China .
Huang G
Tong C
Li G
Wang Z
Xiong Y
Zhang S
Lu J
Guan S
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Źródło:
Immunopharmacology and immunotoxicology [Immunopharmacol Immunotoxicol] 2013 Oct; Vol. 35 (5), pp. 615-21. Date of Electronic Publication: 2013 Aug 28.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Contraceptive Agents, Male/*pharmacology
Gossypol/*pharmacology
Immunosuppressive Agents/*pharmacology
NF-kappa B/*immunology
NFATC Transcription Factors/*immunology
Signal Transduction/*drug effects
Th1 Cells/*immunology
Th2 Cells/*immunology
Transcription Factor AP-1/*immunology
Animals ; Contraceptive Agents, Male/chemistry ; Gossypium/chemistry ; Gossypol/chemistry ; Male ; Mice ; Mice, Inbred BALB C ; NF-kappa B/metabolism ; NFATC Transcription Factors/metabolism ; Plant Roots/chemistry ; Signal Transduction/immunology ; Th1 Cells/metabolism ; Th2 Cells/metabolism ; Transcription Factor AP-1/metabolism
Czasopismo naukowe
Tytuł:
Hedgehog signalling activity of Smoothened requires phosphorylation by protein kinase A and casein kinase I.
Autorzy:
Jia J; Center for Developmental Biology, University of Texas Southwestern Medical Center, Dallas, Texas 75390, USA. />Tong C
Wang B
Luo L
Jiang J
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Źródło:
Nature [Nature] 2004 Dec 23; Vol. 432 (7020), pp. 1045-50.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
MeSH Terms:
Signal Transduction*
Casein Kinase I/*metabolism
Cyclic AMP-Dependent Protein Kinases/*metabolism
Drosophila Proteins/*metabolism
Drosophila melanogaster/*metabolism
Receptors, G-Protein-Coupled/*metabolism
Animals ; Cell Line ; Cell Membrane/metabolism ; Drosophila Proteins/genetics ; Drosophila melanogaster/cytology ; Drosophila melanogaster/enzymology ; Drosophila melanogaster/genetics ; Hedgehog Proteins ; Mutation/genetics ; Phosphorylation ; Receptors, G-Protein-Coupled/genetics ; Smoothened Receptor ; Transgenes/genetics
Czasopismo naukowe
    Wyświetlanie 1-11 z 11

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