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Tytuł :
Protective Effects of Clostridium Butyricum in a Murine Model of Dextran Sodium Sulfate-Induced Colitis That Involve Inhibition of the TLR2 Signaling Pathway and T Helper 17 Cells.
Autorzy :
Xie Y; Department of Gastroenterology, Shengjing Hospital of China Medical University, Shenyang, Liaoning, PR China.
Zhou L; Department of Gastroenterology, Shengjing Hospital of China Medical University, Shenyang, Liaoning, PR China.
Yao X; Department of Gastroenterology, Shengjing Hospital of China Medical University, Shenyang, Liaoning, PR China.
Li Y; Department of Gastroenterology, Shengjing Hospital of China Medical University, Shenyang, Liaoning, PR China. Electronic address: .
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Źródło :
The American journal of the medical sciences [Am J Med Sci] 2020 Aug; Vol. 360 (2), pp. 176-191. Date of Electronic Publication: 2020 May 20.
Typ publikacji :
Journal Article
MeSH Terms :
Clostridium butyricum*
Probiotics*
Colitis/*immunology
Myeloid Differentiation Factor 88/*immunology
Th17 Cells/*immunology
Toll-Like Receptor 2/*immunology
Transcription Factor RelA/*immunology
Animals ; Body Weight ; Colitis/chemically induced ; Dextran Sulfate/toxicity ; Disease Models, Animal ; Drinking Behavior ; Feeding Behavior ; Female ; Gastrointestinal Microbiome ; Inflammatory Bowel Diseases/immunology ; Interleukin-17/genetics ; Interleukin-17/immunology ; Interleukin-17/metabolism ; Interleukin-23/genetics ; Interleukin-23/immunology ; Interleukin-23/metabolism ; Male ; Mice ; Mice, Inbred BALB C ; Myeloid Differentiation Factor 88/genetics ; Myeloid Differentiation Factor 88/metabolism ; Nuclear Receptor Subfamily 1, Group F, Member 3/genetics ; Nuclear Receptor Subfamily 1, Group F, Member 3/immunology ; Nuclear Receptor Subfamily 1, Group F, Member 3/metabolism ; RNA, Messenger/metabolism ; Random Allocation ; Signal Transduction ; Toll-Like Receptor 2/genetics ; Toll-Like Receptor 2/metabolism ; Transcription Factor RelA/genetics ; Transcription Factor RelA/metabolism
Czasopismo naukowe
Tytuł :
Hyperforin induces apoptosis through extrinsic/intrinsic pathways and inhibits EGFR/ERK/NF-κB-mediated anti-apoptotic potential in glioblastoma.
Autorzy :
Hsu FT; Department of Biological Science and Technology, China Medical University, Taichung, Taiwan.
Chen WT; Department of Psychiatry, Zuoying Branch of Kaohsiung Armed Forces General Hospital, Kaohsiung, Taiwan.
Wu CT; Department of Radiation Oncology, Show Chwan Memorial Hospital, Changhua, Taiwan.
Chung JG; Department of Biological Science and Technology, China Medical University, Taichung, Taiwan.; Department of Biotechnology, Asia University, Taichung, Taiwan.
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Źródło :
Environmental toxicology [Environ Toxicol] 2020 Oct; Vol. 35 (10), pp. 1058-1069. Date of Electronic Publication: 2020 Jun 02.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents, Phytogenic/*pharmacology
Apoptosis/*drug effects
Extracellular Signal-Regulated MAP Kinases/*metabolism
Phloroglucinol/*analogs & derivatives
Terpenes/*pharmacology
Transcription Factor RelA/*metabolism
Antineoplastic Agents, Phytogenic/isolation & purification ; Apoptosis Regulatory Proteins/metabolism ; Brain Neoplasms/metabolism ; Brain Neoplasms/pathology ; Cell Line, Tumor ; Cell Survival/drug effects ; ErbB Receptors/metabolism ; Glioblastoma/metabolism ; Glioblastoma/pathology ; Humans ; Hypericum/chemistry ; Phloroglucinol/isolation & purification ; Phloroglucinol/pharmacology ; Signal Transduction ; Terpenes/isolation & purification ; Transcription Factor RelA/genetics
Czasopismo naukowe
Tytuł :
Looking Down on NF-κB.
Autorzy :
Williams LM; Department of Biology, Boston University, Boston, Massachusetts, USA.
Gilmore TD; Department of Biology, Boston University, Boston, Massachusetts, USA .
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Źródło :
Molecular and cellular biology [Mol Cell Biol] 2020 Jul 14; Vol. 40 (15). Date of Electronic Publication: 2020 Jul 14 (Print Publication: 2020).
Typ publikacji :
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Review
MeSH Terms :
Gene Expression Regulation/*genetics
I-kappa B Kinase/*metabolism
NF-kappa B/*metabolism
Transcription Factor RelA/*metabolism
Animals ; Gene Expression Regulation/immunology ; Humans ; I-kappa B Kinase/immunology ; Immunity, Innate/immunology ; NF-kappa B/immunology ; Signal Transduction/physiology ; Transcription Factor RelA/immunology
Czasopismo naukowe
Tytuł :
A20 attenuates hypoxia-induced pulmonary arterial hypertension by inhibiting NF-κB activation and pulmonary artery smooth muscle cell proliferation.
Autorzy :
Chen M; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, 210001, China; Department of Cardiology, The Affiliated Hospital of Yangzhou University, Yangzhou University, Yangzhou, 225012, China.
Ding Z; Department of Cardiology, The Affiliated Hospital of Yangzhou University, Yangzhou University, Yangzhou, 225012, China.
Zhang F; Department of Cardiology, The Affiliated Hospital of Yangzhou University, Yangzhou University, Yangzhou, 225012, China.
Shen H; Department of Cardiology, The Affiliated Hospital of Yangzhou University, Yangzhou University, Yangzhou, 225012, China.
Zhu L; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, 210001, China.
Yang H; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, 210001, China.
Chen S; Department of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, 210001, China. Electronic address: .
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Źródło :
Experimental cell research [Exp Cell Res] 2020 May 15; Vol. 390 (2), pp. 111982. Date of Electronic Publication: 2020 Mar 28.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Hypoxia/*genetics
Myocytes, Smooth Muscle/*metabolism
Pulmonary Arterial Hypertension/*genetics
Transcription Factor RelA/*genetics
Tumor Necrosis Factor alpha-Induced Protein 3/*genetics
Actins/genetics ; Actins/metabolism ; Adenoviridae/genetics ; Adenoviridae/metabolism ; Animals ; Cell Proliferation ; Gene Expression Regulation ; Genetic Vectors/chemistry ; Genetic Vectors/metabolism ; Hypoxia/metabolism ; Hypoxia/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Muscle, Smooth, Vascular/metabolism ; Muscle, Smooth, Vascular/pathology ; Myocytes, Smooth Muscle/pathology ; NF-KappaB Inhibitor alpha/genetics ; NF-KappaB Inhibitor alpha/metabolism ; Primary Cell Culture ; Pulmonary Arterial Hypertension/metabolism ; Pulmonary Arterial Hypertension/pathology ; Pulmonary Artery/metabolism ; Pulmonary Artery/pathology ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Signal Transduction ; Transcription Factor RelA/metabolism ; Tumor Necrosis Factor alpha-Induced Protein 3/antagonists & inhibitors ; Tumor Necrosis Factor alpha-Induced Protein 3/metabolism
Czasopismo naukowe
Tytuł :
Bufalin reverses multidrug resistance by regulating stemness through the CD133/nuclear factor-κB/MDR1 pathway in colorectal cancer.
Autorzy :
Zhan Y; Central Laboratory, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Qiu Y; Interventional Cancer Institute of Chinese Integrative Medicine, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Wang H; Interventional Cancer Institute of Chinese Integrative Medicine, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Wang Z; Department of Pathology, Shuguang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Xu J; Central Laboratory, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Fan G; Interventional Cancer Institute of Chinese Integrative Medicine, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Xu J; Interventional Cancer Institute of Chinese Integrative Medicine, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Li W; Department of General Surgery, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Cao Y; Department of General Surgery, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Le VM; Research Center of Ginseng and Medicinal Materials (CGMM), National Institute of Medicinal Materials, Ho Chi Minh City, Vietnam.
Ly HT; Research Center of Ginseng and Medicinal Materials (CGMM), National Institute of Medicinal Materials, Ho Chi Minh City, Vietnam.
Yuan Z; Interventional Cancer Institute of Chinese Integrative Medicine, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.; Shanghai Putuo Central School of Clinical Medicine, Anhui Medical University, Hefei, China.
Xu K; Central Laboratory, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.; Interventional Cancer Institute of Chinese Integrative Medicine, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.; Shanghai Putuo Central School of Clinical Medicine, Anhui Medical University, Hefei, China.
Yin P; Interventional Cancer Institute of Chinese Integrative Medicine, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.; Department of General Surgery, Putuo Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.; Shanghai Putuo Central School of Clinical Medicine, Anhui Medical University, Hefei, China.
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Źródło :
Cancer science [Cancer Sci] 2020 May; Vol. 111 (5), pp. 1619-1630. Date of Electronic Publication: 2020 Mar 16.
Typ publikacji :
Journal Article
MeSH Terms :
AC133 Antigen/*metabolism
Antineoplastic Agents/*pharmacology
Bufanolides/*pharmacology
Colorectal Neoplasms/*pathology
Drug Resistance, Neoplasm/*drug effects
Transcription Factor RelA/*metabolism
AC133 Antigen/genetics ; ATP Binding Cassette Transporter, Subfamily B/genetics ; ATP Binding Cassette Transporter, Subfamily B/metabolism ; Animals ; Antineoplastic Agents/therapeutic use ; Bufanolides/therapeutic use ; Cell Line, Tumor ; Colorectal Neoplasms/drug therapy ; Colorectal Neoplasms/metabolism ; Doxorubicin/pharmacology ; Doxorubicin/therapeutic use ; Drug Resistance, Multiple/drug effects ; Drug Therapy, Combination ; Gene Expression Regulation, Neoplastic/drug effects ; Humans ; Mice ; Mice, Nude ; Neoplastic Stem Cells/drug effects ; Neoplastic Stem Cells/metabolism ; Neoplastic Stem Cells/pathology ; Signal Transduction ; Transcription Factor RelA/genetics
Czasopismo naukowe
Tytuł :
Krüppel-like factor 3 (KLF3) suppresses NF-κB-driven inflammation in mice.
Autorzy :
Knights AJ; School of Biotechnology and Biomolecular Sciences, University of New South Wales, Sydney, New South Wales 2052, Australia.
Yang L; School of Biotechnology and Biomolecular Sciences, University of New South Wales, Sydney, New South Wales 2052, Australia.
Shah M; School of Biotechnology and Biomolecular Sciences, University of New South Wales, Sydney, New South Wales 2052, Australia.
Norton LJ; School of Biotechnology and Biomolecular Sciences, University of New South Wales, Sydney, New South Wales 2052, Australia.
Green GS; School of Biotechnology and Biomolecular Sciences, University of New South Wales, Sydney, New South Wales 2052, Australia.
Stout ES; School of Biotechnology and Biomolecular Sciences, University of New South Wales, Sydney, New South Wales 2052, Australia.
Vohralik EJ; School of Biotechnology and Biomolecular Sciences, University of New South Wales, Sydney, New South Wales 2052, Australia.
Crossley M; School of Biotechnology and Biomolecular Sciences, University of New South Wales, Sydney, New South Wales 2052, Australia.
Quinlan KGR; School of Biotechnology and Biomolecular Sciences, University of New South Wales, Sydney, New South Wales 2052, Australia .
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Źródło :
The Journal of biological chemistry [J Biol Chem] 2020 May 01; Vol. 295 (18), pp. 6080-6091. Date of Electronic Publication: 2020 Mar 25.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Kruppel-Like Transcription Factors/*metabolism
Transcription Factor RelA/*metabolism
Animals ; Inflammation/genetics ; Inflammation/immunology ; Inflammation/metabolism ; Kruppel-Like Transcription Factors/deficiency ; Macrophages/metabolism ; Mice ; Transcription Factor RelA/genetics ; Transcriptional Activation
Czasopismo naukowe
Tytuł :
Expression of chemokines in macrophage polarization and downregulation of NFκB in aorta allow macrophage polarization by diosgenin in atherosclerosis.
Autorzy :
Binesh A; Department of Biotechnology, Institute of Fisheries Post Graduate Studies, Tamil Nadu Dr. J. Jayalalithaa Fisheries University, Chennai, Tamil Nadu, India.
Devaraj SN; School of Life Sciences, University of Madras, Guindy Campus, Chennai, Tamil Nadu, India.
Devaraj H; School of Life Sciences, University of Madras, Guindy Campus, Chennai, Tamil Nadu, India.
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Źródło :
Journal of biochemical and molecular toxicology [J Biochem Mol Toxicol] 2020 Feb; Vol. 34 (2), pp. e22422. Date of Electronic Publication: 2019 Nov 15.
Typ publikacji :
Journal Article
MeSH Terms :
Cell Polarity*
Aorta/*metabolism
Atherosclerosis/*metabolism
Cytokines/*metabolism
Diosgenin/*pharmacology
Macrophages/*metabolism
Plant Extracts/*pharmacology
Transcription Factor RelA/*metabolism
Animals ; Antigens, CD/genetics ; Antigens, CD/metabolism ; Antigens, Differentiation, Myelomonocytic/genetics ; Antigens, Differentiation, Myelomonocytic/metabolism ; Atherosclerosis/drug therapy ; Atherosclerosis/prevention & control ; CD36 Antigens/genetics ; CD36 Antigens/metabolism ; Cell Differentiation/drug effects ; Cyclooxygenase 2/genetics ; Cyclooxygenase 2/metabolism ; Dexamethasone/pharmacology ; Diet, Atherogenic/adverse effects ; Dioscorea/chemistry ; Diosgenin/therapeutic use ; Humans ; Lipoproteins, LDL/pharmacology ; Male ; Monocytes/metabolism ; Plant Extracts/therapeutic use ; Rats ; Signal Transduction/drug effects ; THP-1 Cells ; Transcription Factor RelA/antagonists & inhibitors ; Transcription Factor RelA/genetics
Czasopismo naukowe
Tytuł :
Giardia lamblia Decreases NF-κB p65 Protein Levels and Modulates LPS-Induced Pro-Inflammatory Response in Macrophages.
Autorzy :
Faria CP; Faculty of Pharmacy, University of Coimbra, Coimbra, Portugal.; Center for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal.
Neves BM; Department of Medical Sciences and Institute of Biomedicine - iBiMED, University of Aveiro, 3810-193, Aveiro, Portugal.
Lourenço Á; Faculty of Pharmacy, University of Coimbra, Coimbra, Portugal.; Center for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal.
Cruz MT; Faculty of Pharmacy, University of Coimbra, Coimbra, Portugal.; Center for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal.
Martins JD; Center for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal.
Silva A; Center for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal.
Pereira S; Faculty of Pharmacy, University of Coimbra, Coimbra, Portugal.; Center for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal.
Sousa MDC; Faculty of Pharmacy, University of Coimbra, Coimbra, Portugal. .; Center for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal. .
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Źródło :
Scientific reports [Sci Rep] 2020 Apr 10; Vol. 10 (1), pp. 6234. Date of Electronic Publication: 2020 Apr 10.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Giardia lamblia/*immunology
Giardiasis/*immunology
Host-Parasite Interactions/*immunology
Macrophages/*immunology
Transcription Factor RelA/*metabolism
Animals ; Blood Buffy Coat/cytology ; Giardiasis/parasitology ; Healthy Volunteers ; Humans ; Lipopolysaccharides/immunology ; Macrophages/metabolism ; Mice ; Peptide Hydrolases/metabolism ; Primary Cell Culture ; Protease Inhibitors/pharmacology ; Proteolysis/drug effects ; Protozoan Proteins/antagonists & inhibitors ; Protozoan Proteins/metabolism ; RAW 264.7 Cells ; Transcription Factor RelA/analysis
Czasopismo naukowe
Tytuł :
PLK1/NF-κB feedforward circuit antagonizes the mono-ADP-ribosyltransferase activity of PARP10 and facilitates HCC progression.
Autorzy :
Tian L; The Department of hepatopancreatobiliary surgery of the Affiliated Hospital, Qingdao University, Qingdao, Shandong, China.; Department of Hepatobiliary Surgery, First Clinical Medical Center of Chinese PLA General Hospital, Beijing, China.
Yao K; The Department of Gynaecology and Obstetrics of the Affiliated Hospital, Qingdao University, Qingdao, Shandong, China.
Liu K; The General Surgery Department, The 971st Hospital of the PLA Navy, Qingdao, Shandong, China.
Han B; The Department of hepatopancreatobiliary surgery of the Affiliated Hospital, Qingdao University, Qingdao, Shandong, China.
Dong H; The Department of General Surgery, Qilu Hospital of Shandong University, Qingdao, Shandong, China.
Zhao W; The Department of hepatopancreatobiliary surgery of the Affiliated Hospital, Qingdao University, Qingdao, Shandong, China.
Jiang W; The Department of hepatopancreatobiliary surgery of the Affiliated Hospital, Qingdao University, Qingdao, Shandong, China.
Qiu F; The Department of hepatopancreatobiliary surgery of the Affiliated Hospital, Qingdao University, Qingdao, Shandong, China.
Qu L; The Department of hepatopancreatobiliary surgery of the Affiliated Hospital, Qingdao University, Qingdao, Shandong, China.
Wu Z; The Department of hepatopancreatobiliary surgery of the Affiliated Hospital, Qingdao University, Qingdao, Shandong, China.
Zhou B; The Department of hepatopancreatobiliary surgery of the Affiliated Hospital, Qingdao University, Qingdao, Shandong, China.
Zhong M; Department of Gastrointestinal Surgery, Zhongshan Hospital of Xiamen University, Xiamen, Fujian, China.; Institute of Gastrointestinal Oncology, School of Medicine, Xiamen University, Xiamen, Fujian, China.
Zhao J; Department of Gastrointestinal Surgery, Zhongshan Hospital of Xiamen University, Xiamen, Fujian, China.; Institute of Gastrointestinal Oncology, School of Medicine, Xiamen University, Xiamen, Fujian, China.
Qiu X; Department of Gastrointestinal Surgery, Zhongshan Hospital of Xiamen University, Xiamen, Fujian, China.; Institute of Gastrointestinal Oncology, School of Medicine, Xiamen University, Xiamen, Fujian, China.
Zhong L; Department of Gastrointestinal Surgery, Zhongshan Hospital of Xiamen University, Xiamen, Fujian, China.; Institute of Gastrointestinal Oncology, School of Medicine, Xiamen University, Xiamen, Fujian, China.
Guo X; The Department of hepatopancreatobiliary surgery of the Affiliated Hospital, Zhongshan University, Guangzhou, Guangdong, China.
Shi T; Department of Pharmacy, the First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, Anhui, China. .
Hong X; Department of Gastrointestinal Surgery, Zhongshan Hospital of Xiamen University, Xiamen, Fujian, China. .; Institute of Gastrointestinal Oncology, School of Medicine, Xiamen University, Xiamen, Fujian, China. .
Lu S; Department of Hepatobiliary Surgery, First Clinical Medical Center of Chinese PLA General Hospital, Beijing, China. .
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Źródło :
Oncogene [Oncogene] 2020 Apr; Vol. 39 (15), pp. 3145-3162. Date of Electronic Publication: 2020 Feb 14.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Carcinoma, Hepatocellular/*pathology
Cell Cycle Proteins/*metabolism
Liver Neoplasms/*pathology
Poly(ADP-ribose) Polymerases/*metabolism
Protein-Serine-Threonine Kinases/*metabolism
Proto-Oncogene Proteins/*metabolism
Transcription Factor RelA/*metabolism
Animals ; Antineoplastic Agents/pharmacology ; Antineoplastic Agents/therapeutic use ; Carcinogenesis/drug effects ; Carcinogenesis/pathology ; Carcinoma, Hepatocellular/mortality ; Carcinoma, Hepatocellular/therapy ; Cell Cycle Proteins/antagonists & inhibitors ; Disease Progression ; Feedback, Physiological ; Female ; HEK293 Cells ; Hepatectomy ; Humans ; Kaplan-Meier Estimate ; Liver/pathology ; Liver/surgery ; Liver Neoplasms/mortality ; Liver Neoplasms/therapy ; Male ; Mice ; Middle Aged ; Mutagenesis, Site-Directed ; Neoplasm Staging ; Nitriles/pharmacology ; Nitriles/therapeutic use ; Phosphorylation/drug effects ; Poly(ADP-ribose) Polymerases/genetics ; Protein-Serine-Threonine Kinases/antagonists & inhibitors ; Proto-Oncogene Proteins/antagonists & inhibitors ; Proto-Oncogene Proteins/genetics ; Pteridines/pharmacology ; Pteridines/therapeutic use ; Signal Transduction/drug effects ; Signal Transduction/genetics ; Staurosporine/pharmacology ; Staurosporine/therapeutic use ; Sulfones/pharmacology ; Sulfones/therapeutic use ; Transcription Factor RelA/antagonists & inhibitors ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
Aptamer-based optical manipulation of protein subcellular localization in cells.
Autorzy :
Xie S; Molecular Science and Biomedicine Laboratory (MBL), State Key Laboratory of Chemo/Bio-Sensing and Chemometrics, College of Chemistry and Chemical Engineering, College of Biology, Collaborative Innovation Center for Chemistry and Molecular Medicine, Hunan University, Changsha, 410082, China.
Du Y; Molecular Science and Biomedicine Laboratory (MBL), State Key Laboratory of Chemo/Bio-Sensing and Chemometrics, College of Chemistry and Chemical Engineering, College of Biology, Collaborative Innovation Center for Chemistry and Molecular Medicine, Hunan University, Changsha, 410082, China.
Zhang Y; Molecular Science and Biomedicine Laboratory (MBL), State Key Laboratory of Chemo/Bio-Sensing and Chemometrics, College of Chemistry and Chemical Engineering, College of Biology, Collaborative Innovation Center for Chemistry and Molecular Medicine, Hunan University, Changsha, 410082, China.
Wang Z; Molecular Science and Biomedicine Laboratory (MBL), State Key Laboratory of Chemo/Bio-Sensing and Chemometrics, College of Chemistry and Chemical Engineering, College of Biology, Collaborative Innovation Center for Chemistry and Molecular Medicine, Hunan University, Changsha, 410082, China.
Zhang D; Molecular Science and Biomedicine Laboratory (MBL), State Key Laboratory of Chemo/Bio-Sensing and Chemometrics, College of Chemistry and Chemical Engineering, College of Biology, Collaborative Innovation Center for Chemistry and Molecular Medicine, Hunan University, Changsha, 410082, China.
He L; Molecular Science and Biomedicine Laboratory (MBL), State Key Laboratory of Chemo/Bio-Sensing and Chemometrics, College of Chemistry and Chemical Engineering, College of Biology, Collaborative Innovation Center for Chemistry and Molecular Medicine, Hunan University, Changsha, 410082, China.
Qiu L; Molecular Science and Biomedicine Laboratory (MBL), State Key Laboratory of Chemo/Bio-Sensing and Chemometrics, College of Chemistry and Chemical Engineering, College of Biology, Collaborative Innovation Center for Chemistry and Molecular Medicine, Hunan University, Changsha, 410082, China. .
Jiang J; Molecular Science and Biomedicine Laboratory (MBL), State Key Laboratory of Chemo/Bio-Sensing and Chemometrics, College of Chemistry and Chemical Engineering, College of Biology, Collaborative Innovation Center for Chemistry and Molecular Medicine, Hunan University, Changsha, 410082, China. .
Tan W; Molecular Science and Biomedicine Laboratory (MBL), State Key Laboratory of Chemo/Bio-Sensing and Chemometrics, College of Chemistry and Chemical Engineering, College of Biology, Collaborative Innovation Center for Chemistry and Molecular Medicine, Hunan University, Changsha, 410082, China. .; Institute of Cancer and Basic Medicine (IBMC), Chinese Academy of Sciences, The Cancer Hospital of the University of Chinese Academy of Sciences, Hangzhou, Zhejiang, 310022, China. .; Institute of Molecular Medicine (IMM), Renji Hospital, State Key Laboratory of Oncogenes and Related Genes, Shanghai Jiao Tong University School of Medicine, and College of Chemistry and Chemical Engineering, Shanghai Jiao Tong University, Shanghai, 200240, China. .
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Źródło :
Nature communications [Nat Commun] 2020 Mar 12; Vol. 11 (1), pp. 1347. Date of Electronic Publication: 2020 Mar 12.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Aptamers, Nucleotide/*genetics
Transcription Factor RelA/*metabolism
Aptamers, Nucleotide/metabolism ; Cell Nucleus/genetics ; Cell Nucleus/metabolism ; Cytoplasm/genetics ; Cytoplasm/metabolism ; Humans ; Optogenetics ; Protein Transport ; Signal Transduction ; Transcription Factor RelA/genetics
Czasopismo naukowe
Tytuł :
Modulation of Microglia Polarization through Silencing of NF-κB p65 by Functionalized Curdlan Nanoparticle-Mediated RNAi.
Autorzy :
Ganbold T; Institute of Mongolian Medicinal Chemistry, School of Chemistry & Chemical Engineering, Inner Mongolia University, Hohhot, Inner Mongolia 010020, P. R. China.; School of Life Sciences, Inner Mongolia University, 24 Zhaojun Road, Hohhot, Inner Mongolia Autonomous Region 010020, PR China.
Bao Q; Institute of Mongolian Medicinal Chemistry, School of Chemistry & Chemical Engineering, Inner Mongolia University, Hohhot, Inner Mongolia 010020, P. R. China.
Zandan J; Institute of Mongolian Medicinal Chemistry, School of Chemistry & Chemical Engineering, Inner Mongolia University, Hohhot, Inner Mongolia 010020, P. R. China.
Hasi A; School of Life Sciences, Inner Mongolia University, 24 Zhaojun Road, Hohhot, Inner Mongolia Autonomous Region 010020, PR China.
Baigude H; Institute of Mongolian Medicinal Chemistry, School of Chemistry & Chemical Engineering, Inner Mongolia University, Hohhot, Inner Mongolia 010020, P. R. China.
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Źródło :
ACS applied materials & interfaces [ACS Appl Mater Interfaces] 2020 Mar 11; Vol. 12 (10), pp. 11363-11374. Date of Electronic Publication: 2020 Mar 02.
Typ publikacji :
Journal Article
MeSH Terms :
Microglia/*drug effects
Nanoparticles/*chemistry
RNA, Small Interfering/*pharmacology
Transcription Factor RelA/*metabolism
beta-Glucans/*pharmacology
Animals ; Cell Physiological Phenomena/drug effects ; Cell Physiological Phenomena/genetics ; Cells, Cultured ; Disease Models, Animal ; Drug Delivery Systems ; Gene Knockdown Techniques ; Infarction, Middle Cerebral Artery/metabolism ; Mice ; Microglia/cytology ; Microglia/metabolism ; Neuroprotective Agents/pharmacology ; RNA Interference ; RNA, Small Interfering/genetics ; Transcription Factor RelA/genetics
Czasopismo naukowe
Tytuł :
Libertellenone M, a diterpene derived from an endophytic fungus Phomopsis sp. S12, protects against DSS-induced colitis via inhibiting both nuclear translocation of NF-κB and NLRP3 inflammasome activation.
Autorzy :
Fan M; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China.
Xiang G; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China.
Chen J; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China.
Gao J; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China.
Xue W; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China.
Wang Y; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China.
Li W; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China.
Zhou L; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China.
Jiao R; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China.
Shen Y; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China. Electronic address: .
Xu Q; State Key Laboratory of Pharmaceutical Biotechnology, School of Life Sciences, Nanjing University, Nanjing 210093, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2020 Mar; Vol. 80, pp. 106144. Date of Electronic Publication: 2020 Jan 09.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*therapeutic use
Colitis/*drug therapy
Inflammasomes/*antagonists & inhibitors
NLR Family, Pyrin Domain-Containing 3 Protein/*antagonists & inhibitors
Transcription Factor RelA/*antagonists & inhibitors
Animals ; Anti-Inflammatory Agents/pharmacology ; Ascomycota ; Colitis/chemically induced ; Colitis/immunology ; Colitis/pathology ; Colon/drug effects ; Colon/immunology ; Colon/pathology ; Cytokines/genetics ; Cytokines/immunology ; Dextran Sulfate ; Female ; Inflammasomes/immunology ; Mice ; Mice, Inbred C57BL ; NLR Family, Pyrin Domain-Containing 3 Protein/immunology ; RAW 264.7 Cells ; Transcription Factor RelA/immunology
Czasopismo naukowe
Tytuł :
Demethyleneberberine attenuates concanavalin A-induced autoimmune hepatitis in mice through inhibition of NF-κB and MAPK signaling.
Autorzy :
Zhang M; State Key Laboratory of Natural Medicines, Department of Biochemistry, China Pharmaceutical University, Nanjing 211198, China.
Li Q; State Key Laboratory of Natural Medicines, Department of Biochemistry, China Pharmaceutical University, Nanjing 211198, China.
Zhou C; State Key Laboratory of Natural Medicines, Department of Biochemistry, China Pharmaceutical University, Nanjing 211198, China.
Zhao Y; State Key Laboratory of Natural Medicines, Department of Biochemistry, China Pharmaceutical University, Nanjing 211198, China.
Li R; State Key Laboratory of Natural Medicines, Department of Biochemistry, China Pharmaceutical University, Nanjing 211198, China.
Zhang Y; State Key Laboratory of Natural Medicines, Department of Biochemistry, China Pharmaceutical University, Nanjing 211198, China. Electronic address: .
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Źródło :
International immunopharmacology [Int Immunopharmacol] 2020 Mar; Vol. 80, pp. 106137. Date of Electronic Publication: 2020 Jan 10.
Typ publikacji :
Journal Article
MeSH Terms :
Berberine/*analogs & derivatives
Hepatitis, Autoimmune/*drug therapy
Mitogen-Activated Protein Kinases/*antagonists & inhibitors
Transcription Factor RelA/*antagonists & inhibitors
Animals ; Berberine/pharmacology ; Berberine/therapeutic use ; Concanavalin A ; Cytokines/genetics ; Cytokines/immunology ; Hepatitis, Autoimmune/immunology ; Liver/drug effects ; Liver/metabolism ; Liver/pathology ; Male ; Mice, Inbred BALB C ; Mitogen-Activated Protein Kinases/immunology ; Signal Transduction/drug effects ; Transcription Factor RelA/immunology
Czasopismo naukowe
Tytuł :
Hydroxytyrosol Inhibits LPS-Induced Neuroinflammatory Responses via Suppression of TLR-4-Mediated NF-κB P65 Activation and ERK Signaling Pathway.
Autorzy :
Zhang L; Tianjin Key Laboratory of Acute Abdomen Disease Associated Organ Injury and ITCWM Repair, Institute of Acute Abdominal Diseases, Tianjin Nankai Hospital, Tianjin 300100, China. Electronic address: .
Zhang J; Tianjin Key Laboratory of Acute Abdomen Disease Associated Organ Injury and ITCWM Repair, Institute of Acute Abdominal Diseases, Tianjin Nankai Hospital, Tianjin 300100, China.
Jiang X; Tianjin Key Laboratory of Acute Abdomen Disease Associated Organ Injury and ITCWM Repair, Institute of Acute Abdominal Diseases, Tianjin Nankai Hospital, Tianjin 300100, China.
Yang L; Tianjin Key Laboratory of Acute Abdomen Disease Associated Organ Injury and ITCWM Repair, Institute of Acute Abdominal Diseases, Tianjin Nankai Hospital, Tianjin 300100, China.
Zhang Q; Tianjin Key Laboratory of Acute Abdomen Disease Associated Organ Injury and ITCWM Repair, Institute of Acute Abdominal Diseases, Tianjin Nankai Hospital, Tianjin 300100, China.
Wang B; Graduate School, Tianjin Medical University, Tianjin 300070, China.
Cui L; Tianjin Key Laboratory of Acute Abdomen Disease Associated Organ Injury and ITCWM Repair, Institute of Acute Abdominal Diseases, Tianjin Nankai Hospital, Tianjin 300100, China.
Wang X; Tianjin Key Laboratory of Acute Abdomen Disease Associated Organ Injury and ITCWM Repair, Institute of Acute Abdominal Diseases, Tianjin Nankai Hospital, Tianjin 300100, China. Electronic address: .
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Źródło :
Neuroscience [Neuroscience] 2020 Feb 01; Vol. 426, pp. 189-200. Date of Electronic Publication: 2019 Dec 19.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Extracellular Signal-Regulated MAP Kinases/*drug effects
Inflammation/*drug therapy
Phenylethyl Alcohol/*analogs & derivatives
Toll-Like Receptor 4/*drug effects
Transcription Factor RelA/*drug effects
Animals ; Anti-Inflammatory Agents/pharmacology ; Down-Regulation/drug effects ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Inflammation/metabolism ; Lipopolysaccharides/pharmacology ; Microglia/metabolism ; Phenylethyl Alcohol/pharmacology ; Signal Transduction/drug effects ; Toll-Like Receptor 4/metabolism ; Transcription Factor RelA/metabolism
Czasopismo naukowe
Tytuł :
RELA is sufficient to mediate interleukin-1 repression of androgen receptor expression and activity in an LNCaP disease progression model.
Autorzy :
Thomas-Jardin SE; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
Dahl H; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
Kanchwala MS; McDermott Center of Human Growth and Development, The University of Texas Southwestern Medical Center, Dallas, Texas.
Ha F; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
Jacob J; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
Soundharrajan R; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
Bautista M; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
Nawas AF; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
Robichaux D; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
Mistry R; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
Anunobi V; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
Xing C; McDermott Center of Human Growth and Development, The University of Texas Southwestern Medical Center, Dallas, Texas.; Department of Bioinformatics, The University of Texas Southwestern Medical Center, Dallas, Texas.; Department of Clinical Sciences, The University of Texas Southwestern Medical Center, Dallas, Texas.
Delk NA; Biological Sciences Department, The University of Texas at Dallas, Richardson, Texas.
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Źródło :
The Prostate [Prostate] 2020 Feb; Vol. 80 (2), pp. 133-145. Date of Electronic Publication: 2019 Nov 15.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
Interleukin-1/*metabolism
Prostatic Neoplasms/*metabolism
Prostatic Neoplasms, Castration-Resistant/*metabolism
Receptors, Androgen/*biosynthesis
Transcription Factor RelA/*metabolism
Cell Line, Tumor ; Disease Progression ; Epigenesis, Genetic ; Gene Expression Regulation, Neoplastic ; Humans ; Interleukin-1alpha/pharmacology ; Male ; NF-kappa B/metabolism ; Prostatic Neoplasms/drug therapy ; Prostatic Neoplasms/genetics ; Prostatic Neoplasms/pathology ; Prostatic Neoplasms, Castration-Resistant/drug therapy ; Prostatic Neoplasms, Castration-Resistant/genetics ; Prostatic Neoplasms, Castration-Resistant/pathology ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; RNA, Neoplasm/genetics ; RNA, Neoplasm/metabolism ; RNA, Small Interfering/administration & dosage ; RNA, Small Interfering/genetics ; RNA-Binding Proteins/genetics ; RNA-Binding Proteins/metabolism ; Receptors, Androgen/genetics ; Transcription Factor RelA/genetics
Czasopismo naukowe
Tytuł :
Ginsenoside Rg3 attenuates endometriosis by inhibiting the viability of human ectopic endometrial stromal cells through the nuclear factor-kappaB signaling pathway.
Autorzy :
Huang R; Department of Laboratory, Women Health Center of Shanxi and Children's Hospital of Shanxi, Taiyuan, China.
Chen S; Department of Obstetrics and Gynaecology, Women Health Center of Shanxi and Children's Hospital of Shanxi, Taiyuan, China. Electronic address: .
Zhao M; Department of Obstetrics and Gynaecology, Women Health Center of Shanxi and Children's Hospital of Shanxi, Taiyuan, China.
Li Z; Department of Obstetrics and Gynaecology, Women Health Center of Shanxi and Children's Hospital of Shanxi, Taiyuan, China.
Zhu L; Department of Laboratory, Women Health Center of Shanxi and Children's Hospital of Shanxi, Taiyuan, China.
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Źródło :
Journal of gynecology obstetrics and human reproduction [J Gynecol Obstet Hum Reprod] 2020 Jan; Vol. 49 (1), pp. 101642. Date of Electronic Publication: 2019 Sep 26.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents, Phytogenic/*pharmacology
Cell Survival/*drug effects
Endometriosis/*pathology
Ginsenosides/*pharmacology
Stromal Cells/*drug effects
Transcription Factor RelA/*metabolism
Adult ; Caspase 3/analysis ; Cell Proliferation/drug effects ; Endometriosis/drug therapy ; Endometriosis/metabolism ; Female ; Humans ; In Vitro Techniques ; Ovarian Diseases/metabolism ; Ovarian Diseases/pathology ; RNA, Messenger/analysis ; Signal Transduction ; Stromal Cells/metabolism ; Transcription Factor RelA/analysis ; Transcription Factor RelA/genetics ; Tumor Necrosis Factor-alpha/pharmacology ; Vascular Endothelial Growth Factor A/analysis ; Vascular Endothelial Growth Factor A/metabolism
Czasopismo naukowe
Tytuł :
Targeting MIAT reduces apoptosis of cardiomyocytes after ischemia/reperfusion injury.
Autorzy :
Chen L; a Department of Internal medicine intensive care , the central hospital of Linyi , Yishui , Shandong , China.
Zhang D; a Department of Internal medicine intensive care , the central hospital of Linyi , Yishui , Shandong , China.
Yu L; a Department of Internal medicine intensive care , the central hospital of Linyi , Yishui , Shandong , China.
Dong H; b Department of Anesthesia , the affiliated hospital of Qingdao University , Qingdao Shandong , China.
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Źródło :
Bioengineered [Bioengineered] 2019 Dec; Vol. 10 (1), pp. 121-132.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis Regulatory Proteins/*genetics
Myocardial Reperfusion Injury/*genetics
Myocardium/*metabolism
Myocytes, Cardiac/*metabolism
RNA, Long Noncoding/*genetics
Transcription Factor RelA/*genetics
Tumor Suppressor Proteins/*genetics
Animals ; Apoptosis/genetics ; Apoptosis Regulatory Proteins/antagonists & inhibitors ; Apoptosis Regulatory Proteins/metabolism ; Caspase 3/genetics ; Caspase 3/metabolism ; Cell Hypoxia ; Cell Line ; Cell Survival ; Gene Expression Regulation ; Humans ; L-Lactate Dehydrogenase/metabolism ; Mice ; Myocardial Reperfusion Injury/metabolism ; Myocardial Reperfusion Injury/pathology ; Myocardial Reperfusion Injury/prevention & control ; Myocardium/pathology ; Myocytes, Cardiac/pathology ; Protein Transport ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; RNA, Long Noncoding/antagonists & inhibitors ; RNA, Long Noncoding/metabolism ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Rats ; Signal Transduction ; Transcription Factor RelA/antagonists & inhibitors ; Transcription Factor RelA/metabolism ; Tumor Suppressor Proteins/antagonists & inhibitors ; Tumor Suppressor Proteins/metabolism ; bcl-2-Associated X Protein/genetics ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
Quietness of circular RNA circ_0054633 alleviates the inflammation and proliferation in lipopolysaccharides-induced acute lung injury model through NF-κB signaling pathway.
Autorzy :
Yang CL; Department of Critical Care Medicine, Jiangxi Provincial People's Hospital Affiliated to Nanchang University, Nanchang, JiangXi, China.
Yang WK; Department of Cardiovascular Surgery, Central People's Hospital of Zhanjiang, Zhanjiang, GuangDong, China.
He ZH; Department of Critical Care Medicine, Jiangxi Provincial People's Hospital Affiliated to Nanchang University, Nanchang, JiangXi, China.
Guo JH; Department of Critical Care Medicine, Jiangxi Provincial People's Hospital Affiliated to Nanchang University, Nanchang, JiangXi, China.
Yang XG; Department of Critical Care Medicine, Jiangxi Provincial People's Hospital Affiliated to Nanchang University, Nanchang, JiangXi, China.
Li HB; Department of Orthopaedics, Jiangxi Provincial People's Hospital Affiliated to Nanchang University, Nanchang, JiangXi 330006, China. Electronic address: .
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Źródło :
Gene [Gene] 2021 Jan 15; Vol. 766, pp. 145153. Date of Electronic Publication: 2020 Sep 17.
Typ publikacji :
Journal Article
MeSH Terms :
Acute Lung Injury/*metabolism
Cell Proliferation/*physiology
Endothelial Cells/*metabolism
Inflammation/*metabolism
NF-kappa B/*metabolism
RNA, Circular/*metabolism
Acute Lung Injury/chemically induced ; Animals ; Apoptosis/drug effects ; Apoptosis/physiology ; Cell Proliferation/drug effects ; Cells, Cultured ; Inflammation/chemically induced ; Interleukin-17/metabolism ; Lipopolysaccharides/pharmacology ; Lung/drug effects ; Lung/metabolism ; Male ; Mice ; Rats ; Rats, Sprague-Dawley ; Transcription Factor RelA/metabolism ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł :
MiR-140-5p/TLR4 /NF-κB signaling pathway: Crucial role in inflammatory response in 16HBE cells induced by dust fall PM 2.5 .
Autorzy :
Chen X; Department of Laboratory Medicine, Affiliated Hospital of Southwest Medical University, Luzhou 646000, China.
Deng T; Southwest University of Science and Technology, Mianyang 621010, China.
Huo T; Southwest University of Science and Technology, Mianyang 621010, China.
Dong F; Southwest University of Science and Technology, Mianyang 621010, China. Electronic address: .
Deng J; Department of Laboratory Medicine, Affiliated Hospital of Southwest Medical University, Luzhou 646000, China; Sichuan Mianyang 404 Hospital, Mianyang 621000, China. Electronic address: .
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Źródło :
Ecotoxicology and environmental safety [Ecotoxicol Environ Saf] 2021 Jan 15; Vol. 208, pp. 111414. Date of Electronic Publication: 2020 Oct 17.
Typ publikacji :
Journal Article
MeSH Terms :
NF-kappa B/*metabolism
Particulate Matter/*toxicity
Cells, Cultured ; Dust ; Humans ; Inflammation ; Lipopolysaccharides ; MicroRNAs/genetics ; Signal Transduction/drug effects ; Toll-Like Receptor 4/metabolism ; Transcription Factor RelA
Czasopismo naukowe
Tytuł :
Rare earth element lanthanum protects against atherosclerosis induced by high-fat diet via down-regulating MAPK and NF-κB pathways.
Autorzy :
Li R; Department of Nutrition and Food Hygiene, School of Public Health, Peking University, Beijing, 100083, China. Electronic address: .
Zhou Y; Department of Nutrition and Food Hygiene, School of Public Health, Peking University, Beijing, 100083, China. Electronic address: .
Liu W; Department of Nutrition and Food Hygiene, School of Public Health, Peking University, Beijing, 100083, China. Electronic address: .
Li Y; Department of Nutrition and Food Hygiene, School of Public Health, Peking University, Beijing, 100083, China. Electronic address: .
Qin Y; Department of Nutrition and Food Hygiene, School of Public Health, Peking University, Beijing, 100083, China. Electronic address: .
Yu L; Department of Nutrition and Food Hygiene, School of Public Health, Peking University, Beijing, 100083, China. Electronic address: .
Chen Y; Department of Nutrition and Food Hygiene, School of Public Health, Peking University, Beijing, 100083, China. Electronic address: .
Xu Y; Department of Nutrition and Food Hygiene, School of Public Health, Peking University, Beijing, 100083, China; Toxicological Research and Risk Assessment for Food Safety, Beijing, 100083, China. Electronic address: .
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Źródło :
Ecotoxicology and environmental safety [Ecotoxicol Environ Saf] 2021 Jan 01; Vol. 207, pp. 111195. Date of Electronic Publication: 2020 Sep 03.
Typ publikacji :
Journal Article
MeSH Terms :
Diet, High-Fat*
Atherosclerosis/*prevention & control
Lanthanum/*metabolism
NF-kappa B/*metabolism
Protective Agents/*metabolism
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Down-Regulation ; Endothelium, Vascular/metabolism ; Inflammation Mediators/metabolism ; Intercellular Adhesion Molecule-1 ; Mice ; Signal Transduction ; Transcription Factor RelA ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe

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