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Wyszukujesz frazę ""Transforming Growth Factor beta"" wg kryterium: Temat


Tytuł:
MicroRNA-499-5p inhibits transforming growth factor-β1-induced Smad2 signaling pathway and suppresses fibroblast proliferation and collagen synthesis in rat by targeting TGFβ-R1.
Autorzy:
Zhao Q; Chinese PLA Medical Academy, Beijing, China.; Department of Cardiovascular Medicine, The Sixth Medical Center, Chinese PLA General Hospital, Beijing, China.
Yang W; Department of Cardiology, Beijing Jishuitan Hospital, The Fourth Clinical Medical College of Peking University, Beijing, China.
Li X; Chinese PLA Medical Academy, Beijing, China.
Yuan H; Department of Cardiovascular Medicine, The Sixth Medical Center, Chinese PLA General Hospital, Beijing, China.
Guo J; Chinese PLA Medical Academy, Beijing, China.
Wang Y; Department of Geriatric Cardiology, Chinese PLA General Hospital, Beijing, China.
Shan Z; Chinese PLA Medical Academy, Beijing, China. .; Department of Cardiovascular Medicine, The Sixth Medical Center, Chinese PLA General Hospital, Beijing, China. .
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Źródło:
Molecular biology reports [Mol Biol Rep] 2023 Dec; Vol. 50 (12), pp. 9757-9767. Date of Electronic Publication: 2023 Sep 07.
Typ publikacji:
Journal Article
MeSH Terms:
Atrial Fibrillation*/genetics
Atrial Fibrillation*/pathology
MicroRNAs*/genetics
MicroRNAs*/metabolism
Receptors, Transforming Growth Factor beta*/genetics
Receptors, Transforming Growth Factor beta*/metabolism
Animals ; Rats ; Cell Proliferation/genetics ; Collagen Type I/metabolism ; Fibroblasts/metabolism ; Fibrosis ; Luciferases/metabolism ; Rats, Sprague-Dawley ; RNA, Messenger/genetics ; Signal Transduction/genetics ; Transforming Growth Factor beta/metabolism ; Transforming Growth Factor beta1/metabolism ; Smad2 Protein/metabolism
Czasopismo naukowe
Tytuł:
Hydronidone ameliorates liver fibrosis by inhibiting activation of hepatic stellate cells via Smad7-mediated degradation of TGFβRI.
Autorzy:
Xu X; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Guo Y; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Luo X; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Shen Z; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Sun Z; Department of Gastroenterology, Huaian First People's Hospital, Nanjing Medical University, Huaian, China.
Shen B; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Zhou C; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Wang J; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Lu J; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Zhang Q; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Ye Y; Continent Pharmaceuticals Co., Ltd., Beijing, China.
Luo Y; Continent Pharmaceuticals Co., Ltd., Beijing, China.
Qu Y; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Cai X; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Dong H; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Lu L; Department of Gastroenterology, Shanghai General Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Pancreatic Diseases, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
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Źródło:
Liver international : official journal of the International Association for the Study of the Liver [Liver Int] 2023 Nov; Vol. 43 (11), pp. 2523-2537. Date of Electronic Publication: 2023 Aug 28.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Liver Cirrhosis*/drug therapy
Signal Transduction*
Transforming Growth Factor beta*/metabolism
Animals ; Mice ; Carbon Tetrachloride/toxicity ; Carbon Tetrachloride/metabolism ; Hepatic Stellate Cells/drug effects ; Hepatic Stellate Cells/metabolism ; Liver/pathology ; Receptor, Transforming Growth Factor-beta Type I ; Smad7 Protein/drug effects ; Smad7 Protein/metabolism
Czasopismo naukowe
Tytuł:
Transforming growth factor beta receptor 2 (Tgfbr2) deficiency in keratocytes results in corneal ectasia.
Autorzy:
Wang YC; Edith Crawley Vision Research Center, Department of Ophthalmology, College of Medicine, University of Cincinnati, Cincinnati, OH, USA; Department of Anesthesiology, School of Medicine, Washington University in St. Louis, MO, USA; School of Optometry, Indiana University, Bloomington, IN, USA. Electronic address: .
Zolnik OB; School of Optometry, Indiana University, Bloomington, IN, USA.
Yasoda S; Department of Ophthalmology, Wakayama Medical University, Wakayama, Japan.
Yeh LK; Department of Ophthalmology, Chang Gung Memorial Hospital, Linkou, Taiwan; College of Medicine, Chang Gung University, Taoyuan, Taiwan.
Yuan Y; Edith Crawley Vision Research Center, Department of Ophthalmology, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.
Kao W; Edith Crawley Vision Research Center, Department of Ophthalmology, College of Medicine, University of Cincinnati, Cincinnati, OH, USA.
Saika S; Department of Ophthalmology, Wakayama Medical University, Wakayama, Japan.
Liu CY; Edith Crawley Vision Research Center, Department of Ophthalmology, College of Medicine, University of Cincinnati, Cincinnati, OH, USA; School of Optometry, Indiana University, Bloomington, IN, USA.
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Źródło:
The ocular surface [Ocul Surf] 2023 Jul; Vol. 29, pp. 557-565. Date of Electronic Publication: 2023 Jun 30.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Cornea*/pathology
Corneal Diseases*/pathology
Receptor, Transforming Growth Factor-beta Type II*/genetics
Receptor, Transforming Growth Factor-beta Type II*/metabolism
Animals ; Humans ; Mice ; Collagen ; Corneal Stroma ; Dilatation, Pathologic/metabolism ; Dilatation, Pathologic/pathology ; Receptors, Transforming Growth Factor beta/genetics ; Receptors, Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Tytuł:
The Antinociceptive Effect of Sympathetic Block is Mediated by Transforming Growth Factor β in a Mouse Model of Radiculopathy.
Autorzy:
Lückemeyer DD; Pain Research Center, Department of Anesthesiology, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA.
Xie W; Pain Research Center, Department of Anesthesiology, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA.
Prudente AS; Pain Research Center, Department of Anesthesiology, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA.
Qualls KA; Pain Research Center, Department of Anesthesiology, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA.
Tonello R; Department of Molecular Pathobiology, Department of Neuroscience and Physiology, Neuroscience Institute, New York University, New York, NY, 10010, USA.
Strong JA; Pain Research Center, Department of Anesthesiology, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA.
Berta T; Pain Research Center, Department of Anesthesiology, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA.
Zhang JM; Pain Research Center, Department of Anesthesiology, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA. .
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Źródło:
Neuroscience bulletin [Neurosci Bull] 2023 Sep; Vol. 39 (9), pp. 1363-1374. Date of Electronic Publication: 2023 May 10.
Typ publikacji:
Journal Article
MeSH Terms:
Transforming Growth Factor beta*/metabolism
Transforming Growth Factor beta*/pharmacology
Radiculopathy*/drug therapy
Radiculopathy*/metabolism
Mice ; Animals ; Receptor, Transforming Growth Factor-beta Type I/metabolism ; Transforming Growth Factor beta1/metabolism ; Hyperalgesia/metabolism ; Pain/metabolism ; Analgesics/pharmacology ; Ganglia, Spinal/metabolism
Czasopismo naukowe
Tytuł:
Mutual regulation between glycosylation and transforming growth factor-β isoforms signaling pathway.
Autorzy:
Chen Z; Laboratory for Functional Glycomics, College of Life Sciences, Northwest University, Xi'an 710069, PR China.
Yu H; Laboratory for Functional Glycomics, College of Life Sciences, Northwest University, Xi'an 710069, PR China.
Chen X; Laboratory for Functional Glycomics, College of Life Sciences, Northwest University, Xi'an 710069, PR China.
Chen W; Laboratory for Functional Glycomics, College of Life Sciences, Northwest University, Xi'an 710069, PR China.
Song W; Laboratory for Functional Glycomics, College of Life Sciences, Northwest University, Xi'an 710069, PR China.
Li Z; Laboratory for Functional Glycomics, College of Life Sciences, Northwest University, Xi'an 710069, PR China. Electronic address: .
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Źródło:
International journal of biological macromolecules [Int J Biol Macromol] 2023 May 01; Vol. 236, pp. 123818. Date of Electronic Publication: 2023 Feb 27.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Receptors, Transforming Growth Factor beta*/metabolism
Signal Transduction*/physiology
Glycosylation ; Transforming Growth Factor beta/metabolism ; Protein Isoforms/metabolism ; Transforming Growth Factors/metabolism ; Transforming Growth Factor beta1/metabolism
Czasopismo naukowe
Tytuł:
Crosstalk between protein kinase C α and transforming growth factor β signaling mediated by Runx2 in intestinal epithelial cells.
Autorzy:
Li X; Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, Nebraska, USA.
Kaur N; Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, Nebraska, USA.
Albahrani M; Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, Nebraska, USA.
Karpf AR; Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, Nebraska, USA.
Black AR; Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, Nebraska, USA.
Black JD; Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, Nebraska, USA. Electronic address: .
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Źródło:
The Journal of biological chemistry [J Biol Chem] 2023 Apr; Vol. 299 (4), pp. 103017. Date of Electronic Publication: 2023 Feb 13.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Colorectal Neoplasms*/genetics
Core Binding Factor Alpha 1 Subunit*/genetics
Core Binding Factor Alpha 1 Subunit*/metabolism
Protein Kinase C-alpha*/genetics
Protein Kinase C-alpha*/metabolism
Receptor, Transforming Growth Factor-beta Type I*/genetics
Receptor, Transforming Growth Factor-beta Type I*/metabolism
Humans ; Epithelial Cells/metabolism ; Intestines ; RNA, Messenger/genetics ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Tytuł:
Synthesis and biological evaluation of N-(3-fluorobenzyl)-4-(1-(methyl-d 3 )-1H-indazol-5-yl)-5-(6-methylpyridin-2-yl)-1H-imidazol-2-amine as a novel, potent ALK5 receptor inhibitor.
Autorzy:
Kang BN; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea; Department of Chemistry, Sungkyunkwan University, Suwon 16419, Republic of Korea.
Kang HJ; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea.
Kim S; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea.
Lee J; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea.
Lee J; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea.
Jeong HJ; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea.
Jeon S; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea.
Shin Y; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea.
Yoon C; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea.
Han C; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea; Department of Medical and Bioscience, The Catholic University of Korea, Bucheon 14662, Republic of Korea.
Seo J; BiSiChem, 3F, Pangyo-ro, 255 beon-gil 74, Bundang-gu, Seongnam 13486, Republic of Korea. Electronic address: .
Yun J; Department of Chemistry, Sungkyunkwan University, Suwon 16419, Republic of Korea. Electronic address: .
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Źródło:
Bioorganic & medicinal chemistry letters [Bioorg Med Chem Lett] 2023 Apr 01; Vol. 85, pp. 129205. Date of Electronic Publication: 2023 Feb 28.
Typ publikacji:
Journal Article
MeSH Terms:
Protein Serine-Threonine Kinases*
Receptors, Transforming Growth Factor beta*/metabolism
Mice ; Animals ; Receptor, Transforming Growth Factor-beta Type I/metabolism ; Amines ; Indazoles/pharmacology ; Cytochrome P-450 Enzyme System/metabolism ; Structure-Activity Relationship ; Protein Kinase Inhibitors/pharmacology
Czasopismo naukowe
Tytuł:
Opposing USP19 splice variants in TGF-β signaling and TGF-β-induced epithelial-mesenchymal transition of breast cancer cells.
Autorzy:
Zhang J; Oncode Institute, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.; Department of Cell Chemical Biology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
van Dinther M; Oncode Institute, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.; Department of Cell Chemical Biology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
Thorikay M; Oncode Institute, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.; Department of Cell Chemical Biology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
Gourabi BM; Department of Anatomy and Embryology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
Kruithof BPT; Department of Cell Chemical Biology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.; HARTZ, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
Ten Dijke P; Oncode Institute, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands. P.ten_.; Department of Cell Chemical Biology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands. P.ten_.
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Źródło:
Cellular and molecular life sciences : CMLS [Cell Mol Life Sci] 2023 Jan 17; Vol. 80 (2), pp. 43. Date of Electronic Publication: 2023 Jan 17.
Typ publikacji:
Journal Article
MeSH Terms:
Transforming Growth Factor beta*/genetics
Transforming Growth Factor beta*/metabolism
Breast Neoplasms*/metabolism
Humans ; Female ; Epithelial-Mesenchymal Transition/genetics ; Receptors, Transforming Growth Factor beta/genetics ; Receptors, Transforming Growth Factor beta/metabolism ; Cell Membrane/metabolism ; Cell Movement/genetics ; Cell Line, Tumor ; Endopeptidases/metabolism
Czasopismo naukowe
Tytuł:
Anti-GARP Antibodies Inhibit Release of TGF-β by Regulatory T Cells via Different Modes of Action, but Do Not Influence Their Function In Vitro.
Autorzy:
Igney FH; Discovery Research, Cancer Immunology & Immune Modulation, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach an der Riss, Germany.
Ebenhoch R; Discovery Research, Structural Research, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach an der Riss, Germany.
Schiele F; Discovery Research, Biotherapeutics Discovery, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach an der Riss, Germany.
Nar H; Discovery Research, Structural Research, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach an der Riss, Germany.
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Źródło:
ImmunoHorizons [Immunohorizons] 2023 Mar 01; Vol. 7 (3), pp. 200-212.
Typ publikacji:
Journal Article
MeSH Terms:
T-Lymphocytes, Regulatory*
Transforming Growth Factor beta*/metabolism
Transforming Growth Factor beta*/pharmacology
Antibodies*/pharmacology
Humans ; Membrane Proteins/immunology ; Receptors, Transforming Growth Factor beta/metabolism ; Transforming Growth Factor beta1/metabolism
Czasopismo naukowe
Tytuł:
The TGF-β/Smad 2/3 signaling pathway is involved in Musashi2-induced invasion and metastasis of colorectal cancer.
Autorzy:
Yu ZL; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Liu J; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Ning ZK; Department of Day Ward, First Affiliated Hospital of Nanchang University, Nanchang, China.
Tian HK; Department of General Surgery, First Affiliated Hospital of Nanchang University, Nanchang, China.
Wu X; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Huang YF; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Wu ZC; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Zong Z; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Zhou TC; Department of Gastroenterological Surgery and Hernia Center, Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital of Sun Yat-Sen University, Guangzhou, Guangdong, China.
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Źródło:
Molecular carcinogenesis [Mol Carcinog] 2023 Feb; Vol. 62 (2), pp. 261-276. Date of Electronic Publication: 2022 Nov 08.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Colorectal Neoplasms*/pathology
Transforming Growth Factor beta*/metabolism
Humans ; Cell Line, Tumor ; Cell Movement ; Cell Proliferation ; Epithelial-Mesenchymal Transition/genetics ; Receptor, Transforming Growth Factor-beta Type II ; Signal Transduction ; Smad2 Protein/genetics ; Smad2 Protein/metabolism ; Smad2 Protein/pharmacology ; Transforming Growth Factor beta1/genetics ; Transforming Growth Factor beta1/metabolism
Czasopismo naukowe
Tytuł:
Shear-mediated ALK5 expression regulates endothelial activation.
Autorzy:
Pang KT; Department of Bioengineering, Imperial College London, United Kingdom.
Ghim M; Department of Bioengineering, Imperial College London, United Kingdom.
Sarathchandra P; Harefield Heart Science Centre, National Heart and Lung Institute, Imperial College London, United Kingdom.
Warboys CM; Comparative Biomedical Sciences, The Royal Veterinary College, United Kingdom.
Yacoub MH; Harefield Heart Science Centre, National Heart and Lung Institute, Imperial College London, United Kingdom.
Chester AH; Harefield Heart Science Centre, National Heart and Lung Institute, Imperial College London, United Kingdom.
Weinberg PD; Department of Bioengineering, Imperial College London, United Kingdom. Electronic address: .
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Źródło:
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2023 Jan 29; Vol. 642, pp. 90-96. Date of Electronic Publication: 2022 Dec 21.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Protein Serine-Threonine Kinases*/metabolism
Receptors, Transforming Growth Factor beta*/metabolism
Animals ; Humans ; Phosphorylation ; Receptor, Transforming Growth Factor-beta Type I/metabolism ; Signal Transduction ; Swine
Czasopismo naukowe
Tytuł:
A versatile Halo- and SNAP-tagged BMP/TGFβ receptor library for quantification of cell surface ligand binding.
Autorzy:
Jatzlau J; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany.
Burdzinski W; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany.; Berlin-Brandenburg School for Regenerative Therapies (BSRT), Berlin, Germany.
Trumpp M; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany.
Obendorf L; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany.
Roßmann K; Leibniz-Forschungsinstitut für Molekulare Pharmakologie, Berlin, Germany.
Ravn K; Department of Biochemistry, University of Cambridge, Cambridge, UK.
Hyvönen M; Department of Biochemistry, University of Cambridge, Cambridge, UK.
Bottanelli F; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany.
Broichhagen J; Leibniz-Forschungsinstitut für Molekulare Pharmakologie, Berlin, Germany.
Knaus P; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany. .; Berlin-Brandenburg School for Regenerative Therapies (BSRT), Berlin, Germany. .
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Źródło:
Communications biology [Commun Biol] 2023 Jan 12; Vol. 6 (1), pp. 34. Date of Electronic Publication: 2023 Jan 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Transforming Growth Factor beta*/metabolism
Bone Morphogenetic Proteins*/metabolism
Bone Morphogenetic Protein Receptors ; Ligands ; Receptors, Transforming Growth Factor beta
Czasopismo naukowe
Tytuł:
Depletion of Tgfbr2 in hepatocytes alleviates liver fibrosis and restores hepatic function in fibrotic mice.
Autorzy:
Liu SQ; Department of Gastroenterology, Changzheng Hospital, Naval Medical University, Shanghai, China.
Deng X; Department of Gastroenterology, Changzheng Hospital, Naval Medical University, Shanghai, China.
Zhu CP; Department of Gastroenterology, Changzheng Hospital, Naval Medical University, Shanghai, China.
Cui YL; Department of Gastroenterology, Changzheng Hospital, Naval Medical University, Shanghai, China.
Xie WF; Department of Gastroenterology, Changzheng Hospital, Naval Medical University, Shanghai, China.
Zhang X; Department of Gastroenterology, Changzheng Hospital, Naval Medical University, Shanghai, China.
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Źródło:
Journal of digestive diseases [J Dig Dis] 2023 Jan; Vol. 24 (1), pp. 39-50. Date of Electronic Publication: 2023 Mar 26.
Typ publikacji:
Journal Article
MeSH Terms:
Hepatocytes*/metabolism
Hepatocytes*/pathology
Liver Cirrhosis*/pathology
Receptor, Transforming Growth Factor-beta Type II*
Animals ; Mice ; Hepatic Stellate Cells/metabolism ; Hepatic Stellate Cells/pathology ; Liver/pathology ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Tytuł:
Mitochondrial dysfunction induces ALK5-SMAD2-mediated hypovascularization and arteriovenous malformations in mouse retinas.
Autorzy:
Zhang H; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA.
Li B; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA.
Huang Q; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA.
López-Giráldez F; Yale Center for Genomic Analysis, Department of Genetics, Yale University School of Medicine, New Haven, CT, USA.
Tanaka Y; Yale Stem Cell Center, Department of Genetics, Yale University School of Medicine, New Haven, CT, USA.; Maisonneuve-Rosemont Hospital Research Center (CRHMR), Department of Medicine, University of Montreal, Montreal, QC, Canada.
Lin Q; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA.
Mehta S; Yale Center for Genomic Analysis, Department of Genetics, Yale University School of Medicine, New Haven, CT, USA.
Wang G; Yale Center for Genomic Analysis, Department of Genetics, Yale University School of Medicine, New Haven, CT, USA.
Graham M; Department of Cell Biology, Yale University School of Medicine, New Haven, CT, USA.
Liu X; Department of Cell Biology, Yale University School of Medicine, New Haven, CT, USA.
Park IH; Yale Center for Genomic Analysis, Department of Genetics, Yale University School of Medicine, New Haven, CT, USA.
Eichmann A; Department of Internal Medicine (Cardiology) and Cellular and Molecular Physiology, Yale University School of Medicine, New Haven, CT, USA.
Min W; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA. .
Zhou JH; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA. .
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Źródło:
Nature communications [Nat Commun] 2022 Dec 10; Vol. 13 (1), pp. 7637. Date of Electronic Publication: 2022 Dec 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Arteriovenous Malformations*/genetics
Arteriovenous Malformations*/metabolism
Smad2 Protein*/genetics
Smad2 Protein*/metabolism
Receptor, Transforming Growth Factor-beta Type I*/genetics
Receptor, Transforming Growth Factor-beta Type I*/metabolism
Animals ; Mice ; Gene Expression Regulation ; Membrane Proteins/metabolism ; Mitochondria/metabolism ; Phosphorylation ; Signal Transduction ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Tytuł:
Genome-wide characterization of the TGF-β gene family and their expression in different tissues during tail regeneration in the Schlegel's Japanese gecko Gekko japonicus.
Autorzy:
Liu Q; College of Life Sciences, Nanjing Normal University, Nanjing 210023, China.
Zhao RM; College of Life Sciences, Nanjing Normal University, Nanjing 210023, China.
Wang DY; College of Life Sciences, Nanjing Normal University, Nanjing 210023, China.
Li P; College of Life Sciences, Nanjing Normal University, Nanjing 210023, China.
Qu YF; College of Life Sciences, Nanjing Normal University, Nanjing 210023, China. Electronic address: .
Ji X; Zhejiang Provincial Key Laboratory for Water Environment and Marine Biological Resources Protection, College of Life and Environmental Sciences, Wenzhou University, Wenzhou 325035, China. Electronic address: .
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Źródło:
International journal of biological macromolecules [Int J Biol Macromol] 2024 Jan; Vol. 255, pp. 128127. Date of Electronic Publication: 2023 Nov 19.
Typ publikacji:
Journal Article
MeSH Terms:
Transforming Growth Factor beta*/genetics
Transforming Growth Factor beta*/metabolism
Genome*
Animals ; Ligands
SCR Organism:
Gekko japonicus
Czasopismo naukowe
Tytuł:
MiR-302d inhibits TGFB-induced EMT and promotes MET in primary human RPE cells.
Autorzy:
Hu X; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
Binter M; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
Hufendiek K; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
Tode J; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
Framme C; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
Fuchs H; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
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Źródło:
PloS one [PLoS One] 2022 Nov 28; Vol. 17 (11), pp. e0278158. Date of Electronic Publication: 2022 Nov 28 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Transforming Growth Factor beta*
MicroRNAs*/genetics
Humans ; Epithelial-Mesenchymal Transition/genetics ; Receptor, Transforming Growth Factor-beta Type II ; Retinal Pigment Epithelium ; Vascular Endothelial Growth Factor A/genetics ; Fibrosis
Czasopismo naukowe
Tytuł:
Activin A Modulates Betaglycan Shedding via the ALK4-SMAD3-Dependent Pathway in Endometriotic Cells.
Autorzy:
Mwaura AN; Faculty of Medicine, Center of Gynecology and Obstetrics, Justus-Liebig-University, D-35392 Giessen, Germany.
Riaz MA; Faculty of Medicine, Center of Gynecology and Obstetrics, Justus-Liebig-University, D-35392 Giessen, Germany.
Maoga JB; Faculty of Medicine, Center of Gynecology and Obstetrics, Justus-Liebig-University, D-35392 Giessen, Germany.
Mecha E; Department of Biochemistry, University of Nairobi, Nairobi P.O. Box 30197-00100, Kenya.
Omwandho COA; Department of Biochemistry, University of Nairobi, Nairobi P.O. Box 30197-00100, Kenya.; Department of Health Sciences, Kirinyaga University, Kerugoya P.O. Box 143-10300, Kenya.
Scheiner-Bobis G; Institute for Veterinary Physiology and Biochemistry, School of Veterinary Medicine, Justus-Liebig-University, D-35392 Giessen, Germany.
Meinhold-Heerlein I; Faculty of Medicine, Center of Gynecology and Obstetrics, Justus-Liebig-University, D-35392 Giessen, Germany.
Konrad L; Faculty of Medicine, Center of Gynecology and Obstetrics, Justus-Liebig-University, D-35392 Giessen, Germany.
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Źródło:
Biomolecules [Biomolecules] 2022 Nov 24; Vol. 12 (12). Date of Electronic Publication: 2022 Nov 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Activins*/pharmacology
Activins*/metabolism
Endometriosis*/metabolism
Receptors, Transforming Growth Factor beta*/metabolism
Activin Receptors, Type I*
Female ; Humans ; Matrix Metalloproteinase 2/genetics ; Matrix Metalloproteinase 2/metabolism ; Matrix Metalloproteinase 3/genetics ; Matrix Metalloproteinase 3/metabolism ; Smad3 Protein/metabolism ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Tytuł:
Regulation of metabolic homeostasis by the TGF-β superfamily receptor ALK7.
Autorzy:
Ibáñez CF; Department of Neuroscience, Karolinska Institute, Stockholm, Sweden.; Peking-Tsinghua Center for Life Sciences, PKU-IDG/McGovern Institute for Brain Research, Peking University School of Life Sciences and Chinese Institute for Brain Research, Beijing, China.; Department of Physiology and Life Sciences Institute, National University of Singapore, Singapore.
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Źródło:
The FEBS journal [FEBS J] 2022 Oct; Vol. 289 (19), pp. 5776-5797. Date of Electronic Publication: 2021 Jul 11.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Insulin-Secreting Cells*/metabolism
Receptors, Transforming Growth Factor beta*/genetics
Activin Receptors, Type I/genetics ; Activin Receptors, Type I/metabolism ; Animals ; Homeostasis ; Humans ; Mammals/metabolism ; Mice ; Signal Transduction/genetics ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Tytuł:
Cyclin A1 (CCNA1) inhibits osteoporosis by suppressing transforming growth factor-beta (TGF-beta) pathway in osteoblasts.
Autorzy:
Du X; Department of Orthopedics, Beijing Geriatric Hospital, No.118 Hot Spring Road, Haidian District 100095, Beijing, China.
Zang C; Department of Orthopedics, Beijing Geriatric Hospital, No.118 Hot Spring Road, Haidian District 100095, Beijing, China.
Wang Q; Department of Orthopedics, Beijing Geriatric Hospital, No.118 Hot Spring Road, Haidian District 100095, Beijing, China. .
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Źródło:
BMC musculoskeletal disorders [BMC Musculoskelet Disord] 2024 Mar 07; Vol. 25 (1), pp. 206. Date of Electronic Publication: 2024 Mar 07.
Typ publikacji:
Journal Article
MeSH Terms:
Anthraquinones*
Osteoporosis*/chemically induced
Transforming Growth Factor beta*/metabolism
Animals ; Female ; Humans ; Mice ; Alkaline Phosphatase/metabolism ; Cell Differentiation ; Cyclin A1/metabolism ; Osteoblasts/metabolism ; Osteogenesis ; Transforming Growth Factors/adverse effects ; Transforming Growth Factors/metabolism
Czasopismo naukowe
Tytuł:
Analysis of immunoinfiltration and EndoMT based on TGF-β signaling pathway-related genes in acute myocardial infarction.
Autorzy:
Shen J; Cardiac Pacing and Electrophysiology Department, The First Affiliated Hospital of Xinjiang Medical University, Urumqi, China. .; Department of Cardiology, Renmin Hospital, Hubei University of Medicine, Shiyan, China. .
Liang J; Cardiac Pacing and Electrophysiology Department, The First Affiliated Hospital of Xinjiang Medical University, Urumqi, China.
Rejiepu M; Cardiac Pacing and Electrophysiology Department, The First Affiliated Hospital of Xinjiang Medical University, Urumqi, China.
Ma Z; Department of Cardiology, Renmin Hospital, Hubei University of Medicine, Shiyan, China.
Zhao J; Department of Cardiology, Renmin Hospital, Hubei University of Medicine, Shiyan, China.
Li J; Department of Cardiology, Renmin Hospital, Hubei University of Medicine, Shiyan, China.
Zhang L; Cardiac Pacing and Electrophysiology Department, The First Affiliated Hospital of Xinjiang Medical University, Urumqi, China. .
Yuan P; Department of Cardiology, Renmin Hospital, Hubei University of Medicine, Shiyan, China. .
Wang J; Department of Cardiology, Renmin Hospital, Hubei University of Medicine, Shiyan, China. .
Tang B; Cardiac Pacing and Electrophysiology Department, The First Affiliated Hospital of Xinjiang Medical University, Urumqi, China. .
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Źródło:
Scientific reports [Sci Rep] 2024 Mar 02; Vol. 14 (1), pp. 5183. Date of Electronic Publication: 2024 Mar 02.
Typ publikacji:
Journal Article
MeSH Terms:
Transforming Growth Factor beta*/metabolism
Myocardial Infarction*/pathology
Rats ; Animals ; Rats, Sprague-Dawley ; Endothelial Cells/metabolism ; Signal Transduction/genetics ; Fibrosis
Czasopismo naukowe

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