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Tytuł:
Molecular characterization and expression of TGFβRI and TGFβRII and its association with litter size in Tibetan sheep.
Autorzy:
Zhang J; College of Agriculture and Animal Husbandry, Key Laboratory of Livestock and Poultry Genetics and Breeding on the Qinghai-Tibet Plateau, Ministry of Agriculture and Rural Affairs, Plateau Livestock Genetic Resources Protection and Innovative Utilization Key Laboratory of Qinghai Province, Qinghai University, Xining, P. R. China.
Li M; College of Agriculture and Animal Husbandry, Key Laboratory of Livestock and Poultry Genetics and Breeding on the Qinghai-Tibet Plateau, Ministry of Agriculture and Rural Affairs, Plateau Livestock Genetic Resources Protection and Innovative Utilization Key Laboratory of Qinghai Province, Qinghai University, Xining, P. R. China.
Sun R; College of Agriculture and Animal Husbandry, Key Laboratory of Livestock and Poultry Genetics and Breeding on the Qinghai-Tibet Plateau, Ministry of Agriculture and Rural Affairs, Plateau Livestock Genetic Resources Protection and Innovative Utilization Key Laboratory of Qinghai Province, Qinghai University, Xining, P. R. China.
He N; College of Agriculture and Animal Husbandry, Key Laboratory of Livestock and Poultry Genetics and Breeding on the Qinghai-Tibet Plateau, Ministry of Agriculture and Rural Affairs, Plateau Livestock Genetic Resources Protection and Innovative Utilization Key Laboratory of Qinghai Province, Qinghai University, Xining, P. R. China.
Wen X; College of Agriculture and Animal Husbandry, Key Laboratory of Livestock and Poultry Genetics and Breeding on the Qinghai-Tibet Plateau, Ministry of Agriculture and Rural Affairs, Plateau Livestock Genetic Resources Protection and Innovative Utilization Key Laboratory of Qinghai Province, Qinghai University, Xining, P. R. China.
Han X; Technology Extension Service of Animal Husbandry of Qinghai, Xining, China.
Luo Z; Technology Extension Service of Animal Husbandry of Qinghai, Xining, China.
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Źródło:
Veterinary medicine and science [Vet Med Sci] 2023 Mar; Vol. 9 (2), pp. 934-944. Date of Electronic Publication: 2023 Jan 07.
Typ publikacji:
Journal Article
MeSH Terms:
Receptors, Transforming Growth Factor beta*/genetics
Receptors, Transforming Growth Factor beta*/metabolism
Transforming Growth Factor beta*/genetics
Transforming Growth Factor beta*/metabolism
Animals ; Female ; Sheep/genetics ; Cattle ; Pregnancy ; Litter Size/genetics ; Tibet ; Receptor, Transforming Growth Factor-beta Type I/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Opposing USP19 splice variants in TGF-β signaling and TGF-β-induced epithelial-mesenchymal transition of breast cancer cells.
Autorzy:
Zhang J; Oncode Institute, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.; Department of Cell Chemical Biology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
van Dinther M; Oncode Institute, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.; Department of Cell Chemical Biology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
Thorikay M; Oncode Institute, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.; Department of Cell Chemical Biology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
Gourabi BM; Department of Anatomy and Embryology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
Kruithof BPT; Department of Cell Chemical Biology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.; HARTZ, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
Ten Dijke P; Oncode Institute, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands. P.ten_.; Department of Cell Chemical Biology, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands. P.ten_.
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Źródło:
Cellular and molecular life sciences : CMLS [Cell Mol Life Sci] 2023 Jan 17; Vol. 80 (2), pp. 43. Date of Electronic Publication: 2023 Jan 17.
Typ publikacji:
Journal Article
MeSH Terms:
Transforming Growth Factor beta*/genetics
Transforming Growth Factor beta*/metabolism
Breast Neoplasms*/metabolism
Humans ; Female ; Epithelial-Mesenchymal Transition/genetics ; Receptors, Transforming Growth Factor beta/genetics ; Receptors, Transforming Growth Factor beta/metabolism ; Cell Membrane/metabolism ; Cell Movement/genetics ; Cell Line, Tumor ; Endopeptidases/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Anti-GARP Antibodies Inhibit Release of TGF-β by Regulatory T Cells via Different Modes of Action, but Do Not Influence Their Function In Vitro.
Autorzy:
Igney FH; Discovery Research, Cancer Immunology & Immune Modulation, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach an der Riss, Germany.
Ebenhoch R; Discovery Research, Structural Research, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach an der Riss, Germany.
Schiele F; Discovery Research, Biotherapeutics Discovery, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach an der Riss, Germany.
Nar H; Discovery Research, Structural Research, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach an der Riss, Germany.
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Źródło:
ImmunoHorizons [Immunohorizons] 2023 Mar 01; Vol. 7 (3), pp. 200-212.
Typ publikacji:
Journal Article
MeSH Terms:
Transforming Growth Factor beta*/metabolism
T-Lymphocytes, Regulatory*
Humans ; Transforming Growth Factor beta1/metabolism ; Membrane Proteins/metabolism ; Receptors, Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
The TGF-β/Smad 2/3 signaling pathway is involved in Musashi2-induced invasion and metastasis of colorectal cancer.
Autorzy:
Yu ZL; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Liu J; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Ning ZK; Department of Day Ward, First Affiliated Hospital of Nanchang University, Nanchang, China.
Tian HK; Department of General Surgery, First Affiliated Hospital of Nanchang University, Nanchang, China.
Wu X; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Huang YF; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Wu ZC; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Zong Z; Department of Gastrointestinal Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, China.
Zhou TC; Department of Gastroenterological Surgery and Hernia Center, Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital of Sun Yat-Sen University, Guangzhou, Guangdong, China.
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Źródło:
Molecular carcinogenesis [Mol Carcinog] 2023 Feb; Vol. 62 (2), pp. 261-276. Date of Electronic Publication: 2022 Nov 08.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Colorectal Neoplasms*/pathology
Transforming Growth Factor beta*/metabolism
Humans ; Cell Line, Tumor ; Cell Movement ; Cell Proliferation ; Epithelial-Mesenchymal Transition/genetics ; Receptor, Transforming Growth Factor-beta Type II ; Signal Transduction ; Smad2 Protein/genetics ; Smad2 Protein/metabolism ; Smad2 Protein/pharmacology ; Transforming Growth Factor beta1/genetics ; Transforming Growth Factor beta1/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Shear-mediated ALK5 expression regulates endothelial activation.
Autorzy:
Pang KT; Department of Bioengineering, Imperial College London, United Kingdom.
Ghim M; Department of Bioengineering, Imperial College London, United Kingdom.
Sarathchandra P; Harefield Heart Science Centre, National Heart and Lung Institute, Imperial College London, United Kingdom.
Warboys CM; Comparative Biomedical Sciences, The Royal Veterinary College, United Kingdom.
Yacoub MH; Harefield Heart Science Centre, National Heart and Lung Institute, Imperial College London, United Kingdom.
Chester AH; Harefield Heart Science Centre, National Heart and Lung Institute, Imperial College London, United Kingdom.
Weinberg PD; Department of Bioengineering, Imperial College London, United Kingdom. Electronic address: .
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Źródło:
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2023 Jan 29; Vol. 642, pp. 90-96. Date of Electronic Publication: 2022 Dec 21.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Protein Serine-Threonine Kinases*/metabolism
Receptors, Transforming Growth Factor beta*/metabolism
Animals ; Humans ; Phosphorylation ; Receptor, Transforming Growth Factor-beta Type I/metabolism ; Signal Transduction ; Swine
Czasopismo naukowe
Szczegóły
Tytuł:
A versatile Halo- and SNAP-tagged BMP/TGFβ receptor library for quantification of cell surface ligand binding.
Autorzy:
Jatzlau J; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany.
Burdzinski W; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany.; Berlin-Brandenburg School for Regenerative Therapies (BSRT), Berlin, Germany.
Trumpp M; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany.
Obendorf L; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany.
Roßmann K; Leibniz-Forschungsinstitut für Molekulare Pharmakologie, Berlin, Germany.
Ravn K; Department of Biochemistry, University of Cambridge, Cambridge, UK.
Hyvönen M; Department of Biochemistry, University of Cambridge, Cambridge, UK.
Bottanelli F; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany.
Broichhagen J; Leibniz-Forschungsinstitut für Molekulare Pharmakologie, Berlin, Germany.
Knaus P; Institute of Chemistry and Biochemistry - Biochemistry, Berlin, Germany. .; Berlin-Brandenburg School for Regenerative Therapies (BSRT), Berlin, Germany. .
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Źródło:
Communications biology [Commun Biol] 2023 Jan 12; Vol. 6 (1), pp. 34. Date of Electronic Publication: 2023 Jan 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Transforming Growth Factor beta*/metabolism
Bone Morphogenetic Proteins*/metabolism
Bone Morphogenetic Protein Receptors ; Ligands ; Receptors, Transforming Growth Factor beta
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Mitochondrial dysfunction induces ALK5-SMAD2-mediated hypovascularization and arteriovenous malformations in mouse retinas.
Autorzy:
Zhang H; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA.
Li B; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA.
Huang Q; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA.
López-Giráldez F; Yale Center for Genomic Analysis, Department of Genetics, Yale University School of Medicine, New Haven, CT, USA.
Tanaka Y; Yale Stem Cell Center, Department of Genetics, Yale University School of Medicine, New Haven, CT, USA.; Maisonneuve-Rosemont Hospital Research Center (CRHMR), Department of Medicine, University of Montreal, Montreal, QC, Canada.
Lin Q; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA.
Mehta S; Yale Center for Genomic Analysis, Department of Genetics, Yale University School of Medicine, New Haven, CT, USA.
Wang G; Yale Center for Genomic Analysis, Department of Genetics, Yale University School of Medicine, New Haven, CT, USA.
Graham M; Department of Cell Biology, Yale University School of Medicine, New Haven, CT, USA.
Liu X; Department of Cell Biology, Yale University School of Medicine, New Haven, CT, USA.
Park IH; Yale Center for Genomic Analysis, Department of Genetics, Yale University School of Medicine, New Haven, CT, USA.
Eichmann A; Department of Internal Medicine (Cardiology) and Cellular and Molecular Physiology, Yale University School of Medicine, New Haven, CT, USA.
Min W; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA. .
Zhou JH; Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, New Haven, CT, USA. .
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Źródło:
Nature communications [Nat Commun] 2022 Dec 10; Vol. 13 (1), pp. 7637. Date of Electronic Publication: 2022 Dec 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Arteriovenous Malformations*/genetics
Arteriovenous Malformations*/metabolism
Smad2 Protein*/genetics
Smad2 Protein*/metabolism
Receptor, Transforming Growth Factor-beta Type I*/genetics
Receptor, Transforming Growth Factor-beta Type I*/metabolism
Animals ; Mice ; Gene Expression Regulation ; Membrane Proteins/metabolism ; Mitochondria/metabolism ; Phosphorylation ; Signal Transduction ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
MiR-302d inhibits TGFB-induced EMT and promotes MET in primary human RPE cells.
Autorzy:
Hu X; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
Binter M; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
Hufendiek K; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
Tode J; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
Framme C; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
Fuchs H; Institute of Ophthalmology, University Eye Hospital, Hannover Medical School, Hannover, Germany.
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Źródło:
PloS one [PLoS One] 2022 Nov 28; Vol. 17 (11), pp. e0278158. Date of Electronic Publication: 2022 Nov 28 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Transforming Growth Factor beta*
MicroRNAs*/genetics
Humans ; Epithelial-Mesenchymal Transition/genetics ; Receptor, Transforming Growth Factor-beta Type II ; Retinal Pigment Epithelium ; Vascular Endothelial Growth Factor A/genetics ; Fibrosis
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Activin A Modulates Betaglycan Shedding via the ALK4-SMAD3-Dependent Pathway in Endometriotic Cells.
Autorzy:
Mwaura AN; Faculty of Medicine, Center of Gynecology and Obstetrics, Justus-Liebig-University, D-35392 Giessen, Germany.
Riaz MA; Faculty of Medicine, Center of Gynecology and Obstetrics, Justus-Liebig-University, D-35392 Giessen, Germany.
Maoga JB; Faculty of Medicine, Center of Gynecology and Obstetrics, Justus-Liebig-University, D-35392 Giessen, Germany.
Mecha E; Department of Biochemistry, University of Nairobi, Nairobi P.O. Box 30197-00100, Kenya.
Omwandho COA; Department of Biochemistry, University of Nairobi, Nairobi P.O. Box 30197-00100, Kenya.; Department of Health Sciences, Kirinyaga University, Kerugoya P.O. Box 143-10300, Kenya.
Scheiner-Bobis G; Institute for Veterinary Physiology and Biochemistry, School of Veterinary Medicine, Justus-Liebig-University, D-35392 Giessen, Germany.
Meinhold-Heerlein I; Faculty of Medicine, Center of Gynecology and Obstetrics, Justus-Liebig-University, D-35392 Giessen, Germany.
Konrad L; Faculty of Medicine, Center of Gynecology and Obstetrics, Justus-Liebig-University, D-35392 Giessen, Germany.
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Źródło:
Biomolecules [Biomolecules] 2022 Nov 24; Vol. 12 (12). Date of Electronic Publication: 2022 Nov 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Activins*/pharmacology
Activins*/metabolism
Endometriosis*/metabolism
Receptors, Transforming Growth Factor beta*/metabolism
Activin Receptors, Type I*
Female ; Humans ; Matrix Metalloproteinase 2/genetics ; Matrix Metalloproteinase 2/metabolism ; Matrix Metalloproteinase 3/genetics ; Matrix Metalloproteinase 3/metabolism ; Smad3 Protein/metabolism ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Regulation of metabolic homeostasis by the TGF-β superfamily receptor ALK7.
Autorzy:
Ibáñez CF; Department of Neuroscience, Karolinska Institute, Stockholm, Sweden.; Peking-Tsinghua Center for Life Sciences, PKU-IDG/McGovern Institute for Brain Research, Peking University School of Life Sciences and Chinese Institute for Brain Research, Beijing, China.; Department of Physiology and Life Sciences Institute, National University of Singapore, Singapore.
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Źródło:
The FEBS journal [FEBS J] 2022 Oct; Vol. 289 (19), pp. 5776-5797. Date of Electronic Publication: 2021 Jul 11.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Insulin-Secreting Cells*/metabolism
Receptors, Transforming Growth Factor beta*/genetics
Activin Receptors, Type I/genetics ; Activin Receptors, Type I/metabolism ; Animals ; Homeostasis ; Humans ; Mammals/metabolism ; Mice ; Signal Transduction/genetics ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
EPDR1 is a noncanonical effector of insulin-mediated angiogenesis regulated by an endothelial-specific TGF-β receptor complex.
Autorzy:
Ahmed T; Department of Chemistry & Biochemistry, University of Arizona, Tucson, Arizona, USA.
Flores PC; Department of Chemistry & Biochemistry, University of Arizona, Tucson, Arizona, USA.
Pan CC; Department of Pharmacology and Cancer Biology, Duke University, Durham, North Carolina, USA.
Ortiz HR; Department of Pharmacology, University of Arizona, Tucson, Arizona, USA.
Lee YS; Department of Pharmacology, University of Arizona, Tucson, Arizona, USA.
Langlais PR; Department of Medicine, University of Arizona, Tucson, Arizona, USA.
Mythreye K; Department of Pathology, University of Alabama at Birmingham, Birmingham, Alabama, USA. Electronic address: .
Lee NY; Department of Chemistry & Biochemistry, University of Arizona, Tucson, Arizona, USA; Department of Pharmacology, University of Arizona, Tucson, Arizona, USA; Comprehensive Cancer Center, University of Arizona, Tucson, Arizona, USA. Electronic address: .
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Źródło:
The Journal of biological chemistry [J Biol Chem] 2022 Sep; Vol. 298 (9), pp. 102297. Date of Electronic Publication: 2022 Jul 21.
Typ publikacji:
Journal Article
MeSH Terms:
Endoglin*/genetics
Endoglin*/metabolism
Growth Differentiation Factor 2*/genetics
Insulin*/metabolism
Neovascularization, Pathologic*/genetics
Neovascularization, Pathologic*/metabolism
Nerve Tissue Proteins*/genetics
Nerve Tissue Proteins*/metabolism
Receptors, Transforming Growth Factor beta*/metabolism
Animals ; Humans ; Mice ; Activin Receptors, Type II/metabolism ; Chlorocebus aethiops ; COS Cells ; Phosphatidylinositol 3-Kinases ; Proteomics ; Receptor, Insulin/genetics ; Receptor, Insulin/metabolism ; Smad1 Protein/metabolism ; Smad5 Protein/metabolism ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Ochratoxin a induces hepatic fibrosis through TGF-β receptor I/Smad2/3 signaling pathway.
Autorzy:
Chae SA; Department of Biotechnology, College of Life Sciences and Biotechnology, Korea University, Seoul, Republic of Korea.
Pyo MC; Department of Biotechnology, College of Life Sciences and Biotechnology, Korea University, Seoul, Republic of Korea.
Yoo HJ; Department of Biotechnology, College of Life Sciences and Biotechnology, Korea University, Seoul, Republic of Korea.
Lee KW; Department of Biotechnology, College of Life Sciences and Biotechnology, Korea University, Seoul, Republic of Korea.; Department of Food Bioscience and Technology, College of Life Sciences and Biotechnology, Korea University, Seoul, Republic of Korea.
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Źródło:
Environmental toxicology [Environ Toxicol] 2022 Aug; Vol. 37 (8), pp. 2084-2095. Date of Electronic Publication: 2022 May 11.
Typ publikacji:
Journal Article
MeSH Terms:
Liver Cirrhosis*/chemically induced
Liver Cirrhosis*/genetics
Liver Cirrhosis*/metabolism
Transforming Growth Factor beta*/metabolism
Epithelial-Mesenchymal Transition ; Fibrosis ; Humans ; Ochratoxins ; RNA, Messenger ; Receptors, Transforming Growth Factor beta/metabolism ; Signal Transduction ; Smad2 Protein/genetics ; Smad2 Protein/metabolism ; Transforming Growth Factor beta1/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
The function of CD146 in human annulus fibrosus cells and mechanism of the regulation by TGF-β.
Autorzy:
Du J; AO Research Institute Davos, Davos, Switzerland.; Department of Orthopedics, University Medical Center Utrecht, Utrecht, the Netherlands.
Guo W; AO Research Institute Davos, Davos, Switzerland.; Department of Spine Surgery, The First Affiliated Hospital of Sun Yat-sen University, Guangzhou, P. R. China.
Häckel S; Department of Orthopaedic Surgery and Traumatology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.
Hoppe S; Department of Orthopaedic Surgery and Traumatology, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland.
Garcia JP; Department of Orthopedics, University Medical Center Utrecht, Utrecht, the Netherlands.
Alini M; AO Research Institute Davos, Davos, Switzerland.
Tryfonidou MA; Faculty of Veterinary Medicine, Utrecht University, Utrecht, the Netherlands.
Creemers LB; Department of Orthopedics, University Medical Center Utrecht, Utrecht, the Netherlands.
Grad S; AO Research Institute Davos, Davos, Switzerland.
Li Z; AO Research Institute Davos, Davos, Switzerland.
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Źródło:
Journal of orthopaedic research : official publication of the Orthopaedic Research Society [J Orthop Res] 2022 Jul; Vol. 40 (7), pp. 1661-1671. Date of Electronic Publication: 2021 Oct 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Annulus Fibrosus*/metabolism
CD146 Antigen*/metabolism
Transforming Growth Factor beta*/metabolism
Extracellular Signal-Regulated MAP Kinases/metabolism ; Humans ; Proto-Oncogene Proteins c-akt ; Receptor, Transforming Growth Factor-beta Type I ; Smad2 Protein ; Smad4 Protein
Czasopismo naukowe
Szczegóły
Tytuł:
Natural plant extracts mediated expression regulation of TGF-β receptors and SMAD genes in human cancer cell lines.
Autorzy:
Jabeen A; Institute of Biochemistry and Biotechnology (UIBB), Pir Mehr Ali Shah, Arid Agriculture University, Rawalpindi, Pakistan.; Institute of Biomedical and Genetic Engineering (IBGE), Islamabad, Pakistan.
Mansoor Q; Institute of Biomedical and Genetic Engineering (IBGE), Islamabad, Pakistan. .
Zainab T; Institute of Biochemistry and Biotechnology (UIBB), Pir Mehr Ali Shah, Arid Agriculture University, Rawalpindi, Pakistan.; National Center for Industrial Biotechnology (NCIB), Pir Mehr Ali Shah, Arid Agriculture University, Rawalpindi, Pakistan.
Farooqi AA; Institute of Biomedical and Genetic Engineering (IBGE), Islamabad, Pakistan.
Qayyum M; Department of Zoology, Pir Mehr Ali Shah, Arid Agriculture University, Rawalpindi, Pakistan.
Mansha A; Institute of Biochemistry and Biotechnology (UIBB), Pir Mehr Ali Shah, Arid Agriculture University, Rawalpindi, Pakistan.
Shakeel A; Institute of Biochemistry and Biotechnology (UIBB), Pir Mehr Ali Shah, Arid Agriculture University, Rawalpindi, Pakistan.
Ahmad MS; Institute of Biochemistry and Biotechnology (UIBB), Pir Mehr Ali Shah, Arid Agriculture University, Rawalpindi, Pakistan. .; Institute of Biomedical and Genetic Engineering (IBGE), Islamabad, Pakistan. .
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Źródło:
Molecular biology reports [Mol Biol Rep] 2022 Jun; Vol. 49 (6), pp. 4171-4178. Date of Electronic Publication: 2022 Mar 17.
Typ publikacji:
Journal Article
MeSH Terms:
Neoplasms*
Receptors, Transforming Growth Factor beta*/genetics
Antioxidants/pharmacology ; Flavonoids/pharmacology ; Humans ; MCF-7 Cells ; Plant Extracts/chemistry ; Plant Extracts/pharmacology ; Transforming Growth Factor beta/genetics ; Transforming Growth Factor beta/metabolism ; Ubiquitin-Protein Ligases
Czasopismo naukowe
Szczegóły
Tytuł:
A juxtamembrane basolateral targeting motif regulates signaling through a TGF-β pathway receptor in Drosophila.
Autorzy:
Peterson AJ; Department of Genetics, Cell Biology & Development and the Developmental Biology Center, University of Minnesota, Minneapolis, Minnesota, United States of America.
Murphy SJ; Center for Individualized Medicine, Mayo Clinic College of Medicine, Rochester, Minnesota, United States of America.
Mundt MG; Department of Genetics, Cell Biology & Development and the Developmental Biology Center, University of Minnesota, Minneapolis, Minnesota, United States of America.
Shimell M; Department of Genetics, Cell Biology & Development and the Developmental Biology Center, University of Minnesota, Minneapolis, Minnesota, United States of America.
Leof EB; Thoracic Diseases Research Unit, Department of Pulmonary and Critical Care Medicine, Mayo Clinic College of Medicine, Rochester, Minnesota, United States of America.
O'Connor MB; Department of Genetics, Cell Biology & Development and the Developmental Biology Center, University of Minnesota, Minneapolis, Minnesota, United States of America.
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Źródło:
PLoS biology [PLoS Biol] 2022 May 20; Vol. 20 (5), pp. e3001660. Date of Electronic Publication: 2022 May 20 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Drosophila*/metabolism
Drosophila Proteins*/genetics
Drosophila Proteins*/metabolism
Transforming Growth Factor beta*/genetics
Transforming Growth Factor beta*/metabolism
Animals ; Cell Membrane/metabolism ; Dogs ; Female ; Ligands ; Madin Darby Canine Kidney Cells ; Protein Serine-Threonine Kinases ; Receptors, Cell Surface/metabolism ; Receptors, Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Inhibition of Integrin αvβ6 Activation of TGF-β Attenuates Tendinopathy.
Autorzy:
Wang X; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
Liu S; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
Yu T; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
An S; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
Deng R; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
Tan X; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
Crane J; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
Zhang W; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
Pan D; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
Wan M; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
Carr A; Nuffield Department of Orthopaedics, Rheumatology and Musculoskeletal Sciences, University of Oxford, Oxford, OX3 7LD, UK.
Cao X; Department of Orthopaedic Surgery, The Johns Hopkins University School of Medicine, Baltimore, MD, 21205, USA.
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Źródło:
Advanced science (Weinheim, Baden-Wurttemberg, Germany) [Adv Sci (Weinh)] 2022 Apr; Vol. 9 (11), pp. e2104469. Date of Electronic Publication: 2022 Feb 15.
Typ publikacji:
Journal Article
MeSH Terms:
Tendinopathy*
Transforming Growth Factor beta*/metabolism
Animals ; Antigens, Neoplasm/genetics ; Antigens, Neoplasm/metabolism ; Integrins/genetics ; Integrins/metabolism ; Mice ; Receptor, Transforming Growth Factor-beta Type II
Czasopismo naukowe
Szczegóły
Tytuł:
Data-based stochastic modeling reveals sources of activity bursts in single-cell TGF-β signaling.
Autorzy:
Kolbe N; Institute of Geometry and Practical Mathematics, RWTH Aachen University, Aachen, Germany.
Hexemer L; Institute of Molecular Biology (IMB), Mainz, Germany.; Department of Systems Biology, Institute for Biomedical Genetics (IBMG), University of Stuttgart, Stuttgart, Germany.
Bammert LM; Institute of Mathematics, Johannes Gutenberg-University, Mainz, Germany.
Loewer A; Systems Biology of the Stress Response, Department of Biology, Technical University of Darmstadt, Darmstadt, Germany.
Lukáčová-Medvid'ová M; Institute of Mathematics, Johannes Gutenberg-University, Mainz, Germany.
Legewie S; Department of Systems Biology, Institute for Biomedical Genetics (IBMG), University of Stuttgart, Stuttgart, Germany.; Stuttgart Research Center for Systems Biology (SRCSB), University of Stuttgart, Stuttgart, Germany.
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Źródło:
PLoS computational biology [PLoS Comput Biol] 2022 Jun 27; Vol. 18 (6), pp. e1010266. Date of Electronic Publication: 2022 Jun 27 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Receptors, Transforming Growth Factor beta*
Signal Transduction*/physiology
Cell Nucleus/metabolism ; Endosomes/metabolism ; Smad Proteins/metabolism ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
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Tytuł:
SMAD proteins: Mediators of diverse outcomes during infection.
Autorzy:
Lai LYS; School of Life and Environmental Sciences, The University of Sydney, Sydney, New South Wales, Australia.
Gracie NP; School of Life and Environmental Sciences, The University of Sydney, Sydney, New South Wales, Australia.
Gowripalan A; Department of Microbiology and Immunology, Albert Einstein College of Medicine, Yeshiva University, Bronx, United States.
Howell LM; School of Life and Environmental Sciences, The University of Sydney, Sydney, New South Wales, Australia.
Newsome TP; School of Life and Environmental Sciences, The University of Sydney, Sydney, New South Wales, Australia. Electronic address: .
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Źródło:
European journal of cell biology [Eur J Cell Biol] 2022 Apr; Vol. 101 (2), pp. 151204. Date of Electronic Publication: 2022 Jan 31.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Smad Proteins*/metabolism
Transforming Growth Factor beta*/metabolism
Fibrosis ; Humans ; Receptors, Transforming Growth Factor beta/metabolism ; Signal Transduction/physiology
Czasopismo naukowe
Szczegóły
Tytuł:
Chordacentrum mineralization is delayed in zebrafish betaglycan-null mutants.
Autorzy:
Molina-Villa T; Department of Cellular and Developmental Biology, Institute of Cellular Physiology, UNAM, México City, Mexico.
Ramírez-Vidal L; Department of Cellular and Developmental Biology, Institute of Cellular Physiology, UNAM, México City, Mexico.
Mendoza V; Department of Cellular and Developmental Biology, Institute of Cellular Physiology, UNAM, México City, Mexico.
Escalante-Alcalde D; Division of Neurosciences, Department of Neural Development and Physiology, Institute of Cellular Physiology, UNAM, México City, Mexico.
López-Casillas F; Department of Cellular and Developmental Biology, Institute of Cellular Physiology, UNAM, México City, Mexico.
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Źródło:
Developmental dynamics : an official publication of the American Association of Anatomists [Dev Dyn] 2022 Jan; Vol. 251 (1), pp. 213-225. Date of Electronic Publication: 2021 Jul 17.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Receptors, Transforming Growth Factor beta*/genetics
Receptors, Transforming Growth Factor beta*/metabolism
Zebrafish*/genetics
Zebrafish*/metabolism
Animals ; Mice ; Proteoglycans/genetics ; Signal Transduction/genetics ; Transforming Growth Factor beta/genetics ; Transforming Growth Factor beta/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Deficiency in Retinal TGFβ Signaling Aggravates Neurodegeneration by Modulating Pro-Apoptotic and MAP Kinase Pathways.
Autorzy:
Bielmeier CB; Institute of Anatomy and Cell Biology, Julius-Maximilians-University Wuerzburg, 97070 Wuerzburg, Germany.
Schmitt SI; Institute of Human Anatomy and Embryology, University of Regensburg, 93053 Regensburg, Germany.
Kleefeldt N; Department of Ophthalmology, University Hospital Wuerzburg, 97070 Wuerzburg, Germany.
Boneva SK; Eye Center, Medical Center, Faculty of Medicine, University of Freiburg, 79106 Freiburg, Germany.
Schlecht A; Institute of Anatomy and Cell Biology, Julius-Maximilians-University Wuerzburg, 97070 Wuerzburg, Germany.
Vallon M; Institute of Anatomy and Cell Biology, Julius-Maximilians-University Wuerzburg, 97070 Wuerzburg, Germany.
Tamm ER; Institute of Human Anatomy and Embryology, University of Regensburg, 93053 Regensburg, Germany.
Hillenkamp J; Department of Ophthalmology, University Hospital Wuerzburg, 97070 Wuerzburg, Germany.
Ergün S; Institute of Anatomy and Cell Biology, Julius-Maximilians-University Wuerzburg, 97070 Wuerzburg, Germany.
Neueder A; Department of Neurology, University of Ulm, 89081 Ulm, Germany.
Braunger BM; Institute of Anatomy and Cell Biology, Julius-Maximilians-University Wuerzburg, 97070 Wuerzburg, Germany.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Feb 27; Vol. 23 (5). Date of Electronic Publication: 2022 Feb 27.
Typ publikacji:
Journal Article
MeSH Terms:
Retinal Degeneration*/genetics
Retinal Degeneration*/metabolism
Transforming Growth Factor beta*/metabolism
Animals ; Disease Models, Animal ; Ependymoglial Cells/metabolism ; Mice ; Mitogen-Activated Protein Kinases/metabolism ; Receptor, Transforming Growth Factor-beta Type II/genetics ; Receptor, Transforming Growth Factor-beta Type II/metabolism ; Retina/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
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