Temat: "Tumor Suppressor Protein p53" - OpacWWW

Skocz do pozycji: 1.

Tytuł:: Characterizing the role of Phlda3 in the development of acute toxicity and malignant transformation of hematopoietic cells induced by total-body irradiation in mice.
Autorzy:: Hasapis S; Department of Radiation Oncology, Duke University School of Medicine, Duke University Medical Center, Box 3813, Durham, NC, 27708, USA.
Caraballo I; Department of Radiation Oncology, Duke University School of Medicine, Duke University Medical Center, Box 3813, Durham, NC, 27708, USA.
Sears TJ; Department of Radiation Oncology, Stanford Cancer Institute, Stanford University, 875 Blake Wilbur Drive, Stanford, CA, 94305-5847, USA.
Brock KD; Department of Radiation Oncology, Duke University School of Medicine, Duke University Medical Center, Box 3813, Durham, NC, 27708, USA.
Cart JB; Department of Radiation Oncology, Duke University School of Medicine, Duke University Medical Center, Box 3813, Durham, NC, 27708, USA.
Moding EJ; Department of Radiation Oncology, Stanford Cancer Institute, Stanford University, 875 Blake Wilbur Drive, Stanford, CA, 94305-5847, USA. .; Stanford Cancer Institute, Stanford University, Stanford, CA, USA. .
Lee CL; Department of Radiation Oncology, Duke University School of Medicine, Duke University Medical Center, Box 3813, Durham, NC, 27708, USA. .; Department of Pathology, Duke University School of Medicine, Durham, NC, 27710, USA. .; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2023 Aug 09; Vol. 13 (1), pp. 12916. Date of Electronic Publication: 2023 Aug 09.
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:: Lymphoma*/genetics
Lymphoma*/radiotherapy
Lymphoma*/metabolism
Thymus Neoplasms*/metabolism
Nuclear Proteins*/genetics
Animals ; Humans ; Mice ; Cell Line ; Cell Transformation, Neoplastic/genetics ; Tumor Suppressor Protein p53/metabolism

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Tytuł:: Human nucleolar protein SURF6/RRP14 participates in early steps of pre-rRNA processing.
Autorzy:: Moraleva A; Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry RAS, Moscow, Russian Federation.
Deryabin A; Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry RAS, Moscow, Russian Federation.
Kordyukova M; Federal Center of Brain Research and Neurotechnologies of the Federal Medical Biological Agency, Moscow, Russian Federation.
Polzikov M; Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry RAS, Moscow, Russian Federation.
Shishova K; Federal Center of Brain Research and Neurotechnologies of the Federal Medical Biological Agency, Moscow, Russian Federation.
Dobrochaeva K; Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry RAS, Moscow, Russian Federation.
Rubtsov Y; Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry RAS, Moscow, Russian Federation.
Rubtsova M; Department of Chemistry, Lomonosov Moscow State University, Moscow, Russian Federation.
Dontsova O; Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry RAS, Moscow, Russian Federation.; Department of Chemistry, Lomonosov Moscow State University, Moscow, Russian Federation.; SkolTech, Moscow, Russian Federation.
Zatsepina O; Shemyakin-Ovchinnikov Institute of Bioorganic Chemistry RAS, Moscow, Russian Federation.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2023 Jul 14; Vol. 18 (7), pp. e0285833. Date of Electronic Publication: 2023 Jul 14 (Print Publication: 2023).
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Nuclear Proteins*/genetics
Nuclear Proteins*/metabolism
RNA Precursors*/genetics
RNA Precursors*/metabolism
Humans ; HeLa Cells ; Mammals/genetics ; Ribosomal Proteins/genetics ; RNA Processing, Post-Transcriptional ; RNA, Ribosomal/metabolism ; Saccharomyces cerevisiae/genetics ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/metabolism

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Skocz do pozycji: 3.

Tytuł:: Nuclear Proteomics of Induced Leukemia Cell Differentiation.
Autorzy:: Novikova S; Laboratory of Systems Biology, Institute of Biomedical Chemistry, 119121 Moscow, Russia.
Tolstova T; Laboratory of Systems Biology, Institute of Biomedical Chemistry, 119121 Moscow, Russia.
Kurbatov L; Laboratory of Systems Biology, Institute of Biomedical Chemistry, 119121 Moscow, Russia.
Farafonova T; Laboratory of Systems Biology, Institute of Biomedical Chemistry, 119121 Moscow, Russia.
Tikhonova O; Laboratory of Systems Biology, Institute of Biomedical Chemistry, 119121 Moscow, Russia.
Soloveva N; Laboratory of Systems Biology, Institute of Biomedical Chemistry, 119121 Moscow, Russia.
Rusanov A; Laboratory of Systems Biology, Institute of Biomedical Chemistry, 119121 Moscow, Russia.
Archakov A; Laboratory of Systems Biology, Institute of Biomedical Chemistry, 119121 Moscow, Russia.
Zgoda V; Laboratory of Systems Biology, Institute of Biomedical Chemistry, 119121 Moscow, Russia.; Pokaż więcej
Źródło:: Cells [Cells] 2022 Oct 14; Vol. 11 (20). Date of Electronic Publication: 2022 Oct 14.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Leukemia, Promyelocytic, Acute*/genetics
Leukemia, Promyelocytic, Acute*/metabolism
Leukemia, Promyelocytic, Acute*/pathology
Nuclear Proteins*/metabolism
Proteome*/metabolism
Granulocytes*/metabolism
Granulocytes*/pathology
Cell Nucleus*/metabolism
Humans ; Caspase 3/metabolism ; Cell Cycle Proteins/metabolism ; Cell Differentiation ; Chromosomal Proteins, Non-Histone/metabolism ; Core Binding Factor Alpha 2 Subunit/metabolism ; Intracellular Signaling Peptides and Proteins/metabolism ; Proteomics ; Tretinoin/pharmacology ; Tretinoin/metabolism ; Tumor Suppressor Protein p53/genetics ; HL-60 Cells

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Skocz do pozycji: 4.

Tytuł:: TP53INP2 Contributes to TGF-β2-Induced Autophagy during the Epithelial-Mesenchymal Transition in Posterior Capsular Opacification Development.
Autorzy:: Cui Y; Eye Center, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou 310009, China.; Zhejiang Provincial Key Lab of Ophthalmology, Hangzhou 310009, China.
Yang H; Eye Center, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou 310009, China.; Zhejiang Provincial Key Lab of Ophthalmology, Hangzhou 310009, China.
Shi S; Eye Center, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou 310009, China.; Zhejiang Provincial Key Lab of Ophthalmology, Hangzhou 310009, China.
Ping X; Eye Center, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou 310009, China.; Zhejiang Provincial Key Lab of Ophthalmology, Hangzhou 310009, China.
Zheng S; GKT School of Medical Education, King's College London, London WC2R 2LS, UK.
Tang X; Eye Center, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou 310009, China.; Zhejiang Provincial Key Lab of Ophthalmology, Hangzhou 310009, China.
Yu X; Eye Center, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou 310009, China.; Zhejiang Provincial Key Lab of Ophthalmology, Hangzhou 310009, China.
Shentu X; Eye Center, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou 310009, China.; Zhejiang Provincial Key Lab of Ophthalmology, Hangzhou 310009, China.; Pokaż więcej
Źródło:: Cells [Cells] 2022 Aug 02; Vol. 11 (15). Date of Electronic Publication: 2022 Aug 02.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Capsule Opacification*/metabolism
Nuclear Proteins/*metabolism
Transforming Growth Factor beta2/*metabolism
Autophagy ; Epithelial-Mesenchymal Transition/genetics ; Humans ; Tumor Suppressor Protein p53

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Skocz do pozycji: 5.

Tytuł:: KLF4, Slug and EMT in Head and Neck Squamous Cell Carcinoma.
Autorzy:: Ingruber J; Department of Otorhinolaryngology and Head and Neck Surgery, Medical University of Innsbruck, University Hospital of Tyrol, A-6020 Innsbruck, Austria.
Savic D; Laboratory for Experimental and Translational Research on Radiation Oncology (EXTRO-Lab), Department of Therapeutic Radiology and Oncology, Medical University of Innsbruck, A-6020 Innsbruck, Austria.; Tyrolean Cancer Research Institute, A-6020 Innsbruck, Austria.
Steinbichler TB; Department of Otorhinolaryngology and Head and Neck Surgery, Medical University of Innsbruck, University Hospital of Tyrol, A-6020 Innsbruck, Austria.
Sprung S; Department of Pathology, Medical University of Innsbruck, A-6020 Innsbruck, Austria.
Fleischer F; Department of Otorhinolaryngology and Head and Neck Surgery, Medical University of Innsbruck, University Hospital of Tyrol, A-6020 Innsbruck, Austria.; Department of Restorative and Operative Dentistry, Medical University of Innsbruck, A-6020 Innsbruck, Austria.
Glueckert R; Department of Otorhinolaryngology and Head and Neck Surgery, Medical University of Innsbruck, University Hospital of Tyrol, A-6020 Innsbruck, Austria.
Schweigl G; Department of Otorhinolaryngology and Head and Neck Surgery, Medical University of Innsbruck, University Hospital of Tyrol, A-6020 Innsbruck, Austria.
Skvortsova II; Laboratory for Experimental and Translational Research on Radiation Oncology (EXTRO-Lab), Department of Therapeutic Radiology and Oncology, Medical University of Innsbruck, A-6020 Innsbruck, Austria.; Tyrolean Cancer Research Institute, A-6020 Innsbruck, Austria.
Riechelmann H; Department of Otorhinolaryngology and Head and Neck Surgery, Medical University of Innsbruck, University Hospital of Tyrol, A-6020 Innsbruck, Austria.
Dudás J; Department of Otorhinolaryngology and Head and Neck Surgery, Medical University of Innsbruck, University Hospital of Tyrol, A-6020 Innsbruck, Austria.; Pokaż więcej
Źródło:: Cells [Cells] 2021 Mar 03; Vol. 10 (3). Date of Electronic Publication: 2021 Mar 03.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Head and Neck Neoplasms/*genetics
Kruppel-Like Transcription Factors/*genetics
Nuclear Proteins/*genetics
Snail Family Transcription Factors/*genetics
Squamous Cell Carcinoma of Head and Neck/*genetics
Tumor Suppressor Protein p53/*genetics
Twist-Related Protein 1/*genetics
Aged ; Aged, 80 and over ; Case-Control Studies ; Cell Line, Tumor ; Cell Movement ; Cell Proliferation ; Epithelial-Mesenchymal Transition/genetics ; Female ; Gene Expression Regulation, Neoplastic ; Head and Neck Neoplasms/metabolism ; Head and Neck Neoplasms/mortality ; Head and Neck Neoplasms/pathology ; Humans ; Kruppel-Like Factor 4 ; Kruppel-Like Transcription Factors/metabolism ; Male ; Middle Aged ; Neoplasm Staging ; Nuclear Proteins/metabolism ; Signal Transduction ; Snail Family Transcription Factors/metabolism ; Squamous Cell Carcinoma of Head and Neck/metabolism ; Squamous Cell Carcinoma of Head and Neck/mortality ; Squamous Cell Carcinoma of Head and Neck/pathology ; Survival Analysis ; Transforming Growth Factor beta1/genetics ; Transforming Growth Factor beta1/metabolism ; Tumor Suppressor Protein p53/metabolism ; Twist-Related Protein 1/metabolism ; Vimentin/genetics ; Vimentin/metabolism ; Zinc Finger E-box-Binding Homeobox 1/genetics ; Zinc Finger E-box-Binding Homeobox 1/metabolism

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Skocz do pozycji: 6.

Tytuł:: Beyond HAT Adaptor: TRRAP Liaisons with Sp1-Mediated Transcription.
Autorzy:: Yin BK; Leibniz Institute on Aging-Fritz Lipmann Institute (FLI), 07745 Jena, Germany.
Wang ZQ; Leibniz Institute on Aging-Fritz Lipmann Institute (FLI), 07745 Jena, Germany.; Faculty of Biological Sciences, Friedrich Schiller University Jena, 07743 Jena, Germany.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2021 Nov 18; Vol. 22 (22). Date of Electronic Publication: 2021 Nov 18.
Typ publikacji:: Journal Article; Review
MeSH Terms:: Transcription, Genetic*
Adaptor Proteins, Signal Transducing/*genetics
Histone Acetyltransferases/*genetics
Neurodegenerative Diseases/*genetics
Neurogenesis/*genetics
Nuclear Proteins/*genetics
Sp1 Transcription Factor/*genetics
Adaptor Proteins, Signal Transducing/metabolism ; Animals ; E2F1 Transcription Factor/genetics ; E2F1 Transcription Factor/metabolism ; Gene Expression Regulation ; Histone Acetyltransferases/metabolism ; Humans ; Multienzyme Complexes/genetics ; Multienzyme Complexes/metabolism ; Neurodegenerative Diseases/metabolism ; Neurodegenerative Diseases/pathology ; Nuclear Proteins/metabolism ; Protein Binding ; Proto-Oncogene Proteins c-myc/genetics ; Proto-Oncogene Proteins c-myc/metabolism ; Signal Transduction ; Sp1 Transcription Factor/metabolism ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/metabolism

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Skocz do pozycji: 7.

Tytuł:: p53-PHLDA3-Akt Network: The Key Regulators of Neuroendocrine Tumorigenesis.
Autorzy:: Chen Y; Laboratory of Fundamental Oncology, National Cancer Center Research Institute, Tsukiji 5-1-1, Chuo-ku, Tokyo 104-0045, Japan.
Ohki R; Laboratory of Fundamental Oncology, National Cancer Center Research Institute, Tsukiji 5-1-1, Chuo-ku, Tokyo 104-0045, Japan.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2020 Jun 08; Vol. 21 (11). Date of Electronic Publication: 2020 Jun 08.
Typ publikacji:: Journal Article; Review
MeSH Terms:: Disease Susceptibility*
Cell Transformation, Neoplastic/*metabolism
Neuroendocrine Tumors/*etiology
Neuroendocrine Tumors/*metabolism
Nuclear Proteins/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Tumor Suppressor Protein p53/*metabolism
Animals ; Biomarkers, Tumor ; Cell Transformation, Neoplastic/genetics ; Disease Models, Animal ; Humans ; Mutation ; Neuroendocrine Tumors/pathology ; Nuclear Proteins/genetics ; Organ Specificity/genetics ; Signal Transduction ; Tumor Suppressor Protein p53/genetics

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Skocz do pozycji: 8.

Tytuł:: p53 mediates PEDF‑induced autophagy in human umbilical vein endothelial cells through sestrin2 signaling.
Autorzy:: Chen T; Orthopedics Surgery Department, Beijing Shijitan Hospital, Capital Medical University, Beijing 100038, P.R. China.
Li T; Orthopedics Surgery Department, Beijing Shijitan Hospital, Capital Medical University, Beijing 100038, P.R. China.
Wang J; Orthopedics Surgery Department, Beijing Shijitan Hospital, Capital Medical University, Beijing 100038, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2019 Aug; Vol. 20 (2), pp. 1443-1450. Date of Electronic Publication: 2019 Jun 03.
Typ publikacji:: Journal Article
MeSH Terms:: Autophagy/*genetics
Eye Proteins/*genetics
Human Umbilical Vein Endothelial Cells/*drug effects
Nerve Growth Factors/*genetics
Nuclear Proteins/*genetics
Serpins/*genetics
Signal Transduction/*genetics
Tumor Suppressor Protein p53/*genetics
Adaptor Proteins, Signal Transducing/genetics ; Adaptor Proteins, Signal Transducing/metabolism ; Autophagy/drug effects ; Cell Cycle Proteins/genetics ; Cell Cycle Proteins/metabolism ; Eye Proteins/metabolism ; Eye Proteins/pharmacology ; Gene Expression Regulation ; Human Umbilical Vein Endothelial Cells/cytology ; Human Umbilical Vein Endothelial Cells/metabolism ; Humans ; Microtubule-Associated Proteins/genetics ; Microtubule-Associated Proteins/metabolism ; Nerve Growth Factors/metabolism ; Nerve Growth Factors/pharmacology ; Nuclear Proteins/antagonists & inhibitors ; Nuclear Proteins/metabolism ; Phosphorylation/drug effects ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Ribosomal Protein S6 Kinases, 70-kDa/genetics ; Ribosomal Protein S6 Kinases, 70-kDa/metabolism ; Sequestosome-1 Protein/genetics ; Sequestosome-1 Protein/metabolism ; Serpins/metabolism ; Serpins/pharmacology ; TOR Serine-Threonine Kinases/genetics ; TOR Serine-Threonine Kinases/metabolism ; Tumor Suppressor Protein p53/antagonists & inhibitors ; Tumor Suppressor Protein p53/metabolism

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Skocz do pozycji: 9.

Tytuł:: A p53/TIAF1/WWOX triad exerts cancer suppression but may cause brain protein aggregation due to p53/WWOX functional antagonism.
Autorzy:: Chou PY; Institute of Molecular Medicine, National Cheng Kung University, College of Medicine, Tainan, Taiwan, 70101, Republic of China.
Lin SR; Institute of Molecular Medicine, National Cheng Kung University, College of Medicine, Tainan, Taiwan, 70101, Republic of China.
Lee MH; Institute of Molecular Medicine, National Cheng Kung University, College of Medicine, Tainan, Taiwan, 70101, Republic of China.
Schultz L; Laboratory of Molecular Immunology, Guthrie Research Institute, Sayre, PA, 18840, USA.
Sze CI; Department of Cell Biology and Anatomy, National Cheng Kung University, College of Medicine, Tainan, Taiwan, 70101, Republic of China.
Chang NS; Institute of Molecular Medicine, National Cheng Kung University, College of Medicine, Tainan, Taiwan, 70101, Republic of China. .; Laboratory of Molecular Immunology, Guthrie Research Institute, Sayre, PA, 18840, USA. .; Department of Neurochemistry, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, NY, 10314, USA. .; Graduate Institute of Biomedical Sciences, College of Medicine, China Medical University, Taichung, 40402, Taiwan, Republic of China. .; Pokaż więcej
Źródło:: Cell communication and signaling : CCS [Cell Commun Signal] 2019 Jul 17; Vol. 17 (1), pp. 76. Date of Electronic Publication: 2019 Jul 17.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:: Apoptosis Regulatory Proteins/*metabolism
Breast Neoplasms/*therapy
Lung Neoplasms/*therapy
Nuclear Proteins/*metabolism
Tumor Suppressor Protein p53/*metabolism
Tumor Suppressor Proteins/*metabolism
WW Domain-Containing Oxidoreductase/*metabolism
Animals ; Apoptosis/drug effects ; Apoptosis Regulatory Proteins/antagonists & inhibitors ; Breast Neoplasms/metabolism ; Breast Neoplasms/pathology ; Cell Movement/drug effects ; Female ; Humans ; Lung Neoplasms/metabolism ; Lung Neoplasms/pathology ; Mice ; Mice, Knockout ; Mice, Nude ; Nuclear Proteins/antagonists & inhibitors ; Protein Aggregates/drug effects ; Signal Transduction/drug effects ; Tumor Cells, Cultured ; Tumor Suppressor Protein p53/antagonists & inhibitors ; Tumor Suppressor Protein p53/deficiency ; Tumor Suppressor Proteins/antagonists & inhibitors ; Tumor Suppressor Proteins/deficiency ; WW Domain-Containing Oxidoreductase/antagonists & inhibitors ; WW Domain-Containing Oxidoreductase/deficiency

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Skocz do pozycji: 10.

Tytuł:: The novel circSLC6A6/miR-1265/C2CD4A axis promotes colorectal cancer growth by suppressing p53 signaling pathway.
Autorzy:: Rong Z; Department of General Surgery, Shanghai General Hospital, Shanghai Jiaotong University School of Medicine, 100 Hai Ning Road, Hongkou District, Shanghai, 200080, China.
Luo Z; Department of General Surgery, Shanghai General Hospital, Shanghai Jiaotong University School of Medicine, 100 Hai Ning Road, Hongkou District, Shanghai, 200080, China.
Fu Z; Department of General Surgery, Shanghai General Hospital, Shanghai Jiaotong University School of Medicine, 100 Hai Ning Road, Hongkou District, Shanghai, 200080, China.
Zhang P; Department of General Surgery, Shanghai General Hospital, Shanghai Jiaotong University School of Medicine, 100 Hai Ning Road, Hongkou District, Shanghai, 200080, China.
Li T; Department of General Surgery, Shanghai General Hospital, Shanghai Jiaotong University School of Medicine, 100 Hai Ning Road, Hongkou District, Shanghai, 200080, China.
Zhang J; Department of General Surgery, Shanghai General Hospital, Shanghai Jiaotong University School of Medicine, 100 Hai Ning Road, Hongkou District, Shanghai, 200080, China.
Zhu Z; Department of General Surgery, Shanghai General Hospital, Shanghai Jiaotong University School of Medicine, 100 Hai Ning Road, Hongkou District, Shanghai, 200080, China.
Yu Z; Department of General Surgery, Shanghai General Hospital, Shanghai Jiaotong University School of Medicine, 100 Hai Ning Road, Hongkou District, Shanghai, 200080, China.
Li Q; Department of Medical Oncology, Shuguang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai, China.; Academy of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Qiu Z; Department of General Surgery, Shanghai General Hospital, Shanghai Jiaotong University School of Medicine, 100 Hai Ning Road, Hongkou District, Shanghai, 200080, China.
Huang C; Department of General Surgery, Shanghai General Hospital, Shanghai Jiaotong University School of Medicine, 100 Hai Ning Road, Hongkou District, Shanghai, 200080, China. .; Pokaż więcej
Źródło:: Journal of experimental & clinical cancer research : CR [J Exp Clin Cancer Res] 2021 Oct 16; Vol. 40 (1), pp. 324. Date of Electronic Publication: 2021 Oct 16.
Typ publikacji:: Journal Article
MeSH Terms:: Colorectal Neoplasms/*metabolism
Membrane Glycoproteins/*metabolism
Membrane Transport Proteins/*metabolism
MicroRNAs/*metabolism
Nuclear Proteins/*metabolism
Transcription Factors/*metabolism
Tumor Suppressor Protein p53/*metabolism
Animals ; Colorectal Neoplasms/genetics ; Colorectal Neoplasms/pathology ; HEK293 Cells ; Heterografts ; Humans ; Male ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; MicroRNAs/genetics ; Nuclear Proteins/genetics ; Signal Transduction ; Transcription Factors/genetics ; Up-Regulation

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Skocz do pozycji: 11.

Tytuł:: The Ins and Outs of HOPS/TMUB1 in biology and pathology.
Autorzy:: Della-Fazia MA; Department of Experimental Medicine, University of Perugia, Italy.
Castelli M; Department of Experimental Medicine, University of Perugia, Italy.
Piobbico D; Department of Experimental Medicine, University of Perugia, Italy.
Pieroni S; Department of Experimental Medicine, University of Perugia, Italy.
Servillo G; Department of Experimental Medicine, University of Perugia, Italy.; Pokaż więcej
Źródło:: The FEBS journal [FEBS J] 2021 May; Vol. 288 (9), pp. 2773-2783. Date of Electronic Publication: 2020 Sep 13.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:: Intracellular Signaling Peptides and Proteins/*genetics
Membrane Proteins/*genetics
Nuclear Proteins/*genetics
Stathmin/*genetics
Tumor Suppressor Protein p53/*genetics
Animals ; Cell Nucleus/genetics ; Gene Expression Regulation/genetics ; Humans ; Inflammation/genetics ; Inflammation/pathology ; Liver/metabolism ; Liver/pathology ; Liver Regeneration/genetics ; Mice ; Nucleophosmin

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Tytuł:: The actin nucleation factors JMY and WHAMM enable a rapid Arp2/3 complex-mediated intrinsic pathway of apoptosis.
Autorzy:: King VL; Department of Molecular and Cell Biology, Institute for Systems Genomics, University of Connecticut, Storrs, Connecticut, United States of America.
Leclair NK; Department of Molecular and Cell Biology, Institute for Systems Genomics, University of Connecticut, Storrs, Connecticut, United States of America.
Coulter AM; Department of Molecular and Cell Biology, Institute for Systems Genomics, University of Connecticut, Storrs, Connecticut, United States of America.
Campellone KG; Department of Molecular and Cell Biology, Institute for Systems Genomics, University of Connecticut, Storrs, Connecticut, United States of America.; Pokaż więcej
Źródło:: PLoS genetics [PLoS Genet] 2021 Apr 19; Vol. 17 (4), pp. e1009512. Date of Electronic Publication: 2021 Apr 19 (Print Publication: 2021).
Typ publikacji:: Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:: Membrane Proteins/*genetics
Microtubule-Associated Proteins/*genetics
Nuclear Proteins/*genetics
Trans-Activators/*genetics
Tumor Suppressor Protein p53/*genetics
Wiskott-Aldrich Syndrome/*genetics
rho GTP-Binding Proteins/*genetics
Actin Cytoskeleton/genetics ; Actin-Related Protein 2/genetics ; Actin-Related Protein 2-3 Complex/genetics ; Actin-Related Protein 3/genetics ; Apoptosis/genetics ; Cytochromes c/genetics ; DNA Damage/genetics ; Humans ; Mitochondria/genetics ; Mitochondria/metabolism ; RNA, Small Interfering/genetics ; Wiskott-Aldrich Syndrome Protein/genetics

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Skocz do pozycji: 13.

Tytuł:: Identification of putative drugs for gastric adenocarcinoma utilizing differentially expressed genes and connectivity map.
Autorzy:: Chen ZX; Department of Medical Oncology, The Second Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Zou XP; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Yan HQ; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Zhang R; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Pang JS; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Qin XG; Department of Gastrointestinal Surgery, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
He RQ; Department of Medical Oncology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Ma J; Department of Medical Oncology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Feng ZB; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Chen G; Department of Pathology, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.
Gan TQ; Department of Medical Oncology, The Second Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi Zhuang Autonomous Region 530021, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2019 Feb; Vol. 19 (2), pp. 1004-1015. Date of Electronic Publication: 2018 Dec 13.
Typ publikacji:: Journal Article
MeSH Terms:: Gene Expression Regulation, Neoplastic*
Adenocarcinoma/*genetics
Antineoplastic Agents/*therapeutic use
Cell Cycle Proteins/*genetics
Cyclin B1/*genetics
Nuclear Proteins/*genetics
Stomach Neoplasms/*genetics
Tumor Suppressor Protein p53/*genetics
Adenocarcinoma/drug therapy ; Adenocarcinoma/metabolism ; Adenocarcinoma/pathology ; Antineoplastic Agents/classification ; Cell Cycle Proteins/metabolism ; Cell Line, Tumor ; Computational Biology/methods ; Cyclin B1/metabolism ; Databases, Genetic ; Gene Expression Profiling ; Gene Ontology ; Gene Regulatory Networks ; Humans ; Molecular Sequence Annotation ; Nuclear Proteins/metabolism ; Pharmacogenetics/methods ; Protein Interaction Mapping ; Signal Transduction ; Stomach Neoplasms/drug therapy ; Stomach Neoplasms/metabolism ; Stomach Neoplasms/pathology ; Tumor Suppressor Protein p53/metabolism

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Skocz do pozycji: 14.

Tytuł:: Oncogene c-fos and mutant R175H p53 regulate expression of Nucleophosmin implicating cancer manifestation.
Autorzy:: Senapati P; Transcription and Disease Laboratory, Molecular Biology and Genetics Unit, Jawaharlal Nehru Centre for Advanced Scientific Research (JNCASR), Bangalore, India.
Dey S; Transcription and Disease Laboratory, Molecular Biology and Genetics Unit, Jawaharlal Nehru Centre for Advanced Scientific Research (JNCASR), Bangalore, India.
Sudarshan D; Transcription and Disease Laboratory, Molecular Biology and Genetics Unit, Jawaharlal Nehru Centre for Advanced Scientific Research (JNCASR), Bangalore, India.
Das S; Transcription and Disease Laboratory, Molecular Biology and Genetics Unit, Jawaharlal Nehru Centre for Advanced Scientific Research (JNCASR), Bangalore, India.
Kumar M; Transcription and Disease Laboratory, Molecular Biology and Genetics Unit, Jawaharlal Nehru Centre for Advanced Scientific Research (JNCASR), Bangalore, India.
Kaypee S; Transcription and Disease Laboratory, Molecular Biology and Genetics Unit, Jawaharlal Nehru Centre for Advanced Scientific Research (JNCASR), Bangalore, India.
Mohiyuddin A; Sri Devaraj Urs Academy of Higher Education and Research (SDUAHER), Kolar, India.
Kodaganur GS; Sri Devaraj Urs Academy of Higher Education and Research (SDUAHER), Kolar, India.; Bangalore Institute of Oncology (BIO), Bangalore, India.
Kundu TK; Transcription and Disease Laboratory, Molecular Biology and Genetics Unit, Jawaharlal Nehru Centre for Advanced Scientific Research (JNCASR), Bangalore, India.; Pokaż więcej
Źródło:: The FEBS journal [FEBS J] 2018 Sep; Vol. 285 (18), pp. 3503-3524. Date of Electronic Publication: 2018 Aug 24.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Gene Expression Regulation, Neoplastic*
Genes, fos*
Mutation*
Carcinoma, Squamous Cell/*pathology
Mouth Neoplasms/*pathology
Nuclear Proteins/*genetics
Tumor Suppressor Protein p53/*metabolism
Carcinoma, Squamous Cell/genetics ; Carcinoma, Squamous Cell/metabolism ; Cell Proliferation ; Humans ; Mouth Neoplasms/genetics ; Mouth Neoplasms/metabolism ; Nuclear Proteins/metabolism ; Nucleophosmin ; Prognosis ; Promoter Regions, Genetic ; Tumor Cells, Cultured ; Tumor Suppressor Protein p53/genetics

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Skocz do pozycji: 15.

Tytuł:: The ribosome biogenesis protein Esf1 is essential for pharyngeal cartilage formation in zebrafish.
Autorzy:: Chen JY; Key Laboratory of Marine Drugs (Ocean University of China), Chinese Ministry of Education, Qingdao, China.; School of Medicine and Pharmacy, Ocean University of China, Qingdao, China.; Laboratory for Marine Drugs and Biological Products, Qingdao National Laboratory for Marine Science and Technology, China.
Tan X; CAS Key Laboratory of Experimental Marine Biology, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, China.
Wang ZH; Key Laboratory of Marine Drugs (Ocean University of China), Chinese Ministry of Education, Qingdao, China.; School of Medicine and Pharmacy, Ocean University of China, Qingdao, China.; Laboratory for Marine Drugs and Biological Products, Qingdao National Laboratory for Marine Science and Technology, China.; CAS Key Laboratory of Experimental Marine Biology, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, China.
Liu YZ; Key Laboratory of Marine Drugs (Ocean University of China), Chinese Ministry of Education, Qingdao, China.; School of Medicine and Pharmacy, Ocean University of China, Qingdao, China.; Laboratory for Marine Drugs and Biological Products, Qingdao National Laboratory for Marine Science and Technology, China.
Zhou JF; Key Laboratory of Marine Drugs (Ocean University of China), Chinese Ministry of Education, Qingdao, China.; School of Medicine and Pharmacy, Ocean University of China, Qingdao, China.; Laboratory for Marine Drugs and Biological Products, Qingdao National Laboratory for Marine Science and Technology, China.
Rong XZ; Key Laboratory of Marine Drugs (Ocean University of China), Chinese Ministry of Education, Qingdao, China.; School of Medicine and Pharmacy, Ocean University of China, Qingdao, China.; Laboratory for Marine Drugs and Biological Products, Qingdao National Laboratory for Marine Science and Technology, China.
Lu L; Key Laboratory of Marine Drugs (Ocean University of China), Chinese Ministry of Education, Qingdao, China.; School of Medicine and Pharmacy, Ocean University of China, Qingdao, China.; Laboratory for Marine Drugs and Biological Products, Qingdao National Laboratory for Marine Science and Technology, China.
Li Y; Key Laboratory of Marine Drugs (Ocean University of China), Chinese Ministry of Education, Qingdao, China.; School of Medicine and Pharmacy, Ocean University of China, Qingdao, China.; Laboratory for Marine Drugs and Biological Products, Qingdao National Laboratory for Marine Science and Technology, China.; Pokaż więcej
Źródło:: The FEBS journal [FEBS J] 2018 Sep; Vol. 285 (18), pp. 3464-3484. Date of Electronic Publication: 2018 Aug 24.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Cartilage/*embryology
Embryo, Nonmammalian/*cytology
Nuclear Proteins/*metabolism
Pharynx/*embryology
Tumor Suppressor Protein p53/*metabolism
Zebrafish/*embryology
Zebrafish Proteins/*metabolism
Animals ; Animals, Genetically Modified/embryology ; Animals, Genetically Modified/genetics ; Animals, Genetically Modified/metabolism ; Body Patterning ; Cartilage/metabolism ; Embryo, Nonmammalian/metabolism ; Mutation ; Nuclear Proteins/genetics ; Pharynx/metabolism ; Ribosomes/metabolism ; Tumor Suppressor Protein p53/genetics ; Zebrafish/genetics ; Zebrafish/metabolism ; Zebrafish Proteins/genetics

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Skocz do pozycji: 16.

Tytuł:: Transcriptomic Analysis of Naïve Human Embryonic Stem Cells Cultured in Three-Dimensional PEG Scaffolds.
Autorzy:: McKee C; Department of Biological Sciences, Oakland University, Rochester, MI 48309, USA.; OU-WB Institute for Stem Cell and Regenerative Medicine, Rochester, MI 48309, USA.
Brown C; Department of Biological Sciences, Oakland University, Rochester, MI 48309, USA.; OU-WB Institute for Stem Cell and Regenerative Medicine, Rochester, MI 48309, USA.
Bakshi S; Department of Biological Sciences, Oakland University, Rochester, MI 48309, USA.; OU-WB Institute for Stem Cell and Regenerative Medicine, Rochester, MI 48309, USA.
Walker K; Department of Biological Sciences, Oakland University, Rochester, MI 48309, USA.; OU-WB Institute for Stem Cell and Regenerative Medicine, Rochester, MI 48309, USA.
Govind CK; Department of Biological Sciences, Oakland University, Rochester, MI 48309, USA.; OU-WB Institute for Stem Cell and Regenerative Medicine, Rochester, MI 48309, USA.
Chaudhry GR; Department of Biological Sciences, Oakland University, Rochester, MI 48309, USA.; OU-WB Institute for Stem Cell and Regenerative Medicine, Rochester, MI 48309, USA.; Pokaż więcej
Źródło:: Biomolecules [Biomolecules] 2020 Dec 28; Vol. 11 (1). Date of Electronic Publication: 2020 Dec 28.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Cell Culture Techniques*
Human Embryonic Stem Cells/*metabolism
Nuclear Proteins/*genetics
Transcription Factors/*genetics
Transcriptome/*genetics
Animals ; Cell Differentiation/genetics ; Cell Line ; Cell Proliferation/genetics ; Cellular Microenvironment/genetics ; Gene Expression Profiling ; Gene Expression Regulation, Developmental/drug effects ; Human Embryonic Stem Cells/cytology ; Humans ; Polyethylene Glycols/pharmacology ; RNA-Seq ; Tumor Suppressor Protein p53/genetics ; Wnt Signaling Pathway/genetics

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Skocz do pozycji: 17.

Tytuł:: Loss of ZC4H2 and RNF220 Inhibits Neural Stem Cell Proliferation and Promotes Neuronal Differentiation.
Autorzy:: Zhang L; State Key Laboratory of Genetic Resources and Evolution, Kunming Institute of Zoology, Chinese Academy of Sciences, Kunming 650223, China.; Kunming College of Life Science, University of Chinese Academy of Sciences, Kunming 650204, China.
Ye M; Kunming College of Life Science, University of Chinese Academy of Sciences, Kunming 650204, China.; Key Laboratory of Animal Models and Human Disease Mechanisms of the Chinese Academy of Sciences and Yunnan Province, and KIZ-CUHK Joint Laboratory of Bioresources and Molecular Research in Common Diseases, Kunming Institute of Zoology, Chinese Academy of Sciences, Kunming 650223, China.
Zhu L; State Key Laboratory of Genetic Resources and Evolution, Kunming Institute of Zoology, Chinese Academy of Sciences, Kunming 650223, China.; Kunming College of Life Science, University of Chinese Academy of Sciences, Kunming 650204, China.
Cha J; State Key Laboratory of Genetic Resources and Evolution, Kunming Institute of Zoology, Chinese Academy of Sciences, Kunming 650223, China.; Kunming College of Life Science, University of Chinese Academy of Sciences, Kunming 650204, China.
Li C; State Key Laboratory of Genetic Resources and Evolution, Kunming Institute of Zoology, Chinese Academy of Sciences, Kunming 650223, China.
Yao YG; Kunming College of Life Science, University of Chinese Academy of Sciences, Kunming 650204, China.; Key Laboratory of Animal Models and Human Disease Mechanisms of the Chinese Academy of Sciences and Yunnan Province, and KIZ-CUHK Joint Laboratory of Bioresources and Molecular Research in Common Diseases, Kunming Institute of Zoology, Chinese Academy of Sciences, Kunming 650223, China.; CAS Center for Excellence in Brain Science and Intelligence Technology, Chinese Academy of Sciences, Shanghai 200031, China.
Mao B; State Key Laboratory of Genetic Resources and Evolution, Kunming Institute of Zoology, Chinese Academy of Sciences, Kunming 650223, China.; Center for Excellence in Animal Evolution and Genetics, Chinese Academy of Sciences, Kunming, Yunnan 650223, China.; Pokaż więcej
Źródło:: Cells [Cells] 2020 Jul 01; Vol. 9 (7). Date of Electronic Publication: 2020 Jul 01.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Cell Proliferation/*genetics
Intracellular Signaling Peptides and Proteins/*metabolism
Neural Stem Cells/*metabolism
Neurogenesis/*genetics
Nuclear Proteins/*metabolism
Ubiquitin-Protein Ligases/*metabolism
Animals ; Cerebellar Cortex/cytology ; Cerebellar Cortex/metabolism ; Cyclin D1/genetics ; Cyclin D1/metabolism ; Cyclin-Dependent Kinase Inhibitor p21/genetics ; Cyclin-Dependent Kinase Inhibitor p21/metabolism ; Down-Regulation ; G1 Phase Cell Cycle Checkpoints/genetics ; Intracellular Signaling Peptides and Proteins/genetics ; Membrane Proteins/genetics ; Membrane Proteins/metabolism ; Mice ; Mice, Knockout ; Nerve Tissue Proteins/genetics ; Nerve Tissue Proteins/metabolism ; Neural Stem Cells/cytology ; Nuclear Proteins/genetics ; RNA-Seq ; Receptor, Notch1/genetics ; Receptor, Notch1/metabolism ; Transcription Factor HES-1/genetics ; Transcription Factor HES-1/metabolism ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/metabolism ; Ubiquitin-Protein Ligases/genetics ; Up-Regulation

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Tytuł:: Role of Junction-Mediating and Regulatory Protein in the Pathogenesis of Glucocorticoid-Induced Endothelial Cell Lesions.
Autorzy:: Zuo W; Peking University China-Japan Friendship School of Clinical Medicine, Beijing, China.
Guo WS; Peking University China-Japan Friendship School of Clinical Medicine, Beijing, China.; Department of Orthopaedic Surgery, Center for Osteonecrosis and Joint Preserving & Reconstruction, China-Japan Friendship Hospital, Beijing, China.
Yu HC; Graduate School of Peking Union Medical College, Beijing, China.
Liu P; Beijing University of Chinese Medicine, Beijing, China.
Zhang QD; Department of Orthopaedic Surgery, Center for Osteonecrosis and Joint Preserving & Reconstruction, China-Japan Friendship Hospital, Beijing, China.; Pokaż więcej
Źródło:: Orthopaedic surgery [Orthop Surg] 2020 Jun; Vol. 12 (3), pp. 964-973. Date of Electronic Publication: 2020 May 04.
Typ publikacji:: Journal Article
MeSH Terms:: Glucocorticoids/*adverse effects
Human Umbilical Vein Endothelial Cells/*drug effects
Nuclear Proteins/*metabolism
Trans-Activators/*metabolism
Apoptosis ; Cell Movement ; Cell Proliferation ; Cells, Cultured ; Femur Head Necrosis/chemically induced ; Femur Head Necrosis/genetics ; Gene Knockdown Techniques ; Humans ; Nuclear Proteins/genetics ; RNA, Messenger/metabolism ; Trans-Activators/genetics ; Tumor Suppressor Protein p53/metabolism

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Skocz do pozycji: 19.

Tytuł:: Mutations in the TP53 gene affected recruitment of 53BP1 protein to DNA lesions, but level of 53BP1 was stable after γ-irradiation that depleted MDC1 protein in specific TP53 mutants.
Autorzy:: Suchánková J; Institute of Biophysics, Academy of Sciences of the Czech Republic, v.v.i., Královopolská 135, 612 00, Brno, Czech Republic.
Legartová S; Institute of Biophysics, Academy of Sciences of the Czech Republic, v.v.i., Královopolská 135, 612 00, Brno, Czech Republic.
Ručková E; Masaryk Memorial Cancer Institute, Žlutý kopec 543/7, 656 53, Brno, Czech Republic.
Vojtěšek B; Masaryk Memorial Cancer Institute, Žlutý kopec 543/7, 656 53, Brno, Czech Republic.
Kozubek S; Institute of Biophysics, Academy of Sciences of the Czech Republic, v.v.i., Královopolská 135, 612 00, Brno, Czech Republic.
Bártová E; Institute of Biophysics, Academy of Sciences of the Czech Republic, v.v.i., Královopolská 135, 612 00, Brno, Czech Republic. .; Pokaż więcej
Źródło:: Histochemistry and cell biology [Histochem Cell Biol] 2017 Sep; Vol. 148 (3), pp. 239-255. Date of Electronic Publication: 2017 Apr 10.
Typ publikacji:: Journal Article
MeSH Terms:: DNA Damage*
Mutation*
Ultraviolet Rays*
Nuclear Proteins/*deficiency
Trans-Activators/*deficiency
Tumor Suppressor Protein p53/*genetics
Tumor Suppressor p53-Binding Protein 1/*metabolism
Adaptor Proteins, Signal Transducing ; Cell Cycle Proteins ; Down-Regulation ; Humans ; Nuclear Proteins/metabolism ; Trans-Activators/metabolism ; Tumor Cells, Cultured ; Tumor Suppressor Protein p53/deficiency ; Tumor Suppressor Protein p53/metabolism

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Tytuł:: p53-Related Transcription Targets of TAp73 in Cancer Cells-Bona Fide or Distorted Reality?
Autorzy:: Wang C; Division of Cellular and Molecular Research, Humphrey Oei Institute of Cancer Research, National Cancer Centre Singapore, Singapore 169610, Singapore.
Teo CR; Cancer and Stem Cell Biology Program, Duke-NUS Medical School, Singapore 169857, Singapore.
Sabapathy K; Division of Cellular and Molecular Research, Humphrey Oei Institute of Cancer Research, National Cancer Centre Singapore, Singapore 169610, Singapore.; Cancer and Stem Cell Biology Program, Duke-NUS Medical School, Singapore 169857, Singapore.; Institute of Molecular and Cell Biology, Biopolis, Singapore 138673, Singapore.; Department of Biochemistry, Yong Loo Lin School of Medicine, National University of Singapore, Singapore 119228, Singapore.; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2020 Feb 17; Vol. 21 (4). Date of Electronic Publication: 2020 Feb 17.
Typ publikacji:: Journal Article; Review
MeSH Terms:: Transcription, Genetic*
Nuclear Proteins/*genetics
Nuclear Proteins/*metabolism
Tumor Suppressor Protein p53/*genetics
Tumor Suppressor Protein p53/*metabolism
Animals ; Cell Line, Tumor ; Cell Proliferation ; Gene Expression Regulation, Neoplastic ; Genes, Tumor Suppressor ; Humans ; Mice ; Tumor Protein p73/metabolism

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