Effect of voluntary exercise upon the metabolic syndrome and gut microbiome composition in mice.
Autorzy:
Moore TM; Division of Cardiology, Department of Medicine, University of California, Los Angeles, California, USA. Terrazas A; Division of Cardiology, Department of Medicine, University of California, Los Angeles, California, USA. Strumwasser AR; Division of Endocrinology, Diabetes, and Hypertension, University of California, Los Angeles, California, USA. Lin AJ; Division of Endocrinology, Diabetes, and Hypertension, University of California, Los Angeles, California, USA. Zhu X; Division of Pediatric Endocrinology, Department of Pediatrics UCLA Children's Discovery and Innovation Institute, Department of Medicine, University of California, Los Angeles, California, USA. Anand ATS; Division of Endocrinology, Diabetes, and Hypertension, University of California, Los Angeles, California, USA. Nguyen CQ; Division of Endocrinology, Diabetes, and Hypertension, University of California, Los Angeles, California, USA. Stiles L; Division of Endocrinology, Diabetes, and Hypertension, University of California, Los Angeles, California, USA. Norheim F; Department of Human Genetics, University of California, Los Angeles, California, USA. Lang JM; Department of Human Genetics, University of California, Los Angeles, California, USA. Hui ST; Division of Cardiology, Department of Medicine, University of California, Los Angeles, California, USA. TurcotteLP; Department of Biological Sciences, Dana & David Dornsife College of Letters, Arts, and Sciences, University of Southern California, Los Angeles, California, USA. Zhou Z; Division of Endocrinology, Diabetes, and Hypertension, University of California, Los Angeles, California, USA.
Age-induced mitochondrial DNA point mutations are inadequate to alter metabolic homeostasis in response to nutrient challenge.
Autorzy:
Moore TM; Department of Biological Sciences, Dana & David Dornsife College of Letters, Arts, and Sciences, University of Southern California, Los Angeles, CA, USA.; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Zhou Z; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Strumwasser AR; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Cohn W; Department of Psychiatry and Biobehavioral Sciences & The Semel Institute for Neuroscience and Human Behavior, University of California, Los Angeles, CA, USA. Lin AJ; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Cory K; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Whitney K; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Ho T; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Ho T; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Lee JL; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Rucker DH; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Hoang AN; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Widjaja K; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Abrishami AD; Division of Cardiology, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Charugundla S; Division of Cardiology, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Stiles L; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Whitelegge JP; Department of Psychiatry and Biobehavioral Sciences & The Semel Institute for Neuroscience and Human Behavior, University of California, Los Angeles, CA, USA. TurcotteLP; Department of Biological Sciences, Dana & David Dornsife College of Letters, Arts, and Sciences, University of Southern California, Los Angeles, CA, USA. Wanagat J; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA. Hevener AL; Division of Endocrinology, Diabetes, and Hypertension, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, CA, USA.; Iris Cantor-UCLA Women's Health Center, University of California, Los Angeles, CA, USA.
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Źródło:
Aging cell [Aging Cell] 2020 Nov; Vol. 19 (11), pp. e13166. Date of Electronic Publication: 2020 Oct 13.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
TNF-α Stress Response Is Reduced Following Load Carriage Training.
Autorzy:
Jensen AE; Innovative Employee Solutions, 9665 Granite Ridge Road, #420, San Diego, CA.; University of Southern California, Biological Sciences, Human and Evolutionary Biology, Dornsife College of Letters, Arts and Sciences, 3616 Trousdale Parkway, AHF 247, Los Angeles, CA.; Warfighter Performance Department, Naval Health Research Center, 140 Sylvester Road, San Diego, CA. Niederberger B; Warfighter Performance Department, Naval Health Research Center, 140 Sylvester Road, San Diego, CA.; Leidos, 10260 Campus Point Drive, San Diego, CA. Jaworski R; Marine Corps System Command, Marine Expeditionary Rifle Squad, 2200 Lester Street, Quantico, VA. Devaney JM; Children's National Medical Center, 111 Michigan Avenue NW, Center for Genetic Medicine Research, Rm. 5700, Washington, DC. TurcotteLP; University of Southern California, Biological Sciences, Human and Evolutionary Biology, Dornsife College of Letters, Arts and Sciences, 3616 Trousdale Parkway, AHF 247, Los Angeles, CA. Kelly KR; Warfighter Performance Department, Naval Health Research Center, 140 Sylvester Road, San Diego, CA.
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Źródło:
Military medicine [Mil Med] 2019 Jan 01; Vol. 184 (1-2), pp. e256-e260.
Typ publikacji:
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
Amelioration of palmitate-induced metabolic dysfunction in L6 muscle cells expressing low levels of receptor-interacting protein 140.
Autorzy:
Constantinescu S; b Department of Math and Science, Marymount California University, Rancho Palos Verdes, California, USA. TurcotteLP; a Department of Biological Sciences, Human and Evolutionary Biology, Dana and David Dornsife College of Letters, Arts and Sciences, University of Southern California, 3560 Watt Way, PED 107, Los Angeles, CA 90089-0652, USA.
Contraction-induced signaling: evidence of convergent cascades in the regulation of muscle fatty acid metabolism.
Autorzy:
TurcotteLP; Department of Biological Sciences, Dana and David Dornsife College of Letters, Arts, and Sciences, University of Southern California, Los Angeles, CA 90089-0652, USA. turcotte@usc.edu Abbott MJ
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