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    Wyświetlanie 1-3 z 3
    Tytuł:
    Renin-Angiotensin System in Huntington's Disease: Evidence from Animal Models and Human Patients.
    Autorzy:
    Kangussu LM; Department of Morphology, Biological Science Institute, Federal University of Minas Gerais, Belo Horizonte 31270-901, MG, Brazil.
    Rocha NP; Department of Neurology, The Mitchell Center for Alzheimer's Disease and Related Brain Disorders, The University of Texas Health Science Center, Houston, TX 77030, USA.; McGovern Medical School, HDSA Center of Excellence at The University of Texas Health Science Center, Houston, TX 77030, USA.; Neuropsychiatry Program, Department of Psychiatry and Behavioral Sciences, McGovern Medical School, University of Texas, Houston, TX 77054, USA.
    Valadão PAC; Department of Morphology, Biological Science Institute, Federal University of Minas Gerais, Belo Horizonte 31270-901, MG, Brazil.
    Machado TCG; Department of Morphology, Biological Science Institute, Federal University of Minas Gerais, Belo Horizonte 31270-901, MG, Brazil.
    Soares KB; Department of Morphology, Biological Science Institute, Federal University of Minas Gerais, Belo Horizonte 31270-901, MG, Brazil.
    Joviano-Santos JV; Department of Morphology, Biological Science Institute, Federal University of Minas Gerais, Belo Horizonte 31270-901, MG, Brazil.; Center for Mathematics, Computing, and Cognition (CMCC), Federal University of ABC (UFABC), São Bernardo do Campo 09612-000, SP, Brazil.
    Latham LB; McGovern Medical School, HDSA Center of Excellence at The University of Texas Health Science Center, Houston, TX 77030, USA.; School of Medicine, University of Washington, Seattle, WA 98195, USA.
    Colpo GD; Neuropsychiatry Program, Department of Psychiatry and Behavioral Sciences, McGovern Medical School, University of Texas, Houston, TX 77054, USA.
    Almeida-Santos AF; Department of Morphology, Biological Science Institute, Federal University of Minas Gerais, Belo Horizonte 31270-901, MG, Brazil.
    Furr Stimming E; McGovern Medical School, HDSA Center of Excellence at The University of Texas Health Science Center, Houston, TX 77030, USA.
    Simões E Silva AC; Department of Pediatrics, School of Medicine, Federal University of Minas Gerais, Belo Horizonte 30130-100, MG, Brazil.
    Teixeira AL; Neuropsychiatry Program, Department of Psychiatry and Behavioral Sciences, McGovern Medical School, University of Texas, Houston, TX 77054, USA.; Department of Psychiatry & Behavioral Sciences, McGovern Medical School, University of Texas Health Science Center, Houston, TX 77054, USA.
    Miranda AS; Department of Morphology, Biological Science Institute, Federal University of Minas Gerais, Belo Horizonte 31270-901, MG, Brazil.
    Guatimosim C; Department of Morphology, Biological Science Institute, Federal University of Minas Gerais, Belo Horizonte 31270-901, MG, Brazil.
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    Źródło:
    International journal of molecular sciences [Int J Mol Sci] 2022 Jul 12; Vol. 23 (14). Date of Electronic Publication: 2022 Jul 12.
    Typ publikacji:
    Journal Article
    MeSH Terms:
    Angiotensin I*/genetics
    Angiotensin I*/metabolism
    Angiotensin-Converting Enzyme 2*/genetics
    Huntington Disease*/genetics
    Peptide Fragments*/genetics
    Peptide Fragments*/metabolism
    Receptor, Angiotensin, Type 1*/genetics
    Receptor, Angiotensin, Type 1*/metabolism
    Renin-Angiotensin System*/genetics
    Renin-Angiotensin System*/physiology
    Angiotensin II/metabolism ; Animals ; Disease Models, Animal ; Humans ; Mice ; Peptidyl-Dipeptidase A/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
    Tytuł:
    Motoneuron-specific loss of VAChT mimics neuromuscular defects seen in congenital myasthenic syndrome.
    Autorzy:
    Joviano-Santos JV; Robarts Research Institute, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.; Departamento de Morfologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Kljakic O; Robarts Research Institute, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.; Department of Anatomy and Cell Biology, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.
    Magalhães-Gomes MPS; Departamento de Morfologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.; Departamento de Medicina, Faculdade Ciências Médicas de Minas Gerais, FCMMG, Belo Horizonte, Brasil.
    Valadão PAC; Departamento de Morfologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    de Oliveira LR; Departamento de Morfologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Prado MAM; Robarts Research Institute, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.; Department of Anatomy and Cell Biology, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.; Department of Physiology and Pharmacology, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.; Brain and Mind Institute, University of Western Ontario, London, Canada.
    Prado VF; Robarts Research Institute, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.; Department of Anatomy and Cell Biology, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.; Department of Physiology and Pharmacology, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.; Brain and Mind Institute, University of Western Ontario, London, Canada.
    Guatimosim C; Departamento de Morfologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
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    Źródło:
    The FEBS journal [FEBS J] 2021 Sep; Vol. 288 (18), pp. 5331-5349. Date of Electronic Publication: 2021 Apr 25.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Acetylcholine/*genetics
    Motor Neurons/*metabolism
    Myasthenic Syndromes, Congenital/*genetics
    Vesicular Acetylcholine Transport Proteins/*genetics
    Acetylcholine/metabolism ; Animals ; Disease Models, Animal ; Humans ; Mice ; Motor Neurons/pathology ; Muscle Contraction/genetics ; Muscle Contraction/physiology ; Muscle, Skeletal/metabolism ; Muscle, Skeletal/physiology ; Myasthenic Syndromes, Congenital/metabolism ; Myasthenic Syndromes, Congenital/pathology ; Neuromuscular Junction/genetics ; Neuromuscular Junction/metabolism ; Neurotransmitter Agents/genetics ; Spinal Cord/metabolism ; Spinal Cord/physiology ; Synaptic Transmission/genetics ; Synaptic Vesicles/metabolism
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknieSzczegóły
    Tytuł:
    Increased oxidative stress and CaMKII activity contribute to electro-mechanical defects in cardiomyocytes from a murine model of Huntington's disease.
    Autorzy:
    Joviano-Santos JV; Department of Morphology, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Santos-Miranda A; Department of Biochemistry and Immunology, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Botelho AFM; Department of Veterinary Clinic and Surgery, Escola de Veterinária, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    de Jesus ICG; Department of Physiology, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Andrade JN; Department of Morphology, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    de Oliveira Barreto T; Department of Biochemistry and Immunology, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Magalhães-Gomes MPS; Department of Morphology, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Valadão PAC; Department of Morphology, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Cruz JDS; Department of Biochemistry and Immunology, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Melo MM; Department of Veterinary Clinic and Surgery, Escola de Veterinária, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Guatimosim S; Department of Physiology, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Guatimosim C; Department of Morphology, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil.
    Pokaż więcej
    Źródło:
    The FEBS journal [FEBS J] 2019 Jan; Vol. 286 (1), pp. 110-123. Date of Electronic Publication: 2018 Dec 20.
    Typ publikacji:
    Journal Article; Research Support, Non-U.S. Gov't
    MeSH Terms:
    Disease Models, Animal*
    Oxidative Stress*
    Arrhythmias, Cardiac/*pathology
    Calcium-Calmodulin-Dependent Protein Kinase Type 2/*metabolism
    Huntington Disease/*physiopathology
    Mitochondria/*pathology
    Myocytes, Cardiac/*pathology
    Animals ; Antioxidants/metabolism ; Arrhythmias, Cardiac/metabolism ; Biomechanical Phenomena ; Electrophysiological Phenomena ; Mice ; Mice, Transgenic ; Mitochondria/metabolism ; Myocytes, Cardiac/metabolism ; Phosphorylation
    Czasopismo naukowe
    Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
      Wyświetlanie 1-3 z 3

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