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Tytuł:
Acacetin alleviates myocardial ischaemia/reperfusion injury by inhibiting oxidative stress and apoptosis via the Nrf-2/HO-1 pathway.
Autorzy:
Wu C; Institute of Cardiovascular Research, Xiamen Cardiovascular Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen, Fujian, China.
Chen RL; Institute of Cardiovascular Research, Xiamen Cardiovascular Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen, Fujian, China.
Wang Y; Institute of Cardiovascular Research, Xiamen Cardiovascular Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen, Fujian, China.
Wu WY; Institute of Cardiovascular Research, Xiamen Cardiovascular Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen, Fujian, China.
Li G; Institute of Cardiovascular Research, Xiamen Cardiovascular Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen, Fujian, China.
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Źródło:
Pharmaceutical biology [Pharm Biol] 2022 Dec; Vol. 60 (1), pp. 553-561.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Flavones/*pharmacology
Myocardial Reperfusion Injury/*drug therapy
Oxidative Stress/*drug effects
Animals ; Anti-Inflammatory Agents/pharmacology ; Antioxidants/pharmacology ; Heme Oxygenase (Decyclizing)/metabolism ; Male ; Myocardial Reperfusion Injury/physiopathology ; NF-E2-Related Factor 2/metabolism ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Rats ; Rats, Sprague-Dawley ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Transgelin-2 Involves in the Apoptosis of Colorectal Cancer Cells Induced by Tanshinone-IIA.
Autorzy:
Zhang Y; Department of Medical Oncology, Shuguang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.; Academy of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Zhao Y; Department of Medical Oncology, Shuguang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.; Academy of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Liu J; Department of Medical Oncology, Shuguang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Li C; Department of Medical Oncology, Shuguang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Feng Y; Academy of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Jiang S; Department of Medical Oncology, Shuguang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.; Academy of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Sun X; Department of Medical Oncology, Shuguang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.; Academy of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Hu X; Department of Medical Oncology, Shuguang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.; Academy of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
Wang Y; Department of Medical Oncology, Shuguang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.; Academy of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.
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Źródło:
Analytical cellular pathology (Amsterdam) [Anal Cell Pathol (Amst)] 2022 Sep 27; Vol. 2022, pp. 9358583. Date of Electronic Publication: 2022 Sep 27 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Colorectal Neoplasms*/drug therapy
Abietanes ; Cell Line, Tumor ; Humans ; Microfilament Proteins ; Muscle Proteins
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Hypoxia Induces Apoptosis of Microglia BV2 by Upregulating Kir2.1 to Activate Mitochondrial-Related Apoptotic Pathways.
Autorzy:
Xie YF; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.
Wang Y; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.
Rong Y; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.
He W; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Pathophysiology, Shihezi University School of Medicine, Shihezi, China.
Yan M; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Pathophysiology, Shihezi University School of Medicine, Shihezi, China.
Li X; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Pathophysiology, Shihezi University School of Medicine, Shihezi, China.
Si J; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.
Li L; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Medical College of Jiaxing University, Jiaxing, Zhejiang, China.
Zhang Y; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.; Clinical Laboratory of the First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.
Ma K; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.
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Źródło:
Disease markers [Dis Markers] 2022 Mar 17; Vol. 2022, pp. 5855889. Date of Electronic Publication: 2022 Mar 17 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*/genetics
Microglia*/metabolism
Cell Hypoxia ; Humans ; Hypoxia/metabolism ; Mitochondria/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Overexpression of lncRNA Dancr inhibits apoptosis and enhances autophagy to protect cardiomyocytes from endoplasmic reticulum stress injury via sponging microRNA-6324.
Autorzy:
Li J; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, School of Basic Medical Sciences, Lanzhou University, Lanzhou, Gansu 730000, P.R. China.
Xie J; Department of Ultrasonic Diagnosis, The First People's Hospital of Lanzhou, Lanzhou, Gansu 730050, P.R. China.
Wang YZ; Gansu Cardiovascular Institute, Lanzhou, Gansu 730050, P.R. China.
Gan YR; Gansu Cardiovascular Institute, Lanzhou, Gansu 730050, P.R. China.
Wei L; Outpatient Department, The First People's Hospital of Lanzhou, Lanzhou, Gansu 730050, P.R. China.
Ding GW; Medical Department, Shijiazhuang People's Medical College, Shijiazhuang, Hebei 050599, P.R. China.
Ding YH; Anesthesiology Department, The First People's Hospital of Lanzhou, Lanzhou, Gansu 730050, P.R. China.
Xie DX; Gansu Cardiovascular Institute, Lanzhou, Gansu 730050, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2021 Feb; Vol. 23 (2). Date of Electronic Publication: 2020 Dec 10.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Autophagy*
Endoplasmic Reticulum Stress*
MicroRNAs/*biosynthesis
Myocytes, Cardiac/*metabolism
RNA, Long Noncoding/*biosynthesis
Animals ; Cell Line ; MicroRNAs/genetics ; Myocytes, Cardiac/pathology ; RNA, Long Noncoding/genetics ; Rats
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Dickkopf‑1/cysteine‑rich angiogenic inducer 61 axis mediates palmitic acid‑induced inflammation and apoptosis of vascular endothelial cells.
Autorzy:
Gan YR; Gansu Cardiovascular Institute, The First People's Hospital of Lanzhou City, Lanzhou, Gansu 730050, P.R. China.
Wei L; Department of Outpatient, The First People's Hospital of Lanzhou City, Lanzhou, Gansu 730050, P.R. China.
Wang YZ; Gansu Cardiovascular Institute, The First People's Hospital of Lanzhou City, Lanzhou, Gansu 730050, P.R. China.
Kou ZK; Gansu Cardiovascular Institute, The First People's Hospital of Lanzhou City, Lanzhou, Gansu 730050, P.R. China.
Liang TX; Gansu Cardiovascular Institute, The First People's Hospital of Lanzhou City, Lanzhou, Gansu 730050, P.R. China.
Ding GW; Department of Clinical Medicine, Shijiazhuang People's Medical College, Shijiazhuang, Hebei 050599, P.R. China.
Ding YH; Department of Anesthesiology, The First People's Hospital of Lanzhou City, Lanzhou, Gansu 730050, P.R. China.
Xie DX; Gansu Cardiovascular Institute, The First People's Hospital of Lanzhou City, Lanzhou, Gansu 730050, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2021 Feb; Vol. 23 (2). Date of Electronic Publication: 2020 Dec 10.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Cysteine-Rich Protein 61/*metabolism
Intercellular Signaling Peptides and Proteins/*metabolism
Palmitic Acid/*adverse effects
Signal Transduction/*drug effects
Apoptosis/genetics ; Cysteine-Rich Protein 61/genetics ; Human Umbilical Vein Endothelial Cells ; Humans ; Inflammation/chemically induced ; Inflammation/genetics ; Inflammation/metabolism ; Intercellular Signaling Peptides and Proteins/genetics ; Palmitic Acid/pharmacology ; Signal Transduction/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
AFB1 Induced Transcriptional Regulation Related to Apoptosis and Lipid Metabolism in Liver of Chicken.
Autorzy:
Liu X; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Kumar Mishra S; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Wang T; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Xu Z; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Zhao X; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Wang Y; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Yin H; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Fan X; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Zeng B; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Yang M; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Yang D; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Ni Q; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Li Y; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Zhang M; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Zhu Q; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
Chen F; Development Research Center of the State Council, Beijing 100010, China.
Li D; Institute of Animal Genetics and Breeding, College of Animal Science and Technology, Sichuan Agricultural University, Chengdu 611130, China.
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Źródło:
Toxins [Toxins (Basel)] 2020 May 04; Vol. 12 (5). Date of Electronic Publication: 2020 May 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Aflatoxin B1/*toxicity
Apoptosis/*drug effects
Hepatocytes/*drug effects
Lipid Metabolism/*drug effects
Liver/*drug effects
RNA/*metabolism
Transcription, Genetic/*drug effects
Animals ; Apoptosis/genetics ; Chickens ; Female ; Gene Expression Regulation ; Gene Regulatory Networks ; Hepatocytes/metabolism ; Hepatocytes/pathology ; Lipid Metabolism/genetics ; Liver/metabolism ; Liver/pathology ; MicroRNAs/genetics ; MicroRNAs/metabolism ; RNA/genetics ; RNA, Long Noncoding/genetics ; RNA, Long Noncoding/metabolism ; RNA, Messenger/genetics ; RNA, Messenger/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Oxidative Stress and Apoptotic Changes in Broiler Chicken Splenocytes Exposed to T-2 Toxin.
Autorzy:
Chen Y; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Han S; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Wang Y; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Li D; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Zhao X; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Zhu Q; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Yin H; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
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Źródło:
BioMed research international [Biomed Res Int] 2019 Nov 25; Vol. 2019, pp. 5493870. Date of Electronic Publication: 2019 Nov 25 (Print Publication: 2019).
Typ publikacji:
Journal Article
MeSH Terms:
Spleen*/cytology
Spleen*/drug effects
Spleen*/pathology
Apoptosis/*drug effects
Oxidative Stress/*drug effects
T-2 Toxin/*toxicity
Animals ; Cells, Cultured ; Chickens ; Dose-Response Relationship, Drug ; Male
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Immunomodulatory Protein from Nectria haematococca Induces Apoptosis in Lung Cancer Cells via the P53 Pathway.
Autorzy:
Wang JJ; Institute of Food Science and Technology, Chinese Academy of Agricultural Sciences, No. 2 Yuan Ming Yuan West Road, Beijing 100193, China. .
Wang Y; Institute of Food Science and Technology, Chinese Academy of Agricultural Sciences, No. 2 Yuan Ming Yuan West Road, Beijing 100193, China. .
Hou L; Institute of Food Science and Technology, Chinese Academy of Agricultural Sciences, No. 2 Yuan Ming Yuan West Road, Beijing 100193, China. .
Xin F; Institute of Food Science and Technology, Chinese Academy of Agricultural Sciences, No. 2 Yuan Ming Yuan West Road, Beijing 100193, China. .
Fan B; Institute of Food Science and Technology, Chinese Academy of Agricultural Sciences, No. 2 Yuan Ming Yuan West Road, Beijing 100193, China. .
Lu C; Institute of Food Science and Technology, Chinese Academy of Agricultural Sciences, No. 2 Yuan Ming Yuan West Road, Beijing 100193, China. .
Zhang L; Institute of Food Science and Technology, Chinese Academy of Agricultural Sciences, No. 2 Yuan Ming Yuan West Road, Beijing 100193, China. .
Wang F; Institute of Food Science and Technology, Chinese Academy of Agricultural Sciences, No. 2 Yuan Ming Yuan West Road, Beijing 100193, China. .
Li S; Institute of Food Science and Technology, Chinese Academy of Agricultural Sciences, No. 2 Yuan Ming Yuan West Road, Beijing 100193, China. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2019 Oct 28; Vol. 20 (21). Date of Electronic Publication: 2019 Oct 28.
Typ publikacji:
Journal Article
MeSH Terms:
Nectria*
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Fungal Proteins/*pharmacology
Lung Neoplasms/*drug therapy
Lung Neoplasms/*metabolism
Tumor Suppressor Protein p53/*metabolism
A549 Cells ; Antineoplastic Agents/therapeutic use ; Biosynthetic Pathways ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Fungal Proteins/metabolism ; Fungal Proteins/therapeutic use ; HEK293 Cells ; Humans ; Phosphatidylinositol 3-Kinases/metabolism ; Proteomics ; Signal Transduction/drug effects ; Tumor Suppressor Protein p53/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
FOXO3 Is Expressed in Ovarian Tissues and Acts as an Apoptosis Initiator in Granulosa Cells of Chickens.
Autorzy:
Cui C; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Han S; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Yin H; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Luo B; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Shen X; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Yang F; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Liu Z; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Zhu Q; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Li D; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
Wang Y; Farm Animal Genetic Resources Exploration and Innovation Key Laboratory of Sichuan Province, Sichuan Agricultural University, Chengdu, Sichuan 611130, China.
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Źródło:
BioMed research international [Biomed Res Int] 2019 Jul 11; Vol. 2019, pp. 6902906. Date of Electronic Publication: 2019 Jul 11 (Print Publication: 2019).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*genetics
Chickens/*genetics
Forkhead Box Protein O3/*genetics
Ovarian Follicle/*metabolism
Animals ; Bcl-2-Like Protein 11/genetics ; Caspase 3/genetics ; Chickens/growth & development ; Female ; Gene Expression Regulation, Developmental/genetics ; Granulosa Cells/metabolism ; Ovarian Follicle/growth & development ; Poly (ADP-Ribose) Polymerase-1/genetics ; Proto-Oncogene Proteins/genetics ; RNA, Messenger/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Taurine protects INS-1 cells from apoptosis induced by Di(2-ethylhexyl) phthalate via reducing oxidative stress and autophagy.
Autorzy:
Li J; a Department of Nutrition and Food Safety , College of Public Health, Dalian Medical University , Dalian , Liaoning , PR China.
Zheng L; a Department of Nutrition and Food Safety , College of Public Health, Dalian Medical University , Dalian , Liaoning , PR China.
Wang X; a Department of Nutrition and Food Safety , College of Public Health, Dalian Medical University , Dalian , Liaoning , PR China.
Yao K; b Department of Orthopedics , The Second Affiliated Hospital of Dalian Medical University , Dalian , Liaoning , PR China.
Shi L; a Department of Nutrition and Food Safety , College of Public Health, Dalian Medical University , Dalian , Liaoning , PR China.
Sun X; c Department of Occupational and Environmental Health , College of Public Health, Dalian Medical University , Dalian , Liaoning , PR China.; d Natural Products Engineering Technology Center, Dalian Medical University , Dalian , Liaoning , PR China.
Yang G; a Department of Nutrition and Food Safety , College of Public Health, Dalian Medical University , Dalian , Liaoning , PR China.
Jiang L; e Department of Internal Medicine , The Afliated Zhong Shan Hospital of Dalian University , Dalian , Liaoning , PR China.
Zhang C; a Department of Nutrition and Food Safety , College of Public Health, Dalian Medical University , Dalian , Liaoning , PR China.
Wang Y; f Department of Endocrinology , the Second Hospital of Chaoyang , Chaoyang , China.
Jiang L; d Natural Products Engineering Technology Center, Dalian Medical University , Dalian , Liaoning , PR China.; g Preventive Medicine Laboratory College of Public Health , Dalian Medical University , Dalian , Liaoning , PR China.
Liu X; a Department of Nutrition and Food Safety , College of Public Health, Dalian Medical University , Dalian , Liaoning , PR China.
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Źródło:
Toxicology mechanisms and methods [Toxicol Mech Methods] 2019 Jul; Vol. 29 (6), pp. 445-456. Date of Electronic Publication: 2019 Jun 04.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Autophagy/*drug effects
Diethylhexyl Phthalate/*toxicity
Environmental Pollutants/*toxicity
Oxidative Stress/*drug effects
Protective Agents/*pharmacology
Taurine/*pharmacology
Animals ; Cell Line ; Cell Survival/drug effects ; Dose-Response Relationship, Drug ; Membrane Potential, Mitochondrial/drug effects ; Rats ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Parkin deficiency elevates hepatic ischemia/reperfusion injury accompanying decreased mitochondrial autophagy, increased apoptosis, impaired DNA damage repair and altered cell cycle distribution.
Autorzy:
Ning XJ; Hubei Key Laboratory of Medical Technology on Transplantation, Institute of Hepatobiliary Diseases, Zhongnan Hospital of Wuhan University, Transplant Center of Wuhan University, Wuhan, Hubei 430071, P.R. China.
Yan X; Hubei Key Laboratory of Medical Technology on Transplantation, Institute of Hepatobiliary Diseases, Zhongnan Hospital of Wuhan University, Transplant Center of Wuhan University, Wuhan, Hubei 430071, P.R. China.
Wang YF; Hubei Key Laboratory of Medical Technology on Transplantation, Institute of Hepatobiliary Diseases, Zhongnan Hospital of Wuhan University, Transplant Center of Wuhan University, Wuhan, Hubei 430071, P.R. China.
Wang R; Hubei Key Laboratory of Medical Technology on Transplantation, Institute of Hepatobiliary Diseases, Zhongnan Hospital of Wuhan University, Transplant Center of Wuhan University, Wuhan, Hubei 430071, P.R. China.
Fan XL; Hubei Key Laboratory of Medical Technology on Transplantation, Institute of Hepatobiliary Diseases, Zhongnan Hospital of Wuhan University, Transplant Center of Wuhan University, Wuhan, Hubei 430071, P.R. China.
Zhong ZB; Hubei Key Laboratory of Medical Technology on Transplantation, Institute of Hepatobiliary Diseases, Zhongnan Hospital of Wuhan University, Transplant Center of Wuhan University, Wuhan, Hubei 430071, P.R. China.
Ye QF; Hubei Key Laboratory of Medical Technology on Transplantation, Institute of Hepatobiliary Diseases, Zhongnan Hospital of Wuhan University, Transplant Center of Wuhan University, Wuhan, Hubei 430071, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2018 Dec; Vol. 18 (6), pp. 5663-5668. Date of Electronic Publication: 2018 Oct 29.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*genetics
Cell Cycle/*genetics
DNA Damage/*genetics
Mitophagy/*genetics
Reperfusion Injury/*genetics
Reperfusion Injury/*metabolism
Ubiquitin-Protein Ligases/*deficiency
Animals ; Disease Models, Animal ; Gene Expression Regulation ; Gene Knockdown Techniques ; Hepatocytes/metabolism ; Liver Diseases/etiology ; Liver Diseases/metabolism ; Liver Diseases/pathology ; Male ; Mitochondria/genetics ; Mitochondria/metabolism ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Rats ; Reperfusion Injury/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
RIPK3-Mediated Necroptosis and Neutrophil Infiltration Are Associated with Poor Prognosis in Patients with Alcoholic Cirrhosis.
Autorzy:
Zhang Z; Department of Infectious Diseases, Peking University First Hospital, Beijing 100034, China.; Xiamen Branch, Zhongshan Hospital, Fudan University, Xiamen 361015, China.
Xie G; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Capital Medical University, Key Laboratory of Remodeling-Related Cardiovascular Diseases, Ministry of Education, Beijing Key Laboratory of Metabolic Disturbance-Related Cardiovascular Disease, Beijing 100069, China.
Liang L; Department of Pathology, Peking University First Hospital, Beijing 100034, China.
Liu H; Department of Pathology, Beijing Youan Hospital, Capital Medical University, Beijing 100069, China.
Pan J; Department of Infectious Diseases, Peking University First Hospital, Beijing 100034, China.
Cheng H; Department of Infectious Diseases, Peking University First Hospital, Beijing 100034, China.
Wang H; Department of Oncology, The First Affiliated Hospital, Institute for Liver Diseases of Anhui Medical University, Hefei 230032, China.
Qu A; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Capital Medical University, Key Laboratory of Remodeling-Related Cardiovascular Diseases, Ministry of Education, Beijing Key Laboratory of Metabolic Disturbance-Related Cardiovascular Disease, Beijing 100069, China.
Wang Y; Department of Infectious Diseases, Peking University First Hospital, Beijing 100034, China.
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Źródło:
Journal of immunology research [J Immunol Res] 2018 Nov 25; Vol. 2018, pp. 1509851. Date of Electronic Publication: 2018 Nov 25 (Print Publication: 2018).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Liver/*pathology
Liver Cirrhosis, Alcoholic/*diagnosis
Neutrophils/*immunology
Receptor-Interacting Protein Serine-Threonine Kinases/*metabolism
Biomarkers/metabolism ; Fibrosis ; Humans ; Liver/metabolism ; Liver Cirrhosis, Alcoholic/immunology ; Necrosis ; Neutrophil Infiltration ; Prognosis ; Retrospective Studies
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MicroRNA-644a promotes apoptosis of hepatocellular carcinoma cells by downregulating the expression of heat shock factor 1.
Autorzy:
Liang W; Department of Gastrointestinal Surgery and Hepatobiliary Surgery, Guilin Medical University, Affiliated Hospital, Guilin, Guangxi, 541001, People's Republic of China.
Liao Y; Department of Gastrointestinal Surgery and Hepatobiliary Surgery, Guilin Medical University, Affiliated Hospital, Guilin, Guangxi, 541001, People's Republic of China.
Li Z; Department of Gastrointestinal Surgery and Hepatobiliary Surgery, Guilin Medical University, Affiliated Hospital, Guilin, Guangxi, 541001, People's Republic of China.
Wang Y; Department of Gastrointestinal Surgery and Hepatobiliary Surgery, Guilin Medical University, Affiliated Hospital, Guilin, Guangxi, 541001, People's Republic of China.
Zheng S; Department of Gastrointestinal Surgery and Hepatobiliary Surgery, Guilin Medical University, Affiliated Hospital, Guilin, Guangxi, 541001, People's Republic of China.
Xu X; Department of Gastrointestinal Surgery and Hepatobiliary Surgery, Guilin Medical University, Affiliated Hospital, Guilin, Guangxi, 541001, People's Republic of China.
Ran F; Department of Gastrointestinal Surgery and Hepatobiliary Surgery, Guilin Medical University, Affiliated Hospital, Guilin, Guangxi, 541001, People's Republic of China.
Tang B; Department of Gastrointestinal Surgery and Hepatobiliary Surgery, Guilin Medical University, Affiliated Hospital, Guilin, Guangxi, 541001, People's Republic of China. .
Wang Z; Department of Gastrointestinal Surgery and Hepatobiliary Surgery, Guilin Medical University, Affiliated Hospital, Guilin, Guangxi, 541001, People's Republic of China. .
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Źródło:
Cell communication and signaling : CCS [Cell Commun Signal] 2018 Jun 14; Vol. 16 (1), pp. 30. Date of Electronic Publication: 2018 Jun 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*genetics
Carcinoma, Hepatocellular/*pathology
Down-Regulation/*genetics
Heat Shock Transcription Factors/*genetics
Liver Neoplasms/*pathology
MicroRNAs/*genetics
3' Untranslated Regions/genetics ; Animals ; Carcinoma, Hepatocellular/genetics ; Cell Proliferation/genetics ; Cell Transformation, Neoplastic ; Disease Progression ; Female ; Humans ; Liver Neoplasms/genetics ; Male ; Mice ; Middle Aged ; Neoplasm Staging ; Tumor Burden/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
LncSOX4 serves an oncogenic role in the tumorigenesis of epithelial ovarian cancer by promoting cell proliferation and inhibiting apoptosis.
Autorzy:
Liu Y; Gynecology Department, Hubei Women and Children's Hospital, Wuhan, Hubei 430070, P.R. China.
Wang Y; Gynecology Department, Hubei Women and Children's Hospital, Wuhan, Hubei 430070, P.R. China.
Yao D; Gynecology Department, Hubei Women and Children's Hospital, Wuhan, Hubei 430070, P.R. China.
Cui D; Urology Department, Hubei Cancer Hospital, Wuhan, Hubei 430079, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2018 Jun; Vol. 17 (6), pp. 8282-8288. Date of Electronic Publication: 2018 Apr 18.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*genetics
Cell Transformation, Neoplastic/*genetics
Neoplasms, Glandular and Epithelial/*genetics
Ovarian Neoplasms/*genetics
RNA, Long Noncoding/*genetics
SOXC Transcription Factors/*genetics
Adult ; Carcinoma, Ovarian Epithelial ; Cell Cycle/drug effects ; Cell Cycle/genetics ; Cell Line, Tumor ; Cell Proliferation ; Female ; Gene Knockout Techniques ; Humans ; Middle Aged ; Neoplasm Metastasis ; Neoplasm Staging ; Neoplasms, Glandular and Epithelial/metabolism ; Neoplasms, Glandular and Epithelial/mortality ; Neoplasms, Glandular and Epithelial/pathology ; Ovarian Neoplasms/metabolism ; Ovarian Neoplasms/mortality ; Ovarian Neoplasms/pathology ; Prognosis
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Effects of Periostracum Cicadae on Cytokines and Apoptosis Regulatory Proteins in an IgA Nephropathy Rat Model.
Autorzy:
Yang L; Xinjiang Academy of Forestry, Urumqi 830000, China. .; Economic Forest Product Quality Inspection and Testing Center of the State Forestry Administration (Urumqi), Urumqi 830052, China. .
Wang Y; Xinjiang Academy of Forestry, Urumqi 830000, China. .; Economic Forest Product Quality Inspection and Testing Center of the State Forestry Administration (Urumqi), Urumqi 830052, China. .
Nuerbiye A; Xinjiang Medical University, Urumqi 830000, China. xjlky_.
Cheng P; Xinjiang Academy of Forestry, Urumqi 830000, China. .; Economic Forest Product Quality Inspection and Testing Center of the State Forestry Administration (Urumqi), Urumqi 830052, China. .
Wang JH; Xinjiang Medical University, Urumqi 830000, China. tcm_.; Harbin Medical University, Harbin 150081, China. tcm_.
Kasimu R; Xinjiang Medical University, Urumqi 830000, China. .
Li H; Xinjiang Academy of Forestry, Urumqi 830000, China. xjlky_.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2018 May 29; Vol. 19 (6). Date of Electronic Publication: 2018 May 29.
Typ publikacji:
Journal Article
MeSH Terms:
Glomerulonephritis, IGA*/drug therapy
Glomerulonephritis, IGA*/metabolism
Glomerulonephritis, IGA*/pathology
Apoptosis/*drug effects
Apoptosis Regulatory Proteins/*metabolism
Complex Mixtures/*pharmacology
Cytokines/*metabolism
Hemiptera/*chemistry
Animals ; Complex Mixtures/chemistry ; Disease Models, Animal ; Male ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Treatment of oral lichen planus using 308-nm excimer laser.
Autorzy:
Liu WB; Department of Dermatology, 404 Hospital of People's Liberation Army, Weihai, Shandong Provence 262200, China.
Sun LW; Department of Dermatology, 404 Hospital of People's Liberation Army, Weihai, Shandong Provence 262200, China.
Yang H; Department of Dermatology, 404 Hospital of People's Liberation Army, Weihai, Shandong Provence 262200, China.
Wang YF; Department of Dermatology, 404 Hospital of People's Liberation Army, Weihai, Shandong Provence 262200, China.
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Źródło:
Dermatologic therapy [Dermatol Ther] 2017 Sep; Vol. 30 (5). Date of Electronic Publication: 2017 Aug 23.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*radiation effects
Lasers, Excimer/*therapeutic use
Lichen Planus, Oral/*radiotherapy
Adult ; Aged ; Female ; Humans ; Lasers, Excimer/adverse effects ; Lichen Planus, Oral/pathology ; Male ; Middle Aged ; Remission Induction/methods ; T-Lymphocytes/radiation effects ; Treatment Outcome
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
iTRAQ Mitoproteome Analysis Reveals Mechanisms of Programmed Cell Death in Arabidopsis thaliana Induced by Ochratoxin A.
Autorzy:
Wang Y; Institute of Food Science and Technology, Chinese Academy of Agricultural Sciences, Beijing 100193, China. .; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Food Science & Nutritional Engineering, China Agricultural University, Beijing 100083, China. .
Peng X; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Food Science & Nutritional Engineering, China Agricultural University, Beijing 100083, China.; College of Food Science and Engineering, Northwest A&F University, Yangling 712100, China.
Yang Z; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Food Science & Nutritional Engineering, China Agricultural University, Beijing 100083, China.
Zhao W; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Food Science & Nutritional Engineering, China Agricultural University, Beijing 100083, China.
Xu W; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Food Science & Nutritional Engineering, China Agricultural University, Beijing 100083, China. .; Beijing Laboratory for Food Quality and Safety, College of Food Science and Nutritional Engineering, China Agricultural University, Beijing 100083, China. .
Hao J; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Food Science & Nutritional Engineering, China Agricultural University, Beijing 100083, China.
Wu W; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Food Science & Nutritional Engineering, China Agricultural University, Beijing 100083, China.
Shen XL; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Food Science & Nutritional Engineering, China Agricultural University, Beijing 100083, China.; School of Public Health, Zunyi Medical University, Zunyi 563000, China.
Luo Y; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Food Science & Nutritional Engineering, China Agricultural University, Beijing 100083, China.; Beijing Laboratory for Food Quality and Safety, College of Food Science and Nutritional Engineering, China Agricultural University, Beijing 100083, China.
Huang K; Beijing Advanced Innovation Center for Food Nutrition and Human Health, College of Food Science & Nutritional Engineering, China Agricultural University, Beijing 100083, China. .; Beijing Laboratory for Food Quality and Safety, College of Food Science and Nutritional Engineering, China Agricultural University, Beijing 100083, China. .
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Źródło:
Toxins [Toxins (Basel)] 2017 May 18; Vol. 9 (5). Date of Electronic Publication: 2017 May 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Arabidopsis/*drug effects
Mitochondria/*drug effects
Mitochondrial Proteins/*metabolism
Ochratoxins/*toxicity
Plant Proteins/*metabolism
Arabidopsis/genetics ; Arabidopsis/metabolism ; DNA Damage ; Gene Expression Regulation, Plant/drug effects ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/metabolism ; Mitochondria/physiology ; Mitochondrial Proteins/genetics ; Plant Leaves/drug effects ; Plant Leaves/genetics ; Plant Leaves/metabolism ; Plant Proteins/genetics ; Proteomics ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Telmisartan suppresses cardiac hypertrophy by inhibiting cardiomyocyte apoptosis via the NFAT/ANP/BNP signaling pathway.
Autorzy:
Li X; Department of Pathology, Heilongjiang Provincial Hospital, Harbin, Heilongjiang 150036, P.R. China.
Lan Y; Intensive Care Unit, Heilongjiang Provincial Hospital, Harbin, Heilongjiang 150036, P.R. China.
Wang Y; Department of Pathology, Heilongjiang Provincial Hospital, Harbin, Heilongjiang 150036, P.R. China.
Nie M; Department of Pathology, Heilongjiang Provincial Hospital, Harbin, Heilongjiang 150036, P.R. China.
Lu Y; Department of Pathology, Heilongjiang Provincial Hospital, Harbin, Heilongjiang 150036, P.R. China.
Zhao E; Department of Oral Pathology, Stomatological Hospital, Harbin Medical University, Harbin, Heilongjiang 150036, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2017 May; Vol. 15 (5), pp. 2574-2582. Date of Electronic Publication: 2017 Mar 14.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Atrial Natriuretic Factor/*metabolism
Benzimidazoles/*pharmacology
Benzoates/*pharmacology
Cardiomegaly/*metabolism
Cardiomegaly/*prevention & control
Myocytes, Cardiac/*metabolism
NFATC Transcription Factors/*metabolism
Natriuretic Peptide, Brain/*metabolism
Signal Transduction/*drug effects
Active Transport, Cell Nucleus/drug effects ; Animals ; Cardiomegaly/pathology ; Cell Nucleus/metabolism ; Cell Nucleus/pathology ; Male ; Mice ; Myocytes, Cardiac/pathology ; Telmisartan
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Costunolide and dehydrocostuslactone combination treatment inhibit breast cancer by inducing cell cycle arrest and apoptosis through c-Myc/p53 and AKT/14-3-3 pathway.
Autorzy:
Peng Z; Department of Molecular Oncology, Eastern Hepatobiliary Surgical Hospital &National Center of Liver Cancer, Second Military Medical University, Shanghai 200438, China.; School of Pharmacy, Shanghai Jiao Tong University, Shanghai 200240, China.
Wang Y; School of Pharmacy, Shanghai Jiao Tong University, Shanghai 200240, China.
Fan J; Department of Molecular Oncology, Eastern Hepatobiliary Surgical Hospital &National Center of Liver Cancer, Second Military Medical University, Shanghai 200438, China.
Lin X; Department of Molecular Oncology, Eastern Hepatobiliary Surgical Hospital &National Center of Liver Cancer, Second Military Medical University, Shanghai 200438, China.
Liu C; Department of Molecular Oncology, Eastern Hepatobiliary Surgical Hospital &National Center of Liver Cancer, Second Military Medical University, Shanghai 200438, China.
Xu Y; Department of Molecular Oncology, Eastern Hepatobiliary Surgical Hospital &National Center of Liver Cancer, Second Military Medical University, Shanghai 200438, China.
Ji W; Department of Molecular Oncology, Eastern Hepatobiliary Surgical Hospital &National Center of Liver Cancer, Second Military Medical University, Shanghai 200438, China.
Yan C; School of Pharmacy, Shanghai Jiao Tong University, Shanghai 200240, China.
Su C; Department of Molecular Oncology, Eastern Hepatobiliary Surgical Hospital &National Center of Liver Cancer, Second Military Medical University, Shanghai 200438, China.
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Źródło:
Scientific reports [Sci Rep] 2017 Jan 24; Vol. 7, pp. 41254. Date of Electronic Publication: 2017 Jan 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/drug effects
Cell Cycle Checkpoints*/drug effects
Signal Transduction*/drug effects
Antineoplastic Combined Chemotherapy Protocols/*therapeutic use
Breast Neoplasms/*drug therapy
Lactones/*therapeutic use
Sesquiterpenes/*therapeutic use
14-3-3 Proteins/metabolism ; Animals ; Antineoplastic Combined Chemotherapy Protocols/pharmacology ; Breast Neoplasms/pathology ; Cell Line, Tumor ; Cyclic AMP/pharmacology ; Female ; Humans ; Lactones/chemistry ; Lactones/pharmacology ; Mice, Inbred BALB C ; Mice, Nude ; Oils, Volatile/pharmacology ; Oils, Volatile/therapeutic use ; Protein Interaction Maps/drug effects ; Proto-Oncogene Proteins c-akt/metabolism ; Proto-Oncogene Proteins c-myc/metabolism ; Sesquiterpenes/chemistry ; Sesquiterpenes/pharmacology ; Tumor Suppressor Protein p53/metabolism ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Stattic Enhances Radiosensitivity and Reduces Radio-Induced Migration and Invasion in HCC Cell Lines through an Apoptosis Pathway.
Autorzy:
Xu G; Department of Radiation Oncology, Affiliated People's Hospital, Jiangsu University, Zhenjiang, Jiangsu 212002, China.
Zhu L; Department of Radiation Oncology, Affiliated People's Hospital, Jiangsu University, Zhenjiang, Jiangsu 212002, China.
Wang Y; Department of Radiation Oncology, Affiliated People's Hospital, Jiangsu University, Zhenjiang, Jiangsu 212002, China.
Shi Y; Department of Radiation Oncology, Affiliated People's Hospital, Jiangsu University, Zhenjiang, Jiangsu 212002, China.
Gong A; School of Medicine, Jiangsu University, Zhenjiang, Jiangsu 212013, China.
Wu C; Department of Radiation Oncology, Affiliated People's Hospital, Jiangsu University, Zhenjiang, Jiangsu 212002, China.
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Źródło:
BioMed research international [Biomed Res Int] 2017; Vol. 2017, pp. 1832494. Date of Electronic Publication: 2017 Oct 26.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Carcinoma, Hepatocellular/*drug therapy
Cell Movement/*drug effects
Cyclic S-Oxides/*pharmacology
Liver Neoplasms/*drug therapy
Neoplasm Invasiveness/*prevention & control
Radiation Tolerance/*drug effects
Apoptosis Regulatory Proteins/metabolism ; Carcinogenesis/drug effects ; Carcinogenesis/metabolism ; Carcinogenesis/pathology ; Carcinoma, Hepatocellular/metabolism ; Carcinoma, Hepatocellular/pathology ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Hep G2 Cells ; Humans ; Liver Neoplasms/metabolism ; Liver Neoplasms/pathology ; STAT3 Transcription Factor/antagonists & inhibitors ; Signal Transduction/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
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