Informacja

Drogi użytkowniku, aplikacja do prawidłowego działania wymaga obsługi JavaScript. Proszę włącz obsługę JavaScript w Twojej przeglądarce.

Wyszukujesz frazę ""Wieser R"" wg kryterium: Autor


Wyświetlanie 1-5 z 5
Tytuł:
Overexpression of primary microRNA 221/222 in acute myeloid leukemia.
Autorzy:
Rommer A; Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090 Vienna, Austria.
Steinleitner K
Hackl H
Schneckenleithner C
Engelmann M
Scheideler M
Vlatkovic I
Kralovics R
Cerny-Reiterer S
Valent P
Sill H
Wieser R
Pokaż więcej
Źródło:
BMC cancer [BMC Cancer] 2013 Jul 29; Vol. 13, pp. 364. Date of Electronic Publication: 2013 Jul 29.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Leukemia, Myeloid, Acute/*genetics
MicroRNAs/*biosynthesis
Adolescent ; Adult ; Aged ; Female ; Gene Expression Regulation, Leukemic ; Humans ; Leukemia, Myeloid, Acute/metabolism ; Male ; MicroRNAs/genetics ; Middle Aged ; Oligonucleotide Array Sequence Analysis ; Reverse Transcriptase Polymerase Chain Reaction ; Transfection ; Young Adult
Czasopismo naukowe
Tytuł:
Molecular and genetic alterations associated with therapy resistance and relapse of acute myeloid leukemia.
Autorzy:
Hackl H; Division of Bioinformatics, Biocenter, Medical University of Innsbruck, Innrain 80, 6020, Innsbruck, Austria.
Astanina K; Department of Medicine I and Comprehensive Cancer Center, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Wien, Austria.
Wieser R; Department of Medicine I and Comprehensive Cancer Center, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Wien, Austria. rotraud.wieser@meduniwien.ac.at.
Pokaż więcej
Źródło:
Journal of hematology & oncology [J Hematol Oncol] 2017 Feb 20; Vol. 10 (1), pp. 51. Date of Electronic Publication: 2017 Feb 20.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Leukemia, Myeloid, Acute/*pathology
Clone Cells ; Disease Progression ; Drug Resistance, Neoplasm/genetics ; Humans ; Leukemia, Myeloid, Acute/genetics ; Recurrence
Czasopismo naukowe
Tytuł:
SOCS2 is part of a highly prognostic 4-gene signature in AML and promotes disease aggressiveness.
Autorzy:
Nguyen CH; Department of Medicine I, Division of Oncology, Medical University of Vienna, Vienna, Austria.; Comprehensive Cancer Center, Vienna, Austria.
Glüxam T; Department of Medicine I, Division of Oncology, Medical University of Vienna, Vienna, Austria.; Comprehensive Cancer Center, Vienna, Austria.
Schlerka A; Department of Medicine I, Division of Oncology, Medical University of Vienna, Vienna, Austria.; Comprehensive Cancer Center, Vienna, Austria.
Bauer K; Department of Medicine I, Division of Oncology, Medical University of Vienna, Vienna, Austria.; Comprehensive Cancer Center, Vienna, Austria.; Institute of Science and Technology Austria, Vienna, Austria.
Grandits AM; Department of Medicine I, Division of Oncology, Medical University of Vienna, Vienna, Austria.; Comprehensive Cancer Center, Vienna, Austria.
Hackl H; Division of Bioinformatics, Biocenter, Medical University of Innsbruck, Innsbruck, Austria.
Dovey O; Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Hinxton, UK.
Zöchbauer-Müller S; Department of Medicine I, Division of Oncology, Medical University of Vienna, Vienna, Austria.; Comprehensive Cancer Center, Vienna, Austria.
Cooper JL; Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Hinxton, UK.
Vassiliou GS; Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Hinxton, UK.
Stoiber D; Ludwig Boltzmann Institute for Cancer Research, Vienna, Austria.; Institute of Pharmacology, Center for Physiology and Pharmacology, Medical University of Vienna, Vienna, Austria.
Wieser R; Department of Medicine I, Division of Oncology, Medical University of Vienna, Vienna, Austria. rotraud.wieser@meduniwien.ac.at.; Comprehensive Cancer Center, Vienna, Austria. rotraud.wieser@meduniwien.ac.at.
Heller G; Department of Medicine I, Division of Oncology, Medical University of Vienna, Vienna, Austria. .; Comprehensive Cancer Center, Vienna, Austria. .; Institute of Pharmacology and Toxicology, Department for Biomedical Sciences, University of Veterinary Medicine Vienna, Vienna, Austria. .
Pokaż więcej
Źródło:
Scientific reports [Sci Rep] 2019 Jun 24; Vol. 9 (1), pp. 9139. Date of Electronic Publication: 2019 Jun 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Leukemia, Myeloid, Acute/*genetics
Leukemia, Myeloid, Acute/*pathology
Suppressor of Cytokine Signaling Proteins/*genetics
Animals ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; Humans ; Leukemia, Myeloid, Acute/diagnosis ; Mice ; Neoplastic Stem Cells/pathology ; Prognosis
Czasopismo naukowe
Tytuł:
CGRP Signaling via CALCRL Increases Chemotherapy Resistance and Stem Cell Properties in Acute Myeloid Leukemia.
Autorzy:
Gluexam T; Division of Oncology, Department of Medicine I, Medical University of Vienna, Waehringer Guertel 18-20, 1090 Vienna, Austria.; Comprehensive Cancer Center, Spitalgasse 23, 1090 Vienna, Austria.
Grandits AM; Division of Oncology, Department of Medicine I, Medical University of Vienna, Waehringer Guertel 18-20, 1090 Vienna, Austria.; Comprehensive Cancer Center, Spitalgasse 23, 1090 Vienna, Austria.
Schlerka A; Division of Oncology, Department of Medicine I, Medical University of Vienna, Waehringer Guertel 18-20, 1090 Vienna, Austria.; Comprehensive Cancer Center, Spitalgasse 23, 1090 Vienna, Austria.
Nguyen CH; Division of Oncology, Department of Medicine I, Medical University of Vienna, Waehringer Guertel 18-20, 1090 Vienna, Austria.; Comprehensive Cancer Center, Spitalgasse 23, 1090 Vienna, Austria.
Etzler J; Division of Oncology, Department of Medicine I, Medical University of Vienna, Waehringer Guertel 18-20, 1090 Vienna, Austria.; Comprehensive Cancer Center, Spitalgasse 23, 1090 Vienna, Austria.
Finkes T; Division of Oncology, Department of Medicine I, Medical University of Vienna, Waehringer Guertel 18-20, 1090 Vienna, Austria.; Comprehensive Cancer Center, Spitalgasse 23, 1090 Vienna, Austria.
Fuchs M; Department I of Internal Medicine, Center for Integrated Oncology Aachen Bonn Cologne Duesseldorf, University of Cologne, Kerpener Str. 62, 50937 Cologne, Germany.
Scheid C; Department I of Internal Medicine, Center for Integrated Oncology Aachen Bonn Cologne Duesseldorf, University of Cologne, Kerpener Str. 62, 50937 Cologne, Germany.
Heller G; Division of Oncology, Department of Medicine I, Medical University of Vienna, Waehringer Guertel 18-20, 1090 Vienna, Austria.; Comprehensive Cancer Center, Spitalgasse 23, 1090 Vienna, Austria.
Hackl H; Institute of Bioinformatics, Biocenter, Medical University of Innsbruck, Innrain 80, 6020 Innsbruck, Austria.
Harrer N; Department for Cancer Research, Boehringer Ingelheim RCV GmbH & Co KG, Dr. Boehringer-Gasse 5-11, A-1121 Vienna, Austria.
Sill H; Division of Hematology, Medical University of Graz, Auenbruggerplatz 38, 8036 Graz, Austria.
Koller E; Third Medical Department, Hanusch Hospital, Heinrich Collinstrasse 30, 1140 Vienna, Austria.
Stoiber D; Institute of Pharmacology, Center for Physiology and Pharmacology, Medical University of Vienna, Waehringer Strasse 13A, 1090 Vienna, Austria.; Division Pharmacology, Department Pharmacology, Physiology and Microbiology, Karl Landsteiner University of Health Sciences, Dr.-Karl-Dorrek-Straße 30, 3500 Krems, Austria.
Sommergruber W; Department of Biotechnology, University of Applied Sciences, Helmut-Qualtinger-Gasse 2, 1030 Vienna, Austria.
Wieser R; Division of Oncology, Department of Medicine I, Medical University of Vienna, Waehringer Guertel 18-20, 1090 Vienna, Austria.; Comprehensive Cancer Center, Spitalgasse 23, 1090 Vienna, Austria.
Pokaż więcej
Źródło:
International journal of molecular sciences [Int J Mol Sci] 2019 Nov 20; Vol. 20 (23). Date of Electronic Publication: 2019 Nov 20.
Typ publikacji:
Journal Article
MeSH Terms:
Drug Resistance, Neoplasm*
Calcitonin Gene-Related Peptide/*metabolism
Calcitonin Receptor-Like Protein/*metabolism
Leukemia, Myeloid, Acute/*metabolism
Neoplastic Stem Cells/*metabolism
Animals ; Antibiotics, Antineoplastic/pharmacology ; Antibiotics, Antineoplastic/therapeutic use ; Apoptosis/drug effects ; Calcitonin Receptor-Like Protein/antagonists & inhibitors ; Cell Line, Tumor ; Daunorubicin/pharmacology ; Daunorubicin/therapeutic use ; Dipeptides/pharmacology ; Dipeptides/therapeutic use ; Female ; Hematopoietic Stem Cells/metabolism ; Humans ; Leukemia, Myeloid, Acute/drug therapy ; Male ; Mice ; Mice, Inbred C57BL ; Middle Aged ; Piperazines ; Quinazolines/pharmacology ; Quinazolines/therapeutic use ; Signal Transduction
Czasopismo naukowe
    Wyświetlanie 1-5 z 5

    Ta witryna wykorzystuje pliki cookies do przechowywania informacji na Twoim komputerze. Pliki cookies stosujemy w celu świadczenia usług na najwyższym poziomie, w tym w sposób dostosowany do indywidualnych potrzeb. Korzystanie z witryny bez zmiany ustawień dotyczących cookies oznacza, że będą one zamieszczane w Twoim komputerze. W każdym momencie możesz dokonać zmiany ustawień dotyczących cookies