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Wyszukujesz frazę ""Wiskott-Aldrich Syndrome Protein Family"" wg kryterium: Temat


Tytuł :
Cell-substrate adhesion drives Scar/WAVE activation and phosphorylation by a Ste20-family kinase, which controls pseudopod lifetime.
Autorzy :
Singh SP; CRUK Beatson Institute, Glasgow, United Kingdom.
Thomason PA; CRUK Beatson Institute, Glasgow, United Kingdom.
Lilla S; CRUK Beatson Institute, Glasgow, United Kingdom.
Schaks M; Zoological Institute, Technische Universität Braunschweig, Braunschweig, Germany & Cell Biology, Helmholtz Centre for Infection Research, Braunschweig, Germany.
Tang Q; Brandeis University, Waltham, Massachusetts, United States of America.
Goode BL; Brandeis University, Waltham, Massachusetts, United States of America.
Machesky LM; CRUK Beatson Institute, Glasgow, United Kingdom.
Rottner K; Zoological Institute, Technische Universität Braunschweig, Braunschweig, Germany & Cell Biology, Helmholtz Centre for Infection Research, Braunschweig, Germany.
Insall RH; CRUK Beatson Institute, Glasgow, United Kingdom.; Institute of Cancer Sciences, University of Glasgow, Glasgow, United Kingdom.
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Źródło :
PLoS biology [PLoS Biol] 2020 Aug 03; Vol. 18 (8), pp. e3000774. Date of Electronic Publication: 2020 Aug 03 (Print Publication: 2020).
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Dictyostelium/*genetics
MAP Kinase Kinase Kinase 3/*genetics
Protozoan Proteins/*genetics
Pseudopodia/*metabolism
Wiskott-Aldrich Syndrome Protein Family/*genetics
Animals ; CRISPR-Cas Systems ; Cell Adhesion ; Cell Line, Tumor ; Chemotaxis/genetics ; Dictyostelium/metabolism ; Dictyostelium/ultrastructure ; Gene Editing/methods ; Gene Expression Regulation ; MAP Kinase Kinase Kinase 3/metabolism ; Melanocytes/metabolism ; Melanocytes/ultrastructure ; Mice ; Mitogen-Activated Protein Kinase 1/genetics ; Mitogen-Activated Protein Kinase 1/metabolism ; Mutation ; NIH 3T3 Cells ; Phenotype ; Phosphorylation ; Ploidies ; Protozoan Proteins/metabolism ; Pseudopodia/genetics ; Pseudopodia/ultrastructure ; Wiskott-Aldrich Syndrome Protein Family/metabolism
Czasopismo naukowe
Tytuł :
MicroRNA-93 targets WASF3 and functions as a metastasis suppressor in breast cancer.
Autorzy :
Shibuya N; Division of Molecular and Cellular Biology, Kobe University Graduate School of Medicine, Kobe, Japan.; Division of Gastrointestinal Surgery, Kobe University Graduate School of Medicine, Kobe, Japan.
Kakeji Y; Division of Gastrointestinal Surgery, Kobe University Graduate School of Medicine, Kobe, Japan.
Shimono Y; Division of Molecular and Cellular Biology, Kobe University Graduate School of Medicine, Kobe, Japan.; Department of Biochemistry, Fujita Health University School of Medicine, Toyoake, Japan.
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Źródło :
Cancer science [Cancer Sci] 2020 Jun; Vol. 111 (6), pp. 2093-2103. Date of Electronic Publication: 2020 May 29.
Typ publikacji :
Journal Article
MeSH Terms :
Breast Neoplasms/*pathology
Gene Expression Regulation, Neoplastic/*genetics
MicroRNAs/*metabolism
Neoplastic Stem Cells/*pathology
Wiskott-Aldrich Syndrome Protein Family/*metabolism
Animals ; Breast Neoplasms/genetics ; Female ; Heterografts ; Humans ; Mice ; MicroRNAs/genetics ; Neoplasm Invasiveness/genetics ; Neoplasm Metastasis ; Neoplastic Stem Cells/metabolism ; Wiskott-Aldrich Syndrome Protein Family/genetics
Czasopismo naukowe
Tytuł :
Wiskott-Aldrich syndrome protein senses irradiation-induced DNA damage to coordinate the cell-protective Golgi dispersal response in human T and B lymphocytes.
Autorzy :
Wen KK; Division of Pediatric Hematology-Oncology, University of Iowa Carver College of Medicine, and the Stead Family University of Iowa Children's Hospital, Iowa City, Iowa.
Han SS; Division of Pediatric Hematology-Oncology, University of Iowa Carver College of Medicine, and the Stead Family University of Iowa Children's Hospital, Iowa City, Iowa.
Vyas YM; Division of Pediatric Hematology-Oncology, University of Iowa Carver College of Medicine, and the Stead Family University of Iowa Children's Hospital, Iowa City, Iowa. Electronic address: .
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Źródło :
The Journal of allergy and clinical immunology [J Allergy Clin Immunol] 2020 Jan; Vol. 145 (1), pp. 324-334. Date of Electronic Publication: 2019 Oct 09.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
B-Lymphocytes/*immunology
DNA Damage/*immunology
Golgi Apparatus/*immunology
Signal Transduction/*immunology
T-Lymphocytes/*immunology
Wiskott-Aldrich Syndrome Protein Family/*immunology
DNA Damage/genetics ; DNA-Activated Protein Kinase/genetics ; DNA-Activated Protein Kinase/immunology ; Golgi Apparatus/genetics ; Humans ; Membrane Proteins/genetics ; Membrane Proteins/immunology ; Wiskott-Aldrich Syndrome/genetics ; Wiskott-Aldrich Syndrome/immunology ; Wiskott-Aldrich Syndrome Protein Family/genetics
Czasopismo naukowe
Tytuł :
Orai1 Promotes Osteosarcoma Metastasis by Activating the Ras-Rac1-WAVE2 Signaling Pathway.
Autorzy :
Guo Y; Department of Spinal Surgery, Hong Hui Hospital, Xi'an Jiao Tong University, Xi'an, Shaanxi, China (mainland).
Zhu J; Department of Spinal Surgery, Hong Hui Hospital, Xi'an Jiao Tong University, Xi'an, Shaanxi, China (mainland).
Wang X; Department of Spinal Surgery, Hong Hui Hospital, Xi'an Jiao Tong University, Xi'an, Shaanxi, China (mainland).
Li R; Department of Spinal Surgery, Hong Hui Hospital, Xi'an Jiao Tong University, Xi'an, Shaanxi, China (mainland).
Jiang K; Department of Spinal Surgery, Hong Hui Hospital, Xi'an Jiao Tong University, Xi'an, Shaanxi, China (mainland).
Chen S; Department of Emergency Medicine, Hong Hui Hospital, Xi'an Jiao Tong University, Xi'an, Shaanxi, China (mainland).
Fan J; Department of Orthopedics, Hong Hui Hospital, Xi'an Jiao Tong University, Xi'an, Shaanxi, China (mainland).
Xue L; Department of Spinal Surgery, Hong Hui Hospital, Xi'an Jiao Tong University, Xi'an, Shaanxi, China (mainland).
Hao D; Department of Spinal Surgery, Hong Hui Hospital, Xi'an Jiao Tong University, Xi'an, Shaanxi, China (mainland).
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Źródło :
Medical science monitor : international medical journal of experimental and clinical research [Med Sci Monit] 2019 Dec 04; Vol. 25, pp. 9227-9236. Date of Electronic Publication: 2019 Dec 04.
Typ publikacji :
Journal Article
MeSH Terms :
Bone Neoplasms/*metabolism
ORAI1 Protein/*metabolism
Osteosarcoma/*metabolism
Wiskott-Aldrich Syndrome Protein Family/*metabolism
rac1 GTP-Binding Protein/*metabolism
ras Proteins/*metabolism
Antineoplastic Agents/pharmacology ; Bone Neoplasms/drug therapy ; Bone Neoplasms/genetics ; Bone Neoplasms/pathology ; Cell Adhesion/genetics ; Cell Line, Tumor ; Cell Movement/genetics ; Humans ; Neoplasm Invasiveness ; Neoplasm Metastasis ; ORAI1 Protein/genetics ; Osteosarcoma/drug therapy ; Osteosarcoma/genetics ; Osteosarcoma/pathology ; RNA, Small Interfering/pharmacology ; Signal Transduction ; Wiskott-Aldrich Syndrome Protein Family/genetics ; rac1 GTP-Binding Protein/genetics ; ras Proteins/genetics
Czasopismo naukowe
Tytuł :
Wasf3 Deficiency Reveals Involvement in Metastasis in a Mouse Model of Breast Cancer.
Autorzy :
Qin H; Georgia Cancer Center, Augusta University, Augusta, Georgia.
Lu S; Georgia Cancer Center, Augusta University, Augusta, Georgia.
Thangaraju M; Department of Biochemistry and Molecular Biology, Augusta University, Augusta, Georgia.
Cowell JK; Georgia Cancer Center, Augusta University, Augusta, Georgia. Electronic address: .
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Źródło :
The American journal of pathology [Am J Pathol] 2019 Dec; Vol. 189 (12), pp. 2450-2458. Date of Electronic Publication: 2019 Sep 19.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Gene Expression Regulation, Neoplastic*
Breast Neoplasms/*pathology
Lung Neoplasms/*secondary
Wiskott-Aldrich Syndrome Protein Family/*metabolism
Animals ; Breast Neoplasms/genetics ; Breast Neoplasms/metabolism ; Cell Movement ; Cohort Studies ; Disease Models, Animal ; Female ; Humans ; Lung Neoplasms/genetics ; Lung Neoplasms/metabolism ; Mice ; Mice, Inbred C57BL ; Neoplasm Invasiveness ; Signal Transduction ; Tumor Cells, Cultured ; Wiskott-Aldrich Syndrome Protein Family/genetics
Czasopismo naukowe
Tytuł :
Cell confinement reveals a branched-actin independent circuit for neutrophil polarity.
Autorzy :
Graziano BR; Cardiovascular Research Institute and Department of Biochemistry and Biophysics, University of California, San Francisco, California, United States of America.
Town JP; Cardiovascular Research Institute and Department of Biochemistry and Biophysics, University of California, San Francisco, California, United States of America.
Sitarska E; Cell Biology and Biophysics Unit, European Molecular Biology Laboratory, Heidelberg, Germany.
Nagy TL; Cardiovascular Research Institute and Department of Biochemistry and Biophysics, University of California, San Francisco, California, United States of America.
Fošnarič M; Laboratory of Clinical Biophysics, Faculty of Medicine, University of Ljubljana, Slovenia.
Penič S; Department of Theoretical Electrotechnics, Mathematics and Physics, Faculty of Electrical Engineering, University of Ljubljana, Slovenia.
Iglič A; Laboratory of Clinical Biophysics, Faculty of Medicine, University of Ljubljana, Slovenia.; Department of Theoretical Electrotechnics, Mathematics and Physics, Faculty of Electrical Engineering, University of Ljubljana, Slovenia.
Kralj-Iglič V; Faculty of Health Sciences, University of Ljubljana, Slovenia.
Gov NS; Department of Chemical and Biological Physics, Weizmann Institute, Rehovot, Israel.
Diz-Muñoz A; Cell Biology and Biophysics Unit, European Molecular Biology Laboratory, Heidelberg, Germany.
Weiner OD; Cardiovascular Research Institute and Department of Biochemistry and Biophysics, University of California, San Francisco, California, United States of America.
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Źródło :
PLoS biology [PLoS Biol] 2019 Oct 10; Vol. 17 (10), pp. e3000457. Date of Electronic Publication: 2019 Oct 10 (Print Publication: 2019).
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms :
Actin-Related Protein 2-3 Complex/*genetics
Actins/*genetics
Cell Polarity/*genetics
Chemotaxis/*genetics
Wiskott-Aldrich Syndrome Protein Family/*genetics
Actin-Related Protein 2-3 Complex/metabolism ; Actins/metabolism ; Biomechanical Phenomena ; CRISPR-Cas Systems ; Cell Adhesion/drug effects ; Cell Membrane/drug effects ; Cell Membrane/metabolism ; Cell Membrane/ultrastructure ; Cell Polarity/drug effects ; Chemotactic Factors/pharmacology ; Chemotaxis/drug effects ; Gene Editing ; Gene Expression Regulation ; HEK293 Cells ; HL-60 Cells ; Humans ; Microscopy, Atomic Force ; N-Formylmethionine Leucyl-Phenylalanine/pharmacology ; Pseudopodia/drug effects ; Pseudopodia/metabolism ; Pseudopodia/ultrastructure ; Signal Transduction ; Surface Properties ; Wiskott-Aldrich Syndrome Protein Family/deficiency
Czasopismo naukowe
Tytuł :
HEM1 deficiency disrupts mTORC2 and F-actin control in inherited immunodysregulatory disease.
Autorzy :
Cook SA; Molecular Development of the Immune System Section, Laboratory of Immune System Biology, and Clinical Genomics Program, National Institute of Allergy and Infectious Diseases (NIAID), National Institutes of Health (NIH), Bethesda, MD, USA.
Comrie WA; Molecular Development of the Immune System Section, Laboratory of Immune System Biology, and Clinical Genomics Program, National Institute of Allergy and Infectious Diseases (NIAID), National Institutes of Health (NIH), Bethesda, MD, USA.; Neomics Pharmaceuticals, LLC, Gaithersburg, MD, USA.
Poli MC; Department of Pediatrics, Baylor College of Medicine, Houston, TX, USA.; Section of Pediatric Immunology, Allergy, and Retrovirology, Texas Children's Hospital, Houston, TX, USA.; Program of Immunogenetics and Translational Immunology, Instituto de Ciencias e Innovación en Medicina, Facultad de Medicina, Clínica Alemana-Universidad del Desarrollo, Santiago, Chile.
Similuk M; Division of Intramural Research, NIAID, NIH, Bethesda, MD, USA.
Oler AJ; Bioinformatics and Computational Biosciences Branch, Office of Cyber Infrastructure and Computational Biology, NIAID, NIH, Bethesda, MD, USA.
Faruqi AJ; Molecular Development of the Immune System Section, Laboratory of Immune System Biology, and Clinical Genomics Program, National Institute of Allergy and Infectious Diseases (NIAID), National Institutes of Health (NIH), Bethesda, MD, USA.
Kuhns DB; Neutrophil Monitoring Laboratory, Leidos Biomedical Research, Inc., Frederick National Laboratory for Cancer Research, Frederick, MD, USA.
Yang S; Roy J. Carver Department of Biochemistry, Biophysics and Molecular Biology, Iowa State University, Ames, IA, USA.
Vargas-Hernández A; Department of Pediatrics, Baylor College of Medicine, Houston, TX, USA.; Section of Pediatric Immunology, Allergy, and Retrovirology, Texas Children's Hospital, Houston, TX, USA.
Carisey AF; Department of Pediatrics, Baylor College of Medicine, Houston, TX, USA.; Section of Pediatric Immunology, Allergy, and Retrovirology, Texas Children's Hospital, Houston, TX, USA.
Fournier B; Laboratory of Lymphocyte Activation and Susceptibility to EBV, INSERM UMR 1163, Paris, France.; University Paris Descartes Sorbonne Paris Cité, Institut des Maladies Génétiques-IMAGINE, Paris, France.
Anderson DE; Advanced Mass Spectrometry Facility, National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK), NIH, Bethesda, MD, USA.
Price S; Laboratory of Clinical Immunology and Microbiology, NIAID, NIH, Bethesda, MD, USA.
Smelkinson M; Biological Imaging Section, Research Technologies Branch, NIAID, NIH, Bethesda, MD, USA.
Abou Chahla W; Department of Pediatric Hematology, Jeanne de Flandre Hospital, Centre Hospitalier Universitaire (CHU), Lille, France.
Forbes LR; Department of Pediatrics, Baylor College of Medicine, Houston, TX, USA.; Section of Pediatric Immunology, Allergy, and Retrovirology, Texas Children's Hospital, Houston, TX, USA.
Mace EM; Department of Pediatrics, Columbia University Irving Medical Center, New York, NY, USA.
Cao TN; Department of Pediatrics, Baylor College of Medicine, Houston, TX, USA.; Section of Pediatric Immunology, Allergy, and Retrovirology, Texas Children's Hospital, Houston, TX, USA.
Coban-Akdemir ZH; Baylor-Hopkins Center for Mendelian Genomics, Houston, TX, USA.; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA.
Jhangiani SN; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA.; Human Genome Sequencing Center, Baylor College of Medicine, Houston, TX, USA.
Muzny DM; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA.; Human Genome Sequencing Center, Baylor College of Medicine, Houston, TX, USA.
Gibbs RA; Baylor-Hopkins Center for Mendelian Genomics, Houston, TX, USA.; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA.; Human Genome Sequencing Center, Baylor College of Medicine, Houston, TX, USA.
Lupski JR; Baylor-Hopkins Center for Mendelian Genomics, Houston, TX, USA.; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA.; Human Genome Sequencing Center, Baylor College of Medicine, Houston, TX, USA.
Orange JS; Department of Pediatrics, Columbia University Irving Medical Center, New York, NY, USA.
Cuvelier GDE; Section of Pediatric Hematology/Oncology/BMT, CancerCare Manitoba, University of Manitoba, Winnipeg, MB, Canada.
Al Hassani M; Sheikh Khalifa Medical City, Abu Dhabi Healthcare Company (SEHA), Abu Dhabi, United Arab Emirates.
Al Kaabi N; Sheikh Khalifa Medical City, Abu Dhabi Healthcare Company (SEHA), Abu Dhabi, United Arab Emirates.
Al Yafei Z; Sheikh Khalifa Medical City, Abu Dhabi Healthcare Company (SEHA), Abu Dhabi, United Arab Emirates.
Jyonouchi S; Division of Allergy and Immunology, Children's Hospital of Philadelphia, Philadelphia, PA, USA.; Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.
Raje N; Division of Allergy, Immunology, Pulmonary, and Sleep Medicine, Children's Mercy Hospital, Kansas City, MO, USA.; Department of Internal Medicine and Pediatrics, University of Missouri Kansas City, Kansas City, MO, USA.
Caldwell JW; Section of Pulmonary, Critical Care, Allergy and Immunological Diseases, Wake Forest University School of Medicine, Winston-Salem, NC, USA.
Huang Y; Department of Pathology and Laboratory Medicine, Children's Hospital of Philadelphia Research Institute and Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.; Department of Pathology, Anatomy, and Cell Biology, Thomas Jefferson University, Philadelphia, PA, USA.
Burkhardt JK; Department of Pathology and Laboratory Medicine, Children's Hospital of Philadelphia Research Institute and Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.
Latour S; Laboratory of Lymphocyte Activation and Susceptibility to EBV, INSERM UMR 1163, Paris, France.; University Paris Descartes Sorbonne Paris Cité, Institut des Maladies Génétiques-IMAGINE, Paris, France.
Chen B; Roy J. Carver Department of Biochemistry, Biophysics and Molecular Biology, Iowa State University, Ames, IA, USA.
ElGhazali G; Sheikh Khalifa Medical City, Abu Dhabi Healthcare Company (SEHA), Abu Dhabi, United Arab Emirates.
Rao VK; Laboratory of Clinical Immunology and Microbiology, NIAID, NIH, Bethesda, MD, USA.
Chinn IK; Department of Pediatrics, Baylor College of Medicine, Houston, TX, USA.; Section of Pediatric Immunology, Allergy, and Retrovirology, Texas Children's Hospital, Houston, TX, USA.
Lenardo MJ; Molecular Development of the Immune System Section, Laboratory of Immune System Biology, and Clinical Genomics Program, National Institute of Allergy and Infectious Diseases (NIAID), National Institutes of Health (NIH), Bethesda, MD, USA. .
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Źródło :
Science (New York, N.Y.) [Science] 2020 Jul 10; Vol. 369 (6500), pp. 202-207.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Actins/*metabolism
Cytokines/*biosynthesis
Immunologic Deficiency Syndromes/*genetics
Lymphoproliferative Disorders/*genetics
Mechanistic Target of Rapamycin Complex 2/*metabolism
Membrane Proteins/*physiology
ADP-Ribosylation Factor 1/metabolism ; CD4-Positive T-Lymphocytes/immunology ; Cell Proliferation ; Humans ; Immunologic Deficiency Syndromes/immunology ; Lymphoproliferative Disorders/immunology ; Membrane Proteins/genetics ; Pedigree ; Phosphorylation ; Wiskott-Aldrich Syndrome Protein Family/chemistry ; Wiskott-Aldrich Syndrome Protein Family/metabolism
Czasopismo naukowe
Tytuł :
Leucine-rich repeat kinase-2 (LRRK2) modulates microglial phenotype and dopaminergic neurodegeneration.
Autorzy :
Dwyer Z; Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada. Electronic address: .
Rudyk C; Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada.
Thompson A; Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada.
Farmer K; Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada.
Fenner B; Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada.
Fortin T; Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada.
Derksen A; Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada.
Sun H; Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada.
Hayley S; Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada. Electronic address: .
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Corporate Authors :
CLINT (Canadian LRRK2 in inflammation team); Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada.
Źródło :
Neurobiology of aging [Neurobiol Aging] 2020 Jul; Vol. 91, pp. 45-55. Date of Electronic Publication: 2020 Feb 27.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Phenotype*
Dopaminergic Neurons/*pathology
Leucine-Rich Repeat Serine-Threonine Protein Kinase-2/*physiology
Microglia/*pathology
Nerve Degeneration/*genetics
Parkinson Disease/*genetics
Parkinson Disease/*pathology
Animals ; CX3C Chemokine Receptor 1/genetics ; CX3C Chemokine Receptor 1/metabolism ; Inflammation/genetics ; Mice, Knockout ; Molecular Targeted Therapy ; Oxidative Stress/genetics ; Parkinson Disease/etiology ; Parkinson Disease/therapy ; Up-Regulation/genetics ; Wiskott-Aldrich Syndrome Protein Family/genetics ; Wiskott-Aldrich Syndrome Protein Family/metabolism
Czasopismo naukowe
Tytuł :
The Wave complex controls epidermal morphogenesis and proliferation by suppressing Wnt-Sox9 signaling.
Autorzy :
Cohen J; Department of Cell and Developmental Biology, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Raviv S; Department of Cell and Developmental Biology, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Adir O; Department of Cell and Developmental Biology, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Padmanabhan K; Department of Cell and Developmental Biology, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Soffer A; Department of Cell and Developmental Biology, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.
Luxenburg C; Department of Cell and Developmental Biology, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel .
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Źródło :
The Journal of cell biology [J Cell Biol] 2019 Apr 01; Vol. 218 (4), pp. 1390-1406. Date of Electronic Publication: 2019 Mar 13.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Proliferation*
Wnt Signaling Pathway*
Adaptor Proteins, Signal Transducing/*metabolism
Cytoskeletal Proteins/*metabolism
Epidermis/*metabolism
Keratinocytes/*metabolism
SOX9 Transcription Factor/*metabolism
Wiskott-Aldrich Syndrome Protein Family/*metabolism
Adaptor Proteins, Signal Transducing/genetics ; Animals ; Cells, Cultured ; Cytoskeletal Proteins/genetics ; Epidermis/abnormalities ; Gene Expression Regulation, Developmental ; Gene Silencing ; Hair Follicle/abnormalities ; Hair Follicle/metabolism ; Mice ; Morphogenesis ; Phosphorylation ; SOX9 Transcription Factor/genetics ; Wiskott-Aldrich Syndrome Protein Family/genetics ; beta Catenin/metabolism
Czasopismo naukowe
Tytuł :
PCARE and WASF3 regulate ciliary F-actin assembly that is required for the initiation of photoreceptor outer segment disk formation.
Autorzy :
Corral-Serrano JC; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.; Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Lamers IJC; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.; Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
van Reeuwijk J; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.; Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Duijkers L; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Hoogendoorn ADM; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Yildirim A; Institute of Molecular Physiology, Johannes Gutenberg University of Mainz, 55099 Mainz, Germany.
Argyrou N; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.; Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Ruigrok RAA; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Letteboer SJF; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Butcher R; Department of Ophthalmology, Ocular Genomics Institute, Massachusetts Eye and Ear, Harvard Medical School, Boston, MA 02114.
van Essen MD; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Sakami S; Department of Pharmacology, School of Medicine, Case Western Reserve University, Cleveland, OH 44106.
van Beersum SEC; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Palczewski K; Department of Pharmacology, School of Medicine, Case Western Reserve University, Cleveland, OH 44106; .
Cheetham ME; UCL Institute of Ophthalmology, University College London, London EC1V 9EL, United Kingdom.
Liu Q; Department of Ophthalmology, Ocular Genomics Institute, Massachusetts Eye and Ear, Harvard Medical School, Boston, MA 02114.
Boldt K; Center of Ophthalmology, Institute for Ophthalmic Research, University of Tübingen, 72076 Tübingen, Germany.
Wolfrum U; Institute of Molecular Physiology, Johannes Gutenberg University of Mainz, 55099 Mainz, Germany.
Ueffing M; Center of Ophthalmology, Institute for Ophthalmic Research, University of Tübingen, 72076 Tübingen, Germany.
Garanto A; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.; Donders Institute for Cognitive Neuroscience, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Roepman R; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands; .; Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
Collin RWJ; Department of Human Genetics, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.; Donders Institute for Cognitive Neuroscience, Radboud University Medical Center, 6525 GA, Nijmegen, The Netherlands.
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Źródło :
Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2020 May 05; Vol. 117 (18), pp. 9922-9931. Date of Electronic Publication: 2020 Apr 20.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cilia/*genetics
Cone-Rod Dystrophies/*genetics
Eye Proteins/*genetics
Rod Cell Outer Segment/*metabolism
Wiskott-Aldrich Syndrome Protein Family/*genetics
Actin-Related Protein 2-3 Complex/genetics ; Actins/genetics ; Animals ; Cilia/pathology ; Cone-Rod Dystrophies/pathology ; Disease Models, Animal ; Gene Expression Regulation/genetics ; Humans ; Mice ; Mice, Knockout ; RNA, Small Interfering/genetics ; Retinal Cone Photoreceptor Cells/metabolism ; Retinal Cone Photoreceptor Cells/pathology ; Rod Cell Outer Segment/pathology
Czasopismo naukowe
Tytuł :
WASF3 Knockdown Sensitizes Gastric Cancer Cells to Oxaliplatin by Inhibiting ATG12-Mediated Autophagy.
Autorzy :
Nie Y; Department of Radiation Oncology and Medical Oncology, Zhongnan Hospital of Wuhan University, Hubei Cancer Clinical Study Center, Hubei Key Laboratory of Tumor Biological Behaviors, Wuhan, China; Department of Gastrointestinal Medical Oncology, Hubei Cancer Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Liang X; Department of Gastrointestinal Medical Oncology, Hubei Cancer Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Liu S; Department of Gastrointestinal Medical Oncology, Hubei Cancer Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Guo F; Department of Pathology, Hubei Cancer Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Fang N; Department of Pathology, Hubei Cancer Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhou F; Department of Radiation Oncology and Medical Oncology, Zhongnan Hospital of Wuhan University, Hubei Cancer Clinical Study Center, Hubei Key Laboratory of Tumor Biological Behaviors, Wuhan, China. Electronic address: .
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Źródło :
The American journal of the medical sciences [Am J Med Sci] 2020 May; Vol. 359 (5), pp. 287-295. Date of Electronic Publication: 2020 Mar 13.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Autophagy*
Autophagy-Related Protein 12/*metabolism
Oxaliplatin/*pharmacology
Stomach Neoplasms/*metabolism
Wiskott-Aldrich Syndrome Protein Family/*genetics
Apoptosis ; Cell Line, Tumor ; Cell Movement ; Cell Proliferation ; Gene Expression Regulation, Neoplastic ; Gene Silencing ; Humans ; Neoplasm Invasiveness ; Neoplasm Metastasis ; RNA, Small Interfering/genetics ; Signal Transduction ; Stomach Neoplasms/genetics
Czasopismo naukowe
Tytuł :
Systematic Nanoscale Analysis of Endocytosis Links Efficient Vesicle Formation to Patterned Actin Nucleation.
Autorzy :
Mund M; Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), Meyerhofstrasse 1, 69117 Heidelberg, Germany.
van der Beek JA; Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), Meyerhofstrasse 1, 69117 Heidelberg, Germany.
Deschamps J; Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), Meyerhofstrasse 1, 69117 Heidelberg, Germany.
Dmitrieff S; Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), Meyerhofstrasse 1, 69117 Heidelberg, Germany.
Hoess P; Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), Meyerhofstrasse 1, 69117 Heidelberg, Germany; Collaboration for joint PhD degree between EMBL and Heidelberg University, Faculty of Biosciences.
Monster JL; Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), Meyerhofstrasse 1, 69117 Heidelberg, Germany.
Picco A; Department of Biochemistry and NCCR Chemical Biology, University of Geneva, Quai Ernest Ansermet 30, 1211 Geneva, Switzerland.
Nédélec F; Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), Meyerhofstrasse 1, 69117 Heidelberg, Germany.
Kaksonen M; Department of Biochemistry and NCCR Chemical Biology, University of Geneva, Quai Ernest Ansermet 30, 1211 Geneva, Switzerland.
Ries J; Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), Meyerhofstrasse 1, 69117 Heidelberg, Germany. Electronic address: .
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Źródło :
Cell [Cell] 2018 Aug 09; Vol. 174 (4), pp. 884-896.e17. Date of Electronic Publication: 2018 Jul 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Actin Cytoskeleton/*metabolism
Actins/*metabolism
Endocytosis/*physiology
Saccharomyces cerevisiae/*metabolism
Saccharomyces cerevisiae Proteins/*metabolism
Secretory Vesicles/*metabolism
Wiskott-Aldrich Syndrome Protein Family/*metabolism
Actins/chemistry ; Cell Membrane/metabolism ; Microscopy, Fluorescence ; Models, Theoretical ; Protein Conformation ; Wiskott-Aldrich Syndrome Protein Family/chemistry
Czasopismo naukowe
Tytuł :
WHIMP links the actin nucleation machinery to Src-family kinase signaling during protrusion and motility.
Autorzy :
Kabrawala S; Department of Molecular and Cell Biology, Institute for Systems Genomics, University of Connecticut, Storrs, Connecticut, United States of America.
Zimmer MD; Department of Molecular and Cell Biology, Institute for Systems Genomics, University of Connecticut, Storrs, Connecticut, United States of America.
Campellone KG; Department of Molecular and Cell Biology, Institute for Systems Genomics, University of Connecticut, Storrs, Connecticut, United States of America.
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Źródło :
PLoS genetics [PLoS Genet] 2020 Mar 20; Vol. 16 (3), pp. e1008694. Date of Electronic Publication: 2020 Mar 20 (Print Publication: 2020).
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Actin Cytoskeleton/*metabolism
Cell Movement/*physiology
Cell Surface Extensions/*physiology
Wiskott-Aldrich Syndrome Protein/*metabolism
src-Family Kinases/*metabolism
Actin-Related Protein 2-3 Complex/metabolism ; Actins/metabolism ; Animals ; Cell Line ; Endocytosis/physiology ; Male ; Mice ; Mice, Inbred C57BL ; NIH 3T3 Cells ; Protein Domains ; Signal Transduction ; Wiskott-Aldrich Syndrome Protein Family/metabolism
Czasopismo naukowe
Tytuł :
Asiatic Acid Interferes with Invasion and Proliferation of Breast Cancer Cells by Inhibiting WAVE3 Activation through PI3K/AKT Signaling Pathway.
Autorzy :
Gou XJ; Baoshan District Hospital of Integrated Traditional Chinese and Western Medicine of Shanghai, Shanghai 201999, China.
Bai HH; School of Pharmacy, Shaanxi University of Traditional Chinese Medicine, Xianyang, Shaanxi 712046, China.
Liu LW; Baoshan District Hospital of Integrated Traditional Chinese and Western Medicine of Shanghai, Shanghai 201999, China.
Chen HY; Baoshan District Hospital of Integrated Traditional Chinese and Western Medicine of Shanghai, Shanghai 201999, China.
Shi Q; Baoshan District Hospital of Integrated Traditional Chinese and Western Medicine of Shanghai, Shanghai 201999, China.
Chang LS; Baoshan District Hospital of Integrated Traditional Chinese and Western Medicine of Shanghai, Shanghai 201999, China.
Ding MM; Baoshan District Hospital of Integrated Traditional Chinese and Western Medicine of Shanghai, Shanghai 201999, China.
Shi Q; Baoshan District Hospital of Integrated Traditional Chinese and Western Medicine of Shanghai, Shanghai 201999, China.
Zhou MX; Baoshan District Hospital of Integrated Traditional Chinese and Western Medicine of Shanghai, Shanghai 201999, China.
Chen WL; Baoshan District Hospital of Integrated Traditional Chinese and Western Medicine of Shanghai, Shanghai 201999, China.
Zhang LM; School of Basic Sciences of Shanxi University of Chinese Medicine, Taiyuan, China.
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Źródło :
BioMed research international [Biomed Res Int] 2020 Feb 10; Vol. 2020, pp. 1874387. Date of Electronic Publication: 2020 Feb 10 (Print Publication: 2020).
Typ publikacji :
Journal Article
MeSH Terms :
Breast Neoplasms/*drug therapy
Cell Proliferation/*drug effects
Pentacyclic Triterpenes/*pharmacology
Wiskott-Aldrich Syndrome Protein Family/*genetics
Animals ; Breast Neoplasms/genetics ; Breast Neoplasms/pathology ; Cell Line, Tumor ; Female ; Gene Expression Regulation, Neoplastic/drug effects ; Humans ; Mice ; Neoplasm Invasiveness/genetics ; Neoplasm Invasiveness/pathology ; Oncogene Protein v-akt/genetics ; Phosphatidylinositol 3-Kinases/genetics ; Signal Transduction/drug effects ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
Tropomyosin-related kinase C (TrkC) enhances podocyte migration by ERK-mediated WAVE2 activation.
Autorzy :
Gromnitza S; Medizinische Klinik D, Universitätsklinikum Münster, Muenster, Germany.
Lepa C; Medizinische Klinik D, Universitätsklinikum Münster, Muenster, Germany.
Weide T; Medizinische Klinik D, Universitätsklinikum Münster, Muenster, Germany.
Schwab A; Institut für Physiologie II, Westfälische-Wilhelms-Universität Münster, Muenster, Germany.
Pavenstädt H; Medizinische Klinik D, Universitätsklinikum Münster, Muenster, Germany.
George B; Medizinische Klinik D, Universitätsklinikum Münster, Muenster, Germany.
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Źródło :
FASEB journal : official publication of the Federation of American Societies for Experimental Biology [FASEB J] 2018 Mar; Vol. 32 (3), pp. 1665-1676. Date of Electronic Publication: 2018 Jan 03.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*
MAP Kinase Signaling System*
Cytoskeleton/*metabolism
Podocytes/*metabolism
Receptor, trkC/*metabolism
Wiskott-Aldrich Syndrome Protein Family/*metabolism
Animals ; Cell Line, Transformed ; Cytoskeleton/genetics ; Mice ; Neurotrophin 3/biosynthesis ; Neurotrophin 3/genetics ; Podocytes/cytology ; Protein Domains ; Receptor, trkC/genetics ; Wiskott-Aldrich Syndrome Protein Family/genetics
Czasopismo naukowe
Tytuł :
WASP family proteins and formins compete in pseudopod- and bleb-based migration.
Autorzy :
Davidson AJ; Cancer Research UK Beatson Institute, Glasgow, Scotland, UK.
Amato C; Cancer Research UK Beatson Institute, Glasgow, Scotland, UK.
Thomason PA; Cancer Research UK Beatson Institute, Glasgow, Scotland, UK.
Insall RH; Cancer Research UK Beatson Institute, Glasgow, Scotland, UK .
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Źródło :
The Journal of cell biology [J Cell Biol] 2018 Feb 05; Vol. 217 (2), pp. 701-714. Date of Electronic Publication: 2017 Nov 30.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Movement*
Dictyostelium/*metabolism
Protozoan Proteins/*metabolism
Pseudopodia/*metabolism
Wiskott-Aldrich Syndrome Protein Family/*metabolism
Cells, Cultured ; Dictyostelium/cytology ; Mutation ; Protozoan Proteins/genetics ; Wiskott-Aldrich Syndrome Protein Family/genetics
Czasopismo naukowe
Tytuł :
SH3BP1-induced Rac-Wave2 pathway activation regulates cervical cancer cell migration, invasion, and chemoresistance to cisplatin.
Autorzy :
Wang J; Department of Oncology, The Second Xiangya Hospital, Central South University, Changsha, P.R. China.
Feng Y; Department of Oncology, The Second Xiangya Hospital, Central South University, Changsha, P.R. China.
Chen X; Department of Oncology, The Second Xiangya Hospital, Central South University, Changsha, P.R. China.
Du Z; Department of Oncology, The Second Xiangya Hospital, Central South University, Changsha, P.R. China.
Jiang S; Department of Oncology, The Second Xiangya Hospital, Central South University, Changsha, P.R. China.
Ma S; Department of Oncology, The Second Xiangya Hospital, Central South University, Changsha, P.R. China.
Zou W; Department of Oncology, The Second Xiangya Hospital, Central South University, Changsha, P.R. China.
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Źródło :
Journal of cellular biochemistry [J Cell Biochem] 2018 Feb; Vol. 119 (2), pp. 1733-1745. Date of Electronic Publication: 2017 Sep 18.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Drug Resistance, Neoplasm*
Cisplatin/*pharmacology
GTPase-Activating Proteins/*genetics
Uterine Cervical Neoplasms/*genetics
Wiskott-Aldrich Syndrome Protein Family/*genetics
rac1 GTP-Binding Protein/*genetics
Cell Line, Tumor ; Cell Movement ; Disease Progression ; Female ; GTPase-Activating Proteins/metabolism ; Gene Expression Regulation, Neoplastic ; HeLa Cells ; Humans ; Neoplasm Invasiveness ; Signal Transduction ; Survival Analysis ; Up-Regulation ; Uterine Cervical Neoplasms/metabolism ; Wiskott-Aldrich Syndrome Protein Family/metabolism ; rac1 GTP-Binding Protein/metabolism
Czasopismo naukowe
Tytuł :
WASP family proteins regulate the mobility of the B cell receptor during signaling activation.
Autorzy :
Rey-Suarez I; Biophysics Program, University of Maryland, College Park, MD, 20742, USA.; National Institute of Biomedical Imaging and Bioengineering, National Institutes of Health, Bethesda, MD, USA.
Wheatley BA; Department of Physics, University of Maryland, College Park, MD, 20742, USA.; Institute for Physical Science and Technology, University of Maryland, College Park, MD, 20742, USA.
Koo P; Department of Molecular & Cellular Biology, Harvard University, Cambridge, MA, 02138, USA.
Bhanja A; Department of Cell Biology & Molecular Genetics, University of Maryland, College Park, MD, 20742, USA.
Shu Z; Department of Cell Biology & Molecular Genetics, University of Maryland, College Park, MD, 20742, USA.; Division of Immunology, Children's Hospital of Chongqing Medical University, Chongqing, China.
Mochrie S; Department of Physics, Yale University, New Haven, CT, 06520, USA.
Song W; Department of Cell Biology & Molecular Genetics, University of Maryland, College Park, MD, 20742, USA.
Shroff H; National Institute of Biomedical Imaging and Bioengineering, National Institutes of Health, Bethesda, MD, USA.
Upadhyaya A; Biophysics Program, University of Maryland, College Park, MD, 20742, USA. .; Department of Physics, University of Maryland, College Park, MD, 20742, USA. .; Institute for Physical Science and Technology, University of Maryland, College Park, MD, 20742, USA. .
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Źródło :
Nature communications [Nat Commun] 2020 Jan 23; Vol. 11 (1), pp. 439. Date of Electronic Publication: 2020 Jan 23.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms :
B-Lymphocytes/*metabolism
Receptors, Antigen, B-Cell/*metabolism
Wiskott-Aldrich Syndrome Protein, Neuronal/*metabolism
Actin-Related Protein 2-3 Complex/metabolism ; Actins/metabolism ; Animals ; Antigens, CD19/metabolism ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Transgenic ; Receptors, Antigen, B-Cell/genetics ; Receptors, IgG/metabolism ; Signal Transduction ; Single Molecule Imaging ; Wiskott-Aldrich Syndrome Protein Family/metabolism ; Wiskott-Aldrich Syndrome Protein, Neuronal/genetics
Czasopismo naukowe
Tytuł :
Deletion of self-reactive CCR7- thymocytes in the absence of MHC expression on thymic epithelial cells.
Autorzy :
Wirasinha RC; Infection and Immunity Program, Monash Biomedicine Discovery Institute and Department of Biochemistry and Molecular Biology, Monash University, Melbourne, VIC, Australia.
Chan A; Infection and Immunity Program, Monash Biomedicine Discovery Institute and Department of Biochemistry and Molecular Biology, Monash University, Melbourne, VIC, Australia.
Yap JY; Department of Immunology and Infectious Disease, The John Curtin School of Medical Research, The Australian National University, Canberra, ACT, Australia.
Hu DY; Department of Immunology and Infectious Disease, The John Curtin School of Medical Research, The Australian National University, Canberra, ACT, Australia.
Teh CE; Walter and Eliza Hall Institute of Medical Research, Melbourne, VIC, Australia.; Department of Medical Biology, University of Melbourne, Melbourne, VIC, Australia.
Gray DHD; Walter and Eliza Hall Institute of Medical Research, Melbourne, VIC, Australia.; Department of Medical Biology, University of Melbourne, Melbourne, VIC, Australia.
Goodnow CC; Immunology Division, Garvan Institute of Medical Research, Sydney, NSW, Australia.; St Vincent's Clinical School, University of New South Wales, Sydney, NSW, Australia.
Daley SR; Infection and Immunity Program, Monash Biomedicine Discovery Institute and Department of Biochemistry and Molecular Biology, Monash University, Melbourne, VIC, Australia. .
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Źródło :
Cell death and differentiation [Cell Death Differ] 2019 Dec; Vol. 26 (12), pp. 2727-2739. Date of Electronic Publication: 2019 Apr 24.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Receptors, CCR7/*deficiency
Thymus Gland/*metabolism
Animals ; Apoptosis ; Epithelial Cells/cytology ; Epithelial Cells/immunology ; Epithelial Cells/metabolism ; Humans ; Mice ; Mice, Inbred C57BL ; Mice, Transgenic ; Receptors, Antigen, T-Cell, alpha-beta ; Receptors, CCR7/metabolism ; Thymus Gland/cytology ; Thymus Gland/immunology ; Wiskott-Aldrich Syndrome Protein Family
Czasopismo naukowe
Tytuł :
Arf GTPase interplay with Rho GTPases in regulation of the actin cytoskeleton.
Autorzy :
Singh V; Department of Pathology, University of Cambridge , Cambridge , UK.
Davidson AC; Department of Pathology, University of Cambridge , Cambridge , UK.
Hume PJ; Department of Pathology, University of Cambridge , Cambridge , UK.
Humphreys D; Department of Biomedical Science, University of Sheffield , Sheffield , UK.
Koronakis V; Department of Pathology, University of Cambridge , Cambridge , UK.
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Źródło :
Small GTPases [Small GTPases] 2019 Nov; Vol. 10 (6), pp. 411-418. Date of Electronic Publication: 2017 Nov 03.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms :
ADP-Ribosylation Factors/*metabolism
Actin Cytoskeleton/*metabolism
rho GTP-Binding Proteins/*metabolism
Animals ; Escherichia coli/metabolism ; Humans ; Salmonella/metabolism ; Wiskott-Aldrich Syndrome Protein Family/metabolism
Czasopismo naukowe

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