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Wyszukujesz frazę ""Wu, Junlu"" wg kryterium: Autor


Tytuł :
Correction to: Long non-coding RNA HOTTIP enhances IL-6 expression to potentiate immune escape of ovarian cancer cells by upregulating the expression of PD-L1 in neutrophils
Autorzy :
Shang, Anquan
Wang, Weiwei
Gu, Chenzheng
Chen, Chen
Zeng, Bingjie
Yang, Yibao
Ji, Ping
Sun, Junjun
Wu, Junlu
Lu, Wenying
Sun, Zujun
Li, Dong
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Źródło :
Journal of Experimental & Clinical Cancer Research. 39(1)
Czasopismo naukowe
Tytuł :
PLAC8 inhibits oral squamous cell carcinogenesis and epithelial-mesenchymal transition via the Wnt/β-catenin and PI3K/Akt/GSK3β signaling pathways.
Autorzy :
Wu J; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, P.R. China.
Wang X; Department of Radiology, School and Hospital of Stomatology, Tongji University, Shanghai Engineering Research Center of Tooth Restoration and Regeneration, Shanghai 200072, P.R. China.
Shang A; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, P.R. China.
Vella G; Department of Internal Medicine V Pulmonology, Allergology and Respiratory Critical Care Medicine, Saarland University, Homburg D-66421, Germany.
Sun Z; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, P.R. China.
Ji P; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, P.R. China.
Yang D; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, P.R. China.
Wan A; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, P.R. China.
Yao Y; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, P.R. China.
Li D; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, P.R. China.
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Źródło :
Oncology letters [Oncol Lett] 2020 Nov; Vol. 20 (5), pp. 128. Date of Electronic Publication: 2020 Aug 19.
Typ publikacji :
Journal Article
Czasopismo naukowe
Tytuł :
Rig-G is a growth inhibitory factor of lung cancer cells that suppresses STAT3 and NF-κB.
Autorzy :
Li D; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, 200065 Shanghai, China.
Sun J; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, 200065 Shanghai, China.
Liu W; Department of General Surgery, Shanghai Tongji Hospital, Tongji University School of Medicine, 200065 Shanghai, China.
Wang X; Department of Pharmacy, Putuo People's Hospital, 200060 Shanghai, China.
Bals R; Department of Internal Medicine V - Pulmonology, Allergology, Respiratory Intensive Care Medicine, Saarland University Hospital, 66424 Homburg, Germany.
Wu J; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, 200065 Shanghai, China.
Quan W; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, 200065 Shanghai, China.
Yao Y; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, 200065 Shanghai, China.
Zhang Y; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, 200065 Shanghai, China.
Zhou H; Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, 200065 Shanghai, China.
Wu K; Institute of Pathology, Charité Medical University, 10117 Berlin, Germany.
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Źródło :
Oncotarget [Oncotarget] 2016 Oct 04; Vol. 7 (40), pp. 66032-66050.
Typ publikacji :
Journal Article
MeSH Terms :
Biomarkers, Tumor/*metabolism
Carcinoma, Non-Small-Cell Lung/*pathology
Intracellular Signaling Peptides and Proteins/*metabolism
Lung Neoplasms/*pathology
NF-kappa B/*metabolism
STAT3 Transcription Factor/*metabolism
Animals ; Apoptosis ; Biomarkers, Tumor/genetics ; Carcinoma, Non-Small-Cell Lung/genetics ; Carcinoma, Non-Small-Cell Lung/metabolism ; Cell Proliferation ; Deubiquitinating Enzyme CYLD/genetics ; Deubiquitinating Enzyme CYLD/metabolism ; Down-Regulation ; Female ; Humans ; I-kappa B Proteins/genetics ; I-kappa B Proteins/metabolism ; Intracellular Signaling Peptides and Proteins/genetics ; Lung Neoplasms/genetics ; Lung Neoplasms/metabolism ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; MicroRNAs/genetics ; NF-kappa B/genetics ; PTEN Phosphohydrolase/genetics ; PTEN Phosphohydrolase/metabolism ; Phosphorylation ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; STAT3 Transcription Factor/genetics ; Tumor Cells, Cultured ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
[Mechanisms of antimicrobial peptide cathelicidin secreted by non-tumorous cells for lung tumor growth].
Autorzy :
Yao Y; Department of Clinical Laboratory, Tongji Hospital, University of Tongji, Shanghai 200065, China.
Wu J; Department of Clinical Laboratory, Tongji Hospital, University of Tongji, Shanghai 200065, China.
Quan W; Department of Clinical Laboratory, Tongji Hospital, University of Tongji, Shanghai 200065, China.
Wan H; Department of Clinical Laboratory, Tongji Hospital, University of Tongji, Shanghai 200065, China.
Li D; Department of Clinical Laboratory, Tongji Hospital, University of Tongji, Shanghai 200065, China; Email: .
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Źródło :
Zhonghua yi xue za zhi [Zhonghua Yi Xue Za Zhi] 2015 Oct 13; Vol. 95 (38), pp. 3142-6.
Typ publikacji :
Journal Article
MeSH Terms :
Lung Neoplasms*
Animals ; Anti-Infective Agents ; Antimicrobial Cationic Peptides ; Carcinoma, Lewis Lung ; Cathelicidins ; Disease Progression ; Kaplan-Meier Estimate ; Lung ; Macrophages ; Mice ; Neutrophils
Czasopismo naukowe

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