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Tytuł :
MicroRNA-338-5p alleviates neuronal apoptosis via directly targeting BCL2L11 in APP/PS1 mice.
Autorzy :
Li J; Department of Anesthesiology, Sun Yat-sen Memorial Hospital, Sun Yat-sen University, Guangzhou 510120, China.; Laboratory of RNA and Major Diseases of Brain and Hearts, Sun Yat-sen University, Guangzhou 510120, China.; Guangdong Province Key Laboratory of Brain Function and Disease, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou 510080, China.
Li D; Department of Pediatrics, Sun Yat-sen Memorial Hospital, Sun Yat-sen University, Guangzhou 510120, China.; Laboratory of RNA and Major Diseases of Brain and Hearts, Sun Yat-sen University, Guangzhou 510120, China.
Zhou H; Department of Cardiology, Sun Yat-sen Memorial Hospital, Sun Yat-sen University, Guangzhou 510120, China.; Laboratory of RNA and Major Diseases of Brain and Hearts, Sun Yat-sen University, Guangzhou 510120, China.
Wu G; Department of Anesthesiology, Sun Yat-sen Memorial Hospital, Sun Yat-sen University, Guangzhou 510120, China.; Laboratory of RNA and Major Diseases of Brain and Hearts, Sun Yat-sen University, Guangzhou 510120, China.
He Z; Department of Intensive Care Unit, Sun Yat-sen Memorial Hospital, Sun Yat-sen University, Guangzhou 510120, China.
Liao W; Department of Intensive Care Unit, Sun Yat-sen Memorial Hospital, Sun Yat-sen University, Guangzhou 510120, China.
Li Y; Department of Anesthesiology, Sun Yat-sen Memorial Hospital, Sun Yat-sen University, Guangzhou 510120, China.; Laboratory of RNA and Major Diseases of Brain and Hearts, Sun Yat-sen University, Guangzhou 510120, China.; Guangdong Province Key Laboratory of Brain Function and Disease, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou 510080, China.
Zhi Y; Department of Intensive Care Unit, Sun Yat-sen Memorial Hospital, Sun Yat-sen University, Guangzhou 510120, China.
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Źródło :
Aging [Aging (Albany NY)] 2020 Oct 21; Vol. 12 (20), pp. 20728-20742. Date of Electronic Publication: 2020 Oct 21.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Alzheimer Disease/*etiology
Apoptosis/*physiology
Bcl-2-Like Protein 11/*physiology
MicroRNAs/*physiology
Neurons/*physiology
Animals ; Cells, Cultured ; Cognitive Dysfunction ; Male ; Mice ; Mice, Inbred C57BL
Czasopismo naukowe
Tytuł :
Silencing of circFoxO3 Protects HT22 Cells from Glutamate-Induced Oxidative Injury via Regulating the Mitochondrial Apoptosis Pathway.
Autorzy :
Lin SP; Department of Emergency, The Second Affiliated Hospital of Guangzhou Medical University, 250 Changgang East Road, Guangzhou, Guangdong, 510260, People's Republic of China.
Hu J; Department of Emergency, The Second Affiliated Hospital of Guangzhou Medical University, 250 Changgang East Road, Guangzhou, Guangdong, 510260, People's Republic of China.
Wei JX; Department of Emergency, The Second Affiliated Hospital of Guangzhou Medical University, 250 Changgang East Road, Guangzhou, Guangdong, 510260, People's Republic of China.
Ye S; Department of Geriatrics, The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou, People's Republic of China.
Bu J; Department of Emergency, The Second Affiliated Hospital of Guangzhou Medical University, 250 Changgang East Road, Guangzhou, Guangdong, 510260, People's Republic of China.
Xu W; Guangzhou Medical University, Guangzhou, People's Republic of China.
Chen S; Guangzhou Medical University, Guangzhou, People's Republic of China.
Wu Y; Department of Emergency, The Second Affiliated Hospital of Guangzhou Medical University, 250 Changgang East Road, Guangzhou, Guangdong, 510260, People's Republic of China.
Wu G; Department of Emergency, The Second Affiliated Hospital of Guangzhou Medical University, 250 Changgang East Road, Guangzhou, Guangdong, 510260, People's Republic of China.
Zhu L; Department of Emergency, The Second Affiliated Hospital of Guangzhou Medical University, 250 Changgang East Road, Guangzhou, Guangdong, 510260, People's Republic of China.
Lin PY; Department of Emergency, The Second Affiliated Hospital of Guangzhou Medical University, 250 Changgang East Road, Guangzhou, Guangdong, 510260, People's Republic of China.
Chen XH; Department of Emergency, The Second Affiliated Hospital of Guangzhou Medical University, 250 Changgang East Road, Guangzhou, Guangdong, 510260, People's Republic of China. .
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Źródło :
Cellular and molecular neurobiology [Cell Mol Neurobiol] 2020 Oct; Vol. 40 (7), pp. 1231-1242. Date of Electronic Publication: 2020 Mar 05.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Glutamic Acid/*pharmacology
Hippocampus/*metabolism
Mitochondria/*metabolism
RNA, Circular/*metabolism
Reactive Oxygen Species/*metabolism
Animals ; Apoptosis/physiology ; Cell Line ; Cell Survival/drug effects ; Forkhead Box Protein O3/genetics ; Glutamic Acid/metabolism ; Mice ; Mitochondria/drug effects ; Neurons/metabolism ; Neuroprotective Agents/pharmacology ; Oxidative Stress/drug effects ; Signal Transduction/drug effects
Czasopismo naukowe
Tytuł :
Vacancy-induced toxicity of CoSe 2 nanomaterials in rat lung macrophages.
Autorzy :
Wu G; Tianjin Key Laboratory of Environmental Remediation and Pollution Control, College of Environmental Science and Engineering, Nankai University, Tianjin, China.
Chen X; Tianjin Key Laboratory of Environmental Remediation and Pollution Control, College of Environmental Science and Engineering, Nankai University, Tianjin, China.
Zhang Z; Tianjin Key Laboratory of Environmental Remediation and Pollution Control, College of Environmental Science and Engineering, Nankai University, Tianjin, China.
Zhu N; Key Laboratory of Molecular Microbiology and Technology, Ministry of Education, College of Life Sciences, Nankai University, Tianjin, China.
Yu Q; Key Laboratory of Molecular Microbiology and Technology, Ministry of Education, College of Life Sciences, Nankai University, Tianjin, China.
Liu H; School of Chemical Science and Engineering, Tongji University, Shanghai, China.
Liu L; Tianjin Key Laboratory of Environmental Remediation and Pollution Control, College of Environmental Science and Engineering, Nankai University, Tianjin, China.
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Źródło :
Nanotoxicology [Nanotoxicology] 2020 Sep; Vol. 14 (7), pp. 968-984. Date of Electronic Publication: 2020 Jul 07.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Epithelial Cells/*drug effects
Lung/*drug effects
Macrophages, Alveolar/*drug effects
Nanostructures/*toxicity
Selenium Compounds/*toxicity
Animals ; Cell Line, Tumor ; Cell Survival/drug effects ; Endoplasmic Reticulum Stress/drug effects ; Epithelial Cells/metabolism ; Epithelial Cells/pathology ; Humans ; Macrophages, Alveolar/metabolism ; Macrophages, Alveolar/pathology ; Membrane Potential, Mitochondrial/drug effects ; Nanostructures/chemistry ; Protein Corona/chemistry ; Rats ; Reactive Oxygen Species/metabolism ; Selenium Compounds/chemistry ; Solubility ; Surface Properties ; Tissue Distribution
Czasopismo naukowe
Tytuł :
Taurine attenuates isoproterenol-induced H9c2 cardiomyocytes hypertrophy by improving antioxidative ability and inhibiting calpain-1-mediated apoptosis.
Autorzy :
Li W; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China.
Yang J; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China.
Lyu Q; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China.
Wu G; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China.
Lin S; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China.
Yang Q; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China. .
Hu J; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China. .
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Źródło :
Molecular and cellular biochemistry [Mol Cell Biochem] 2020 Jun; Vol. 469 (1-2), pp. 119-132. Date of Electronic Publication: 2020 Apr 18.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Calpain/*metabolism
Cardiomegaly/*metabolism
Isoproterenol/*adverse effects
Membrane Potential, Mitochondrial/*drug effects
Oxidative Stress/*drug effects
Taurine/*pharmacology
Animals ; Apoptotic Protease-Activating Factor 1/metabolism ; Atrial Natriuretic Factor/metabolism ; Calcium/metabolism ; Calcium-Binding Proteins/metabolism ; Caspase 3/metabolism ; Caspase 9/metabolism ; Cell Line ; Cytochromes c/metabolism ; Isoproterenol/pharmacology ; Mitochondria/metabolism ; Myocytes, Cardiac ; Natriuretic Peptide, Brain/metabolism ; Natriuretic Peptides ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Rats ; Ventricular Myosins/metabolism ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
Glycine Attenuates LPS-Induced Apoptosis and Inflammatory Cell Infiltration in Mouse Liver.
Autorzy :
Zhang Y; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science, China Agricultural University, Beijing 100193, China.
Jia H; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science, China Agricultural University, Beijing 100193, China.
Jin Y; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science, China Agricultural University, Beijing 100193, China.
Liu N; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science, China Agricultural University, Beijing 100193, China.
Chen J; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science, China Agricultural University, Beijing 100193, China.
Yang Y; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science, China Agricultural University, Beijing 100193, China.
Dai Z; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science, China Agricultural University, Beijing 100193, China.
Wang C; State Key Laboratory of Agrobiotechnology, College of Biological Sciences, China Agricultural University, Beijing 100193, China.
Wu G; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science, China Agricultural University, Beijing 100193, China.; Department of Animal Science, Texas A&M University, College Station, TX 77843, USA.
Wu Z; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science, China Agricultural University, Beijing 100193, China.; Beijing Advanced Innovation Center for Food Nutrition and Human Health, China Agricultural University, Beijing 100193, China.
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Źródło :
The Journal of nutrition [J Nutr] 2020 May 01; Vol. 150 (5), pp. 1116-1125.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Chemical and Drug Induced Liver Injury/*prevention & control
Glycine/*pharmacology
Inflammation/*prevention & control
Lipopolysaccharides/*administration & dosage
Liver/*drug effects
Alanine Transaminase/blood ; Animals ; Aspartate Aminotransferases/blood ; Chemical and Drug Induced Liver Injury/pathology ; Hydrogen Peroxide/analysis ; L-Lactate Dehydrogenase/blood ; Liver/chemistry ; Macrophages/pathology ; Male ; Malondialdehyde/analysis ; Mice ; Mice, Inbred C57BL ; Neutrophils/pathology ; Peroxidase/metabolism
Czasopismo naukowe
Tytuł :
Taurine prevents cardiomyocyte apoptosis by inhibiting the calpain-1/cytochrome c pathway during RVH in broilers.
Autorzy :
Li W; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China.
Yang J; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China.
Lyu Q; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China.
Wu G; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China.
Lin S; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China.
Yang Q; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China. .
Hu J; Liaoning Provincial Key Laboratory of Zoonosis, College of Animal Science and Veterinary Medicine, Shenyang Agricultural University, Shenyang, 110866, Liaoning, People's Republic of China. .
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Źródło :
Amino acids [Amino Acids] 2020 Mar; Vol. 52 (3), pp. 453-463. Date of Electronic Publication: 2020 Feb 27.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Calpain/*antagonists & inhibitors
Cytochromes c/*antagonists & inhibitors
Hypertrophy, Right Ventricular/*prevention & control
Myocytes, Cardiac/*drug effects
Taurine/*pharmacology
Animals ; Caspase 3/metabolism ; Caspase 9/metabolism ; Chickens ; Hypertrophy, Right Ventricular/enzymology ; Hypertrophy, Right Ventricular/pathology ; Metabolic Networks and Pathways/drug effects ; Myocytes, Cardiac/enzymology ; Myocytes, Cardiac/pathology ; Taurine/administration & dosage ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
Neurotoxicity of Mn 3 O 4 nanoparticles: Apoptosis and dopaminergic neurons damage pathway.
Autorzy :
Chen X; Tianjin Key Laboratory of Environmental Remediation and Pollution Control, College of Environmental Science and Engineering, Nankai University, Tianjin, 300071, China.
Wu G; Tianjin Key Laboratory of Environmental Remediation and Pollution Control, College of Environmental Science and Engineering, Nankai University, Tianjin, 300071, China.
Zhang Z; Tianjin Key Laboratory of Environmental Remediation and Pollution Control, College of Environmental Science and Engineering, Nankai University, Tianjin, 300071, China.
Ma X; Shanxi Provincial Research Academy of Environmental Science, Xinghua Street NO.11, Taiyuan, Shanxi, 030027, China. Electronic address: .
Liu L; Tianjin Key Laboratory of Environmental Remediation and Pollution Control, College of Environmental Science and Engineering, Nankai University, Tianjin, 300071, China. Electronic address: .
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Źródło :
Ecotoxicology and environmental safety [Ecotoxicol Environ Saf] 2020 Jan 30; Vol. 188, pp. 109909. Date of Electronic Publication: 2019 Nov 15.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Dopaminergic Neurons/*drug effects
Metal Nanoparticles/*toxicity
Oxides/*toxicity
Animals ; Calcium Channels/genetics ; Calcium Channels/metabolism ; Cell Survival ; Dopa Decarboxylase/genetics ; Dopa Decarboxylase/metabolism ; Dopamine/metabolism ; Dopaminergic Neurons/metabolism ; Dopaminergic Neurons/pathology ; Manganese Compounds/chemistry ; Metal Nanoparticles/chemistry ; Oxidative Stress/drug effects ; Oxides/chemistry ; PC12 Cells ; Rats ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł :
Nd 2 O 3 Nanoparticles Induce Toxicity and Cardiac/Cerebrovascular Abnormality in Zebrafish Embryos via the Apoptosis Pathway.
Autorzy :
Chen Y; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.; Beijing Key Laboratory of Hypoxic Conditioning Translational Medicine, Xuanwu Hospital, Capital Medical University, Beijing, People's Republic of China.
Zhu W; School of Pharmacy, Baotou Medical College, Baotou 014040, People's Republic of China.
Shu F; Third Hospital of Baotou, Baotou, People's Republic of China.
Fan Y; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.
Yang N; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.
Wu T; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.
Ji L; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.
Xie W; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.
Bade R; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.
Jiang S; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.
Liu X; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.
Shao G; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.; Beijing Key Laboratory of Hypoxic Conditioning Translational Medicine, Xuanwu Hospital, Capital Medical University, Beijing, People's Republic of China.
Wu G; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.
Jia X; Biomedicine Research Center, Neuroscience Institute, Baotou Medical College, Baotou 014040, People's Republic of China.; Inner Mongolia Key Laboratory of Hypoxic Translational Medicine, Baotou Medical College, Baotou 014040, People's Republic of China.; Beijing Key Laboratory of Hypoxic Conditioning Translational Medicine, Xuanwu Hospital, Capital Medical University, Beijing, People's Republic of China.
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Źródło :
International journal of nanomedicine [Int J Nanomedicine] 2020 Jan 22; Vol. 15, pp. 387-400. Date of Electronic Publication: 2020 Jan 22 (Print Publication: 2020).
Typ publikacji :
Journal Article; Video-Audio Media
MeSH Terms :
Apoptosis/*drug effects
Cardiovascular Abnormalities/*chemically induced
Metal Nanoparticles/*toxicity
Neodymium/*toxicity
Oxides/*toxicity
Zebrafish/*embryology
Animals ; Animals, Genetically Modified ; Arrhythmias, Cardiac/chemically induced ; Dose-Response Relationship, Drug ; Embryo, Nonmammalian/drug effects ; Female ; Heart Rate/drug effects ; Larva/drug effects ; Male ; Toxicity Tests ; Zebrafish/genetics
Czasopismo naukowe
Tytuł :
Gastrodin Attenuates Neuronal Apoptosis and Neurological Deficits after Experimental Intracerebral Hemorrhage.
Autorzy :
Liu XC; Department of neurosurgery, Taizhou Hospital of Zhejiang Province, Linhai, Zhejiang, People's Republic of China.
Wu CZ; Department of neurosurgery, Taizhou Hospital of Zhejiang Province, Linhai, Zhejiang, People's Republic of China.
Hu XF; Department of neurosurgery, Taizhou Hospital of Zhejiang Province, Linhai, Zhejiang, People's Republic of China.
Wang TL; Department of neurosurgery, Taizhou Hospital of Zhejiang Province, Linhai, Zhejiang, People's Republic of China.
Jin XP; Department of neurosurgery, Taizhou Hospital of Zhejiang Province, Linhai, Zhejiang, People's Republic of China.
Ke SF; Department of neurosurgery, Taizhou Hospital of Zhejiang Province, Linhai, Zhejiang, People's Republic of China.
Wang E; Department of neurosurgery, Taizhou Hospital of Zhejiang Province, Linhai, Zhejiang, People's Republic of China.
Wu G; Department of neurosurgery, Taizhou Hospital of Zhejiang Province, Linhai, Zhejiang, People's Republic of China. Electronic address: .
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Źródło :
Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association [J Stroke Cerebrovasc Dis] 2020 Jan; Vol. 29 (1), pp. 104483. Date of Electronic Publication: 2019 Nov 11.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Benzyl Alcohols/*pharmacology
Cerebral Cortex/*drug effects
Cerebral Hemorrhage/*drug therapy
Glucosides/*pharmacology
Neurons/*drug effects
Neuroprotective Agents/*pharmacology
Animals ; Apoptosis Regulatory Proteins/metabolism ; Behavior, Animal/drug effects ; Brain Edema/metabolism ; Brain Edema/pathology ; Brain Edema/prevention & control ; Cells, Cultured ; Cerebral Cortex/metabolism ; Cerebral Cortex/pathology ; Cerebral Cortex/physiopathology ; Cerebral Hemorrhage/metabolism ; Cerebral Hemorrhage/pathology ; Cerebral Hemorrhage/physiopathology ; Disease Models, Animal ; Male ; Motor Activity/drug effects ; Neurons/metabolism ; Neurons/pathology ; Oxidative Stress/drug effects ; Rats, Sprague-Dawley ; Signal Transduction
Czasopismo naukowe
Tytuł :
miR-96-5p Regulates Proliferation, Migration, and Apoptosis of Vascular Smooth Muscle Cell Induced by Angiotensin II via Targeting NFAT5.
Autorzy :
Tian L; Department of Cardiology, Jiangdu People's Hospital, Yangzhou, China.
Cai D; Department of Cardiology, Jiangdu People's Hospital, Yangzhou, China.
Zhuang D; Department of Cardiology, Jiangdu People's Hospital, Yangzhou, China.
Wang W; Department of Cardiology, Jiangdu People's Hospital, Yangzhou, China.
Wang X; Department of Cardiology, Jiangdu People's Hospital, Yangzhou, China.
Bian X; Department of Cardiology, Jiangdu People's Hospital, Yangzhou, China.
Xu R; Department of Nephrology, Jiangdu People's Hospital, Yangzhou, China.
Wu G; Department of Cardiology, Xi'an Central Hospital of Xi'an Jiaotong University, Xi'an, China, .
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Źródło :
Journal of vascular research [J Vasc Res] 2020; Vol. 57 (2), pp. 86-96. Date of Electronic Publication: 2020 Feb 11.
Typ publikacji :
Journal Article
MeSH Terms :
Angiotensin II/*pharmacology
Apoptosis/*drug effects
MicroRNAs/*physiology
Muscle, Smooth, Vascular/*cytology
Myocytes, Smooth Muscle/*physiology
Transcription Factors/*genetics
Cell Movement/drug effects ; Cell Proliferation/drug effects ; Cells, Cultured ; Humans ; Transcription Factors/physiology
Czasopismo naukowe
Tytuł :
miR-429 suppresses cell growth and induces apoptosis of human thyroid cancer cell by targeting ZEB1.
Autorzy :
Wu G; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Zheng H; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Xu J; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Guo Y; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Zheng G; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Ma C; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Hao S; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Liu X; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Chen H; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Wei S; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Song X; b Department of Otolaryngology-Head and Neck Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
Wang X; a Department of Thyroid Surgery , The Affiliated Yantai Yuhuangding Hospital of Qingdao University , Yantai , China.
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Źródło :
Artificial cells, nanomedicine, and biotechnology [Artif Cells Nanomed Biotechnol] 2019 Dec; Vol. 47 (1), pp. 548-554.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*genetics
MicroRNAs/*genetics
Thyroid Neoplasms/*pathology
Zinc Finger E-box-Binding Homeobox 1/*genetics
Base Sequence ; Cell Line, Tumor ; Cell Movement ; Cell Proliferation/genetics ; Down-Regulation ; Humans ; Neoplasm Invasiveness
Czasopismo naukowe
Tytuł :
Perilipin 5 deficiency promotes atherosclerosis progression through accelerating inflammation, apoptosis, and oxidative stress.
Autorzy :
Zhou PL; Department of Interventional Radiology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China.
Li M; Department of Interventional Radiology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China.
Han XW; Department of Interventional Radiology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China.
Bi YH; Department of Interventional Radiology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China.
Zhang WG; Department of Interventional Radiology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China.
Wu ZY; Department of Interventional Radiology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China.
Wu G; Department of Interventional Radiology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan, China.
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Źródło :
Journal of cellular biochemistry [J Cell Biochem] 2019 Nov; Vol. 120 (11), pp. 19107-19123. Date of Electronic Publication: 2019 Jul 11.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Oxidative Stress*
Atherosclerosis/*metabolism
Intracellular Signaling Peptides and Proteins/*deficiency
Muscle Proteins/*deficiency
Animals ; Atherosclerosis/genetics ; Atherosclerosis/pathology ; Caspase 3/genetics ; Caspase 3/metabolism ; Inflammation/genetics ; Inflammation/metabolism ; Inflammation/pathology ; Intracellular Signaling Peptides and Proteins/metabolism ; Mice ; Mice, Knockout, ApoE ; Muscle Proteins/metabolism ; Poly(ADP-ribose) Polymerases/genetics ; Poly(ADP-ribose) Polymerases/metabolism ; RAW 264.7 Cells
Czasopismo naukowe
Tytuł :
Hydrogen inhalation protects hypoxic-ischemic brain damage by attenuating inflammation and apoptosis in neonatal rats.
Autorzy :
Wu G; 1 Department of Pediatrics, Third Xiangya Hospital, Central South University, Changsha 410013, China.
Chen Z; 1 Department of Pediatrics, Third Xiangya Hospital, Central South University, Changsha 410013, China.
Wang P; 1 Department of Pediatrics, Third Xiangya Hospital, Central South University, Changsha 410013, China.
Zhao M; 1 Department of Pediatrics, Third Xiangya Hospital, Central South University, Changsha 410013, China.
Fujino M; 2 Division of Transplantation Immunology, National Research Institute for Child Health and Development, Tokyo 157-8535, Japan.; 3 AIDS Research Center, National Institute of Infectious Diseases, Tokyo 162-8640, Japan.
Zhang C; 1 Department of Pediatrics, Third Xiangya Hospital, Central South University, Changsha 410013, China.
Zhou W; 1 Department of Pediatrics, Third Xiangya Hospital, Central South University, Changsha 410013, China.
Hirano SI; 4 MiZ Co., Ltd, Kanagawa 247-0062, Japan.
Li XK; 1 Department of Pediatrics, Third Xiangya Hospital, Central South University, Changsha 410013, China.; 2 Division of Transplantation Immunology, National Research Institute for Child Health and Development, Tokyo 157-8535, Japan.
Zhao L; 1 Department of Pediatrics, Third Xiangya Hospital, Central South University, Changsha 410013, China.
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Źródło :
Experimental biology and medicine (Maywood, N.J.) [Exp Biol Med (Maywood)] 2019 Sep; Vol. 244 (12), pp. 1017-1027. Date of Electronic Publication: 2019 Jun 12.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Brain Injuries/*prevention & control
Hydrogen/*pharmacology
Hypoxia-Ischemia, Brain/*prevention & control
Administration, Inhalation ; Animals ; Brain Injuries/metabolism ; Brain Injuries/pathology ; Female ; Gene Expression Regulation/drug effects ; Hydrogen/pharmacokinetics ; Hypoxia-Ischemia, Brain/metabolism ; Hypoxia-Ischemia, Brain/pathology ; Inflammation/metabolism ; Inflammation/pathology ; Inflammation/prevention & control ; Male ; Microglia/metabolism ; Microglia/pathology ; Nerve Tissue Proteins/biosynthesis ; Rats ; Rats, Sprague-Dawley ; Time Factors
Czasopismo naukowe
Tytuł :
Amelioration of Enterotoxigenic Escherichia coli -Induced Intestinal Barrier Disruption by Low-Molecular-Weight Chitosan in Weaned Pigs is Related to Suppressed Intestinal Inflammation and Apoptosis.
Autorzy :
Wan J; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China.
Zhang J; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China.
Wu G; Shanghai Institute of Applied Physics, Chinese Academy of Sciences, Shanghai 201800, China.
Chen D; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China.
Yu B; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China.
Huang Z; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China.
Luo Y; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China.
Zheng P; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China.
Luo J; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China.
Mao X; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China.
Yu J; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China.
He J; Institute of Animal Nutrition, Sichuan Agricultural University, Chengdu 611130, China. .
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2019 Jul 16; Vol. 20 (14). Date of Electronic Publication: 2019 Jul 16.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Anti-Inflammatory Agents/*therapeutic use
Chitosan/*therapeutic use
Escherichia coli Infections/*drug therapy
Intestinal Mucosa/*drug effects
Animals ; Anti-Inflammatory Agents/pharmacology ; Chitosan/pharmacology ; Enterotoxigenic Escherichia coli ; Interleukin-6/genetics ; Intestinal Mucosa/metabolism ; Intestinal Mucosa/pathology ; Occludin/genetics ; Occludin/metabolism ; Swine ; Toll-Like Receptor 4/genetics ; Toll-Like Receptor 4/metabolism ; Transcription Factor RelA/genetics ; Transcription Factor RelA/metabolism ; Tumor Necrosis Factor-alpha/genetics ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł :
FAM46C inhibits lipopolysaccharides-induced myocardial dysfunction via downregulating cellular adhesion molecules and inhibiting apoptosis.
Autorzy :
Tan J; Department of Critical Care Medicine, Huashan Hospital, Fudan University, No. 12 Middle Urumqi Road, Shanghai, PR China.
Sun T; Department of Cardiology, Huashan Hospital, Fudan University, No. 12 Middle Urumqi Road, Shanghai, PR China.
Shen J; Department of Critical Care Medicine, Huashan Hospital, Fudan University, No. 12 Middle Urumqi Road, Shanghai, PR China.
Zhu H; Department of Critical Care Medicine, Huashan Hospital, Fudan University, No. 12 Middle Urumqi Road, Shanghai, PR China.
Gong Y; Department of Critical Care Medicine, Huashan Hospital, Fudan University, No. 12 Middle Urumqi Road, Shanghai, PR China.
Zhu H; Department of Critical Care Medicine, Huashan Hospital, Fudan University, No. 12 Middle Urumqi Road, Shanghai, PR China.
Wu G; Department of Critical Care Medicine, Huashan Hospital, Fudan University, No. 12 Middle Urumqi Road, Shanghai, PR China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2019 Jul 15; Vol. 229, pp. 1-12. Date of Electronic Publication: 2019 Mar 22.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Cardiomyopathies/*pathology
Cell Adhesion Molecules/*metabolism
Myocytes, Cardiac/*pathology
Polynucleotide Adenylyltransferase/*metabolism
Proteins/*metabolism
Sepsis/*complications
Animals ; Cardiomyopathies/etiology ; Cardiomyopathies/metabolism ; Case-Control Studies ; Cell Adhesion Molecules/genetics ; Cell Proliferation ; Humans ; Lipopolysaccharides/toxicity ; MAP Kinase Signaling System ; Male ; Mice ; Mice, Inbred C57BL ; Myocytes, Cardiac/metabolism ; Nucleotidyltransferases ; Polynucleotide Adenylyltransferase/genetics ; Proteins/genetics ; Sepsis/chemically induced ; Signal Transduction ; Toll-Like Receptor 4/genetics ; Toll-Like Receptor 4/metabolism
Czasopismo naukowe
Tytuł :
Luteolin attenuates high glucose-induced podocyte injury via suppressing NLRP3 inflammasome pathway.
Autorzy :
Yu Q; Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, Jiangsu 210009, PR China.
Zhang M; State Key Laboratory of Natural Medicines, Department of Physiology, China Pharmaceutical University, Nanjing, Jiangsu 210009, PR China.
Qian L; Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, Jiangsu 210009, PR China.
Wen D; Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, Jiangsu 210009, PR China.
Wu G; Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, Jiangsu 210009, PR China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2019 May 15; Vol. 225, pp. 1-7. Date of Electronic Publication: 2019 Mar 30.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Glucose/*pharmacology
Inflammasomes/*drug effects
Inflammation/*prevention & control
Luteolin/*pharmacology
NLR Family, Pyrin Domain-Containing 3 Protein/*metabolism
Podocytes/*drug effects
Animals ; Cells, Cultured ; Humans ; Inflammasomes/metabolism ; Inflammation/chemically induced ; Inflammation/pathology ; Mice ; NLR Family, Pyrin Domain-Containing 3 Protein/genetics ; Podocytes/metabolism ; Podocytes/pathology ; Reactive Oxygen Species/metabolism ; Signal Transduction
Czasopismo naukowe
Tytuł :
Grem2 mediates podocyte apoptosis in high glucose milieu.
Autorzy :
Wen H; Key Laboratory for Aging and Regenerative Medicine, School of Pharmacy, Southwest Medical University, Luzhou, Sichuan, China; Feinstein Institute for Medical Research and Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, Manhasset, NY, United States.
Kumar V; Feinstein Institute for Medical Research and Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, Manhasset, NY, United States.
Mishra A; Feinstein Institute for Medical Research and Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, Manhasset, NY, United States.
Song S; Feinstein Institute for Medical Research and Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, Manhasset, NY, United States.
Aslam R; Feinstein Institute for Medical Research and Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, Manhasset, NY, United States.
Hussain A; Feinstein Institute for Medical Research and Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, Manhasset, NY, United States.
Wang H; Feinstein Institute for Medical Research and Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, Manhasset, NY, United States; Department of Emergency Medicine, North Shore University Hospital, Manhasset, NY, United States.
Zhou X; Key Laboratory for Aging and Regenerative Medicine, School of Pharmacy, Southwest Medical University, Luzhou, Sichuan, China.
He X; Key Laboratory for Aging and Regenerative Medicine, School of Pharmacy, Southwest Medical University, Luzhou, Sichuan, China.
Wu G; Key Laboratory for Aging and Regenerative Medicine, School of Pharmacy, Southwest Medical University, Luzhou, Sichuan, China.
Luo H; Key Laboratory for Aging and Regenerative Medicine, School of Pharmacy, Southwest Medical University, Luzhou, Sichuan, China.
Lan X; Key Laboratory for Aging and Regenerative Medicine, School of Pharmacy, Southwest Medical University, Luzhou, Sichuan, China; Feinstein Institute for Medical Research and Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, Manhasset, NY, United States. Electronic address: .
Malhotra A; Feinstein Institute for Medical Research and Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, Manhasset, NY, United States.
Singhal PC; Feinstein Institute for Medical Research and Donald and Barbara Zucker School of Medicine at Hofstra/Northwell, Manhasset, NY, United States. Electronic address: .
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Źródło :
Biochimie [Biochimie] 2019 May; Vol. 160, pp. 113-121. Date of Electronic Publication: 2019 Mar 01.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Diabetes Mellitus, Experimental/*complications
Diabetes Mellitus, Type 2/*complications
Diabetic Nephropathies/*pathology
Glucose/*pharmacology
Intercellular Signaling Peptides and Proteins/*metabolism
Podocytes/*pathology
Animals ; Cytokines ; Diabetic Nephropathies/etiology ; Diabetic Nephropathies/metabolism ; Female ; Humans ; Intercellular Signaling Peptides and Proteins/genetics ; Male ; Mice ; Mice, Obese ; Phosphorylation ; Podocytes/drug effects ; Podocytes/metabolism ; Signal Transduction ; Sweetening Agents/pharmacology ; Up-Regulation
Czasopismo naukowe
Tytuł :
Protective Effects of Functional Amino Acids on Apoptosis, Inflammatory Response, and Pulmonary Fibrosis in Lipopolysaccharide-Challenged Mice.
Autorzy :
Ma X; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science , China Agricultural University , Beijing 100193 , China.
Zhang Y; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science , China Agricultural University , Beijing 100193 , China.
Jiang D; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science , China Agricultural University , Beijing 100193 , China.
Yang Y; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science , China Agricultural University , Beijing 100193 , China.
Wu G; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science , China Agricultural University , Beijing 100193 , China.; Department of Animal Science , Texas A&M University , College Station , Texas 77843 , United States.
Wu Z; State Key Laboratory of Animal Nutrition, Department of Animal Nutrition and Feed Science , China Agricultural University , Beijing 100193 , China.
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Źródło :
Journal of agricultural and food chemistry [J Agric Food Chem] 2019 May 01; Vol. 67 (17), pp. 4915-4922. Date of Electronic Publication: 2019 Apr 19.
Typ publikacji :
Journal Article
MeSH Terms :
Amino Acids/*administration & dosage
Apoptosis/*drug effects
Protective Agents/*administration & dosage
Pulmonary Fibrosis/*drug therapy
Animals ; Chemokines/genetics ; Chemokines/immunology ; Cytokines/genetics ; Cytokines/immunology ; Humans ; Lipopolysaccharides/adverse effects ; Lung/drug effects ; Lung/immunology ; Lung/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Neutrophils/drug effects ; Neutrophils/immunology ; Pulmonary Fibrosis/genetics ; Pulmonary Fibrosis/immunology ; Pulmonary Fibrosis/physiopathology
Czasopismo naukowe
Tytuł :
Blockade of Cyclophilin D Attenuates Oxidative Stress-Induced Cell Death in Human Dental Pulp Cells.
Autorzy :
Huang S; Department of Prosthodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.; Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.; Department of Oral Implantology and Prosthetic Dentistry, Academic Centre for Dentistry Amsterdam (ACTA), University of Amsterdam and Vrije Universiteit Amsterdam, 1081 LA Amsterdam, Netherlands.
Zheng B; Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.; Department of Endodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.
Jin X; Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.; Department of Endodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.
Yu Q; Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.; Department of Endodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.
Zhang X; Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.; Department of Endodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.
Sun X; Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.
Chen Y; Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.
Ren X; Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.; Department of Endodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.
Wismeijer D; Department of Oral Implantology and Prosthetic Dentistry, Academic Centre for Dentistry Amsterdam (ACTA), University of Amsterdam and Vrije Universiteit Amsterdam, 1081 LA Amsterdam, Netherlands.
Ma J; Department of Prosthodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.; Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.
Zhang C; Comprehensive Dental Care, Endodontics, Faculty of Dentistry, The University of Hong Kong, Pokfulam, Hong Kong.
Wu G; Department of Oral Implantology and Prosthetic Dentistry, Academic Centre for Dentistry Amsterdam (ACTA), University of Amsterdam and Vrije Universiteit Amsterdam, 1081 LA Amsterdam, Netherlands.
Pan Y; Institute of Stomatology, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.; Department of Endodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China.
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Źródło :
Oxidative medicine and cellular longevity [Oxid Med Cell Longev] 2019 Apr 04; Vol. 2019, pp. 1729013. Date of Electronic Publication: 2019 Apr 04 (Print Publication: 2019).
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*/drug effects
Oxidative Stress*/drug effects
Cyclophilin D/*antagonists & inhibitors
Dental Pulp/*pathology
Acetylcysteine/pharmacology ; Cyclophilin D/metabolism ; Cyclosporine/pharmacology ; Humans ; Hydrogen Peroxide/toxicity ; Mitochondria/drug effects ; Mitochondria/pathology ; RNA, Small Interfering/metabolism
Czasopismo naukowe
Tytuł :
Prion Protein Protects Cancer Cells against Endoplasmic Reticulum Stress Induced Apoptosis.
Autorzy :
Gao Z; State Key Laboratory of Virology, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan, 430071, China.
Peng M; Department of Oncology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Chen L; Department of Oncology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Yang X; Department of the First Abdominal Surgery, Jiangxi Tumor Hospital, Nanchang, 330029, China.
Li H; State Key Laboratory of Virology, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan, 430071, China.
Shi R; State Key Laboratory of Virology, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan, 430071, China.
Wu G; State Key Laboratory of Virology, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan, 430071, China.
Cai L; State Key Laboratory of Virology, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan, 430071, China.
Song Q; Department of Oncology, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
Li C; State Key Laboratory of Virology, Wuhan Institute of Virology, Chinese Academy of Sciences, Wuhan, 430071, China. .
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Źródło :
Virologica Sinica [Virol Sin] 2019 Apr; Vol. 34 (2), pp. 222-234. Date of Electronic Publication: 2019 Apr 24.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Endoplasmic Reticulum Stress*
Prion Proteins/*genetics
Activating Transcription Factor 4/genetics ; Brefeldin A/pharmacology ; Cell Line, Tumor ; Glycosylation ; Humans ; Lung/cytology ; Lung/drug effects ; Prion Proteins/metabolism ; Protein Synthesis Inhibitors/pharmacology ; Signal Transduction ; Thapsigargin/pharmacology ; Tunicamycin/pharmacology ; Unfolded Protein Response
Czasopismo naukowe

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