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Tytuł:
CircRNA_0092516 regulates chondrocyte proliferation and apoptosis in osteoarthritis through the miR-337-3p/PTEN axis.
Autorzy:
Huang Z; Department of Orthopedics, The Third Affiliated Hospital of Suchow University, No. 185 Juqian Road, Changzhou 213000, Jiangsu Province, China.
Ma W; Department of Orthopedics, The Third Affiliated Hospital of Suchow University, No. 185 Juqian Road, Changzhou 213000, Jiangsu Province, China.
Xiao J; Department of Orthopedics, The Third Affiliated Hospital of Suchow University, No. 185 Juqian Road, Changzhou 213000, Jiangsu Province, China.
Dai X; Department of Orthopedics, The Third Affiliated Hospital of Suchow University, No. 185 Juqian Road, Changzhou 213000, Jiangsu Province, China.
Ling W; Department of Orthopedics, The Third Affiliated Hospital of Suchow University, No. 185 Juqian Road, Changzhou 213000, Jiangsu Province, China.
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Źródło:
Journal of biochemistry [J Biochem] 2021 Apr 29; Vol. 169 (4), pp. 467-475.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Cell Proliferation*
Signal Transduction*
Chondrocytes/*metabolism
MicroRNAs/*metabolism
PTEN Phosphohydrolase/*metabolism
RNA, Circular/*metabolism
Aged ; Cell Line ; Chondrocytes/pathology ; Female ; Humans ; Male ; MicroRNAs/genetics ; Middle Aged ; PTEN Phosphohydrolase/genetics ; RNA, Circular/genetics
Czasopismo naukowe
Tytuł:
Vitamin D3 ameliorates nitrogen mustard-induced cutaneous inflammation by inactivating the NLRP3 inflammasome through the SIRT3-SOD2-mtROS signaling pathway.
Autorzy:
Dong X; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.
He Y; Department of Ultrasound, Xinqiao Hospital, Third Military Medical University (Army Medical University), Chongqing, China.
Ye F; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.
Zhao Y; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.
Cheng J; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.
Xiao J; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.
Yu W; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.
Zhao J; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.
Sai Y; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.
Dan G; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.
Chen M; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Pathology and Southwest Cancer Centre, Southwest Hospital, Third Military Medical University (Army Medical University), Chongqing, China.
Zou Z; Department of Chemical Defense Medicine, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.; Institute of Toxicology, School of Military Preventive Medicine, Third Military Medical University (Army Medical University), Chongqing, China.
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Źródło:
Clinical and translational medicine [Clin Transl Med] 2021 Feb; Vol. 11 (2), pp. e312.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Dermatitis, Contact/*etiology
Inflammasomes/*drug effects
Mechlorethamine/*toxicity
NLR Family, Pyrin Domain-Containing 3 Protein/*metabolism
Reactive Oxygen Species/*metabolism
Signal Transduction/*drug effects
Sirtuin 3/*metabolism
Superoxide Dismutase/*metabolism
Vitamin D/*therapeutic use
Animals ; Dermatitis, Contact/drug therapy ; Female ; HaCaT Cells/drug effects ; HaCaT Cells/metabolism ; Humans ; Inflammasomes/physiology ; Keratinocytes/drug effects ; Keratinocytes/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mitochondria/drug effects ; Mitochondria/metabolism
Czasopismo naukowe
Tytuł:
Exogenous hydrogen sulfide reduces atrial remodeling and atrial fibrillation induced by diabetes mellitus via activation of the PI3K/Akt/eNOS pathway.
Autorzy:
Xue X; Center for Comprehensive Treatment of Atrial Fibrillation, Department of Cardiothoracic Surgery, Changzheng Hospital, Second Military Medical University, Shanghai 200003, P.R. China.
Ling X; Center for Comprehensive Treatment of Atrial Fibrillation, Department of Cardiothoracic Surgery, Changzheng Hospital, Second Military Medical University, Shanghai 200003, P.R. China.
Xi W; Center for Comprehensive Treatment of Atrial Fibrillation, Department of Cardiothoracic Surgery, Changzheng Hospital, Second Military Medical University, Shanghai 200003, P.R. China.
Wang P; Center for Comprehensive Treatment of Atrial Fibrillation, Department of Cardiothoracic Surgery, Changzheng Hospital, Second Military Medical University, Shanghai 200003, P.R. China.
Sun J; Department of Rehabilitation, Elderly Rehabilitation Hospital, Suzhou Red Cross Society, Suzhou, Jiangsu 215009, P.R. China.
Yang Q; Center for Comprehensive Treatment of Atrial Fibrillation, Department of Cardiothoracic Surgery, Changzheng Hospital, Second Military Medical University, Shanghai 200003, P.R. China.
Xiao J; Center for Comprehensive Treatment of Atrial Fibrillation, Department of Cardiothoracic Surgery, Changzheng Hospital, Second Military Medical University, Shanghai 200003, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2020 Sep; Vol. 22 (3), pp. 1759-1766. Date of Electronic Publication: 2020 Jun 30.
Typ publikacji:
Journal Article
MeSH Terms:
Atrial Fibrillation/*prevention & control
Diabetes Mellitus, Experimental/*drug therapy
Hydrogen Sulfide/*administration & dosage
Signal Transduction/*drug effects
Vascular Remodeling/*drug effects
Animals ; Atrial Fibrillation/etiology ; Atrial Fibrillation/metabolism ; Cell Culture Techniques ; Diabetes Mellitus, Experimental/chemically induced ; Diabetes Mellitus, Experimental/complications ; Diabetes Mellitus, Experimental/metabolism ; Disease Models, Animal ; Gene Expression Regulation/drug effects ; Humans ; Hydrogen Sulfide/pharmacology ; Male ; Nitric Oxide Synthase Type III/metabolism ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Rats ; Rats, Sprague-Dawley ; Streptozocin ; Treatment Outcome
Czasopismo naukowe
Tytuł:
Preemptive intrathecal administration of endomorphins relieves inflammatory pain in male mice via inhibition of p38 MAPK signaling and regulation of inflammatory cytokines.
Autorzy:
Zhang T; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Zhang N; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Zhang R; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Zhao W; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Chen Y; Department of Neurology, School of Medicine, Duke University, Durham, North Carolina, 27710, USA.
Wang Z; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Xu B; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Zhang M; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Shi X; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Zhang Q; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Guo Y; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Xiao J; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Chen D; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China.
Fang Q; Key Laboratory of Preclinical Study for New Drugs of Gansu Province, and Institute of Physiology, School of Basic Medical Sciences, Lanzhou University, 199 Donggang West Road, Lanzhou, 730000, People's Republic of China. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2018 Nov 15; Vol. 15 (1), pp. 320. Date of Electronic Publication: 2018 Nov 15.
Typ publikacji:
Journal Article
MeSH Terms:
Analgesics, Opioid/*therapeutic use
Oligopeptides/*therapeutic use
Pain/*drug therapy
Signal Transduction/*physiology
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Disease Models, Animal ; Freund's Adjuvant ; Ganglia, Spinal/cytology ; Gene Expression Regulation/drug effects ; Inflammation/chemically induced ; Inflammation/complications ; Injections, Spinal ; Male ; Mice ; Neurons/drug effects ; Opioid Peptides ; Pain/etiology ; Pain/pathology ; Pain Threshold ; Signal Transduction/drug effects ; Time Factors
Czasopismo naukowe
Tytuł:
ROS‑mediated autophagy through the AMPK signaling pathway protects INS‑1 cells from human islet amyloid polypeptide‑induced cytotoxicity.
Autorzy:
Xia G; Department of Endocrinology and Metabolic Disease, Tianjin Medical University General Hospital, Tianjin 300052, P.R. China.
Zhu T; Department of Endocrinology and Metabolic Disease, Tianjin Medical University General Hospital, Tianjin 300052, P.R. China.
Li X; Department of Endocrinology and Metabolic Disease, Tianjin Medical University General Hospital, Tianjin 300052, P.R. China.
Jin Y; Department of Endocrinology and Metabolic Disease, Tianjin Medical University General Hospital, Tianjin 300052, P.R. China.
Zhou J; Department of Endocrinology and Metabolic Disease, Tianjin Medical University General Hospital, Tianjin 300052, P.R. China.
Xiao J; Department of Endocrinology and Metabolic Disease, Tianjin Medical University General Hospital, Tianjin 300052, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2018 Sep; Vol. 18 (3), pp. 2744-2752. Date of Electronic Publication: 2018 Jul 03.
Typ publikacji:
Journal Article
MeSH Terms:
AMP-Activated Protein Kinases/*metabolism
Autophagy/*drug effects
Islet Amyloid Polypeptide/*toxicity
Reactive Oxygen Species/*metabolism
Signal Transduction/*drug effects
AMP-Activated Protein Kinases/antagonists & inhibitors ; Adenine/analogs & derivatives ; Adenine/pharmacology ; Cell Line ; Humans ; Malondialdehyde/metabolism ; Microtubule-Associated Proteins/metabolism ; Oxidative Stress/drug effects ; RNA-Binding Proteins/metabolism
Czasopismo naukowe
Tytuł:
Inhibition of PKR ameliorates lipopolysaccharide-induced acute lung injury by suppressing NF-κB pathway in mice.
Autorzy:
Li Y; a Department of Anesthesiology , Ruijin Hospital, Shanghai Jiao Tong University School of Medicine , Shanghai , China.
Xiao J; a Department of Anesthesiology , Ruijin Hospital, Shanghai Jiao Tong University School of Medicine , Shanghai , China.
Tan Y; a Department of Anesthesiology , Ruijin Hospital, Shanghai Jiao Tong University School of Medicine , Shanghai , China.
Wang J; b Department of Anesthesiology and Intensive Care , Changhai Hospital, Second Military Medical University , Shanghai , China.
Zhang Y; b Department of Anesthesiology and Intensive Care , Changhai Hospital, Second Military Medical University , Shanghai , China.
Deng X; b Department of Anesthesiology and Intensive Care , Changhai Hospital, Second Military Medical University , Shanghai , China.
Luo Y; a Department of Anesthesiology , Ruijin Hospital, Shanghai Jiao Tong University School of Medicine , Shanghai , China.
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Źródło:
Immunopharmacology and immunotoxicology [Immunopharmacol Immunotoxicol] 2017 Aug; Vol. 39 (4), pp. 165-172. Date of Electronic Publication: 2017 May 17.
Typ publikacji:
Journal Article
MeSH Terms:
Acute Lung Injury/*chemically induced
Acute Lung Injury/*metabolism
Anti-Inflammatory Agents/*pharmacology
NF-kappa B/*metabolism
Signal Transduction/*drug effects
eIF-2 Kinase/*antagonists & inhibitors
Animals ; Apoptosis/drug effects ; Interleukin-1beta/metabolism ; Interleukin-6/metabolism ; Lipopolysaccharides/pharmacology ; Lung/drug effects ; Lung/metabolism ; Male ; Mice ; Mice, Inbred BALB C ; Phosphorylation/drug effects ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł:
Overexpression of G protein-coupled receptor GPR87 promotes pancreatic cancer aggressiveness and activates NF-κB signaling pathway.
Autorzy:
Wang L; Department of Emergency Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1277 JieFang Avenue, Wuhan, 430022, China.
Zhou W; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China.
Zhong Y; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China.
Huo Y; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China.
Fan P; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China.
Zhan S; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China.
Xiao J; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China.
Jin X; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China.
Gou S; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China.
Yin T; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China.
Wu H; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China.
Liu T; Department of Pancreatic Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China. .; Department of Digestive Surgical Oncology, Cancer Center, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, People's Republic of China. .
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Źródło:
Molecular cancer [Mol Cancer] 2017 Mar 14; Vol. 16 (1), pp. 61. Date of Electronic Publication: 2017 Mar 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Signal Transduction*
NF-kappa B/*metabolism
Pancreatic Neoplasms/*genetics
Pancreatic Neoplasms/*metabolism
Receptors, Lysophosphatidic Acid/*genetics
Animals ; Biomarkers ; Cell Line, Tumor ; Disease Models, Animal ; Disease Progression ; Gene Expression ; Gene Silencing ; Heterografts ; Humans ; Mice ; Pancreatic Neoplasms/mortality ; Pancreatic Neoplasms/pathology ; Prognosis ; Receptors, Lysophosphatidic Acid/metabolism
Czasopismo naukowe
Tytuł:
Valproate Attenuates Endoplasmic Reticulum Stress-Induced Apoptosis in SH-SY5Y Cells via the AKT/GSK3β Signaling Pathway.
Autorzy:
Li Z; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. LiZM_.
Wu F; Science and Education division, Cixi People's Hospital, Wenzhou Medical University, Ningbo 315300, China. .
Zhang X; Ningbo Medical Treatment Center, Li Huili Hospital, Ningbo 315000, China. .
Chai Y; Department of neurosurgery, The second Affiliated Hospital, Nanchang University, Nanchang 330006, China. .
Chen D; Emergency Department, The Second Affiliated Hospital, Wenzhou Medical University, Wenzhou 325035, China. .
Yang Y; Emergency Department, The Second Affiliated Hospital, Wenzhou Medical University, Wenzhou 325035, China. .
Xu K; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Yin J; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Li R; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Shi H; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Wang Z; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Li X; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .; Institute of Life Sciences, Wenzhou University, Wenzhou 325035, China. .
Xiao J; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Zhang H; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2017 Feb 08; Vol. 18 (2). Date of Electronic Publication: 2017 Feb 08.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Endoplasmic Reticulum Stress/*drug effects
Glycogen Synthase Kinase 3 beta/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Signal Transduction/*drug effects
Valproic Acid/*pharmacology
Cell Line, Tumor ; Cell Survival/drug effects ; Endoplasmic Reticulum Chaperone BiP ; Humans ; JNK Mitogen-Activated Protein Kinases/metabolism ; Matrix Metalloproteinase 9/metabolism ; Neurons/drug effects ; Neurons/metabolism ; Phosphorylation ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Thapsigargin/pharmacology ; Transcription Factor CHOP/metabolism ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł:
Insights into significant pathways and gene interaction networks in peripheral blood mononuclear cells for early diagnosis of hepatocellular carcinoma.
Autorzy:
Jiang JX; Department of Biliary-Hepatic Surgery, Affiliated Hospital of Guiyang Medical College, Guiyang, Guizhou Province, China.
Yu C; Department of Biliary-Hepatic Surgery, Affiliated Hospital of Guiyang Medical College, Guiyang, Guizhou Province, China.
Li ZP; Department of Biliary-Hepatic Surgery, Affiliated Hospital of Guiyang Medical College, Guiyang, Guizhou Province, China.
Xiao J; Department of Biliary-Hepatic Surgery, Affiliated Hospital of Guiyang Medical College, Guiyang, Guizhou Province, China.
Zhang H; Department of Biliary-Hepatic Surgery, Affiliated Hospital of Guiyang Medical College, Guiyang, Guizhou Province, China.
Chen MY; Department of Biliary-Hepatic Surgery, Affiliated Hospital of Guiyang Medical College, Guiyang, Guizhou Province, China.
Sun CY; Department of Biliary-Hepatic Surgery, Affiliated Hospital of Guiyang Medical College, Guiyang, Guizhou Province, China.
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Źródło:
Journal of cancer research and therapeutics [J Cancer Res Ther] 2016 Apr-Jun; Vol. 12 (2), pp. 981-9.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Regulatory Networks*
Signal Transduction*
Carcinoma, Hepatocellular/*genetics
Carcinoma, Hepatocellular/*metabolism
Leukocytes, Mononuclear/*metabolism
Neoplasms/*genetics
Neoplasms/*metabolism
Biomarkers ; Carcinoma, Hepatocellular/diagnosis ; Cluster Analysis ; Computational Biology/methods ; Databases, Nucleic Acid ; Early Detection of Cancer ; Gene Expression Regulation, Neoplastic ; Humans ; Molecular Sequence Annotation ; Neoplasms/diagnosis ; Reproducibility of Results
Czasopismo naukowe
Tytuł:
PTEN Inhibits Cell Proliferation, Promotes Cell Apoptosis, and Induces Cell Cycle Arrest via Downregulating the PI3K/AKT/ hTERT Pathway in Lung Adenocarcinoma A549 Cells.
Autorzy:
Lu XX; Department of Geriatrics, Xiangya Hospital of Central South University, Changsha, Hunan 410008, China.
Cao LY; Department of Geriatrics, Xiangya Hospital of Central South University, Changsha, Hunan 410008, China.
Chen X; Department of Respiratory Medicine, Xiangya Hospital of Central South University, Changsha, Hunan 410008, China.
Xiao J; Department of Geriatrics, Xiangya Hospital of Central South University, Changsha, Hunan 410008, China.
Zou Y; Department of Geriatrics, Xiangya Hospital of Central South University, Changsha, Hunan 410008, China.
Chen Q; Department of Geriatrics, Xiangya Hospital of Central South University, Changsha, Hunan 410008, China.
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Źródło:
BioMed research international [Biomed Res Int] 2016; Vol. 2016, pp. 2476842. Date of Electronic Publication: 2016 Oct 16.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Cell Cycle Checkpoints*
Down-Regulation*
Gene Expression Regulation, Neoplastic*
Signal Transduction*
Adenocarcinoma/*metabolism
Lung Neoplasms/*metabolism
PTEN Phosphohydrolase/*metabolism
Phosphatidylinositol 3-Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Telomerase/*metabolism
Adenocarcinoma/drug therapy ; Adenocarcinoma/genetics ; Adenocarcinoma/pathology ; Amino Acid Substitution ; Cell Line, Tumor ; Chromones/pharmacology ; Humans ; Lung Neoplasms/drug therapy ; Lung Neoplasms/genetics ; Lung Neoplasms/pathology ; Morpholines/pharmacology ; Mutation, Missense ; PTEN Phosphohydrolase/genetics ; Phosphatidylinositol 3-Kinases/genetics ; Proto-Oncogene Proteins c-akt/genetics ; Telomerase/genetics
Czasopismo naukowe
Tytuł:
The Role of bFGF in the Excessive Activation of Astrocytes Is Related to the Inhibition of TLR4/NFκB Signals.
Autorzy:
Ye L; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Yang Y; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Zhang X; Department of Pharmacy, Ningbo Medical Treatment Center, Li Huili Hospital, Ningbo 315040, China. .
Cai P; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Li R; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Chen D; Emergency Department, The Second Affiliated Hospital, Wenzhou Medical University, Wenzhou 325000, China. .
Wei X; Department of Neurosurgery, Cixi People's Hospital, Wenzhou Medical University, Cixi, Ningbo 315300, China. .
Zhang X; Department of Gastroenterology, Ningbo Medical Treatment Center Li Huili Hospital, Ningbo 315040, China. .
Xu H; Department of Orthopaedics, the Second Affiliated Hospital, Wenzhou Medical University, Wenzhou 325000, China. .
Xiao J; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Li X; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Lin L; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
Zhang H; Key Laboratory of Biotechnology and Pharmaceutical Engineering, School of Pharmaceutical Sciences, Wenzhou Medical University, Wenzhou 325035, China. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2015 Dec 28; Vol. 17 (1). Date of Electronic Publication: 2015 Dec 28.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
NF-kappa B*
Signal Transduction*
Toll-Like Receptor 4*
Astrocytes/*pathology
Fibroblast Growth Factor 2/*physiology
Gliosis/*physiopathology
Animals ; Astrocytes/metabolism ; Astrocytes/physiology ; Cells, Cultured ; Cytokines/genetics ; Down-Regulation ; Glial Fibrillary Acidic Protein/genetics ; Gliosis/metabolism ; Neurocan/genetics ; Rats ; Vimentin/genetics
Czasopismo naukowe
Tytuł:
Dasatinib enhances antitumor activity of paclitaxel in ovarian cancer through Src signaling.
Autorzy:
Xiao J; Department of Gynecology, Sun Yat-sen University Cancer Center, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Guangzhou, Guangdong 510060, P.R. China.
Xu M; Department of Obstetrics and Gynecology, The First Affiliated Hospital, Sun Yat‑sen University, Guangzhou, Guangdong 510080, P.R. China.
Hou T; Department of Gynecology, Sun Yat-sen University Cancer Center, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Guangzhou, Guangdong 510060, P.R. China.
Huang Y; Department of Gynecology, Sun Yat-sen University Cancer Center, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Guangzhou, Guangdong 510060, P.R. China.
Yang C; Department of Gynecology, Sun Yat-sen University Cancer Center, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Guangzhou, Guangdong 510060, P.R. China.
Li J; Department of Gynecology, Sun Yat-sen University Cancer Center, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Guangzhou, Guangdong 510060, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2015 Sep; Vol. 12 (3), pp. 3249-3256. Date of Electronic Publication: 2015 May 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents/*therapeutic use
Dasatinib/*therapeutic use
Ovarian Neoplasms/*drug therapy
Ovary/*drug effects
Paclitaxel/*therapeutic use
Signal Transduction/*drug effects
src-Family Kinases/*metabolism
Animals ; Antineoplastic Agents/pharmacology ; Cell Line, Tumor ; Dasatinib/pharmacology ; Drug Synergism ; Enzyme Activation/drug effects ; Female ; Humans ; Mice, Inbred BALB C ; Ovarian Neoplasms/metabolism ; Ovarian Neoplasms/pathology ; Ovary/metabolism ; Ovary/pathology ; Paclitaxel/pharmacology
Czasopismo naukowe
Tytuł:
Hydrogen Peroxide Induce Human Cytomegalovirus Replication through the Activation of p38-MAPK Signaling Pathway.
Autorzy:
Xiao J; Beijing Key Laboratory of Blood Safety and Supply Technologies, Beijing 100850, China. .; Beijing Institute of Transfusion Medicine, 27 (9) Taiping Road, Beijing 100850, China. .
Deng J; Beijing Key Laboratory of Blood Safety and Supply Technologies, Beijing 100850, China. .; Beijing Institute of Transfusion Medicine, 27 (9) Taiping Road, Beijing 100850, China. .
Lv L; Beijing Key Laboratory of Blood Safety and Supply Technologies, Beijing 100850, China. .; Beijing Institute of Transfusion Medicine, 27 (9) Taiping Road, Beijing 100850, China. .
Kang Q; Beijing Key Laboratory of Blood Safety and Supply Technologies, Beijing 100850, China. .; Beijing Institute of Transfusion Medicine, 27 (9) Taiping Road, Beijing 100850, China. .
Ma P; Beijing Key Laboratory of Blood Safety and Supply Technologies, Beijing 100850, China. .; Beijing Institute of Transfusion Medicine, 27 (9) Taiping Road, Beijing 100850, China. .
Yan F; Beijing Key Laboratory of Blood Safety and Supply Technologies, Beijing 100850, China. .; Beijing Institute of Transfusion Medicine, 27 (9) Taiping Road, Beijing 100850, China. .
Song X; Beijing Key Laboratory of Blood Safety and Supply Technologies, Beijing 100850, China. .; Beijing Institute of Transfusion Medicine, 27 (9) Taiping Road, Beijing 100850, China. .
Gao B; Beijing Key Laboratory of Blood Safety and Supply Technologies, Beijing 100850, China. .; Beijing Institute of Transfusion Medicine, 27 (9) Taiping Road, Beijing 100850, China. .
Zhang Y; Beijing Key Laboratory of Blood Safety and Supply Technologies, Beijing 100850, China. .; Beijing Institute of Transfusion Medicine, 27 (9) Taiping Road, Beijing 100850, China. .
Xu J; Beijing Key Laboratory of Blood Safety and Supply Technologies, Beijing 100850, China. .; Beijing Institute of Transfusion Medicine, 27 (9) Taiping Road, Beijing 100850, China. .
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Źródło:
Viruses [Viruses] 2015 Jun 04; Vol. 7 (6), pp. 2816-33. Date of Electronic Publication: 2015 Jun 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Signal Transduction*
Cytomegalovirus/*drug effects
Cytomegalovirus/*physiology
Hydrogen Peroxide/*metabolism
Virus Replication/*drug effects
p38 Mitogen-Activated Protein Kinases/*metabolism
Acetylcysteine/metabolism ; Animals ; Antioxidants/metabolism ; Cells, Cultured ; Cytomegalovirus Infections/virology ; Disease Models, Animal ; Fibroblasts/virology ; Genes, Immediate-Early ; Humans ; Male ; Mice, Inbred BALB C ; Oxidants/metabolism ; Promoter Regions, Genetic/drug effects ; Transcription, Genetic
Czasopismo naukowe
Tytuł:
Glycosylation of KSHV encoded vGPCR functions in its signaling and tumorigenicity.
Autorzy:
Wu H; Key Laboratory of Protein Chemistry and Developmental Biology of Ministry of Education of China, College of Life Science, Hunan Normal University, Changsha 410081, China. .
Liu L; Division of Pediatric Neurology, Children's Medical Center, The Second Xiangya Hospital, Central South University, Changsha 410011, China. .
Xiao J; Key Laboratory of Protein Chemistry and Developmental Biology of Ministry of Education of China, College of Life Science, Hunan Normal University, Changsha 410081, China. .
Chi M; Key Laboratory of Protein Chemistry and Developmental Biology of Ministry of Education of China, College of Life Science, Hunan Normal University, Changsha 410081, China. .
Qu Y; Key Laboratory of Protein Chemistry and Developmental Biology of Ministry of Education of China, College of Life Science, Hunan Normal University, Changsha 410081, China. .
Feng H; Key Laboratory of Protein Chemistry and Developmental Biology of Ministry of Education of China, College of Life Science, Hunan Normal University, Changsha 410081, China. .
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Źródło:
Viruses [Viruses] 2015 Mar 31; Vol. 7 (4), pp. 1627-41. Date of Electronic Publication: 2015 Mar 31.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Carcinogenesis*
Oncogenes*
Signal Transduction*
Herpesvirus 8, Human/*physiology
Receptors, Chemokine/*metabolism
Animals ; DNA Mutational Analysis ; Disease Models, Animal ; Fluorescent Antibody Technique ; Glycosylation ; Herpesvirus 8, Human/genetics ; Heterografts/pathology ; Mice ; Mutant Proteins/genetics ; Mutant Proteins/metabolism ; Protein Transport ; Receptors, Chemokine/genetics ; Sarcoma, Kaposi/pathology ; Sarcoma, Kaposi/virology
Czasopismo naukowe
Tytuł:
bFGF regulates autophagy and ubiquitinated protein accumulation induced by myocardial ischemia/reperfusion via the activation of the PI3K/Akt/mTOR pathway.
Autorzy:
Wang ZG; 1] School of Pharmacy, Key Laboratory of Biotechnology and Pharmaceutical Engineering, Wenzhou Medical University, Wenzhou 325035, China [2] Department of Biochemistry and Molecular Biology, College of Basic Medical Science, Jilin University, Changchun, 130012, China.
Wang Y; 1] School of Pharmacy, Key Laboratory of Biotechnology and Pharmaceutical Engineering, Wenzhou Medical University, Wenzhou 325035, China [2] Department of Biochemistry and Molecular Biology, College of Basic Medical Science, Jilin University, Changchun, 130012, China.
Huang Y; School of Pharmacy, Key Laboratory of Biotechnology and Pharmaceutical Engineering, Wenzhou Medical University, Wenzhou 325035, China.
Lu Q; Department of Pediatric Cardiology, The Second Affiliated Hospital, Wenzhou Medical University, Wenzhou, 325000, China.
Zheng L; Department of Ultrasound, The Second Affiliated Hospital, Wenzhou Medical University, Wenzhou, 325000, China.
Hu D; Department of Medical Immunology, Medical School, Anhui University of Science and Technology, Huainan 232001, China.
Feng WK; School of Pharmacy, Key Laboratory of Biotechnology and Pharmaceutical Engineering, Wenzhou Medical University, Wenzhou 325035, China.
Liu YL; School of Pharmacy, Key Laboratory of Biotechnology and Pharmaceutical Engineering, Wenzhou Medical University, Wenzhou 325035, China.
Ji KT; Department of Pediatric Cardiology, The Second Affiliated Hospital, Wenzhou Medical University, Wenzhou, 325000, China.
Zhang HY; School of Pharmacy, Key Laboratory of Biotechnology and Pharmaceutical Engineering, Wenzhou Medical University, Wenzhou 325035, China.
Fu XB; Institute of Basic Medical Science, Chinese PLA General Hospital, Beijing 100853, China.
Li XK; 1] School of Pharmacy, Key Laboratory of Biotechnology and Pharmaceutical Engineering, Wenzhou Medical University, Wenzhou 325035, China [2] Department of Biochemistry and Molecular Biology, College of Basic Medical Science, Jilin University, Changchun, 130012, China.
Chu MP; Department of Pediatric Cardiology, The Second Affiliated Hospital, Wenzhou Medical University, Wenzhou, 325000, China.
Xiao J; School of Pharmacy, Key Laboratory of Biotechnology and Pharmaceutical Engineering, Wenzhou Medical University, Wenzhou 325035, China.
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Źródło:
Scientific reports [Sci Rep] 2015 Mar 19; Vol. 5, pp. 9287. Date of Electronic Publication: 2015 Mar 19.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Signal Transduction*/drug effects
Fibroblast Growth Factor 2/*metabolism
Myocardial Reperfusion Injury/*metabolism
Phosphatidylinositol 3-Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
TOR Serine-Threonine Kinases/*metabolism
Ubiquitinated Proteins/*metabolism
Animals ; Apoptosis/drug effects ; Cell Line ; Disease Models, Animal ; Echocardiography ; Fibroblast Growth Factor 2/pharmacology ; Fibrosis ; Gene Silencing ; Humans ; Male ; Mice ; Myocardial Reperfusion Injury/diagnosis ; Myocardial Reperfusion Injury/genetics ; Myocardial Reperfusion Injury/physiopathology ; Myocytes, Cardiac/drug effects ; Myocytes, Cardiac/metabolism ; RNA Interference ; Rats
Czasopismo naukowe
Tytuł:
Jagged-1 signaling suppresses the IL-6 and TGF-β treatment-induced Th17 cell differentiation via the reduction of RORγt/IL-17A/IL-17F/IL-23a/IL-12rb1.
Autorzy:
Wang Y; 1] Institute of Tissue Transplantation and Immunology, Department of Immunobiology, Jinan University, Guangzhou 510632, China [2] Key Laboratory of Functional Protein Research of Guangdong Higher Education Institutes, Jinan University, Guangzhou 510632, China.
Xing F; 1] Institute of Tissue Transplantation and Immunology, Department of Immunobiology, Jinan University, Guangzhou 510632, China [2] Key Laboratory of Functional Protein Research of Guangdong Higher Education Institutes, Jinan University, Guangzhou 510632, China.
Ye S; Institute of Tissue Transplantation and Immunology, Department of Immunobiology, Jinan University, Guangzhou 510632, China.
Xiao J; Institute of Tissue Transplantation and Immunology, Department of Immunobiology, Jinan University, Guangzhou 510632, China.
Di J; Institute of Tissue Transplantation and Immunology, Department of Immunobiology, Jinan University, Guangzhou 510632, China.
Zeng S; Institute of Tissue Transplantation and Immunology, Department of Immunobiology, Jinan University, Guangzhou 510632, China.
Liu J; Department of Stomatology, Jinan University, Guangzhou 510632, China.
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Źródło:
Scientific reports [Sci Rep] 2015 Feb 04; Vol. 5, pp. 8234. Date of Electronic Publication: 2015 Feb 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Signal Transduction*
Calcium-Binding Proteins/*metabolism
Cytokines/*metabolism
Cytokines/*pharmacology
Intercellular Signaling Peptides and Proteins/*metabolism
Membrane Proteins/*metabolism
Nuclear Receptor Subfamily 1, Group F, Member 3/*metabolism
Th17 Cells/*drug effects
Th17 Cells/*metabolism
Animals ; Cell Differentiation/drug effects ; Cell Differentiation/genetics ; Gene Expression Regulation ; Interleukin-17/metabolism ; Interleukin-17/pharmacology ; Interleukin-6/pharmacology ; Jagged-1 Protein ; Mice ; Receptors, Notch/metabolism ; Serrate-Jagged Proteins ; T-Lymphocyte Subsets/cytology ; T-Lymphocyte Subsets/drug effects ; T-Lymphocyte Subsets/metabolism ; Th17 Cells/cytology
Czasopismo naukowe
Tytuł:
Glucocorticoids retard skeletal muscle development and myoblast protein synthesis through a mechanistic target of rapamycin (mTOR)-signaling pathway in broilers (Gallus gallus domesticus).
Autorzy:
Wang X; a Department of Animal Science , Shandong Agricultural University , Taian , Shandong , PR China.
Jia Q; a Department of Animal Science , Shandong Agricultural University , Taian , Shandong , PR China.
Xiao J; a Department of Animal Science , Shandong Agricultural University , Taian , Shandong , PR China.
Jiao H; a Department of Animal Science , Shandong Agricultural University , Taian , Shandong , PR China.
Lin H; a Department of Animal Science , Shandong Agricultural University , Taian , Shandong , PR China.
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Źródło:
Stress (Amsterdam, Netherlands) [Stress] 2015; Vol. 18 (6), pp. 686-98. Date of Electronic Publication: 2015 Sep 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Dexamethasone/*pharmacology
Glucocorticoids/*pharmacology
Muscle Development/*drug effects
Myoblasts/*drug effects
Signal Transduction/*drug effects
TOR Serine-Threonine Kinases/*metabolism
AMP-Activated Protein Kinases/metabolism ; Animals ; Chickens ; Male ; Muscle, Skeletal/drug effects ; Muscle, Skeletal/metabolism ; Myoblasts/metabolism ; Phosphorylation/drug effects ; Protein Serine-Threonine Kinases/metabolism ; Sirolimus
Czasopismo naukowe
Tytuł:
Progesterone and Src family inhibitor PP1 synergistically inhibit cell migration and invasion of human basal phenotype breast cancer cells.
Autorzy:
Xie M; Department of Geriatrics, Xiangya Hospital, Central South University, 87 Xiangya Road, Changsha, Hunan 410008, China ; Department of Respiratory, Xiangya Hospital, Central South University, 87 Xiangya Road, Changsha, Hunan 410008, China ; Laboratory of Cancer Experimental Therapy, Atlanta Research & Educational Foundation (151F), Atlanta VA Medical Center, 1670 Clairmont Road, Decatur, GA 30033, USA.
Zhou L; Laboratory of Cancer Experimental Therapy, Atlanta Research & Educational Foundation (151F), Atlanta VA Medical Center, 1670 Clairmont Road, Decatur, GA 30033, USA ; Department of Oncology, Shanghai Seventh People's Hospital, 358 Datong Road, Pudong New District, Shanghai 200137, China.
Chen X; Department of Respiratory, Xiangya Hospital, Central South University, 87 Xiangya Road, Changsha, Hunan 410008, China.
Gainey LO; Laboratory of Cancer Experimental Therapy, Atlanta Research & Educational Foundation (151F), Atlanta VA Medical Center, 1670 Clairmont Road, Decatur, GA 30033, USA.
Xiao J; Department of Geriatrics, Xiangya Hospital, Central South University, 87 Xiangya Road, Changsha, Hunan 410008, China ; Department of Respiratory, Xiangya Hospital, Central South University, 87 Xiangya Road, Changsha, Hunan 410008, China.
Nanes MS; Medical Service, Atlanta VA Medical Center and Division of Endocrinology, Metabolism, and Lipids, Department of Medicine, Emory University School of Medicine, Decatur, GA 30033, USA.
Hou A; Department of Oncology, Shanghai Seventh People's Hospital, 358 Datong Road, Pudong New District, Shanghai 200137, China.
You S; Laboratory of Cancer Experimental Therapy, Atlanta Research & Educational Foundation (151F), Atlanta VA Medical Center, 1670 Clairmont Road, Decatur, GA 30033, USA.
Chen Q; Department of Geriatrics, Xiangya Hospital, Central South University, 87 Xiangya Road, Changsha, Hunan 410008, China ; Department of Respiratory, Xiangya Hospital, Central South University, 87 Xiangya Road, Changsha, Hunan 410008, China.
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Źródło:
BioMed research international [Biomed Res Int] 2015; Vol. 2015, pp. 426429. Date of Electronic Publication: 2015 May 17.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Retracted Publication
MeSH Terms:
Breast Neoplasms/*drug therapy
Cell Movement/*drug effects
Neoplasm Proteins/*antagonists & inhibitors
Progesterone/*pharmacology
Pyrazoles/*pharmacology
Pyrimidines/*pharmacology
Signal Transduction/*drug effects
src-Family Kinases/*antagonists & inhibitors
Breast Neoplasms/enzymology ; Breast Neoplasms/genetics ; Breast Neoplasms/pathology ; Cell Line, Tumor ; Female ; Humans ; Neoplasm Invasiveness ; Neoplasm Proteins/genetics ; Neoplasm Proteins/metabolism ; Signal Transduction/genetics
Czasopismo naukowe
Tytuł:
Epigallocatechin gallate attenuates fibrosis, oxidative stress, and inflammation in non-alcoholic fatty liver disease rat model through TGF/SMAD, PI3 K/Akt/FoxO1, and NF-kappa B pathways.
Autorzy:
Xiao J; Center for Gene and Cell Engineering, Shenzhen Institute of Advanced Technology, Chinese Academy of Sciences, Shenzhen, China, jia.xiao@siat.ac.cn.
Ho CT
Liong EC
Nanji AA
Leung TM
Lau TY
Fung ML
Tipoe GL
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Źródło:
European journal of nutrition [Eur J Nutr] 2014 Feb; Vol. 53 (1), pp. 187-99. Date of Electronic Publication: 2013 Mar 21.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Signal Transduction*
Antioxidants/*pharmacology
Catechin/*analogs & derivatives
Fatty Liver/*drug therapy
Oxidative Stress/*drug effects
Alanine Transaminase/blood ; Animals ; Aspartate Aminotransferases/blood ; Catechin/pharmacology ; Cyclooxygenase 2/metabolism ; Diet, High-Fat ; Down-Regulation ; Fatty Liver/pathology ; Female ; Fibrosis ; Forkhead Transcription Factors/metabolism ; Inflammation/metabolism ; Inflammation/pathology ; Liver/drug effects ; Liver/metabolism ; Matrix Metalloproteinase 2/metabolism ; NF-kappa B/metabolism ; Nerve Tissue Proteins/metabolism ; Nitric Oxide Synthase Type II/metabolism ; Non-alcoholic Fatty Liver Disease ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Rats ; Rats, Sprague-Dawley ; Smad Proteins/metabolism ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł:
Glucotoxicity and α cell dysfunction: involvement of the PI3K/Akt pathway in glucose-induced insulin resistance in rat islets and clonal αTC1-6 cells.
Autorzy:
Shen XX; Endocrinology and Cardiac Disease Clinical Center, Fuwai Hospital, Peking Union Medical College and Chinese Academy of Medical Sciences, Beijing, PR China. />Li HL
Pan L
Hong J
Xiao J
Hermansen K
Jeppesen PB
Li GW
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Źródło:
Endocrine research [Endocr Res] 2012; Vol. 37 (1), pp. 12-24. Date of Electronic Publication: 2011 Oct 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Glucagon-Secreting Cells/*metabolism
Glucose/*pharmacology
Insulin Resistance/*physiology
Phosphatidylinositol 3-Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Signal Transduction/*physiology
Animals ; Cells, Cultured ; Glucagon/metabolism ; Glucagon-Secreting Cells/drug effects ; Glucose/metabolism ; Insulin/metabolism ; Insulin Secretion ; Islets of Langerhans/drug effects ; Islets of Langerhans/metabolism ; Male ; Rats ; Rats, Sprague-Dawley ; Signal Transduction/drug effects
Czasopismo naukowe

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