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Wyszukujesz frazę ""Xie, Lai-Hua"" wg kryterium: Autor


Wyświetlanie 1-12 z 12
Tytuł:
Molecular Mechanisms of Ferroptosis and Relevance to Cardiovascular Disease.
Autorzy:
Xie LH; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers University, Newark, NJ 07103, USA.
Fefelova N; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers University, Newark, NJ 07103, USA.
Pamarthi SH; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers University, Newark, NJ 07103, USA.
Gwathmey JK; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers University, Newark, NJ 07103, USA.
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Źródło:
Cells [Cells] 2022 Sep 01; Vol. 11 (17). Date of Electronic Publication: 2022 Sep 01.
Typ publikacji:
Journal Article; Review; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
COVID-19*
Cardiovascular Diseases*
Ferroptosis*
Reperfusion Injury*
Humans ; Iron/metabolism ; Lipid Peroxidation
Czasopismo naukowe
Tytuł:
Calcium and Heart Failure: How Did We Get Here and Where Are We Going?
Autorzy:
Siri-Angkul N; Department of Cell Biology and Molecular Medicine, Rutgers University-New Jersey Medical School, Newark, NJ 07103, USA.; Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, Chiang Mai 50200, Thailand.; Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai 50200, Thailand.
Dadfar B; Department of General Medicine, School of Medicine, Mazandaran University of Medical Sciences, Sari 1471655836, Iran.
Jaleel R; School of International Education, Zhengzhou University, Zhengzhou 450001, China.
Naushad J; Weill Cornell Medicine Qatar, Doha P. O. Box 24144, Qatar.
Parambathazhath J; Process Dynamics Laboratories, Al Khor 679576, Qatar.
Doye AA; Gwathmey Inc., Cambridge, MA 02138, USA.
Xie LH; Department of Cell Biology and Molecular Medicine, Rutgers University-New Jersey Medical School, Newark, NJ 07103, USA.
Gwathmey JK; Department of Cell Biology and Molecular Medicine, Rutgers University-New Jersey Medical School, Newark, NJ 07103, USA.; Department of Medicine, Boston University School of Medicine, Boston, MA 02118, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Jul 09; Vol. 22 (14). Date of Electronic Publication: 2021 Jul 09.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Ca(2+) Mg(2+)-ATPase/*metabolism
Calcium/*metabolism
Heart Failure/*drug therapy
Myocardial Contraction/*physiology
Sarcoplasmic Reticulum/*metabolism
Adenosine Triphosphatases/metabolism ; Adrenergic beta-1 Receptor Agonists/pharmacology ; Adrenergic beta-1 Receptor Agonists/therapeutic use ; Adrenergic beta-Antagonists/pharmacology ; Animals ; Antioxidants/pharmacology ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/antagonists & inhibitors ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/metabolism ; Cardiotonic Agents/pharmacology ; Dobutamine/pharmacology ; Dobutamine/therapeutic use ; Heart Failure/physiopathology ; Humans
Czasopismo naukowe
Tytuł:
Mitochondrial depolarization promotes calcium alternans: Mechanistic insights from a ventricular myocyte model.
Autorzy:
Pandey V; Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, California, United States of America.
Xie LH; Department of Cell Biology and Molecular Medicine, Rutgers, New Jersey Medical School, Newark, New Jersey, United States of America.
Qu Z; Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, California, United States of America.; Department of Computational Medicine, David Geffen School of Medicine, University of California, Los Angeles, California, United States of America.
Song Z; Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, California, United States of America.; Peng Cheng Laboratory, Shenzhen, Guangdong, China.
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Źródło:
PLoS computational biology [PLoS Comput Biol] 2021 Jan 25; Vol. 17 (1), pp. e1008624. Date of Electronic Publication: 2021 Jan 25 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Arrhythmias, Cardiac*/metabolism
Arrhythmias, Cardiac*/physiopathology
Models, Cardiovascular*
Action Potentials/*physiology
Calcium/*metabolism
Mitochondria/*physiology
Animals ; Cells, Cultured ; Computational Biology ; Computer Simulation ; Heart Ventricles/cytology ; Mitochondria/enzymology ; Mitochondria/metabolism ; Myocytes, Cardiac/metabolism ; Myocytes, Cardiac/physiology ; Rabbits ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł:
The effects of macrophages on cardiomyocyte calcium-handling function using in vitro culture models.
Autorzy:
Hitscherich PG; Department of Biomedical Engineering, New Jersey Institute of Technology, Newark, New Jersey.
Xie LH; Department of Cell Biology and Molecular Medicine, Rutgers New Jersey Medical School, Newark, New Jersey.
Del Re D; Department of Cell Biology and Molecular Medicine, Rutgers New Jersey Medical School, Newark, New Jersey.
Lee EJ; Department of Biomedical Engineering, New Jersey Institute of Technology, Newark, New Jersey.
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Źródło:
Physiological reports [Physiol Rep] 2019 Jul; Vol. 7 (13), pp. e14137.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms:
Calcium Signaling*
Myocytes, Cardiac/*metabolism
Animals ; Cellular Microenvironment ; Coculture Techniques ; Culture Media, Conditioned/pharmacology ; Mice ; Myocytes, Cardiac/drug effects ; Osteopontin/genetics ; Osteopontin/metabolism ; RAW 264.7 Cells ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/genetics ; Sarcoplasmic Reticulum Calcium-Transporting ATPases/metabolism ; Troponin I/genetics ; Troponin I/metabolism
Czasopismo naukowe
Tytuł:
The valosin-containing protein is a novel mediator of mitochondrial respiration and cell survival in the heart in vivo.
Autorzy:
Lizano P; Department of Cell Biology and Molecular Medicine; New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, NJ, 07103, USA.
Rashed E; Department of Cell Biology and Molecular Medicine; New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, NJ, 07103, USA.
Stoll S; Division of Physiology, Department of Basic Science, School of Medicine, Loma Linda University, Loma Linda, CA, 92324, USA.
Zhou N; Division of Physiology, Department of Basic Science, School of Medicine, Loma Linda University, Loma Linda, CA, 92324, USA.
Wen H; Department of Cell Biology and Molecular Medicine; New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, NJ, 07103, USA.
Hays TT; Division of Physiology, Department of Basic Science, School of Medicine, Loma Linda University, Loma Linda, CA, 92324, USA.
Qin G; Department of Biomedical Engineering, University of Alabama at Birmingham (UAB),Birmingham, AL, 35294, USA.
Xie LH; Department of Cell Biology and Molecular Medicine; New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, NJ, 07103, USA.
Depre C; Department of Cell Biology and Molecular Medicine; New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, NJ, 07103, USA.
Qiu H; Department of Cell Biology and Molecular Medicine; New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, NJ, 07103, USA.; Division of Physiology, Department of Basic Science, School of Medicine, Loma Linda University, Loma Linda, CA, 92324, USA.
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Źródło:
Scientific reports [Sci Rep] 2017 Apr 20; Vol. 7, pp. 46324. Date of Electronic Publication: 2017 Apr 20.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Respiration*/genetics
Cell Survival*/genetics
Mitochondria/*metabolism
Myocardium/*metabolism
Valosin Containing Protein/*metabolism
Adenosine Diphosphate/metabolism ; Animals ; Biomarkers ; Female ; Gene Deletion ; Gene Expression ; Male ; Mice ; Mice, Transgenic ; Mitochondria/genetics ; Mitochondrial Membrane Transport Proteins/genetics ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Models, Biological ; Myocardial Reperfusion Injury/genetics ; Myocardial Reperfusion Injury/metabolism ; Myocardial Reperfusion Injury/pathology ; Myocytes, Cardiac/metabolism ; Nitric Oxide Synthase Type II/genetics ; Nitric Oxide Synthase Type II/metabolism ; Oxygen Consumption ; Valosin Containing Protein/genetics
Czasopismo naukowe
Tytuł:
The Zinc Ion Chelating Agent TPEN Attenuates Neuronal Death/apoptosis Caused by Hypoxia/ischemia Via Mediating the Pathophysiological Cascade Including Excitotoxicity, Oxidative Stress, and Inflammation.
Autorzy:
Wang WM; College of Life Sciences, Nankai University, Tianjin, China.; Department of Pharmacology, Second Military Medical University, Shanghai, China.
Liu Z; College of Life Sciences, Nankai University, Tianjin, China.
Liu AJ; Department of Pharmacology, Second Military Medical University, Shanghai, China.
Wang YX; College of Life Sciences, Nankai University, Tianjin, China.
Wang HG; College of Life Sciences, Nankai University, Tianjin, China.
An D; College of Life Sciences, Nankai University, Tianjin, China.
Heng B; College of Life Sciences, Nankai University, Tianjin, China.
Xie LH; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, New Brunswick, NJ, USA.
Duan JL; Department of Gerontology, Xin Hua Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Liu YQ; College of Life Sciences, Nankai University, Tianjin, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2015 Sep; Vol. 21 (9), pp. 708-17. Date of Electronic Publication: 2015 Jul 20.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Cell Hypoxia/*drug effects
Ethylenediamines/*pharmacology
Ischemia/*drug therapy
Neurons/*drug effects
Neuroprotective Agents/*pharmacology
Animals ; Apoptosis/physiology ; Brain/drug effects ; Brain/pathology ; Brain/physiopathology ; Brain Ischemia/drug therapy ; Brain Ischemia/pathology ; Brain Ischemia/physiopathology ; Cell Hypoxia/physiology ; Chelating Agents/pharmacology ; Disease Models, Animal ; Female ; Glucose/deficiency ; Infarction, Middle Cerebral Artery ; Ischemia/pathology ; Ischemia/physiopathology ; Neuroimmunomodulation/drug effects ; Neuroimmunomodulation/physiology ; Neurons/pathology ; Neurons/physiology ; Oxidative Stress/drug effects ; Oxidative Stress/physiology ; PC12 Cells ; Rats ; Rats, Inbred SHR ; Receptors, AMPA/metabolism ; Receptors, N-Methyl-D-Aspartate/metabolism ; Zinc/metabolism
Czasopismo naukowe
Tytuł:
Selective Connexin43 Inhibition Prevents Isoproterenol-Induced Arrhythmias and Lethality in Muscular Dystrophy Mice.
Autorzy:
Gonzalez JP; Department of Cell Biology and Molecular Medicine, Rutgers Biomedical and Health Sciences, New Jersey Medical School, Newark, NJ USA.
Ramachandran J; Department of Pharmacology and Physiology, Rutgers Biomedical and Health Sciences, New Jersey Medical School, Newark, NJ USA.
Xie LH; Department of Pharmacology and Physiology, Rutgers Biomedical and Health Sciences, New Jersey Medical School, Newark, NJ USA.
Contreras JE; Department of Pharmacology and Physiology, Rutgers Biomedical and Health Sciences, New Jersey Medical School, Newark, NJ USA.
Fraidenraich D; Department of Cell Biology and Molecular Medicine, Rutgers Biomedical and Health Sciences, New Jersey Medical School, Newark, NJ USA.
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Źródło:
Scientific reports [Sci Rep] 2015 Aug 27; Vol. 5, pp. 13490. Date of Electronic Publication: 2015 Aug 27.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Arrhythmias, Cardiac/*drug therapy
Connexin 43/*antagonists & inhibitors
Muscular Dystrophy, Animal/*complications
Muscular Dystrophy, Duchenne/*complications
Adolescent ; Animals ; Arrhythmias, Cardiac/pathology ; Connexin 43/metabolism ; Disease Models, Animal ; Female ; Humans ; Isoproterenol ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Inbred mdx ; Muscular Dystrophy, Animal/pathology ; Muscular Dystrophy, Duchenne/pathology ; Peptides/pharmacology ; Peptides/therapeutic use ; Up-Regulation/drug effects ; Up-Regulation/genetics
Czasopismo naukowe
Tytuł:
Cardiac specific expression of threonine 5 to alanine mutant sarcolipin results in structural remodeling and diastolic dysfunction.
Autorzy:
Shanmugam M; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Li D; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Gao S; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Fefelova N; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Shah V; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Voit A; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Pachon R; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Yehia G; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Xie LH; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Babu GJ; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
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Źródło:
PloS one [PLoS One] 2015 Feb 11; Vol. 10 (2), pp. e0115822. Date of Electronic Publication: 2015 Feb 11 (Print Publication: 2015).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Mutation*
Muscle Proteins/*genetics
Myocardium/*metabolism
Myocardium/*pathology
Proteolipids/*genetics
Threonine/*genetics
Ventricular Dysfunction/*genetics
Ventricular Remodeling/*genetics
Animals ; Calcium/metabolism ; Diastole/genetics ; Gene Expression ; Heart Atria/metabolism ; Hemodynamics ; Mice ; Mice, Transgenic ; Muscle Proteins/metabolism ; Organ Specificity/genetics ; Proteolipids/metabolism ; Sarcoplasmic Reticulum/metabolism ; Threonine/metabolism
Czasopismo naukowe
Tytuł:
Store-operated Ca2+ entry (SOCE) regulates melanoma proliferation and cell migration.
Autorzy:
Umemura M; Cardiovascular Research Institute, Yokohama City University School of Medicine, Yokohama, Japan.
Baljinnyam E; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Feske S; Department of Pathology, New York University School of Medicine, New York, New York, United States of America.
De Lorenzo MS; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Xie LH; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Feng X; Cardiovascular Research Institute, Yokohama City University School of Medicine, Yokohama, Japan.
Oda K; Cardiovascular Research Institute, Yokohama City University School of Medicine, Yokohama, Japan.
Makino A; Cardiovascular Research Institute, Yokohama City University School of Medicine, Yokohama, Japan.
Fujita T; Cardiovascular Research Institute, Yokohama City University School of Medicine, Yokohama, Japan.
Yokoyama U; Cardiovascular Research Institute, Yokohama City University School of Medicine, Yokohama, Japan.
Iwatsubo M; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
Chen S; Department of Chemical Biology, Susan Lehman Cullen Laboratory of Cancer Research in the Ernest Mario School of Pharmacy, Rutgers, The State University of New Jersey, Piscataway, New Jersey, United States of America.
Goydos JS; Division of Surgical Oncology, Department of Surgery, Robert Wood Johnson Medical School, Rutgers, The State University of New Jersey, Piscataway, New Jersey, United States of America.
Ishikawa Y; Cardiovascular Research Institute, Yokohama City University School of Medicine, Yokohama, Japan.
Iwatsubo K; Cardiovascular Research Institute, Yokohama City University School of Medicine, Yokohama, Japan ; Department of Cell Biology and Molecular Medicine, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey, United States of America.
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Źródło:
PloS one [PLoS One] 2014 Feb 21; Vol. 9 (2), pp. e89292. Date of Electronic Publication: 2014 Feb 21 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Calcium/*metabolism
Calcium Signaling/*physiology
Cell Movement/*physiology
Cell Proliferation/*physiology
Melanocytes/*metabolism
Melanoma/*metabolism
Skin Neoplasms/*metabolism
Animals ; Calcium Channels/metabolism ; Cell Line, Tumor ; Endoplasmic Reticulum/metabolism ; Humans ; Melanoma/pathology ; Mice ; Skin Neoplasms/pathology
Czasopismo naukowe
Tytuł:
Modulation of intracellular calcium waves and triggered activities by mitochondrial ca flux in mouse cardiomyocytes.
Autorzy:
Zhao Z; Department of Pharmacology, School of Medicine, Xi'an Jiaotong University, Xi'an, China ; Department of Cell Biology and Molecular Medicine, Rutgers, New Jersey Medical School, Newark, New Jersey, United States of America.
Gordan R; Department of Cell Biology and Molecular Medicine, Rutgers, New Jersey Medical School, Newark, New Jersey, United States of America.
Wen H; Department of Cell Biology and Molecular Medicine, Rutgers, New Jersey Medical School, Newark, New Jersey, United States of America ; Department of Reproductive and Genetic Toxicology, National Center for Safety Evaluation of Drugs, National Institutes for Food and Drug Control,  Beijing, P.R. China.
Fefelova N; Department of Cell Biology and Molecular Medicine, Rutgers, New Jersey Medical School, Newark, New Jersey, United States of America.
Zang WJ; Department of Pharmacology, School of Medicine, Xi'an Jiaotong University, Xi'an, China.
Xie LH; Department of Cell Biology and Molecular Medicine, Rutgers, New Jersey Medical School, Newark, New Jersey, United States of America.
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Źródło:
PloS one [PLoS One] 2013 Nov 07; Vol. 8 (11), pp. e80574. Date of Electronic Publication: 2013 Nov 07 (Print Publication: 2013).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Calcium/*metabolism
Mitochondria/*drug effects
Mitochondria/*metabolism
Myocytes, Cardiac/*drug effects
Myocytes, Cardiac/*metabolism
Animals ; Antimycin A/pharmacology ; Calcium Signaling/drug effects ; Cells, Cultured ; Kaempferols/pharmacology ; Membrane Potential, Mitochondrial/drug effects ; Mice ; Oligomycins/pharmacology
Czasopismo naukowe
Tytuł:
Ginsenoside Re: pharmacological effects on cardiovascular system.
Autorzy:
Peng L; Section of Cardiology, Internal Medicine, JiShuiTan Hospital, Beijing 100035, China.
Sun S
Xie LH
Wicks SM
Xie JT
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Źródło:
Cardiovascular therapeutics [Cardiovasc Ther] 2012 Aug; Vol. 30 (4), pp. e183-8. Date of Electronic Publication: 2011 Apr 01.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Review
MeSH Terms:
Cardiovascular Agents/*pharmacology
Cardiovascular System/*drug effects
Ginsenosides/*pharmacology
Action Potentials ; Angiogenesis Inducing Agents/pharmacology ; Animals ; Anti-Arrhythmia Agents/pharmacology ; Cardiotonic Agents/pharmacology ; Cardiovascular Agents/chemistry ; Cardiovascular System/physiopathology ; Dose-Response Relationship, Drug ; Ginsenosides/chemistry ; Heart Conduction System/drug effects ; Heart Conduction System/physiopathology ; Heart Rate/drug effects ; Humans ; Myocardial Contraction/drug effects ; Neovascularization, Physiologic/drug effects
Czasopismo naukowe
    Wyświetlanie 1-12 z 12

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