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Wyświetlanie 1-5 z 5
Tytuł :
Bone marrow cells are differentiated into MDSCs by BCC-Ex through down-regulating the expression of CXCR4 and activating STAT3 signalling pathway.
Autorzy :
Liu QW; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Chen Y; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Li JY; School of Chemistry, Biology and Material Science, East China University of Technology, Nanchang, China.
Xiao L; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Zhang WJ; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Zhao JL; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Gu HC; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life and Science, Nanchang University, Nanchang, China.
Wu HY; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Zuo GS; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Deng KY; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life and Science, Nanchang University, Nanchang, China.
Xin HB; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life and Science, Nanchang University, Nanchang, China.
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Źródło :
Journal of cellular and molecular medicine [J Cell Mol Med] 2021 May 06. Date of Electronic Publication: 2021 May 06.
Typ publikacji :
Journal Article
Czasopismo naukowe
Tytuł :
Human amniotic mesenchymal stem cells inhibit hepatocellular carcinoma in tumour-bearing mice.
Autorzy :
Liu QW; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Li JY; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life and Science, Nanchang University, Nanchang, China.
Zhang XC; Department of Obstetrics and Gynecology, The First Affiliated Hospital of Nanchang University, Nanchang, China.
Liu Y; Department of Respiratory and Critical Care Medicine, The First Affiliated Hospital of Nanchang University, Nanchang, China.
Liu QY; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Xiao L; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Zhang WJ; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Wu HY; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Deng KY; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life and Science, Nanchang University, Nanchang, China.
Xin HB; The National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life and Science, Nanchang University, Nanchang, China.
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Źródło :
Journal of cellular and molecular medicine [J Cell Mol Med] 2020 Sep; Vol. 24 (18), pp. 10525-10541. Date of Electronic Publication: 2020 Aug 14.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Mesenchymal Stem Cell Transplantation*
Amnion/*cytology
Carcinoma, Hepatocellular/*therapy
Liver Neoplasms/*therapy
Adaptor Proteins, Signal Transducing/antagonists & inhibitors ; Adaptor Proteins, Signal Transducing/genetics ; Adaptor Proteins, Signal Transducing/physiology ; Adipogenesis ; Animals ; Apoptosis ; Carcinoma, Hepatocellular/pathology ; Female ; Genes, Reporter ; Hep G2 Cells/transplantation ; Humans ; Insulin-Like Growth Factor Binding Protein 3/antagonists & inhibitors ; Insulin-Like Growth Factor Binding Protein 3/genetics ; Insulin-Like Growth Factor Binding Protein 3/physiology ; Intercellular Signaling Peptides and Proteins/genetics ; Intercellular Signaling Peptides and Proteins/physiology ; Liver Neoplasms/pathology ; Male ; Mesenchymal Stem Cells/cytology ; Mesenchymal Stem Cells/metabolism ; Mice ; Mice, Inbred BALB C ; Mice, Inbred NOD ; Mice, Nude ; Mice, SCID ; Neoplasm Proteins/antagonists & inhibitors ; Neoplasm Proteins/genetics ; Neoplasm Proteins/physiology ; Osteogenesis ; Paracrine Communication ; Pregnancy ; RNA Interference ; RNA, Small Interfering/genetics ; RNA, Small Interfering/pharmacology ; Signal Transduction ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
FKBP12.6 protects heart from AngII-induced hypertrophy through inhibiting Ca /calmodulin-mediated signalling pathways in vivo and in vitro.
Autorzy :
Xiao YF; Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life Science, Nanchang University, Nanchang, China.
Zeng ZX; Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life Science, Nanchang University, Nanchang, China.
Guan XH; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Wang LF; Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life Science, Nanchang University, Nanchang, China.
Wang CJ; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Shi H; Georgia Cancer Center, Augusta University, Augusta, GA, USA.
Shou W; Riley Heart Research Center, Herman B Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, IN, USA.
Deng KY; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Xin HB; Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life Science, Nanchang University, Nanchang, China.
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Źródło :
Journal of cellular and molecular medicine [J Cell Mol Med] 2018 Jul; Vol. 22 (7), pp. 3638-3651. Date of Electronic Publication: 2018 Apr 22.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Calcium/*metabolism
Calmodulin/*metabolism
Cardiomegaly/*metabolism
Tacrolimus Binding Proteins/*metabolism
Angiotensin II/metabolism ; Angiotensin II/toxicity ; Animals ; Calcineurin/metabolism ; Calcium-Calmodulin-Dependent Protein Kinase Type 2/metabolism ; Cardiomegaly/chemically induced ; Cardiomegaly/pathology ; Cell Line ; Gene Expression ; Glycogen Synthase Kinase 3 beta/metabolism ; Male ; Mice, Inbred C57BL ; Mice, Transgenic ; Myocardium/metabolism ; Myocardium/pathology ; Myocytes, Cardiac/metabolism ; Myocytes, Cardiac/pathology ; Proto-Oncogene Proteins c-akt/metabolism ; Tacrolimus Binding Proteins/genetics
Czasopismo naukowe
Tytuł :
CD38 deficiency suppresses adipogenesis and lipogenesis in adipose tissues through activating Sirt1/PPARγ signaling pathway.
Autorzy :
Wang LF; Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life Sciences, Nanchang University, Nanchang, China.
Miao LJ; Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life Sciences, Nanchang University, Nanchang, China.
Wang XN; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Huang CC; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Qian YS; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Huang X; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Wang XL; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Jin WZ; Institute of Zoology, Chinese Academy of Sciences, Beijing, China.
Ji GJ; National Laboratory of Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Beijing, China.
Fu M; Department of Basic Medical Science, Shock/Trauma Research Center, School of Medicine, University of Missouri Kansas City, Kansas City, MO, USA.
Deng KY; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Xin HB; Institute of Translational Medicine, Nanchang University, Nanchang, China.
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Źródło :
Journal of cellular and molecular medicine [J Cell Mol Med] 2018 Jan; Vol. 22 (1), pp. 101-110. Date of Electronic Publication: 2017 Aug 16.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Adipogenesis*
Lipogenesis*
Signal Transduction*
ADP-ribosyl Cyclase 1/*deficiency
Adipose Tissue/*metabolism
PPAR gamma/*metabolism
Sirtuin 1/*metabolism
ADP-ribosyl Cyclase 1/metabolism ; Adipocytes/metabolism ; Animals ; Cell Differentiation ; Embryo, Mammalian/cytology ; Fibroblasts/metabolism ; Mice ; NAD/metabolism
Czasopismo naukowe
Tytuł :
CD38 promotes angiotensin II-induced cardiac hypertrophy.
Autorzy :
Guan XH; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Hong X; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Zhao N; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Liu XH; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Xiao YF; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Chen TT; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Deng LB; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Wang XL; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Wang JB; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Ji GJ; National Laboratory of Biomacromolecules, Institute of Biophysics Chinese Academy of Sciences, Beijing, China.
Fu M; Department of Basic Medical Science, Shock/Trauma Research Center, School of Medicine, University of Missouri Kansas City, Kansas City, MO, USA.
Deng KY; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Xin HB; Institute of Translational Medicine, Nanchang University, Nanchang, China.
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Źródło :
Journal of cellular and molecular medicine [J Cell Mol Med] 2017 Aug; Vol. 21 (8), pp. 1492-1502. Date of Electronic Publication: 2017 Mar 12.
Typ publikacji :
Journal Article
MeSH Terms :
ADP-ribosyl Cyclase 1/*genetics
Angiotensin II/*pharmacology
Cardiomegaly/*genetics
Membrane Glycoproteins/*genetics
Myocardium/*metabolism
Myocytes, Cardiac/*metabolism
ADP-ribosyl Cyclase 1/antagonists & inhibitors ; ADP-ribosyl Cyclase 1/deficiency ; Animals ; Atrial Natriuretic Factor/genetics ; Atrial Natriuretic Factor/metabolism ; Calcium/metabolism ; Cardiomegaly/chemically induced ; Cardiomegaly/metabolism ; Cardiomegaly/pathology ; Cell Line ; Forkhead Box Protein O3/genetics ; Forkhead Box Protein O3/metabolism ; Gene Expression Regulation ; Membrane Glycoproteins/antagonists & inhibitors ; Membrane Glycoproteins/deficiency ; Mice ; Mice, Knockout ; Mitogen-Activated Protein Kinase 1/genetics ; Mitogen-Activated Protein Kinase 1/metabolism ; Mitogen-Activated Protein Kinase 3/genetics ; Mitogen-Activated Protein Kinase 3/metabolism ; Myocardium/pathology ; Myocytes, Cardiac/drug effects ; Myocytes, Cardiac/pathology ; NFATC Transcription Factors/genetics ; NFATC Transcription Factors/metabolism ; Natriuretic Peptide, Brain/genetics ; Natriuretic Peptide, Brain/metabolism ; Nerve Tissue Proteins/genetics ; Nerve Tissue Proteins/metabolism ; Phosphorylation ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Rats ; Reactive Oxygen Species/metabolism ; Signal Transduction ; Sirtuins/genetics ; Sirtuins/metabolism
Czasopismo naukowe
    Wyświetlanie 1-5 z 5

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