Informacja

Drogi użytkowniku, aplikacja do prawidłowego działania wymaga obsługi JavaScript. Proszę włącz obsługę JavaScript w Twojej przeglądarce.

Przeglądasz jako GOŚĆ

Wyszukujesz frazę ""Xin HB"" wg kryterium: Autor


Wyświetlanie 1-7 z 7
Tytuł :
CD38 Deficiency Promotes Inflammatory Response through Activating Sirt1/NF- κ B-Mediated Inhibition of TLR2 Expression in Macrophages.
Autorzy :
Qian Y; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Chen C; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Ma L; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Wang Z; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Wang LF; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Zuo L; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Yang Y; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Huang X; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Jiang M; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Wang X; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Shi H; Georgia Cancer Center, Augusta University, Augusta, GA 30912, USA.
Fu M; Department of Basic Medical Science, Shock/Trauma Research Center, School of Medicine, University of Missouri-Kansas City, Kansas City, MO 64108, USA.
Deng KY; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Xin HB; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Pokaż więcej
Źródło :
Mediators of inflammation [Mediators Inflamm] 2018 May 27; Vol. 2018, pp. 8736949. Date of Electronic Publication: 2018 May 27 (Print Publication: 2018).
Typ publikacji :
Journal Article
MeSH Terms :
ADP-ribosyl Cyclase 1/*deficiency
Inflammation/*metabolism
Macrophages/*metabolism
Macrophages, Peritoneal/*metabolism
NF-kappa B/*metabolism
Sirtuin 1/*metabolism
ADP-ribosyl Cyclase 1/genetics ; ADP-ribosyl Cyclase 1/metabolism ; Animals ; Blotting, Western ; Computational Biology ; Inflammation/genetics ; Mice ; RAW 264.7 Cells ; Signal Transduction/genetics ; Signal Transduction/physiology ; Sirtuin 1/genetics ; Toll-Like Receptor 2/metabolism
Czasopismo naukowe
Tytuł :
CD38 Deficiency Protects Heart from High Fat Diet-Induced Oxidative Stress Via Activating Sirt3/FOXO3 Pathway.
Autorzy :
Wang LF; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Huang CC; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Xiao YF; Institute of Translational Medicine, Nanchang University, Nanchang, China.; College of Life Sciences, Nanchang University, Nanchang, China.
Guan XH; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Wang XN; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Cao Q; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Liu Y; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Huang X; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Deng LB; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Deng KY; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Xin HB; Institute of Translational Medicine, Nanchang University, Nanchang, China.; College of Life Sciences, Nanchang University, Nanchang, China.
Pokaż więcej
Źródło :
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology [Cell Physiol Biochem] 2018; Vol. 48 (6), pp. 2350-2363. Date of Electronic Publication: 2018 Aug 16.
Typ publikacji :
Journal Article
MeSH Terms :
Diet, High-Fat*
Oxidative Stress*
ADP-ribosyl Cyclase 1/*genetics
Forkhead Box Protein O3/*metabolism
Membrane Glycoproteins/*genetics
Sirtuin 3/*metabolism
ADP-ribosyl Cyclase 1/antagonists & inhibitors ; ADP-ribosyl Cyclase 1/metabolism ; Animals ; Cell Line ; Glutathione/metabolism ; Male ; Membrane Glycoproteins/antagonists & inhibitors ; Membrane Glycoproteins/metabolism ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Myocardium/metabolism ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; RNA Interference ; RNA, Small Interfering/metabolism ; Rats ; Reactive Oxygen Species/metabolism ; Signal Transduction ; Superoxide Dismutase/metabolism
Czasopismo naukowe
Tytuł :
CD38 deficiency suppresses adipogenesis and lipogenesis in adipose tissues through activating Sirt1/PPARγ signaling pathway.
Autorzy :
Wang LF; Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life Sciences, Nanchang University, Nanchang, China.
Miao LJ; Institute of Translational Medicine, Nanchang University, Nanchang, China.; School of Life Sciences, Nanchang University, Nanchang, China.
Wang XN; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Huang CC; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Qian YS; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Huang X; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Wang XL; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Jin WZ; Institute of Zoology, Chinese Academy of Sciences, Beijing, China.
Ji GJ; National Laboratory of Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Beijing, China.
Fu M; Department of Basic Medical Science, Shock/Trauma Research Center, School of Medicine, University of Missouri Kansas City, Kansas City, MO, USA.
Deng KY; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Xin HB; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Pokaż więcej
Źródło :
Journal of cellular and molecular medicine [J Cell Mol Med] 2018 Jan; Vol. 22 (1), pp. 101-110. Date of Electronic Publication: 2017 Aug 16.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Adipogenesis*
Lipogenesis*
Signal Transduction*
ADP-ribosyl Cyclase 1/*deficiency
Adipose Tissue/*metabolism
PPAR gamma/*metabolism
Sirtuin 1/*metabolism
ADP-ribosyl Cyclase 1/metabolism ; Adipocytes/metabolism ; Animals ; Cell Differentiation ; Embryo, Mammalian/cytology ; Fibroblasts/metabolism ; Mice ; NAD/metabolism
Czasopismo naukowe
Tytuł :
CD38 promotes angiotensin II-induced cardiac hypertrophy.
Autorzy :
Guan XH; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Hong X; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Zhao N; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Liu XH; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Xiao YF; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Chen TT; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Deng LB; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Wang XL; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Wang JB; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Ji GJ; National Laboratory of Biomacromolecules, Institute of Biophysics Chinese Academy of Sciences, Beijing, China.
Fu M; Department of Basic Medical Science, Shock/Trauma Research Center, School of Medicine, University of Missouri Kansas City, Kansas City, MO, USA.
Deng KY; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Xin HB; Institute of Translational Medicine, Nanchang University, Nanchang, China.
Pokaż więcej
Źródło :
Journal of cellular and molecular medicine [J Cell Mol Med] 2017 Aug; Vol. 21 (8), pp. 1492-1502. Date of Electronic Publication: 2017 Mar 12.
Typ publikacji :
Journal Article
MeSH Terms :
ADP-ribosyl Cyclase 1/*genetics
Angiotensin II/*pharmacology
Cardiomegaly/*genetics
Membrane Glycoproteins/*genetics
Myocardium/*metabolism
Myocytes, Cardiac/*metabolism
ADP-ribosyl Cyclase 1/antagonists & inhibitors ; ADP-ribosyl Cyclase 1/deficiency ; Animals ; Atrial Natriuretic Factor/genetics ; Atrial Natriuretic Factor/metabolism ; Calcium/metabolism ; Cardiomegaly/chemically induced ; Cardiomegaly/metabolism ; Cardiomegaly/pathology ; Cell Line ; Forkhead Box Protein O3/genetics ; Forkhead Box Protein O3/metabolism ; Gene Expression Regulation ; Membrane Glycoproteins/antagonists & inhibitors ; Membrane Glycoproteins/deficiency ; Mice ; Mice, Knockout ; Mitogen-Activated Protein Kinase 1/genetics ; Mitogen-Activated Protein Kinase 1/metabolism ; Mitogen-Activated Protein Kinase 3/genetics ; Mitogen-Activated Protein Kinase 3/metabolism ; Myocardium/pathology ; Myocytes, Cardiac/drug effects ; Myocytes, Cardiac/pathology ; NFATC Transcription Factors/genetics ; NFATC Transcription Factors/metabolism ; Natriuretic Peptide, Brain/genetics ; Natriuretic Peptide, Brain/metabolism ; Nerve Tissue Proteins/genetics ; Nerve Tissue Proteins/metabolism ; Phosphorylation ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Rats ; Reactive Oxygen Species/metabolism ; Signal Transduction ; Sirtuins/genetics ; Sirtuins/metabolism
Czasopismo naukowe
Tytuł :
CD38 Deficiency Protects the Heart from Ischemia/Reperfusion Injury through Activating SIRT1/FOXOs-Mediated Antioxidative Stress Pathway.
Autorzy :
Guan XH; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Liu XH; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Hong X; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Zhao N; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Xiao YF; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Wang LF; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Tang L; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Jiang K; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Qian YS; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Deng KY; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Ji G; National Laboratory of Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Beijing 100101, China.
Fu M; Department of Basic Medical Science, Shock/Trauma Research Center, School of Medicine, University of Missouri-Kansas City, Kansas City, MO 64108, USA.
Xin HB; Institute of Translational Medicine, Nanchang University, Nanchang 330031, China.
Pokaż więcej
Źródło :
Oxidative medicine and cellular longevity [Oxid Med Cell Longev] 2016; Vol. 2016, pp. 7410257. Date of Electronic Publication: 2016 Jul 31.
Typ publikacji :
Journal Article
MeSH Terms :
Oxidative Stress*
ADP-ribosyl Cyclase/*deficiency
ADP-ribosyl Cyclase 1/*deficiency
Forkhead Box Protein O3/*metabolism
Membrane Glycoproteins/*deficiency
Myocardial Infarction/*prevention & control
Myocardial Reperfusion Injury/*prevention & control
Myocardium/*enzymology
Nerve Tissue Proteins/*metabolism
Sirtuin 1/*metabolism
ADP-ribosyl Cyclase/genetics ; ADP-ribosyl Cyclase 1/genetics ; Animals ; Apoptosis ; Calcium/metabolism ; Cell Line ; Disease Models, Animal ; Genotype ; Male ; Membrane Glycoproteins/genetics ; Mice, Inbred C57BL ; Mice, Knockout ; Myocardial Infarction/enzymology ; Myocardial Infarction/genetics ; Myocardial Infarction/pathology ; Myocardial Reperfusion Injury/enzymology ; Myocardial Reperfusion Injury/genetics ; Myocardial Reperfusion Injury/pathology ; Myocardium/pathology ; NADPH Oxidase 4 ; NADPH Oxidases/metabolism ; Phenotype ; RNA Interference ; Rats ; Reactive Oxygen Species/metabolism ; Signal Transduction ; Transfection
Czasopismo naukowe
Tytuł :
Disruption of CD38 gene enhances cardiac functions by elevating serum testosterone in the male null mice.
Autorzy :
Gan L; Laboratory of Cardiovascular Disease, State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, Chengdu 610041, PR China.
Jiang W
Xiao YF
Deng L
Gu LD
Guo ZY
Zhou ZC
Wu D
Xin HB
Pokaż więcej
Źródło :
Life sciences [Life Sci] 2011 Sep 26; Vol. 89 (13-14), pp. 491-7. Date of Electronic Publication: 2011 Aug 03.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Myocardial Contraction*
ADP-ribosyl Cyclase 1/*genetics
Heart/*physiology
Testosterone/*blood
Androgen Antagonists/pharmacology ; Animals ; Calcium/metabolism ; Contractile Proteins/genetics ; Echocardiography ; Flutamide/pharmacology ; Gene Deletion ; Gene Expression Regulation ; Heart/drug effects ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; RNA, Messenger/genetics ; Receptors, Androgen/genetics
Czasopismo naukowe
Tytuł :
Mouse embryonic fibroblasts from CD38 knockout mice are resistant to oxidative stresses through inhibition of reactive oxygen species production and Ca(2+) overload.
Autorzy :
Ge Y; Laboratory of Cardiovascular Diseases, West China Hospital, The State Key Laboratory of Biotherapy, Sichuan University, Chengdu 610041, People's Republic of China.
Jiang W
Gan L
Wang L
Sun C
Ni P
Liu Y
Wu S
Gu L
Zheng W
Lund FE
Xin HB
Pokaż więcej
Źródło :
Biochemical and biophysical research communications [Biochem Biophys Res Commun] 2010 Aug 20; Vol. 399 (2), pp. 167-72. Date of Electronic Publication: 2010 Jul 16.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
ADP-ribosyl Cyclase 1/*genetics
Calcium/*metabolism
Oxidative Stress/*genetics
Reactive Oxygen Species/*metabolism
Animals ; Embryo, Mammalian/cytology ; Fibroblasts/enzymology ; Mice ; Mice, Knockout ; NADH, NADPH Oxidoreductases/biosynthesis ; NADPH Oxidase 1
Czasopismo naukowe
    Wyświetlanie 1-7 z 7

    Ta witryna wykorzystuje pliki cookies do przechowywania informacji na Twoim komputerze. Pliki cookies stosujemy w celu świadczenia usług na najwyższym poziomie, w tym w sposób dostosowany do indywidualnych potrzeb. Korzystanie z witryny bez zmiany ustawień dotyczących cookies oznacza, że będą one zamieszczane w Twoim komputerze. W każdym momencie możesz dokonać zmiany ustawień dotyczących cookies