Autor: "Y. Lu" - OpacWWW - Prolib Integro

Skocz do pozycji: 1.

Tytuł:: Cuproptosis-related risk score based on machine learning algorithm predicts prognosis and characterizes tumor microenvironment in head and neck squamous carcinomas.
Autorzy:: Ye M; Medical Cosmetic Center, First Affiliated Hospital of Shantou University Medical College, Shantou, 515041, Guangdong, People's Republic of China. .
Zhang G; Shantou University Medical College, Shantou, 515041, Guangdong, People's Republic of China.
Lu Y; Shantou University Medical College, Shantou, 515041, Guangdong, People's Republic of China.
Ren S; Medical Cosmetic Center, First Affiliated Hospital of Shantou University Medical College, Shantou, 515041, Guangdong, People's Republic of China. .
Ji Y; Medical Cosmetic Center, First Affiliated Hospital of Shantou University Medical College, Shantou, 515041, Guangdong, People's Republic of China. .; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2023 Jul 22; Vol. 13 (1), pp. 11870. Date of Electronic Publication: 2023 Jul 22.
Typ publikacji:: Journal Article
MeSH Terms:: Head and Neck Neoplasms*
Tumor Microenvironment*
Apoptosis*
Humans ; Algorithms ; Machine Learning ; Prognosis ; Risk Factors ; Squamous Cell Carcinoma of Head and Neck ; Copper

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Skocz do pozycji: 2.

Tytuł:: Astaxanthin ameliorates oxidative stress and neuronal apoptosis via SIRT1/NRF2/Prx2/ASK1/p38 after traumatic brain injury in mice.
Autorzy:: Zhang XS; Department of Neurosurgery, Beijing Friendship Hospital, Capital Medical University, Beijing, China.; Department of Neurosurgery, Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medical School, Nanjing, China.
Lu Y; Department of Neurosurgery, Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medical School, Nanjing, China.
Li W; Department of Pharmacy, Beijing Boai Hospital, China Rehabilitation Research Center, Capital Medical University, Beijing, China.
Tao T; Department of Neurosurgery, Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medical School, Nanjing, China.
Peng L; Department of Neurosurgery, Beijing Friendship Hospital, Capital Medical University, Beijing, China.
Wang WH; Department of Neurosurgery, Beijing Friendship Hospital, Capital Medical University, Beijing, China.
Gao S; Department of Neurosurgery, Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medical School, Nanjing, China.
Liu C; Department of Neurosurgery, Beijing Friendship Hospital, Capital Medical University, Beijing, China.
Zhuang Z; Department of Neurosurgery, Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medical School, Nanjing, China.
Xia DY; Department of Neurosurgery, The First Affiliated Hospital of Wannan Medical College, Wuhu, China.
Hang CH; Department of Neurosurgery, Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medical School, Nanjing, China.
Li W; Department of Neurosurgery, Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medical School, Nanjing, China.; Pokaż więcej
Źródło:: British journal of pharmacology [Br J Pharmacol] 2021 Mar; Vol. 178 (5), pp. 1114-1132. Date of Electronic Publication: 2021 Jan 15.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis*
Brain Injuries, Traumatic*/drug therapy
Oxidative Stress*
Xanthophylls/*pharmacology
Animals ; MAP Kinase Kinase Kinase 5 ; Mice ; Mice, Knockout ; NF-E2-Related Factor 2/metabolism ; Peroxiredoxins ; Sirtuin 1/metabolism ; p38 Mitogen-Activated Protein Kinases

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Skocz do pozycji: 3.

Tytuł:: Knockdown of GCN2 inhibits high glucose-induced oxidative stress and apoptosis in retinal pigment epithelial cells.
Autorzy:: Zhang X; Department of Ophthalmology, The Second Affiliated Hospital, Medical College, Xi'an Jiaotong University, Xi'an, China.
He N; Department of Ophthalmology, The Second Affiliated Hospital, Medical College, Xi'an Jiaotong University, Xi'an, China.
Xing Y; Department of Ophthalmology, The Second Affiliated Hospital, Medical College, Xi'an Jiaotong University, Xi'an, China.
Lu Y; Department of Ophthalmology, The Second Affiliated Hospital, Medical College, Xi'an Jiaotong University, Xi'an, China.; Pokaż więcej
Źródło:: Clinical and experimental pharmacology & physiology [Clin Exp Pharmacol Physiol] 2020 Apr; Vol. 47 (4), pp. 591-598. Date of Electronic Publication: 2020 Jan 23.
Typ publikacji:: Journal Article
MeSH Terms:: Gene Knockdown Techniques*
Apoptosis/*genetics
Glucose/*pharmacology
Oxidative Stress/*genetics
Protein Serine-Threonine Kinases/*deficiency
Protein Serine-Threonine Kinases/*genetics
Retinal Pigment Epithelium/*cytology
Apoptosis/drug effects ; Cell Line ; Dose-Response Relationship, Drug ; Heme Oxygenase-1/metabolism ; Humans ; NF-E2-Related Factor 2/metabolism ; Oxidative Stress/drug effects

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Skocz do pozycji: 4.

Tytuł:: Transient Receptor Potential Canonical 5-Scramblase Signaling Complex Mediates Neuronal Phosphatidylserine Externalization and Apoptosis.
Autorzy:: Guo J; School of Basic Medical Sciences, Anhui Medical University, Hefei, Anhui 230032, China.
Li J; School of Basic Medical Sciences, Anhui Medical University, Hefei, Anhui 230032, China.
Xia L; School of Basic Medical Sciences, Anhui Medical University, Hefei, Anhui 230032, China.
Wang Y; School of Basic Medical Sciences, Anhui Medical University, Hefei, Anhui 230032, China.
Zhu J; School of Basic Medical Sciences, Anhui Medical University, Hefei, Anhui 230032, China.
Du J; School of Basic Medical Sciences, Anhui Medical University, Hefei, Anhui 230032, China.
Lu Y; Brown Foundation Institute of Molecular Medicine, University of Texas, McGovern Medical School, Houston, TX 77030, USA.
Liu G; Institute of Biomedical and Health, School of Life and Health Science, Anhui Science & Technology University, Fengyang, Anhui 233100, China.
Yao X; School of Biomedical Sciences, Chinese University of Hong Kong, Hong Kong, China.
Shen B; School of Basic Medical Sciences, Anhui Medical University, Hefei, Anhui 230032, China.; Pokaż więcej
Źródło:: Cells [Cells] 2020 Feb 26; Vol. 9 (3). Date of Electronic Publication: 2020 Feb 26.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis*
Exocytosis*
Neurons/*metabolism
Phosphatidylserines/*metabolism
Phospholipid Transfer Proteins/*metabolism
TRPC Cation Channels/*metabolism
Animals ; Cell Membrane/metabolism ; HEK293 Cells ; Humans ; Ion Channel Gating ; Mice, Knockout ; Protein Binding ; Reperfusion Injury/pathology ; Reperfusion Injury/physiopathology ; Signal Transduction

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Skocz do pozycji: 5.

Tytuł:: N -Methylparoxetine Blocked Autophagic Flux and Induced Apoptosis by Activating ROS-MAPK Pathway in Non-Small Cell Lung Cancer Cells.
Autorzy:: Wang K; Research Center of Integrative Medicine, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.; Department of Pathology, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Chen B; Research Center of Integrative Medicine, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.; Department of Biochemistry, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Yin T; Research Center of Integrative Medicine, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.; Department of Biochemistry, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Zhan Y; Department of Biochemistry, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Lu Y; Department of Pathology, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Zhang Y; Department of Pathology, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Chen J; Department of Biochemistry, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Wu W; Department of Biochemistry, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Zhou S; Department of Biochemistry, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Mao W; Research Center of Integrative Medicine, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.; Department of Pathology, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Tan Y; Department of Biochemistry, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Du B; Department of Pathology, Guangzhou University of Chinese Medicine, Guangzhou 510006, China.
Liu X; Department of Anaesthesia and Intensive Care, The Chinese University of Hong Kong, Hong Kong SAR 999077, China.
Ho HI; Department of Anaesthesia and Intensive Care, The Chinese University of Hong Kong, Hong Kong SAR 999077, China. .
Xiao J; Research Center of Integrative Medicine, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou 510006, China. .; Department of Biochemistry, Guangzhou University of Chinese Medicine, Guangzhou 510006, China. .; Pokaż więcej
Źródło:: International journal of molecular sciences [Int J Mol Sci] 2019 Jul 11; Vol. 20 (14). Date of Electronic Publication: 2019 Jul 11.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Autophagy/*drug effects
Carcinoma, Non-Small-Cell Lung/*metabolism
Lung Neoplasms/*metabolism
MAP Kinase Signaling System/*drug effects
Paroxetine/*pharmacology
Reactive Oxygen Species/*metabolism
Cell Line, Tumor ; Cell Proliferation/drug effects ; Humans ; JNK Mitogen-Activated Protein Kinases/metabolism ; Lysosomes/metabolism ; Mitochondria/drug effects ; Mitochondria/metabolism ; Molecular Structure ; Paroxetine/analogs & derivatives ; Paroxetine/chemistry ; p38 Mitogen-Activated Protein Kinases/metabolism

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Skocz do pozycji: 6.

Tytuł:: Small nucleolar RNA Sf-15 regulates proliferation and apoptosis of Spodoptera frugiperda Sf9 cells.
Autorzy:: Wu B; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Huang L; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Qiu W; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Liu X; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Shen Y; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Lu Y; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Yang Z; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Li X; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Cui B; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Xu S; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Qiao H; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Qiu R; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Yao L; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China.
Kan Y; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China. .
Li D; China-UK-NYNU-RRes Joint Laboratory of Insect Biology, Henan Key Laboratory of Insect Biology in Funiu Mountain, Nanyang Normal University, 1638 Wolong Road, Nanyang, 473061, Henan, China. .; Pokaż więcej
Źródło:: BMC molecular biology [BMC Mol Biol] 2019 Apr 11; Vol. 20 (1), pp. 12. Date of Electronic Publication: 2019 Apr 11.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis/*genetics
Cell Proliferation/*genetics
RNA, Small Nucleolar/*genetics
Spodoptera/*genetics
Animals ; Gene Expression Profiling ; Sf9 Cells

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Skocz do pozycji: 7.

Tytuł:: Liraglutide protects renal mesangial cells against hyperglycemia‑mediated mitochondrial apoptosis by activating the ERK‑Yap signaling pathway and upregulating Sirt3 expression.
Autorzy:: Li J; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Li N; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Yan S; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Lu Y; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Miao X; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Gu Z; Department of Geriatric Endocrinology, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.
Shao Y; Department of Outpatients, Chinese People's Liberation Army General Hospital, Beijing 100853, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2019 Apr; Vol. 19 (4), pp. 2849-2860. Date of Electronic Publication: 2019 Feb 08.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Hyperglycemia/*metabolism
Liraglutide/*pharmacology
Mesangial Cells/*drug effects
Mesangial Cells/*metabolism
Mitochondria/*drug effects
Mitochondria/*metabolism
Cell Cycle Proteins ; Cells, Cultured ; Dose-Response Relationship, Drug ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Gene Expression Regulation/drug effects ; Glucose/metabolism ; Hyperglycemia/genetics ; Ion Channel Gating ; Membrane Potential, Mitochondrial/drug effects ; Mitochondrial Membrane Transport Proteins/metabolism ; Mitochondrial Permeability Transition Pore ; Nuclear Proteins/metabolism ; Oxidative Stress/drug effects ; Protective Agents/pharmacology ; Reactive Oxygen Species/metabolism ; Signal Transduction/drug effects ; Sirtuin 3/genetics ; Transcription Factors/metabolism

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Skocz do pozycji: 8.

Tytuł:: Glucagon-like peptide 2 decreases osteoclasts by stimulating apoptosis dependent on nitric oxide synthase.
Autorzy:: Lu Y; Departments of Geriatrics, Zhongshan Hospital, Fudan University, Shanghai, China.
Lu D; School of Life Science and Technology, Tongji University, Shanghai, China.
Hu Y; Departments of Geriatrics, Zhongshan Hospital, Fudan University, Shanghai, China.; Pokaż więcej
Źródło:: Cell proliferation [Cell Prolif] 2018 Aug; Vol. 51 (4), pp. e12443. Date of Electronic Publication: 2018 Feb 19.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis*/drug effects
Glucagon-Like Peptide 2/*metabolism
Nitric Oxide Synthase Type II/*metabolism
Animals ; Caspase 3/metabolism ; Cell Differentiation ; Female ; Glucagon-Like Peptide 2/genetics ; Mice ; Nitric Oxide/metabolism ; Osteoclasts/cytology ; Osteoclasts/metabolism ; Proto-Oncogene Proteins c-bcl-2/metabolism ; RANK Ligand/pharmacology ; RAW 264.7 Cells ; Rats ; Rats, Sprague-Dawley ; Signal Transduction/drug effects ; Smad2 Protein/metabolism ; Smad3 Protein/metabolism ; Tartrate-Resistant Acid Phosphatase/blood ; Transforming Growth Factor beta/metabolism

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Skocz do pozycji: 9.

Tytuł:: Salvianolic acid A, a novel PI3K/Akt inhibitor, induces cell apoptosis and suppresses tumor growth in acute myeloid leukemia.
Autorzy:: Pei R; a Department of Pathology, Zhejiang Provincial Key Laboratory of Pathophysiology , Ningbo University School of Medicine , Ningbo , China.; b Department of Hematology , Yinzhou People's Hospital, Ningbo University School of Medicine , Ningbo , China.
Si T; a Department of Pathology, Zhejiang Provincial Key Laboratory of Pathophysiology , Ningbo University School of Medicine , Ningbo , China.; b Department of Hematology , Yinzhou People's Hospital, Ningbo University School of Medicine , Ningbo , China.
Lu Y; a Department of Pathology, Zhejiang Provincial Key Laboratory of Pathophysiology , Ningbo University School of Medicine , Ningbo , China.; b Department of Hematology , Yinzhou People's Hospital, Ningbo University School of Medicine , Ningbo , China.
Zhou JX; a Department of Pathology, Zhejiang Provincial Key Laboratory of Pathophysiology , Ningbo University School of Medicine , Ningbo , China.
Jiang L; a Department of Pathology, Zhejiang Provincial Key Laboratory of Pathophysiology , Ningbo University School of Medicine , Ningbo , China.; Pokaż więcej
Źródło:: Leukemia & lymphoma [Leuk Lymphoma] 2018 Aug; Vol. 59 (8), pp. 1959-1967. Date of Electronic Publication: 2017 Nov 22.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Phosphoinositide-3 Kinase Inhibitors*
Xenograft Model Antitumor Assays*
Apoptosis/*drug effects
Caffeic Acids/*pharmacology
Lactates/*pharmacology
Leukemia, Myeloid, Acute/*drug therapy
Proto-Oncogene Proteins c-akt/*antagonists & inhibitors
Animals ; Cell Line, Tumor ; Female ; Humans ; Leukemia, Myeloid, Acute/pathology ; Male ; Mice, Inbred BALB C ; Mice, Nude ; Middle Aged ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; THP-1 Cells ; Tumor Burden/drug effects

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Skocz do pozycji: 10.

Tytuł:: Propofol prevents human umbilical vein endothelial cell injury from Ang II-induced apoptosis by activating the ACE2-(1-7)-Mas axis and eNOS phosphorylation.
Autorzy:: Zhang L; Department of Anesthesiology, Affiliated Hospital of Guangdong Medical University, Zhanjiang, China.
Wang J; Department of Anesthesiology, Affiliated Hospital of Guangdong Medical University, Zhanjiang, China.
Liang J; Department of Anesthesiology, Affiliated Hospital of Guangdong Medical University, Zhanjiang, China.
Feng D; Guangdong Key Laboratory of Age-related Cardiac-cerebral Vascular Disease, Institute of Neurology, Affiliated Hospital of Guangdong Medical University, Guangdong Medical University, Zhanjiang, Guangdong, China.
Deng F; Department of Anesthesiology, Affiliated Hospital of Guangdong Medical University, Zhanjiang, China.
Yang Y; Department of Anesthesiology, Affiliated Hospital of Guangdong Medical University, Zhanjiang, China.
Lu Y; Department of Anesthesiology, Affiliated Hospital of Guangdong Medical University, Zhanjiang, China.
Hu Z; Department of Anesthesiology, Affiliated Hospital of Guangdong Medical University, Zhanjiang, China.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2018 Jul 11; Vol. 13 (7), pp. e0199373. Date of Electronic Publication: 2018 Jul 11 (Print Publication: 2018).
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Angiotensin II/*pharmacology
Apoptosis/*drug effects
Human Umbilical Vein Endothelial Cells/*drug effects
Human Umbilical Vein Endothelial Cells/*metabolism
Nitric Oxide Synthase Type III/*metabolism
Peptidyl-Dipeptidase A/*metabolism
Propofol/*pharmacology
Protective Agents/*pharmacology
Proto-Oncogene Proteins/*metabolism
Receptors, G-Protein-Coupled/*metabolism
Angiotensin-Converting Enzyme 2 ; Animals ; Humans ; Nitric Oxide/metabolism ; Oxidative Stress/drug effects ; Proto-Oncogene Mas

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Skocz do pozycji: 11.

Tytuł:: Inactivation of deubiquitinase CYLD enhances therapeutic antibody production in Chinese hamster ovary cells.
Autorzy:: Lu Y; Institute of Physical Science and Information Technology, Anhui University, Hefei, Anhui, 230601, People's Republic of China.
Zhou Q; Institute of Physical Science and Information Technology, Anhui University, Hefei, Anhui, 230601, People's Republic of China.
Han Q; Institute of Physical Science and Information Technology, Anhui University, Hefei, Anhui, 230601, People's Republic of China.
Wu P; Institute of Physical Science and Information Technology, Anhui University, Hefei, Anhui, 230601, People's Republic of China.
Zhang L; Institute of Physical Science and Information Technology, Anhui University, Hefei, Anhui, 230601, People's Republic of China.
Zhu L; Institute of Physical Science and Information Technology, Anhui University, Hefei, Anhui, 230601, People's Republic of China.
Weaver DT; Institute of Physical Science and Information Technology, Anhui University, Hefei, Anhui, 230601, People's Republic of China.
Xu C; Institute of Physical Science and Information Technology, Anhui University, Hefei, Anhui, 230601, People's Republic of China. .
Zhang B; Institute of Physical Science and Information Technology, Anhui University, Hefei, Anhui, 230601, People's Republic of China. .; Pokaż więcej
Źródło:: Applied microbiology and biotechnology [Appl Microbiol Biotechnol] 2018 Jul; Vol. 102 (14), pp. 6081-6093. Date of Electronic Publication: 2018 May 15.
Typ publikacji:: Journal Article
MeSH Terms:: Cell Engineering*
Gene Silencing*
Apoptosis/*genetics
Cell Proliferation/*genetics
Deubiquitinating Enzyme CYLD/*genetics
Immunoglobulin G/*biosynthesis
Animals ; Antibodies, Bispecific/biosynthesis ; CHO Cells ; CRISPR-Cas Systems ; Cricetinae ; Cricetulus ; Gene Deletion ; Genes, Tumor Suppressor ; Humans ; Rituximab/biosynthesis ; Trastuzumab/biosynthesis

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Skocz do pozycji: 12.

Tytuł:: Fibrinogen links podocyte injury with Toll-like receptor 4 and is associated with disease activity in FSGS patients.
Autorzy:: Wang H; Division of Nephrology, Jinling Hospital, Southern Medical University, Nanjing, China.; National Clinical Research Center of Kidney Diseases, Jinling Hospital, Nanjing University School of Medicine, Nanjing, China.
Zheng C; National Clinical Research Center of Kidney Diseases, Jinling Hospital, Nanjing University School of Medicine, Nanjing, China.
Xu X; National Clinical Research Center of Kidney Diseases, Jinling Hospital, Nanjing University School of Medicine, Nanjing, China.
Zhao Y; National Clinical Research Center of Kidney Diseases, Jinling Hospital, Nanjing University School of Medicine, Nanjing, China.
Lu Y; National Clinical Research Center of Kidney Diseases, Jinling Hospital, Nanjing University School of Medicine, Nanjing, China.
Liu Z; Division of Nephrology, Jinling Hospital, Southern Medical University, Nanjing, China.; National Clinical Research Center of Kidney Diseases, Jinling Hospital, Nanjing University School of Medicine, Nanjing, China.; Pokaż więcej
Źródło:: Nephrology (Carlton, Vic.) [Nephrology (Carlton)] 2018 May; Vol. 23 (5), pp. 418-429.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis*
Fibrinogen/*metabolism
Glomerulosclerosis, Focal Segmental/*metabolism
Podocytes/*metabolism
Toll-Like Receptor 4/*metabolism
Actin Cytoskeleton/metabolism ; Actin Cytoskeleton/pathology ; Adolescent ; Adult ; Albuminuria/chemically induced ; Albuminuria/metabolism ; Albuminuria/pathology ; Animals ; Cell Line ; Disease Models, Animal ; Doxorubicin ; Female ; Fibrinogen/urine ; Glomerulosclerosis, Focal Segmental/chemically induced ; Glomerulosclerosis, Focal Segmental/drug therapy ; Glomerulosclerosis, Focal Segmental/pathology ; Glucocorticoids/therapeutic use ; Humans ; Male ; Mice, Inbred BALB C ; Phosphorylation ; Podocytes/pathology ; Prednisone/therapeutic use ; RNA Interference ; Remission Induction ; Signal Transduction ; Toll-Like Receptor 4/genetics ; Transcription Factor RelA/metabolism ; Transfection ; Treatment Outcome ; Young Adult ; p38 Mitogen-Activated Protein Kinases/metabolism

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Skocz do pozycji: 13.

Tytuł:: Downregulation of TRIM28 inhibits growth and increases apoptosis of nude mice with non‑small cell lung cancer xenografts.
Autorzy:: Liu L; Department of Immunology, Basic Medical Institute, Chengde Medical College, Chengde, Hebei 067000, P.R. China.
Zhang L; Department of Community Care, Nursing Institute, Chengde Medical College, Chengde, Hebei 067000, P.R. China.
Wang J; Department of Immunology, Basic Medical Institute, Chengde Medical College, Chengde, Hebei 067000, P.R. China.
Zhao X; Department of Immunology, Basic Medical Institute, Chengde Medical College, Chengde, Hebei 067000, P.R. China.
Xu Q; Department of Central Laboratory, Chengde Medical College, Chengde, Hebei 067000, P.R. China.
Lu Y; Department of Pathology, Basic Medical Institute, Chengde Medical College, Chengde, Hebei 067000, P.R. China.
Zuo Y; Department of Pathology, Basic Medical Institute, Chengde Medical College, Chengde, Hebei 067000, P.R. China.
Chen L; Department of Central Laboratory, Chengde Medical College, Chengde, Hebei 067000, P.R. China.
Du J; Grade 2013, Clinical Institute, Chengde Medical College, Chengde, Hebei 067000, P.R. China.
Lian Y; Grade 2013, Clinical Institute, Chengde Medical College, Chengde, Hebei 067000, P.R. China.
Zhang Q; Grade 2013, Clinical Institute, Chengde Medical College, Chengde, Hebei 067000, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2018 Jan; Vol. 17 (1), pp. 835-842. Date of Electronic Publication: 2017 Nov 03.
Typ publikacji:: Journal Article
MeSH Terms:: Gene Expression Regulation, Neoplastic*
Apoptosis/*genetics
Carcinoma, Non-Small-Cell Lung/*genetics
Carcinoma, Non-Small-Cell Lung/*pathology
Lung Neoplasms/*genetics
Lung Neoplasms/*pathology
Tripartite Motif-Containing Protein 28/*genetics
Adult ; Aged ; Animals ; Apoptosis Regulatory Proteins/genetics ; Apoptosis Regulatory Proteins/metabolism ; Autoantibodies/blood ; Autoantibodies/immunology ; Carcinoma, Non-Small-Cell Lung/immunology ; Case-Control Studies ; Cell Cycle ; Cell Line, Tumor ; Cell Proliferation ; Female ; Gene Knockdown Techniques ; Humans ; Lung Neoplasms/immunology ; Male ; Mice ; Mice, Nude ; Middle Aged ; RNA Interference ; RNA, Small Interfering ; Tripartite Motif-Containing Protein 28/immunology ; Tripartite Motif-Containing Protein 28/metabolism

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Skocz do pozycji: 14.

Tytuł:: Triptolide inhibits proliferation, differentiation and induces apoptosis of osteoblastic MC3T3‑E1 cells.
Autorzy:: Ji W; Department of Rheumatology and Immunology, Affiliated Hospital of Nanjing University of Traditional Chinese Medicine, Nanjing, Jiangsu 210029, P.R. China.
Liu S; Laboratory of Clinical Pharmacology, Affiliated Hospital of Nanjing University of Traditional Chinese Medicine, Nanjing, Jiangsu 210029, P.R. China.
Zhao X; Department of Rheumatology and Immunology, Affiliated Hospital of Nanjing University of Traditional Chinese Medicine, Nanjing, Jiangsu 210029, P.R. China.
Guo Y; Department of Rheumatology and Immunology, Affiliated Hospital of Nanjing University of Traditional Chinese Medicine, Nanjing, Jiangsu 210029, P.R. China.
Xia S; Hematology‑Oncology Department, Zhenjiang Hospital of Integrated Chinese and Western Medicine, Zhenjiang, Jiangsu 212000, P.R. China.
Lu Y; College of Basic Medical Sciences, Nanjing University of Traditional Chinese Medicine, Nanjing, Jiangsu 210023, P.R. China.
Yin M; Hematology‑Oncology Department, Zhenjiang Hospital of Integrated Chinese and Western Medicine, Zhenjiang, Jiangsu 212000, P.R. China.
Xu X; College of Nursing, Nanjing University of Traditional Chinese Medicine, Nanjing, Jiangsu 210023, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2017 Nov; Vol. 16 (5), pp. 7391-7397. Date of Electronic Publication: 2017 Sep 21.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Cell Differentiation/*drug effects
Cell Proliferation/*drug effects
Diterpenes/*toxicity
Phenanthrenes/*toxicity
Alkaline Phosphatase/metabolism ; Animals ; Calcium/metabolism ; Cell Cycle Checkpoints/drug effects ; Cell Line ; Epoxy Compounds/toxicity ; Flow Cytometry ; Mice ; Osteoblasts/cytology ; Osteoblasts/drug effects ; Osteoblasts/metabolism ; Osteogenesis/drug effects ; Tripterygium/chemistry ; Tripterygium/metabolism

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Skocz do pozycji: 15.

Tytuł:: Bortezomib could down-regulate the expression of RANKL, inhibit cell proliferation and induce cell apoptosis in the human myeloma cell line RPMI 8226 by activating casepase-3.
Autorzy:: Lin L; Department of Hematology, Yinzhou Hospital Affiliated to Medical School of Ningbo University, Ningbo 315040, Zhejiang, China.
Chen D; Department of Hematology, Yinzhou Hospital Affiliated to Medical School of Ningbo University, Ningbo 315040, Zhejiang, China.
Xiang ZF; Department of Radiation Oncology, Ningbo Medical Center Lihuili Hospital, Ningbo 315040, Zhejiang, China.
Pei RZ; Department of Hematology, Yinzhou Hospital Affiliated to Medical School of Ningbo University, Ningbo 315040, Zhejiang, China.
Zhang PS; Department of Hematology, Yinzhou Hospital Affiliated to Medical School of Ningbo University, Ningbo 315040, Zhejiang, China.
Liu XH; Department of Hematology, Yinzhou Hospital Affiliated to Medical School of Ningbo University, Ningbo 315040, Zhejiang, China.
Du XH; Department of Hematology, Yinzhou Hospital Affiliated to Medical School of Ningbo University, Ningbo 315040, Zhejiang, China.
Lu Y; Department of Hematology, Yinzhou Hospital Affiliated to Medical School of Ningbo University, Ningbo 315040, Zhejiang, China.; Pokaż więcej
Źródło:: Cancer biomarkers : section A of Disease markers [Cancer Biomark] 2017 Aug 23; Vol. 20 (2), pp. 217-224.
Typ publikacji:: Journal Article
MeSH Terms:: Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Bortezomib/*pharmacology
Caspase 3/*metabolism
Gene Expression Regulation, Neoplastic/*drug effects
RANK Ligand/*genetics
Cell Cycle/drug effects ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Enzyme Activation/drug effects ; Humans ; Multiple Myeloma/genetics ; Multiple Myeloma/metabolism ; RANK Ligand/metabolism

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Skocz do pozycji: 16.

Tytuł:: Telmisartan suppresses cardiac hypertrophy by inhibiting cardiomyocyte apoptosis via the NFAT/ANP/BNP signaling pathway.
Autorzy:: Li X; Department of Pathology, Heilongjiang Provincial Hospital, Harbin, Heilongjiang 150036, P.R. China.
Lan Y; Intensive Care Unit, Heilongjiang Provincial Hospital, Harbin, Heilongjiang 150036, P.R. China.
Wang Y; Department of Pathology, Heilongjiang Provincial Hospital, Harbin, Heilongjiang 150036, P.R. China.
Nie M; Department of Pathology, Heilongjiang Provincial Hospital, Harbin, Heilongjiang 150036, P.R. China.
Lu Y; Department of Pathology, Heilongjiang Provincial Hospital, Harbin, Heilongjiang 150036, P.R. China.
Zhao E; Department of Oral Pathology, Stomatological Hospital, Harbin Medical University, Harbin, Heilongjiang 150036, P.R. China.; Pokaż więcej
Źródło:: Molecular medicine reports [Mol Med Rep] 2017 May; Vol. 15 (5), pp. 2574-2582. Date of Electronic Publication: 2017 Mar 14.
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*drug effects
Atrial Natriuretic Factor/*metabolism
Benzimidazoles/*pharmacology
Benzoates/*pharmacology
Cardiomegaly/*metabolism
Cardiomegaly/*prevention & control
Myocytes, Cardiac/*metabolism
NFATC Transcription Factors/*metabolism
Natriuretic Peptide, Brain/*metabolism
Signal Transduction/*drug effects
Active Transport, Cell Nucleus/drug effects ; Animals ; Cardiomegaly/pathology ; Cell Nucleus/metabolism ; Cell Nucleus/pathology ; Male ; Mice ; Myocytes, Cardiac/pathology ; Telmisartan

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Skocz do pozycji: 17.

Tytuł:: Litchi seed extracts diminish prostate cancer progression via induction of apoptosis and attenuation of EMT through Akt/GSK-3β signaling.
Autorzy:: Guo H; Center for Translational Medicine, Guangxi Medical University, 22 Shuangyong Road, Nanning 530021, China.; Key Laboratory of Longevity and Aging-related Disease, Chinese Ministry of Education, 22 Shuangyong Road, Nanning 530021, China.
Luo H; College of Pharmacy, Guangxi University of Chinese Medicine, 179 Mingxiu Dong Road, Nanning 530001, China.
Yuan H; Department of Pharmaceutical Sciences, College of Pharmacy, University of Michigan, 1600 Huron Parkway, Ann Arbor, MI 48109, USA.
Xia Y; Center for Translational Medicine, Guangxi Medical University, 22 Shuangyong Road, Nanning 530021, China.; Key Laboratory of Longevity and Aging-related Disease, Chinese Ministry of Education, 22 Shuangyong Road, Nanning 530021, China.; Xinxiang Central Hospital of Henan, 56 Jinsui Road, Xinxiang 453000, China.
Shu P; Department of Pharmaceutical Sciences, College of Pharmacy, University of Michigan, 1600 Huron Parkway, Ann Arbor, MI 48109, USA.
Huang X; Center for Translational Medicine, Guangxi Medical University, 22 Shuangyong Road, Nanning 530021, China.; Key Laboratory of Longevity and Aging-related Disease, Chinese Ministry of Education, 22 Shuangyong Road, Nanning 530021, China.
Lu Y; Center for Translational Medicine, Guangxi Medical University, 22 Shuangyong Road, Nanning 530021, China.; Key Laboratory of Longevity and Aging-related Disease, Chinese Ministry of Education, 22 Shuangyong Road, Nanning 530021, China.
Liu X; Center for Translational Medicine, Guangxi Medical University, 22 Shuangyong Road, Nanning 530021, China.; Key Laboratory of Longevity and Aging-related Disease, Chinese Ministry of Education, 22 Shuangyong Road, Nanning 530021, China.
Keller ET; Department of Urology and Pathology, School of Medicine, University of Michigan, 2800 Plymouth Road, Ann Arbor, MI 48109, USA.
Sun D; Department of Pharmaceutical Sciences, College of Pharmacy, University of Michigan, 1600 Huron Parkway, Ann Arbor, MI 48109, USA.
Deng J; College of Pharmacy, Guangxi University of Chinese Medicine, 179 Mingxiu Dong Road, Nanning 530001, China.
Zhang J; Center for Translational Medicine, Guangxi Medical University, 22 Shuangyong Road, Nanning 530021, China.; Key Laboratory of Longevity and Aging-related Disease, Chinese Ministry of Education, 22 Shuangyong Road, Nanning 530021, China.; Department of Urology and Pathology, School of Medicine, University of Michigan, 2800 Plymouth Road, Ann Arbor, MI 48109, USA.; Southern University of Science and Technology, School of Medicine, 1088 Xueyuan Blvd., Nanshan District, Shenzhen, 518055, China.; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2017 Jan 30; Vol. 7, pp. 41656. Date of Electronic Publication: 2017 Jan 30.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Signal Transduction*
Antineoplastic Agents/*therapeutic use
Apoptosis/*drug effects
Epithelial-Mesenchymal Transition/*drug effects
Litchi/*chemistry
Plant Extracts/*therapeutic use
Prostatic Neoplasms/*drug therapy
Animals ; Antineoplastic Agents/pharmacology ; Cadherins/metabolism ; Cell Line, Tumor ; Cyclin-Dependent Kinase Inhibitor p21/metabolism ; Cyclin-Dependent Kinase Inhibitor p27/metabolism ; Cyclins/metabolism ; Glycogen Synthase Kinase 3 beta/metabolism ; Humans ; Male ; Mice ; Mice, Nude ; Plant Extracts/pharmacology ; Proto-Oncogene Proteins c-akt/metabolism ; Snail Family Transcription Factors/metabolism ; Vimentin/metabolism

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Skocz do pozycji: 18.

Tytuł:: The interplay between p16 serine phosphorylation and arginine methylation determines its function in modulating cellular apoptosis and senescence.
Autorzy:: Lu Y; The Institute of Genetics and Cytology, Northeast Normal University, Changchun 130024, P. R. China.
Ma W; The Institute of Genetics and Cytology, Northeast Normal University, Changchun 130024, P. R. China.
Li Z; The Institute of Genetics and Cytology, Northeast Normal University, Changchun 130024, P. R. China.
Lu J; The Institute of Genetics and Cytology, Northeast Normal University, Changchun 130024, P. R. China.
Wang X; The Institute of Genetics and Cytology, Northeast Normal University, Changchun 130024, P. R. China.; School of Life Sciences, Northeast Normal University, Changchun 130024, P. R. China.; Pokaż więcej
Źródło:: Scientific reports [Sci Rep] 2017 Jan 25; Vol. 7, pp. 41390. Date of Electronic Publication: 2017 Jan 25.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis*/genetics
Cellular Senescence*
Arginine/*metabolism
Cyclin-Dependent Kinase Inhibitor p16/*metabolism
Phosphoserine/*metabolism
Serine/*metabolism
Arginine/genetics ; Cell Line ; Cell Proliferation/genetics ; Cyclin-Dependent Kinase 4/metabolism ; Cyclin-Dependent Kinase Inhibitor p16/genetics ; Humans ; Hydrogen Peroxide/pharmacology ; Methylation ; Models, Biological ; Mutant Proteins/metabolism ; Mutation/genetics ; Phosphorylation ; Serine/genetics

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Skocz do pozycji: 19.

Tytuł:: Role of BmDredd during Apoptosis of Silk Gland in Silkworm, Bombyx mori.
Autorzy:: Chen RT; Institute of Sericulture and Apiculture, College of Animal Sciences, Zhejiang University, Hangzhou, People's Republic of China.
Jiao P; Institute of Sericulture and Apiculture, College of Animal Sciences, Zhejiang University, Hangzhou, People's Republic of China.
Liu Z; Institute of Sericulture and Apiculture, College of Animal Sciences, Zhejiang University, Hangzhou, People's Republic of China.
Lu Y; Institute of Sericulture and Apiculture, College of Animal Sciences, Zhejiang University, Hangzhou, People's Republic of China.
Xin HH; Institute of Sericulture and Apiculture, College of Animal Sciences, Zhejiang University, Hangzhou, People's Republic of China.
Zhang DP; Institute of Sericulture and Apiculture, College of Animal Sciences, Zhejiang University, Hangzhou, People's Republic of China.
Miao YG; Institute of Sericulture and Apiculture, College of Animal Sciences, Zhejiang University, Hangzhou, People's Republic of China.; Pokaż więcej
Źródło:: PloS one [PLoS One] 2017 Jan 09; Vol. 12 (1), pp. e0169404. Date of Electronic Publication: 2017 Jan 09 (Print Publication: 2017).
Typ publikacji:: Journal Article
MeSH Terms:: Apoptosis/*genetics
Bombyx/*physiology
Caspases/*genetics
Insect Proteins/*genetics
Silk/*metabolism
Animals ; Caspase Inhibitors/pharmacology ; Caspases/metabolism ; Gene Expression/drug effects ; Insect Proteins/metabolism ; Metamorphosis, Biological

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Skocz do pozycji: 20.

Tytuł:: Celecoxib suppresses autophagy and enhances cytotoxicity of imatinib in imatinib-resistant chronic myeloid leukemia cells.
Autorzy:: Lu Y; Department of Blood Transfusion, the Third Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.; Department of Hematology, the Third Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Liu LL; Department of Hematology, the Third Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Liu SS; State Key Laboratory of Oncology in South China, Department of VIP Region, Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, China.
Fang ZG; Department of Hematology, the Third Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Zou Y; Department of Blood Transfusion, the Third Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.
Deng XB; Cancer Hospital of Guangzhou Medical University, Guangzhou, China.
Long ZJ; Department of Hematology, the Third Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.; Institute of Hematology, Sun Yat-sen University, Guangzhou, China.
Liu Q; Department of Hematology, the Third Affiliated Hospital, Sun Yat-sen University, Guangzhou, China.; Institute of Hematology, Sun Yat-sen University, Guangzhou, China.
Lin DJ; Department of Blood Transfusion, the Third Affiliated Hospital, Sun Yat-sen University, Guangzhou, China. .; Department of Hematology, the Third Affiliated Hospital, Sun Yat-sen University, Guangzhou, China. .; Institute of Hematology, Sun Yat-sen University, Guangzhou, China. .; Pokaż więcej
Źródło:: Journal of translational medicine [J Transl Med] 2016 Sep 20; Vol. 14, pp. 270. Date of Electronic Publication: 2016 Sep 20.
Typ publikacji:: Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:: Apoptosis/*drug effects
Autophagy/*drug effects
Celecoxib/*pharmacology
Celecoxib/*therapeutic use
Drug Resistance, Neoplasm/*drug effects
Imatinib Mesylate/*therapeutic use
Leukemia, Myelogenous, Chronic, BCR-ABL Positive/*drug therapy
Leukemia, Myelogenous, Chronic, BCR-ABL Positive/*pathology
Adult ; Cell Line, Tumor ; Female ; G1 Phase Cell Cycle Checkpoints/drug effects ; Humans ; Imatinib Mesylate/pharmacology ; Lysosomes/drug effects ; Lysosomes/metabolism ; Male ; Middle Aged ; Necrosis ; Neoplasm Staging

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