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Wyszukujesz frazę ""Yamamoto, Mizuki"" wg kryterium: Autor


Tytuł :
The Relaxation of Reproducible Resistance Modulations in a Au/(Pb,La)(Zr, Ti)O3/(La,Sr)2CuO4 Thin Film in High Temperatures.
Autorzy :
Yamada, Kazumasa
Kaku, Shigeru
Yamamoto, Mizuki
Matsumoto, Daisuke
Watanabe, Yukio
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Temat :
ELECTRONIC modulation
FERROELECTRIC thin films
THIN films
FERROELECTRIC crystals
ANNEALING of crystals
ELECTRONS
FERROELECTRICITY
Źródło :
Ferroelectrics; 2007, Vol. 357 Issue 1, p70-73, 4p, 1 Diagram, 2 Graphs
Czasopismo naukowe
Tytuł :
Inactivation of NF-kappaB components by covalent binding of (-)-dehydroxymethylepoxyquinomicin to specific cysteine residues.
Autorzy :
Yamamoto M; Center for Chemical Biology, School of Fundamental Science and Technology, Keio University, Yokohama 223-0061, Japan.
Horie R
Takeiri M
Kozawa I
Umezawa K
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Źródło :
Journal of medicinal chemistry [J Med Chem] 2008 Sep 25; Vol. 51 (18), pp. 5780-8. Date of Electronic Publication: 2008 Aug 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Benzamides/*metabolism
Cyclohexanones/*metabolism
Cysteine/*metabolism
NF-kappa B/*antagonists & inhibitors
Animals ; Base Sequence ; Binding Sites ; Cell Line, Tumor ; DNA Primers ; Electrophoretic Mobility Shift Assay ; Humans ; Magnetic Resonance Spectroscopy ; Mass Spectrometry ; Mutagenesis, Site-Directed ; Surface Plasmon Resonance
Czasopismo naukowe
Tytuł :
Epigenetic alteration of the NF-κB-inducing kinase (NIK) gene is involved in enhanced NIK expression in basal-like breast cancer.
Autorzy :
Yamamoto M; Department of Cancer Biology, Division of Cellular and Molecular Biology Laboratory of Stem Cell Therapy, Institute of Medical Science, University of Tokyo, Shirokane-dai, Minato-ku, Tokyo, Japan.
Ito T
Shimizu T
Ishida T
Semba K
Watanabe S
Yamaguchi N
Inoue J
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Źródło :
Cancer science [Cancer Sci] 2010 Nov; Vol. 101 (11), pp. 2391-7. Date of Electronic Publication: 2010 Aug 24.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Epigenesis, Genetic*
Breast Neoplasms/*genetics
Gene Expression Regulation, Neoplastic/*genetics
Protein-Serine-Threonine Kinases/*genetics
Acetylation/drug effects ; Azacitidine/pharmacology ; Blotting, Western ; Breast Neoplasms/metabolism ; Breast Neoplasms/pathology ; Cell Line ; Cell Line, Tumor ; CpG Islands/genetics ; DNA Methylation/drug effects ; Enzyme Inhibitors/pharmacology ; Female ; Gene Expression Regulation, Neoplastic/drug effects ; Histones/metabolism ; Humans ; NF-kappa B/metabolism ; Protein-Serine-Threonine Kinases/metabolism ; Receptor, ErbB-2/genetics ; Receptors, Estrogen/genetics ; Receptors, Progesterone/genetics ; Reverse Transcriptase Polymerase Chain Reaction ; Valproic Acid/pharmacology
Czasopismo naukowe
Tytuł :
NEIL1 mRNA splicing variants are expressed in normal mouse organs.
Autorzy :
Yamamoto R; Department of Advanced Pathobiology, Graduate School of Life & Environmental Sciences, Osaka Prefecture University, Izumisano, Osaka 598-8531, Japan. yamamoto@vet.osakafu-u.ac.jp
Yamamoto M
Kusaka H
Masatsugu H
Matsuyama S
Tajima T
Ide H
Kubo K
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Źródło :
Journal of radiation research [J Radiat Res] 2012; Vol. 53 (2), pp. 234-41.
Typ publikacji :
Journal Article
MeSH Terms :
Alternative Splicing/*genetics
DNA Glycosylases/*genetics
Genetic Variation/*genetics
Organ Specificity/*genetics
RNA Splicing/*genetics
RNA, Messenger/*genetics
Viscera/*physiology
Animals ; Mice ; Mice, Inbred ICR ; Reference Values ; Tissue Distribution
Czasopismo naukowe
Tytuł :
NF-κB non-cell-autonomously regulates cancer stem cell populations in the basal-like breast cancer subtype.
Autorzy :
Yamamoto M; Division of Cellular and Molecular Biology, Department of Cancer Biology, Institute of Medical Science, University of Tokyo, Shirokane-dai, Minato-ku, Tokyo 108-8639, Japan.
Taguchi Y
Ito-Kureha T
Semba K
Yamaguchi N
Inoue J
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Źródło :
Nature communications [Nat Commun] 2013; Vol. 4, pp. 2299.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Calcium-Binding Proteins/*biosynthesis
Carcinoma, Basal Cell/*metabolism
Intercellular Signaling Peptides and Proteins/*biosynthesis
Membrane Proteins/*biosynthesis
NF-kappa B/*metabolism
Neoplastic Stem Cells/*metabolism
Triple Negative Breast Neoplasms/*metabolism
Calcium-Binding Proteins/metabolism ; Cell Line, Tumor ; Female ; Gene Expression Regulation, Neoplastic ; Humans ; Intercellular Signaling Peptides and Proteins/metabolism ; Jagged-1 Protein ; Membrane Proteins/metabolism ; Neoplasm Recurrence, Local/metabolism ; Protein-Serine-Threonine Kinases/biosynthesis ; Protein-Serine-Threonine Kinases/genetics ; Protein-Serine-Threonine Kinases/metabolism ; RNA Interference ; RNA, Small Interfering ; Receptors, Notch/metabolism ; Serrate-Jagged Proteins ; Signal Transduction
Czasopismo naukowe
Tytuł :
Tropomodulin 1 expression driven by NF-κB enhances breast cancer growth.
Autorzy :
Ito-Kureha T; Division of Cellular and Molecular Biology, Institute of Medical Science, University of Tokyo, Tokyo, Japan. Cell Signal Unit, Okinawa Institute of Science and Technology, Okinawa, Japan.
Koshikawa N; Division of Cancer Cell Research, Institute of Medical Science, University of Tokyo, Tokyo, Japan.
Yamamoto M; Department of Life Science and Medical Bio-science, Waseda University, Tokyo, Japan.
Semba K; Department of Life Science and Medical Bio-science, Waseda University, Tokyo, Japan.
Yamaguchi N; Department of Molecular Cell Biology, Graduate School of Pharmaceutical Sciences, Chiba University, Chiba, Japan.
Yamamoto T; Cell Signal Unit, Okinawa Institute of Science and Technology, Okinawa, Japan.
Seiki M; Graduate School of Medicine, Kochi University, Kochi, Japan.
Inoue J; Division of Cellular and Molecular Biology, Institute of Medical Science, University of Tokyo, Tokyo, Japan. .
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Źródło :
Cancer research [Cancer Res] 2015 Jan 01; Vol. 75 (1), pp. 62-72. Date of Electronic Publication: 2014 Nov 14.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
NF-kappa B/*metabolism
Triple Negative Breast Neoplasms/*metabolism
Tropomodulin/*biosynthesis
Animals ; Cell Line, Tumor ; Cell Proliferation/physiology ; Female ; Heterografts ; Humans ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; NF-kappa B/genetics ; Signal Transduction ; Tissue Array Analysis ; Triple Negative Breast Neoplasms/genetics ; Triple Negative Breast Neoplasms/immunology ; Triple Negative Breast Neoplasms/pathology ; Tropomodulin/genetics ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
Enhanced expression of retinoic acid receptor alpha (RARA) induces epithelial-to-mesenchymal transition and disruption of mammary acinar structures.
Autorzy :
Doi A; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, 2-2 Wakamatsu-cho, Shinjuku-ku, Tokyo 162-8480, Japan. Electronic address: .
Ishikawa K; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, 2-2 Wakamatsu-cho, Shinjuku-ku, Tokyo 162-8480, Japan; Japan Biological Informatics Consortium (JBiC), 2-45 Aomi, Koto-ku, Tokyo 135-8073, Japan. Electronic address: .
Shibata N; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, 2-2 Wakamatsu-cho, Shinjuku-ku, Tokyo 162-8480, Japan.
Ito E; Division of Gene Expression Analysis, Translational Research Center (Tokyo Branch), Fukushima Medical University, Shibuya-ku, Tokyo 151-0051, Japan.
Fujimoto J; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, 2-2 Wakamatsu-cho, Shinjuku-ku, Tokyo 162-8480, Japan; Japan Biological Informatics Consortium (JBiC), 2-45 Aomi, Koto-ku, Tokyo 135-8073, Japan.
Yamamoto M; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, 2-2 Wakamatsu-cho, Shinjuku-ku, Tokyo 162-8480, Japan.
Shiga H; Japan Biological Informatics Consortium (JBiC), 2-45 Aomi, Koto-ku, Tokyo 135-8073, Japan.
Mochizuki H; Japan Biological Informatics Consortium (JBiC), 2-45 Aomi, Koto-ku, Tokyo 135-8073, Japan.
Kawamura Y; Japan Biological Informatics Consortium (JBiC), 2-45 Aomi, Koto-ku, Tokyo 135-8073, Japan.
Goshima N; Quantitative Proteomics Team, Molecular Profiling Research Center for Drug Discovery (molprof), National Institute of Advanced Industrial Science and Technology (AIST), 2-4-7 Aomi, Koto-ku, Tokyo 135-0064, Japan.
Semba K; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, 2-2 Wakamatsu-cho, Shinjuku-ku, Tokyo 162-8480, Japan; Division of Gene Function Analysis, Translational Research Center, Fukushima Medical University, 1 Hikarigaoka, Fukushima-city, Fukushima 960-1295, Japan. Electronic address: .
Watanabe S; Division of Gene Expression Analysis, Translational Research Center (Tokyo Branch), Fukushima Medical University, Shibuya-ku, Tokyo 151-0051, Japan.
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Źródło :
Molecular oncology [Mol Oncol] 2015 Feb; Vol. 9 (2), pp. 355-64. Date of Electronic Publication: 2014 Sep 22.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Epithelial-Mesenchymal Transition*
Breast Neoplasms/*metabolism
Cell Transformation, Neoplastic/*metabolism
Mammary Glands, Human/*metabolism
Neoplasm Proteins/*biosynthesis
Receptors, Retinoic Acid/*biosynthesis
Breast Neoplasms/genetics ; Breast Neoplasms/pathology ; Cell Line, Tumor ; Cell Transformation, Neoplastic/genetics ; Cell Transformation, Neoplastic/pathology ; Female ; Gene Expression Regulation, Neoplastic ; Humans ; Mammary Glands, Human/pathology ; Neoplasm Proteins/genetics ; Receptors, Retinoic Acid/genetics ; Retinoic Acid Receptor alpha
Czasopismo naukowe

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