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Tytuł :
Enhancing the chemotherapy effect of Apatinib on gastric cancer by co-treating with salidroside to reprogram the tumor hypoxia micro-environment and induce cell apoptosis.
Autorzy :
Zhang Z; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Yang W; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Ma F; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Ma Q; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Zhang B; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Zhang Y; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Liu Y; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Liu H; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Hua Y; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
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Źródło :
Drug delivery [Drug Deliv] 2020 Dec; Vol. 27 (1), pp. 691-702.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents/*administration & dosage
Apoptosis/*drug effects
Glucosides/*administration & dosage
Phenols/*administration & dosage
Pyridines/*administration & dosage
Stomach Neoplasms/*pathology
Tumor Hypoxia/*drug effects
Tumor Microenvironment/*drug effects
Animals ; Antineoplastic Agents/pharmacokinetics ; Antineoplastic Agents/pharmacology ; Cell Line, Tumor ; Cell Movement/drug effects ; Cell Proliferation/drug effects ; Drug Carriers/chemistry ; Drug Liberation ; Glucosides/pharmacology ; Humans ; Male ; Mice, Inbred BALB C ; Molecular Targeted Therapy ; Nanoparticles/chemistry ; Phenols/pharmacology ; Pyridines/pharmacology ; Stomach Neoplasms/drug therapy ; Treatment Outcome ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
Pyridoxine induces monocyte-macrophages death as specific treatment of acute myeloid leukemia.
Autorzy :
Yang W; The Second Affiliated Hospital, The State Key Laboratory of Respiratory Disease, Guangdong Provincial Key Laboratory of Allergy & Clinical Immunology, Guangzhou Medical University, Guangzhou, 510260, PR China.
Liu S; The Second Affiliated Hospital, The State Key Laboratory of Respiratory Disease, Guangdong Provincial Key Laboratory of Allergy & Clinical Immunology, Guangzhou Medical University, Guangzhou, 510260, PR China.
Li Y; The Second Affiliated Hospital, The State Key Laboratory of Respiratory Disease, Guangdong Provincial Key Laboratory of Allergy & Clinical Immunology, Guangzhou Medical University, Guangzhou, 510260, PR China.
Wang Y; School of Basic Medical Sciences, Guangzhou Medical University, Guangzhou, 510260, PR China.
Deng Y; The Second Affiliated Hospital, The State Key Laboratory of Respiratory Disease, Guangdong Provincial Key Laboratory of Allergy & Clinical Immunology, Guangzhou Medical University, Guangzhou, 510260, PR China.
Sun W; The Second Affiliated Hospital, The State Key Laboratory of Respiratory Disease, Guangdong Provincial Key Laboratory of Allergy & Clinical Immunology, Guangzhou Medical University, Guangzhou, 510260, PR China.
Huang H; The Second Affiliated Hospital, Guangzhou Medical University, Guangzhou, 510260, PR China.
Xie J; The Second Affiliated Hospital, The State Key Laboratory of Respiratory Disease, Guangdong Provincial Key Laboratory of Allergy & Clinical Immunology, Guangzhou Medical University, Guangzhou, 510260, PR China.
He A; The Second Affiliated Hospital, The State Key Laboratory of Respiratory Disease, Guangdong Provincial Key Laboratory of Allergy & Clinical Immunology, Guangzhou Medical University, Guangzhou, 510260, PR China.
Chen H; The Second Affiliated Hospital, The State Key Laboratory of Respiratory Disease, Guangdong Provincial Key Laboratory of Allergy & Clinical Immunology, Guangzhou Medical University, Guangzhou, 510260, PR China.
Tao A; The Second Affiliated Hospital, The State Key Laboratory of Respiratory Disease, Guangdong Provincial Key Laboratory of Allergy & Clinical Immunology, Guangzhou Medical University, Guangzhou, 510260, PR China. Electronic address: .
Yan J; The Second Affiliated Hospital, The State Key Laboratory of Respiratory Disease, Guangdong Provincial Key Laboratory of Allergy & Clinical Immunology, Guangzhou Medical University, Guangzhou, 510260, PR China. Electronic address: .
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Źródło :
Cancer letters [Cancer Lett] 2020 Nov 01; Vol. 492, pp. 96-105. Date of Electronic Publication: 2020 Aug 26.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Leukemia, Myeloid, Acute/*drug therapy
Macrophages/*drug effects
Monocytes/*drug effects
Pyridoxine/*pharmacology
Animals ; Cells, Cultured ; Humans ; Leukemia, Myeloid, Acute/pathology ; Macrophages/pathology ; Mice ; Mice, Inbred C57BL ; Monocytes/pathology ; Oligopeptides/pharmacology ; Pyridoxine/therapeutic use ; Pyroptosis/drug effects
Czasopismo naukowe
Tytuł :
Cantharidic acid induces apoptosis in human nasopharyngeal carcinoma cells through p38-mediated upregulation of caspase activation.
Autorzy :
Chen YC; Department of Medical Research, Chung Shan Medical University Hospital, Taichung, Taiwan.
Chen PN; Institute of Biochemistry, Microbiology and Immunology, Chung Shan Medical University, Taichung, Taiwan.
Lin CW; Institute of Oral Sciences, Chung Shan Medical University, Taichung, Taiwan.; Department of Dentistry, Chung Shan Medical University Hospital, Taichung, Taiwan.
Yang WE; Department of Medical Research, Chung Shan Medical University Hospital, Taichung, Taiwan.; Institute of Medicine, Chung Shan Medical University, Taichung, Taiwan.
Ho YT; Institute of Medicine, Chung Shan Medical University, Taichung, Taiwan.
Yang SF; Department of Medical Research, Chung Shan Medical University Hospital, Taichung, Taiwan.; Institute of Medicine, Chung Shan Medical University, Taichung, Taiwan.
Chuang CY; School of Medicine, Chung Shan Medical University, Taichung, Taiwan.; Department of Otolaryngology, Chung Shan Medical University Hospital, Taichung, Taiwan.
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Źródło :
Environmental toxicology [Environ Toxicol] 2020 May; Vol. 35 (5), pp. 619-627. Date of Electronic Publication: 2020 Jan 09.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Cantharidin/*analogs & derivatives
Caspases/*metabolism
Transcriptional Activation/*drug effects
p38 Mitogen-Activated Protein Kinases/*metabolism
Cantharidin/pharmacology ; Cell Line, Tumor ; Cell Survival/drug effects ; Humans ; MAP Kinase Signaling System/drug effects ; Nasopharyngeal Carcinoma/metabolism ; Nasopharyngeal Carcinoma/pathology ; Nasopharyngeal Neoplasms/metabolism ; Nasopharyngeal Neoplasms/pathology ; Signal Transduction ; Up-Regulation
Czasopismo naukowe
Tytuł :
Fisetin improves lead-induced neuroinflammation, apoptosis and synaptic dysfunction in mice associated with the AMPK/SIRT1 and autophagy pathway.
Autorzy :
Yang W; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tongshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China; Shanxi Key Laboratory of Ecological Animal Science and Environmental Veterinary Medicine, College of Animal Science and Veterinary Medicine, Shanxi Agricultural University, Taigu, Shanxi, 030801, China.
Tian ZK; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tongshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China.
Yang HX; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tongshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China.
Feng ZJ; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tongshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China.
Sun JM; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tongshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China.
Jiang H; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tongshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China.
Cheng C; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tongshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China.
Ming QL; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tongshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China.
Liu CM; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tongshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China. Electronic address: .
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Źródło :
Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association [Food Chem Toxicol] 2019 Dec; Vol. 134, pp. 110824. Date of Electronic Publication: 2019 Sep 17.
Typ publikacji :
Journal Article
MeSH Terms :
Adenylate Kinase/*metabolism
Apoptosis/*drug effects
Autophagy/*drug effects
Flavonoids/*therapeutic use
Inflammation/*drug therapy
Lead/*toxicity
Sirtuin 1/*metabolism
Synapses/*drug effects
Animals ; Brain/drug effects ; Brain/physiopathology ; Flavonoids/pharmacology ; Male ; Mice ; Mice, Inbred ICR ; Phosphorylation ; Synapses/physiology
Czasopismo naukowe
Tytuł :
Maternal control of suspensor programmed cell death via gibberellin signaling.
Autorzy :
Shi C; State Key Laboratory of Hybrid Rice, College of Life Sciences, Wuhan University, 430072, Wuhan, China.
Luo P; State Key Laboratory of Hybrid Rice, College of Life Sciences, Wuhan University, 430072, Wuhan, China.
Du YT; State Key Laboratory of Hybrid Rice, College of Life Sciences, Wuhan University, 430072, Wuhan, China.
Chen H; State Key Laboratory of Hybrid Rice, College of Life Sciences, Wuhan University, 430072, Wuhan, China.
Huang X; State Key Laboratory of Hybrid Rice, College of Life Sciences, Wuhan University, 430072, Wuhan, China.
Cheng TH; State Key Laboratory of Hybrid Rice, College of Life Sciences, Wuhan University, 430072, Wuhan, China.
Luo A; State Key Laboratory of Hybrid Rice, College of Life Sciences, Wuhan University, 430072, Wuhan, China.
Li HJ; State Key Laboratory of Molecular and Developmental Biology, Institute of Genetics and Developmental Biology, Chinese Academy of Sciences, 100101, Beijing, China.
Yang WC; State Key Laboratory of Molecular and Developmental Biology, Institute of Genetics and Developmental Biology, Chinese Academy of Sciences, 100101, Beijing, China.
Zhao P; State Key Laboratory of Hybrid Rice, College of Life Sciences, Wuhan University, 430072, Wuhan, China. .
Sun MX; State Key Laboratory of Hybrid Rice, College of Life Sciences, Wuhan University, 430072, Wuhan, China. .
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Źródło :
Nature communications [Nat Commun] 2019 Aug 02; Vol. 10 (1), pp. 3484. Date of Electronic Publication: 2019 Aug 02.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Gene Expression Regulation, Developmental*
Gene Expression Regulation, Plant*
Apoptosis/*physiology
Gibberellins/*metabolism
Seeds/*growth & development
Arabidopsis/growth & development ; CRISPR-Cas Systems/genetics ; Cell Communication/physiology ; Plant Proteins/genetics ; Plant Proteins/metabolism ; Plants, Genetically Modified ; Promoter Regions, Genetic ; Signal Transduction/physiology ; Tobacco/genetics ; Tobacco/growth & development ; Transcription Factors/genetics ; Transcription Factors/metabolism
Czasopismo naukowe
Tytuł :
Downregulation of ENDOCAN in myeloid leukemia cells inhibits proliferation and promotes apoptosis by suppressing nuclear factor‑κB activity.
Autorzy :
Sun L; Department of Hematology, Shengjing Hospital of China Medical University, Shenyang, Liaoning 110004, P.R. China.
Sun C; Anorectal Department, Shenyang Anorectal Hospital, Shenyang, Liaoning 110054, P.R. China.
Sun J; Intensive Care Unit, The Fourth Affiliated Hospital of China Medical University, Shenyang, Liaoning 110032, P.R. China.
Yang W; Department of Hematology, Shengjing Hospital of China Medical University, Shenyang, Liaoning 110004, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2019 Apr; Vol. 19 (4), pp. 3247-3254. Date of Electronic Publication: 2019 Feb 19.
Typ publikacji :
Journal Article
MeSH Terms :
Gene Expression Regulation, Leukemic*
Apoptosis/*genetics
Leukemia, Myeloid/*genetics
Leukemia, Myeloid/*metabolism
NF-kappa B/*metabolism
Neoplasm Proteins/*genetics
Proteoglycans/*genetics
Cell Cycle Checkpoints/genetics ; Cell Line, Tumor ; Cell Proliferation ; Gene Silencing ; HL-60 Cells ; Humans ; K562 Cells ; Leukemia, Myeloid/pathology ; U937 Cells
Czasopismo naukowe
Tytuł :
[Deoxynivalenol induced mRNA expressions of inflammation and apoptosis in BeWo cells].
Autorzy :
Yu M; Wuxi School of Medicine, Jiangnan University, Wuxi 214122, China.
Hou W; Tongji Hospital, Wuhan 430030, China.
Chen L; Tongji Medical College of Huazhong University of Science & Technology, Wuhan 430030, China.
Chen L; Wuxi School of Medicine, Jiangnan University, Wuxi 214122, China.
Yang W; Tongji Medical College of Huazhong University of Science & Technology, Wuhan 430030, China.
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Źródło :
Wei sheng yan jiu = Journal of hygiene research [Wei Sheng Yan Jiu] 2019 Jan; Vol. 48 (1), pp. 94-98.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*/drug effects
Cytokines*/metabolism
RNA, Messenger*/drug effects
RNA, Messenger*/metabolism
Trichothecenes*/pharmacology
Humans ; Inflammation
Czasopismo naukowe
Tytuł :
Ribosomal protein S6 kinase 1 promotes the survival of photoreceptors in retinitis pigmentosa.
Autorzy :
Lin B; School of Optometry, The Hong Kong Polytechnic University, Hung Hom, Kowloon, Hong Kong. .
Xiong G; Department of Ophthalmology, University of Hong Kong, Pokfulam, Hong Kong.
Yang W; School of Optometry, The Hong Kong Polytechnic University, Hung Hom, Kowloon, Hong Kong.
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Źródło :
Cell death & disease [Cell Death Dis] 2018 Nov 15; Vol. 9 (12), pp. 1141. Date of Electronic Publication: 2018 Nov 15.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*genetics
Retinitis Pigmentosa/*genetics
Ribosomal Protein S6 Kinases, 90-kDa/*genetics
TOR Serine-Threonine Kinases/*genetics
Adaptor Proteins, Signal Transducing ; Animals ; Cell Cycle Proteins/genetics ; Cell Survival/genetics ; Disease Models, Animal ; Electroretinography ; Eukaryotic Initiation Factors/genetics ; Gene Expression Regulation/genetics ; Humans ; Mice ; PTEN Phosphohydrolase/genetics ; Retina/metabolism ; Retina/pathology ; Retinal Cone Photoreceptor Cells/metabolism ; Retinal Cone Photoreceptor Cells/pathology ; Retinal Rod Photoreceptor Cells/metabolism ; Retinal Rod Photoreceptor Cells/pathology ; Retinitis Pigmentosa/metabolism ; Retinitis Pigmentosa/pathology ; Signal Transduction/genetics
Czasopismo naukowe
Tytuł :
[Effects of FBI-1 silencing on proliferation and apoptosis of triple-negative breast cancer cell line MDA-MB-231].
Autorzy :
Chen MJ; Department of Breast Surgery, The Affiliated Tumor Hospital of Guangxi Medical University, Nanning 530021, China.
Wang L; Department of Breast Surgery, Liuzhou People's Hospital, Liuzhou 545006, China.
Yang WP; Department of Breast Surgery, The Affiliated Tumor Hospital of Guangxi Medical University, Nanning 530021, China.
Qin QH; Department of Breast Surgery, The Affiliated Tumor Hospital of Guangxi Medical University, Nanning 530021, China.
Tan QX; Department of Breast Surgery, The Affiliated Tumor Hospital of Guangxi Medical University, Nanning 530021, China.
Lian B; Department of Breast Surgery, The Affiliated Tumor Hospital of Guangxi Medical University, Nanning 530021, China.
Wei CY; Department of Breast Surgery, The Affiliated Tumor Hospital of Guangxi Medical University, Nanning 530021, China. .
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Źródło :
Sheng li xue bao : [Acta physiologica Sinica] [Sheng Li Xue Bao] 2018 Oct 25; Vol. 70 (5), pp. 497-503.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Cell Proliferation*
RNA Interference*
DNA-Binding Proteins/*genetics
Transcription Factors/*genetics
Triple Negative Breast Neoplasms/*genetics
Animals ; Caspase 3/metabolism ; Cell Line, Tumor ; Humans ; Mice ; Mice, Nude ; Proto-Oncogene Proteins c-bcl-2/metabolism ; RNA, Messenger ; RNA, Small Interfering ; Survivin/metabolism ; Triple Negative Breast Neoplasms/pathology ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
Effects of microRNA-133b on retinal vascular endothelial cell proliferation and apoptosis through angiotensinogen-mediated angiotensin II- extracellular signal-regulated kinase 1/2 signalling pathway in rats with diabetic retinopathy.
Autorzy :
Liu TT; Department of Ophthalmology, First Hospital of Jilin University, Changchun, China.
Hao Q; Department of Ophthalmology, First Hospital of Jilin University, Changchun, China.
Zhang Y; Department of Otorhinolaryngology, Head and Neck Surgery, First Hospital of Jilin University, Changchun, China.
Li ZH; Department of Ophthalmology, People's Hospital of Changchun City, Changchun, China.
Cui ZH; Department of Ophthalmology, First Hospital of Jilin University, Changchun, China.
Yang W; Department of Ophthalmology, First Hospital of Jilin University, Changchun, China.
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Źródło :
Acta ophthalmologica [Acta Ophthalmol] 2018 Aug; Vol. 96 (5), pp. e626-e635. Date of Electronic Publication: 2018 Feb 28.
Typ publikacji :
Journal Article
MeSH Terms :
Diabetes Mellitus, Experimental*
Gene Expression Regulation*
Angiotensin II/*metabolism
Apoptosis/*genetics
Diabetic Retinopathy/*genetics
MAP Kinase Signaling System/*physiology
MicroRNAs/*genetics
Animals ; Blotting, Western ; Cell Proliferation ; Cells, Cultured ; Diabetic Retinopathy/metabolism ; Diabetic Retinopathy/pathology ; Flow Cytometry ; Immunohistochemistry ; Male ; MicroRNAs/biosynthesis ; RNA/genetics ; Rats ; Rats, Sprague-Dawley ; Real-Time Polymerase Chain Reaction ; Signal Transduction
Czasopismo naukowe
Tytuł :
Structural characterization of a polysaccharide from the flower buds of Tussilago farfara, and its effect on proliferation and apoptosis of A549 human non-small lung cancer cell line.
Autorzy :
Qu H; Department of Hematology and Oncology, Inner Mongolia Forestry General Hospital, Yakeshi 022150, China.
Yang W; Department of Thoracic Surgery, Beijing Chest Hospital, Capital Medical University, Beijing 101149, China.
Li J; Department of Oncology, Beijing Chest Hospital, Capital Medical University, Beijing 101149, China. Electronic address: .
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Źródło :
International journal of biological macromolecules [Int J Biol Macromol] 2018 Jul 01; Vol. 113, pp. 849-858. Date of Electronic Publication: 2018 Mar 02.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Carcinoma, Non-Small-Cell Lung/*pathology
Flowers/*chemistry
Lung Neoplasms/*pathology
Polysaccharides/*chemistry
Polysaccharides/*pharmacology
Tussilago/*chemistry
A549 Cells ; Antineoplastic Agents/chemistry ; Antineoplastic Agents/pharmacology ; Cell Cycle/drug effects ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Humans ; Molecular Weight ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction/drug effects
Czasopismo naukowe
Tytuł :
Fisetin induces apoptosis through mitochondrial apoptosis pathway in human uveal melanoma cells.
Autorzy :
Wang K; Institute of Medicine, Chung Shan Medical University, Taichung, Taiwan.; Department of Ophthalmology, Sijhih Cathay General Hospital, New Taipei City, Taiwan.; Department of Ophthalmology, Cathay General Hospital, Taipei, Taiwan.
Hu DN; Tissue Culture Center, New York Eye and Ear Infirmary of Mount Sinai, Icahn School of Medicine at Mount Sinai, New York, New York.; Department of Ophthalmology, New York Eye and Ear Infirmary of Mount Sinai, Icahn School of Medicine at Mount Sinai, New York, New York.
Lin HW; Department of Optometry, Asia University, Taichung, Taiwan.; Department of Medical Research, China Medical University Hospital, China Medical University, Taichung, Taiwan.
Yang WE; Institute of Medicine, Chung Shan Medical University, Taichung, Taiwan.; Department of Medical Research, Chung Shan Medical University Hospital, Taichung, Taiwan.
Hsieh YH; Institute of Biochemistry, Microbiology and Immunology, Chung Shan Medical University, Taichung, Taiwan.
Chien HW; Institute of Medicine, Chung Shan Medical University, Taichung, Taiwan.; Department of Ophthalmology, Sijhih Cathay General Hospital, New Taipei City, Taiwan.; Department of Ophthalmology, Cathay General Hospital, Taipei, Taiwan.
Yang SF; Institute of Medicine, Chung Shan Medical University, Taichung, Taiwan.; Department of Medical Research, Chung Shan Medical University Hospital, Taichung, Taiwan.
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Źródło :
Environmental toxicology [Environ Toxicol] 2018 May; Vol. 33 (5), pp. 527-534. Date of Electronic Publication: 2018 Jan 31.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Flavonoids/*pharmacology
Melanoma/*pathology
Mitochondria/*drug effects
Uveal Neoplasms/*pathology
Cell Line, Tumor ; Cell Survival/drug effects ; Cytochromes c/metabolism ; Humans ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/metabolism ; Proto-Oncogene Proteins c-bcl-2/metabolism
SCR Disease Name :
Uveal melanoma
Czasopismo naukowe
Tytuł :
Protective effect of rutin against carbon tetrachloride-induced oxidative stress, inflammation and apoptosis in mouse kidney associated with the ceramide, MAPKs, p53 and calpain activities.
Autorzy :
Ma JQ; School of Chemical Engineering, Sichuan University of Science and Engineering, No. 180, Huixing Road, 643000, Zigong City, Sichuan Province, PR China. Electronic address: .
Liu CM; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tangshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China.
Yang W; School of Life Science, Jiangsu Normal University, No.101, Shanghai Road, Tangshan New Area, 221116, Xuzhou City, Jiangsu Province, PR China.
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Źródło :
Chemico-biological interactions [Chem Biol Interact] 2018 Apr 25; Vol. 286, pp. 26-33. Date of Electronic Publication: 2018 Mar 06.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Inflammation/*prevention & control
Kidney/*drug effects
Oxidative Stress/*drug effects
Rutin/*pharmacology
Animals ; Calpain/metabolism ; Carbon Tetrachloride/toxicity ; Caspase 3/metabolism ; Ceramides/metabolism ; Cytochromes c/metabolism ; Inflammation/chemically induced ; Kidney/metabolism ; Male ; Mice ; Mice, Inbred ICR ; Mitogen-Activated Protein Kinases/metabolism ; Protective Agents/pharmacology ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Reactive Oxygen Species/metabolism ; Tumor Suppressor Protein p53/metabolism ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł :
Autophagy promotes escape from phosphatidylinositol 3-kinase inhibition in estrogen receptor-positive breast cancer.
Autorzy :
Yang W; Department of Molecular and Systems Biology, Norris Cotton Cancer Center, Geisel School of Medicine at Dartmouth, Lebanon, New Hampshire, USA.
Hosford SR; Department of Molecular and Systems Biology, Norris Cotton Cancer Center, Geisel School of Medicine at Dartmouth, Lebanon, New Hampshire, USA.
Traphagen NA; Department of Molecular and Systems Biology, Norris Cotton Cancer Center, Geisel School of Medicine at Dartmouth, Lebanon, New Hampshire, USA.
Shee K; Department of Molecular and Systems Biology, Norris Cotton Cancer Center, Geisel School of Medicine at Dartmouth, Lebanon, New Hampshire, USA.
Demidenko E; Community and Family Medicine, Norris Cotton Cancer Center, Geisel School of Medicine at Dartmouth, Lebanon, New Hampshire, USA; and.
Liu S; Department of Molecular and Systems Biology, Norris Cotton Cancer Center, Geisel School of Medicine at Dartmouth, Lebanon, New Hampshire, USA.
Miller TW; Department of Molecular and Systems Biology, Norris Cotton Cancer Center, Geisel School of Medicine at Dartmouth, Lebanon, New Hampshire, USA.; Comprehensive Breast Program, Norris Cotton Cancer Center, Geisel School of Medicine at Dartmouth, Lebanon, New Hampshire, USA.
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Źródło :
FASEB journal : official publication of the Federation of American Societies for Experimental Biology [FASEB J] 2018 Mar; Vol. 32 (3), pp. 1222-1235. Date of Electronic Publication: 2018 Jan 03.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Autophagy*
Phosphoinositide-3 Kinase Inhibitors*
Apoptosis/*drug effects
Breast Neoplasms/*pathology
Chloroquine/*pharmacology
Receptors, Estrogen/*metabolism
Animals ; Antimalarials/pharmacology ; Breast Neoplasms/drug therapy ; Breast Neoplasms/metabolism ; Cell Proliferation/drug effects ; Female ; Humans ; Mice ; Mice, Inbred NOD ; Mice, SCID ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction ; Tumor Cells, Cultured ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
DLP1-dependent mitochondrial fragmentation and redistribution mediate prion-associated mitochondrial dysfunction and neuronal death.
Autorzy :
Li C; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Wang D; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Wu W; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Yang W; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Ali Shah SZ; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Zhao Y; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Duan Y; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Wang L; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Zhou X; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Zhao D; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Yang L; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
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Źródło :
Aging cell [Aging Cell] 2018 Feb; Vol. 17 (1). Date of Electronic Publication: 2017 Nov 22.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*physiology
Dynamins/*metabolism
Mitochondria/*metabolism
Neurons/*metabolism
Animals ; Cell Line ; Mice ; Mitochondrial Dynamics/physiology ; Nerve Degeneration/pathology ; Parkinson Disease/metabolism
Czasopismo naukowe
Tytuł :
Down-regulation of long non-coding RNA MALAT1 by RNA interference inhibits proliferation and induces apoptosis in multiple myeloma.
Autorzy :
Liu H; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Wang H; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Wu B; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Yao K; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Liao A; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Miao M; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Li Y; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Yang W; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
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Źródło :
Clinical and experimental pharmacology & physiology [Clin Exp Pharmacol Physiol] 2017 Oct; Vol. 44 (10), pp. 1032-1041. Date of Electronic Publication: 2017 Aug 24.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*genetics
Down-Regulation/*genetics
Multiple Myeloma/*pathology
RNA, Long Noncoding/*genetics
Animals ; Carcinogenesis/genetics ; Cell Cycle Checkpoints/genetics ; Cell Line, Tumor ; Cell Proliferation/genetics ; Gene Silencing ; Humans ; RNA Interference ; RNA, Long Noncoding/metabolism
Czasopismo naukowe
Tytuł :
FSTL1 contributes to tumor progression via attenuating apoptosis in a AKT/GSK-3β - dependent manner in hepatocellular carcinoma.
Autorzy :
Yang W; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.
Wu Y; Department of General Surgery, Xi'an Dangcheng Hospital, Xi'an 710051, Shaanxi, China.; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.
Wang C; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.
Liu Z; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.
Xu M; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.; Department of Bioengineering and Therapeutic Sciences, Schools of Pharmacy and Medicine, University of California, San Francisco, CA 94143, USA.
Zheng X; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.
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Źródło :
Cancer biomarkers : section A of Disease markers [Cancer Biomark] 2017 Jul 19; Vol. 20 (1), pp. 75-85.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Carcinoma, Hepatocellular/*metabolism
Follistatin-Related Proteins/*metabolism
Glycogen Synthase Kinase 3 beta/*metabolism
Liver Neoplasms/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Carcinoma, Hepatocellular/genetics ; Carcinoma, Hepatocellular/surgery ; Cell Line, Tumor ; Disease Progression ; Female ; Follistatin-Related Proteins/genetics ; Hep G2 Cells ; Humans ; Kaplan-Meier Estimate ; Liver Neoplasms/genetics ; Liver Neoplasms/surgery ; Middle Aged ; Multivariate Analysis ; Prognosis ; RNA Interference ; Signal Transduction
Czasopismo naukowe
Tytuł :
Hippocampal Apoptosis and Cognitive Function in Dysbindin-1B Mice.
Autorzy :
Yang W; State Key Laboratory of Medical Molecular Biology,Department of Biochemistry and Molecular Biology,Institute of Basic Medical Sciences,CAMS and PUMC,Beijing 100005,China.
Xu Q; State Key Laboratory of Medical Molecular Biology,Department of Biochemistry and Molecular Biology,Institute of Basic Medical Sciences,CAMS and PUMC,Beijing 100005,China.
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Źródło :
Zhongguo yi xue ke xue yuan xue bao. Acta Academiae Medicinae Sinicae [Zhongguo Yi Xue Ke Xue Yuan Xue Bao] 2017 Jun 20; Vol. 39 (3), pp. 307-311.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Cognition*
Dysbindin/*metabolism
Hippocampus/*pathology
Animals ; Anxiety ; Carrier Proteins ; Locomotion ; Male ; Mice ; Mice, Inbred C57BL ; Neurons/pathology ; Spatial Memory
Czasopismo naukowe
Tytuł :
PRAS40 alleviates neurotoxic prion peptide-induced apoptosis via mTOR-AKT signaling.
Autorzy :
Yang W; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.; Hebei Institute of Animal Science and Veterinary Medicine, Baoding, China.
Yang LF; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Song ZQ; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Shah SZA; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Cui YY; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Li CS; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Zhao HF; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Gao HL; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Zhou XM; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Zhao DM; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
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Źródło :
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2017 May; Vol. 23 (5), pp. 416-427. Date of Electronic Publication: 2017 Mar 14.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*physiology
Neurons/*metabolism
Phosphoproteins/*metabolism
Pregnancy Proteins/*toxicity
Proto-Oncogene Proteins c-akt/*metabolism
TOR Serine-Threonine Kinases/*metabolism
Animals ; Cell Line, Tumor ; Drug Evaluation, Preclinical ; Mice ; Phosphoproteins/genetics ; Prion Diseases/drug therapy ; Reactive Oxygen Species/metabolism ; Signal Transduction ; Transfection
Czasopismo naukowe
Tytuł :
Antileukemic activity of an arsenomolybdate in the human HL-60 and U937 leukemia cells.
Autorzy :
Li C; School of Public Health, Jilin University, Changchun, Jilin 130021, PR China.
Cao H; School of Public Health, Jilin University, Changchun, Jilin 130021, PR China.
Sun J; School of Public Health, Jilin University, Changchun, Jilin 130021, PR China.
Tian R; School of Public Health, Jilin University, Changchun, Jilin 130021, PR China.
Li D; College of Basic Medical Science, Jilin University, Changchun, Jilin 130021, PR China.
Qi Y; School of Public Health, Jilin University, Changchun, Jilin 130021, PR China. Electronic address: .
Yang W; College of Basic Medical Science, Jilin University, Changchun, Jilin 130021, PR China. Electronic address: .
Li J; School of Public Health, Jilin University, Changchun, Jilin 130021, PR China.
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Źródło :
Journal of inorganic biochemistry [J Inorg Biochem] 2017 Mar; Vol. 168, pp. 67-75. Date of Electronic Publication: 2016 Dec 16.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Arsenicals/*pharmacology
Caspase 3/*drug effects
Leukemia, Promyelocytic, Acute/*drug therapy
Molybdenum/*pharmacology
Antineoplastic Agents/chemical synthesis ; Antineoplastic Agents/chemistry ; Arsenicals/chemical synthesis ; Arsenicals/chemistry ; Blotting, Western ; Cell Cycle/drug effects ; Cell Proliferation/drug effects ; Drug Stability ; Flow Cytometry ; Fluorescence ; HL-60 Cells ; Humans ; Inhibitory Concentration 50 ; Molybdenum/chemistry ; U937 Cells
Czasopismo naukowe

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