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Tytuł:
Neuronal Apoptosis Preventive Potential of Sophocarpine via Suppression of Aβ-Accumulation and Down-Regulation of Inflammatory Response.
Autorzy:
Zhu X; Changchun University of Chinese Medicine, 130117, Changchun, Jilin, China.
Gu Z; Neurology, Third Affiliated Clinical Hospital of Changchun University of Traditional Chinese Medicine, 130117, Changchun, Jilin, China.
Yu Y; Neurology, Songyuan Central Hospital, 138000, Songyuan, Jilin, China.
Yang W; GCP Office, Affiliated Hospital of Changchun University of Traditional Chinese Medicine, 130021, Changchun, Jilin, China.
Li M; Neurology, Third Affiliated Clinical Hospital of Changchun University of Traditional Chinese Medicine, 130117, Changchun, Jilin, China.
Li Y; Ultrasound Electrophysiological Diagnostic Center, Third Affiliated Clinical Hospital of Changchun University of Traditional Chinese Medicine, 130117, Changchun, Jilin, China.
Zhang P; Changchun University of Chinese Medicine, 130117, Changchun, Jilin, China.
Wang J; Changchun University of Chinese Medicine, 130117, Changchun, Jilin, China.
Zhao J; Neurology, Affiliated Hospital of Changchun University of Traditional Chinese Medicine, 130021, Changchun, Jilin, China. .
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Źródło:
Doklady. Biochemistry and biophysics [Dokl Biochem Biophys] 2021 Mar; Vol. 497 (1), pp. 116-122. Date of Electronic Publication: 2021 Apr 24.
Typ publikacji:
Journal Article
MeSH Terms:
Alkaloids/*pharmacology
Amyloid beta-Peptides/*metabolism
Apoptosis/*drug effects
Neurons/*drug effects
Neuroprotective Agents/*pharmacology
Animals ; Cyclooxygenase 2/metabolism ; Inflammation/metabolism ; Inflammation/pathology ; NF-kappa B/metabolism ; Neurons/cytology ; Neurons/metabolism ; Neurons/pathology ; PC12 Cells ; Rats ; Signal Transduction/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Enhancing the chemotherapy effect of Apatinib on gastric cancer by co-treating with salidroside to reprogram the tumor hypoxia micro-environment and induce cell apoptosis.
Autorzy:
Zhang Z; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Yang W; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Ma F; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Ma Q; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Zhang B; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Zhang Y; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Liu Y; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Liu H; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
Hua Y; Department of General Surgery, Affiliated Tumor Hospital of Zhengzhou University, Henan Cancer Hospital, Henan, Zhengzhou.
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Źródło:
Drug delivery [Drug Deliv] 2020 Dec; Vol. 27 (1), pp. 691-702.
Typ publikacji:
Journal Article
MeSH Terms:
Antineoplastic Agents/*administration & dosage
Apoptosis/*drug effects
Glucosides/*administration & dosage
Phenols/*administration & dosage
Pyridines/*administration & dosage
Stomach Neoplasms/*pathology
Tumor Hypoxia/*drug effects
Tumor Microenvironment/*drug effects
Animals ; Antineoplastic Agents/pharmacokinetics ; Antineoplastic Agents/pharmacology ; Cell Line, Tumor ; Cell Movement/drug effects ; Cell Proliferation/drug effects ; Drug Carriers/chemistry ; Drug Liberation ; Glucosides/pharmacology ; Humans ; Male ; Mice, Inbred BALB C ; Molecular Targeted Therapy ; Nanoparticles/chemistry ; Phenols/pharmacology ; Pyridines/pharmacology ; Stomach Neoplasms/drug therapy ; Treatment Outcome ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Downregulation of ENDOCAN in myeloid leukemia cells inhibits proliferation and promotes apoptosis by suppressing nuclear factor‑κB activity.
Autorzy:
Sun L; Department of Hematology, Shengjing Hospital of China Medical University, Shenyang, Liaoning 110004, P.R. China.
Sun C; Anorectal Department, Shenyang Anorectal Hospital, Shenyang, Liaoning 110054, P.R. China.
Sun J; Intensive Care Unit, The Fourth Affiliated Hospital of China Medical University, Shenyang, Liaoning 110032, P.R. China.
Yang W; Department of Hematology, Shengjing Hospital of China Medical University, Shenyang, Liaoning 110004, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2019 Apr; Vol. 19 (4), pp. 3247-3254. Date of Electronic Publication: 2019 Feb 19.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Expression Regulation, Leukemic*
Apoptosis/*genetics
Leukemia, Myeloid/*genetics
Leukemia, Myeloid/*metabolism
NF-kappa B/*metabolism
Neoplasm Proteins/*genetics
Proteoglycans/*genetics
Cell Cycle Checkpoints/genetics ; Cell Line, Tumor ; Cell Proliferation ; Gene Silencing ; HL-60 Cells ; Humans ; K562 Cells ; Leukemia, Myeloid/pathology ; U937 Cells
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Effects of microRNA-133b on retinal vascular endothelial cell proliferation and apoptosis through angiotensinogen-mediated angiotensin II- extracellular signal-regulated kinase 1/2 signalling pathway in rats with diabetic retinopathy.
Autorzy:
Liu TT; Department of Ophthalmology, First Hospital of Jilin University, Changchun, China.
Hao Q; Department of Ophthalmology, First Hospital of Jilin University, Changchun, China.
Zhang Y; Department of Otorhinolaryngology, Head and Neck Surgery, First Hospital of Jilin University, Changchun, China.
Li ZH; Department of Ophthalmology, People's Hospital of Changchun City, Changchun, China.
Cui ZH; Department of Ophthalmology, First Hospital of Jilin University, Changchun, China.
Yang W; Department of Ophthalmology, First Hospital of Jilin University, Changchun, China.
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Źródło:
Acta ophthalmologica [Acta Ophthalmol] 2018 Aug; Vol. 96 (5), pp. e626-e635. Date of Electronic Publication: 2018 Feb 28.
Typ publikacji:
Journal Article
MeSH Terms:
Diabetes Mellitus, Experimental*
Gene Expression Regulation*
Angiotensin II/*metabolism
Apoptosis/*genetics
Diabetic Retinopathy/*genetics
MAP Kinase Signaling System/*physiology
MicroRNAs/*genetics
Animals ; Blotting, Western ; Cell Proliferation ; Cells, Cultured ; Diabetic Retinopathy/metabolism ; Diabetic Retinopathy/pathology ; Flow Cytometry ; Immunohistochemistry ; Male ; MicroRNAs/biosynthesis ; RNA/genetics ; Rats ; Rats, Sprague-Dawley ; Real-Time Polymerase Chain Reaction ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
DLP1-dependent mitochondrial fragmentation and redistribution mediate prion-associated mitochondrial dysfunction and neuronal death.
Autorzy:
Li C; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Wang D; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Wu W; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Yang W; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Ali Shah SZ; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Zhao Y; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Duan Y; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Wang L; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Zhou X; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Zhao D; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
Yang L; National Animal Transmissible Spongiform Encephalopathy Laboratory, State Key Laboratories for Agrobiotechnology, Key Laboratory of Animal Epidemiology and Zoonosis, College of Veterinary Medicine, Ministry of Agriculture, China Agricultural University, Beijing, 100193, China.
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Źródło:
Aging cell [Aging Cell] 2018 Feb; Vol. 17 (1). Date of Electronic Publication: 2017 Nov 22.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*physiology
Dynamins/*metabolism
Mitochondria/*metabolism
Neurons/*metabolism
Animals ; Cell Line ; Mice ; Mitochondrial Dynamics/physiology ; Nerve Degeneration/pathology ; Parkinson Disease/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Down-regulation of long non-coding RNA MALAT1 by RNA interference inhibits proliferation and induces apoptosis in multiple myeloma.
Autorzy:
Liu H; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Wang H; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Wu B; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Yao K; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Liao A; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Miao M; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Li Y; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
Yang W; Department of Hematology, Affiliated Shengjing Hospital of China Medical University, Shenyang, Liaoning Province, China.
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Źródło:
Clinical and experimental pharmacology & physiology [Clin Exp Pharmacol Physiol] 2017 Oct; Vol. 44 (10), pp. 1032-1041. Date of Electronic Publication: 2017 Aug 24.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*genetics
Down-Regulation/*genetics
Multiple Myeloma/*pathology
RNA, Long Noncoding/*genetics
Animals ; Carcinogenesis/genetics ; Cell Cycle Checkpoints/genetics ; Cell Line, Tumor ; Cell Proliferation/genetics ; Gene Silencing ; Humans ; RNA Interference ; RNA, Long Noncoding/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
FSTL1 contributes to tumor progression via attenuating apoptosis in a AKT/GSK-3β - dependent manner in hepatocellular carcinoma.
Autorzy:
Yang W; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.
Wu Y; Department of General Surgery, Xi'an Dangcheng Hospital, Xi'an 710051, Shaanxi, China.; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.
Wang C; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.
Liu Z; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.
Xu M; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.; Department of Bioengineering and Therapeutic Sciences, Schools of Pharmacy and Medicine, University of California, San Francisco, CA 94143, USA.
Zheng X; Department of Hepatobiliary Surgery, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, Shaanxi, China.
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Źródło:
Cancer biomarkers : section A of Disease markers [Cancer Biomark] 2017 Jul 19; Vol. 20 (1), pp. 75-85.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Carcinoma, Hepatocellular/*metabolism
Follistatin-Related Proteins/*metabolism
Glycogen Synthase Kinase 3 beta/*metabolism
Liver Neoplasms/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Carcinoma, Hepatocellular/genetics ; Carcinoma, Hepatocellular/surgery ; Cell Line, Tumor ; Disease Progression ; Female ; Follistatin-Related Proteins/genetics ; Hep G2 Cells ; Humans ; Kaplan-Meier Estimate ; Liver Neoplasms/genetics ; Liver Neoplasms/surgery ; Middle Aged ; Multivariate Analysis ; Prognosis ; RNA Interference ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
PRAS40 alleviates neurotoxic prion peptide-induced apoptosis via mTOR-AKT signaling.
Autorzy:
Yang W; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.; Hebei Institute of Animal Science and Veterinary Medicine, Baoding, China.
Yang LF; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Song ZQ; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Shah SZA; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Cui YY; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Li CS; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Zhao HF; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Gao HL; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Zhou XM; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
Zhao DM; National Animal Transmissible Spongiform Encephalopathy Laboratory and Key Laboratory of Animal Epidemiology and Zoonosis of Ministry of Agriculture, College of Veterinary Medicine and State Key Laboratory of Agrobiotechnology, China Agricultural University, Beijing, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2017 May; Vol. 23 (5), pp. 416-427. Date of Electronic Publication: 2017 Mar 14.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*physiology
Neurons/*metabolism
Phosphoproteins/*metabolism
Pregnancy Proteins/*toxicity
Proto-Oncogene Proteins c-akt/*metabolism
TOR Serine-Threonine Kinases/*metabolism
Animals ; Cell Line, Tumor ; Drug Evaluation, Preclinical ; Mice ; Phosphoproteins/genetics ; Prion Diseases/drug therapy ; Reactive Oxygen Species/metabolism ; Signal Transduction ; Transfection
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Docetaxel-Loaded Self-Assembly Stearic Acid-Modified Bletilla striata Polysaccharide Micelles and Their Anticancer Effect: Preparation, Characterization, Cellular Uptake and In Vitro Evaluation.
Autorzy:
Guan Q; Department of Pharmaceutics, School of Pharmacy, Jilin University, No. 1266, Fujin Road, Changchun 130021, China. .
Sun D; Department of Pharmaceutics, School of Pharmacy, Jilin University, No. 1266, Fujin Road, Changchun 130021, China. .
Zhang G; Department of Pharmaceutics, School of Pharmacy, Jilin University, No. 1266, Fujin Road, Changchun 130021, China. .
Sun C; Department of Pharmaceutics, School of Pharmacy, Jilin University, No. 1266, Fujin Road, Changchun 130021, China. .
Wang M; Department of Pharmaceutics, School of Pharmacy, Jilin University, No. 1266, Fujin Road, Changchun 130021, China. .
Ji D; Department of Pharmaceutics, School of Pharmacy, Jilin University, No. 1266, Fujin Road, Changchun 130021, China. .
Yang W; Department of Neurology, Second Hospital of Jilin University, Changchun 130041, China. .
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2016 Dec 02; Vol. 21 (12). Date of Electronic Publication: 2016 Dec 02.
Typ publikacji:
Journal Article
MeSH Terms:
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Drug Carriers/*pharmacology
Neoplasms/*drug therapy
Polysaccharides/*pharmacology
Taxoids/*pharmacology
Cell Line, Tumor ; Docetaxel ; Drug Liberation ; HeLa Cells ; Hep G2 Cells ; Humans ; Hydrophobic and Hydrophilic Interactions ; MCF-7 Cells ; Micelles ; Nuclear Magnetic Resonance, Biomolecular ; Particle Size ; Plant Roots/chemistry ; Solubility ; Stearic Acids/chemistry
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
The protein PprI provides protection against radiation injury in human and mouse cells.
Autorzy:
Shi Y; Department of Radiation Toxicology, School of Radiological Medicine and Protection, Medical College of Soochow University, Collaborative Innovation Center of Radiation Medicine of Jiangsu Higher Education Institutions, Soochow University, Suzhou, Jiangsu 215123, China.
Wu W; Department of Radiation Toxicology, School of Radiological Medicine and Protection, Medical College of Soochow University, Collaborative Innovation Center of Radiation Medicine of Jiangsu Higher Education Institutions, Soochow University, Suzhou, Jiangsu 215123, China.
Qiao H; Department of Radiation Toxicology, School of Radiological Medicine and Protection, Medical College of Soochow University, Collaborative Innovation Center of Radiation Medicine of Jiangsu Higher Education Institutions, Soochow University, Suzhou, Jiangsu 215123, China.
Yue L; Department of Radiation Toxicology, School of Radiological Medicine and Protection, Medical College of Soochow University, Collaborative Innovation Center of Radiation Medicine of Jiangsu Higher Education Institutions, Soochow University, Suzhou, Jiangsu 215123, China.
Ren L; Department of Radiation Toxicology, School of Radiological Medicine and Protection, Medical College of Soochow University, Collaborative Innovation Center of Radiation Medicine of Jiangsu Higher Education Institutions, Soochow University, Suzhou, Jiangsu 215123, China.
Zhang S; Department of Radiation Genetics, School of Radiological Medicine and Protection, Medical College of Soochow University, Collaborative Innovation Center of Radiation Medicine of Jiangsu Higher Education Institutions, Soochow University, Suzhou, Jiangsu 215123, China.
Yang W; Department of Radiobiology, School of Radiological Medicine and Protection, Medical College of Soochow University, Collaborative Innovation Center of Radiation Medicine of Jiangsu Higher Education Institutions, Soochow University, Suzhou, Jiangsu 215123, China.
Yang Z; Department of Radiation Toxicology, School of Radiological Medicine and Protection, Medical College of Soochow University, Collaborative Innovation Center of Radiation Medicine of Jiangsu Higher Education Institutions, Soochow University, Suzhou, Jiangsu 215123, China.
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Źródło:
Scientific reports [Sci Rep] 2016 May 25; Vol. 6, pp. 26664. Date of Electronic Publication: 2016 May 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/drug effects
Apoptosis*/radiation effects
Bacterial Proteins/*pharmacology
Deinococcus/*genetics
Gamma Rays/*adverse effects
Radiation Injuries, Experimental/*prevention & control
Radiation-Protective Agents/*pharmacology
Animals ; Bacterial Proteins/genetics ; Human Umbilical Vein Endothelial Cells ; Humans ; Male ; Mice ; Mice, Inbred BALB C ; Radiation Injuries, Experimental/genetics ; Radiation Injuries, Experimental/metabolism ; Radiation Injuries, Experimental/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Hydrogen-Rich Saline Attenuates Cardiac and Hepatic Injury in Doxorubicin Rat Model by Inhibiting Inflammation and Apoptosis.
Autorzy:
Gao Y; Department of Cardiology, The Fourth Affiliated Hospital of Harbin Medical University, 37 Yiyuan Street, Harbin, Heilongjiang 150001, China.
Yang H; Department of Cardiology, The First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, Harbin, Heilongjiang 150001, China.
Fan Y; Department of Cardiology, The First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, Harbin, Heilongjiang 150001, China.
Li L; Department of Cardiology, The First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, Harbin, Heilongjiang 150001, China.
Fang J; Department of Cardiology, The First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, Harbin, Heilongjiang 150001, China.
Yang W; Department of Cardiology, The First Affiliated Hospital of Harbin Medical University, 23 Youzheng Street, Harbin, Heilongjiang 150001, China.
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Źródło:
Mediators of inflammation [Mediators Inflamm] 2016; Vol. 2016, pp. 1320365. Date of Electronic Publication: 2016 Dec 26.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Chemical and Drug Induced Liver Injury/*drug therapy
Doxorubicin/*adverse effects
Heart Injuries/*drug therapy
Hydrogen/*therapeutic use
Sodium Chloride/*therapeutic use
Animals ; Anti-Inflammatory Agents/chemistry ; Anti-Inflammatory Agents/therapeutic use ; Antioxidants/metabolism ; Aspartate Aminotransferases/blood ; Echocardiography ; Heart/drug effects ; Heart Injuries/chemically induced ; Hydrogen/chemistry ; Inflammation/drug therapy ; Male ; Malondialdehyde/blood ; Natriuretic Peptide, Brain/metabolism ; Oxidative Stress ; Rats ; Rats, Wistar ; Reactive Oxygen Species/metabolism ; Serum Albumin/metabolism ; Sodium Chloride/chemistry
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Anticancer effect of 2,7-dihydroxy-3-methylanthraquinone on human gastric cancer SGC-7901 cells in vitro and in vivo.
Autorzy:
Zhu H; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Zheng Z; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Zhang J; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Liu X; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Liu Y; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Yang W; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Liu Y; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Zhang T; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Zhao Y; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Liu Y; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Su X; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
Gu X; a Department of Gastric Surgery , Liaoning Cancer Hospital & Institute , Shenyang , PR China.
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Źródło:
Pharmaceutical biology [Pharm Biol] 2016; Vol. 54 (2), pp. 285-92. Date of Electronic Publication: 2015 Apr 08.
Typ publikacji:
Journal Article
MeSH Terms:
Anthraquinones/*pharmacology
Antineoplastic Agents, Phytogenic/*therapeutic use
Apoptosis/*drug effects
Drugs, Chinese Herbal/*therapeutic use
Hedyotis/*chemistry
Stomach Neoplasms/*drug therapy
Animals ; Anthraquinones/chemistry ; Anthraquinones/isolation & purification ; Anthraquinones/therapeutic use ; Antineoplastic Agents, Phytogenic/isolation & purification ; Antineoplastic Agents, Phytogenic/pharmacology ; Apoptosis/genetics ; Cell Culture Techniques ; Cell Line, Tumor ; Cell Survival/drug effects ; Dose-Response Relationship, Drug ; Drugs, Chinese Herbal/isolation & purification ; Drugs, Chinese Herbal/pharmacology ; Flow Cytometry ; Gene Expression Regulation, Neoplastic/drug effects ; Humans ; Mice, Nude ; Mitochondria/drug effects ; Mitochondria/metabolism ; Stomach Neoplasms/genetics ; Stomach Neoplasms/metabolism ; Stomach Neoplasms/pathology ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Diet-induced obesity exacerbates auditory degeneration via hypoxia, inflammation, and apoptosis signaling pathways in CD/1 mice.
Autorzy:
Hwang JH; Graduate Institute of Clinical Medicine, College of Medicine, National Taiwan University, Taipei, Taiwan.
Hsu CJ
Yu WH
Liu TC
Yang WS
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Źródło:
PloS one [PLoS One] 2013 Apr 26; Vol. 8 (4), pp. e60730. Date of Electronic Publication: 2013 Apr 26 (Print Publication: 2013).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Signal Transduction*
Cochlea/*pathology
Hearing Loss/*etiology
Hypoxia/*etiology
Inflammation/*etiology
Obesity/*complications
Animals ; Diet ; Disease Progression ; Hearing Loss/pathology ; Hypoxia/pathology ; Immunohistochemistry ; Inflammation/pathology ; Male ; Mice ; Obesity/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
1-benzyl-2-phenylbenzimidazole (BPB), a benzimidazole derivative, induces cell apoptosis in human chondrosarcoma through intrinsic and extrinsic pathways.
Autorzy:
Liu JF; Central Laboratory, Shin Kong Wu Ho-Su Memorial Hospital, No.95, Wunchang Road, Shihlin District, Taipei City 111, Taiwan. .
Huang YL
Yang WH
Chang CS
Tang CH
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2012 Dec 04; Vol. 13 (12), pp. 16472-88. Date of Electronic Publication: 2012 Dec 04.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Benzimidazoles/*pharmacology
Bone Neoplasms/*pathology
Chondrosarcoma/*pathology
Animals ; Benzimidazoles/chemistry ; Cells, Cultured ; Humans ; Male ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Mice, SCID ; Signal Transduction/drug effects ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Apoptosis of the dermal papilla cells of hair follicle associated with the expression of gene HSPCO16 in vitro.
Autorzy:
Yang WB; Department of Dermatology, Southwest Hospital, Third Military Medical University, Chongqing 400038, China.
Hao F
Song ZQ
Yang XC
Ni B
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Źródło:
Experimental dermatology [Exp Dermatol] 2005 Mar; Vol. 14 (3), pp. 209-14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*genetics
Hair Follicle/*physiology
Proteins/*genetics
3T3 Cells ; Animals ; Cadaver ; Cells, Cultured ; Cloning, Molecular ; Gene Expression ; Hair Follicle/cytology ; Hair Follicle/metabolism ; Humans ; In Situ Hybridization ; In Situ Nick-End Labeling ; Mice ; Reverse Transcriptase Polymerase Chain Reaction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Deep learning nomogram based on Gd-EOB-DTPA MRI for predicting early recurrence in hepatocellular carcinoma after hepatectomy
Autorzy:
Yan, Meng
Zhang, XiaoAff1, Aff2
Zhang, Bin
Geng, Zhijun
Xie, Chuanmiao
Yang, Wei
Zhang, Shuixing
Qi, Zhendong
Lin, Ting
Ke, Qiying
Li, XinmingAff5, IDs00330023094190_cor11
Wang, ShutongAff7, IDs00330023094190_cor12
Quan, XianyueAff5, IDs00330023094190_cor13
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Źródło:
European Radiology. 33(7):4949-4961
Czasopismo naukowe
Szczegóły
Wyświetlanie 1-20 z 500

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