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Tytuł:
Icaritin activates Nrf2/Keap1 signaling to protect neuronal cells from oxidative stress.
Autorzy:
Xu Y; State Key Laboratory of Generic Manufacture Technology of Chinese Traditional Medicine, Linyi, China.; Linyi Key Laboratory for Immunopharmacology and Immunotoxicology of Natural Medicine, Lunan Pharmaceutical Group Co. Ltd., Linyi, China.; Lunan Pharmaceutical Group Co. Ltd., Linyi, China.
Lu X; State Key Laboratory of Generic Manufacture Technology of Chinese Traditional Medicine, Linyi, China.; Linyi Key Laboratory for Immunopharmacology and Immunotoxicology of Natural Medicine, Lunan Pharmaceutical Group Co. Ltd., Linyi, China.; Lunan Pharmaceutical Group Co. Ltd., Linyi, China.
Zhang L; State Key Laboratory of Generic Manufacture Technology of Chinese Traditional Medicine, Linyi, China.; Lunan Pharmaceutical Group Co. Ltd., Linyi, China.
Wang L; State Key Laboratory of Generic Manufacture Technology of Chinese Traditional Medicine, Linyi, China.; Linyi Key Laboratory for Immunopharmacology and Immunotoxicology of Natural Medicine, Lunan Pharmaceutical Group Co. Ltd., Linyi, China.; Lunan Pharmaceutical Group Co. Ltd., Linyi, China.
Zhang G; State Key Laboratory of Generic Manufacture Technology of Chinese Traditional Medicine, Linyi, China.; Linyi Key Laboratory for Immunopharmacology and Immunotoxicology of Natural Medicine, Lunan Pharmaceutical Group Co. Ltd., Linyi, China.; Lunan Pharmaceutical Group Co. Ltd., Linyi, China.
Yao J; State Key Laboratory of Generic Manufacture Technology of Chinese Traditional Medicine, Linyi, China.; Linyi Key Laboratory for Immunopharmacology and Immunotoxicology of Natural Medicine, Lunan Pharmaceutical Group Co. Ltd., Linyi, China.; Lunan Pharmaceutical Group Co. Ltd., Linyi, China.
Sun C; State Key Laboratory of Generic Manufacture Technology of Chinese Traditional Medicine, Linyi, China.; Linyi Key Laboratory for Immunopharmacology and Immunotoxicology of Natural Medicine, Lunan Pharmaceutical Group Co. Ltd., Linyi, China.; Lunan Pharmaceutical Group Co. Ltd., Linyi, China.
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Źródło:
Chemical biology & drug design [Chem Biol Drug Des] 2021 Jan; Vol. 97 (1), pp. 111-120. Date of Electronic Publication: 2020 Jul 26.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Flavonoids/*pharmacology
Oxidative Stress/*drug effects
Signal Transduction/*drug effects
Antioxidants/chemistry ; Cell Line, Tumor ; Cell Survival/drug effects ; Heme Oxygenase-1/metabolism ; Humans ; Hydrogen Peroxide/pharmacology ; Kelch-Like ECH-Associated Protein 1/metabolism ; Malondialdehyde/metabolism ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/metabolism ; NAD(P)H Dehydrogenase (Quinone)/metabolism ; NF-E2-Related Factor 2/metabolism ; Reactive Oxygen Species/metabolism ; Superoxide Dismutase/metabolism ; Up-Regulation/drug effects ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł:
The Synthetic Flavonoid Derivative GL-V9 Induces Apoptosis and Autophagy in Cutaneous Squamous Cell Carcinoma via Suppressing AKT-Regulated HK2 and mTOR Signals.
Autorzy:
Zhu Y; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Carcinogenesis and Intervention, China Pharmaceutical University, 24 Tongjiaxiang, Nanjing 210009, China.; School of Medicine & Holistic Integrative Medicine, Nanjing University of Chinese Medicine, 138 Xianlin Rd, Nanjing 210033, China.
Liu M; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Carcinogenesis and Intervention, China Pharmaceutical University, 24 Tongjiaxiang, Nanjing 210009, China.
Yao J; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Carcinogenesis and Intervention, China Pharmaceutical University, 24 Tongjiaxiang, Nanjing 210009, China.
Guo Q; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Carcinogenesis and Intervention, China Pharmaceutical University, 24 Tongjiaxiang, Nanjing 210009, China.
Wei L; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Carcinogenesis and Intervention, China Pharmaceutical University, 24 Tongjiaxiang, Nanjing 210009, China.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2020 Oct 30; Vol. 25 (21). Date of Electronic Publication: 2020 Oct 30.
Typ publikacji:
Journal Article
MeSH Terms:
Carcinoma, Squamous Cell*/drug therapy
Carcinoma, Squamous Cell*/metabolism
Carcinoma, Squamous Cell*/pathology
Skin Neoplasms*/drug therapy
Skin Neoplasms*/metabolism
Skin Neoplasms*/pathology
Apoptosis/*drug effects
Autophagy/*drug effects
Flavonoids/*pharmacology
Animals ; Cell Line, Tumor ; Female ; Flavonoids/chemistry ; Humans ; Mice ; Mice, Nude ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł:
Kallikrein 12 Regulates Innate Resistance of Murine Macrophages against Mycobacterium bovis Infection by Modulating Autophagy and Apoptosis.
Autorzy:
Sabir N; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. .
Hussain T; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. .
Liao Y; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. liaoyi_.
Wang J; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. .
Song Y; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. .
Shahid M; Department of Clinical Veterinary Medicine, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. drshahid_.
Cheng G; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. .
Mangi MH; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. .
Yao J; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. mole-yao@hotmail.com.
Yang L; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. .
Zhao D; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. .
Zhou X; Key Laboratory of Animal Epidemiology and Zoonosis, Ministry of Agriculture, National Animal Transmissible Spongiform Encephalopathy Laboratory, College of Veterinary Medicine, China Agricultural University, Beijing 100193, China. .
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Źródło:
Cells [Cells] 2019 May 05; Vol. 8 (5). Date of Electronic Publication: 2019 May 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Autophagy*
Immunity, Innate*
Kallikreins/*metabolism
Macrophages/*pathology
Mycobacterium bovis/*physiology
Tuberculosis, Bovine/*immunology
Tuberculosis, Bovine/*microbiology
Animals ; Cattle ; Cytokines/metabolism ; Macrophages/microbiology ; Mice ; Mice, Inbred C57BL ; Microbial Viability ; RAW 264.7 Cells ; Signal Transduction ; Tuberculosis, Bovine/pathology
Czasopismo naukowe
Tytuł:
Arsenic trioxide decreases the amount and inhibits the function of regulatory T cells, which may contribute to its efficacy in the treatment of acute promyelocytic leukemia.
Autorzy:
Xu W; a Department of Immunology , Harbin Medical University , Harbin , People's Republic of China.
Li X; b Department of Hematology , The First Affiliated Hospital of Harbin Medical University , Harbin , People's Republic of China.
Quan L; c Department of Hematology , Harbin Medical University Cancer Hospital , Harbin , People's Republic of China.
Yao J; a Department of Immunology , Harbin Medical University , Harbin , People's Republic of China.
Mu G; a Department of Immunology , Harbin Medical University , Harbin , People's Republic of China.
Guo J; a Department of Immunology , Harbin Medical University , Harbin , People's Republic of China.
Wang Y; a Department of Immunology , Harbin Medical University , Harbin , People's Republic of China.
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Źródło:
Leukemia & lymphoma [Leuk Lymphoma] 2018 Mar; Vol. 59 (3), pp. 650-659. Date of Electronic Publication: 2017 Jul 06.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Arsenic Trioxide/*pharmacology
Cell Proliferation/*drug effects
Leukemia, Promyelocytic, Acute/*immunology
T-Lymphocytes, Regulatory/*immunology
Antineoplastic Agents/pharmacology ; Case-Control Studies ; Follow-Up Studies ; Humans ; Leukemia, Promyelocytic, Acute/blood ; Leukemia, Promyelocytic, Acute/drug therapy ; Leukemia, Promyelocytic, Acute/pathology ; Prognosis ; T-Lymphocytes, Regulatory/drug effects
Czasopismo naukowe
Tytuł:
5-Aminolevulinic Acid-Mediated Sonodynamic Therapy Inhibits RIPK1/RIPK3-Dependent Necroptosis in THP-1-Derived Foam Cells.
Autorzy:
Tian F; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.
Yao J; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.
Yan M; Department of Pathophysiology and the Key Laboratory of Cardiovascular Pathophysiology, Harbin Medical University, the Key Laboratory of Cardiovascular Research (Harbin Medical University), Ministry of Education, Harbin 150081, P. R. China.
Sun X; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.
Wang W; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.
Gao W; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.
Tian Z; Department of Pathophysiology and the Key Laboratory of Cardiovascular Pathophysiology, Harbin Medical University, the Key Laboratory of Cardiovascular Research (Harbin Medical University), Ministry of Education, Harbin 150081, P. R. China.
Guo S; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.
Dong Z; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.
Li B; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.
Gao T; Department of Forensic Medicine, Harbin Medical University, Harbin 150001, P. R. China.
Shan P; Department of Vascular Surgery, the First Affiliated Hospital of Harbin Medical University, Harbin 150001, P. R. China.
Liu B; Department of Vascular Surgery, the First Affiliated Hospital of Harbin Medical University, Harbin 150001, P. R. China.
Wang H; Department of Vascular Surgery, the First Affiliated Hospital of Harbin Medical University, Harbin 150001, P. R. China.
Cheng J; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.
Gao Q; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.
Zhang Z; Laboratory of Photo- and Sono-theranostic Technologies and Condensed Matter Science and Technology Institute, Harbin Institute of Technology, Harbin 150080, P. R. China.
Cao W; Laboratory of Photo- and Sono-theranostic Technologies and Condensed Matter Science and Technology Institute, Harbin Institute of Technology, Harbin 150080, P. R. China.; Materials Research Institute, The Pennsylvania State University, University Park 16802, PA, USA.
Tian Y; Department of Cardiology, the First Affiliated Hospital, Cardiovascular Institute, Harbin Medical University, Harbin, 150001, P. R. China.; Department of Pathophysiology and the Key Laboratory of Cardiovascular Pathophysiology, Harbin Medical University, the Key Laboratory of Cardiovascular Research (Harbin Medical University), Ministry of Education, Harbin 150081, P. R. China.; Heilongjiang Academy of Medical Sciences, Harbin 150086, P. R. China.
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Źródło:
Scientific reports [Sci Rep] 2016 Feb 25; Vol. 6, pp. 21992. Date of Electronic Publication: 2016 Feb 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Necrosis*
Ultrasonic Waves*
Aminolevulinic Acid/*pharmacology
Foam Cells/*metabolism
Receptor-Interacting Protein Serine-Threonine Kinases/*metabolism
Atherosclerosis/metabolism ; Atherosclerosis/pathology ; Atherosclerosis/therapy ; Caspase 3/metabolism ; Caspase 8/metabolism ; Cell Line, Tumor ; Foam Cells/pathology ; Foam Cells/ultrastructure ; Humans ; Imidazoles/metabolism ; Indoles/metabolism
Czasopismo naukowe
Tytuł:
Extracellular Acidification Acts as a Key Modulator of Neutrophil Apoptosis and Functions.
Autorzy:
Cao S; State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
Liu P; State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
Zhu H; State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
Gong H; State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
Yao J; Hematopoietic Stem Cell Transplantation Center, Institute of Hematology and Blood Diseases Hospital, Peking Union Medical College and Chinese Academy of Medical Sciences, Tianjin, China.
Sun Y; State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
Geng G; State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
Wang T; State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
Feng S; Hematopoietic Stem Cell Transplantation Center, Institute of Hematology and Blood Diseases Hospital, Peking Union Medical College and Chinese Academy of Medical Sciences, Tianjin, China.
Han M; Hematopoietic Stem Cell Transplantation Center, Institute of Hematology and Blood Diseases Hospital, Peking Union Medical College and Chinese Academy of Medical Sciences, Tianjin, China.
Zhou J; State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
Xu Y; State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China.
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Źródło:
PloS one [PLoS One] 2015 Sep 04; Vol. 10 (9), pp. e0137221. Date of Electronic Publication: 2015 Sep 04 (Print Publication: 2015).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*immunology
Chemotaxis/*immunology
Neutrophils/*immunology
Respiratory Burst/*immunology
Actins/genetics ; Actins/immunology ; Apoptosis/drug effects ; Caspase 3/genetics ; Caspase 3/immunology ; Chemotaxis/drug effects ; Escherichia coli/immunology ; Gene Expression Regulation ; Humans ; Hydrogen-Ion Concentration ; Immunity, Innate ; N-Formylmethionine Leucyl-Phenylalanine/pharmacology ; Neutrophils/cytology ; Neutrophils/drug effects ; Phagocytosis/drug effects ; Phosphorylation ; Primary Cell Culture ; Proto-Oncogene Proteins c-akt/genetics ; Proto-Oncogene Proteins c-akt/immunology ; Reactive Oxygen Species/immunology ; Reactive Oxygen Species/metabolism ; Respiratory Burst/drug effects ; Signal Transduction
Czasopismo naukowe
Tytuł:
Ischemia/reperfusion-induced CHOP expression promotes apoptosis and impairs renal function recovery: the role of acidosis and GPR4.
Autorzy:
Dong B; Department of Transplantation and Hepatic Surgery, Renji Hospital, School of Medicine, Shanghai Jiaotong University, Shanghai, China; Department of Urology, First Hospital of Jilin University, Changchun, China.
Zhou H; Department of Urology, First Hospital of Jilin University, Changchun, China.
Han C; Department of Urology, The Affiliated School of Clinical Medicine of Xuzhou Medical College, Xuzhou Central Hospital, Xuzhou, China.
Yao J; Animal Facility of Renji Hospital, School of Medicine, Shanghai Jiaotong University, Shanghai, China.
Xu L; The Central Laboratory of Renji Hospital, School of Medicine, Shanghai Jiaotong University, Shanghai, China.
Zhang M; Department of Transplantation and Hepatic Surgery, Renji Hospital, School of Medicine, Shanghai Jiaotong University, Shanghai, China.
Fu Y; Department of Urology, First Hospital of Jilin University, Changchun, China.
Xia Q; Department of Transplantation and Hepatic Surgery, Renji Hospital, School of Medicine, Shanghai Jiaotong University, Shanghai, China.
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Źródło:
PloS one [PLoS One] 2014 Oct 24; Vol. 9 (10), pp. e110944. Date of Electronic Publication: 2014 Oct 24 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/drug effects
Recovery of Function*/drug effects
Acidosis/*metabolism
Receptors, G-Protein-Coupled/*metabolism
Reperfusion Injury/*metabolism
Reperfusion Injury/*pathology
Transcription Factor CHOP/*metabolism
8-Bromo Cyclic Adenosine Monophosphate/pharmacology ; Acidosis/complications ; Animals ; Bone Marrow Transplantation ; Caspase 3/metabolism ; Cyclic AMP/metabolism ; Epithelial Cells/drug effects ; Epithelial Cells/metabolism ; Gene Knockdown Techniques ; Human Umbilical Vein Endothelial Cells/drug effects ; Human Umbilical Vein Endothelial Cells/metabolism ; Hydrochloric Acid/pharmacology ; Hypoxia/complications ; Hypoxia/metabolism ; Kidney/blood supply ; Kidney/physiopathology ; Male ; Mice, Inbred C57BL ; Mice, Knockout ; Microcirculation/drug effects ; Regional Blood Flow/drug effects
Czasopismo naukowe
Tytuł:
Heat shock protein B6 potently increases non-small cell lung cancer growth.
Autorzy:
Chen S; Department of Cardiothoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu 215006, P.R. China.
Huang H; Department of Cardiothoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu 215006, P.R. China.
Yao J; Department of Cardiothoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu 215006, P.R. China.
Pan L; Department of Cardiothoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu 215006, P.R. China.
Ma H; Department of Cardiothoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu 215006, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2014 Aug; Vol. 10 (2), pp. 677-82. Date of Electronic Publication: 2014 May 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Carcinoma, Lewis Lung/*pathology
HSP20 Heat-Shock Proteins/*pharmacology
Lung Neoplasms/*pathology
Animals ; Carcinoma, Lewis Lung/blood supply ; Cell Line, Tumor ; Cell Movement/drug effects ; Cell Proliferation/drug effects ; Fibroblast Growth Factor 2/genetics ; Fibroblast Growth Factor 2/metabolism ; HSP20 Heat-Shock Proteins/genetics ; HSP20 Heat-Shock Proteins/metabolism ; Intercellular Adhesion Molecule-1/genetics ; Intercellular Adhesion Molecule-1/metabolism ; Lung Neoplasms/blood supply ; Lymphatic Metastasis ; Mice ; Mice, Inbred BALB C ; Neovascularization, Pathologic ; Recombinant Proteins/biosynthesis ; Recombinant Proteins/genetics ; Recombinant Proteins/pharmacology ; Transplantation, Homologous ; Vascular Endothelial Growth Factor A/genetics ; Vascular Endothelial Growth Factor A/metabolism
Czasopismo naukowe
Tytuł:
Epithelial-mesenchymal transition and apoptosis of renal tubular epithelial cells are associated with disease progression in patients with IgA nephropathy.
Autorzy:
Yao J; Institute of Pathology, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, P.R. China.
Ke Z; Department of Pathology, Hubei University of Medicine, Shiyan, Hubei 442000, P.R. China.
Wang X; Institute of Pathology, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, P.R. China.
Peng F; Department of Pathology, Hubei University of Medicine, Shiyan, Hubei 442000, P.R. China.
Li B; Department of Pathology, Hubei University of Medicine, Shiyan, Hubei 442000, P.R. China.
Wu R; Institute of Pathology, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2014 Jul; Vol. 10 (1), pp. 39-44. Date of Electronic Publication: 2014 Apr 24.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Glomerulonephritis, IGA/*pathology
Kidney Tubules/*metabolism
Actins/metabolism ; Adolescent ; Adult ; Collagen/metabolism ; Disease Progression ; Epithelial Cells/metabolism ; Epithelial Cells/pathology ; Epithelial-Mesenchymal Transition ; Female ; Glomerulonephritis, IGA/metabolism ; Humans ; Hyperplasia/metabolism ; Kidney Tubules/pathology ; Male ; Middle Aged ; Neoplasm Grading ; Proteins/analysis ; Sclerosis/metabolism ; Vimentin/metabolism ; Young Adult
Czasopismo naukowe
Tytuł:
Cordycepin as a sensitizer to tumour necrosis factor (TNF)-α-induced apoptosis through eukaryotic translation initiation factor 2α (eIF2α)- and mammalian target of rapamycin complex 1 (mTORC1)-mediated inhibition of nuclear factor (NF)-κB.
Autorzy:
Kadomatsu M; Department of Molecular Signaling, Interdisciplinary Graduate School of Medicine and Engineering, University of Yamanashi, Chuo, Yamanashi, Japan.
Nakajima S
Kato H
Gu L
Chi Y
Yao J
Kitamura M
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Źródło:
Clinical and experimental immunology [Clin Exp Immunol] 2012 Jun; Vol. 168 (3), pp. 325-32.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Cordyceps/*immunology
Deoxyadenosines/*pharmacology
Epithelial Cells/*drug effects
Animals ; Cell Line ; Cell Survival/drug effects ; Epithelial Cells/metabolism ; Epithelial Cells/pathology ; Kidney Tubules/pathology ; NF-kappa B/antagonists & inhibitors ; Nucleoside Transport Proteins/metabolism ; Phosphorylation/drug effects ; Rats ; Receptor, Adenosine A3/metabolism ; Signal Transduction/drug effects ; Sirolimus/pharmacology ; TOR Serine-Threonine Kinases/metabolism ; Tumor Necrosis Factor-alpha/immunology ; Tumor Necrosis Factor-alpha/metabolism ; eIF-2 Kinase/metabolism
Czasopismo naukowe
Tytuł:
Sporamin induce apoptosis in human tongue carcinoma cells by down-regulating Akt/GSK-3 signaling.
Autorzy:
Yao J; School of Medicine, Taizhou University, Jiaojiang District, Taizhou, Zhejiang 318000, China. />Qian C
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Źródło:
Fundamental & clinical pharmacology [Fundam Clin Pharmacol] 2011 Apr; Vol. 25 (2), pp. 229-36.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Plant Proteins/*pharmacology
Tongue Neoplasms/*drug therapy
Antineoplastic Agents/administration & dosage ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Dose-Response Relationship, Drug ; Down-Regulation/drug effects ; Glycogen Synthase Kinase 3/drug effects ; Glycogen Synthase Kinase 3/metabolism ; Humans ; Phosphorylation/drug effects ; Plant Proteins/administration & dosage ; Proto-Oncogene Proteins c-akt/drug effects ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction/drug effects ; Tongue Neoplasms/pathology
Czasopismo naukowe
Tytuł:
Antisense oligonucleotide against miR-21 inhibits migration and induces apoptosis in leukemic K562 cells.
Autorzy:
Hu H; Department of Biochemistry and Molecular Biology, Medical College of Jinan University, Guangzhou 510632, China.
Li Y
Gu J
Zhu X
Dong D
Yao J
Lin C
Fei J
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Źródło:
Leukemia & lymphoma [Leuk Lymphoma] 2010 Apr; Vol. 51 (4), pp. 694-701.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Cell Movement/*drug effects
Leukemia, Myelogenous, Chronic, BCR-ABL Positive/*pathology
MicroRNAs/*antagonists & inhibitors
Oligonucleotides, Antisense/*pharmacology
Apoptosis/genetics ; Apoptosis Regulatory Proteins/genetics ; Apoptosis Regulatory Proteins/metabolism ; Cell Movement/genetics ; Down-Regulation/drug effects ; Down-Regulation/genetics ; Drug Evaluation, Preclinical ; Gene Expression Regulation, Leukemic ; Humans ; K562 Cells ; Leukemia, Myelogenous, Chronic, BCR-ABL Positive/genetics ; MicroRNAs/genetics ; RNA-Binding Proteins/genetics ; RNA-Binding Proteins/metabolism ; Validation Studies as Topic
Czasopismo naukowe
Tytuł:
Atypical, bidirectional regulation of cadmium-induced apoptosis via distinct signaling of unfolded protein response.
Autorzy:
Yokouchi M; Department of Molecular Signaling, Interdisciplinary Graduate School of Medicine and Engineering, University of Yamanashi, Chuo, Yamanashi 409-3898, Japan.
Hiramatsu N
Hayakawa K
Kasai A
Takano Y
Yao J
Kitamura M
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Źródło:
Cell death and differentiation [Cell Death Differ] 2007 Aug; Vol. 14 (8), pp. 1467-74. Date of Electronic Publication: 2007 Apr 27.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Apoptosis/*physiology
Cadmium/*toxicity
Protein Denaturation/*drug effects
Activating Transcription Factor 6/metabolism ; Animals ; DNA-Binding Proteins/metabolism ; Endoplasmic Reticulum/drug effects ; Endoplasmic Reticulum/metabolism ; Endoplasmic Reticulum Chaperone BiP ; Eukaryotic Initiation Factor-2/metabolism ; Gene Expression/drug effects ; Heat-Shock Proteins/genetics ; JNK Mitogen-Activated Protein Kinases/metabolism ; Kidney Tubules, Proximal/drug effects ; Kidney Tubules, Proximal/metabolism ; Kidney Tubules, Proximal/pathology ; LLC-PK1 Cells ; Models, Biological ; Molecular Chaperones/genetics ; Nuclear Proteins/metabolism ; Regulatory Factor X Transcription Factors ; Signal Transduction/drug effects ; Swine ; Transcription Factor CHOP/genetics ; Transcription Factors ; eIF-2 Kinase/metabolism
Czasopismo naukowe
Tytuł:
Increased population of CD4(+)CD25(high), regulatory T cells with their higher apoptotic and proliferating status in peripheral blood of acute myeloid leukemia patients.
Autorzy:
Wang X; Center for Stem Cell Research and Application, Institute of Hematology, Union Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zheng J
Liu J
Yao J
He Y
Li X
Yu J
Yang J
Liu Z
Huang S
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Źródło:
European journal of haematology [Eur J Haematol] 2005 Dec; Vol. 75 (6), pp. 468-76.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Proliferation*
Apoptosis/*immunology
CD4-Positive T-Lymphocytes/*immunology
Leukemia, Myeloid, Acute/*immunology
Receptors, Interleukin-2/*immunology
Adolescent ; Adult ; CD4 Lymphocyte Count/methods ; CD4-Positive T-Lymphocytes/pathology ; Cells, Cultured ; Coculture Techniques ; Cytokines/blood ; Cytokines/immunology ; Female ; Humans ; Ki-67 Antigen/immunology ; Leukemia, Myeloid, Acute/blood ; Leukemia, Myeloid, Acute/pathology ; Male ; Middle Aged ; Receptors, Interleukin-2/blood
Czasopismo naukowe
Tytuł:
By blocking hexokinase-2 phosphorylation, limonin suppresses tumor glycolysis and induces cell apoptosis in hepatocellular carcinoma
Autorzy:
Yao J
Liu J
Zhao W
Pokaż więcej
Temat:
Apoptosis
Limonin
Tumor glycolysis
Hexokinase-2
Neoplasms. Tumors. Oncology. Including cancer and carcinogens
RC254-282
Źródło:
OncoTargets and Therapy, Vol Volume 11, Pp 3793-3803 (2018)
Opis pliku:
electronic resource
Relacje:
https://www.dovepress.com/by-blocking-hexokinase-2-phosphorylation-limonin-suppresses-tumor-glyc-peer-reviewed-article-OTT; https://doaj.org/toc/1178-6930
Dostęp URL:
https://doaj.org/article/7828ebf1c06247218b4d3fac0a6ca5a4  Link otwiera się w nowym oknie
Czasopismo naukowe

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