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Tytuł:
Cuproptosis-Related LncRNA-Based Prediction of the Prognosis and Immunotherapy Response in Papillary Renal Cell Carcinoma.
Autorzy:
Pang Y; Department of Bioinformatics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.; Department of Biophysics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.
Wang Y; Department of Biophysics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.
Zhou X; Department of Bioinformatics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.; Department of Biophysics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.
Ni Z; Department of Bioinformatics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.; Department of Biophysics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.
Chen W; Department of Bioinformatics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.; Department of Biophysics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.
Liu Y; Department of Bioinformatics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.; Department of Biophysics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.
Du W; Department of Bioinformatics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.; Department of Biophysics, School of Life Sciences, Xuzhou Medical University, Xuzhou 221004, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Jan 11; Vol. 24 (2). Date of Electronic Publication: 2023 Jan 11.
Typ publikacji:
Journal Article
MeSH Terms:
Carcinoma, Renal Cell*/genetics
Carcinoma, Renal Cell*/therapy
Kidney Neoplasms*/genetics
Kidney Neoplasms*/therapy
RNA, Long Noncoding*/genetics
Apoptosis*
Humans ; Creatine Kinase, Mitochondrial Form ; Immunotherapy ; Phosphatidylinositol 3-Kinases ; Tumor Microenvironment/genetics ; Copper
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Identification of cuproptosis-related long noncoding RNA signature for predicting prognosis and immunotherapy response in bladder cancer.
Autorzy:
Huang G; Department of Urology, Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Huang Y; Department of Urology, Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Zhang C; Department of Urology, Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Jiang Y; Department of Urology, Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Ye Z; Department of Urology, Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
He C; Jiangxi Key Laboratory of Molecular Medicine, Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Yu F; Department of Obstetrics and Gynecology, First Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Chen Z; Department of Urology, Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
Xi X; Department of Urology, Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China. .
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Źródło:
Scientific reports [Sci Rep] 2022 Dec 10; Vol. 12 (1), pp. 21386. Date of Electronic Publication: 2022 Dec 10.
Typ publikacji:
Journal Article
MeSH Terms:
RNA, Long Noncoding*/genetics
Urinary Bladder Neoplasms*/genetics
Urinary Bladder Neoplasms*/therapy
Apoptosis*
Humans ; Immunotherapy ; Prognosis ; Risk Factors ; Copper
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Hypoxia Induces Apoptosis of Microglia BV2 by Upregulating Kir2.1 to Activate Mitochondrial-Related Apoptotic Pathways.
Autorzy:
Xie YF; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.
Wang Y; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.
Rong Y; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.
He W; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Pathophysiology, Shihezi University School of Medicine, Shihezi, China.
Yan M; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Pathophysiology, Shihezi University School of Medicine, Shihezi, China.
Li X; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Pathophysiology, Shihezi University School of Medicine, Shihezi, China.
Si J; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.
Li L; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Medical College of Jiaxing University, Jiaxing, Zhejiang, China.
Zhang Y; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.; Clinical Laboratory of the First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.
Ma K; Key Laboratory of Xinjiang Endemic and Ethnic Diseases, Ministry of Education, Shihezi University School of Medicine, Shihezi, China.; NHC Key Laboratory of Prevention and Treatment of Central Asia High Incidence Diseases, First Affiliated Hospital, Shihezi University School of Medicine, Shihezi, China.; Department of Physiology, Shihezi University School of Medicine, Shihezi, China.
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Źródło:
Disease markers [Dis Markers] 2022 Mar 17; Vol. 2022, pp. 5855889. Date of Electronic Publication: 2022 Mar 17 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*/genetics
Microglia*/metabolism
Cell Hypoxia ; Humans ; Hypoxia/metabolism ; Mitochondria/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Live cell tracking of macrophage efferocytosis during Drosophila embryo development in vivo.
Autorzy:
Raymond MH; Center for Cell Clearance, University of Virginia, Charlottesville, VA, USA.; Department of Microbiology, Immunology, and Cancer Biology, University of Virginia, Charlottesville, VA, USA.; Neuroscience Graduate Program, University of Virginia, Charlottesville, VA, USA.
Davidson AJ; Centre for Inflammation Research, University of Edinburgh, Edinburgh BioQuarter, Edinburgh, UK.
Shen Y; Department of Chemistry, University of Alberta, Edmonton, Canada.
Tudor DR; Centre for Regenerative Medicine, University of Edinburgh, Edinburgh BioQuarter, Edinburgh, UK.
Lucas CD; Centre for Inflammation Research, University of Edinburgh, Edinburgh BioQuarter, Edinburgh, UK.
Morioka S; Center for Cell Clearance, University of Virginia, Charlottesville, VA, USA.; Department of Microbiology, Immunology, and Cancer Biology, University of Virginia, Charlottesville, VA, USA.; Department of Medicine, Division of Nephrology and CIIR, University of Virginia, Charlottesville, VA, USA.
Perry JSA; Immunology Program, Memorial Sloan Kettering Cancer Center, New York, NY 10065, USA.
Krapivkina J; University of Bordeaux, CNRS, Interdisciplinary Institute for Neuroscience (IINS), UMR 5297, Bordeaux, France.
Perrais D; University of Bordeaux, CNRS, Interdisciplinary Institute for Neuroscience (IINS), UMR 5297, Bordeaux, France.
Schumacher LJ; Centre for Regenerative Medicine, University of Edinburgh, Edinburgh BioQuarter, Edinburgh, UK.
Campbell RE; Department of Chemistry, University of Alberta, Edmonton, Canada.; Department of Chemistry, School of Science, The University of Tokyo, Bunkyo-ku, Tokyo, 113-0033, Japan.
Wood W; Centre for Inflammation Research, University of Edinburgh, Edinburgh BioQuarter, Edinburgh, UK.
Ravichandran KS; Center for Cell Clearance, University of Virginia, Charlottesville, VA, USA.; Department of Microbiology, Immunology, and Cancer Biology, University of Virginia, Charlottesville, VA, USA.; VIB/UGent Inflammation Research Centre, and Biomedical Molecular Biology, Ghent University, Belgium.; Division of Immunobiology, Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, MO, USA.
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Źródło:
Science (New York, N.Y.) [Science] 2022 Mar 11; Vol. 375 (6585), pp. 1182-1187. Date of Electronic Publication: 2022 Mar 10.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Cell Tracking*
Embryonic Development*
Phagocytosis*
Drosophila/*embryology
Macrophages/*physiology
Animals ; Hydrogen-Ion Concentration
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Chemical Characterization, Antitumor, and Immune-Enhancing Activities of Polysaccharide from Sargassum pallidum .
Autorzy:
Gao Y; College of Marine Science and Engineering, Qingdao Agricultural University, Qingdao 266109, China.
Li Y; College of Life Sciences, Qingdao Agricultural University, Qingdao 266109, China.
Niu Y; College of Life Sciences, Qingdao Agricultural University, Qingdao 266109, China.
Ju H; College of Life Sciences, Qingdao Agricultural University, Qingdao 266109, China.
Chen R; College of Life Sciences, Qingdao Agricultural University, Qingdao 266109, China.
Li B; College of Marine Science and Engineering, Qingdao Agricultural University, Qingdao 266109, China.
Song X; College of Agronomy, Qingdao Agricultural University, Qingdao 266109, China.
Song L; College of Marine Science and Biological Engineering, Qingdao University of Science and Technology, Qingdao 266042, China.; Shandong Provincial Key Laboratory of Biochemical Engineering, Qingdao 266042, China.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2021 Dec 13; Vol. 26 (24). Date of Electronic Publication: 2021 Dec 13.
Typ publikacji:
Journal Article
MeSH Terms:
Antineoplastic Agents*/chemistry
Antineoplastic Agents*/pharmacology
Immunomodulating Agents*/chemistry
Immunomodulating Agents*/pharmacology
Apoptosis/*drug effects
Melanoma, Experimental/*drug therapy
Sargassum/*chemistry
A549 Cells ; Animals ; Drug Screening Assays, Antitumor ; Hep G2 Cells ; Humans ; Melanoma, Experimental/metabolism ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Microcystin-Induced Immunotoxicity in Fishes: A Scoping Review.
Autorzy:
Lin W; Hunan Provincial Collaborative Innovation Center for Efficient and Health Production of Fisheries, Hunan Provincial Key Laboratory for Health Aquaculture and Product Processing in Dongting Lake Area, Hunan Provincial Key Laboratory for Molecular Immunity Technology of Aquatic Animal Diseases, Hunan Engineering Research Center of Aquatic Organism Resources and Environmental Ecology, Zoology Key Laboratory of Hunan Higher Education, College of Life and Environmental Sciences, Hunan University of Arts and Science, Changde 415000, China.; Department of Biological and Agricultural Engineering, University of California, Davis, CA 95616, USA.; Department of Fisheries Resources and Environment, College of Fisheries, Huazhong Agricultural University, Wuhan 430070, China.
Hung TC; Department of Biological and Agricultural Engineering, University of California, Davis, CA 95616, USA.
Kurobe T; Department of Anatomy, Physiology, and Cell Biology, University of California, Davis, CA 95616, USA.
Wang Y; Department of Biological and Agricultural Engineering, University of California, Davis, CA 95616, USA.
Yang P; Hunan Provincial Collaborative Innovation Center for Efficient and Health Production of Fisheries, Hunan Provincial Key Laboratory for Health Aquaculture and Product Processing in Dongting Lake Area, Hunan Provincial Key Laboratory for Molecular Immunity Technology of Aquatic Animal Diseases, Hunan Engineering Research Center of Aquatic Organism Resources and Environmental Ecology, Zoology Key Laboratory of Hunan Higher Education, College of Life and Environmental Sciences, Hunan University of Arts and Science, Changde 415000, China.
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Źródło:
Toxins [Toxins (Basel)] 2021 Oct 29; Vol. 13 (11). Date of Electronic Publication: 2021 Oct 29.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Fishes*/immunology
Fishes*/metabolism
Apoptosis/*drug effects
Immunotoxins/*toxicity
Inflammation/*immunology
Microcystins/*toxicity
Oxidative Stress/*drug effects
Animals ; Fish Diseases/chemically induced ; Fish Diseases/immunology ; Inflammation/chemically induced ; Protein Phosphatase 1/immunology ; Protein Phosphatase 2/immunology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
CdGAP promotes prostate cancer metastasis by regulating epithelial-to-mesenchymal transition, cell cycle progression, and apoptosis.
Autorzy:
Mehra C; Cancer Research Program, Research Institute of the McGill University Health Centre, Montréal, QC, Canada.; Department of Anatomy and Cell Biology, McGill University, Montréal, QC, Canada.
Chung JH; Cancer Research Program, Research Institute of the McGill University Health Centre, Montréal, QC, Canada.; Department of Anatomy and Cell Biology, McGill University, Montréal, QC, Canada.
He Y; Cancer Research Program, Research Institute of the McGill University Health Centre, Montréal, QC, Canada.; Department of Anatomy and Cell Biology, McGill University, Montréal, QC, Canada.
Lara-Márquez M; Cancer Research Program, Research Institute of the McGill University Health Centre, Montréal, QC, Canada.; Department of Anatomy and Cell Biology, McGill University, Montréal, QC, Canada.
Goyette MA; Institut de Recherches Cliniques de Montréal, Université de Montréal, Montréal, QC, Canada.
Boufaied N; Cancer Research Program, Research Institute of the McGill University Health Centre, Montréal, QC, Canada.
Barrès V; Centre de Recherche du Centre Hospitalier de l'Université de Montréal et Institut du Cancer de Montréal, Montréal, QC, Canada.
Ouellet V; Centre de Recherche du Centre Hospitalier de l'Université de Montréal et Institut du Cancer de Montréal, Montréal, QC, Canada.
Guérard KP; Cancer Research Program, Research Institute of the McGill University Health Centre, Montréal, QC, Canada.
Delliaux C; Institut de Recherches Cliniques de Montréal, Université de Montréal, Montréal, QC, Canada.
Saad F; Centre de Recherche du Centre Hospitalier de l'Université de Montréal et Institut du Cancer de Montréal, Montréal, QC, Canada.; Department of Surgery, Université de Montréal, Montréal, QC, Canada.
Lapointe J; Cancer Research Program, Research Institute of the McGill University Health Centre, Montréal, QC, Canada.; Division of Urology, Department of Surgery, McGill University, Montréal, QC, Canada.
Côté JF; Department of Anatomy and Cell Biology, McGill University, Montréal, QC, Canada.; Institut de Recherches Cliniques de Montréal, Université de Montréal, Montréal, QC, Canada.
Labbé DP; Cancer Research Program, Research Institute of the McGill University Health Centre, Montréal, QC, Canada. .; Department of Anatomy and Cell Biology, McGill University, Montréal, QC, Canada. .; Division of Urology, Department of Surgery, McGill University, Montréal, QC, Canada. .
Lamarche-Vane N; Cancer Research Program, Research Institute of the McGill University Health Centre, Montréal, QC, Canada. .; Department of Anatomy and Cell Biology, McGill University, Montréal, QC, Canada. .
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Źródło:
Communications biology [Commun Biol] 2021 Sep 07; Vol. 4 (1), pp. 1042. Date of Electronic Publication: 2021 Sep 07.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Cell Cycle*
Epithelial-Mesenchymal Transition*
Neoplasm Metastasis*
Prostatic Neoplasms/*pathology
Animals ; Male ; Mice ; Mice, Nude
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
circRNA-Mediated Inhibin-Activin Balance Regulation in Ovarian Granulosa Cell Apoptosis and Follicular Atresia.
Autorzy:
Ma M; College of Animal Science and Technology, Nanjing Agriculture University, Nanjing 210095, China.
Wang H; College of Animal Science and Technology, Nanjing Agriculture University, Nanjing 210095, China.
Zhang Y; College of Animal Science and Technology, Nanjing Agriculture University, Nanjing 210095, China.
Zhang J; College of Animal Science and Food Engineering, Jinling Institute of Technology, Nanjing 211169, China.
Liu J; College of Animal Science and Food Engineering, Jinling Institute of Technology, Nanjing 211169, China.
Pan Z; College of Animal Science and Technology, Nanjing Agriculture University, Nanjing 210095, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Aug 24; Vol. 22 (17). Date of Electronic Publication: 2021 Aug 24.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Activins/*genetics
Follicular Atresia/*metabolism
Granulosa Cells/*metabolism
Inhibins/*genetics
MicroRNAs/*genetics
RNA, Circular/*metabolism
Animals ; Female ; Follicular Atresia/physiology ; Gene Expression Regulation ; Granulosa Cells/physiology ; Inhibins/metabolism ; MicroRNAs/metabolism ; Ovary/metabolism ; Ovary/physiology ; Sus scrofa/genetics ; Sus scrofa/metabolism ; Sus scrofa/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Chiral nanomaterials for tumor therapy: autophagy, apoptosis, and photothermal ablation.
Autorzy:
Peng Z; Department of Breast Surgery, Southwest Hospital, Army Medical University, Chongqing, 400038, China.
Yuan L; Department of Breast Surgery, Southwest Hospital, Army Medical University, Chongqing, 400038, China.
XuHong J; Department of Breast Surgery, Southwest Hospital, Army Medical University, Chongqing, 400038, China.
Tian H; Department of Breast Surgery, Southwest Hospital, Army Medical University, Chongqing, 400038, China.
Zhang Y; Department of Breast Surgery, Southwest Hospital, Army Medical University, Chongqing, 400038, China. .
Deng J; Institute of Burn Research, Southwest Hospital, State Key Lab of Trauma, Burn and Combined Injury, Army Medical University, Chongqing, 400038, China. .
Qi X; Department of Breast Surgery, Southwest Hospital, Army Medical University, Chongqing, 400038, China. .
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Źródło:
Journal of nanobiotechnology [J Nanobiotechnology] 2021 Jul 22; Vol. 19 (1), pp. 220. Date of Electronic Publication: 2021 Jul 22.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Apoptosis/*drug effects
Autophagy/*drug effects
Nanostructures/*therapeutic use
Neoplasms/*therapy
Photothermal Therapy/*methods
Polymers/*chemistry
Animals ; Antineoplastic Agents/chemistry ; Antineoplastic Agents/pharmacology ; Antineoplastic Agents/therapeutic use ; Biocompatible Materials/chemistry ; Biocompatible Materials/therapeutic use ; Humans ; Nanomedicine ; Nanostructures/chemistry
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Oridonin Promotes Apoptosis and Restrains the Viability and Migration of Bladder Cancer by Impeding TRPM7 Expression via the ERK and AKT Signaling Pathways.
Autorzy:
Che X; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
Zhan J; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
Zhao F; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
Zhong Z; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
Chen M; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
Han R; Department of Urology, The Second Hospital of Tianjin Medical University, Tianjin Institute of Urology, 300211 Tianjin, China.
Wang Y; Department of Urology, The Second Affiliated Hospital of Hainan Medical University, 570311 Hainan, China.
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Źródło:
BioMed research international [Biomed Res Int] 2021 Jul 05; Vol. 2021, pp. 4340950. Date of Electronic Publication: 2021 Jul 05 (Print Publication: 2021).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*/drug effects
Apoptosis*/genetics
Cell Movement*/drug effects
Cell Movement*/genetics
MAP Kinase Signaling System*/drug effects
MAP Kinase Signaling System*/genetics
Diterpenes, Kaurane/*pharmacology
Protein Serine-Threonine Kinases/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
TRPM Cation Channels/*metabolism
Urinary Bladder Neoplasms/*pathology
Animals ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Proliferation/genetics ; Cell Survival/drug effects ; Cell Survival/genetics ; Female ; Humans ; Mice, Inbred C57BL ; Protein Serine-Threonine Kinases/genetics ; TRPM Cation Channels/genetics ; Tumor Stem Cell Assay ; Urinary Bladder Neoplasms/genetics ; Xenograft Model Antitumor Assays ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Tanshinones induce tumor cell apoptosis via directly targeting FHIT.
Autorzy:
Zhou X; Center for Chemical Biology, Institute of Interdisciplinary Integrative Medicine Research, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Pan Y; Center for Chemical Biology, Institute of Interdisciplinary Integrative Medicine Research, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Wang Y; Center for Chemical Biology, Institute of Interdisciplinary Integrative Medicine Research, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Wang B; Center for Chemical Biology, Institute of Interdisciplinary Integrative Medicine Research, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Yan Y; Shanghai Innovation Center of TCM Health Service, Shanghai University of Traditional Chinese Medicine, Shanghai, China.
Qu Y; Center for Chemical Biology, Institute of Interdisciplinary Integrative Medicine Research, Shanghai University of Traditional Chinese Medicine, Shanghai, China. .
Ke X; Center for Chemical Biology, Institute of Interdisciplinary Integrative Medicine Research, Shanghai University of Traditional Chinese Medicine, Shanghai, China. .
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Źródło:
Scientific reports [Sci Rep] 2021 Jun 09; Vol. 11 (1), pp. 12217. Date of Electronic Publication: 2021 Jun 09.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Abietanes/*pharmacology
Acid Anhydride Hydrolases/*antagonists & inhibitors
Colorectal Neoplasms/*drug therapy
Hydrolases/*antagonists & inhibitors
Neoplasm Proteins/*antagonists & inhibitors
Salvia miltiorrhiza/*chemistry
Abietanes/chemistry ; Colorectal Neoplasms/metabolism ; Colorectal Neoplasms/pathology ; Humans ; Tumor Cells, Cultured
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Sacubitril/valsartan inhibits ox‑LDL‑induced MALAT1 expression, inflammation and apoptosis by suppressing the TLR4/NF‑κB signaling pathway in HUVECs.
Autorzy:
Bai W; Department of Internal Medicine, Hebei Medical University, Shijiazhuang, Hebei 050017, P.R. China.
Huo T; Department of Neurology, Hebei General Hospital, Shijiazhuang, Hebei 050051, P.R. China.
Chen X; Department of Cardiology, Hebei General Hospital, Shijiazhuang, Hebei 050051, P.R. China.
Song X; Department of Cardiology, Hebei General Hospital, Shijiazhuang, Hebei 050051, P.R. China.
Meng C; Department of Cardiology, Hebei General Hospital, Shijiazhuang, Hebei 050051, P.R. China.
Dang Y; Department of Cardiology, Hebei General Hospital, Shijiazhuang, Hebei 050051, P.R. China.
Rong C; Department of Cardiology, Hebei General Hospital, Shijiazhuang, Hebei 050051, P.R. China.
Dou L; Department of Cardiology, Hebei General Hospital, Shijiazhuang, Hebei 050051, P.R. China.
Qi X; Department of Cardiology, Hebei General Hospital, Shijiazhuang, Hebei 050051, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2021 Jun; Vol. 23 (6). Date of Electronic Publication: 2021 Mar 31.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Aminobutyrates/*pharmacology
Anti-Inflammatory Agents/*pharmacology
Human Umbilical Vein Endothelial Cells/*drug effects
RNA, Long Noncoding/*genetics
Tetrazoles/*pharmacology
Valsartan/*pharmacology
Biphenyl Compounds ; Drug Combinations ; Human Umbilical Vein Endothelial Cells/metabolism ; Humans ; Lipoproteins, LDL/pharmacology ; NF-kappa B/metabolism ; RNA, Long Noncoding/metabolism ; Signal Transduction ; Toll-Like Receptor 4/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Inhibition of MAGL activates the Keap1/Nrf2 pathway to attenuate glucocorticoid-induced osteonecrosis of the femoral head.
Autorzy:
Yang N; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
Sun H; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
Xue Y; Department of Orthopaedics, Changshu Hospital Affiliated to Nanjing University of Traditional Chinese Medicine, Changshu, China.
Zhang W; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
Wang H; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
Tao H; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
Liang X; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
Li M; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
Xu Y; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
Chen L; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
Zhang L; Department of Orthopaedics, Beijing Friendship Hospital, Capital Medical University, Beijing, China.
Huang L; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
Geng D; Department of Orthopaedics, The First Affiliated Hospital of Soochow University, Soochow University, Suzhou, China.
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Źródło:
Clinical and translational medicine [Clin Transl Med] 2021 Jun; Vol. 11 (6), pp. e447.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Femur Head/*drug effects
Glucocorticoids/*toxicity
Kelch-Like ECH-Associated Protein 1/*metabolism
Monoacylglycerol Lipases/*antagonists & inhibitors
NF-E2-Related Factor 2/*metabolism
Osteonecrosis/*prevention & control
Animals ; Cell Differentiation ; Femur Head/pathology ; Kelch-Like ECH-Associated Protein 1/genetics ; Male ; Mesenchymal Stem Cells/drug effects ; Mesenchymal Stem Cells/metabolism ; Mesenchymal Stem Cells/pathology ; NF-E2-Related Factor 2/genetics ; Osteoblasts/drug effects ; Osteoblasts/metabolism ; Osteoblasts/pathology ; Osteonecrosis/chemically induced ; Osteonecrosis/metabolism ; Osteonecrosis/pathology ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Cediranib Induces Apoptosis, G1 Phase Cell Cycle Arrest, and Autophagy in Non-Small-Cell Lung Cancer Cell A549 In Vitro.
Autorzy:
Guo M; School of Pharmacy, Lanzhou University, Lanzhou, Gansu 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou, Guangdong 516029, China.
Liu Z; Gansu Key Laboratory of Environmental Friendly Composites and Biomass Utilization, College of Chemical Engineering, Northwest Minzu University, Lanzhou, Gansu 730030, China.
Si J; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou, Guangdong 516029, China.; Institute of Modern Physics, Chinese Academy of Sciences, Lanzhou 730000, China.
Zhang J; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou, Guangdong 516029, China.; Institute of Modern Physics, Chinese Academy of Sciences, Lanzhou 730000, China.
Zhao J; Medical College of Northwest Minzu University, Lanzhou, Gansu 730030, China.
Guo Z; Medical College of Northwest Minzu University, Lanzhou, Gansu 730030, China.
Xie Y; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou, Guangdong 516029, China.; Institute of Modern Physics, Chinese Academy of Sciences, Lanzhou 730000, China.
Zhang H; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou, Guangdong 516029, China.; Institute of Modern Physics, Chinese Academy of Sciences, Lanzhou 730000, China.
Gan L; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou, Guangdong 516029, China.; Institute of Modern Physics, Chinese Academy of Sciences, Lanzhou 730000, China.
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Źródło:
BioMed research international [Biomed Res Int] 2021 Mar 29; Vol. 2021, pp. 5582648. Date of Electronic Publication: 2021 Mar 29 (Print Publication: 2021).
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Autophagy/*drug effects
Carcinoma, Non-Small-Cell Lung/*pathology
G1 Phase Cell Cycle Checkpoints/*drug effects
Lung Neoplasms/*pathology
Quinazolines/*pharmacology
A549 Cells ; Carcinoma, Non-Small-Cell Lung/metabolism ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Humans ; Lung Neoplasms/metabolism ; MAP Kinase Signaling System/drug effects ; Proto-Oncogene Proteins c-akt/metabolism ; Quinazolines/chemistry ; Receptors, Vascular Endothelial Growth Factor/metabolism ; TOR Serine-Threonine Kinases/metabolism ; Tumor Stem Cell Assay
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
miR-140-5p regulates vascular smooth muscle cell viability, migration and apoptosis by targeting ROBO4 gene expression in atherosclerosis.
Autorzy:
Luo Y; Department of Vascular Surgery, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400010, P.R. China.
Li Y; Department of Gynecology, Jialing Branch of Nanchong Central Hospital, Nanchong, Sichuan 637919, P.R. China.
Peng H; Department of Anorectal, Nanchong Central Hospital, Nanchong, Sichuan 637000, P.R. China.
Zhao Y; Department of Vascular Surgery, The First Affiliated Hospital of Chongqing Medical University, Chongqing 400010, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2021 Mar; Vol. 23 (3). Date of Electronic Publication: 2021 Jan 26.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Cell Movement*
Gene Expression Regulation*
Atherosclerosis/*metabolism
MicroRNAs/*metabolism
Muscle, Smooth, Vascular/*metabolism
Myocytes, Smooth Muscle/*metabolism
Receptors, Cell Surface/*biosynthesis
Aged ; Atherosclerosis/genetics ; Atherosclerosis/pathology ; Cell Survival ; Cells, Cultured ; Female ; Humans ; Male ; MicroRNAs/genetics ; Middle Aged ; Muscle, Smooth, Vascular/pathology ; Myocytes, Smooth Muscle/pathology ; Receptors, Cell Surface/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Synergistic killing effect of paclitaxel and honokiol in non-small cell lung cancer cells through paraptosis induction.
Autorzy:
Li XQ; Department of Medical Oncology, Affiliated Hospital of Jiangsu University, Zhenjiang, 212001, Jiangsu, China.
Ren J; Department of Medical Oncology, Affiliated Hospital of Jiangsu University, Zhenjiang, 212001, Jiangsu, China.
Wang Y; Center of Experimental Medicine, Affiliated Hospital of Jiangsu University, Zhenjiang, 212001, Jiangsu, China.
Su JY; Department of Pulmonary Medicine, Affiliated Hospital of Jiangsu University, Zhenjiang, 212001, Jiangsu, China.
Zhu YM; Department of Pulmonary Medicine, Affiliated Hospital of Jiangsu University, Zhenjiang, 212001, Jiangsu, China.
Chen CG; Department of Pulmonary Medicine, Affiliated Hospital of Jiangsu University, Zhenjiang, 212001, Jiangsu, China.
Long WG; Department of Pathology, Affiliated Hospital of Jiangsu University, Zhenjiang, 212001, Jiangsu, China.
Jiang Q; Center of Experimental Medicine, Affiliated Hospital of Jiangsu University, Zhenjiang, 212001, Jiangsu, China.
Li J; Department of Pulmonary Medicine, Affiliated Hospital of Jiangsu University, Zhenjiang, 212001, Jiangsu, China. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2021 Feb; Vol. 44 (1), pp. 135-150. Date of Electronic Publication: 2020 Sep 16.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*/drug effects
Biphenyl Compounds/*pharmacology
Carcinoma, Non-Small-Cell Lung/*pathology
Lignans/*pharmacology
Lung Neoplasms/*pathology
Paclitaxel/*pharmacology
Animals ; Calcium/metabolism ; Carcinoma, Non-Small-Cell Lung/ultrastructure ; Cell Line, Tumor ; Drug Synergism ; Endoplasmic Reticulum Stress/drug effects ; Humans ; Lung Neoplasms/ultrastructure ; Male ; Mice, Inbred BALB C ; Mice, Nude ; Microtubule-Associated Proteins/metabolism ; Mitochondria/drug effects ; Mitochondria/metabolism ; Proteasome Endopeptidase Complex/metabolism ; Ubiquitin-Activating Enzymes/metabolism ; Vacuoles/drug effects ; Vacuoles/metabolism ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Alantolactone induces apoptosis in THP-1 cells through STAT3, survivin inhibition, and intrinsic apoptosis pathway.
Autorzy:
Ahmad B; College of Basic Medical Sciences, Dalian Medical University, Dalian, China.
Gamallat Y; College of Basic Medical Sciences, Dalian Medical University, Dalian, China.; Guangzhou Institute of Pediatrics, Guangzhou Women and Childrens Medical Center, Guangzhou, China.
Su P; College of Basic Medical Sciences, Dalian Medical University, Dalian, China.
Husain A; College of Basic Medical Sciences, Dalian Medical University, Dalian, China.
Rehman AU; College of Basic Medical Sciences, Dalian Medical University, Dalian, China.
Zaky MY; College of Basic Medical Sciences, Dalian Medical University, Dalian, China.
Bakheet AMH; The Third Affiliated Hospital of Sun-yat Sen University, Guangzhou, China.
Tahir N; College of Basic Medical Sciences, Dalian Medical University, Dalian, China.
Xin Y; College of Basic Medical Sciences, Dalian Medical University, Dalian, China.
Liang W; Stem Cell Clinical Research Center, National Joint Engineering Laboratory, Regenerative Medicine Center, The First Affiliated Hospital of Dalian Medical, Dalian City, China.
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Źródło:
Chemical biology & drug design [Chem Biol Drug Des] 2021 Feb; Vol. 97 (2), pp. 266-272. Date of Electronic Publication: 2020 Sep 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Lactones/*pharmacology
STAT3 Transcription Factor/*metabolism
Sesquiterpenes, Eudesmane/*pharmacology
Signal Transduction/*drug effects
Survivin/*metabolism
Cell Survival/drug effects ; Humans ; Inula/chemistry ; Inula/metabolism ; Mitochondria/drug effects ; Mitochondria/metabolism ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; STAT3 Transcription Factor/antagonists & inhibitors ; Survivin/antagonists & inhibitors ; THP-1 Cells ; bcl-2-Associated X Protein/genetics ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The hard clam genome reveals massive expansion and diversification of inhibitors of apoptosis in Bivalvia.
Autorzy:
Song H; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; Center for Ocean Mega-Science, Chinese Academy of Sciences, Qingdao, 266071, China.; CAS Engineering Laboratory for Marine Ranching, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.
Guo X; Haskin Shellfish Research Laboratory, Department of Marine and Coastal Sciences, Rutgers University, Port Norris, NJ, USA.
Sun L; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; Center for Ocean Mega-Science, Chinese Academy of Sciences, Qingdao, 266071, China.; CAS Engineering Laboratory for Marine Ranching, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; University of the Chinese Academy of Sciences, Beijing, 100049, China.
Wang Q; Novogene Bioinformatics Institute, Beijing, 100029, China.
Han F; Novogene Bioinformatics Institute, Beijing, 100029, China.
Wang H; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; Center for Ocean Mega-Science, Chinese Academy of Sciences, Qingdao, 266071, China.; University of the Chinese Academy of Sciences, Beijing, 100049, China.
Wray GA; Duke University, 130 Science Dr, Durham, NC, 27708, USA.
Davidson P; Duke University, 130 Science Dr, Durham, NC, 27708, USA.
Wang Q; University of the Chinese Academy of Sciences, Beijing, 100049, China.; Research and Development Center for Efficient Utilization of Coastal Bioresources, Yantai Institute of Coastal Zone Research, Chinese Academy of Sciences, Yantai, 264003, China.
Hu Z; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; University of the Chinese Academy of Sciences, Beijing, 100049, China.
Zhou C; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; University of the Chinese Academy of Sciences, Beijing, 100049, China.
Yu Z; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; Center for Ocean Mega-Science, Chinese Academy of Sciences, Qingdao, 266071, China.; CAS Engineering Laboratory for Marine Ranching, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.
Yang M; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; University of the Chinese Academy of Sciences, Beijing, 100049, China.
Feng J; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; Center for Ocean Mega-Science, Chinese Academy of Sciences, Qingdao, 266071, China.; CAS Engineering Laboratory for Marine Ranching, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.
Shi P; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; University of the Chinese Academy of Sciences, Beijing, 100049, China.
Zhou Y; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; Center for Ocean Mega-Science, Chinese Academy of Sciences, Qingdao, 266071, China.; CAS Engineering Laboratory for Marine Ranching, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; University of the Chinese Academy of Sciences, Beijing, 100049, China.
Zhang L; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China.; Center for Ocean Mega-Science, Chinese Academy of Sciences, Qingdao, 266071, China.; CAS Engineering Laboratory for Marine Ranching, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China.; University of the Chinese Academy of Sciences, Beijing, 100049, China.
Zhang T; CAS Key Laboratory of Marine Ecology and Environmental Sciences, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China. .; Laboratory for Marine Ecology and Environmental Science, Qingdao National Laboratory for Marine Science and Technology, Qingdao, 266237, China. .; Center for Ocean Mega-Science, Chinese Academy of Sciences, Qingdao, 266071, China. .; CAS Engineering Laboratory for Marine Ranching, Institute of Oceanology, Chinese Academy of Sciences, Qingdao, 266071, China. .; University of the Chinese Academy of Sciences, Beijing, 100049, China. .
Pokaż więcej
Źródło:
BMC biology [BMC Biol] 2021 Jan 25; Vol. 19 (1), pp. 15. Date of Electronic Publication: 2021 Jan 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Genome*
Apoptosis/*genetics
Inhibitor of Apoptosis Proteins/*genetics
Mercenaria/*physiology
Animals ; Inhibitor of Apoptosis Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Crosstalk of the Caspase Family and Mammalian Target of Rapamycin Signaling.
Autorzy:
Yan J; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.; College of Life Sciences, University of Chinese Academy of Sciences, Beijing 101408, China.
Xie Y; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.
Si J; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.
Gan L; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.
Li H; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.
Sun C; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.
Di C; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.
Zhang J; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.; College of Life Sciences, University of Chinese Academy of Sciences, Beijing 101408, China.
Huang G; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.; College of Life Sciences, University of Chinese Academy of Sciences, Beijing 101408, China.
Zhang X; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.; College of Life Sciences, University of Chinese Academy of Sciences, Beijing 101408, China.
Zhang H; Institute of Modern Physics, Chinese Academy of Sciences, 509 Nanchang Road, Lanzhou 730000, China.; Advanced Energy Science and Technology Guangdong Laboratory, Huizhou 516029, China.; Key Laboratory of Heavy Ion Radiation Biology and Medicine of Chinese Academy of Sciences, Lanzhou 730000, China.; Key Laboratory of Heavy Ion Radiation Medicine of Gansu Province, Lanzhou 730000, China.
Pokaż więcej
Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Jan 15; Vol. 22 (2). Date of Electronic Publication: 2021 Jan 15.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Apoptosis/*physiology
Caspases/*metabolism
Cell Proliferation/*physiology
Signal Transduction/*physiology
TOR Serine-Threonine Kinases/*metabolism
Animals ; Endoplasmic Reticulum/metabolism ; Endoplasmic Reticulum Stress/physiology ; Humans ; Inflammation/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Effects of the E1 activating enzyme UBA2 on porcine oocyte maturation, apoptosis, and embryonic development in vitro.
Autorzy:
Xu D; Department of Animal Science, College of Agriculture, Yanbian University, Yanji, China.; Jilin Medical University, Jilin, China.
Bi J; College of Life Sciences, Xiamen University, Fujian, China.
Guan Y; College of Life Sciences, Xiamen University, Fujian, China.
Luo X; Department of Animal Science, College of Agriculture, Yanbian University, Yanji, China.
Chen X; Department of Animal Science, College of Agriculture, Yanbian University, Yanji, China.
Lv Y; Department of Animal Science, College of Agriculture, Yanbian University, Yanji, China.
Jin Y; Department of Animal Science, College of Agriculture, Yanbian University, Yanji, China.
Pokaż więcej
Źródło:
Animal science journal = Nihon chikusan Gakkaiho [Anim Sci J] 2021 Jan-Dec; Vol. 92 (1), pp. e13548.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Apoptosis/*genetics
Embryonic Development/*drug effects
Embryonic Development/*genetics
Gene Expression/*genetics
Gene Expression Regulation, Developmental/*drug effects
Oocytes/*growth & development
Oogenesis/*drug effects
Oogenesis/*genetics
SUMO-1 Protein/*genetics
SUMO-1 Protein/*metabolism
Ubiquitin-Activating Enzymes/*pharmacology
Animals ; Cell Survival/drug effects ; Cell Survival/genetics ; Embryo Culture Techniques/veterinary ; Female ; Fertilization in Vitro ; In Vitro Oocyte Maturation Techniques/veterinary ; Pregnancy ; Swine
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
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