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Tytuł :
[The NOXA promoter could function as an active enhancer to regulate the expression of BCL2 in the apoptosis response].
Autorzy :
Qin ZY; Key Laboratory of Human Functional Genomics of Jiangsu Province, Nanjing Medical University, Nanjing 211166, China.; Department of Cell Biology, Nanjing Medical University, Nanjing 211166, China.
Shi X; Key Laboratory of Human Functional Genomics of Jiangsu Province, Nanjing Medical University, Nanjing 211166, China.; Department of Cell Biology, Nanjing Medical University, Nanjing 211166, China.
Cao PP; Key Laboratory of Human Functional Genomics of Jiangsu Province, Nanjing Medical University, Nanjing 211166, China.; Department of Cell Biology, Nanjing Medical University, Nanjing 211166, China.
Chu Y; Key Laboratory of Human Functional Genomics of Jiangsu Province, Nanjing Medical University, Nanjing 211166, China.; Department of Cell Biology, Nanjing Medical University, Nanjing 211166, China.
Guan W; The First Affiliated Hospital of Nanjing Medical University, Nanjing 210029,China.
Yang N; Key Laboratory of Human Functional Genomics of Jiangsu Province, Nanjing Medical University, Nanjing 211166, China.; Department of Biochemistry and Molecular Biology, Nanjing Medical University, Nanjing 211166, China.
Cheng H; Key Laboratory of Human Functional Genomics of Jiangsu Province, Nanjing Medical University, Nanjing 211166, China.; The First Affiliated Hospital of Nanjing Medical University, Nanjing 210029,China.
Sun YJ; Key Laboratory of Human Functional Genomics of Jiangsu Province, Nanjing Medical University, Nanjing 211166, China.; Department of Cell Biology, Nanjing Medical University, Nanjing 211166, China.
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Źródło :
Yi chuan = Hereditas [Yi Chuan] 2020 Nov 20; Vol. 42 (11), pp. 1110-1121.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*/genetics
Gene Expression Regulation, Developmental*
Promoter Regions, Genetic*
Chromatin Immunoprecipitation ; Humans ; MCF-7 Cells ; Polymerase Chain Reaction ; Proto-Oncogene Proteins c-bcl-2/genetics
Czasopismo naukowe
Tytuł :
Calcitriol attenuates renal tubular epithelial cells apoptosis via inhibiting p38MAPK signaling in diabetic nephropathy.
Autorzy :
Guo Y; Department of Nephrology, The First Hospital of Jiaxing, First Affilated Hospital of Jiaxing Universty, Jiaxing, 314000, Zhejiang, China.
Xie X; Institute of Nephrology, School of Medicine, ZhongDa Hospital, Southeast University, Nanjing, 210009, Jiangsu, China.
Zhao Y; Institute of Nephrology, School of Medicine, ZhongDa Hospital, Southeast University, Nanjing, 210009, Jiangsu, China.
Zhou M; Department of Nephrology, The Affiliated Huaian NO.1 People's Hospital of Nanjing Medical Universty, Huaian, 223001, Jiangsu, China.
Yang Y; Institute of Nephrology, School of Medicine, ZhongDa Hospital, Southeast University, Nanjing, 210009, Jiangsu, China.
Zhang X; Institute of Nephrology, School of Medicine, ZhongDa Hospital, Southeast University, Nanjing, 210009, Jiangsu, China. .
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Źródło :
Acta diabetologica [Acta Diabetol] 2020 Nov; Vol. 57 (11), pp. 1327-1335. Date of Electronic Publication: 2020 Jun 18.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Calcitriol/*administration & dosage
Diabetic Nephropathies/*drug therapy
Epithelial Cells/*drug effects
Kidney Tubules/*drug effects
Animals ; Diabetic Nephropathies/metabolism ; Diabetic Nephropathies/physiopathology ; Epithelial Cells/cytology ; Epithelial Cells/metabolism ; Humans ; Kidney Tubules/cytology ; Kidney Tubules/metabolism ; MAP Kinase Signaling System/drug effects ; Male ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł :
Role of Bm30kc6 gene in cell apoptosis and the silk gland degradation signaling pathway in Bombyx mori L.
Autorzy :
Xiao Y; College of Animal Sciences, Zhejiang University, Hangzhou, China.
Li LL; College of Animal Sciences, Zhejiang University, Hangzhou, China.
Bibi A; College of Animal Sciences, Zhejiang University, Hangzhou, China.
Zhang N; Zhejiang Agriculture & Forestry University, Hangzhou, China.
Chen T; College of Animal Sciences, Zhejiang University, Hangzhou, China.
Mo Y; College of Animal Sciences, Zhejiang University, Hangzhou, China.
Yue W; Zhejiang Agriculture & Forestry University, Hangzhou, China.
Miao Y; College of Animal Sciences, Zhejiang University, Hangzhou, China.
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Źródło :
Archives of insect biochemistry and physiology [Arch Insect Biochem Physiol] 2020 Nov; Vol. 105 (3), pp. e21741. Date of Electronic Publication: 2020 Oct 01.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*genetics
Bombyx/*genetics
Bombyx/*growth & development
Animals ; Bombyx/metabolism ; Caspase 3/genetics ; Caspase 3/metabolism ; Caspase Inhibitors/pharmacology ; Dactinomycin/pharmacology ; Ecdysone/pharmacology ; Gene Expression Regulation, Developmental ; Hemolymph/metabolism ; Insect Proteins/genetics ; Insect Proteins/metabolism ; RNA, Small Interfering ; Signal Transduction ; Ultraviolet Rays
Czasopismo naukowe
Tytuł :
Modulating TRADD to restore cellular homeostasis and inhibit apoptosis.
Autorzy :
Xu D; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.; Interdisciplinary Research Center on Biology and Chemistry, Shanghai Institute of Organic Chemistry, Chinese Academy of Sciences, Shanghai, China.
Zhao H; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Jin M; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Zhu H; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Shan B; Interdisciplinary Research Center on Biology and Chemistry, Shanghai Institute of Organic Chemistry, Chinese Academy of Sciences, Shanghai, China.
Geng J; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Dziedzic SA; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Amin P; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Mifflin L; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Naito MG; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Najafov A; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Xing J; Department of Pediatrics and Human Development, Michigan State University, Grand Rapids, MI, USA.
Yan L; Interdisciplinary Research Center on Biology and Chemistry, Shanghai Institute of Organic Chemistry, Chinese Academy of Sciences, Shanghai, China.
Liu J; State Key Laboratory of Bio-organic and Natural Products Chemistry, Shanghai Institute of Organic Chemistry, Chinese Academy of Sciences, Shanghai, China.
Qin Y; Interdisciplinary Research Center on Biology and Chemistry, Shanghai Institute of Organic Chemistry, Chinese Academy of Sciences, Shanghai, China.
Hu X; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Wang H; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Zhang M; Interdisciplinary Research Center on Biology and Chemistry, Shanghai Institute of Organic Chemistry, Chinese Academy of Sciences, Shanghai, China.
Manuel VJ; Department of Cell Biology, Harvard Medical School, Boston, MA, USA.
Tan L; Interdisciplinary Research Center on Biology and Chemistry, Shanghai Institute of Organic Chemistry, Chinese Academy of Sciences, Shanghai, China.
He Z; Interdisciplinary Research Center on Biology and Chemistry, Shanghai Institute of Organic Chemistry, Chinese Academy of Sciences, Shanghai, China.; Center for Neurodegenerative Disease Research, Institute on Aging, Department of Pathology and Laboratory, University of Pennsylvania School of Medicine, Philadelphia, PA, USA.
Sun ZJ; Department of Cancer Biology, Dana Farber Cancer Institute, Boston, MA, USA.
Lee VMY; Center for Neurodegenerative Disease Research, Institute on Aging, Department of Pathology and Laboratory, University of Pennsylvania School of Medicine, Philadelphia, PA, USA.
Wagner G; Department of Biochemistry and Molecular Pharmacology, Harvard Medical School, Boston, MA, USA.
Yuan J; Department of Cell Biology, Harvard Medical School, Boston, MA, USA. .
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Źródło :
Nature [Nature] 2020 Nov; Vol. 587 (7832), pp. 133-138. Date of Electronic Publication: 2020 Sep 23.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Homeostasis/*drug effects
TNF Receptor-Associated Death Domain Protein/*antagonists & inhibitors
TNF Receptor-Associated Death Domain Protein/*metabolism
Animals ; Autophagy/drug effects ; Baculoviral IAP Repeat-Containing 3 Protein/metabolism ; Beclin-1/chemistry ; Beclin-1/metabolism ; Bortezomib/antagonists & inhibitors ; Bortezomib/pharmacology ; Cell Line ; Humans ; Huntingtin Protein/metabolism ; Inhibitor of Apoptosis Proteins/metabolism ; Male ; Mice ; Models, Molecular ; Neurofibrillary Tangles/metabolism ; Proteome/metabolism ; Receptor-Interacting Protein Serine-Threonine Kinases/chemistry ; Receptor-Interacting Protein Serine-Threonine Kinases/metabolism ; TNF Receptor-Associated Death Domain Protein/chemistry ; TNF Receptor-Associated Death Domain Protein/deficiency ; TNF Receptor-Associated Factor 2/metabolism ; Ubiquitination ; alpha-Synuclein/metabolism ; tau Proteins/metabolism
Czasopismo naukowe
Tytuł :
Tβ4 suppresses lincRNA-p21-mediated hepatic apoptosis and fibrosis by inhibiting PI3K-AKT-NF-κB pathway.
Autorzy :
Yang L; Department of Infectious Disease, The First Affiliated Hospital of Dalian Medical University, Dalian, Liaoning 116011, China.
Fu WL; Department of Hepatobiliary Surgery, Municipal Friendship Hospital, Dalian, Liaoning 116001, China.
Zhu Y; Department of Infectious Disease, The First Affiliated Hospital of Dalian Medical University, Dalian, Liaoning 116011, China.
Wang XG; Department of Hepatobiliary Surgery, Municipal Friendship Hospital, Dalian, Liaoning 116001, China. Electronic address: .
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Źródło :
Gene [Gene] 2020 Oct 20; Vol. 758, pp. 144946. Date of Electronic Publication: 2020 Jul 07.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Liver/*injuries
Liver Cirrhosis/*prevention & control
RNA, Long Noncoding/*genetics
Thymosin/*pharmacology
Actins/metabolism ; Animals ; Cell Proliferation/drug effects ; Collagen/metabolism ; Hepatocytes/pathology ; I-kappa B Proteins/antagonists & inhibitors ; Liver/metabolism ; Liver Cirrhosis/drug therapy ; Mice ; Mice, Inbred C57BL ; Phosphatidylinositol 3-Kinases/metabolism ; Phosphoinositide-3 Kinase Inhibitors/pharmacology ; Proto-Oncogene Proteins c-akt/antagonists & inhibitors ; Tissue Inhibitor of Metalloproteinase-1/metabolism
Czasopismo naukowe
Tytuł :
Reactivating latent HIV with PKC agonists induces resistance to apoptosis and is associated with phosphorylation and activation of BCL2.
Autorzy :
French AJ; Division of Infectious Diseases, Mayo Clinic, Rochester, Minnesota, United States of America.
Natesampillai S; Division of Infectious Diseases, Mayo Clinic, Rochester, Minnesota, United States of America.
Krogman A; Division of Infectious Diseases, Mayo Clinic, Rochester, Minnesota, United States of America.
Correia C; Division of Oncology Research, Mayo Clinic, Rochester, Minnesota, United States of America.
Peterson KL; Division of Oncology Research, Mayo Clinic, Rochester, Minnesota, United States of America.
Alto A; Division of Infectious Diseases, Mayo Clinic, Rochester, Minnesota, United States of America.
Chandrasekar AP; Division of Infectious Diseases, Mayo Clinic, Rochester, Minnesota, United States of America.
Misra A; Division of Infectious Diseases, Mayo Clinic, Rochester, Minnesota, United States of America.
Li Y; Division of Biomedical Statistics and Informatics, Mayo Clinic, Rochester, Minnesota, United States of America.
Kaufmann SH; Division of Oncology Research, Mayo Clinic, Rochester, Minnesota, United States of America.; Department of Molecular Pharmacology & Experimental Therapeutics, Mayo Clinic, Rochester, Minnesota, United States of America.
Badley AD; Division of Infectious Diseases, Mayo Clinic, Rochester, Minnesota, United States of America.; Department of Molecular Medicine, Mayo Clinic, Rochester, Minnesota, United States of America.
Cummins NW; Division of Infectious Diseases, Mayo Clinic, Rochester, Minnesota, United States of America.
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Źródło :
PLoS pathogens [PLoS Pathog] 2020 Oct 19; Vol. 16 (10), pp. e1008906. Date of Electronic Publication: 2020 Oct 19 (Print Publication: 2020).
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
HIV Infections/*virology
HIV-1/*pathogenicity
Protein Kinase C/*chemistry
Virus Latency/*drug effects
CD4-Positive T-Lymphocytes/virology ; HIV Infections/drug therapy ; Humans ; Phosphorylation ; Protein Kinase C/metabolism ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Virus Activation/drug effects ; bcl-Associated Death Protein/metabolism
Czasopismo naukowe
Tytuł :
Total Flavonoids Isolated from Diospyros kaki L. f. Leaves Induced Apoptosis and Oxidative Stress in Human Cancer Cells.
Autorzy :
Chen LI; International Ginseng Institute, School of Agriculture, Middle Tennessee State University, Murfreesboro, TN, U.S.A.; Department of Pharmacology, College of Medicine, Guangxi University of Science and Technology, Liuzhou, P.R. China.
Guo Y; International Ginseng Institute, School of Agriculture, Middle Tennessee State University, Murfreesboro, TN, U.S.A.; Faculty of International Education, Guangxi University of Chinese Medicine, Nanning, P.R. China.
Alsaif G; Department of Biology, Middle Tennessee State University, Murfreesboro, TN, U.S.A.
Gao Y; International Ginseng Institute, School of Agriculture, Middle Tennessee State University, Murfreesboro, TN, U.S.A. ying..
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Źródło :
Anticancer research [Anticancer Res] 2020 Sep; Vol. 40 (9), pp. 5201-5210.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents, Phytogenic/*pharmacology
Apoptosis/*drug effects
Diospyros/*chemistry
Flavonoids/*pharmacology
Oxidative Stress/*drug effects
Plant Extracts/*pharmacology
Plant Leaves/*chemistry
Antineoplastic Agents, Phytogenic/chemistry ; Antineoplastic Agents, Phytogenic/isolation & purification ; Antioxidants/chemistry ; Antioxidants/pharmacology ; Cell Line, Tumor ; Dose-Response Relationship, Drug ; Flavonoids/chemistry ; Flavonoids/isolation & purification ; Humans ; Plant Extracts/chemistry ; Plant Extracts/isolation & purification ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł :
Delayed recanalization after MCAO ameliorates ischemic stroke by inhibiting apoptosis via HGF/c-Met/STAT3/Bcl-2 pathway in rats.
Autorzy :
Tang H; Department of Neurosurgery, Affiliated Haikou Hospital, Xiangya Medical College of Central South University, Haikou, Hainan 570000, China; Department of Physiology and Pharmacology, Loma Linda University, Loma Linda, CA 92354, USA.
Gamdzyk M; Department of Physiology and Pharmacology, Loma Linda University, Loma Linda, CA 92354, USA.
Huang L; Department of Physiology and Pharmacology, Loma Linda University, Loma Linda, CA 92354, USA; Department of Neurosurgery, Loma Linda University, Loma Linda, CA 92354, USA.
Gao L; Department of Neurosurgery, Affiliated Haikou Hospital, Xiangya Medical College of Central South University, Haikou, Hainan 570000, China; Department of Physiology and Pharmacology, Loma Linda University, Loma Linda, CA 92354, USA.
Lenahan C; Department of Physiology and Pharmacology, Loma Linda University, Loma Linda, CA 92354, USA; Department of Neurosurgery, Loma Linda University, Loma Linda, CA 92354, USA.
Kang R; Department of Physiology and Pharmacology, Loma Linda University, Loma Linda, CA 92354, USA.
Tang J; Department of Physiology and Pharmacology, Loma Linda University, Loma Linda, CA 92354, USA.
Xia Y; Department of Neurosurgery, Affiliated Haikou Hospital, Xiangya Medical College of Central South University, Haikou, Hainan 570000, China. Electronic address: .
Zhang JH; Department of Physiology and Pharmacology, Loma Linda University, Loma Linda, CA 92354, USA; Department of Neurosurgery, Loma Linda University, Loma Linda, CA 92354, USA; Department of Anesthesiology, Loma Linda University Medical Center, Loma Linda, CA 92354, USA. Electronic address: .
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Źródło :
Experimental neurology [Exp Neurol] 2020 Aug; Vol. 330, pp. 113359. Date of Electronic Publication: 2020 May 16.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*physiology
Hepatocyte Growth Factor/*metabolism
Infarction, Middle Cerebral Artery/*metabolism
Ischemic Stroke/*metabolism
Proto-Oncogene Proteins c-met/*metabolism
Animals ; Infarction, Middle Cerebral Artery/pathology ; Ischemic Stroke/pathology ; Male ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Rats ; Rats, Sprague-Dawley ; STAT3 Transcription Factor/metabolism ; Signal Transduction/physiology
Czasopismo naukowe
Tytuł :
CisPG21 and CisCEL16 are involved in the regulation of the degradation of cell walls during secretory cavity cell programmed cell death in the fruits of Citrus sinensis (L.) Osbeck.
Autorzy :
Tong P; State Key Laboratory for Conservation and Utilization of Subtropical Agro-bioresources, College of Life Sciences, South China Agricultural University, Guangzhou, 510642, China. Electronic address: .
Huai B; State Key Laboratory for Conservation and Utilization of Subtropical Agro-bioresources, College of Life Sciences, South China Agricultural University, Guangzhou, 510642, China. Electronic address: .
Chen Y; Shaanxi University of Chinese Medicine, Xianyang, 712046, China. Electronic address: .
Bai M; State Key Laboratory for Conservation and Utilization of Subtropical Agro-bioresources, College of Life Sciences, South China Agricultural University, Guangzhou, 510642, China. Electronic address: .
Wu H; State Key Laboratory for Conservation and Utilization of Subtropical Agro-bioresources, College of Life Sciences, South China Agricultural University, Guangzhou, 510642, China; Guangdong Key Laboratory for Innovative Development and Utilization of Forest Plant Germplasm, College of Forestry, South China Agricultural University, Guangzhou, 510642, China. Electronic address: .
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Źródło :
Plant science : an international journal of experimental plant biology [Plant Sci] 2020 Aug; Vol. 297, pp. 110540. Date of Electronic Publication: 2020 May 30.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Cell Wall/*metabolism
Citrus sinensis/*genetics
Fruit/*metabolism
Genes, Plant/*physiology
Cell Wall/physiology ; Cellulase/metabolism ; Cellulose/metabolism ; Citrus sinensis/metabolism ; Citrus sinensis/physiology ; Fruit/physiology ; Genes, Plant/genetics ; In Situ Hybridization ; In Situ Nick-End Labeling ; Pectins/metabolism ; Polygalacturonase/metabolism ; Real-Time Polymerase Chain Reaction
Czasopismo naukowe
Tytuł :
miR-134-5p inhibition reduces infarct-induced cardiomyocyte apoptosis via Creb1 upregulation.
Autorzy :
Yang J; Department of Emergency Medicine, the First Affiliated Hospital of Nanchang University, No. 17, Yong Wai Zheng Street, Nanchang, China. Electronic address: .
Liu S; Department of Emergency Medicine, the First Affiliated Hospital of Nanchang University, No. 17, Yong Wai Zheng Street, Nanchang, China. Electronic address: .
Wang H; Department of Emergency Medicine, the First Affiliated Hospital of Nanchang University, No. 17, Yong Wai Zheng Street, Nanchang, China. Electronic address: .
Liu Y; Department of Emergency Medicine, the First Affiliated Hospital of Nanchang University, No. 17, Yong Wai Zheng Street, Nanchang, China. Electronic address: .
Liu Y; Department of Emergency Medicine, the First Affiliated Hospital of Nanchang University, No. 17, Yong Wai Zheng Street, Nanchang, China. Electronic address: .
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Źródło :
Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association [J Stroke Cerebrovasc Dis] 2020 Aug; Vol. 29 (8), pp. 104850. Date of Electronic Publication: 2020 May 29.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Antagomirs/*administration & dosage
Cyclic AMP Response Element-Binding Protein/*metabolism
MicroRNAs/*antagonists & inhibitors
Myocardial Infarction/*prevention & control
Myocytes, Cardiac/*metabolism
3' Untranslated Regions ; Animals ; Binding Sites ; Cell Hypoxia ; Cells, Cultured ; Cyclic AMP Response Element-Binding Protein/genetics ; Disease Models, Animal ; Extracellular Signal-Regulated MAP Kinases/metabolism ; Hydrogen Peroxide/toxicity ; JNK Mitogen-Activated Protein Kinases/metabolism ; MicroRNAs/genetics ; MicroRNAs/metabolism ; Myocardial Infarction/genetics ; Myocardial Infarction/metabolism ; Myocardial Infarction/pathology ; Myocytes, Cardiac/pathology ; Oxidative Stress ; Signal Transduction ; Up-Regulation
Czasopismo naukowe
Tytuł :
A near-infrared light-controlled, ultrasensitive one-step photoelectrochemical detection of dual cell apoptosis indicators in living cancer cells.
Autorzy :
Zheng YN; Chongqing Engineering Laboratory of Nanomaterials & Sensor Technologies, College of Chemistry and Chemical Engineering, Southwest University, Chongqing 400715, P. R. China. .
Xiong CY; Chongqing Engineering Laboratory of Nanomaterials & Sensor Technologies, College of Chemistry and Chemical Engineering, Southwest University, Chongqing 400715, P. R. China. .
Zhuo Y; Chongqing Engineering Laboratory of Nanomaterials & Sensor Technologies, College of Chemistry and Chemical Engineering, Southwest University, Chongqing 400715, P. R. China. .
Chai YQ; Chongqing Engineering Laboratory of Nanomaterials & Sensor Technologies, College of Chemistry and Chemical Engineering, Southwest University, Chongqing 400715, P. R. China. .
Liang WB; Chongqing Engineering Laboratory of Nanomaterials & Sensor Technologies, College of Chemistry and Chemical Engineering, Southwest University, Chongqing 400715, P. R. China. .
Yuan R; Chongqing Engineering Laboratory of Nanomaterials & Sensor Technologies, College of Chemistry and Chemical Engineering, Southwest University, Chongqing 400715, P. R. China. .
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Źródło :
Chemical communications (Cambridge, England) [Chem Commun (Camb)] 2020 Jul 28; Vol. 56 (60), pp. 8488-8491.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*/drug effects
Infrared Rays*
Electrochemical Techniques/*methods
Phosphatidylserines/*analysis
Sodium-Potassium-Exchanging ATPase/*analysis
Cell Line, Tumor ; Doxorubicin/pharmacology ; Humans ; Kinetics ; Methylene Blue/chemistry ; Microscopy, Confocal ; Quantum Dots/chemistry
Czasopismo naukowe
Tytuł :
Protective effect of CTRP6 on cerebral ischemia/reperfusion injury by attenuating inflammation, oxidative stress and apoptosis in PC12 cells.
Autorzy :
Li Y; Rehabilitation Centre, Beijing Xiaotangshan Hospital, Beijing 102211, P.R. China.
Sun J; Rehabilitation Centre, Beijing Xiaotangshan Hospital, Beijing 102211, P.R. China.
Gu L; Rehabilitation Centre, Beijing Xiaotangshan Hospital, Beijing 102211, P.R. China.
Gao X; Department of Neurology, General Hospital of The Yangtze River Shipping and Wuhan Brain Hospital, Wuhan, Hubei 430010, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2020 Jul; Vol. 22 (1), pp. 344-352. Date of Electronic Publication: 2020 May 04.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Oxidative Stress*
Adipokines/*metabolism
Brain Ischemia/*metabolism
Inflammation/*metabolism
Reperfusion Injury/*metabolism
Animals ; Brain Ischemia/pathology ; Cell Survival ; PC12 Cells ; Rats ; Reperfusion Injury/pathology ; Signal Transduction
Czasopismo naukowe
Tytuł :
Dequalinium chloride inhibits the growth of human glioma cells in vitro and vivo: a study on molecular mechanism and potential targeted agents.
Autorzy :
Yu Y; Department of Neurosurgery, The First Hospital of Jilin University, 71 Xinmin Avenue, Changchun, Jilin, 130021, China.
Yang B; Department of Hematology, The second Medical Center & National Clinical Research Center for Geriatric Diseases, Chinese PLA General Hospital, 28 Fuxing Road, Beijing, 100853, China.
Yu J; Department of Neurosurgery, The First Hospital of Jilin University, 71 Xinmin Avenue, Changchun, Jilin, 130021, China.
Zhao G; Department of Neurosurgery, The First Hospital of Jilin University, 71 Xinmin Avenue, Changchun, Jilin, 130021, China.
Chen F; Department of Neurosurgery, The First Hospital of Jilin University, 71 Xinmin Avenue, Changchun, Jilin, 130021, China. .
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Źródło :
Acta neurochirurgica [Acta Neurochir (Wien)] 2020 Jul; Vol. 162 (7), pp. 1683-1690. Date of Electronic Publication: 2020 May 14.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Brain Neoplasms/*metabolism
Cell Proliferation/*drug effects
Dequalinium/*pharmacology
Glioma/*metabolism
Brain Neoplasms/genetics ; Cell Line, Tumor ; Glioma/genetics ; Humans
Czasopismo naukowe
Tytuł :
A Gold(I) Complex Containing an Oleanolic Acid Derivative as a Potential Anti-Ovarian-Cancer Agent by Inhibiting TrxR and Activating ROS-Mediated ERS.
Autorzy :
Bian M; School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, 210023, P. R. China.
Sun Y; School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, 210023, P. R. China.
Liu Y; School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, 210023, P. R. China.
Xu Z; School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, 210023, P. R. China.
Fan R; School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, 210023, P. R. China.
Liu Z; School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, 210023, P. R. China.
Liu W; School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, 210023, P. R. China.; State Key Laboratory of Natural Medicines, China Pharmaceutical University, Nanjing, 210009, P. R. China.; State Key Laboratory of Coordination Chemistry, Nanjing University, Nanjing, 210023, P. R. China.
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Źródło :
Chemistry (Weinheim an der Bergstrasse, Germany) [Chemistry] 2020 Jun 02; Vol. 26 (31), pp. 7092-7108. Date of Electronic Publication: 2020 Mar 25.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents/*pharmacology
Antioxidants/*pharmacology
Apoptosis/*drug effects
Cell Survival/*drug effects
Coordination Complexes/*pharmacology
Gold/*chemistry
Oleanolic Acid/*chemistry
Ovarian Neoplasms/*drug therapy
Reactive Oxygen Species/*metabolism
Thioredoxin-Disulfide Reductase/*antagonists & inhibitors
Antineoplastic Agents/chemistry ; Cell Cycle Checkpoints/drug effects ; Cell Line, Tumor ; Coordination Complexes/chemistry ; Endoplasmic Reticulum Stress ; Female ; Gold/pharmacology ; Humans ; Oxidation-Reduction ; Thioredoxin-Disulfide Reductase/chemistry
Czasopismo naukowe
Tytuł :
Fast Activation and Tracing of Caspase-3 Involved Cell Apoptosis by Combined Electrostimulation and Smart Signal-Amplified SERS Nanoprobes.
Autorzy :
Qi G; State Key Laboratory of Electroanalytical Chemistry, Changchun Institute of Applied Chemistry, Chinese Academy of Sciences, Changchun 130022, Jilin P. R. China.; University of Chinese Academy of Sciences, Beijing 100049, P. R. China.
Sun D; State Key Laboratory of Supramolecular Structure and Materials, Jilin University, Changchun 130012, P. R. China.
Tian Y; State Key Laboratory of Supramolecular Structure and Materials, Jilin University, Changchun 130012, P. R. China.
Xu C; State Key Laboratory of Electroanalytical Chemistry, Changchun Institute of Applied Chemistry, Chinese Academy of Sciences, Changchun 130022, Jilin P. R. China.; University of Science and Technology of China, Hefei 230026, P. R. China.
Zhang Y; State Key Laboratory of Electroanalytical Chemistry, Changchun Institute of Applied Chemistry, Chinese Academy of Sciences, Changchun 130022, Jilin P. R. China.; University of Science and Technology of China, Hefei 230026, P. R. China.
Wang D; State Key Laboratory of Electroanalytical Chemistry, Changchun Institute of Applied Chemistry, Chinese Academy of Sciences, Changchun 130022, Jilin P. R. China.; University of Science and Technology of China, Hefei 230026, P. R. China.
Ma K; State Key Laboratory of Electroanalytical Chemistry, Changchun Institute of Applied Chemistry, Chinese Academy of Sciences, Changchun 130022, Jilin P. R. China.; University of Science and Technology of China, Hefei 230026, P. R. China.
Xu S; State Key Laboratory of Supramolecular Structure and Materials, Jilin University, Changchun 130012, P. R. China.
Jin Y; State Key Laboratory of Electroanalytical Chemistry, Changchun Institute of Applied Chemistry, Chinese Academy of Sciences, Changchun 130022, Jilin P. R. China.; University of Chinese Academy of Sciences, Beijing 100049, P. R. China.
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Źródło :
Analytical chemistry [Anal Chem] 2020 Jun 02; Vol. 92 (11), pp. 7861-7868. Date of Electronic Publication: 2020 May 22.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis*
Caspase 3/*metabolism
Gold/*chemistry
Metal Nanoparticles/*chemistry
Caspase 3/analysis ; Cell Line, Tumor ; Electric Stimulation Therapy ; Humans ; Spectrum Analysis, Raman
Czasopismo naukowe
Tytuł :
Ribophorin II is upregulated in myelodysplastic syndromes and prevents apoptosis and cell cycle progression.
Autorzy :
Ren J; Department of Hematology, Second Hospital of Hebei Medical University, Key Laboratory of Hematology of Hebei Province, Shijiazhuang, Hebei 050000, China.
Wang Y; Department of Hematology, Second Hospital of Hebei Medical University, Key Laboratory of Hematology of Hebei Province, Shijiazhuang, Hebei 050000, China.
Wang L; Department of Hematology, Second Hospital of Hebei Medical University, Key Laboratory of Hematology of Hebei Province, Shijiazhuang, Hebei 050000, China.
Guo X; Department of Hematology, Second Hospital of Hebei Medical University, Key Laboratory of Hematology of Hebei Province, Shijiazhuang, Hebei 050000, China.
Guo X; Department of Hematology, Second Hospital of Hebei Medical University, Key Laboratory of Hematology of Hebei Province, Shijiazhuang, Hebei 050000, China.
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Źródło :
Experimental biology and medicine (Maywood, N.J.) [Exp Biol Med (Maywood)] 2020 Jun; Vol. 245 (12), pp. 1009-1015. Date of Electronic Publication: 2020 May 25.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*genetics
Cell Cycle Checkpoints/*genetics
Hexosyltransferases/*genetics
Myelodysplastic Syndromes/*genetics
Proteasome Endopeptidase Complex/*genetics
Up-Regulation/*genetics
Cell Line, Tumor ; Cell Proliferation/genetics ; Core Binding Factor Alpha 1 Subunit/genetics ; Down-Regulation/genetics ; Enhancer of Zeste Homolog 2 Protein/genetics ; Gene Expression Regulation, Neoplastic/genetics ; Humans ; RNA, Messenger/genetics
Czasopismo naukowe
Tytuł :
Chronic Copper Exposure Induces Hypospermatogenesis in Mice by Increasing Apoptosis Without Affecting Testosterone Secretion.
Autorzy :
Chen H; College of Veterinary Medicine, South China Agricultural University, Guangzhou, 510642, China.
Kang Z; College of Veterinary Medicine, South China Agricultural University, Guangzhou, 510642, China.
Qiao N; College of Veterinary Medicine, South China Agricultural University, Guangzhou, 510642, China.
Liu G; College of Veterinary Medicine, South China Agricultural University, Guangzhou, 510642, China.
Huang K; College of Veterinary Medicine, South China Agricultural University, Guangzhou, 510642, China.
Wang X; College of Veterinary Medicine, South China Agricultural University, Guangzhou, 510642, China.
Pang C; College of Veterinary Medicine, South China Agricultural University, Guangzhou, 510642, China.
Zeng Q; College of Veterinary Medicine, South China Agricultural University, Guangzhou, 510642, China.
Tang Z; College of Veterinary Medicine, South China Agricultural University, Guangzhou, 510642, China. .
Li Y; College of Veterinary Medicine, South China Agricultural University, Guangzhou, 510642, China. .
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Źródło :
Biological trace element research [Biol Trace Elem Res] 2020 Jun; Vol. 195 (2), pp. 472-480. Date of Electronic Publication: 2019 Aug 23.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Copper Sulfate/*pharmacology
Spermatogenesis/*drug effects
Testis/*drug effects
Testosterone/*metabolism
Administration, Oral ; Animals ; Copper Sulfate/administration & dosage ; Copper Sulfate/analysis ; Male ; Mice ; Testis/metabolism ; Testis/pathology ; Testosterone/blood
Czasopismo naukowe
Tytuł :
Regulatory effects of Prohibitin 1 on proliferation and apoptosis of pulmonary arterial smooth muscle cells in monocrotaline-induced PAH rats.
Autorzy :
Cao YY; Department of Pharmacology, Xiangya School of Pharmaceutical Science, Central South University, Changsha 410013, China.
Ba HX; Department of Pharmacology, Xiangya School of Pharmaceutical Science, Central South University, Changsha 410013, China.
Li Y; Department of Health Management, The Third Xiangya Hospital, Central South University, Changsha 410013, China. Electronic address: .
Tang SY; Xiangya Nursing School, Central South University, Changsha 410013, China.
Luo ZQ; Department of Physiology, Xiangya School of Medicine, Central South University, Changsha 410013, China.
Li XH; Department of Pharmacology, Xiangya School of Pharmaceutical Science, Central South University, Changsha 410013, China; Hunan Key Laboratory for Bioanalysis of Complex Matrix Samples, Changsha 411000, China. Electronic address: .
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Źródło :
Life sciences [Life Sci] 2020 Jun 01; Vol. 250, pp. 117548. Date of Electronic Publication: 2020 Mar 12.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*
Hypertension, Pulmonary/*pathology
Monocrotaline/*pharmacology
Myocytes, Smooth Muscle/*cytology
Repressor Proteins/*metabolism
Animals ; Cell Proliferation ; Disease Models, Animal ; Gene Silencing ; Heart Ventricles/drug effects ; Hemodynamics ; Hypertension, Pulmonary/chemically induced ; Lung/metabolism ; Male ; Phosphatidylinositol 3-Kinases/metabolism ; Pulmonary Artery/cytology ; RNA, Small Interfering/metabolism ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł :
HIF‑1α affects trophoblastic apoptosis involved in the onset of preeclampsia by regulating FOXO3a under hypoxic conditions.
Autorzy :
Zhang Z; Department of Clinical Laboratory, The Third Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, P.R. China.
Huang C; Department of Clinical Laboratory, The Third Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, P.R. China.
Wang P; Department of Clinical Laboratory, The Third Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, P.R. China.
Gao J; Department of Clinical Laboratory, The Third Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, P.R. China.
Liu X; Department of Clinical Laboratory, The Third Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, P.R. China.
Li Y; Department of Clinical Laboratory, The Third Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, P.R. China.
Yan S; Department of Obstetrics and Gynecology, The Third Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, P.R. China.
Shi Y; Department of Clinical Laboratory, The Third Affiliated Hospital of Zhengzhou University, Zhengzhou, Henan 450052, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2020 Jun; Vol. 21 (6), pp. 2484-2492. Date of Electronic Publication: 2020 Apr 01.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*/drug effects
Cell Hypoxia*
Forkhead Box Protein O3/*metabolism
Hypoxia-Inducible Factor 1, alpha Subunit/*metabolism
Pre-Eclampsia/*pathology
Adult ; Case-Control Studies ; Cell Line ; Cobalt/pharmacology ; Female ; Forkhead Box Protein O3/antagonists & inhibitors ; Forkhead Box Protein O3/genetics ; Humans ; Hypoxia-Inducible Factor 1, alpha Subunit/antagonists & inhibitors ; Hypoxia-Inducible Factor 1, alpha Subunit/genetics ; Placenta/metabolism ; Pre-Eclampsia/metabolism ; Pregnancy ; RNA Interference ; RNA, Small Interfering/metabolism ; Trophoblasts/cytology ; Trophoblasts/metabolism ; Up-Regulation/drug effects
Czasopismo naukowe
Tytuł :
Inonotus obliquus polysaccharides induces apoptosis of lung cancer cells and alters energy metabolism via the LKB1/AMPK axis.
Autorzy :
Jiang S; Jiangxi Provincial Key Laboratory of Tumor Pathogens and Molecular Pathology and Department of Pathophysiology, Nanchang University Jiangxi Medical College, China.
Shi F; Jiangxi Provincial Key Laboratory of Tumor Pathogens and Molecular Pathology and Department of Pathophysiology, Nanchang University Jiangxi Medical College, China.
Lin H; Jiangxi Provincial Key Laboratory of Tumor Pathogens and Molecular Pathology and Department of Pathophysiology, Nanchang University Jiangxi Medical College, China.
Ying Y; Jiangxi Provincial Key Laboratory of Tumor Pathogens and Molecular Pathology and Department of Pathophysiology, Nanchang University Jiangxi Medical College, China.
Luo L; Department of Gastroenterology, Research Institute of Digestive Diseases, The First Affiliated Hospital, Nanchang University, Nanchang, China.
Huang D; Department of Gastroenterology, Research Institute of Digestive Diseases, The First Affiliated Hospital, Nanchang University, Nanchang, China.
Luo Z; Jiangxi Provincial Key Laboratory of Tumor Pathogens and Molecular Pathology and Department of Pathophysiology, Nanchang University Jiangxi Medical College, China. Electronic address: .
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Źródło :
International journal of biological macromolecules [Int J Biol Macromol] 2020 May 15; Vol. 151, pp. 1277-1286. Date of Electronic Publication: 2019 Nov 18.
Typ publikacji :
Journal Article
MeSH Terms :
AMP-Activated Protein Kinases/*metabolism
Apoptosis/*drug effects
Energy Metabolism/*drug effects
Fungal Polysaccharides/*pharmacology
Inonotus/*chemistry
Protein-Serine-Threonine Kinases/*metabolism
Signal Transduction/*drug effects
Adenosine Triphosphate/metabolism ; Animals ; Antineoplastic Agents/chemistry ; Antineoplastic Agents/pharmacology ; Cell Line, Tumor ; Cell Survival/drug effects ; Fungal Polysaccharides/chemistry ; Humans ; Male ; Membrane Potential, Mitochondrial/drug effects ; Mice
Czasopismo naukowe

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