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Wyszukujesz frazę ""Yoshida, Y"" wg kryterium: Autor


Tytuł:
Carbon-ion beam irradiation kills X-ray-resistant p53-null cancer cells by inducing mitotic catastrophe.
Autorzy:
Amornwichet N; Department of Radiation Oncology, Gunma University Graduate School of Medicine, Maebashi, Gunma, Japan; Department of Radiology, Chulalongkorn University, Pathumwan, Bangkok, Thailand.
Oike T; Department of Radiation Oncology, Gunma University Graduate School of Medicine, Maebashi, Gunma, Japan; Division of Genome Biology, National Cancer Center Research Institute, Chuo-ku, Tokyo, Japan.
Shibata A; Advanced Scientific Research Leaders Development Unit, Gunma University, Maebashi, Gunma, Japan.
Ogiwara H; Division of Genome Biology, National Cancer Center Research Institute, Chuo-ku, Tokyo, Japan.
Tsuchiya N; Division of Genome Biology, National Cancer Center Research Institute, Chuo-ku, Tokyo, Japan.
Yamauchi M; Division of Radiation Biology and Protection, Atomic Bomb Disease Institute, Nagasaki University, Sakamoto, Nagasaki, Japan.
Saitoh Y; Department of Radiation Oncology, Gunma University Graduate School of Medicine, Maebashi, Gunma, Japan.
Sekine R; Advanced Scientific Research Leaders Development Unit, Gunma University, Maebashi, Gunma, Japan.
Isono M; Gunma University Heavy Ion Medical Center, Maebashi, Gunma, Japan.
Yoshida Y; Gunma University Heavy Ion Medical Center, Maebashi, Gunma, Japan.
Ohno T; Gunma University Heavy Ion Medical Center, Maebashi, Gunma, Japan.
Kohno T; Division of Genome Biology, National Cancer Center Research Institute, Chuo-ku, Tokyo, Japan.
Nakano T; Department of Radiation Oncology, Gunma University Graduate School of Medicine, Maebashi, Gunma, Japan.
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Źródło:
PloS one [PLoS One] 2014 Dec 22; Vol. 9 (12), pp. e115121. Date of Electronic Publication: 2014 Dec 22 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Radiation, Ionizing*
Apoptosis/*radiation effects
Tumor Suppressor Protein p53/*genetics
Cell Line, Tumor ; DNA Breaks, Double-Stranded/radiation effects ; G2 Phase Cell Cycle Checkpoints/radiation effects ; HCT116 Cells ; Heavy Ion Radiotherapy ; Histones/metabolism ; Humans ; M Phase Cell Cycle Checkpoints/radiation effects ; Phosphorylation ; Tumor Suppressor Protein p53/deficiency ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
Tytuł:
Inhibition of phosphodiesterase-4 (PDE4) activity triggers luminal apoptosis and AKT dephosphorylation in a 3-D colonic-crypt model.
Autorzy:
Tsunoda T; Department of Cell Biology, Faculty of Medicine, Fukuoka University, Fukuoka 814-0180, Japan.
Ota T
Fujimoto T
Doi K
Tanaka Y
Yoshida Y
Ogawa M
Matsuzaki H
Hamabashiri M
Tyson DR
Kuroki M
Miyamoto S
Shirasawa S
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Źródło:
Molecular cancer [Mol Cancer] 2012 Jul 25; Vol. 11, pp. 46. Date of Electronic Publication: 2012 Jul 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*genetics
Colorectal Neoplasms/*genetics
Colorectal Neoplasms/*metabolism
Cyclic Nucleotide Phosphodiesterases, Type 4/*genetics
Proto-Oncogene Proteins c-akt/*metabolism
Apoptosis/drug effects ; Cell Line, Tumor ; Cluster Analysis ; Cyclic Nucleotide Phosphodiesterases, Type 4/metabolism ; Enzyme Activation/drug effects ; Gene Expression Profiling ; Gene Expression Regulation, Neoplastic/drug effects ; Genes, ras ; HCT116 Cells ; Humans ; Phosphodiesterase 4 Inhibitors/pharmacology ; Phosphorylation/drug effects ; RNA Interference ; Recurrence ; Rolipram/pharmacology ; Spheroids, Cellular ; Tight Junctions/metabolism ; Tumor Cells, Cultured
Czasopismo naukowe
Tytuł:
Turning point in apoptosis/necrosis induced by hydrogen peroxide.
Autorzy:
Saito Y; National Institute of Advanced Industrial Science and Technology (AIST), Human Stress Signal Research Center (HSSRC), Ikeda, Osaka 563 8577, Japan. />Nishio K
Ogawa Y
Kimata J
Kinumi T
Yoshida Y
Noguchi N
Niki E
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Źródło:
Free radical research [Free Radic Res] 2006 Jun; Vol. 40 (6), pp. 619-30.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Hydrogen Peroxide/*pharmacology
Adenosine Triphosphate/metabolism ; Caspases/metabolism ; Cell Line, Tumor ; Cytochromes c/metabolism ; Cytosol/metabolism ; Enzyme Activation/drug effects ; Enzyme Inhibitors/pharmacology ; Humans ; Mitochondria/metabolism ; Necrosis/chemically induced ; Poly(ADP-ribose) Polymerase Inhibitors ; Poly(ADP-ribose) Polymerases/metabolism
Czasopismo naukowe
Tytuł:
Thalamic retrograde degeneration in the congenitally hydrocephalic rat is attributable to apoptotic cell death.
Autorzy:
Mori F; Department of Neuropathology, Institute of Brain Science, Hirosaki University School of Medicine, Japan. />Tanji K
Yoshida Y
Wakabayashi K
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Źródło:
Neuropathology : official journal of the Japanese Society of Neuropathology [Neuropathology] 2002 Sep; Vol. 22 (3), pp. 186-93.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Hydrocephalus/*pathology
Retrograde Degeneration/*pathology
Thalamus/*ultrastructure
Animals ; Animals, Newborn ; DNA, Single-Stranded ; Hydrocephalus/genetics ; Immunohistochemistry ; In Situ Nick-End Labeling ; Microscopy, Electron ; Rats ; Retrograde Degeneration/genetics ; Thalamus/growth & development
Czasopismo naukowe
Tytuł:
Mild hypothermia mitigates post-ischemic neuronal death following focal cerebral ischemia in rat brain: immunohistochemical study of Fas, caspase-3 and TUNEL.
Autorzy:
Phanithi PB; Department of Pathology, Research Institute for Brain and Blood Vessels, Akita, Japan.
Yoshida Y
Santana A
Su M
Kawamura S
Yasui N
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Źródło:
Neuropathology : official journal of the Japanese Society of Neuropathology [Neuropathology] 2000 Dec; Vol. 20 (4), pp. 273-82.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Hypothermia, Induced*
Apoptosis/*physiology
Brain/*metabolism
Brain Ischemia/*metabolism
Caspases/*metabolism
Neurons/*metabolism
fas Receptor/*metabolism
Animals ; Brain/pathology ; Brain/physiopathology ; Brain Ischemia/pathology ; Brain Ischemia/physiopathology ; Cardiovascular Physiological Phenomena ; Caspase 3 ; Cerebral Infarction/pathology ; Immunohistochemistry ; In Situ Nick-End Labeling ; Neurons/pathology ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł:
Adenovirus-mediated transfer of p53 and Fas ligand drastically enhances apoptosis in gliomas.
Autorzy:
Shinoura N; Department of Molecular Biotherapy Research, Cancer Chemotherapy Center, Japanese Foundation for Cancer Research, Tokyo.
Yoshida Y
Asai A
Kirino T
Hamada H
Pokaż więcej
Źródło:
Cancer gene therapy [Cancer Gene Ther] 2000 May; Vol. 7 (5), pp. 732-8.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Gene Transfer Techniques*
Proto-Oncogene Proteins c-bcl-2*
Adenoviridae/*genetics
Brain Neoplasms/*therapy
Genes, p53/*genetics
Genetic Therapy/*methods
Glioma/*therapy
Membrane Glycoproteins/*genetics
Brain Neoplasms/metabolism ; Cell Separation ; DNA, Complementary/metabolism ; Diploidy ; Dose-Response Relationship, Drug ; Fas Ligand Protein ; Flow Cytometry ; Glioma/metabolism ; Humans ; Immunoblotting ; Lac Operon ; Proto-Oncogene Proteins/metabolism ; Transduction, Genetic ; Tumor Cells, Cultured ; Up-Regulation ; bcl-2-Associated X Protein
Czasopismo naukowe
Tytuł:
Adenovirus-mediated overexpression of Fas induces apoptosis of gliomas.
Autorzy:
Shinoura N; Department of Molecular Biotherapy Research, Cancer Chemotherapy Center, Japanese Foundation for Cancer Research, Tokyo.
Ohashi M
Yoshida Y
Kirino T
Asai A
Hashimoto M
Hamada H
Pokaż więcej
Źródło:
Cancer gene therapy [Cancer Gene Ther] 2000 Feb; Vol. 7 (2), pp. 224-32.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/genetics
Apoptosis*/immunology
Adenoviridae/*genetics
Glioma/*pathology
fas Receptor/*biosynthesis
fas Receptor/*genetics
Glioma/immunology ; Glioma/prevention & control ; Glioma/virology ; Humans ; Myelin Basic Protein/genetics ; Promoter Regions, Genetic/immunology ; Proto-Oncogene Proteins c-bcl-2/physiology ; Tumor Cells, Cultured ; bcl-X Protein ; fas Receptor/physiology
Czasopismo naukowe
Tytuł:
Relative level of expression of Bax and Bcl-XL determines the cellular fate of apoptosis/necrosis induced by the overexpression of Bax.
Autorzy:
Shinoura N; Department of Molecular Biotherapy Research, Cancer Chemotherapy Center, 1-37-1 Kami-Ikebukuro, Toshima-ku, Tokyo 170-8455, Japan.
Yoshida Y
Asai A
Kirino T
Hamada H
Pokaż więcej
Źródło:
Oncogene [Oncogene] 1999 Oct 07; Vol. 18 (41), pp. 5703-13.
Typ publikacji:
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation, Neoplastic*
Apoptosis/*genetics
Proto-Oncogene Proteins/*physiology
Proto-Oncogene Proteins c-bcl-2/*physiology
Brain Neoplasms/pathology ; Caspase 3 ; Caspases/metabolism ; Cell Nucleus/ultrastructure ; Glioblastoma/pathology ; Humans ; Mitochondria/metabolism ; Necrosis ; Neoplasm Proteins/metabolism ; Permeability ; Proto-Oncogene Proteins/biosynthesis ; Proto-Oncogene Proteins/genetics ; Proto-Oncogene Proteins c-bcl-2/biosynthesis ; Proto-Oncogene Proteins c-bcl-2/genetics ; Recombinant Fusion Proteins/physiology ; Transfection ; Tumor Cells, Cultured/pathology ; Vacuoles/ultrastructure ; bcl-2-Associated X Protein ; bcl-X Protein
Czasopismo naukowe
Tytuł:
Anti-tumor activities of selective HSP90α/β inhibitor, TAS-116, in combination with bortezomib in multiple myeloma.
Autorzy:
Suzuki, R
Hideshima, T
Mimura, N
Minami, J
Ohguchi, H
Kikuchi, S
Yoshida, Y
Gorgun, G
Cirstea, D
Cottini, F
Jakubikova, J
Tai, Y-T
Chauhan, D
Richardson, P G
Munshi, N C
Utsugi, T
Anderson, K C
Pokaż więcej
Temat:
HEAT shock proteins
MULTIPLE myeloma treatment
CELL-mediated cytotoxicity
APOPTOSIS
ONCOLOGY research
Źródło:
Leukemia (08876924); Feb2015, Vol. 29 Issue 2, p510-514, 5p
Czasopismo naukowe

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