Informacja

Drogi użytkowniku, aplikacja do prawidłowego działania wymaga obsługi JavaScript. Proszę włącz obsługę JavaScript w Twojej przeglądarce.

English (EN)·Polski (PL)·Ukraiński (UK)
Przejdź do strony domowej biblioteki Uniwersytet Ekonomiczny we Wrocławiu Link otwiera się w nowym oknie Logo biblioteki Prolib Integro - strona głównaUniwersytet Ekonomiczny we Wrocławiu

Wyszukujesz frazę ""Zhang, Bo"" wg kryterium: Autor

  Lista filtrów
Wyrównaj
Wyświetlanie 1-20 z 390
Tytuł:
Targeting cell death pathways for cancer therapy: recent developments in necroptosis, pyroptosis, ferroptosis, and cuproptosis research.
Autorzy:
Tong X; Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, No. 270 Dong'An Road, Shanghai, 200032, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Tang R; Shanghai Pancreatic Cancer Institute, No. 270 Dong'An Road, Shanghai, 200032, China.; Pancreatic Cancer Institute, Fudan University, Shanghai, China.
Xiao M; Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, No. 270 Dong'An Road, Shanghai, 200032, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Xu J; Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, No. 270 Dong'An Road, Shanghai, 200032, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Wang W; Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, No. 270 Dong'An Road, Shanghai, 200032, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Zhang B; Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, No. 270 Dong'An Road, Shanghai, 200032, China.; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Liu J; Shanghai Pancreatic Cancer Institute, No. 270 Dong'An Road, Shanghai, 200032, China.; Pancreatic Cancer Institute, Fudan University, Shanghai, China.
Yu X; Shanghai Pancreatic Cancer Institute, No. 270 Dong'An Road, Shanghai, 200032, China. .; Pancreatic Cancer Institute, Fudan University, Shanghai, China. .
Shi S; Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, No. 270 Dong'An Road, Shanghai, 200032, China. .; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China. .
Pokaż więcej
Źródło:
Journal of hematology & oncology [J Hematol Oncol] 2022 Dec 08; Vol. 15 (1), pp. 174. Date of Electronic Publication: 2022 Dec 08.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms:
Neoplasms*/drug therapy
Apoptosis*
Humans ; Cell Death ; Copper ; Ferroptosis ; Pyroptosis ; Necroptosis
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Selenium deficiency causes apoptosis through endoplasmic reticulum stress in swine small intestine.
Autorzy:
Zheng Y; College of Veterinary Medicine, Northeast Agricultural University, Harbin, China.
Zhang B; Fushun Center for Animal Epidemic Disease Prevention and Control, Fushun, China.
Guan H; College of Animal Science and Veterinary Medicine, Southwest Minzu University, Chengdu, China.
Jiao X; China Institute of Water Resources and Hydropower Research, Beijing, China.
Yang J; College of Veterinary Medicine, Northeast Agricultural University, Harbin, China.
Cai J; College of Veterinary Medicine, Northeast Agricultural University, Harbin, China.
Liu Q; College of Veterinary Medicine, Northeast Agricultural University, Harbin, China.
Zhang Z; College of Veterinary Medicine, Northeast Agricultural University, Harbin, China.; Key Laboratory of the Provincial Education, Department of Heilongjiang for Common Animal Disease Prevention and Treatment, Northeast Agricultural University, Harbin, China.
Pokaż więcej
Źródło:
BioFactors (Oxford, England) [Biofactors] 2021 Sep; Vol. 47 (5), pp. 788-800. Date of Electronic Publication: 2021 Jun 15.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*physiology
Endoplasmic Reticulum Stress/*physiology
Intestine, Small/*physiopathology
Selenium/*deficiency
Animals ; Disease Models, Animal ; Swine
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
LncRNA NR024118 is downregulated in sepsis and inhibits LPS‑induced apoptosis of cardiomyocytes.
Autorzy:
Qin G; ICU Department, Guangxi Zhuang Autonomous National Hospital, Nanning, Guangxi Zhuang Autonomous Region 530001, P.R. China.
Wei L; ICU Department, Guangxi Zhuang Autonomous National Hospital, Nanning, Guangxi Zhuang Autonomous Region 530001, P.R. China.
Jiang F; ICU Department, Guangxi Zhuang Autonomous National Hospital, Nanning, Guangxi Zhuang Autonomous Region 530001, P.R. China.
Li J; ICU Department, Guangxi Zhuang Autonomous National Hospital, Nanning, Guangxi Zhuang Autonomous Region 530001, P.R. China.
Zhang B; ICU Department, Guangxi Zhuang Autonomous National Hospital, Nanning, Guangxi Zhuang Autonomous Region 530001, P.R. China.
Pan D; ICU Department, Guangxi Zhuang Autonomous National Hospital, Nanning, Guangxi Zhuang Autonomous Region 530001, P.R. China.
Lu X; ICU Department, Guangxi Zhuang Autonomous National Hospital, Nanning, Guangxi Zhuang Autonomous Region 530001, P.R. China.
Pokaż więcej
Źródło:
Molecular medicine reports [Mol Med Rep] 2021 Jun; Vol. 23 (6). Date of Electronic Publication: 2021 Apr 13.
Typ publikacji:
Journal Article
MeSH Terms:
Down-Regulation*
Apoptosis/*drug effects
Lipopolysaccharides/*adverse effects
Myocytes, Cardiac/*metabolism
RNA, Long Noncoding/*metabolism
Sepsis/*metabolism
Adult ; Aged ; Female ; Gene Expression Regulation/drug effects ; Humans ; Interleukin-1beta ; Interleukin-6 ; Male ; Middle Aged ; NF-kappa B ; RNA, Long Noncoding/genetics ; Signal Transduction/drug effects ; Up-Regulation/drug effects
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Isoalantolactone inhibits IKKβ kinase activity to interrupt the NF-κB/COX-2-mediated signaling cascade and induces apoptosis regulated by the mitochondrial translocation of cofilin in glioblastoma.
Autorzy:
Xing JS; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.
Wang X; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.; Department of Neurosurgery, The Third People's Hospital of Dalian, Non-Directly Affiliated Hospital of Dalian Medical University, Dalian, China.
Lan YL; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.
Lou JC; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.
Ma B; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.
Zhu T; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.
Zhang H; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.
Wang D; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.
Yu Z; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.
Yuan Z; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.
Li XY; Department of Endocrinology, Dalian Municipal Central Hospital, Affiliated of Dalian Medical University, Dalian, China.
Zhang B; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, China.; Department of Neurosurgery, Shenzhen People's Hospital, Shenzhen, China.
Pokaż więcej
Źródło:
Cancer medicine [Cancer Med] 2019 Apr; Vol. 8 (4), pp. 1655-1670. Date of Electronic Publication: 2019 Feb 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Cyclooxygenase 2/*metabolism
I-kappa B Kinase/*antagonists & inhibitors
Mitochondria/*drug effects
Mitochondria/*metabolism
NF-kappa B/*metabolism
Sesquiterpenes/*pharmacology
Signal Transduction/*drug effects
Actin Depolymerizing Factors/metabolism ; Animals ; Cell Cycle Checkpoints/drug effects ; Cell Line, Tumor ; Cell Movement/drug effects ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Disease Models, Animal ; Dose-Response Relationship, Drug ; Glioblastoma/metabolism ; Heterografts ; Humans ; I-kappa B Kinase/metabolism ; Male ; Mice ; Molecular Structure ; Protein Transport/drug effects ; Sesquiterpenes/chemistry
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Marinobufagenin inhibits glioma growth through sodium pump α1 subunit and ERK signaling-mediated mitochondrial apoptotic pathway.
Autorzy:
Lan YL; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, 116023, China.; Department of Pharmacy, Dalian Medical University, Dalian, 116044, China.; Department of Physiology, Dalian Medical University, Dalian, 116044, China.
Wang X; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, 116023, China.
Lou JC; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, 116023, China.
Xing JS; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, 116023, China.
Zou S; Department of Physiology, Dalian Medical University, Dalian, 116044, China.
Yu ZL; Department of Pharmacy, Dalian Medical University, Dalian, 116044, China.
Ma XC; Department of Pharmacy, Dalian Medical University, Dalian, 116044, China.
Wang H; Department of Neurology, The Second Affiliated Hospital of Dalian Medical University, Dalian, 116023, China.
Zhang B; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, 116023, China.
Pokaż więcej
Źródło:
Cancer medicine [Cancer Med] 2018 May; Vol. 7 (5), pp. 2034-2047. Date of Electronic Publication: 2018 Mar 26.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Bufanolides/*pharmacology
Extracellular Signal-Regulated MAP Kinases/*metabolism
Membrane Potential, Mitochondrial/*drug effects
Sodium-Potassium-Exchanging ATPase/*antagonists & inhibitors
Animals ; Cell Cycle/drug effects ; Cell Line, Tumor ; Cell Movement/drug effects ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Cytochromes c/metabolism ; Glioma/drug therapy ; Humans ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Mitochondria/physiology ; RNA Interference ; RNA, Small Interfering/genetics ; Signal Transduction/drug effects ; Sodium-Potassium-Exchanging ATPase/genetics ; Sodium-Potassium-Exchanging ATPase/metabolism ; Transcription Factor RelA/metabolism ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Flavonoids of Kudzu Root Fermented by Eurtotium cristatum Protected Rat Pheochromocytoma Line 12 (PC12) Cells against H₂O₂-Induced Apoptosis.
Autorzy:
Zhang B; College of Food Science and Technology, Nanjing Agricultural University, Nanjing 210095, China. vip_.; College of Food Science and Technology, Bohai University, Jinzhou 121013, China. vip_.
Li W; Jiangsu Key Construction Laboratory of Food Resource Development and Quality Safe, Xuzhou Institute Technology, Xuzhou 221008, China. .
Dong M; College of Food Science and Technology, Nanjing Agricultural University, Nanjing 210095, China. .
Pokaż więcej
Źródło:
International journal of molecular sciences [Int J Mol Sci] 2017 Dec 19; Vol. 18 (12). Date of Electronic Publication: 2017 Dec 19.
Typ publikacji:
Journal Article
MeSH Terms:
Antioxidants/*pharmacology
Apoptosis/*drug effects
Flavonoids/*pharmacology
Neuroprotective Agents/*pharmacology
Plant Extracts/*pharmacology
Pueraria/*chemistry
Animals ; Antioxidants/chemistry ; Fermentation ; Flavonoids/analysis ; Hydrogen Peroxide/toxicity ; Neuroprotective Agents/chemistry ; PC12 Cells ; Plant Extracts/chemistry ; Plant Roots/chemistry ; Rats
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
GATA4 is highly expressed in childhood acute lymphoblastic leukemia, promotes cell proliferation and inhibits apoptosis by activating BCL2 and MDM2.
Autorzy:
Han Q; Department of Pediatrics, Daqing Oilfield General Hospital, Daqing, Heilongjiang 163000, P.R. China.
Xu X; Department of Pediatrics, Daqing Oilfield General Hospital, Daqing, Heilongjiang 163000, P.R. China.
Li J; Department of Pediatrics, Daqing Oilfield General Hospital, Daqing, Heilongjiang 163000, P.R. China.
Wang J; Department of Pediatrics, Daqing Oilfield General Hospital, Daqing, Heilongjiang 163000, P.R. China.
Bai L; Department of Pediatrics, Daqing Oilfield General Hospital, Daqing, Heilongjiang 163000, P.R. China.
Wang A; Department of Pediatrics, Daqing Oilfield General Hospital, Daqing, Heilongjiang 163000, P.R. China.
Wang W; Department of Pediatrics, Daqing Oilfield General Hospital, Daqing, Heilongjiang 163000, P.R. China.
Zhang B; Department of Pediatric Neurology, The First Hospital of Jilin University, Changchun, Jilin 130000, P.R. China.
Pokaż więcej
Źródło:
Molecular medicine reports [Mol Med Rep] 2017 Nov; Vol. 16 (5), pp. 6290-6298. Date of Electronic Publication: 2017 Aug 28.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*genetics
Cell Proliferation/*genetics
GATA4 Transcription Factor/*genetics
Gene Expression Regulation/*genetics
Precursor Cell Lymphoblastic Leukemia-Lymphoma/*genetics
Proto-Oncogene Proteins c-bcl-2/*genetics
Proto-Oncogene Proteins c-mdm2/*genetics
Cell Line ; Cell Line, Tumor ; Humans ; Jurkat Cells ; Precursor Cell Lymphoblastic Leukemia-Lymphoma/pathology ; Signal Transduction/genetics ; Transcriptional Activation/genetics ; Tumor Suppressor Protein p53/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Long non‑coding RNA MEG3 contributes to cisplatin‑induced apoptosis via inhibition of autophagy in human glioma cells.
Autorzy:
Ma B; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, Liaoning 116023, P.R. China.
Gao Z; Department of Clinical Laboratory, The Second Affiliated Hospital of Dalian Medical University, Dalian, Liaoning 116023, P.R. China.
Lou J; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, Liaoning 116023, P.R. China.
Zhang H; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, Liaoning 116023, P.R. China.
Yuan Z; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, Liaoning 116023, P.R. China.
Wu Q; Department of Neurology, Dalian Municipal Central Hospital Affiliated to Dalian Medical University, Dalian, Liaoning 116033, P.R. China.
Li X; Department of Endocrinology, Dalian Municipal Central Hospital Affiliated to Dalian Medical University, Dalian, Liaoning 116033, P.R. China.
Zhang B; Department of Neurosurgery, The Second Affiliated Hospital of Dalian Medical University, Dalian, Liaoning 116023, P.R. China.
Pokaż więcej
Źródło:
Molecular medicine reports [Mol Med Rep] 2017 Sep; Vol. 16 (3), pp. 2946-2952. Date of Electronic Publication: 2017 Jun 30.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Autophagy/*drug effects
Brain Neoplasms/*genetics
Brain Neoplasms/*pathology
Cisplatin/*pharmacology
Glioma/*genetics
Glioma/*pathology
RNA, Long Noncoding/*metabolism
Apoptosis/genetics ; Autophagy/genetics ; Cell Line, Tumor ; Humans ; RNA, Long Noncoding/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Homoharringtonine induces apoptosis and inhibits STAT3 via IL-6/JAK1/STAT3 signal pathway in Gefitinib-resistant lung cancer cells.
Autorzy:
Cao W; 1] School of Life Sciences, Tsinghua University, Beijing, 100084, China [2] The Shenzhen Key Laboratory of Gene and Antibody Therapy, State Key Laboratory of Health Science and Technology (prep), Center for Biotechnology &Biomedicine and Division of Life &Health Sciences, Graduate School at Shenzhen, Tsinghua University, Shenzhen, Guangdong, 518055, China.
Liu Y; 1] The Shenzhen Key Laboratory of Gene and Antibody Therapy, State Key Laboratory of Health Science and Technology (prep), Center for Biotechnology &Biomedicine and Division of Life &Health Sciences, Graduate School at Shenzhen, Tsinghua University, Shenzhen, Guangdong, 518055, China [2] School of Basic Medical Sciences, Hubei University of Medicine, Shiyan, 442000, Hubei, China.
Zhang R; 1] School of Life Sciences, Tsinghua University, Beijing, 100084, China [2] The Shenzhen Key Laboratory of Gene and Antibody Therapy, State Key Laboratory of Health Science and Technology (prep), Center for Biotechnology &Biomedicine and Division of Life &Health Sciences, Graduate School at Shenzhen, Tsinghua University, Shenzhen, Guangdong, 518055, China.
Zhang B; National Laboratory of Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Beijing, 100101, China.
Wang T; 1] The Shenzhen Key Laboratory of Gene and Antibody Therapy, State Key Laboratory of Health Science and Technology (prep), Center for Biotechnology &Biomedicine and Division of Life &Health Sciences, Graduate School at Shenzhen, Tsinghua University, Shenzhen, Guangdong, 518055, China [2] The Key Laboratory of Bioorganic Phosphorus Chemistry &Chemical Biology (Ministry of Education), Department of Chemistry, Tsinghua University, Beijing, 100084, China.
Zhu X; 1] School of Life Sciences, Tsinghua University, Beijing, 100084, China [2] The Shenzhen Key Laboratory of Gene and Antibody Therapy, State Key Laboratory of Health Science and Technology (prep), Center for Biotechnology &Biomedicine and Division of Life &Health Sciences, Graduate School at Shenzhen, Tsinghua University, Shenzhen, Guangdong, 518055, China.
Mei L; 1] School of Life Sciences, Tsinghua University, Beijing, 100084, China [2] The Shenzhen Key Laboratory of Gene and Antibody Therapy, State Key Laboratory of Health Science and Technology (prep), Center for Biotechnology &Biomedicine and Division of Life &Health Sciences, Graduate School at Shenzhen, Tsinghua University, Shenzhen, Guangdong, 518055, China.
Chen H; 1] School of Life Sciences, Tsinghua University, Beijing, 100084, China [2] The Shenzhen Key Laboratory of Gene and Antibody Therapy, State Key Laboratory of Health Science and Technology (prep), Center for Biotechnology &Biomedicine and Division of Life &Health Sciences, Graduate School at Shenzhen, Tsinghua University, Shenzhen, Guangdong, 518055, China.
Zhang H; 1] School of Life Sciences, Tsinghua University, Beijing, 100084, China [2] The Shenzhen Key Laboratory of Gene and Antibody Therapy, State Key Laboratory of Health Science and Technology (prep), Center for Biotechnology &Biomedicine and Division of Life &Health Sciences, Graduate School at Shenzhen, Tsinghua University, Shenzhen, Guangdong, 518055, China.
Ming P; Laboratory of Zhuhai People's Hospital, Zhuhai, Guangdong, 519000, China.
Huang L; 1] School of Life Sciences, Tsinghua University, Beijing, 100084, China [2] The Shenzhen Key Laboratory of Gene and Antibody Therapy, State Key Laboratory of Health Science and Technology (prep), Center for Biotechnology &Biomedicine and Division of Life &Health Sciences, Graduate School at Shenzhen, Tsinghua University, Shenzhen, Guangdong, 518055, China [3] The Key Laboratory of Bioorganic Phosphorus Chemistry &Chemical Biology (Ministry of Education), Department of Chemistry, Tsinghua University, Beijing, 100084, China.
Pokaż więcej
Źródło:
Scientific reports [Sci Rep] 2015 Jul 13; Vol. 5, pp. 8477. Date of Electronic Publication: 2015 Jul 13.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents/*toxicity
Apoptosis/*drug effects
Drug Resistance, Neoplasm/*drug effects
Harringtonines/*toxicity
Quinazolines/*toxicity
Signal Transduction/*drug effects
Animals ; Antineoplastic Agents/chemistry ; Antineoplastic Agents/therapeutic use ; Carcinoma, Non-Small-Cell Lung/drug therapy ; Carcinoma, Non-Small-Cell Lung/metabolism ; Carcinoma, Non-Small-Cell Lung/pathology ; Cell Line, Tumor ; Cell Survival/drug effects ; Docetaxel ; Drug Synergism ; Enzyme-Linked Immunosorbent Assay ; ErbB Receptors/genetics ; ErbB Receptors/metabolism ; Gefitinib ; Harringtonines/chemistry ; Harringtonines/therapeutic use ; Homoharringtonine ; Humans ; Interleukin-6/analysis ; Interleukin-6/metabolism ; Janus Kinase 1/metabolism ; Lung Neoplasms/drug therapy ; Lung Neoplasms/metabolism ; Lung Neoplasms/pathology ; Male ; Mice ; Mice, Nude ; Mitochondria/drug effects ; Mitochondria/metabolism ; Phosphorylation/drug effects ; STAT3 Transcription Factor/metabolism ; Taxoids/therapeutic use ; Taxoids/toxicity ; Transplantation, Heterologous
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Atrazine Causes Autophagy- and Apoptosis-Related Neurodegenerative Effects in Dopaminergic Neurons in the Rat Nigrostriatal Dopaminergic System.
Autorzy:
Song XY; Department of Hygienic Toxicology, School of Public Health, Harbin Medical University, 157 Baojian Road, Nan Gang District, Harbin 150081, China. .
Li JN; Department of Hygienic Toxicology, School of Public Health, Harbin Medical University, 157 Baojian Road, Nan Gang District, Harbin 150081, China. .
Wu YP; Department of Hygienic Toxicology, School of Public Health, Harbin Medical University, 157 Baojian Road, Nan Gang District, Harbin 150081, China. Wuyanping_.
Zhang B; Department of Hygienic Toxicology, School of Public Health, Harbin Medical University, 157 Baojian Road, Nan Gang District, Harbin 150081, China. sky_zhangbo_.
Li BX; Department of Hygienic Toxicology, School of Public Health, Harbin Medical University, 157 Baojian Road, Nan Gang District, Harbin 150081, China. .
Pokaż więcej
Źródło:
International journal of molecular sciences [Int J Mol Sci] 2015 Jun 12; Vol. 16 (6), pp. 13490-506. Date of Electronic Publication: 2015 Jun 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Autophagy*
Atrazine/*toxicity
Dopaminergic Neurons/*drug effects
Herbicides/*toxicity
Animals ; Apoptosis Regulatory Proteins/metabolism ; Atrazine/adverse effects ; Beclin-1 ; Caspase 9/metabolism ; Dopaminergic Neurons/metabolism ; Herbicides/adverse effects ; Male ; Mesencephalon/cytology ; Mesencephalon/drug effects ; Mesencephalon/metabolism ; Mesencephalon/ultrastructure ; Microtubule-Associated Proteins/metabolism ; Mitochondria/drug effects ; Mitochondria/metabolism ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Rats ; Rats, Wistar ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Low-frequency low energy ultrasound combined with microbubbles induces distinct apoptosis of A7r5 cells.
Autorzy:
Zhang B; Department of Echocardiography, Tongji University Affiliated Shanghai East Hospital, Shanghai 200120, P.R. China.
Zhou H; Shanghai Acoustics Laboratory, Chinese Academy of Sciences, Shanghai 200233, P.R. China.
Cheng Q; Institute of Acoustics, Tongji University, Shanghai 200092, P.R. China.
Lei L; Department of Echocardiography, Tongji University Affiliated Shanghai East Hospital, Shanghai 200120, P.R. China.
Hu B; Department of Ultrasound in Medicine, Shanghai Institute of Ultrasound in Medicine, Shanghai Jiao Tong University Affiliated 6th People's Hospital, Shanghai 200233, P.R. China.
Pokaż więcej
Źródło:
Molecular medicine reports [Mol Med Rep] 2014 Dec; Vol. 10 (6), pp. 3282-8. Date of Electronic Publication: 2014 Oct 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Contrast Media/*pharmacology
Microbubbles/*therapeutic use
Animals ; Cell Death/drug effects ; Cell Line ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Muscle, Smooth, Vascular/drug effects ; Muscle, Smooth, Vascular/metabolism ; Myocytes, Smooth Muscle/drug effects ; Myocytes, Smooth Muscle/metabolism ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Rats ; Ultrasonics/methods ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
The biological effect of large single doses: a possible role for non-targeted effects in cell inactivation.
Autorzy:
Veldwijk MR; Department of Radiation Oncology, Universitätsmedizin Mannheim, Medical Faculty Mannheim, University of Heidelberg, Mannheim, Germany.
Zhang B; Department of Radiation Oncology, Universitätsmedizin Mannheim, Medical Faculty Mannheim, University of Heidelberg, Mannheim, Germany.
Wenz F; Department of Radiation Oncology, Universitätsmedizin Mannheim, Medical Faculty Mannheim, University of Heidelberg, Mannheim, Germany.
Herskind C; Department of Radiation Oncology, Universitätsmedizin Mannheim, Medical Faculty Mannheim, University of Heidelberg, Mannheim, Germany.
Pokaż więcej
Źródło:
PloS one [PLoS One] 2014 Jan 22; Vol. 9 (1), pp. e84991. Date of Electronic Publication: 2014 Jan 22 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*radiation effects
Cell Cycle/*radiation effects
Cytokines/*metabolism
Histones/*metabolism
Animals ; Breast Neoplasms/metabolism ; Breast Neoplasms/pathology ; Cell Line ; Cell Line, Tumor ; Cell Survival/radiation effects ; Culture Media, Conditioned/metabolism ; Dose-Response Relationship, Radiation ; Female ; Flow Cytometry ; G1 Phase/radiation effects ; Humans ; MCF-7 Cells ; Microscopy, Fluorescence ; Time Factors
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Down-regulation of AP-4 inhibits proliferation, induces cell cycle arrest and promotes apoptosis in human gastric cancer cells.
Autorzy:
Liu X; Department of General Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Zhang B
Guo Y
Liang Q
Wu C
Wu L
Tao K
Wang G
Chen J
Pokaż więcej
Źródło:
PloS one [PLoS One] 2012; Vol. 7 (5), pp. e37096. Date of Electronic Publication: 2012 May 16.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adaptor Protein Complex 4/*genetics
Apoptosis/*genetics
Cell Cycle Checkpoints/*genetics
Stomach Neoplasms/*genetics
Stomach Neoplasms/*pathology
Adaptor Protein Complex 4/metabolism ; Caspase 9/genetics ; Caspase 9/metabolism ; Cell Growth Processes/genetics ; Cell Line, Tumor ; Cyclin D1/genetics ; Cyclin D1/metabolism ; Cyclin-Dependent Kinase Inhibitor p21/genetics ; Cyclin-Dependent Kinase Inhibitor p21/metabolism ; Down-Regulation ; Gene Expression Regulation, Neoplastic ; Humans ; Proto-Oncogene Proteins c-bcl-2 ; RNA, Messenger/genetics ; RNA, Small Interfering/genetics ; Stomach Neoplasms/metabolism ; bcl-X Protein/genetics ; bcl-X Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Gefitinib analogue V1801 induces apoptosis of T790M EGFR-harboring lung cancer cells by up-regulation of the BH-3 only protein Noxa.
Autorzy:
Zhang B; Division of Molecular Carcinogenesis and Targeted Therapy for Cancer, State Key Laboratory of Biomembrane and Membrane Biotechnology, Institute of Zoology, Chinese Academy of Sciences, Beijing, China.
Jiao J
Liu Y
Guo LX
Zhou B
Li GQ
Yao ZJ
Zhou GB
Pokaż więcej
Źródło:
PloS one [PLoS One] 2012; Vol. 7 (11), pp. e48748. Date of Electronic Publication: 2012 Nov 21.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
ErbB Receptors/*genetics
Lung Neoplasms/*pathology
Mutation/*genetics
Protein Kinase Inhibitors/*pharmacology
Proto-Oncogene Proteins c-bcl-2/*metabolism
Quinazolines/*pharmacology
Animals ; Antineoplastic Agents/pharmacology ; Carcinoma, Non-Small-Cell Lung/drug therapy ; Carcinoma, Non-Small-Cell Lung/genetics ; Carcinoma, Non-Small-Cell Lung/pathology ; Cell Line, Tumor ; Drug Resistance, Neoplasm/drug effects ; Gefitinib ; Humans ; Lung Neoplasms/drug therapy ; Lung Neoplasms/genetics ; Mice ; Mice, Nude ; Proto-Oncogene Proteins c-myc/metabolism ; Treatment Outcome ; Up-Regulation/drug effects ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Tanshinone IIA suppresses lung injury and apoptosis, and modulates protein kinase B and extracellular signal-regulated protein kinase pathways in rats challenged with seawater exposure.
Autorzy:
Li JH; Department of Respiration, Tangdu Hospital, Fourth Military Medical University, Xi'an, China.
Xu M
Xie XY
Fan QX
Mu DG
Zhang Y
Cao FL
Wang YX
Zhao PT
Zhang B
Jin FG
Li ZC
Pokaż więcej
Źródło:
Clinical and experimental pharmacology & physiology [Clin Exp Pharmacol Physiol] 2011 Apr; Vol. 38 (4), pp. 269-77.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Abietanes/*pharmacology
Acute Lung Injury/*drug therapy
Apoptosis/*drug effects
MAP Kinase Signaling System/*drug effects
Mitogen-Activated Protein Kinase 1/*metabolism
Mitogen-Activated Protein Kinase 3/*metabolism
Proto-Oncogene Proteins c-akt/*metabolism
Acute Lung Injury/enzymology ; Acute Lung Injury/pathology ; Animals ; Apoptosis/physiology ; Caspase 3/genetics ; Caspase 3/metabolism ; Mitogen-Activated Protein Kinases/metabolism ; Oxygen/blood ; Oxygen/metabolism ; Partial Pressure ; Rats ; Rats, Sprague-Dawley ; Seawater ; bcl-Associated Death Protein/genetics ; bcl-Associated Death Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Protein kinase Cdelta stimulates proteasome-dependent degradation of C/EBPalpha during apoptosis induction of leukemic cells.
Autorzy:
Zhao M; Institute of Health Sciences, Shanghai Institutes for Biological Sciences of Chinese Academy of Sciences and Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Duan XF
Zhao XY
Zhang B
Lu Y
Liu W
Cheng JK
Chen GQ
Pokaż więcej
Źródło:
PloS one [PLoS One] 2009 Aug 07; Vol. 4 (8), pp. e6552. Date of Electronic Publication: 2009 Aug 07.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*/drug effects
CCAAT-Enhancer-Binding Protein-alpha/*metabolism
Leukemia/*pathology
Proteasome Endopeptidase Complex/*metabolism
Protein Kinase C-delta/*metabolism
Base Sequence ; Blotting, Western ; CCAAT-Enhancer-Binding Protein-alpha/genetics ; Caspase 3/metabolism ; Cell Line, Tumor ; DNA Primers ; Down-Regulation ; Etoposide/pharmacology ; Gene Silencing ; Humans ; Hydrolysis ; Leukemia/enzymology ; Leukemia/metabolism ; RNA, Messenger/genetics ; RNA, Small Interfering ; Reverse Transcriptase Polymerase Chain Reaction ; Transcription, Genetic ; Ubiquitination
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
CircYIPF6 regulates glioma cell proliferation, apoptosis, and glycolysis through targeting miR-760 to modulate PTBP1 expression
Autorzy:
Lei Dan
Xiao Wenyong
Zhang Bo
Pokaż więcej
Temat:
glioma
circyipf6
mir-760
ptbp1
proliferation
apoptosis
glycolysis
Neurosciences. Biological psychiatry. Neuropsychiatry
RC321-571
Źródło:
Translational Neuroscience, Vol 14, Iss 1, Pp 191-94 (2023)
Opis pliku:
electronic resource
Relacje:
https://doaj.org/toc/2081-6936
Dostęp URL:
https://doaj.org/article/99042dc3fc2745bbb7350a4934b4e5ad  Link otwiera się w nowym oknie
Czasopismo naukowe
Szczegóły
Wyświetlanie 1-20 z 390

Ta witryna wykorzystuje pliki cookies do przechowywania informacji na Twoim komputerze. Pliki cookies stosujemy w celu świadczenia usług na najwyższym poziomie, w tym w sposób dostosowany do indywidualnych potrzeb. Korzystanie z witryny bez zmiany ustawień dotyczących cookies oznacza, że będą one zamieszczane w Twoim komputerze. W każdym momencie możesz dokonać zmiany ustawień dotyczących cookies