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Tytuł :
1,25‑Dihydroxyvitamin D3 enhances the susceptibility of anaplastic thyroid cancer cells to adriamycin‑induced apoptosis by increasing the generation of reactive oxygen species.
Autorzy :
Zhang T; Department of Thyroid Surgery, The First Hospital of China Medical University, Shenyang, Liaoning 110001, P.R. China.
He L; Department of Thyroid Surgery, The First Hospital of China Medical University, Shenyang, Liaoning 110001, P.R. China.
Sun W; Department of Thyroid Surgery, The First Hospital of China Medical University, Shenyang, Liaoning 110001, P.R. China.
Qin Y; Department of Thyroid Surgery, The First Hospital of China Medical University, Shenyang, Liaoning 110001, P.R. China.
Zhang P; Department of Thyroid Surgery, The First Hospital of China Medical University, Shenyang, Liaoning 110001, P.R. China.
Zhang H; Department of Thyroid Surgery, The First Hospital of China Medical University, Shenyang, Liaoning 110001, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2019 Sep; Vol. 20 (3), pp. 2641-2648. Date of Electronic Publication: 2019 Jul 25.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Calcitriol/*pharmacology
Doxorubicin/*pharmacology
Reactive Oxygen Species/*metabolism
Thyroid Carcinoma, Anaplastic/*etiology
Thyroid Carcinoma, Anaplastic/*metabolism
Biomarkers ; Caspase 3/metabolism ; Cell Line, Tumor ; Cell Survival/drug effects ; Enzyme Activation/drug effects ; Humans ; Thyroid Carcinoma, Anaplastic/pathology
Czasopismo naukowe
Tytuł :
Ivermectin induces cell cycle arrest and apoptosis of HeLa cells via mitochondrial pathway.
Autorzy :
Zhang P; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai, China.
Zhang Y; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai, China.
Liu K; Shandong Key Laboratory of Chemical Medicine, Shandong Academy of Pharmaceutical Sciences, Jinan, China.
Liu B; Vegetable Technical Extension Station Qingpu District Shanghai, Shanghai, China.
Xu W; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai, China.
Gao J; College of Life and Environmental Sciences, Shanghai Normal University, Shanghai, China.
Ding L; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai, China.
Tao L; Shanghai Key Laboratory of Chemical Biology, School of Pharmacy, East China University of Science and Technology, Shanghai, China.
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Źródło :
Cell proliferation [Cell Prolif] 2019 Mar; Vol. 52 (2), pp. e12543. Date of Electronic Publication: 2018 Dec 04.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents/*pharmacology
Antiparasitic Agents/*pharmacology
Apoptosis/*drug effects
Cell Cycle Checkpoints/*drug effects
Ivermectin/*pharmacology
Mitochondria/*drug effects
Uterine Cervical Neoplasms/*drug therapy
Cell Survival/drug effects ; Female ; HeLa Cells ; Humans ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/metabolism ; Mitochondria/pathology ; Uterine Cervical Neoplasms/metabolism ; Uterine Cervical Neoplasms/pathology
Czasopismo naukowe
Tytuł :
In Vitro and In Vivo Antimetastatic Effects of ZSTK474 on Prostate Cancer DU145 Cells.
Autorzy :
Liu J; Tianjin Key Laboratory on Technologies Enabling Development of Clinical Therapeutics and Diagnostics, School of Pharmaceutical Sciences, Tianjin Medical University, Tianjin 300070, China.; Research Center of Basic Medical Sciences, Tianjin Medical University, Tianjin 300070, China.
Tan X; Tianjin Key Laboratory on Technologies Enabling Development of Clinical Therapeutics and Diagnostics, School of Pharmaceutical Sciences, Tianjin Medical University, Tianjin 300070, China.; Research Center of Basic Medical Sciences, Tianjin Medical University, Tianjin 300070, China.; State Key Laboratory of Toxicology and Medical Countermeasures, Beijing Institute of Pharmacology and Toxicology, Beijing 100850, China.
Zhao W; Tianjin Key Laboratory on Technologies Enabling Development of Clinical Therapeutics and Diagnostics, School of Pharmaceutical Sciences, Tianjin Medical University, Tianjin 300070, China.; Research Center of Basic Medical Sciences, Tianjin Medical University, Tianjin 300070, China.
Liu J; Tianjin Key Laboratory on Technologies Enabling Development of Clinical Therapeutics and Diagnostics, School of Pharmaceutical Sciences, Tianjin Medical University, Tianjin 300070, China.; Research Center of Basic Medical Sciences, Tianjin Medical University, Tianjin 300070, China.; State Key Laboratory of Toxicology and Medical Countermeasures, Beijing Institute of Pharmacology and Toxicology, Beijing 100850, China.
Xing X; State Key Laboratory of Modern Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China.
Fan G; State Key Laboratory of Modern Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China.
Zhang P; Department of Anatomy and Histology, School of Basic Medical Sciences, Tianjin Medical University, Tianjin 300070, China.
Zhang Z; Tianjin Key Laboratory on Technologies Enabling Development of Clinical Therapeutics and Diagnostics, School of Pharmaceutical Sciences, Tianjin Medical University, Tianjin 300070, China.
Zhong Y; State Key Laboratory of Toxicology and Medical Countermeasures, Beijing Institute of Pharmacology and Toxicology, Beijing 100850, China.
Kong D; Tianjin Key Laboratory on Technologies Enabling Development of Clinical Therapeutics and Diagnostics, School of Pharmaceutical Sciences, Tianjin Medical University, Tianjin 300070, China.; Research Center of Basic Medical Sciences, Tianjin Medical University, Tianjin 300070, China.
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Źródło :
Current cancer drug targets [Curr Cancer Drug Targets] 2019; Vol. 19 (4), pp. 321-329.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis*
Cell Movement*
Bone Neoplasms/*drug therapy
Neovascularization, Pathologic/*drug therapy
Phosphatidylinositol 3-Kinases/*chemistry
Prostatic Neoplasms/*drug therapy
Triazines/*pharmacology
Animals ; Bone Neoplasms/metabolism ; Bone Neoplasms/secondary ; Cell Proliferation ; Humans ; In Vitro Techniques ; Male ; Mice ; Mice, Inbred BALB C ; Mice, Nude ; Prostatic Neoplasms/metabolism ; Prostatic Neoplasms/pathology ; Signal Transduction ; Tumor Cells, Cultured ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
Phosphatidylinositol 3-Kinase/Akt signal pathway resists the apoptosis and inflammation in human extravillous trophoblasts induced by Porphyromonas gingivalis.
Autorzy :
Guo H; MOST KLOS & KLOBM, School & Hospital of Stomatology, Wuhan University, Wuhan, Hubei, China.
Ren H; Department of Orthodontics, Xiangyang Hospital of Stomatology, Xiangyang, Hubei, China.
Liang S; MOST KLOS & KLOBM, School & Hospital of Stomatology, Wuhan University, Wuhan, Hubei, China.
Ji Y; MOST KLOS & KLOBM, School & Hospital of Stomatology, Wuhan University, Wuhan, Hubei, China.
Jiang H; MOST KLOS & KLOBM, School & Hospital of Stomatology, Wuhan University, Wuhan, Hubei, China.
Zhang P; Department of Pediatric Dentistry, School of Dentistry, University of Alabama at Birmingham, USA.
Du M; MOST KLOS & KLOBM, School & Hospital of Stomatology, Wuhan University, Wuhan, Hubei, China. Electronic address: .
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Źródło :
Molecular immunology [Mol Immunol] 2018 Dec; Vol. 104, pp. 100-107.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*immunology
Bacteroidaceae Infections/*immunology
Phosphatidylinositol 3-Kinases/*immunology
Porphyromonas gingivalis/*immunology
Proto-Oncogene Proteins c-akt/*immunology
Signal Transduction/*immunology
Trophoblasts/*immunology
Bacteroidaceae Infections/pathology ; Cell Line ; Humans ; Inflammation/immunology ; Inflammation/microbiology ; Inflammation/pathology ; Trophoblasts/microbiology ; Trophoblasts/pathology
Czasopismo naukowe
Tytuł :
Zinc enhances chemosensitivity to paclitaxel in PC‑3 prostate cancer cells.
Autorzy :
Zhang P; Department of Pathophysiology, Prostate Diseases Prevention and Treatment Research Center, School of Basic Medical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China.
Li Y; Department of Pathophysiology, Prostate Diseases Prevention and Treatment Research Center, School of Basic Medical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China.
Tang X; Department of Pathophysiology, Prostate Diseases Prevention and Treatment Research Center, School of Basic Medical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China.
Guo R; Department of Pathophysiology, Prostate Diseases Prevention and Treatment Research Center, School of Basic Medical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China.
Li J; Department of Pathophysiology, Prostate Diseases Prevention and Treatment Research Center, School of Basic Medical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China.
Chen YY; Department of Pathophysiology, Prostate Diseases Prevention and Treatment Research Center, School of Basic Medical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China.
Guo H; Department of Pathophysiology, Prostate Diseases Prevention and Treatment Research Center, School of Basic Medical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China.
Su J; Department of Pathophysiology, Prostate Diseases Prevention and Treatment Research Center, School of Basic Medical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China.
Sun L; Department of Pathophysiology, Prostate Diseases Prevention and Treatment Research Center, School of Basic Medical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China.
Liu Y; Department of Pathophysiology, Prostate Diseases Prevention and Treatment Research Center, School of Basic Medical Sciences, Jilin University, Changchun, Jilin 130021, P.R. China.
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Źródło :
Oncology reports [Oncol Rep] 2018 Oct; Vol. 40 (4), pp. 2269-2277. Date of Electronic Publication: 2018 Aug 02.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents, Phytogenic/*pharmacology
Apoptosis/*drug effects
Drug Resistance, Neoplasm/*drug effects
Mitochondria/*pathology
Paclitaxel/*pharmacology
Prostatic Neoplasms/*pathology
Zinc/*pharmacology
Cell Proliferation/drug effects ; Humans ; Male ; Mitochondria/drug effects ; Mitochondria/metabolism ; Prostatic Neoplasms/drug therapy ; Prostatic Neoplasms/metabolism ; Tumor Cells, Cultured
Czasopismo naukowe
Tytuł :
EGb761 Ameliorates Neuronal Apoptosis and Promotes Angiogenesis in Experimental Intracerebral Hemorrhage via RSK1/GSK3β Pathway.
Autorzy :
Pan C; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China.
Liu N; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China.
Zhang P; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China.
Wu Q; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China.
Deng H; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China.
Xu F; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China.
Lian L; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China.
Liang Q; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China.
Hu Y; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China.
Zhu S; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China. .
Tang Z; Department of Neurology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430030, People's Republic of China. .
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Źródło :
Molecular neurobiology [Mol Neurobiol] 2018 Feb; Vol. 55 (2), pp. 1556-1567. Date of Electronic Publication: 2017 Feb 09.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Ginkgo biloba*
Apoptosis/*drug effects
Cerebral Hemorrhage/*drug therapy
Glycogen Synthase Kinase 3 beta/*metabolism
Neovascularization, Physiologic/*drug effects
Neuroprotective Agents/*pharmacology
Plant Extracts/*pharmacology
Ribosomal Protein S6 Kinases, 90-kDa/*metabolism
Animals ; Cerebral Hemorrhage/metabolism ; Disease Models, Animal ; Male ; Mice ; Neurons/drug effects ; Neurons/metabolism ; Neuroprotective Agents/therapeutic use ; Phosphorylation/drug effects ; Plant Extracts/therapeutic use ; Signal Transduction/drug effects
Czasopismo naukowe
Tytuł :
Calcitonin gene‑related peptide reduces Porphyromonas gingivalis LPS‑induced TNF‑α release and apoptosis in osteoblasts.
Autorzy :
Zhou Y; Department of Oral and Maxillofacial Surgery, Xinqiao Hospital, Third Military Medical University, Chongqing 400037, P.R. China.
Zhang H; Department of Oral and Maxillofacial Surgery, Xinqiao Hospital, Third Military Medical University, Chongqing 400037, P.R. China.
Zhang G; Department of Oral and Maxillofacial Surgery, Xinqiao Hospital, Third Military Medical University, Chongqing 400037, P.R. China.
He Y; Department of Stomatology, General Hospital of Chengdu Military Region of PLA, Chengdu, Sichuan 610083, P.R. China.
Zhang P; Department of Oral and Maxillofacial Surgery, Xinqiao Hospital, Third Military Medical University, Chongqing 400037, P.R. China.
Sun Z; Jiebao Biotechnology, Wuhan, Hubei 430012, P.R. China.
Gao Y; Department of Oral and Maxillofacial Surgery, Xinqiao Hospital, Third Military Medical University, Chongqing 400037, P.R. China.
Tan Y; Department of Oral and Maxillofacial Surgery, Xinqiao Hospital, Third Military Medical University, Chongqing 400037, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2018 Feb; Vol. 17 (2), pp. 3246-3254. Date of Electronic Publication: 2017 Dec 06.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Calcitonin Gene-Related Peptide/*pharmacology
Lipopolysaccharides/*toxicity
Porphyromonas gingivalis/*metabolism
Tumor Necrosis Factor-alpha/*metabolism
Animals ; Caspase 3/metabolism ; Caspase 8/metabolism ; Cells, Cultured ; Cytokines/analysis ; Enzyme-Linked Immunosorbent Assay ; Mice ; Mice, Inbred BALB C ; Osteoblasts/cytology ; Osteoblasts/drug effects ; Osteoblasts/metabolism ; Tumor Necrosis Factor-alpha/analysis
Czasopismo naukowe
Tytuł :
Ginsenoside‑Rg5 treatment inhibits apoptosis of chondrocytes and degradation of cartilage matrix in a rat model of osteoarthritis.
Autorzy :
Zhang P; The Disease Prevention Center of Anyang Hospital of Traditional Chinese Medicine of Henan Province, Nanyang, Henan 455000, P.R. China.
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Źródło :
Oncology reports [Oncol Rep] 2017 Mar; Vol. 37 (3), pp. 1497-1502. Date of Electronic Publication: 2017 Jan 19.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Cartilage, Articular/*drug effects
Chondrocytes/*drug effects
Ginsenosides/*pharmacology
Knee Joint/*drug effects
Osteoarthritis/*drug therapy
Animals ; Blotting, Western ; Cartilage, Articular/metabolism ; Cartilage, Articular/pathology ; Cells, Cultured ; Chondrocytes/metabolism ; Chondrocytes/pathology ; Disease Models, Animal ; Flow Cytometry ; Knee Joint/metabolism ; Knee Joint/pathology ; Male ; Matrix Metalloproteinase 13/genetics ; Matrix Metalloproteinase 13/metabolism ; Osteoarthritis/metabolism ; Osteoarthritis/pathology ; RNA, Messenger/genetics ; Rats ; Rats, Wistar ; Real-Time Polymerase Chain Reaction ; Reverse Transcriptase Polymerase Chain Reaction ; Tissue Inhibitor of Metalloproteinase-1/genetics ; Tissue Inhibitor of Metalloproteinase-1/metabolism
Czasopismo naukowe
Tytuł :
Neddylation Inhibition Activates the Extrinsic Apoptosis Pathway through ATF4-CHOP-DR5 Axis in Human Esophageal Cancer Cells.
Autorzy :
Chen P; College of Basic Medical Sciences, Zhengzhou University; Collaborative Innovation Center of Henan Province for Cancer Chemoprevention, Zhengzhou, China. Cancer Institute, Fudan University Shanghai Cancer Center; Collaborative Innovation Center of Cancer Medicine; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China. .
Hu T; College of Basic Medical Sciences, Zhengzhou University; Collaborative Innovation Center of Henan Province for Cancer Chemoprevention, Zhengzhou, China.
Liang Y; Cancer Institute, Fudan University Shanghai Cancer Center; Collaborative Innovation Center of Cancer Medicine; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Li P; College of Basic Medical Sciences, Zhengzhou University; Collaborative Innovation Center of Henan Province for Cancer Chemoprevention, Zhengzhou, China.
Chen X; College of Basic Medical Sciences, Zhengzhou University; Collaborative Innovation Center of Henan Province for Cancer Chemoprevention, Zhengzhou, China.
Zhang J; College of Basic Medical Sciences, Zhengzhou University; Collaborative Innovation Center of Henan Province for Cancer Chemoprevention, Zhengzhou, China.
Ma Y; College of Basic Medical Sciences, Zhengzhou University; Collaborative Innovation Center of Henan Province for Cancer Chemoprevention, Zhengzhou, China.
Hao Q; College of Basic Medical Sciences, Zhengzhou University; Collaborative Innovation Center of Henan Province for Cancer Chemoprevention, Zhengzhou, China.
Wang J; Department of Pathology, Linzhou Cancer Hospital, Linzhou, China.
Zhang P; Cancer Institute, Fudan University Shanghai Cancer Center; Collaborative Innovation Center of Cancer Medicine; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China.
Zhang Y; Department of Clinical Laboratory, Huadong Hospital; Shanghai Key Laboratory of Clinical Geriatric Medicine; Research Center on Aging and Medicine, Fudan University, Shanghai, China.
Zhao H; Department of Clinical Laboratory, Huadong Hospital; Shanghai Key Laboratory of Clinical Geriatric Medicine; Research Center on Aging and Medicine, Fudan University, Shanghai, China.
Yang S; College of Basic Medical Sciences, Zhengzhou University; Collaborative Innovation Center of Henan Province for Cancer Chemoprevention, Zhengzhou, China.
Yu J; College of Pharmacy, Seoul National University, Seoul, Korea.
Jeong LS; College of Pharmacy, Seoul National University, Seoul, Korea.
Qi H; AntiCancer Biotech (Beijing) Co., Ltd., Beijing, China.
Yang M; AntiCancer Biotech (Beijing) Co., Ltd., Beijing, China. Department of Surgery, University of California, San Diego, California. AntiCancer, Inc., San Diego, California.
Hoffman RM; AntiCancer Biotech (Beijing) Co., Ltd., Beijing, China. Department of Surgery, University of California, San Diego, California. AntiCancer, Inc., San Diego, California.
Dong Z; College of Basic Medical Sciences, Zhengzhou University; Collaborative Innovation Center of Henan Province for Cancer Chemoprevention, Zhengzhou, China. .
Jia L; Cancer Institute, Fudan University Shanghai Cancer Center; Collaborative Innovation Center of Cancer Medicine; Department of Oncology, Shanghai Medical College, Fudan University, Shanghai, China. .
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Źródło :
Clinical cancer research : an official journal of the American Association for Cancer Research [Clin Cancer Res] 2016 Aug 15; Vol. 22 (16), pp. 4145-57. Date of Electronic Publication: 2016 Mar 16.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis*/drug effects
Apoptosis*/genetics
Signal Transduction*/drug effects
Activating Transcription Factor 4/*metabolism
Esophageal Neoplasms/*metabolism
Receptors, TNF-Related Apoptosis-Inducing Ligand/*metabolism
Transcription Factor CHOP/*metabolism
Animals ; BH3 Interacting Domain Death Agonist Protein/metabolism ; Biomarkers ; Caspase 8/metabolism ; Cell Line, Tumor ; Cyclopentanes/pharmacology ; Disease Models, Animal ; Esophageal Neoplasms/drug therapy ; Esophageal Neoplasms/mortality ; Esophageal Neoplasms/pathology ; Gene Silencing ; Humans ; Mice ; Models, Biological ; Prognosis ; Pyrimidines/pharmacology ; RNA Interference ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł :
ERβ up-regulation was involved in silibinin-induced growth inhibition of human breast cancer MCF-7 cells.
Autorzy :
Zheng N; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Liu L; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Liu W; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Zhang P; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Huang H; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Zang L; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Hayashi T; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Tashiro S; Institute for Clinical and Biomedical Sciences, Kyoto 603-8072, Japan.
Onodera S; Department of Clinical and Biomedical Sciences, Showa Pharmaceutical University, Tokyo 194-8543, Japan.
Xia M; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China. Electronic address: .
Ikejima T; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China. Electronic address: .
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Źródło :
Archives of biochemistry and biophysics [Arch Biochem Biophys] 2016 Feb 01; Vol. 591, pp. 141-9. Date of Electronic Publication: 2016 Jan 06.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Cell Proliferation/*drug effects
Estrogen Receptor beta/*metabolism
Mitochondria/*metabolism
Silymarin/*administration & dosage
Up-Regulation/*drug effects
Humans ; MCF-7 Cells ; Mitochondria/drug effects ; Silybin
Czasopismo naukowe
Tytuł :
Ginsenoside Rg3 induces apoptosis in human multiple myeloma cells via the activation of Bcl-2-associated X protein.
Autorzy :
Luo Y; Department of Hematology, Second Affiliated Hospital of Chongqing Medical University, Chongqing 404000, P.R. China.
Zhang P; Department of Hematology, Second Affiliated Hospital of Chongqing Medical University, Chongqing 404000, P.R. China.
Zeng HQ; Department of Hematology, Second Affiliated Hospital of Chongqing Medical University, Chongqing 404000, P.R. China.
Lou SF; Department of Hematology, Second Affiliated Hospital of Chongqing Medical University, Chongqing 404000, P.R. China.
Wang DX; Department of Hematology, Second Affiliated Hospital of Chongqing Medical University, Chongqing 404000, P.R. China.
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Źródło :
Molecular medicine reports [Mol Med Rep] 2015 Sep; Vol. 12 (3), pp. 3557-3562. Date of Electronic Publication: 2015 May 19.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents, Phytogenic/*pharmacology
Apoptosis/*drug effects
Ginsenosides/*pharmacology
Multiple Myeloma/*drug therapy
bcl-2-Associated X Protein/*metabolism
Antineoplastic Agents, Phytogenic/chemistry ; Cell Line, Tumor ; Ginsenosides/chemistry ; Humans ; Multiple Myeloma/metabolism ; Multiple Myeloma/pathology ; Panax/chemistry
Czasopismo naukowe
Tytuł :
ERα down-regulation plays a key role in silibinin-induced autophagy and apoptosis in human breast cancer MCF-7 cells.
Autorzy :
Zheng N; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Zhang P; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Huang H; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Liu W; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Hayashi T; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Zang L; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Zhang Y; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Liu L; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Xia M; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China.
Tashiro S; Institute for Clinical and Biomedical Sciences, Kyoto 603-8072, Japan.
Onodera S; Department of Clinical and Biomedical Sciences, Showa Pharmaceutical University, Tokyo 194-8543, Japan.
Ikejima T; China-Japan Research Institute of Medical and Pharmaceutical Sciences, Shenyang Pharmaceutical University, Shenyang 110016, China. Electronic address: .
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Źródło :
Journal of pharmacological sciences [J Pharmacol Sci] 2015 Jul; Vol. 128 (3), pp. 97-107. Date of Electronic Publication: 2015 May 12.
Typ publikacji :
Journal Article
MeSH Terms :
Apoptosis/*drug effects
Apoptosis/*genetics
Autophagy/*drug effects
Autophagy/*genetics
Breast Neoplasms/*genetics
Breast Neoplasms/*pathology
Down-Regulation/*drug effects
Down-Regulation/*genetics
Estrogen Receptor alpha/*physiology
Gene Expression/*drug effects
Gene Expression/*genetics
Silymarin/*pharmacology
Female ; Humans ; MAP Kinase Signaling System/genetics ; MAP Kinase Signaling System/physiology ; MCF-7 Cells ; Silybin ; TOR Serine-Threonine Kinases/genetics ; TOR Serine-Threonine Kinases/metabolism
Czasopismo naukowe
Tytuł :
[Study on effect of tetramethylpyrazine on proliferation and apoptosis of leukemic U937 cells and its mechanism].
Autorzy :
Wang XJ
Yang GC
Chen HX
Zhang P
Xu YH
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Źródło :
Zhongguo Zhong yao za zhi = Zhongguo zhongyao zazhi = China journal of Chinese materia medica [Zhongguo Zhong Yao Za Zhi] 2015 Jun; Vol. 40 (11), pp. 2186-90.
Typ publikacji :
English Abstract; Journal Article
MeSH Terms :
Apoptosis/*drug effects
Cell Proliferation/*drug effects
Leukemia/*drug therapy
Pyrazines/*pharmacology
Cell Cycle/drug effects ; Cyclin-Dependent Kinase 2/analysis ; Humans ; Proto-Oncogene Proteins c-bcl-2/analysis ; Pyrazines/therapeutic use ; U937 Cells
Czasopismo naukowe
Tytuł :
Nuclear envelope proteins Nesprin2 and LaminA regulate proliferation and apoptosis of vascular endothelial cells in response to shear stress.
Autorzy :
Han Y; Institute of Mechanobiology & Medical Engineering, School of Life Sciences & Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Wang L; Institute of Mechanobiology & Medical Engineering, School of Life Sciences & Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Yao QP; Institute of Mechanobiology & Medical Engineering, School of Life Sciences & Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Zhang P; Institute of Mechanobiology & Medical Engineering, School of Life Sciences & Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Liu B; Institute of Mechanobiology & Medical Engineering, School of Life Sciences & Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Wang GL; Institute of Mechanobiology & Medical Engineering, School of Life Sciences & Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Shen BR; Institute of Mechanobiology & Medical Engineering, School of Life Sciences & Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Cheng B; Department of Bioengineering, University of CA, San Diego, USA.
Wang Y; Institute of Mechanobiology & Medical Engineering, School of Life Sciences & Biotechnology, Shanghai Jiao Tong University, Shanghai, China; Department of Bioengineering, University of CA, San Diego, USA.
Jiang ZL; Institute of Mechanobiology & Medical Engineering, School of Life Sciences & Biotechnology, Shanghai Jiao Tong University, Shanghai, China.
Qi YX; Institute of Mechanobiology & Medical Engineering, School of Life Sciences & Biotechnology, Shanghai Jiao Tong University, Shanghai, China. Electronic address: .
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Źródło :
Biochimica et biophysica acta [Biochim Biophys Acta] 2015 May; Vol. 1853 (5), pp. 1165-73. Date of Electronic Publication: 2015 Feb 23.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis*
Shear Strength*
Stress, Mechanical*
Endothelial Cells/*metabolism
Lamin Type A/*metabolism
Nerve Tissue Proteins/*metabolism
Nuclear Envelope/*metabolism
Animals ; Cell Proliferation ; Cells, Cultured ; Endothelial Cells/cytology ; Endothelial Cells/ultrastructure ; Gene Regulatory Networks ; Models, Biological ; Phosphorylation ; RNA Interference ; Rats ; Transcription Factors/metabolism
Czasopismo naukowe
Tytuł :
Phenotypic characterization of craniofacial bone marrow stromal cells: unique properties of enhanced osteogenesis, cell recruitment, autophagy, and apoptosis resistance.
Autorzy :
Dong W; Department of Oral and Maxillofacial Surgery, School of Stomatology, Nanjing Medical University, 136 Hanzhong Road, Nanjing, 210029, Jiangsu Province, China.
Ge J
Zhang P
Fu Y
Zhang Z
Cheng J
Jiang H
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Źródło :
Cell and tissue research [Cell Tissue Res] 2014 Oct; Vol. 358 (1), pp. 165-75. Date of Electronic Publication: 2014 Jun 14.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis*
Autophagy*
Osteogenesis*
Bone Marrow Cells/*metabolism
Mandible/*metabolism
Animals ; Bone Marrow Cells/cytology ; Cell Hypoxia ; Mandible/cytology ; Nanog Homeobox Protein ; Organic Anion Transporters, Sodium-Independent/metabolism ; Rats ; Rats, Sprague-Dawley ; Stromal Cells/cytology ; Stromal Cells/pathology ; Transcription Factors/metabolism
Czasopismo naukowe
Tytuł :
Inhibition of hedgehog signaling by GANT58 induces apoptosis and shows synergistic antitumor activity with AKT inhibitor in acute T cell leukemia cells.
Autorzy :
Hou X; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China. Electronic address: .
Chen X; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China.
Zhang P; Department of Neurosurgery, Qi Lu Hospital, Shandong University, Jinan, PR China.
Fan Y; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China.
Ma A; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China.
Pang T; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China.
Song Z; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China.
Jin Y; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China.
Hao W; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China.
Liu F; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China.
Wang W; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China.
Wang Y; Department of Pediatrics, Provincial Hospital Affiliated to Shandong University, Shandong University, Jinan, PR China. Electronic address: .
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Źródło :
Biochimie [Biochimie] 2014 Jun; Vol. 101, pp. 50-9. Date of Electronic Publication: 2014 Jan 03.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Oxadiazoles/*pharmacology
Phosphorylcholine/*analogs & derivatives
Precursor T-Cell Lymphoblastic Leukemia-Lymphoma/*drug therapy
Pyridines/*pharmacology
Thiophenes/*pharmacology
Drug Screening Assays, Antitumor ; Drug Synergism ; Hedgehog Proteins/metabolism ; Humans ; Jurkat Cells ; MAP Kinase Signaling System ; Phosphorylcholine/pharmacology ; Proto-Oncogene Proteins c-akt/antagonists & inhibitors ; Transcription Factors/antagonists & inhibitors ; Transcription Factors/metabolism ; Veratrum Alkaloids/pharmacology ; Zinc Finger Protein GLI1
Czasopismo naukowe
Tytuł :
The contribution of Cdc2 in rotenone-induced G2/M arrest and caspase-3-dependent apoptosis.
Autorzy :
Wang H; Department of Neurology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, 1277 Jie Fang Road, Wuhan, 430022, Hubei, China.
Zhang Z
Huang J
Zhang P
Xiong N
Wang T
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Źródło :
Journal of molecular neuroscience : MN [J Mol Neurosci] 2014 May; Vol. 53 (1), pp. 31-40. Date of Electronic Publication: 2013 Dec 12.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis*
G2 Phase Cell Cycle Checkpoints*
Cyclin-Dependent Kinases/*metabolism
Rotenone/*pharmacology
CDC2 Protein Kinase ; Caspase 3/genetics ; Caspase 3/metabolism ; Cell Line, Tumor ; Cyclin-Dependent Kinases/genetics ; Humans ; Neurons/drug effects ; Neurons/metabolism
Czasopismo naukowe
Tytuł :
Mitochondria are the primary target in the induction of apoptosis by chiral ruthenium(II) polypyridyl complexes in cancer cells.
Autorzy :
Wang JQ; MOE Key Laboratory of Bioinorganic and Synthetic Chemistry, State Key Laboratory of Optoelectronic Materials and Technologies, School of Chemistry and Chemical Engineering, Sun Yat-Sen University, Guangzhou, 510275, People's Republic of China.
Zhang PY
Qian C
Hou XJ
Ji LN
Chao H
Pokaż więcej
Źródło :
Journal of biological inorganic chemistry : JBIC : a publication of the Society of Biological Inorganic Chemistry [J Biol Inorg Chem] 2014 Mar; Vol. 19 (3), pp. 335-48. Date of Electronic Publication: 2013 Nov 28.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Antineoplastic Agents/*administration & dosage
Apoptosis/*drug effects
Drug Delivery Systems/*methods
Mitochondria/*drug effects
Pyridines/*administration & dosage
Ruthenium/*administration & dosage
Antineoplastic Agents/chemistry ; Apoptosis/physiology ; Cell Line, Tumor ; HeLa Cells ; Hep G2 Cells ; Humans ; MCF-7 Cells ; Membrane Potential, Mitochondrial/drug effects ; Membrane Potential, Mitochondrial/physiology ; Mitochondria/pathology ; Pyridines/chemistry ; Ruthenium/chemistry
Czasopismo naukowe
Tytuł :
β-elemene induces caspase-dependent apoptosis in human glioma cells in vitro through the upregulation of Bax and Fas/ FasL and downregulation of Bcl-2.
Autorzy :
Li CL; Department of Neurosurgery, The Second Affiliated Hospital of Harbin Medical University, Harbin, China E-mail : , .
Chang L
Guo L
Zhao D
Liu HB
Wang QS
Zhang P
Du WZ
Liu X
Zhang HT
Liu Y
Zhang Y
Xie JH
Ming JG
Cui YQ
Sun Y
Zhang ZR
Jiang CL
Pokaż więcej
Źródło :
Asian Pacific journal of cancer prevention : APJCP [Asian Pac J Cancer Prev] 2014; Vol. 15 (23), pp. 10407-12.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Fas Ligand Protein/*drug effects
Glioma/*genetics
Proto-Oncogene Proteins c-bcl-2/*drug effects
RNA, Messenger/*drug effects
Sesquiterpenes/*pharmacology
bcl-2-Associated X Protein/*drug effects
fas Receptor/*drug effects
Apoptosis/genetics ; Blotting, Western ; Caspases/drug effects ; Caspases/genetics ; Caspases/metabolism ; Cell Line, Tumor ; Down-Regulation/drug effects ; Drug Screening Assays, Antitumor ; Fas Ligand Protein/genetics ; Fas Ligand Protein/metabolism ; Glioma/metabolism ; Humans ; In Vitro Techniques ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; RNA, Messenger/metabolism ; Reverse Transcriptase Polymerase Chain Reaction ; Up-Regulation/drug effects ; bcl-2-Associated X Protein/genetics ; bcl-2-Associated X Protein/metabolism ; fas Receptor/genetics ; fas Receptor/metabolism
Czasopismo naukowe
Tytuł :
Trichostatin A protects against cisplatin-induced ototoxicity by regulating expression of genes related to apoptosis and synaptic function.
Autorzy :
Wang P; Department of Otolaryngology-Head and Neck Surgery, First Hospital of Jilin University, Changchun 130021, China.
Zhang P
Huang J
Li M
Chen X
Pokaż więcej
Źródło :
Neurotoxicology [Neurotoxicology] 2013 Jul; Vol. 37, pp. 51-62. Date of Electronic Publication: 2013 Apr 01.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Apoptosis/*drug effects
Basilar Membrane/*drug effects
Cisplatin/*toxicity
Histone Deacetylase Inhibitors/*pharmacology
Hydroxamic Acids/*pharmacology
Neuronal Plasticity/*drug effects
Synaptic Transmission/*drug effects
Animals ; Animals, Newborn ; Apoptosis/genetics ; Basilar Membrane/metabolism ; Basilar Membrane/pathology ; Cytoprotection ; Gene Expression Profiling/methods ; Gene Expression Regulation ; Hair Cells, Auditory/drug effects ; Hair Cells, Auditory/metabolism ; Hair Cells, Auditory/pathology ; Neuronal Plasticity/genetics ; Oligonucleotide Array Sequence Analysis ; Organ Culture Techniques ; RNA, Messenger/metabolism ; Rats ; Rats, Wistar ; Real-Time Polymerase Chain Reaction ; Signal Transduction/drug effects ; Signal Transduction/genetics ; Spiral Ganglion/drug effects ; Spiral Ganglion/metabolism ; Spiral Ganglion/pathology ; Synaptic Transmission/genetics ; Time Factors
Czasopismo naukowe

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