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Wyszukujesz frazę ""Zhao, M."" wg kryterium: Autor


Tytuł:
Design, Synthesis of Hydrogen Peroxide Response AIE Fluorescence Probes Based on Imidazo [1,2-a] Pyridine.
Autorzy:
Tong L; Department of Veterinary Medicine, Shanxi Agricultural University, Jinzhong 030801, China.
Yang Y; Department of Veterinary Medicine, Shanxi Agricultural University, Jinzhong 030801, China.
Zhang L; Department of Veterinary Medicine, Shanxi Agricultural University, Jinzhong 030801, China.
Tao J; Department of Mining Engineering, Shanxi Institute of Technology, Yangquan 045000, China.
Sun B; Department of Mining Engineering, Shanxi Institute of Technology, Yangquan 045000, China.
Song C; Department of Veterinary Medicine, Shanxi Agricultural University, Jinzhong 030801, China.
Qi M; Department of Veterinary Medicine, Shanxi Agricultural University, Jinzhong 030801, China.
Yang F; Department of Mining Engineering, Shanxi Institute of Technology, Yangquan 045000, China.
Zhao M; Department of Mining Engineering, Shanxi Institute of Technology, Yangquan 045000, China.; Yangquan Technology Innovation Center of Carbon Dioxide Capture, Utilization and Storage, Shanxi Institute of Technology, Yangquan 045000, China.
Jiang J; Department of Veterinary Medicine, Shanxi Agricultural University, Jinzhong 030801, China.; Department of Mining Engineering, Shanxi Institute of Technology, Yangquan 045000, China.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2024 Feb 16; Vol. 29 (4). Date of Electronic Publication: 2024 Feb 16.
Typ publikacji:
Journal Article
MeSH Terms:
Hydrogen Peroxide*
Oxidative Stress*
Humans ; Fluorescence ; Reactive Oxygen Species ; Fluorescent Dyes ; Pyridines
Czasopismo naukowe
Tytuł:
Antioxidant Effects and Cytoprotective Potentials of Herbal Tea against H 2 O 2 -Induced Oxidative Damage by Activating Heme Oxygenase1 Pathway.
Autorzy:
Zhang KX; Center for Disease Control and Prevention of Guangdong Province, Guangzhou 511430, China.; Department of Medical Statistics and Epidemiology, Guangdong Provincial Key Laboratory of Food, Nutrition and Health, Guangdong Engineering Technology Research Center of Nutrition Translation, School of Public Health, Sun Yat-Sen University, Guangzhou 510000, China.
Tan JB; Center for Disease Control and Prevention of Guangdong Province, Guangzhou 511430, China.
Xie CL; School of Pharmaceutical Science (Shenzhen), Sun Yat-Sen University, Guangzhou 510000, China.
Zheng RB; Guangzhou Wanlaoji Pharmaceutical Co., Ltd., Guangzhou 510403, China.
Huang XD; Guangzhou Wanlaoji Pharmaceutical Co., Ltd., Guangzhou 510403, China.
Zhang MM; Center for Disease Control and Prevention of Guangdong Province, Guangzhou 511430, China.
Zhao M; Center for Disease Control and Prevention of Guangdong Province, Guangzhou 511430, China.
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Źródło:
BioMed research international [Biomed Res Int] 2020 Jul 05; Vol. 2020, pp. 7187946. Date of Electronic Publication: 2020 Jul 05 (Print Publication: 2020).
Typ publikacji:
Journal Article
MeSH Terms:
Teas, Herbal*
Antioxidants/*pharmacology
Cytoprotection/*drug effects
Heme Oxygenase-1/*metabolism
Hydrogen Peroxide/*toxicity
Oxidative Stress/*drug effects
Biphenyl Compounds/chemistry ; Cell Cycle/drug effects ; Cell Line ; Cell Survival/drug effects ; Cytoprotection/genetics ; Gene Expression Regulation/drug effects ; Humans ; Hydroxybenzoates/analysis ; Oxidative Stress/genetics ; Picrates/chemistry ; Protective Agents/pharmacology ; Transcriptome/genetics
Czasopismo naukowe
Tytuł:
Exhaustive-exercise-induced oxidative stress alteration of erythrocyte oxygen release capacity.
Autorzy:
Xiong Y; a Key Laboratory of Biorheological Science and Technology (Chongqing University), Ministry of Education, Chongqing 400030, P.R. China.; b Department of Anatomy, School of Basic Medicine, Binzhou Medical University, Yantai, P.R. China.
Xiong Y; c Department of Pathophysiology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences (CAMS), School of Basic Medicine, Peking Union Medical College, Beijing (PUMC), P.R. China.
Wang Y; b Department of Anatomy, School of Basic Medicine, Binzhou Medical University, Yantai, P.R. China.
Zhao Y; a Key Laboratory of Biorheological Science and Technology (Chongqing University), Ministry of Education, Chongqing 400030, P.R. China.
Li Y; a Key Laboratory of Biorheological Science and Technology (Chongqing University), Ministry of Education, Chongqing 400030, P.R. China.
Ren Y; a Key Laboratory of Biorheological Science and Technology (Chongqing University), Ministry of Education, Chongqing 400030, P.R. China.
Wang R; a Key Laboratory of Biorheological Science and Technology (Chongqing University), Ministry of Education, Chongqing 400030, P.R. China.
Zhao M; b Department of Anatomy, School of Basic Medicine, Binzhou Medical University, Yantai, P.R. China.
Hao Y; b Department of Anatomy, School of Basic Medicine, Binzhou Medical University, Yantai, P.R. China.
Liu H; b Department of Anatomy, School of Basic Medicine, Binzhou Medical University, Yantai, P.R. China.
Wang X; a Key Laboratory of Biorheological Science and Technology (Chongqing University), Ministry of Education, Chongqing 400030, P.R. China.
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Źródło:
Canadian journal of physiology and pharmacology [Can J Physiol Pharmacol] 2018 Sep; Vol. 96 (9), pp. 953-962. Date of Electronic Publication: 2018 May 24.
Typ publikacji:
Journal Article
MeSH Terms:
Oxidative Stress*
Physical Conditioning, Animal*
Erythrocytes/*metabolism
Oxygen/*metabolism
Animals ; Carbonic Anhydrase II/metabolism ; Hemoglobins/metabolism ; Kinetics ; Male ; Membrane Proteins/metabolism ; Phosphorylation ; Rats ; Rats, Wistar
Czasopismo naukowe
Tytuł:
Selenocystine inhibits JEG-3 cell growth in vitro and in vivo by triggering oxidative damage-mediated S-phase arrest and apoptosis.
Autorzy:
Zhao M; Department of Orthopaedics, Taishan Hospital Affiliated to Taishan Medical University, Taian, Shandong, China.
Hou Y; Department of Biochemistry, School of Basic Medicine, Taishan Medical University, Taian, Shandong, China.
Fu X; Department of Biochemistry, School of Basic Medicine, Taishan Medical University, Taian, Shandong, China.
Li D; Department of Biochemistry, School of Basic Medicine, Taishan Medical University, Taian, Shandong, China.
Sun J; Department of Biochemistry, School of Basic Medicine, Taishan Medical University, Taian, Shandong, China.
Fu X; Department of Biochemistry, School of Basic Medicine, Taishan Medical University, Taian, Shandong, China.
Wei Z; Department of Oncology, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, Shandong, China.
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Źródło:
Journal of cancer research and therapeutics [J Cancer Res Ther] 2018; Vol. 14 (7), pp. 1540-1548.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*drug effects
Cystine/*analogs & derivatives
Organoselenium Compounds/*pharmacology
Oxidative Stress/*drug effects
S Phase Cell Cycle Checkpoints/*drug effects
Animals ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Choriocarcinoma/metabolism ; Cystine/pharmacology ; Disease Models, Animal ; Female ; Humans ; Mice ; Reactive Oxygen Species/metabolism ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł:
Oxidative Stress in Hypoxic-Ischemic Encephalopathy: Molecular Mechanisms and Therapeutic Strategies.
Autorzy:
Zhao M; Department of Pediatrics, the Third Xiangya Hospital, Central South University, Changsha 410006, China. .
Zhu P; Guangdong Cardiovascular Institute, Guangdong General Hospital, Guangdong Academy of Medical Sciences, Guangzhou 510100, China. .
Fujino M; National Research Institute for Child Health and Development, 2-10-1 Okura, Setagaya-ku, Tokyo 157-8535, Japan. .; National Institute of Infectious Diseases, 1-23-1 Toyama, Shinjuku-ku, Tokyo 162-8640, Japan. .
Zhuang J; Guangdong Cardiovascular Institute, Guangdong General Hospital, Guangdong Academy of Medical Sciences, Guangzhou 510100, China. .
Guo H; Guangdong Cardiovascular Institute, Guangdong General Hospital, Guangdong Academy of Medical Sciences, Guangzhou 510100, China. .
Sheikh I; Department of Pediatrics, the Third Xiangya Hospital, Central South University, Changsha 410006, China. Dr_.
Zhao L; Department of Pediatrics, the Third Xiangya Hospital, Central South University, Changsha 410006, China. .
Li XK; Department of Pediatrics, the Third Xiangya Hospital, Central South University, Changsha 410006, China. .; National Research Institute for Child Health and Development, 2-10-1 Okura, Setagaya-ku, Tokyo 157-8535, Japan. .
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2016 Dec 10; Vol. 17 (12). Date of Electronic Publication: 2016 Dec 10.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Oxidative Stress*
Hypoxia-Ischemia, Brain/*pathology
Hypoxia-Ischemia, Brain/*therapy
Animals ; Antioxidants/metabolism ; Biomarkers/metabolism ; Humans ; Hypoxia-Ischemia, Brain/metabolism ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł:
Iron overload by Superparamagnetic Iron Oxide Nanoparticles is a High Risk Factor in Cirrhosis by a Systems Toxicology Assessment.
Autorzy:
Wei Y; Department of Nanomedicine &Biopharmaceuticals, National Engineering Research Center for Nanomedicine, Huazhong University of Science and Technology, Wuhan, Hubei, China.
Zhao M; Department of Nanomedicine &Biopharmaceuticals, National Engineering Research Center for Nanomedicine, Huazhong University of Science and Technology, Wuhan, Hubei, China.
Yang F; Department of Nanomedicine &Biopharmaceuticals, National Engineering Research Center for Nanomedicine, Huazhong University of Science and Technology, Wuhan, Hubei, China.
Mao Y; Department of Nanomedicine &Biopharmaceuticals, National Engineering Research Center for Nanomedicine, Huazhong University of Science and Technology, Wuhan, Hubei, China.
Xie H; Division of Viral Products, Office of Vaccines Research and Review, Center for Biologics Evaluation and Research, United States Food and Drug Administration, Silver Spring, Maryland, United States of America.
Zhou Q; Department of Nanomedicine &Biopharmaceuticals, National Engineering Research Center for Nanomedicine, Huazhong University of Science and Technology, Wuhan, Hubei, China.
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Źródło:
Scientific reports [Sci Rep] 2016 Jun 30; Vol. 6, pp. 29110. Date of Electronic Publication: 2016 Jun 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Liver/*drug effects
Liver Cirrhosis/*physiopathology
Magnetite Nanoparticles/*adverse effects
Oxidative Stress/*drug effects
Animals ; Contrast Media/adverse effects ; Contrast Media/therapeutic use ; Disease Models, Animal ; Humans ; Iron Overload/chemically induced ; Iron Overload/physiopathology ; Liver/physiopathology ; Liver Cirrhosis/chemically induced ; Magnetic Resonance Imaging ; Magnetite Nanoparticles/therapeutic use ; Mice ; Theranostic Nanomedicine ; Tissue Distribution/drug effects
Czasopismo naukowe
Tytuł:
p, p'-Dichlorodiphenyldichloroethylene induces colorectal adenocarcinoma cell proliferation through oxidative stress.
Autorzy:
Song L; Institute of Biotechnology, Key Laboratory of Chemical Biology and Molecular Engineering of National Ministry of Education, Shanxi University, Taiyuan, China; MOE Key Lab of Environmental Remediation and Ecosystem Health, College of Environmental and Resource Sciences, Zhejiang University, Hangzhou, China.
Liu J; Institute of Biotechnology, Key Laboratory of Chemical Biology and Molecular Engineering of National Ministry of Education, Shanxi University, Taiyuan, China.
Jin X; Institute of Biotechnology, Key Laboratory of Chemical Biology and Molecular Engineering of National Ministry of Education, Shanxi University, Taiyuan, China.
Li Z; Institute of Biotechnology, Key Laboratory of Chemical Biology and Molecular Engineering of National Ministry of Education, Shanxi University, Taiyuan, China; College of Life Science, Zhejiang Chinese Medical University, Hangzhou, China.
Zhao M; Research Center of Environmental Science, Zhejiang University of Technology, Hangzhou, China.
Liu W; MOE Key Lab of Environmental Remediation and Ecosystem Health, College of Environmental and Resource Sciences, Zhejiang University, Hangzhou, China.
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Źródło:
PloS one [PLoS One] 2014 Nov 11; Vol. 9 (11), pp. e112700. Date of Electronic Publication: 2014 Nov 11 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adenocarcinoma/*pathology
Colorectal Neoplasms/*pathology
Dichlorodiphenyl Dichloroethylene/*toxicity
Oxidative Stress/*drug effects
Adenocarcinoma/chemically induced ; Adenocarcinoma/metabolism ; Catalase/metabolism ; Cell Line, Tumor/drug effects ; Cell Proliferation/drug effects ; Colorectal Neoplasms/chemically induced ; Colorectal Neoplasms/metabolism ; Dose-Response Relationship, Drug ; Humans ; Reactive Oxygen Species/metabolism ; Superoxide Dismutase/metabolism ; Transcription Factors/genetics ; Transcription Factors/metabolism ; Wnt Signaling Pathway/drug effects ; Zinc Finger Protein GLI1 ; beta Catenin/genetics ; beta Catenin/metabolism
Czasopismo naukowe
Tytuł:
Aerobic interval training protects against myocardial infarction-induced oxidative injury by enhancing antioxidase system and mitochondrial biosynthesis.
Autorzy:
Jiang HK; Department of Pharmacology, College of Medicine, Xi'an Jiaotong University, Xi'an, China; Department of Physical Education, Nan Yang Institute of Technology, Nan Yang, China.
Miao Y
Wang YH
Zhao M
Feng ZH
Yu XJ
Liu JK
Zang WJ
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Źródło:
Clinical and experimental pharmacology & physiology [Clin Exp Pharmacol Physiol] 2014 Mar; Vol. 41 (3), pp. 192-201.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antioxidants/*metabolism
Mitochondria/*metabolism
Mitochondrial Turnover/*physiology
Myocardial Infarction/*physiopathology
Oxidative Stress/*physiology
Physical Conditioning, Animal/*physiology
AMP-Activated Protein Kinases/metabolism ; Animals ; Male ; Mitochondria/physiology ; Myocardial Infarction/metabolism ; NF-E2-Related Factor 2/metabolism ; Oxidation-Reduction ; Phosphatidylinositol 3-Kinase/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Rats ; Rats, Sprague-Dawley ; Sirtuin 3/metabolism
Czasopismo naukowe
Tytuł:
Free iron catalyzes oxidative damage to hematopoietic cells/mesenchymal stem cells in vitro and suppresses hematopoiesis in iron overload patients.
Autorzy:
Lu W; The First Central Clinical College of Tianjin Medical University, Tianjin First Central Hospital, Tianjin, China.
Zhao M; The First Central Clinical College of Tianjin Medical University, Tianjin First Central Hospital, Tianjin, China.
Rajbhandary S; The First Central Clinical College of Tianjin Medical University, Tianjin First Central Hospital, Tianjin, China.
Xie F; The First Central Clinical College of Tianjin Medical University, Tianjin First Central Hospital, Tianjin, China.
Chai X; The First Central Clinical College of Tianjin Medical University, Tianjin First Central Hospital, Tianjin, China.
Mu J; The First Central Clinical College of Tianjin Medical University, Tianjin First Central Hospital, Tianjin, China.
Meng J; The First Central Clinical College of Tianjin Medical University, Tianjin First Central Hospital, Tianjin, China.
Liu Y; Chinese Academy of Medical Sciences, Tianjin, China.
Jiang Y; The First Central Clinical College of Tianjin Medical University, Tianjin First Central Hospital, Tianjin, China.
Xu X; Key Lab for Critical Care Medicine of the Ministry of Health, Tianjin First Central Hospital, Tianjin, China.
Meng A; Institute of Radiation Medicine, Chinese Academy of Medical Science and Peking Union Medical Collage, Tianjin, China.
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Źródło:
European journal of haematology [Eur J Haematol] 2013 Sep; Vol. 91 (3), pp. 249-261. Date of Electronic Publication: 2013 Jul 15.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Hematopoiesis*
Oxidative Stress*
Hematopoietic Stem Cells/*metabolism
Iron/*metabolism
Iron Overload/*metabolism
Mesenchymal Stem Cells/*metabolism
Adult ; Aged ; Apoptosis ; Bone Marrow Cells/cytology ; Bone Marrow Cells/metabolism ; Cell Cycle ; Cell Proliferation ; Colony-Forming Units Assay ; Deferoxamine/therapeutic use ; Female ; G1 Phase Cell Cycle Checkpoints ; Hematopoietic Stem Cells/cytology ; Humans ; Iron Overload/drug therapy ; Male ; Middle Aged ; Signal Transduction
Czasopismo naukowe
Tytuł:
Protective effect of Homer 1a against hydrogen peroxide-induced oxidative stress in PC12 cells.
Autorzy:
Luo P; Department of Neurosurgery, Xijing Hospital, Fourth Military Medical University, Xi'an, PR China.
Chen T
Zhao Y
Xu H
Huo K
Zhao M
Yang Y
Fei Z
Pokaż więcej
Źródło:
Free radical research [Free Radic Res] 2012 Jun; Vol. 46 (6), pp. 766-76. Date of Electronic Publication: 2012 Apr 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Carrier Proteins/*metabolism
Hydrogen Peroxide/*pharmacology
Oxidative Stress/*drug effects
Animals ; Apoptosis/drug effects ; Carrier Proteins/biosynthesis ; Carrier Proteins/genetics ; Cell Survival/drug effects ; Down-Regulation/drug effects ; Homer Scaffolding Proteins ; Lipid Peroxidation/drug effects ; Membrane Potential, Mitochondrial/drug effects ; Mitochondria/drug effects ; Mitochondria/metabolism ; PC12 Cells ; Rats ; Transfection
Czasopismo naukowe

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