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Wyszukujesz frazę ""Zhou, P."" wg kryterium: Autor


Tytuł:
MiR-206 improves intervertebral disk degeneration by targeting GJA1.
Autorzy:
Zhou P; Department of Orthopedics and Joints, The Third Affiliated Hospital of Soochow University, No. 185, Juqian Street, Tianning District, Changzhou City, 213000, Jiangsu Province, China.
Xu P; Department of Orthopedics and Joints, The Third Affiliated Hospital of Soochow University, No. 185, Juqian Street, Tianning District, Changzhou City, 213000, Jiangsu Province, China.
Yu W; Department of Orthopedics and Joints, The Third Affiliated Hospital of Soochow University, No. 185, Juqian Street, Tianning District, Changzhou City, 213000, Jiangsu Province, China.
Li H; Department of Orthopedics and Joints, The Third Affiliated Hospital of Soochow University, No. 185, Juqian Street, Tianning District, Changzhou City, 213000, Jiangsu Province, China. .
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Źródło:
Journal of orthopaedic surgery and research [J Orthop Surg Res] 2022 Mar 12; Vol. 17 (1), pp. 157. Date of Electronic Publication: 2022 Mar 12.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis/*genetics
Connexin 43/*genetics
Intervertebral Disc Degeneration/*genetics
MicroRNAs/*genetics
Nucleus Pulposus/*metabolism
Animals ; Intervertebral Disc Degeneration/diagnosis ; Intervertebral Disc Degeneration/pathology ; Lipopolysaccharides/pharmacology ; MicroRNAs/metabolism ; Rats
Czasopismo naukowe
Tytuł:
Matrine derivate MASM protects murine MC3T3-E1 osteoblastic cells against dexamethasone-induced apoptosis via the regulation of USP14/p53.
Autorzy:
Zhou P; Department of Emergency, Changhai Hospital, Naval Military Medical University , Shanghai , China.; Department of Orthopedics, Changhai Hospital, Naval Military Medical University , Shanghai , China.
Xia D; Department of Emergency, Changhai Hospital, Naval Military Medical University , Shanghai , China.
Wang Y; Department of Emergency, Changhai Hospital, Naval Military Medical University , Shanghai , China.; Department of Orthopedics, Changhai Hospital, Naval Military Medical University , Shanghai , China.
Lv H; Department of Logistics, The Nine Nine Zero Hospital of the People's Liberation Army , Henan , China.
Wang Z; Department of Nutrition, Armed Police Henan Corps Hospital , Henan , China.
Xing M; College of Mechanical Engineering, University of Manitoba , Winnipeg , Canada.
Zhao Q; Department of Organic Chemistry, School of Pharmcy, Naval Military Medical University , Shanghai , China.
Xu S; Department of Emergency, Changhai Hospital, Naval Military Medical University , Shanghai , China.; Department of Orthopedics, Changhai Hospital, Naval Military Medical University , Shanghai , China.
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Źródło:
Artificial cells, nanomedicine, and biotechnology [Artif Cells Nanomed Biotechnol] 2019 Dec; Vol. 47 (1), pp. 3720-3728.
Typ publikacji:
Journal Article
MeSH Terms:
Alkaloids/*pharmacology
Apoptosis/*drug effects
Dexamethasone/*adverse effects
Osteoblasts/*cytology
Osteoblasts/*drug effects
Quinolizines/*pharmacology
Tumor Suppressor Protein p53/*metabolism
Ubiquitin Thiolesterase/*metabolism
3T3 Cells ; Animals ; Cytoprotection/drug effects ; Mice ; Osteoblasts/metabolism ; Ubiquitination/drug effects ; Up-Regulation/drug effects ; Matrines
Czasopismo naukowe
Tytuł:
Spleen tyrosine kinase promotes NLR family pyrin domain containing 3 inflammasome‑mediated IL‑1β secretion via c‑Jun N‑terminal kinase activation and cell apoptosis during diabetic nephropathy.
Autorzy:
Qiao Y; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Tian X; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Men L; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Li S; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Chen Y; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Xue M; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Hu Y; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Zhou P; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Long G; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Shi Y; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Liu R; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Liu Y; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Qi Z; Department of Histology and Embryology, School of Medicine, Nankai University, Tianjin 300071, P.R. China.
Cui Y; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
Shen Y; School of Medical Laboratory, Tianjin Medical University, Tianjin 300203, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2018 Aug; Vol. 18 (2), pp. 1995-2008. Date of Electronic Publication: 2018 Jun 14.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Diabetes Mellitus, Experimental/*metabolism
Diabetic Nephropathies/*metabolism
Inflammasomes/*metabolism
Interleukin-1beta/*metabolism
JNK Mitogen-Activated Protein Kinases/*metabolism
NLR Family, Pyrin Domain-Containing 3 Protein/*metabolism
Syk Kinase/*metabolism
Animals ; Diabetes Mellitus, Experimental/pathology ; Diabetic Nephropathies/pathology ; Male ; Rats ; Rats, Sprague-Dawley
Czasopismo naukowe
Tytuł:
YAP knockdown inhibits proliferation and induces apoptosis of human prostate cancer DU145 cells.
Autorzy:
Jin X; Department of Urology, The Affiliated Taizhou People's Hospital of Nantong University, Taizhou, Jiangsu 225300, P.R. China.
Zhao W; Department of Urology, The Affiliated Taizhou People's Hospital of Nantong University, Taizhou, Jiangsu 225300, P.R. China.
Zhou P; Department of Urology, The Affiliated Taizhou People's Hospital of Nantong University, Taizhou, Jiangsu 225300, P.R. China.
Niu T; Department of Urology, The Affiliated Taizhou People's Hospital of Nantong University, Taizhou, Jiangsu 225300, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2018 Mar; Vol. 17 (3), pp. 3783-3788. Date of Electronic Publication: 2017 Dec 27.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Expression Regulation, Neoplastic*
Adaptor Proteins, Signal Transducing/*genetics
Apoptosis/*genetics
Epithelial Cells/*metabolism
Phosphoproteins/*genetics
Prostate/*metabolism
Adaptor Proteins, Signal Transducing/antagonists & inhibitors ; Adaptor Proteins, Signal Transducing/metabolism ; Caspase 3/genetics ; Caspase 3/metabolism ; Cell Line ; Cell Line, Tumor ; Cell Proliferation ; Connective Tissue Growth Factor/genetics ; Connective Tissue Growth Factor/metabolism ; Cysteine-Rich Protein 61/genetics ; Cysteine-Rich Protein 61/metabolism ; Epithelial Cells/pathology ; Humans ; Male ; Phosphoproteins/antagonists & inhibitors ; Phosphoproteins/metabolism ; Prostate/pathology ; Proto-Oncogene Proteins c-bcl-2/genetics ; Proto-Oncogene Proteins c-bcl-2/metabolism ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Signal Transduction ; Transcription Factors ; Transcription, Genetic ; YAP-Signaling Proteins ; bcl-2-Associated X Protein/genetics ; bcl-2-Associated X Protein/metabolism
Czasopismo naukowe
Tytuł:
cfa-miR-143 Promotes Apoptosis via the p53 Pathway in Canine Influenza Virus H3N2-Infected Cells.
Autorzy:
Zhou P; College of Veterinary Medicine, South China Agricultural University, Guangzhou 510642, China. .; Guangdong Provincial Key Laboratory of Prevention and Control for Severe Clinical Animal Diseases, Guangzhou 510642, China. .; Guangdong Provincial Pet Engineering Technology Research Center, Guangzhou 510642, China. .
Tu L; College of Veterinary Medicine, South China Agricultural University, Guangzhou 510642, China. .; Guangdong Provincial Key Laboratory of Prevention and Control for Severe Clinical Animal Diseases, Guangzhou 510642, China. .; Guangdong Provincial Pet Engineering Technology Research Center, Guangzhou 510642, China. .
Lin X; College of Veterinary Medicine, South China Agricultural University, Guangzhou 510642, China. .; Guangdong Provincial Key Laboratory of Prevention and Control for Severe Clinical Animal Diseases, Guangzhou 510642, China. .; Guangdong Provincial Pet Engineering Technology Research Center, Guangzhou 510642, China. .
Hao X; College of Veterinary Medicine, South China Agricultural University, Guangzhou 510642, China. .; Guangdong Provincial Key Laboratory of Prevention and Control for Severe Clinical Animal Diseases, Guangzhou 510642, China. .; Guangdong Provincial Pet Engineering Technology Research Center, Guangzhou 510642, China. .
Zheng Q; College of Veterinary Medicine, South China Agricultural University, Guangzhou 510642, China. qingxu_.; Guangdong Provincial Key Laboratory of Prevention and Control for Severe Clinical Animal Diseases, Guangzhou 510642, China. qingxu_.; Guangdong Provincial Pet Engineering Technology Research Center, Guangzhou 510642, China. qingxu_.
Zeng W; College of Veterinary Medicine, South China Agricultural University, Guangzhou 510642, China. .; Guangdong Provincial Key Laboratory of Prevention and Control for Severe Clinical Animal Diseases, Guangzhou 510642, China. .; Guangdong Provincial Pet Engineering Technology Research Center, Guangzhou 510642, China. .
Zhang X; College of Veterinary Medicine, South China Agricultural University, Guangzhou 510642, China. .; Guangdong Provincial Key Laboratory of Prevention and Control for Severe Clinical Animal Diseases, Guangzhou 510642, China. .; Guangdong Provincial Pet Engineering Technology Research Center, Guangzhou 510642, China. .
Zheng Y; College of Veterinary Medicine, South China Agricultural University, Guangzhou 510642, China. .; Guangdong Provincial Key Laboratory of Prevention and Control for Severe Clinical Animal Diseases, Guangzhou 510642, China. .; Guangdong Provincial Pet Engineering Technology Research Center, Guangzhou 510642, China. .
Wang L; College of Veterinary Medicine, South China Agricultural University, Guangzhou 510642, China. .; Guangdong Provincial Key Laboratory of Prevention and Control for Severe Clinical Animal Diseases, Guangzhou 510642, China. .; Guangdong Provincial Pet Engineering Technology Research Center, Guangzhou 510642, China. .
Li S; College of Veterinary Medicine, South China Agricultural University, Guangzhou 510642, China. .; Guangdong Provincial Key Laboratory of Prevention and Control for Severe Clinical Animal Diseases, Guangzhou 510642, China. .; Guangdong Provincial Pet Engineering Technology Research Center, Guangzhou 510642, China. .
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Źródło:
Viruses [Viruses] 2017 Nov 25; Vol. 9 (12). Date of Electronic Publication: 2017 Nov 25.
Typ publikacji:
Journal Article
MeSH Terms:
Apoptosis*
Dog Diseases/*virology
Influenza A Virus, H3N2 Subtype/*pathogenicity
Influenza, Human/*virology
MicroRNAs/*genetics
Orthomyxoviridae Infections/*veterinary
Animals ; Caspase 3/genetics ; Caspase 3/metabolism ; Cell Count ; Dogs ; Down-Regulation ; Genes, Reporter ; HEK293 Cells ; Humans ; Influenza A Virus, H3N2 Subtype/genetics ; Lung/virology ; Madin Darby Canine Kidney Cells ; Orthomyxoviridae Infections/virology ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/metabolism ; Up-Regulation
Czasopismo naukowe
Tytuł:
New insight into mitochondrial changes in vascular endothelial cells irradiated by gamma ray.
Autorzy:
Hu S; a Department of Cardiology , Chinese PLA General Hospital , Beijing , China.
Gao Y; b Beijing Institute of Radiation Medicine , Beijing , China.
Zhou H; a Department of Cardiology , Chinese PLA General Hospital , Beijing , China.
Kong F; b Beijing Institute of Radiation Medicine , Beijing , China.
Xiao F; b Beijing Institute of Radiation Medicine , Beijing , China.
Zhou P; b Beijing Institute of Radiation Medicine , Beijing , China.
Chen Y; a Department of Cardiology , Chinese PLA General Hospital , Beijing , China.
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Źródło:
International journal of radiation biology [Int J Radiat Biol] 2017 May; Vol. 93 (5), pp. 470-476. Date of Electronic Publication: 2017 Feb 08.
Typ publikacji:
Journal Article
MeSH Terms:
Gamma Rays*
Apoptosis/*physiology
Endothelial Cells/*physiology
Membrane Potential, Mitochondrial/*physiology
Mitochondria/*physiology
Mitochondrial Membrane Transport Proteins/*physiology
Apoptosis/radiation effects ; Cells, Cultured ; Dose-Response Relationship, Radiation ; Endothelial Cells/radiation effects ; Endothelial Cells/ultrastructure ; Humans ; Membrane Potential, Mitochondrial/radiation effects ; Mitochondria/radiation effects ; Mitochondria/ultrastructure ; Mitochondrial Membrane Transport Proteins/radiation effects ; Mitochondrial Membrane Transport Proteins/ultrastructure ; Mitochondrial Permeability Transition Pore ; Radiation Dosage ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Tytuł:
Effects of internalized gold nanoparticles with respect to cytotoxicity and invasion activity in lung cancer cells.
Autorzy:
Liu Z; Department of Geriatrics, the Second Affiliated Hospital, Nanjing Medical University, Jiangsu, China.
Wu Y; Department of Geriatrics, the Second Affiliated Hospital, Nanjing Medical University, Jiangsu, China.
Guo Z; Department of Geriatrics, the Second Affiliated Hospital, Nanjing Medical University, Jiangsu, China.
Liu Y; Department of Geriatrics, the Second Affiliated Hospital, Nanjing Medical University, Jiangsu, China.
Shen Y; Department of Geriatrics, the Second Affiliated Hospital, Nanjing Medical University, Jiangsu, China.
Zhou P; Department of Geriatrics, the Second Affiliated Hospital, Nanjing Medical University, Jiangsu, China.
Lu X; Department of Geriatrics, the Second Affiliated Hospital, Nanjing Medical University, Jiangsu, China.
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Źródło:
PloS one [PLoS One] 2014 Jun 05; Vol. 9 (6), pp. e99175. Date of Electronic Publication: 2014 Jun 05 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*drug effects
Endocytosis/*drug effects
Gold/*chemistry
Metal Nanoparticles/*toxicity
Cell Cycle Checkpoints/drug effects ; Cell Line, Tumor ; Cell Movement/drug effects ; Citric Acid/chemistry ; Humans ; Intercellular Adhesion Molecule-1/metabolism ; Lung Neoplasms ; Matrix Metalloproteinase 9/metabolism ; Metal Nanoparticles/chemistry ; Metal Nanoparticles/ultrastructure ; Particle Size
Czasopismo naukowe
Tytuł:
6-Shogaol induces apoptosis in human leukemia cells through a process involving caspase-mediated cleavage of eIF2α.
Autorzy:
Liu Q; State Key Laboratory of Natural Medicines, China Pharmaceutical University, Nanjing 210009, China. .
Peng YB
Zhou P
Qi LW
Zhang M
Gao N
Liu EH
Li P
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Źródło:
Molecular cancer [Mol Cancer] 2013 Nov 12; Vol. 12 (1), pp. 135. Date of Electronic Publication: 2013 Nov 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents, Phytogenic/*pharmacology
Apoptosis/*drug effects
Catechols/*pharmacology
Eukaryotic Initiation Factor-2/*metabolism
Leukemia/*drug therapy
Animals ; Antineoplastic Agents, Phytogenic/chemistry ; Caspase 3/metabolism ; Caspase 7/metabolism ; Catechols/chemistry ; Enzyme Activation ; Eukaryotic Initiation Factor-2/chemistry ; HL-60 Cells ; Humans ; Jurkat Cells ; Leukemia/enzymology ; Leukemia/pathology ; Leukocytes, Mononuclear/drug effects ; Leukocytes, Mononuclear/physiology ; Mice ; Mice, Inbred NOD ; Mice, Nude ; Mice, SCID ; Molecular Docking Simulation ; Phosphorylation ; Protein Processing, Post-Translational ; Proteolysis ; U937 Cells ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł:
The tumor suppressor Caliban regulates DNA damage-induced apoptosis through p53-dependent and -independent activity.
Autorzy:
Wang Y; CAS Key Laboratory for Biological Effects of Nanomaterials and Nanosafety, Institute of High Energy Physics, Chinese Academy of Sciences, Beijing, China.
Wang Z
Joshi BH
Puri RK
Stultz B
Yuan Q
Bai Y
Zhou P
Yuan Z
Hursh DA
Bi X
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Źródło:
Oncogene [Oncogene] 2013 Aug 15; Vol. 32 (33), pp. 3857-66. Date of Electronic Publication: 2012 Sep 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*physiology
DNA Damage/*physiology
Drosophila Proteins/*metabolism
Tumor Suppressor Proteins/*metabolism
Animals ; Carcinoma, Non-Small-Cell Lung/genetics ; Carcinoma, Non-Small-Cell Lung/metabolism ; Cell Line, Tumor ; Drosophila ; Drosophila Proteins/genetics ; Gene Knockout Techniques ; Humans ; Immunohistochemistry ; Mice ; Mice, Nude ; Microscopy, Confocal ; Microscopy, Electron, Scanning ; Neoplasms, Experimental/genetics ; Neoplasms, Experimental/metabolism ; Reverse Transcriptase Polymerase Chain Reaction ; Transplantation, Heterologous ; Tumor Suppressor Protein p53/metabolism ; Tumor Suppressor Proteins/genetics
Czasopismo naukowe
Tytuł:
6-Shogaol induces apoptosis in human hepatocellular carcinoma cells and exhibits anti-tumor activity in vivo through endoplasmic reticulum stress.
Autorzy:
Hu R; State Key Laboratory of Natural Medicines, China Pharmaceutical University, Nanjing, China.
Zhou P
Peng YB
Xu X
Ma J
Liu Q
Zhang L
Wen XD
Qi LW
Gao N
Li P
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Źródło:
PloS one [PLoS One] 2012; Vol. 7 (6), pp. e39664. Date of Electronic Publication: 2012 Jun 29.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents/*pharmacology
Apoptosis/*drug effects
Carcinoma, Hepatocellular/*pathology
Catechols/*pharmacology
Endoplasmic Reticulum Stress/*drug effects
Liver Neoplasms/*pathology
Animals ; Antineoplastic Agents/administration & dosage ; Antineoplastic Agents/chemistry ; Carcinoma, Hepatocellular/enzymology ; Catechols/administration & dosage ; Catechols/chemistry ; Cell Line, Tumor ; Cell Survival/drug effects ; Cinnamates/pharmacology ; Dose-Response Relationship, Drug ; Drug Screening Assays, Antitumor ; Drug Synergism ; Electrophoresis, Gel, Two-Dimensional ; Eukaryotic Initiation Factor-2/metabolism ; Humans ; Liver Neoplasms/enzymology ; Mice ; Neoplasm Proteins/metabolism ; Phosphorylation/drug effects ; RNA, Small Interfering/metabolism ; Thiourea/analogs & derivatives ; Thiourea/pharmacology ; Time Factors ; Transcription Factor CHOP/metabolism ; Unfolded Protein Response/drug effects ; Up-Regulation/drug effects ; Xenograft Model Antitumor Assays ; eIF-2 Kinase/metabolism
Czasopismo naukowe
Tytuł:
Design, synthesis, and in vitro biological evaluation of novel HDAC inhibitors bearing C-1 phenyl substituted tetrahydroisoquinoline Cap moiety as anti-tumor therapeutic agents
Autorzy:
Wang, Jie
Zhou, Chi
Li, Bo
Liu, Huaqing
Zhang, HuiAff1, IDs00044024032062_cor5
Liu, LeiAff1, IDs00044024032062_cor6
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Źródło:
Medicinal Chemistry Research. 33(4):687-702
Czasopismo naukowe
Tytuł:
A novel 2-iminobenzimidazole compound, XYA1353, displays in vitro and in vivo anti-myeloma activity via targeting NF-κB signaling
Autorzy:
Gao, Jian
Zhou, Jian
Zhang, Menghui
Zhang, Yan
Zeng, Yindi
Li, Shihao
Xu, KailinAff2, Aff4, IDs11010023047646_cor7
Yao, RuosiAff2, Aff4, Aff5, IDs11010023047646_cor8
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Źródło:
Molecular and Cellular Biochemistry: An International Journal for Chemical Biology in Health and Disease. 479(4):843-857
Czasopismo naukowe
Tytuł:
Multi-dimensional characterization of apoptosis in the tumor microenvironment and therapeutic relevance in melanoma
Autorzy:
Ye, JingAff1, Aff2, Aff3, Aff4, Aff5
Wei, Benliang
Zhou, GuoweiAff1, Aff2, Aff3, Aff4, Aff5
Xu, YantaoAff1, Aff2, Aff3, Aff4, Aff5
He, YiAff1, Aff2, Aff3, Aff4, Aff5
Hu, XihengAff1, Aff2, Aff7, Aff8, IDs13402024009300_cor6
Chen, XiangAff1, Aff2, Aff3, Aff4, Aff5, Aff7, IDs13402024009300_cor7
Zhang, GuanxiongAff1, Aff2, Aff3, Aff4, Aff5, IDs13402024009300_cor8
Liu, HongAff1, Aff2, Aff3, Aff4, Aff5, Aff6, IDs13402024009300_cor9
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Źródło:
Cellular Oncology. :1-21
Czasopismo naukowe
Tytuł:
Upregulation of tumor suppressor PIAS3 by Honokiol promotes tumor cell apoptosis via selective inhibition of STAT3 tyrosine 705 phosphorylation
Autorzy:
Fei, Yue
Zhang, Xiaoyan
Wang, Xiaohui
Sun, Yifei
He, Jin
Liu, Xia
Song, Zheng
Li, Lanfang
Qiu, Lihua
Qian, Zhengzi
Zhou, Shiyong
Liu, Xianming
Zhang, HuilaiAff1, IDs1141802301757z_cor13
Wang, XianhuoAff1, IDs1141802301757z_cor14
Pokaż więcej
Źródło:
Journal of Natural Medicines. 78(2):285-295
Czasopismo naukowe

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