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Tytuł :
Comparison of baseline laboratory findings of macrophage activation syndrome complicating systemic juvenile idiopathic arthritis and multisystem inflammatory syndrome in children.
Autorzy :
Aydın F; Division of Pediatric Rheumatology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Çelikel E; Division of Pediatric Rheumatology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Ekici Tekin Z; Division of Pediatric Rheumatology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Coşkun S; Division of Pediatric Rheumatology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Sezer M; Division of Pediatric Rheumatology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Karagöl C; Division of Pediatric Rheumatology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Kaplan MM; Division of Pediatric Rheumatology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Tekgöz N; Division of Pediatric Rheumatology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Kurt T; Division of Pediatric Rheumatology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Özcan S; Division of Pediatric Intensive Care Unit, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Kavurt AV; Division of Pediatric Cardiology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Özkaya Parlakay A; Division of Pediatric Infectious Disease, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
Çelikel Acar B; Division of Pediatric Rheumatology, Department of Pediatrics, Ministry of Health, Ankara City Hospital, Ankara, Turkey.
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Źródło :
International journal of rheumatic diseases [Int J Rheum Dis] 2021 Apr; Vol. 24 (4), pp. 542-547. Date of Electronic Publication: 2021 Feb 07.
Typ publikacji :
Comparative Study; Journal Article
MeSH Terms :
Macrophage Activation*
C-Reactive Protein/*metabolism
COVID-19/*diagnosis
Ferritins/*blood
Fibrinogen/*metabolism
Macrophage Activation Syndrome/*diagnosis
Systemic Inflammatory Response Syndrome/*diagnosis
Biomarkers/blood ; COVID-19/blood ; COVID-19/complications ; Child ; Female ; Follow-Up Studies ; Humans ; Leukocyte Count ; Macrophage Activation Syndrome/blood ; Macrophage Activation Syndrome/etiology ; Male ; Retrospective Studies ; SARS-CoV-2 ; Systemic Inflammatory Response Syndrome/blood ; Systemic Inflammatory Response Syndrome/complications
SCR Disease Name :
pediatric multisystem inflammatory disease, COVID-19 related
Czasopismo naukowe
Tytuł :
Shear and Integrin Outside-In Signaling Activate NADPH-Oxidase 2 to Promote Platelet Activation.
Autorzy :
Xu Z; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).
Liang Y; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).
Delaney MK; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).; Dupage Medical Technology, Inc (M.K.D.).
Zhang Y; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).
Kim K; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).; Korean Medicine-Application Center, Korea Institute of Oriental Medicine, Daegu (K.K.).
Li J; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).
Bai Y; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).
Cho J; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).
Ushio-Fukai M; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).; Department of Medicine (Cardiology), Vascular Biology Center, Medical College of Georgia at Augusta University (M.U.-F.).
Cheng N; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).
Du X; Department of Pharmacology, University of Illinois at Chicago (Z.X., Y.L., M.K.D., Y.Z., K.K., J.L., Y.B., J.C., M.U.-F., N.C., X.D.).
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Źródło :
Arteriosclerosis, thrombosis, and vascular biology [Arterioscler Thromb Vasc Biol] 2021 May 05; Vol. 41 (5), pp. 1638-1653. Date of Electronic Publication: 2021 Mar 11.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Mechanotransduction, Cellular*
Platelet Activation*
Blood Platelets/*enzymology
Hydrogen Peroxide/*metabolism
Integrin beta3/*metabolism
NADPH Oxidase 2/*metabolism
Platelet Glycoprotein GPIIb-IIIa Complex/*metabolism
Animals ; Enzyme Activation ; Female ; GTP-Binding Protein alpha Subunits, G12-G13/genetics ; GTP-Binding Protein alpha Subunits, G12-G13/metabolism ; Integrin beta3/genetics ; Male ; Mice, Inbred C57BL ; Mice, Knockout ; NADPH Oxidase 2/genetics ; NADPH Oxidases/metabolism ; Phosphatidylinositol 3-Kinase/metabolism ; Phosphorylation ; Platelet Factor 4/genetics ; Platelet Factor 4/metabolism ; Platelet Glycoprotein GPIIb-IIIa Complex/genetics ; Proto-Oncogene Proteins c-akt/metabolism ; Stress, Mechanical ; Syk Kinase/metabolism
Czasopismo naukowe
Tytuł :
Dynamic regulation of B cell complement signaling is integral to germinal center responses.
Autorzy :
Cumpelik A; Renal Division, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, USA.; Translational Transplant Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Heja D; Renal Division, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, USA.; Translational Transplant Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, USA.; eGenesis Inc., Cambridge, MA, USA.
Hu Y; Renal Division, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, USA.; Translational Transplant Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Varano G; Department of Oncological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.; Department of Translational Medicine, Laboratory for Advanced Therapy Technologies, University of Ferrara, Ferrara, Italy.
Ordikhani F; Renal Division, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, USA.; Translational Transplant Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, USA.; Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Roberto MP; Department of Oncological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.; Graduate School of Biomedical Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
He Z; Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Homann D; Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Lira SA; Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Dominguez-Sola D; Department of Oncological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA. .; Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA. .; Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA. .; Department of Pathology, Icahn School of Medicine at Mount Sinai, New York, NY, USA. .
Heeger PS; Renal Division, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, USA. .; Translational Transplant Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, USA. .; Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA. .
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Źródło :
Nature immunology [Nat Immunol] 2021 Jun; Vol. 22 (6), pp. 757-768. Date of Electronic Publication: 2021 May 24.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Complement Activation*
B-Lymphocytes/*immunology
Complement C3a/*metabolism
Complement C5a/*metabolism
Germinal Center/*immunology
Animals ; Animals, Genetically Modified ; B-Lymphocytes/metabolism ; CD55 Antigens/genetics ; CD55 Antigens/metabolism ; CD59 Antigens/metabolism ; Cell Line, Tumor ; Clonal Hematopoiesis/immunology ; Germinal Center/cytology ; Germinal Center/metabolism ; Humans ; Lymphocyte Activation ; Mice ; Palatine Tonsil/cytology ; Palatine Tonsil/pathology ; Proto-Oncogene Proteins c-bcl-6/metabolism ; Receptor, Anaphylatoxin C5a/genetics ; Receptor, Anaphylatoxin C5a/metabolism ; Receptors, Antigen, B-Cell/metabolism ; Receptors, Complement/genetics ; Receptors, Complement/metabolism ; Signal Transduction/immunology ; TOR Serine-Threonine Kinases/metabolism
Czasopismo naukowe
Tytuł :
Overexpression of the PTPN22 Autoimmune Risk Variant LYP-620W Fails to Restrain Human CD4 T Cell Activation.
Autorzy :
Perry DJ; Department of Pathology, Immunology, and Laboratory Medicine, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL; and.
Peters LD; Department of Pathology, Immunology, and Laboratory Medicine, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL; and.
Lakshmi PS; Department of Pathology, Immunology, and Laboratory Medicine, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL; and.
Zhang L; Department of Pathology, Immunology, and Laboratory Medicine, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL; and.
Han Z; Department of Pathology, Immunology, and Laboratory Medicine, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL; and.
Wasserfall CH; Department of Pathology, Immunology, and Laboratory Medicine, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL; and.
Mathews CE; Department of Pathology, Immunology, and Laboratory Medicine, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL; and.
Atkinson MA; Department of Pathology, Immunology, and Laboratory Medicine, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL; and.; Department of Pediatrics, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL.
Brusko TM; Department of Pathology, Immunology, and Laboratory Medicine, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL; and .; Department of Pediatrics, University of Florida Diabetes Institute, College of Medicine, Gainesville, FL.
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Źródło :
Journal of immunology (Baltimore, Md. : 1950) [J Immunol] 2021 Aug 01; Vol. 207 (3), pp. 849-859. Date of Electronic Publication: 2021 Jul 23.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Protein Tyrosine Phosphatase, Non-Receptor Type 22/*metabolism
Signaling Lymphocytic Activation Molecule Associated Protein/*metabolism
T-Lymphocyte Subsets/*immunology
T-Lymphocytes, Regulatory/*immunology
Autoimmunity ; Cell Differentiation ; Cell Proliferation ; Cells, Cultured ; Gene Expression Regulation ; Genetic Variation ; Humans ; Immune Tolerance ; Immunologic Memory ; Lymphocyte Activation/genetics ; Mutation/genetics ; Protein Tyrosine Phosphatase, Non-Receptor Type 22/genetics ; Signaling Lymphocytic Activation Molecule Associated Protein/genetics
Czasopismo naukowe
Tytuł :
IL36 is a critical upstream amplifier of neutrophilic lung inflammation in mice.
Autorzy :
Koss CK; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.; Department of Biology, University of Konstanz, Konstanz, Germany.
Wohnhaas CT; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.; Department of Biology, University of Konstanz, Konstanz, Germany.
Baker JR; Airway Disease, National Heart and Lung Institute, Imperial College London, London, UK.
Tilp C; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.
Przibilla M; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.
Lerner C; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.
Frey S; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.
Keck M; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.
Williams CMM; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.; WRDM, Inflammation and Immunology Research Unit, Pfizer, Cambridge, MA, USA.
Peter D; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.
Ramanujam M; Boehringer Ingelheim Pharmaceuticals Inc., Ridgefield, CT, USA.
Fine J; Boehringer Ingelheim Pharmaceuticals Inc., Ridgefield, CT, USA.
Gantner F; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.; Department of Biology, University of Konstanz, Konstanz, Germany.
Thomas M; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany.
Barnes PJ; Airway Disease, National Heart and Lung Institute, Imperial College London, London, UK.
Donnelly LE; Airway Disease, National Heart and Lung Institute, Imperial College London, London, UK.
El Kasmi KC; Boehringer Ingelheim Pharma GmbH & Co KG, Biberach, Germany. .
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Źródło :
Communications biology [Commun Biol] 2021 Feb 08; Vol. 4 (1), pp. 172. Date of Electronic Publication: 2021 Feb 08.
Typ publikacji :
Journal Article
MeSH Terms :
Neutrophil Activation*
Interleukin-1/*metabolism
Lung/*metabolism
Neutrophils/*metabolism
Orthomyxoviridae Infections/*metabolism
Pneumonia, Viral/*metabolism
Receptors, Interleukin-1/*metabolism
Animals ; Cells, Cultured ; Cigarette Smoking ; Disease Models, Animal ; Female ; Fibroblasts/immunology ; Fibroblasts/metabolism ; Humans ; Influenza A Virus, H1N1 Subtype/pathogenicity ; Interleukin-1/genetics ; Lung/immunology ; Lung/virology ; Macrophage Activation ; Macrophages, Alveolar/immunology ; Macrophages, Alveolar/metabolism ; Male ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Mice, Knockout ; Neutrophils/immunology ; Neutrophils/virology ; Orthomyxoviridae Infections/genetics ; Orthomyxoviridae Infections/immunology ; Orthomyxoviridae Infections/virology ; Pneumonia, Viral/genetics ; Pneumonia, Viral/immunology ; Pneumonia, Viral/virology ; Receptors, Interleukin-1/genetics ; Signal Transduction
Czasopismo naukowe
Tytuł :
Transactivation of Herpes Simplex Virus 1 (HSV-1) Infected Cell Protein 4 Enhancer by Glucocorticoid Receptor and Stress-Induced Transcription Factors Requires Overlapping Krüppel-Like Transcription Factor 4/Sp1 Binding Sites.
Autorzy :
Ostler JB; Oklahoma State University College of Veterinary Medicine, Department of Veterinary Pathobiology, Stillwater, Oklahoma, USA.
Thunuguntla P; Oklahoma State University College of Veterinary Medicine, Department of Veterinary Pathobiology, Stillwater, Oklahoma, USA.
Hendrickson BY; Oklahoma State University College of Veterinary Medicine, Department of Veterinary Pathobiology, Stillwater, Oklahoma, USA.
Jones C; Oklahoma State University College of Veterinary Medicine, Department of Veterinary Pathobiology, Stillwater, Oklahoma, USA .
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Źródło :
Journal of virology [J Virol] 2021 Jan 28; Vol. 95 (4). Date of Electronic Publication: 2021 Jan 28 (Print Publication: 2021).
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms :
Herpes Simplex*/immunology
Herpes Simplex*/virology
Virus Activation*
Virus Latency*
Herpesvirus 1, Human/*physiology
Immediate-Early Proteins/*immunology
Receptors, Glucocorticoid/*immunology
Animals ; Cell Line, Tumor ; Chlorocebus aethiops ; Gene Expression Regulation, Viral ; Humans ; Kruppel-Like Transcription Factors/immunology ; Mice ; Promyelocytic Leukemia Zinc Finger Protein/immunology ; Snail Family Transcription Factors/immunology ; Transcriptional Activation ; Vero Cells
Czasopismo naukowe
Tytuł :
Thromboinflammation Supports Complement Activation in Cancer Patients With COVID-19.
Autorzy :
Peerschke EI; Department of Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, United States.
Valentino A; Department of Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, United States.
So RJ; Department of Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, United States.
Shulman S; Department of Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, United States.
Ravinder; Department of Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, United States.
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Źródło :
Frontiers in immunology [Front Immunol] 2021 Aug 18; Vol. 12, pp. 716361. Date of Electronic Publication: 2021 Aug 18 (Print Publication: 2021).
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms :
COVID-19/*immunology
Complement Activation/*immunology
Inflammation/*immunology
Neoplasms/*immunology
SARS-CoV-2/*immunology
Thrombosis/*immunology
Adolescent ; Adult ; Aged ; Aged, 80 and over ; Biomarkers/blood ; COVID-19/blood ; COVID-19/virology ; Complement Inactivating Agents/blood ; Female ; Fibrin Fibrinogen Degradation Products/metabolism ; Humans ; Inflammation/blood ; Male ; Middle Aged ; Neoplasms/blood ; Platelet Activation/immunology ; Retrospective Studies ; SARS-CoV-2/physiology ; Thrombosis/blood ; Young Adult
Czasopismo naukowe
Tytuł :
The rise of complement in ANCA-associated vasculitis: from marginal player to target of modern therapy.
Autorzy :
Trivioli G; Nephrology Unit, Meyer Children's Hospital, Firenze, Italy.; Department of Biomedical Experimental and Clinical Sciences 'Mario Serio', University of Firenze, Firenze, Italy.
Vaglio A; Nephrology Unit, Meyer Children's Hospital, Firenze, Italy.; Department of Biomedical Experimental and Clinical Sciences 'Mario Serio', University of Firenze, Firenze, Italy.
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Źródło :
Clinical and experimental immunology [Clin Exp Immunol] 2020 Dec; Vol. 202 (3), pp. 403-406. Date of Electronic Publication: 2020 Oct 05.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Neutrophil Activation*
Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/*immunology
Antibodies, Antineutrophil Cytoplasmic/*immunology
Complement Activation/*immunology
Complement C5a/*immunology
Neutrophils/*immunology
Animals ; Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/drug therapy ; Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/pathology ; Complement Activation/drug effects ; Glomerulonephritis/drug therapy ; Glomerulonephritis/immunology ; Glomerulonephritis/pathology ; Humans ; Mice
Czasopismo naukowe
Tytuł :
Iron Released after Cryo-Thermal Therapy Induced M1 Macrophage Polarization, Promoting the Differentiation of CD4 T Cells into CTLs.
Autorzy :
Wang S; School of Biomedical Engineering, Shanghai Jiao Tong University, Shanghai 200030, China.; School of Biomedical Engineering and Med-X Research Institute, Shanghai Jiao Tong University, Shanghai 200030, China.
Cheng M; School of Biomedical Engineering, Shanghai Jiao Tong University, Shanghai 200030, China.; School of Biomedical Engineering and Med-X Research Institute, Shanghai Jiao Tong University, Shanghai 200030, China.
Peng P; School of Biomedical Engineering, Shanghai Jiao Tong University, Shanghai 200030, China.; School of Biomedical Engineering and Med-X Research Institute, Shanghai Jiao Tong University, Shanghai 200030, China.
Lou Y; School of Biomedical Engineering, Shanghai Jiao Tong University, Shanghai 200030, China.; School of Biomedical Engineering and Med-X Research Institute, Shanghai Jiao Tong University, Shanghai 200030, China.
Zhang A; School of Biomedical Engineering, Shanghai Jiao Tong University, Shanghai 200030, China.; School of Biomedical Engineering and Med-X Research Institute, Shanghai Jiao Tong University, Shanghai 200030, China.
Liu P; School of Biomedical Engineering, Shanghai Jiao Tong University, Shanghai 200030, China.; School of Biomedical Engineering and Med-X Research Institute, Shanghai Jiao Tong University, Shanghai 200030, China.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Jun 29; Vol. 22 (13). Date of Electronic Publication: 2021 Jun 29.
Typ publikacji :
Journal Article
MeSH Terms :
CD4-Positive T-Lymphocytes/*drug effects
Cryotherapy/*adverse effects
Iron/*metabolism
Iron/*pharmacology
Macrophage Activation/*drug effects
T-Lymphocytes, Cytotoxic/*drug effects
Animals ; CD4-Positive T-Lymphocytes/physiology ; Cell Differentiation/drug effects ; Cell Differentiation/immunology ; Cell Polarity/drug effects ; Cells, Cultured ; Female ; Iron Chelating Agents/pharmacology ; Lymphocyte Activation/drug effects ; Macrophage Activation/physiology ; Macrophages/drug effects ; Macrophages/physiology ; Mice ; Mice, Inbred BALB C ; RAW 264.7 Cells ; T-Lymphocytes, Cytotoxic/physiology
Czasopismo naukowe
Tytuł :
Functional selective FPR1 signaling in favor of an activation of the neutrophil superoxide generating NOX2 complex.
Autorzy :
Lind S; Department of Rheumatology and Inflammation Research, Institute of Medicine, University of Gothenburg, Gothenburg, Sweden.
Dahlgren C; Department of Rheumatology and Inflammation Research, Institute of Medicine, University of Gothenburg, Gothenburg, Sweden.
Holmdahl R; Medical Inflammation Research, Department of Medical Biochemistry and Biophysics, Karolinska Institute, Stockholm, Sweden.
Olofsson P; Medical Inflammation Research, Department of Medical Biochemistry and Biophysics, Karolinska Institute, Stockholm, Sweden.
Forsman H; Department of Rheumatology and Inflammation Research, Institute of Medicine, University of Gothenburg, Gothenburg, Sweden.
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Źródło :
Journal of leukocyte biology [J Leukoc Biol] 2021 Jun; Vol. 109 (6), pp. 1105-1120. Date of Electronic Publication: 2020 Oct 11.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Signal Transduction*/drug effects
NADPH Oxidase 2/*metabolism
Neutrophil Activation/*immunology
Neutrophils/*immunology
Neutrophils/*metabolism
Receptors, Formyl Peptide/*metabolism
Superoxides/*metabolism
Calcium/metabolism ; Chemotaxis/genetics ; Cytoskeleton/metabolism ; Drug Discovery ; Enzyme Activation ; HL-60 Cells ; Humans ; NADPH Oxidases/metabolism ; Neutrophil Activation/genetics ; Phosphorylation ; Protein Binding ; Reactive Oxygen Species/metabolism ; Receptors, Formyl Peptide/agonists
Czasopismo naukowe
Tytuł :
Th1-Polarized, Dengue Virus-Activated Human Mast Cells Induce Endothelial Transcriptional Activation and Permeability.
Autorzy :
Syenina A; Program in Emerging Infectious Diseases, Duke-National University of Singapore, Singapore 169857, Singapore.
Saron WAA; Program in Emerging Infectious Diseases, Duke-National University of Singapore, Singapore 169857, Singapore.
Jagaraj CJ; Program in Emerging Infectious Diseases, Duke-National University of Singapore, Singapore 169857, Singapore.
Bibi S; Laboratory of Molecular and Cellular Oncology, LBPA CNRS UMR8113, Ecole Normale Supérieure de Cachan, 94235 Cachan, France.
Arock M; Laboratory of Molecular and Cellular Oncology, LBPA CNRS UMR8113, Ecole Normale Supérieure de Cachan, 94235 Cachan, France.; Laboratory of Hematology, Pitié-Salpêtrière Hospital, Pierre et Marie Curie University, 75005 Paris, France.
Gubler DJ; Program in Emerging Infectious Diseases, Duke-National University of Singapore, Singapore 169857, Singapore.
Rathore APS; Pathology Department, Duke University Medical Center, Durham, NC 27710, USA.
Abraham SN; Program in Emerging Infectious Diseases, Duke-National University of Singapore, Singapore 169857, Singapore.; Pathology Department, Duke University Medical Center, Durham, NC 27710, USA.; Immunology Department, Duke University Medical Center, Durham, NC 27710, USA.; Molecular Genetics and Microbiology Departments, Duke University Medical Center, Durham, NC 27710, USA.
St John AL; Program in Emerging Infectious Diseases, Duke-National University of Singapore, Singapore 169857, Singapore.; Pathology Department, Duke University Medical Center, Durham, NC 27710, USA.; Department of Microbiology and Immunology, National University of Singapore, Singapore 117545, Singapore.; SingHealth Duke-NUS Global Health Institute, Singapore 169857, Singapore.
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Źródło :
Viruses [Viruses] 2020 Dec 02; Vol. 12 (12). Date of Electronic Publication: 2020 Dec 02.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Transcriptional Activation*
Dengue/*immunology
Dengue/*metabolism
Dengue Virus/*immunology
Endothelium, Vascular/*metabolism
Mast Cells/*physiology
Th1 Cells/*physiology
Biomarkers ; Capillary Permeability ; Cell Degranulation/immunology ; Dengue/virology ; Endothelial Cells ; Endothelium, Vascular/immunology ; Fluorescent Antibody Technique ; Gene Expression Profiling ; Histocytochemistry ; Host-Pathogen Interactions/immunology ; Humans ; Lymphocyte Activation ; Macrophages/immunology ; Macrophages/metabolism ; Mast Cells/cytology
Czasopismo naukowe
Tytuł :
Resveratrol suppresses the reprogramming of macrophages into an endotoxin-tolerant state through the activation of AMP-activated protein kinase.
Autorzy :
Quan H; Department of Plastic Surgery, Affiliated Hospital of Zunyi Medical University, Zunyi, China; Department of Anesthesiology and Pain Medicine, Chonnam National University Medical School, Gwangju, South Korea.
Yin M; Department of Anesthesiology and Pain Medicine, Chonnam National University Medical School, Gwangju, South Korea; The Brain Korea 21 Project, Center for Biomedical Human Resources at Chonnam National University, Gwangju, South Korea.
Kim J; Department of Anesthesiology and Pain Medicine, Chonnam National University Medical School, Gwangju, South Korea.
Jang EA; Department of Anesthesiology and Pain Medicine, Chonnam National University Medical School, Gwangju, South Korea.
Yang SH; Department of Anesthesiology and Pain Medicine, Chonnam National University Medical School, Gwangju, South Korea.
Bae HB; Department of Anesthesiology and Pain Medicine, Chonnam National University Medical School, Gwangju, South Korea; The Brain Korea 21 Project, Center for Biomedical Human Resources at Chonnam National University, Gwangju, South Korea; Department of Anesthesiology and Pain Medicine, Chonnam National University Hwasun Hospital, Hwasun-gun, Jeollanamdo, South Korea. Electronic address: .
Jeong S; Department of Anesthesiology and Pain Medicine, Chonnam National University Medical School, Gwangju, South Korea; Department of Anesthesiology and Pain Medicine, Chonnam National University Hwasun Hospital, Hwasun-gun, Jeollanamdo, South Korea. Electronic address: .
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Źródło :
European journal of pharmacology [Eur J Pharmacol] 2021 May 15; Vol. 899, pp. 173993. Date of Electronic Publication: 2021 Mar 03.
Typ publikacji :
Journal Article
MeSH Terms :
AMP-Activated Protein Kinases/*metabolism
Endotoxemia/*drug therapy
Immunologic Factors/*pharmacology
Macrophage Activation/*drug effects
Macrophages/*drug effects
Resveratrol/*pharmacology
Animals ; Calcium Signaling ; Calcium-Calmodulin-Dependent Protein Kinase Kinase/metabolism ; Disease Models, Animal ; Endotoxemia/chemically induced ; Endotoxemia/enzymology ; Enzyme Activation ; Inflammation Mediators/metabolism ; Interleukin-1 Receptor-Associated Kinases/metabolism ; Interleukin-6/genetics ; Interleukin-6/metabolism ; Lipopolysaccharides ; Macrophages/enzymology ; Male ; Mice ; Mice, Inbred BALB C ; Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases/metabolism ; RAW 264.7 Cells ; Tumor Necrosis Factor-alpha/genetics ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Tytuł :
Unexpected macrophage activation syndrome in a healthy young woman: a case report.
Autorzy :
Saviano A; Department of Emergency Medicine, Fondazione Policlinico A. Gemelli IRCCS, Rome, Italy. .
Petrucci M
Tilli P
Pignataro G
Petruzziello C
Giuliano G
Ojetti V
Covino M
Franceschi F
Candelli M
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Źródło :
European review for medical and pharmacological sciences [Eur Rev Med Pharmacol Sci] 2020 Jul; Vol. 24 (13), pp. 7320-7323.
Typ publikacji :
Case Reports
MeSH Terms :
Macrophage Activation*
Macrophage Activation Syndrome/*diagnosis
Macrophages/*immunology
Adult ; Disease Progression ; Fatal Outcome ; Female ; Humans ; Macrophage Activation Syndrome/complications ; Macrophage Activation Syndrome/immunology ; Macrophage Activation Syndrome/therapy ; Multiple Organ Failure/etiology ; Treatment Failure
Raport
Tytuł :
The role of interleukin-18 in the diagnosis and monitoring of hemophagocytic lymphohistiocytosis/macrophage activation syndrome - a systematic review.
Autorzy :
Krei JM; Department of Clinical Biochemistry, Aarhus University Hospital, Aarhus, Denmark.
Møller HJ; Department of Clinical Biochemistry, Aarhus University Hospital, Aarhus, Denmark.; Department of Clinical Medicine, Aarhus University, Aarhus, Denmark.
Larsen JB; Department of Clinical Biochemistry, Aarhus University Hospital, Aarhus, Denmark.
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Źródło :
Clinical and experimental immunology [Clin Exp Immunol] 2021 Feb; Vol. 203 (2), pp. 174-182. Date of Electronic Publication: 2020 Nov 23.
Typ publikacji :
Journal Article; Meta-Analysis; Systematic Review
MeSH Terms :
Interleukin-18/*immunology
Lymphohistiocytosis, Hemophagocytic/*diagnosis
Macrophage Activation Syndrome/*diagnosis
Macrophages/*immunology
Monitoring, Immunologic/*methods
Animals ; Diagnosis, Differential ; Humans ; Lymphohistiocytosis, Hemophagocytic/immunology ; Macrophage Activation ; Macrophage Activation Syndrome/immunology ; Phenotype
Czasopismo naukowe
Tytuł :
Amyloid precursor protein 770 is specifically expressed and released from platelets.
Autorzy :
Miura S; Preparing Section for New Faculty of Medical Science, Fukushima Medical University, Fukushima, Japan.
Yoshihisa A; Department of Cardiovascular Medicine, Fukushima Medical University, Fukushima, Japan.
Misaka T; Department of Cardiovascular Medicine, Fukushima Medical University, Fukushima, Japan.
Yamaki T; Department of Cardiovascular Medicine, Fukushima Medical University, Fukushima, Japan.
Kojima T; Department of Neurosurgery, Fukushima Medical University, Fukushima, Japan.
Toyokawa M; Preparing Section for New Faculty of Medical Science, Fukushima Medical University, Fukushima, Japan.
Ogawa K; Preparing Section for New Faculty of Medical Science, Fukushima Medical University, Fukushima, Japan.
Shimura H; Department of Laboratory Medicine, Fukushima Medical University, Fukushima, Japan.
Yamamoto N; Department of Biochemistry, School of Pharmaceutical Sciences, Ohu University, Fukushima, Japan.
Kasahara K; Laboratory of Biomembrane, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan.
Takeishi Y; Department of Cardiovascular Medicine, Fukushima Medical University, Fukushima, Japan.
Kitazume S; Preparing Section for New Faculty of Medical Science, Fukushima Medical University, Fukushima, Japan. Electronic address: .
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Źródło :
The Journal of biological chemistry [J Biol Chem] 2020 Sep 18; Vol. 295 (38), pp. 13194-13201. Date of Electronic Publication: 2020 Jul 23.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Gene Expression Regulation*
Platelet Activation*
Amyloid beta-Protein Precursor/*biosynthesis
Blood Platelets/*metabolism
CD40 Antigens/metabolism ; Endothelial Cells/metabolism ; Humans ; Lymphocyte Activation ; T-Lymphocytes/metabolism
Czasopismo naukowe
Tytuł :
Transcriptome profiling and protease inhibition experiments identify proteases that activate H3N2 influenza A and influenza B viruses in murine airways.
Autorzy :
Harbig A; Institute of Virology, Philipps-University, 35043 Marburg, Germany.
Mernberger M; Institute of Molecular Oncology, Member of the German Center for Lung Research, Philipps-University, 35043 Marburg, Germany.
Bittel L; Institute of Virology, Philipps-University, 35043 Marburg, Germany.
Pleschka S; Institute of Medical Virology, Justus Liebig University, 35390 Giessen, Germany.
Schughart K; Department of Infection Genetics, Helmholtz Centre for Infection Research, 38124 Braunschweig, Germany.; University of Veterinary Medicine Hannover, 30559 Hannover, Germany.; Department of Microbiology, Immunology, and Biochemistry, University of Tennessee Health Science Center, Memphis, Tennessee 38163.
Steinmetzer T; Institute of Pharmaceutical Chemistry, Philipps-University, 35043 Marburg, Germany.
Stiewe T; Institute of Molecular Oncology, Member of the German Center for Lung Research, Philipps-University, 35043 Marburg, Germany.; Genomics Core Facility, Philipps-University, 35043 Marburg, Germany.
Nist A; Genomics Core Facility, Philipps-University, 35043 Marburg, Germany.
Böttcher-Friebertshäuser E; Institute of Virology, Philipps-University, 35043 Marburg, Germany .
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Źródło :
The Journal of biological chemistry [J Biol Chem] 2020 Aug 14; Vol. 295 (33), pp. 11388-11407. Date of Electronic Publication: 2020 Apr 17.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Virus Activation*/drug effects
Influenza A Virus, H3N2 Subtype/*physiology
Influenza B virus/*physiology
Influenza, Human/*enzymology
Orthomyxoviridae Infections/*enzymology
Peptide Hydrolases/*metabolism
Animals ; Cell Line ; Dogs ; Enzyme Activation/drug effects ; Gene Expression Profiling ; HEK293 Cells ; Host-Pathogen Interactions/drug effects ; Humans ; Influenza A Virus, H3N2 Subtype/drug effects ; Influenza B virus/drug effects ; Influenza, Human/drug therapy ; Influenza, Human/genetics ; Influenza, Human/virology ; Lung/enzymology ; Lung/metabolism ; Lung/virology ; Madin Darby Canine Kidney Cells ; Membrane Proteins/genetics ; Membrane Proteins/metabolism ; Mice ; Orthomyxoviridae Infections/drug therapy ; Orthomyxoviridae Infections/genetics ; Orthomyxoviridae Infections/virology ; Peptide Hydrolases/genetics ; Protease Inhibitors/pharmacology ; Serine Endopeptidases/genetics ; Serine Endopeptidases/metabolism
Czasopismo naukowe
Tytuł :
Programmed cell death 1-expressing CD56-negative natural killer (NK) cell expansion is a hallmark of chronic NK cell activation during dasatinib treatment.
Autorzy :
Ishiyama KI; Department of Hematology and Oncology, Graduate School of Medicine, Kyoto University, Kyoto, Japan.; Department of Microbiology and Immunology, University of California San Francisco, San Francisco, CA, USA.
Kitawaki T; Department of Hematology and Oncology, Graduate School of Medicine, Kyoto University, Kyoto, Japan.
Otsuka Y; Department of Hematology and Oncology, Graduate School of Medicine, Kyoto University, Kyoto, Japan.
Takaori-Kondo A; Department of Hematology and Oncology, Graduate School of Medicine, Kyoto University, Kyoto, Japan.
Kadowaki N; Division of Hematology, Rheumatology and Respiratory Medicine, Department of Internal Medicine, Faculty of Medicine, Kagawa University, Kagawa, Japan.
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Źródło :
Cancer science [Cancer Sci] 2021 Feb; Vol. 112 (2), pp. 523-536. Date of Electronic Publication: 2020 Dec 16.
Typ publikacji :
Journal Article
MeSH Terms :
Antineoplastic Agents/*therapeutic use
Dasatinib/*therapeutic use
Killer Cells, Natural/*immunology
Leukemia/*drug therapy
Leukemia/*immunology
Lymphocyte Activation/*drug effects
B7-H1 Antigen/immunology ; CD56 Antigen/immunology ; Cytomegalovirus Infections/complications ; Humans ; Killer Cells, Natural/drug effects ; Lymphocyte Activation/immunology ; Lymphocyte Subsets/drug effects ; Lymphocyte Subsets/immunology ; Virus Activation/immunology
Czasopismo naukowe
Tytuł :
Inhibition of succinate dehydrogenase activity impairs human T cell activation and function.
Autorzy :
Nastasi C; LEO Foundation Skin Immunology Research Center & Department of Immunology and Microbiology, University of Copenhagen, Panum Institute, The Maersk tower, 07.12.76, Blegdamsvej 3C, 2200, Copenhagen, Denmark.
Willerlev-Olsen A; LEO Foundation Skin Immunology Research Center & Department of Immunology and Microbiology, University of Copenhagen, Panum Institute, The Maersk tower, 07.12.76, Blegdamsvej 3C, 2200, Copenhagen, Denmark.
Dalhoff K; MS-Omics, Vedbæk, Denmark.
Ford SL; LEO Foundation Skin Immunology Research Center & Department of Immunology and Microbiology, University of Copenhagen, Panum Institute, The Maersk tower, 07.12.76, Blegdamsvej 3C, 2200, Copenhagen, Denmark.
Gadsbøll AØ; LEO Foundation Skin Immunology Research Center & Department of Immunology and Microbiology, University of Copenhagen, Panum Institute, The Maersk tower, 07.12.76, Blegdamsvej 3C, 2200, Copenhagen, Denmark.
Buus TB; LEO Foundation Skin Immunology Research Center & Department of Immunology and Microbiology, University of Copenhagen, Panum Institute, The Maersk tower, 07.12.76, Blegdamsvej 3C, 2200, Copenhagen, Denmark.
Gluud M; LEO Foundation Skin Immunology Research Center & Department of Immunology and Microbiology, University of Copenhagen, Panum Institute, The Maersk tower, 07.12.76, Blegdamsvej 3C, 2200, Copenhagen, Denmark.
Danielsen M; MS-Omics, Vedbæk, Denmark.
Litman T; Department of Immunology and Microbiology, University of Copenhagen, Copenhagen, Denmark.; LEO Pharma A/S, Ballerup, Denmark.
Bonefeld CM; LEO Foundation Skin Immunology Research Center & Department of Immunology and Microbiology, University of Copenhagen, Panum Institute, The Maersk tower, 07.12.76, Blegdamsvej 3C, 2200, Copenhagen, Denmark.
Geisler C; LEO Foundation Skin Immunology Research Center & Department of Immunology and Microbiology, University of Copenhagen, Panum Institute, The Maersk tower, 07.12.76, Blegdamsvej 3C, 2200, Copenhagen, Denmark.
Ødum N; LEO Foundation Skin Immunology Research Center & Department of Immunology and Microbiology, University of Copenhagen, Panum Institute, The Maersk tower, 07.12.76, Blegdamsvej 3C, 2200, Copenhagen, Denmark.
Woetmann A; LEO Foundation Skin Immunology Research Center & Department of Immunology and Microbiology, University of Copenhagen, Panum Institute, The Maersk tower, 07.12.76, Blegdamsvej 3C, 2200, Copenhagen, Denmark. .
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Źródło :
Scientific reports [Sci Rep] 2021 Jan 14; Vol. 11 (1), pp. 1458. Date of Electronic Publication: 2021 Jan 14.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Cell Proliferation/*drug effects
Enzyme Inhibitors/*pharmacology
Gene Expression Regulation/*drug effects
Lymphocyte Activation/*drug effects
Succinate Dehydrogenase/*antagonists & inhibitors
T-Lymphocytes/*immunology
Cytokines/biosynthesis ; Cytokines/immunology ; Enzyme Activation/drug effects ; Enzyme Activation/immunology ; Gene Expression Regulation/immunology ; Humans ; Succinate Dehydrogenase/immunology
Czasopismo naukowe
Tytuł :
Adiponectin Enhances B-Cell Proliferation and Differentiation via Activation of Akt1/STAT3 and Exacerbates Collagen-Induced Arthritis.
Autorzy :
Che N; Department of Rheumatology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China.
Sun X; Department of Rheumatology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China.
Gu L; Department of Rheumatology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China.
Wang X; Department of Pathology, Shenzhen Institute of Research and Innovation, The University of Hong Kong, Hong Kong, China.; Chongqing International Institute for Immunology, Hong Kong, China.
Shi J; Clinical Medical Science of the First Clinical Medical College, Nanjing Medical University, Nanjing, China.
Sun Y; Clinical Medical Science of the First Clinical Medical College, Nanjing Medical University, Nanjing, China.
Xu L; Department of Rheumatology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China.
Liu R; Department of Rheumatology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China.
Wang J; Department of Rheumatology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China.
Zhu F; Department of Rheumatology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China.
Peng N; Department of Rheumatology and Nephrology, The Second People's Hospital of China Three Gorges University, Yichang, China.
Xiao F; Department of Pathology, Shenzhen Institute of Research and Innovation, The University of Hong Kong, Hong Kong, China.; Chongqing International Institute for Immunology, Hong Kong, China.
Hu D; Department of Rheumatology and Nephrology, The Second People's Hospital of China Three Gorges University, Yichang, China.
Lu L; Department of Pathology, Shenzhen Institute of Research and Innovation, The University of Hong Kong, Hong Kong, China.; Chongqing International Institute for Immunology, Hong Kong, China.
Qiu W; Department of Immunology, Key Laboratory of Immunological Environment and Disease, Nanjing Medical University, Nanjing, China.
Zhang M; Department of Rheumatology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China.
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Źródło :
Frontiers in immunology [Front Immunol] 2021 Mar 18; Vol. 12, pp. 626310. Date of Electronic Publication: 2021 Mar 18 (Print Publication: 2021).
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Adiponectin/*toxicity
Arthritis, Experimental/*enzymology
B-Lymphocytes/*drug effects
Cell Differentiation/*drug effects
Cell Proliferation/*drug effects
Lymphocyte Activation/*drug effects
Proto-Oncogene Proteins c-akt/*metabolism
STAT3 Transcription Factor/*metabolism
Animals ; Arthritis, Experimental/chemically induced ; Arthritis, Experimental/immunology ; Arthritis, Experimental/pathology ; B-Lymphocytes/enzymology ; B-Lymphocytes/immunology ; Collagen Type II ; Enzyme Activation ; Male ; Mice, Inbred C57BL ; Mice, Inbred DBA ; Phosphatidylinositol 3-Kinase/metabolism ; Positive Regulatory Domain I-Binding Factor 1/genetics ; Positive Regulatory Domain I-Binding Factor 1/metabolism ; Proto-Oncogene Proteins c-akt/genetics ; Receptors, Adiponectin/agonists ; Receptors, Adiponectin/genetics ; Receptors, Adiponectin/metabolism ; STAT3 Transcription Factor/genetics ; Signal Transduction
Czasopismo naukowe
Tytuł :
Diffuse interstitial pneumonia-like/macrophage activation syndrome-like changes in patients with COVID-19 correlate with length of illness.
Autorzy :
Felix JC; Department of Pathology, Medical College of Wisconsin, 9200 W. Wisconsin Avenue, Milwaukee, WI 53226, United States of America. Electronic address: .
Sheinin YM; Department of Pathology, Medical College of Wisconsin, 9200 W. Wisconsin Avenue, Milwaukee, WI 53226, United States of America. Electronic address: .
Suster D; Rutgers University Medical School, 185 South Orange Avenue, Newark, NJ 07103, United States of America.
Ronen N; Department of Pathology, Medical College of Wisconsin, 9200 W. Wisconsin Avenue, Milwaukee, WI 53226, United States of America. Electronic address: .
Ratiani M; Department of Pathology, Medical College of Wisconsin, 9200 W. Wisconsin Avenue, Milwaukee, WI 53226, United States of America. Electronic address: .
Vanden Heuvel T; Department of Pathology, Medical College of Wisconsin, 9200 W. Wisconsin Avenue, Milwaukee, WI 53226, United States of America. Electronic address: .
Winge E; Department of Pathology, Medical College of Wisconsin, 9200 W. Wisconsin Avenue, Milwaukee, WI 53226, United States of America. Electronic address: .
Patton MD; Department of Pathology, Medical College of Wisconsin, 9200 W. Wisconsin Avenue, Milwaukee, WI 53226, United States of America. Electronic address: .
Rau MJ; Department of Pathology, Medical College of Wisconsin, 9200 W. Wisconsin Avenue, Milwaukee, WI 53226, United States of America. Electronic address: .
Ge L; Department of Pathology, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, United States of America. Electronic address: .
Sun Y; Department of Pathology, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, United States of America. Electronic address: .
Udhane SS; Department of Pathology, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, United States of America. Electronic address: .
Langenheim JF; Department of Pathology, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, United States of America. Electronic address: .
Rui H; Department of Pathology, Medical College of Wisconsin, 8701 Watertown Plank Rd, Milwaukee, WI 53226, United States of America. Electronic address: .
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Źródło :
Annals of diagnostic pathology [Ann Diagn Pathol] 2021 Aug; Vol. 53, pp. 151744. Date of Electronic Publication: 2021 Apr 19.
Typ publikacji :
Journal Article
MeSH Terms :
COVID-19/*pathology
Immunohistochemistry/*methods
Lung/*pathology
Lung Diseases, Interstitial/*pathology
Macrophage Activation Syndrome/*pathology
Adult ; Aged ; Aged, 80 and over ; Autopsy ; COVID-19/complications ; COVID-19/diagnosis ; COVID-19/virology ; Capsid Proteins/metabolism ; Comorbidity ; Female ; Humans ; Lung/metabolism ; Lung Diseases, Interstitial/etiology ; Lung Diseases, Interstitial/virology ; Lymphocytes/metabolism ; Lymphocytes/pathology ; Macrophage Activation Syndrome/etiology ; Macrophage Activation Syndrome/virology ; Macrophages/pathology ; Male ; Middle Aged ; Pulmonary Alveoli/immunology ; Pulmonary Alveoli/pathology ; SARS-CoV-2/genetics ; Sick Leave
Czasopismo naukowe

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