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Tytuł:
Modeling 5-FU-Induced Chemotherapy Selection of a Drug-Resistant Cancer Stem Cell Subpopulation.
Autorzy:
Ramović Hamzagić A; Faculty of Medical Sciences, Department of Genetics, University of Kragujevac, 34000 Kragujevac, Serbia.; Serbia for Harm Reduction of Biological and Chemical Hazards, Faculty of Medical Sciences, University of Kragujevac, 34000 Kragujevac, Serbia.
Cvetković D; Faculty of Medical Sciences, Department of Genetics, University of Kragujevac, 34000 Kragujevac, Serbia.; Serbia for Harm Reduction of Biological and Chemical Hazards, Faculty of Medical Sciences, University of Kragujevac, 34000 Kragujevac, Serbia.
Gazdić Janković M; Faculty of Medical Sciences, Department of Genetics, University of Kragujevac, 34000 Kragujevac, Serbia.; Serbia for Harm Reduction of Biological and Chemical Hazards, Faculty of Medical Sciences, University of Kragujevac, 34000 Kragujevac, Serbia.
Milivojević Dimitrijević N; Institute for Information Technologies Kragujevac, University of Kragujevac, Liceja Kneževine Srbije 1A, 34000 Kragujevac, Serbia.
Nikolić D; Institute for Information Technologies Kragujevac, University of Kragujevac, Liceja Kneževine Srbije 1A, 34000 Kragujevac, Serbia.; Bioengineering Research and Development Center (BioIRC), Prvoslava Stojanovica 6, 34000 Kragujevac, Serbia.
Živanović M; Institute for Information Technologies Kragujevac, University of Kragujevac, Liceja Kneževine Srbije 1A, 34000 Kragujevac, Serbia.
Kastratović N; Faculty of Medical Sciences, Department of Genetics, University of Kragujevac, 34000 Kragujevac, Serbia.; Serbia for Harm Reduction of Biological and Chemical Hazards, Faculty of Medical Sciences, University of Kragujevac, 34000 Kragujevac, Serbia.
Petrović I; Faculty of Medical Sciences, Department of Pathophysiology, University of Kragujevac, 34000 Kragujevac, Serbia.
Nikolić S; Faculty of Medical Sciences, Department of Genetics, University of Kragujevac, 34000 Kragujevac, Serbia.; Serbia for Harm Reduction of Biological and Chemical Hazards, Faculty of Medical Sciences, University of Kragujevac, 34000 Kragujevac, Serbia.
Jovanović M; Faculty of Sciences, University of Kragujevac, Radoja Domanovića 12, 34000 Kragujevac, Serbia.
Šeklić D; Institute for Information Technologies Kragujevac, University of Kragujevac, Liceja Kneževine Srbije 1A, 34000 Kragujevac, Serbia.
Filipović N; Bioengineering Research and Development Center (BioIRC), Prvoslava Stojanovica 6, 34000 Kragujevac, Serbia.; Faculty of Engineering, University of Kragujevac, Sestre Janjić 6, 34000 Kragujevac, Serbia.
Ljujić B; Faculty of Medical Sciences, Department of Genetics, University of Kragujevac, 34000 Kragujevac, Serbia.; Serbia for Harm Reduction of Biological and Chemical Hazards, Faculty of Medical Sciences, University of Kragujevac, 34000 Kragujevac, Serbia.
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Źródło:
Current oncology (Toronto, Ont.) [Curr Oncol] 2024 Feb 25; Vol. 31 (3), pp. 1221-1234. Date of Electronic Publication: 2024 Feb 25.
Typ publikacji:
Journal Article
MeSH Terms:
Artificial Intelligence*
Neoplasms*/pathology
Humans ; Cell Line, Tumor ; Aldehyde Dehydrogenase 1 Family ; Fluorouracil/pharmacology ; Neoplastic Stem Cells/metabolism ; Neoplastic Stem Cells/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Are embryonic stem cell markers and ALDH1A1 relevant in the context of breast cancer estrogen positivity?
Autorzy:
Mousa NA; Clinical Sciences Department, College of Medicine, University of Sharjah, Sharjah, United Arab Emirates.
Hussein A; Family and Community Medicine and Behavioural Sciences Department, College of Medicine, University of Sharjah, Sharjah, United Arab Emirates.
Elemam NM; Clinical Sciences Department, College of Medicine, University of Sharjah, Sharjah, United Arab Emirates.; Research Institute for Medical and Health Sciences, University of Sharjah, Sharjah, United Arab Emirates.
Mohammed G; Clinical Sciences Department, College of Medicine, University of Sharjah, Sharjah, United Arab Emirates.
Elwany M; Medical Research Institute, Alexandria University, Alexandria, Egypt.
Basha T; Clinical Sciences Department, College of Medicine, University of Sharjah, Sharjah, United Arab Emirates.
AlHammadi AA; Clinical Sciences Department, College of Medicine, University of Sharjah, Sharjah, United Arab Emirates.
Majzob RS; Clinical Sciences Department, College of Medicine, University of Sharjah, Sharjah, United Arab Emirates.
Talaat IM; Clinical Sciences Department, College of Medicine, University of Sharjah, Sharjah, United Arab Emirates.; Medical Research Institute, Alexandria University, Alexandria, Egypt.; Pathology Department, Faculty of Medicine, Alexandria University, Alexandria, Egypt.
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Źródło:
Cancer medicine [Cancer Med] 2024 Feb; Vol. 13 (3), pp. e7004.
Typ publikacji:
Journal Article
MeSH Terms:
Breast Neoplasms*/pathology
Humans ; Female ; Prospective Studies ; Biomarkers, Tumor/genetics ; Biomarkers, Tumor/metabolism ; Prognosis ; Estrogens ; Embryonic Stem Cells/metabolism ; Aldehyde Dehydrogenase 1 Family ; Retinal Dehydrogenase/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Co-expression of cancer stem cell markers, SALL4/ALDH1A1, is associated with tumor aggressiveness and poor survival in patients with serous ovarian carcinoma.
Autorzy:
Sharbatoghli M; Oncopathology Research Center, Iran University of Medical Sciences (IUMS), Tehran, Iran.
Shamshiripour P; Oncopathology Research Center, Iran University of Medical Sciences (IUMS), Tehran, Iran.
Fattahi F; Oncopathology Research Center, Iran University of Medical Sciences (IUMS), Tehran, Iran.; Department of Molecular Medicine, Faculty of Advanced Technologies in Medicine, Iran University of Medical Sciences, Tehran, Iran.
Kalantari E; Oncopathology Research Center, Iran University of Medical Sciences (IUMS), Tehran, Iran.
Habibi Shams Z; Department of Pathology, Faculty of Medicine, Iran University of Medical Sciences, Tehran, Iran.
Panahi M; Department of Pathology, Faculty of Medicine, Iran University of Medical Sciences, Tehran, Iran.
Totonchi M; Department of Stem Cells and Developmental Biology, Cell Science Research Center, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran, Iran.; Department of Genetics, Reproductive Biomedicine Research Center, Royan Institute for Reproductive Biomedicine, ACECR, Tehran, Iran.
Asadi-Lari Z; Department of Biology, Faculty of Science, University of Toronto, Toronto, Canada.
Madjd Z; Oncopathology Research Center, Iran University of Medical Sciences (IUMS), Tehran, Iran. .; Department of Molecular Medicine, Faculty of Advanced Technologies in Medicine, Iran University of Medical Sciences, Tehran, Iran. .
Saeednejad Zanjani L; Oncopathology Research Center, Iran University of Medical Sciences (IUMS), Tehran, Iran. .
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Źródło:
Journal of ovarian research [J Ovarian Res] 2022 Jan 28; Vol. 15 (1), pp. 17. Date of Electronic Publication: 2022 Jan 28.
Typ publikacji:
Journal Article
MeSH Terms:
Aldehyde Dehydrogenase 1 Family/*metabolism
Biomarkers, Tumor/*metabolism
Ovarian Neoplasms/*metabolism
Retinal Dehydrogenase/*metabolism
Transcription Factors/*metabolism
Adolescent ; Adult ; Aged ; Aldehyde Dehydrogenase 1 Family/genetics ; Biomarkers, Tumor/genetics ; Female ; Gene Expression Regulation, Neoplastic ; Humans ; Kaplan-Meier Estimate ; Middle Aged ; Neoplastic Stem Cells ; Ovarian Neoplasms/genetics ; Ovarian Neoplasms/mortality ; Ovarian Neoplasms/pathology ; Ovary/metabolism ; Ovary/pathology ; Prognosis ; Protein Interaction Maps ; Retinal Dehydrogenase/genetics ; Transcription Factors/genetics ; Young Adult
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
AMBRA1 Negatively Regulates the Function of ALDH1B1, a Cancer Stem Cell Marker, by Controlling Its Ubiquitination.
Autorzy:
Baek SH; Department of Systems Biology, College of Life Science and Biotechnology, Yonsei University, Seoul 03722, Korea.; BK21 Yonsei Education & Research Center for Biosystems, Yonsei University, Seoul 03722, Korea.
Jang YK; Department of Systems Biology, College of Life Science and Biotechnology, Yonsei University, Seoul 03722, Korea.; BK21 Yonsei Education & Research Center for Biosystems, Yonsei University, Seoul 03722, Korea.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2021 Nov 08; Vol. 22 (21). Date of Electronic Publication: 2021 Nov 08.
Typ publikacji:
Journal Article
MeSH Terms:
Adaptor Proteins, Signal Transducing/*metabolism
Aldehyde Dehydrogenase 1 Family/*metabolism
Aldehyde Dehydrogenase, Mitochondrial/*metabolism
Neoplastic Stem Cells/*pathology
TNF Receptor-Associated Factor 6/*metabolism
Ubiquitin-Protein Ligases/*metabolism
beta Catenin/*metabolism
Aldehyde Dehydrogenase 1 Family/antagonists & inhibitors ; Aldehyde Dehydrogenase, Mitochondrial/antagonists & inhibitors ; Cell Line ; Cell Line, Tumor ; Humans ; Neoplastic Stem Cells/metabolism ; Ubiquitination ; Wnt Signaling Pathway
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Presence and Potential Role of ALDH1A2 in the Glioblastoma Microenvironment.
Autorzy:
Sanders S; Department of Cancer Biology, Wake Forest School of Medicine, Winston Salem, NC 27157, USA.; Brain Tumor Center of Excellence, Wake Forest Baptist Medical Center Comprehensive Cancer Center, Winston Salem, NC 27157, USA.
Herpai DM; Department of Cancer Biology, Wake Forest School of Medicine, Winston Salem, NC 27157, USA.; Brain Tumor Center of Excellence, Wake Forest Baptist Medical Center Comprehensive Cancer Center, Winston Salem, NC 27157, USA.
Rodriguez A; Department of Neurosurgery, Jackson T. Stephens Spine and Neuroscience Institute, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA.; Winthrop P. Rockefeller Cancer Institute, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA.
Huang Y; Department of Cancer Biology, Wake Forest School of Medicine, Winston Salem, NC 27157, USA.; Brain Tumor Center of Excellence, Wake Forest Baptist Medical Center Comprehensive Cancer Center, Winston Salem, NC 27157, USA.
Chou J; Department of Cancer Biology, Wake Forest School of Medicine, Winston Salem, NC 27157, USA.
Hsu FC; Department of Biostatistics and Data Science, Wake Forest School of Medicine, Winston Salem, NC 27157, USA.
Seals D; Biology Department, Appalachian State University, Boone, NC 28608, USA.
Mott R; Department of Pathology, Wake Forest School of Medicine, Winston Salem, NC 27157, USA.
Miller LD; Department of Cancer Biology, Wake Forest School of Medicine, Winston Salem, NC 27157, USA.
Debinski W; Department of Cancer Biology, Wake Forest School of Medicine, Winston Salem, NC 27157, USA.; Brain Tumor Center of Excellence, Wake Forest Baptist Medical Center Comprehensive Cancer Center, Winston Salem, NC 27157, USA.
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Źródło:
Cells [Cells] 2021 Sep 20; Vol. 10 (9). Date of Electronic Publication: 2021 Sep 20.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation, Neoplastic*
Aldehyde Dehydrogenase 1 Family/*metabolism
Glioblastoma/*pathology
Macrophages/*immunology
Matrix Metalloproteinase 2/*metabolism
Matrix Metalloproteinase 9/*metabolism
Retinal Dehydrogenase/*metabolism
Aldehyde Dehydrogenase 1 Family/genetics ; Aldehyde Dehydrogenase 1 Family/immunology ; Apoptosis ; Brain Neoplasms/genetics ; Brain Neoplasms/immunology ; Brain Neoplasms/metabolism ; Brain Neoplasms/pathology ; Cell Movement ; Cell Proliferation ; Glioblastoma/genetics ; Glioblastoma/immunology ; Glioblastoma/metabolism ; Humans ; Matrix Metalloproteinase 2/genetics ; Matrix Metalloproteinase 9/genetics ; Retinal Dehydrogenase/genetics ; Retinal Dehydrogenase/immunology ; Tretinoin/metabolism ; Tumor Cells, Cultured ; Tumor Microenvironment
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
The Molecular Context of Oxidant Stress Response in Cancer Establishes ALDH1A1 as a Critical Target: What This Means for Acute Myeloid Leukemia.
Autorzy:
Dancik GM; Department of Computer Science, Eastern Connecticut State University, Willimantic, CT 06226, USA.
Varisli L; Department of Molecular Biology and Genetics, Science Faculty, Dicle University, Diyarbakir 21280, Turkey.
Vlahopoulos SA; First Department of Pediatrics, National and Kapodistrian University of Athens, Thivon & Levadeias 8, 11527 Athens, Greece.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 May 27; Vol. 24 (11). Date of Electronic Publication: 2023 May 27.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Leukemia, Myeloid, Acute*/genetics
Antineoplastic Agents*
Humans ; Mice ; Animals ; Oxidants ; Retinal Dehydrogenase/genetics ; Retinal Dehydrogenase/metabolism ; Aldehyde Dehydrogenase/genetics ; Aldehyde Dehydrogenase/metabolism ; Proteins ; RNA ; Aldehyde Dehydrogenase 1 Family
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Activation of ALDH1A1 by omeprazole reduces cell oxidative stress damage.
Autorzy:
Calleja LF; Departamento de Bioquímica, Instituto Nacional de Cardiología 'Ignacio Chávez', Ciudad de México, México.
Yoval-Sánchez B; Departamento de Bioquímica, Instituto Nacional de Cardiología 'Ignacio Chávez', Ciudad de México, México.
Hernández-Esquivel L; Departamento de Bioquímica, Instituto Nacional de Cardiología 'Ignacio Chávez', Ciudad de México, México.
Gallardo-Pérez JC; Departamento de Bioquímica, Instituto Nacional de Cardiología 'Ignacio Chávez', Ciudad de México, México.
Sosa-Garrocho M; Departamento de Biología Celular y Desarrollo, Instituto de Fisiología Celular, Universidad Nacional Autónoma de México, Ciudad de México, México.
Marín-Hernández Á; Departamento de Bioquímica, Instituto Nacional de Cardiología 'Ignacio Chávez', Ciudad de México, México.
Jasso-Chávez R; Departamento de Bioquímica, Instituto Nacional de Cardiología 'Ignacio Chávez', Ciudad de México, México.
Macías-Silva M; Departamento de Biología Celular y Desarrollo, Instituto de Fisiología Celular, Universidad Nacional Autónoma de México, Ciudad de México, México.
Salud Rodríguez-Zavala J; Departamento de Bioquímica, Instituto Nacional de Cardiología 'Ignacio Chávez', Ciudad de México, México.
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Źródło:
The FEBS journal [FEBS J] 2021 Jul; Vol. 288 (13), pp. 4064-4080. Date of Electronic Publication: 2021 Jan 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Aldehyde Dehydrogenase 1 Family/*metabolism
Lipid Peroxidation/*drug effects
Omeprazole/*pharmacology
Oxidative Stress/*drug effects
Aldehyde Dehydrogenase 1 Family/genetics ; Animals ; Cell Survival/drug effects ; Cells, Cultured ; Enzyme Activation/drug effects ; Enzyme Activation/radiation effects ; Female ; Humans ; Hydrogen Peroxide/pharmacology ; Light ; Oxidants/pharmacology ; Rats, Wistar ; Reactive Oxygen Species/metabolism ; Rats
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
ALDH1 expression predicts progression of premalignant lesions to cancer in Type I endometrial carcinomas.
Autorzy:
Mah V; 4525 MacDonald Research Laboratories, Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, CA, 90095, USA.
Elshimali Y; Division of Cancer Research and Training, Department of Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, USA.
Chu A; Department of Pediatrics, David Geffen School of Medicine at UCLA, Los Angeles, USA.
Moatamed NA; 4525 MacDonald Research Laboratories, Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, CA, 90095, USA.
Uzzell JP; 4525 MacDonald Research Laboratories, Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, CA, 90095, USA.
Tsui J; 4525 MacDonald Research Laboratories, Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, CA, 90095, USA.
Schettler S; 4525 MacDonald Research Laboratories, Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, CA, 90095, USA.
Shakeri H; 4525 MacDonald Research Laboratories, Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, CA, 90095, USA.
Wadehra M; 4525 MacDonald Research Laboratories, Department of Pathology and Laboratory Medicine, David Geffen School of Medicine at UCLA, Los Angeles, CA, 90095, USA. .; Division of Cancer Research and Training, Department of Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, USA. .; Jonsson Comprehensive Cancer Center, David Geffen School of Medicine at UCLA, Los Angeles, USA. .
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Źródło:
Scientific reports [Sci Rep] 2021 Jun 07; Vol. 11 (1), pp. 11949. Date of Electronic Publication: 2021 Jun 07.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Gene Expression Regulation, Neoplastic*
Aldehyde Dehydrogenase 1 Family/*genetics
Biomarkers, Tumor/*genetics
Endometrial Hyperplasia/*genetics
Endometrial Neoplasms/*genetics
Precancerous Conditions/*genetics
Adult ; Aged ; Aged, 80 and over ; Aldehyde Dehydrogenase 1 Family/metabolism ; Biomarkers, Tumor/metabolism ; Cell Line, Tumor ; Disease Progression ; Endometrial Hyperplasia/enzymology ; Endometrial Hyperplasia/pathology ; Endometrial Neoplasms/enzymology ; Endometrial Neoplasms/pathology ; Female ; Humans ; Kaplan-Meier Estimate ; Middle Aged ; Precancerous Conditions/enzymology ; Precancerous Conditions/pathology ; Prognosis
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Association study between genetic variants in retinol metabolism pathway genes and prostate cancer risk.
Autorzy:
Cao D; Department of Urology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, People's Republic of China.
Meng Y; Department of Genetic Toxicology, The Key Laboratory of Modern Toxicology of Ministry of Education, School of Public Health, Nanjing Medical University, Nanjing, Jiangsu, People's Republic of China.
Li S; Department of Genetic Toxicology, The Key Laboratory of Modern Toxicology of Ministry of Education, School of Public Health, Nanjing Medical University, Nanjing, Jiangsu, People's Republic of China.
Xin J; Department of Genetic Toxicology, The Key Laboratory of Modern Toxicology of Ministry of Education, School of Public Health, Nanjing Medical University, Nanjing, Jiangsu, People's Republic of China.
Ben S; Department of Genetic Toxicology, The Key Laboratory of Modern Toxicology of Ministry of Education, School of Public Health, Nanjing Medical University, Nanjing, Jiangsu, People's Republic of China.
Cheng Y; Department of Urology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, People's Republic of China.
Wang M; Department of Genetic Toxicology, The Key Laboratory of Modern Toxicology of Ministry of Education, School of Public Health, Nanjing Medical University, Nanjing, Jiangsu, People's Republic of China.
Hua L; Department of Urology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, People's Republic of China.
Cheng G; Department of Urology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, People's Republic of China.
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Źródło:
Cancer medicine [Cancer Med] 2020 Dec; Vol. 9 (24), pp. 9462-9470. Date of Electronic Publication: 2020 Oct 17.
Typ publikacji:
Journal Article
MeSH Terms:
Polymorphism, Single Nucleotide*
Aldehyde Dehydrogenase 1 Family/*metabolism
Prostatic Neoplasms/*genetics
Prostatic Neoplasms/*metabolism
Retinal Dehydrogenase/*metabolism
Vitamin A/*metabolism
Aged ; Aldehyde Dehydrogenase 1 Family/genetics ; Computational Biology/methods ; Databases, Genetic ; Genetic Predisposition to Disease ; Humans ; Male ; Neoplasm Staging ; Prostatic Neoplasms/pathology ; Quantitative Trait Loci ; Retinal Dehydrogenase/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
High expression of aldehyde dehydrogenase 1 and tissue necrosis factor alpha may relate to chronic infection of buffalo mammary gland.
Autorzy:
Thakur S; School of Animal Biotechnology, Guru Angad Dev Veterinary and Animal Sciences University, Ludhiana, India.
Choudhary S; School of Animal Biotechnology, Guru Angad Dev Veterinary and Animal Sciences University, Ludhiana, India.
Pathak D; Department of Veterinary Anatomy, College of Veterinary Science, Guru Angad Dev Veterinary and Animal Sciences University, Ludhiana, India.
Choudhary RK; School of Animal Biotechnology, Guru Angad Dev Veterinary and Animal Sciences University, Ludhiana, India.; Department of Animal and Veterinary Sciences, University of Vermont, Burlington, New Jersey, USA.
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Źródło:
Animal biotechnology [Anim Biotechnol] 2020 Jun; Vol. 31 (3), pp. 276-281. Date of Electronic Publication: 2019 Mar 04.
Typ publikacji:
Journal Article
MeSH Terms:
Mammary Glands, Animal*/chemistry
Mammary Glands, Animal*/enzymology
Mammary Glands, Animal*/metabolism
Mastitis, Bovine*/enzymology
Mastitis, Bovine*/metabolism
Aldehyde Dehydrogenase 1 Family/*metabolism
Hepatocyte Nuclear Factor 4/*metabolism
Tumor Necrosis Factor-alpha/*metabolism
Aldehyde Dehydrogenase 1 Family/genetics ; Animals ; Buffaloes/genetics ; Buffaloes/metabolism ; Cattle ; Hepatocyte Nuclear Factor 4/genetics ; Tumor Necrosis Factor-alpha/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Prognostic Value of Cancer Stem Cell Markers (CSCs) Expression-ALDH1A1, CD133, CD44-For Survival and Long-Term Follow-Up of Ovarian Cancer Patients.
Autorzy:
Izycka N; Department of Gynecology, Obstetrics and Gynecologic Oncology, Division of Gynecologic Oncology, Poznan University of Medical Sciences, Polna 33 St., 60-535 Poznań, Poland.
Rucinski M; Department of Histology and Embryology, Poznan University of Medical Sciences, Święcickiego 6 St., 61-781 Poznań, Poland.
Andrzejewska M; Department of Histology and Embryology, Poznan University of Medical Sciences, Święcickiego 6 St., 61-781 Poznań, Poland.
Szubert S; Department of Gynecology, Obstetrics and Gynecologic Oncology, Division of Gynecologic Oncology, Poznan University of Medical Sciences, Polna 33 St., 60-535 Poznań, Poland.
Nowak-Markwitz E; Department of Gynecology, Obstetrics and Gynecologic Oncology, Division of Gynecologic Oncology, Poznan University of Medical Sciences, Polna 33 St., 60-535 Poznań, Poland.
Sterzynska K; Department of Histology and Embryology, Poznan University of Medical Sciences, Święcickiego 6 St., 61-781 Poznań, Poland.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Jan 25; Vol. 24 (3). Date of Electronic Publication: 2023 Jan 25.
Typ publikacji:
Journal Article
MeSH Terms:
Hyaluronan Receptors*/metabolism
Ovarian Neoplasms*/diagnosis
Ovarian Neoplasms*/drug therapy
Ovarian Neoplasms*/metabolism
Female ; Humans ; Aldehyde Dehydrogenase 1 Family/metabolism ; Biomarkers, Tumor/metabolism ; Carcinoma, Ovarian Epithelial/pathology ; Follow-Up Studies ; Neoplastic Stem Cells/metabolism ; Prognosis ; Retinal Dehydrogenase/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
ALDH1-Mediated Autophagy Sensitizes Glioblastoma Cells to Ferroptosis.
Autorzy:
Wu Y; Department of Neuropathology, Institute of Pathology, School of Medicine, Technical University Munich, 81675 Munich, Germany.
Kram H; Department of Neuropathology, Institute of Pathology, School of Medicine, Technical University Munich, 81675 Munich, Germany.
Gempt J; Department of Neurosurgery, Klinikum Rechts der Isar, School of Medicine, Technical University Munich, 81675 Munich, Germany.
Liesche-Starnecker F; Institute of Pathology and Molecular Diagnostics, Medical Faculty, University of Augsburg, 86156 Augsburg, Germany.
Wu W; Department of Radiology, Molecular Imaging Program at Stanford, Stanford University, Stanford, CA 94305, USA.
Schlegel J; Department of Neuropathology, Institute of Pathology, School of Medicine, Technical University Munich, 81675 Munich, Germany.
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Źródło:
Cells [Cells] 2022 Dec 12; Vol. 11 (24). Date of Electronic Publication: 2022 Dec 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Ferroptosis*/genetics
Glioblastoma*/genetics
Glioblastoma*/metabolism
Humans ; Aldehyde Dehydrogenase 1 Family ; Autophagy ; Glioma/metabolism ; Neoplasm Recurrence, Local
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Exploratory Metabolomics Underscores the Folate Enzyme ALDH1L1 as a Regulator of Glycine and Methylation Reactions.
Autorzy:
Rushing BR; Nutrition Research Institute, UNC Chapel Hill, Kannapolis, NC 28081, USA.; Department of Nutrition, UNC Chapel Hill, Chapel Hill, NC 27599, USA.
Fogle HM; Nutrition Research Institute, UNC Chapel Hill, Kannapolis, NC 28081, USA.; Department of Nutrition, UNC Chapel Hill, Chapel Hill, NC 27599, USA.
Sharma J; Nutrition Research Institute, UNC Chapel Hill, Kannapolis, NC 28081, USA.
You M; Nutrition Research Institute, UNC Chapel Hill, Kannapolis, NC 28081, USA.
McCormac JP; Nutrition Research Institute, UNC Chapel Hill, Kannapolis, NC 28081, USA.
Molina S; Nutrition Research Institute, UNC Chapel Hill, Kannapolis, NC 28081, USA.
Sumner S; Nutrition Research Institute, UNC Chapel Hill, Kannapolis, NC 28081, USA.; Department of Nutrition, UNC Chapel Hill, Chapel Hill, NC 27599, USA.
Krupenko NI; Nutrition Research Institute, UNC Chapel Hill, Kannapolis, NC 28081, USA.; Department of Nutrition, UNC Chapel Hill, Chapel Hill, NC 27599, USA.
Krupenko SA; Nutrition Research Institute, UNC Chapel Hill, Kannapolis, NC 28081, USA.; Department of Nutrition, UNC Chapel Hill, Chapel Hill, NC 27599, USA.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2022 Dec 01; Vol. 27 (23). Date of Electronic Publication: 2022 Dec 01.
Typ publikacji:
Journal Article
MeSH Terms:
Folic Acid*/metabolism
Neoplasms*
Humans ; Glycine/metabolism ; Retinal Dehydrogenase/metabolism ; Methylation ; Aldehyde Dehydrogenase 1 Family/metabolism ; S-Adenosylmethionine/metabolism ; Metabolomics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Chromium (VI)-induced ALDH1A1/EGF axis promotes lung cancer progression.
Autorzy:
Ge X; Department of Nutrition and Food Hygiene, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, Jiangsu, China.; Jiangsu Key Lab of Cancer Biomarkers, Prevention and Treatment, Jiangsu Collaborative Innovation Center for Cancer Personalized Medicine, Nanjing Medical University, Nanjing, Jiangsu, China.; The Key Laboratory of Modern Toxicology of Ministry of Education, Nanjing Medical University, Nanjing, Jiangsu, China.
Li M; Department of Nutrition and Food Hygiene, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, Jiangsu, China.; Jiangsu Key Lab of Cancer Biomarkers, Prevention and Treatment, Jiangsu Collaborative Innovation Center for Cancer Personalized Medicine, Nanjing Medical University, Nanjing, Jiangsu, China.
Song GX; Department of Pathology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, China.
Zhang Z; Department of Nutrition and Food Hygiene, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, Jiangsu, China.; Jiangsu Key Lab of Cancer Biomarkers, Prevention and Treatment, Jiangsu Collaborative Innovation Center for Cancer Personalized Medicine, Nanjing Medical University, Nanjing, Jiangsu, China.
Yin J; Department of Neurosurgery, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China.
Ge Z; Department of Nutrition and Food Hygiene, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, Jiangsu, China.; Jiangsu Key Lab of Cancer Biomarkers, Prevention and Treatment, Jiangsu Collaborative Innovation Center for Cancer Personalized Medicine, Nanjing Medical University, Nanjing, Jiangsu, China.
Shi Z; Department of Neurosurgery, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China.
Liu LZ; Department of Pathology, Anatomy and Cell Biology, Department of Medical Oncology, Thomas Jefferson University, Philadelphia, Pennsylvania, USA.
Jiang BH; The Academy of Medical Science, Zhengzhou University, Zhengzhou, 450000, China.
Qian X; Department of Nutrition and Food Hygiene, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing, Jiangsu, China.; Jiangsu Key Lab of Cancer Biomarkers, Prevention and Treatment, Jiangsu Collaborative Innovation Center for Cancer Personalized Medicine, Nanjing Medical University, Nanjing, Jiangsu, China.; The Key Laboratory of Modern Toxicology of Ministry of Education, Nanjing Medical University, Nanjing, Jiangsu, China.
Shen H; Department of Oncology, The First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, China.; Department of Oncology, Sir Run Run Hospital, Nanjing Medical University, Nanjing, Jiangsu, China.
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Źródło:
Clinical and translational medicine [Clin Transl Med] 2022 Dec; Vol. 12 (12), pp. e1136.
Typ publikacji:
Journal Article
MeSH Terms:
Lung Neoplasms*/genetics
Carcinoma, Non-Small-Cell Lung*
Carcinoma, Squamous Cell*
Tics*
Humans ; Aldehyde Dehydrogenase/genetics ; Epidermal Growth Factor ; Neoplastic Processes ; Carcinogenesis ; Cell Transformation, Neoplastic/genetics ; Lung ; Aldehyde Dehydrogenase 1 Family ; Retinal Dehydrogenase/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Quantitative Proteomic Analysis of Zearalenone Exposure on Uterine Development in Weaned Gilts.
Autorzy:
Liu X; Colloge of Animal Sciences and Veterinary Medicine, Shandong Agricultural University, No. 61 Daizong Street, Tai'an 271018, China.
Wang Z; Shandong Muge Animal Husbandry Equipment Co., Ltd., Yucheng 251200, China.
Jiang Y; Zhongcheng Feed Technology Co., Ltd., Feicheng 271600, China.
Huang L; Colloge of Animal Sciences and Veterinary Medicine, Shandong Agricultural University, No. 61 Daizong Street, Tai'an 271018, China.
Yuan X; Department of Life Sciences, Shandong Agricultural University, No. 61 Daizong Street, Tai'an 271018, China.
Li Y; Colloge of Animal Sciences and Veterinary Medicine, Shandong Agricultural University, No. 61 Daizong Street, Tai'an 271018, China.
Jiao N; Colloge of Animal Sciences and Veterinary Medicine, Shandong Agricultural University, No. 61 Daizong Street, Tai'an 271018, China.
Yang W; Colloge of Animal Sciences and Veterinary Medicine, Shandong Agricultural University, No. 61 Daizong Street, Tai'an 271018, China.
Jiang S; Colloge of Animal Sciences and Veterinary Medicine, Shandong Agricultural University, No. 61 Daizong Street, Tai'an 271018, China.
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Źródło:
Toxins [Toxins (Basel)] 2022 Oct 09; Vol. 14 (10). Date of Electronic Publication: 2022 Oct 09.
Typ publikacji:
Journal Article; Randomized Controlled Trial, Veterinary; Research Support, Non-U.S. Gov't
MeSH Terms:
Zearalenone*/toxicity
Animals ; Female ; Aldehyde Dehydrogenase 1 Family ; Chromogranins ; Creatine Kinase ; Gonadotropin-Releasing Hormone ; Luteinizing Hormone ; Neprilysin ; Peroxidase ; Phosphatidylinositol 3-Kinase ; Phosphatidylinositol 3-Kinases ; Proteome ; Proteomics ; Proto-Oncogene Proteins c-akt ; Sus scrofa ; Swine
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Aldehyde Dehydrogenase 1B1 Is Implicated in DNA Damage Response in Human Colorectal Adenocarcinoma.
Autorzy:
Tsochantaridis I; Department of Molecular Biology & Genetics, Faculty of Health Sciences, Democritus University of Thrace, 68100 Alexandroupolis, Greece.
Kontopoulos A; Department of Molecular Biology & Genetics, Faculty of Health Sciences, Democritus University of Thrace, 68100 Alexandroupolis, Greece.
Voulgaridou GP; Department of Molecular Biology & Genetics, Faculty of Health Sciences, Democritus University of Thrace, 68100 Alexandroupolis, Greece.
Tsifintaris M; Department of Molecular Biology & Genetics, Faculty of Health Sciences, Democritus University of Thrace, 68100 Alexandroupolis, Greece.
Triantafyllou C; Department of Molecular Biology & Genetics, Faculty of Health Sciences, Democritus University of Thrace, 68100 Alexandroupolis, Greece.
Pappa A; Department of Molecular Biology & Genetics, Faculty of Health Sciences, Democritus University of Thrace, 68100 Alexandroupolis, Greece.
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Źródło:
Cells [Cells] 2022 Jun 24; Vol. 11 (13). Date of Electronic Publication: 2022 Jun 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adenocarcinoma*/genetics
Colorectal Neoplasms*/genetics
Colorectal Neoplasms*/pathology
Aldehyde Dehydrogenase 1 Family/*metabolism
Aldehyde Dehydrogenase, Mitochondrial/*metabolism
Aldehyde Dehydrogenase/metabolism ; Aldehyde Dehydrogenase, Mitochondrial/genetics ; DNA Damage ; DNA-Binding Proteins/metabolism ; Humans ; Ubiquitin-Protein Ligases/metabolism ; X-ray Repair Cross Complementing Protein 1/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Butylidenephthalide Abrogates the Snail-Induced Cancer Stemness in Oral Carcinomas.
Autorzy:
Chen PY; School of Dentistry, Chung Shan Medical University, Taichung 402, Taiwan.; Department of Dentistry, Chung Shan Medical University Hospital, Taichung 402, Taiwan.
Chao SC; Institute of Oral Sciences, Chung Shan Medical University, Taichung 402, Taiwan.; Department of Medical Research and Education, Lo-Hsu Medical Foundation, Lotung Poh-Ai Hospital, Yilan 265, Taiwan.
Hsieh PL; Department of Anatomy, School of Medicine, China Medical University, Taichung 404, Taiwan.
Liao YW; Department of Medical Research, Chung Shan Medical University Hospital, Taichung 402, Taiwan.
Chu PM; Department of Anatomy, School of Medicine, China Medical University, Taichung 404, Taiwan.
Harn HJ; Buddhist Tzu Chi Bioinnovation Center, Tzu Chi Foundation, Hualien 970, Taiwan.; Bioinnovation Center, Buddhist Tzu Chi Medical Foundation, Hualien 970, Taiwan.; Department of Pathology, Buddhist Tzu Chi General Hospital and Tzu Chi University, Hualien 970, Taiwan.
Yu CC; School of Dentistry, Chung Shan Medical University, Taichung 402, Taiwan.; Department of Dentistry, Chung Shan Medical University Hospital, Taichung 402, Taiwan.; Institute of Oral Sciences, Chung Shan Medical University, Taichung 402, Taiwan.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 May 31; Vol. 23 (11). Date of Electronic Publication: 2022 May 31.
Typ publikacji:
Journal Article
MeSH Terms:
Carcinoma*/metabolism
Mouth Neoplasms*/pathology
Aldehyde Dehydrogenase 1 Family ; Animals ; Cell Line, Tumor ; Humans ; Hyaluronan Receptors/metabolism ; Isoenzymes/metabolism ; Mice ; Neoplasm Recurrence, Local/pathology ; Neoplastic Stem Cells/metabolism ; Phthalic Anhydrides ; Retinal Dehydrogenase/metabolism ; Snail Family Transcription Factors/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Triple-negative expression (ALDH1A1-/CD133-/mutant p53-) cases in lung adenocarcinoma had a good prognosis.
Autorzy:
Yamashita N; Department of Education for Medical Research Base, Hyogo College of Medicine, 1-1 Mukogawa-cho, Nishinomiya, 663-8501, Japan.; Department of Thoracic Surgery, Shin-Komonji Hospital, Fukuoka, Japan.
So T; Department of Thoracic Surgery, Shin-Komonji Hospital, Fukuoka, Japan.
Miyata T; Department of Thoracic Surgery, Shin-Kuki General Hospital, Saitama, Japan.
Yoshimatsu T; Department of Thoracic Surgery, Fukuoka-Wajiro Hospital, Fukuoka, Japan.
Nakano R; Department of Pathology, Fukuoka-Wajiro Hospital, Fukuoka, Japan.
Oyama T; Department of Education for Medical Research Base, Hyogo College of Medicine, 1-1 Mukogawa-cho, Nishinomiya, 663-8501, Japan.; Imamitsu Home Care Clinic, Kitakyusyu, Japan.
Matsunaga W; Department of Education for Medical Research Base, Hyogo College of Medicine, 1-1 Mukogawa-cho, Nishinomiya, 663-8501, Japan.
Gotoh A; Department of Education for Medical Research Base, Hyogo College of Medicine, 1-1 Mukogawa-cho, Nishinomiya, 663-8501, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2022 Jan 27; Vol. 12 (1), pp. 1473. Date of Electronic Publication: 2022 Jan 27.
Typ publikacji:
Journal Article
MeSH Terms:
Adenocarcinoma of Lung/*mortality
Biomarkers, Tumor/*analysis
Lung/*pathology
Lung Neoplasms/*mortality
AC133 Antigen/analysis ; AC133 Antigen/metabolism ; Adenocarcinoma of Lung/pathology ; Adenocarcinoma of Lung/surgery ; Aged ; Aldehyde Dehydrogenase 1 Family/analysis ; Aldehyde Dehydrogenase 1 Family/metabolism ; Biomarkers, Tumor/genetics ; Biomarkers, Tumor/metabolism ; Female ; Humans ; Immunohistochemistry ; Kaplan-Meier Estimate ; Lung/surgery ; Lung Neoplasms/pathology ; Lung Neoplasms/surgery ; Male ; Mutation ; Pneumonectomy ; Prognosis ; Retinal Dehydrogenase/analysis ; Retinal Dehydrogenase/metabolism ; Retrospective Studies ; Risk Assessment/methods ; Tumor Suppressor Protein p53/analysis ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Generation of Cancer Stem/Initiating Cells by Cell-Cell Fusion.
Autorzy:
Dittmar T; Institute of Immunology, Center for Biomedical Education and Research (ZBAF), University of Witten/Herdecke, Stockumer Str. 10, 58448 Witten, Germany.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Apr 19; Vol. 23 (9). Date of Electronic Publication: 2022 Apr 19.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Neoplasms*/metabolism
Neoplastic Stem Cells*/metabolism
Aldehyde Dehydrogenase 1 Family ; Biomarkers/metabolism ; Cell Communication ; Cell Fusion ; Humans
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Wyświetlanie 1-20 z 603

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