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Tytuł:
Molecular Mechanisms of Macroautophagy, Microautophagy, and Chaperone-Mediated Autophagy.
Autorzy:
Yamamoto H; Department of Molecular Oncology, Institute for Advanced Medical Sciences, Nippon Medical School.
Matsui T; Department of Molecular Oncology, Institute for Advanced Medical Sciences, Nippon Medical School.
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Źródło:
Journal of Nippon Medical School = Nippon Ika Daigaku zasshi [J Nippon Med Sch] 2024 Mar 09; Vol. 91 (1), pp. 2-9. Date of Electronic Publication: 2023 Jun 02.
Typ publikacji:
Review; Journal Article
MeSH Terms:
Chaperone-Mediated Autophagy*
Neurodegenerative Diseases*/metabolism
Humans ; Microautophagy ; Macroautophagy ; Autophagy
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Insights on E1-like enzyme ATG7: functional regulation and relationships with aging-related diseases.
Autorzy:
Liu J; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China.; Department of Anus and Intestine Surgery, First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China.
Xiao Y; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China.
Cao L; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China.
Lu S; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China.
Zhang S; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China.
Yang R; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China.
Wang Y; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China.
Zhang N; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Department of Cardiology, First Hospital of China Medical University, Key Laboratory of Environmental Stress and Chronic Disease Control and Prevention, Ministry of Education, China Medical University, Shenyang, Liaoning, China.
Yu Y; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China.
Wang X; Department of Thoracic Surgery, Shengjing Hospital of China Medical University, Shenyang, Liaoning, China.
Guo W; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China.
Wang Z; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China.; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China.
Xu H; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China. .; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China. .
Xing C; Department of Anus and Intestine Surgery, First Affiliated Hospital of China Medical University, Shenyang, Liaoning, China. .
Song X; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China. .; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China. .
Cao L; The College of Basic Medical Science, Health Sciences Institute, China Medical University, Shenyang, Liaoning, China. .; Key Laboratory of Cell Biology of Ministry of Public Health, Key Laboratory of Medical Cell Biology of Ministry of Education, Key Laboratory of Precision Diagnosis and Treatment of Gastrointestinal Tumors (China Medical University), Ministry of Education, Liaoning Province Collaborative Innovation Center of Aging Related Disease Diagnosis and Treatment and Prevention, China Medical University, Shenyang, Liaoning, China. .
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Źródło:
Communications biology [Commun Biol] 2024 Mar 29; Vol. 7 (1), pp. 382. Date of Electronic Publication: 2024 Mar 29.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Autophagosomes*/metabolism
Autophagy*/genetics
Autophagy-Related Protein 7/genetics ; Ubiquitin-Activating Enzymes/metabolism ; Ubiquitin-Conjugating Enzymes/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Lipid droplet accumulation in Wdr45-deficient cells caused by impairment of chaperone-mediated autophagic degradation of Fasn.
Autorzy:
Xiong Q; Institutes of Biomedical Sciences, Shanxi Provincial Key Laboratory for Medical Molecular Cell Biology, Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education, Shanxi University, Taiyuan, 030006, China. .
Sun H; Institutes of Biomedical Sciences, Shanxi Provincial Key Laboratory for Medical Molecular Cell Biology, Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education, Shanxi University, Taiyuan, 030006, China.
Wang Y; Institutes of Biomedical Sciences, Shanxi Provincial Key Laboratory for Medical Molecular Cell Biology, Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education, Shanxi University, Taiyuan, 030006, China.
Xu Q; Institutes of Biomedical Sciences, Shanxi Provincial Key Laboratory for Medical Molecular Cell Biology, Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education, Shanxi University, Taiyuan, 030006, China.
Zhang Y; Institutes of Biomedical Sciences, Shanxi Provincial Key Laboratory for Medical Molecular Cell Biology, Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education, Shanxi University, Taiyuan, 030006, China.
Xu M; Institutes of Biomedical Sciences, Shanxi Provincial Key Laboratory for Medical Molecular Cell Biology, Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education, Shanxi University, Taiyuan, 030006, China.
Zhao Z; Institutes of Biomedical Sciences, Shanxi Provincial Key Laboratory for Medical Molecular Cell Biology, Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education, Shanxi University, Taiyuan, 030006, China.
Li P; Institutes of Biomedical Sciences, Shanxi Provincial Key Laboratory for Medical Molecular Cell Biology, Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education, Shanxi University, Taiyuan, 030006, China. .
Wu C; Institutes of Biomedical Sciences, Shanxi Provincial Key Laboratory for Medical Molecular Cell Biology, Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education, Shanxi University, Taiyuan, 030006, China. .
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Źródło:
Lipids in health and disease [Lipids Health Dis] 2024 Mar 28; Vol. 23 (1), pp. 91. Date of Electronic Publication: 2024 Mar 28.
Typ publikacji:
Journal Article
MeSH Terms:
Chaperone-Mediated Autophagy*
Neurodegenerative Diseases*/genetics
Neurodegenerative Diseases*/metabolism
Humans ; Carrier Proteins/genetics ; Carrier Proteins/metabolism ; Lipid Droplets/metabolism ; Autophagy/genetics ; Fatty Acid Synthases/metabolism ; Lipids
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Involvement of Lipophagy and Chaperone-Mediated Autophagy in the Pathogenesis of Non-Alcoholic Fatty Liver Disease by Regulation of Lipid Droplets.
Autorzy:
Mastoridou EM; Department of Anatomy-Histology-Embryology, Faculty of Medicine, School of Health Sciences, University of Ioannina, 45110 Ioannina, Greece.
Goussia AC; Department of Pathology, Faculty of Medicine, School of Health Sciences, University of Ioannina, 45110 Ioannina, Greece.
Kanavaros P; Department of Anatomy-Histology-Embryology, Faculty of Medicine, School of Health Sciences, University of Ioannina, 45110 Ioannina, Greece.
Charchanti AV; Department of Anatomy-Histology-Embryology, Faculty of Medicine, School of Health Sciences, University of Ioannina, 45110 Ioannina, Greece.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Nov 02; Vol. 24 (21). Date of Electronic Publication: 2023 Nov 02.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Non-alcoholic Fatty Liver Disease*/metabolism
Chaperone-Mediated Autophagy*
Humans ; Lipid Droplets/metabolism ; Lipid Metabolism ; Hepatocytes/metabolism ; Autophagy/physiology ; Perilipins/metabolism ; Liver/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The Intrinsically Disordered N Terminus in Atg12 from Yeast Is Necessary for the Functional Structure of the Protein.
Autorzy:
Popelka H; Life Sciences Institute, University of Michigan, Ann Arbor, MI 48109, USA.
Lahiri V; Life Sciences Institute, University of Michigan, Ann Arbor, MI 48109, USA.; Department of Molecular, Cellular and Developmental Biology, University of Michigan, Ann Arbor, MI 48109, USA.
Hawkins WD; Life Sciences Institute, University of Michigan, Ann Arbor, MI 48109, USA.; Department of Molecular, Cellular and Developmental Biology, University of Michigan, Ann Arbor, MI 48109, USA.
da Veiga Leprevost F; Department of Pathology, Michigan Medicine, University of Michigan, Ann Arbor, MI 48109, USA.
Nesvizhskii AI; Department of Pathology, Michigan Medicine, University of Michigan, Ann Arbor, MI 48109, USA.
Klionsky DJ; Life Sciences Institute, University of Michigan, Ann Arbor, MI 48109, USA.; Department of Molecular, Cellular and Developmental Biology, University of Michigan, Ann Arbor, MI 48109, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Oct 10; Vol. 24 (20). Date of Electronic Publication: 2023 Oct 10.
Typ publikacji:
Journal Article
MeSH Terms:
Autophagy-Related Protein 12*
Saccharomyces cerevisiae*/genetics
Saccharomyces cerevisiae*/metabolism
Saccharomyces cerevisiae Proteins*/metabolism
Autophagy ; Autophagy-Related Protein 5 ; Autophagy-Related Proteins/genetics ; Ubiquitin/genetics ; Ubiquitin/metabolism ; Ubiquitin-Protein Ligases
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Integrated Multi-Omics Analyses Reveal That Autophagy-Mediated Cellular Metabolism Is Required for the Initiation of Pollen Germination.
Autorzy:
Zhou X; College of Life Sciences, South-Central Minzu University, Wuhan 430074, China.
Zhang Q; College of Life Sciences, South-Central Minzu University, Wuhan 430074, China.
Zhao Y; College of Life Sciences, South-Central Minzu University, Wuhan 430074, China.
Ding S; College of Life Sciences, South-Central Minzu University, Wuhan 430074, China.
Yu GH; College of Life Sciences, South-Central Minzu University, Wuhan 430074, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Oct 09; Vol. 24 (19). Date of Electronic Publication: 2023 Oct 09.
Typ publikacji:
Journal Article
MeSH Terms:
Multiomics*
Autophagy*/genetics
Autophagy-Related Proteins/genetics ; Autophagy-Related Protein 12/genetics ; Pollen/genetics ; Pollen/metabolism ; Germination/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Targeting RPA promotes autophagic flux and the antitumor response to radiation in nasopharyngeal carcinoma.
Autorzy:
Feng Y; Department of Clinical Laboratory, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Institute of Gastroenterology, Guangzhou, 510655, China.; Department of General Surgery, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.
Jiang Y; Guangdong Institute of Gastroenterology, Guangzhou, 510655, China.; Department of General Surgery, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Department of General Surgery (Department of Gastric Surgery Section 2, ), The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.
Liu J; Department of Clinical Laboratory, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Institute of Gastroenterology, Guangzhou, 510655, China.; Department of General Surgery, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.
Liu J; Department of Clinical Laboratory, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Institute of Gastroenterology, Guangzhou, 510655, China.; Department of General Surgery, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.
Shi M; Department of Clinical Laboratory, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Institute of Gastroenterology, Guangzhou, 510655, China.; Department of General Surgery, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.
Chen J; Department of Clinical Laboratory, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Institute of Gastroenterology, Guangzhou, 510655, China.; Department of General Surgery, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.
Zhang J; Department of Clinical Laboratory, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Institute of Gastroenterology, Guangzhou, 510655, China.; Department of General Surgery, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.
Tian Y; Department of Clinical Laboratory, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Institute of Gastroenterology, Guangzhou, 510655, China.; Department of General Surgery, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China.
Yang X; Department of Clinical Laboratory, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China. .; Guangdong Institute of Gastroenterology, Guangzhou, 510655, China. .; Department of General Surgery, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China. .; Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China. .; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China. .
Liu H; Department of Clinical Laboratory, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China. .; Guangdong Institute of Gastroenterology, Guangzhou, 510655, China. .; Department of General Surgery, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China. .; Guangdong Provincial Key Laboratory of Colorectal and Pelvic Floor Diseases, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China. .; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510655, China. .
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Źródło:
Journal of translational medicine [J Transl Med] 2023 Oct 19; Vol. 21 (1), pp. 738. Date of Electronic Publication: 2023 Oct 19.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antineoplastic Agents*/therapeutic use
Autophagy*/drug effects
Autophagy*/genetics
Nasopharyngeal Carcinoma*/drug therapy
Nasopharyngeal Carcinoma*/genetics
Nasopharyngeal Carcinoma*/radiotherapy
Nasopharyngeal Neoplasms*/drug therapy
Nasopharyngeal Neoplasms*/genetics
Nasopharyngeal Neoplasms*/radiotherapy
Radiation Tolerance*/drug effects
Radiation Tolerance*/genetics
Replication Protein A*/antagonists & inhibitors
Replication Protein A*/genetics
Humans ; Apoptosis ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; Autophagy-Related Protein 5/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Model Organisms to Study Autophagy.
Autorzy:
Xiong Q; Shanxi Provincial Key Laboratory of Medical Molecular Cell Biology, Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education, Institutes of Biomedical Sciences, Shanxi University, No. 92 Wucheng Road, Taiyuan 030006, China.
Eichinger L; Center for Biochemistry, Medical Faculty, University of Cologne, Joseph-Stelzmann-Str. 52, 50931 Cologne, Germany.
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Źródło:
Cells [Cells] 2023 Sep 05; Vol. 12 (18). Date of Electronic Publication: 2023 Sep 05.
Typ publikacji:
Editorial; Research Support, Non-U.S. Gov't
MeSH Terms:
Autophagy*
Eukaryotic Cells*
Humans ; Autophagy-Related Proteins ; Kinetics
Raport
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interactome of Arabidopsis ATG5 Suggests Functions beyond Autophagy.
Autorzy:
Elander PH; Department of Molecular Sciences, Uppsala BioCenter, Swedish University of Agricultural Sciences and Linnean Center for Plant Biology, 750-07 Uppsala, Sweden.
Holla S; Department of Molecular Sciences, Uppsala BioCenter, Swedish University of Agricultural Sciences and Linnean Center for Plant Biology, 750-07 Uppsala, Sweden.
Sabljić I; Department of Molecular Sciences, Uppsala BioCenter, Swedish University of Agricultural Sciences and Linnean Center for Plant Biology, 750-07 Uppsala, Sweden.
Gutierrez-Beltran E; Instituto de Bioquımica Vegetal y Fotosıntesis, Universidad de Sevilla and Consejo Superior de Investigaciones Cientıficas, 41092 Sevilla, Spain.; Departamento de Bioquimica Vegetal y Biologia Molecular, Facultad de Biologia, Universidad de Sevilla, 41012 Sevilla, Spain.
Willems P; Department of Plant Biotechnology and Bioinformatics, Ghent University, 9052 Ghent, Belgium.; Department of Biomolecular Medicine, Ghent University, 9000 Ghent, Belgium.
Bozhkov PV; Department of Molecular Sciences, Uppsala BioCenter, Swedish University of Agricultural Sciences and Linnean Center for Plant Biology, 750-07 Uppsala, Sweden.
Minina EA; Department of Molecular Sciences, Uppsala BioCenter, Swedish University of Agricultural Sciences and Linnean Center for Plant Biology, 750-07 Uppsala, Sweden.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Aug 01; Vol. 24 (15). Date of Electronic Publication: 2023 Aug 01.
Typ publikacji:
Journal Article
MeSH Terms:
Arabidopsis*/metabolism
Autophagy-Related Protein 5*/genetics
Autophagy-Related Protein 5*/metabolism
Autophagy/genetics ; Chromatography, Liquid ; Tandem Mass Spectrometry
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Activation of Chaperone-Mediated Autophagy Inhibits the Aryl Hydrocarbon Receptor Function by Degrading This Receptor in Human Lung Epithelial Carcinoma A549 Cells.
Autorzy:
Xiong R; Department of Pharmaceutics & Medicinal Chemistry, Thomas J. Long School of Pharmacy, University of the Pacific, Stockton, CA 95211, USA.
Shao D; Department of Pharmaceutics & Medicinal Chemistry, Thomas J. Long School of Pharmacy, University of the Pacific, Stockton, CA 95211, USA.
Do S; Department of Pharmaceutics & Medicinal Chemistry, Thomas J. Long School of Pharmacy, University of the Pacific, Stockton, CA 95211, USA.
Chan WK; Department of Pharmaceutics & Medicinal Chemistry, Thomas J. Long School of Pharmacy, University of the Pacific, Stockton, CA 95211, USA.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Oct 12; Vol. 24 (20). Date of Electronic Publication: 2023 Oct 12.
Typ publikacji:
Journal Article
MeSH Terms:
Carcinoma*
Chaperone-Mediated Autophagy*
Lung Neoplasms*/metabolism
Humans ; A549 Cells ; Autophagy/genetics ; Chloroquine/pharmacology ; Ligands ; Lung/pathology ; Receptors, Aryl Hydrocarbon/genetics ; Receptors, Aryl Hydrocarbon/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
miR-30a-3p Regulates Autophagy in the Involution of Mice Mammary Glands.
Autorzy:
Tian L; Faculty of Food Science and Engineering, Kunming University of Science and Technology, Kunming 650500, China.
Guo S; Faculty of Food Science and Engineering, Kunming University of Science and Technology, Kunming 650500, China.
Zhao Z; Faculty of Food Science and Engineering, Kunming University of Science and Technology, Kunming 650500, China.
Chen Y; Faculty of Food Science and Engineering, Kunming University of Science and Technology, Kunming 650500, China.
Wang C; Key Laboratory of Dairy Science of Education Ministry, College of Veterinary Medicine, Northeast Agricultural University, Harbin 150030, China.
Li Q; Key Laboratory of Dairy Science of Education Ministry, College of Veterinary Medicine, Northeast Agricultural University, Harbin 150030, China.
Li Y; School of Medicine, Kunming University of Science and Technology, Kunming 650500, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Sep 20; Vol. 24 (18). Date of Electronic Publication: 2023 Sep 20.
Typ publikacji:
Journal Article
MeSH Terms:
Autophagy*/genetics
MicroRNAs*/genetics
Mammary Glands, Animal*/growth & development
Mammary Glands, Animal*/metabolism
Animals ; Female ; Mice ; Autophagy-Related Protein 5 ; Epithelial Cells ; Lactation/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Signal-Retaining Autophagy Indicator as a Quantitative Imaging Method for ER-Phagy.
Autorzy:
Jimenez-Moreno N; Cancer Research UK Scotland Centre, Institute of Genetics and Cancer, University of Edinburgh, Edinburgh EH4 2XR, UK.
Salomo-Coll C; Cancer Research UK Scotland Centre, Institute of Genetics and Cancer, University of Edinburgh, Edinburgh EH4 2XR, UK.
Murphy LC; MRC Human Genetics Unit, Institute of Genetics and Cancer, University of Edinburgh, Edinburgh EH4 2XU, UK.
Wilkinson S; Cancer Research UK Scotland Centre, Institute of Genetics and Cancer, University of Edinburgh, Edinburgh EH4 2XR, UK.
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Źródło:
Cells [Cells] 2023 Apr 11; Vol. 12 (8). Date of Electronic Publication: 2023 Apr 11.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Membrane Proteins*/metabolism
Autophagy*
Endoplasmic Reticulum/metabolism ; Endoplasmic Reticulum Stress ; Autophagy-Related Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Chaperone-mediated autophagy promotes breast cancer angiogenesis via regulation of aerobic glycolysis.
Autorzy:
Chen R; Tibetan Traditional Medical College, Lhasa, China.
Li P; Yantai Mountain Hospital, Yantai, Shandong, China.
Fu Y; General Hospital, Western Theater Command, Chengdu, Sichuan, China.
Wu Z; Tibetan Traditional Medical College, Lhasa, China.
Xu L; Tibetan Traditional Medical College, Lhasa, China.
Wang J; General Hospital of Tibet Area Military Command, Lhasa, China.
Chen S; Army Medical University (Third Military Medical University), Chongqing, China.
Yang M; Army Medical University (Third Military Medical University), Chongqing, China.
Peng B; Army Medical University (Third Military Medical University), Chongqing, China.
Yang Y; General Hospital, Western Theater Command, Chengdu, Sichuan, China.
Zhang H; General Hospital of Tibet Area Military Command, Lhasa, China.
Han Q; General Hospital of Tibet Area Military Command, Lhasa, China.; Army Medical University (Third Military Medical University), Chongqing, China.
Li S; Army Medical University (Third Military Medical University), Chongqing, China.
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Źródło:
PloS one [PLoS One] 2023 Mar 13; Vol. 18 (3), pp. e0281577. Date of Electronic Publication: 2023 Mar 13 (Print Publication: 2023).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Chaperone-Mediated Autophagy*
Breast Neoplasms*/pathology
Humans ; Female ; Autophagy ; Culture Media, Conditioned ; Lysosomal-Associated Membrane Protein 2/metabolism ; Lysosomal Membrane Proteins/metabolism ; Human Umbilical Vein Endothelial Cells/metabolism ; Glycolysis ; Cell Line, Tumor
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Upregulated Chaperone-Mediated Autophagy May Perform a Key Role in Reduced Cancer Incidence in Huntington's Disease.
Autorzy:
Hasholt LF; Institute of Cellular and Molecular Medicine, University of Copenhagen, Copenhagen, Denmark.
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Źródło:
Journal of Huntington's disease [J Huntingtons Dis] 2023; Vol. 12 (4), pp. 371-376.
Typ publikacji:
Journal Article
MeSH Terms:
Huntington Disease*/metabolism
Chaperone-Mediated Autophagy*
Neoplasms*
Animals ; Humans ; Incidence ; Autophagy/genetics ; Huntingtin Protein
Czasopismo naukowe
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Tytuł:
Osthole Suppresses Knee Osteoarthritis Development by Enhancing Autophagy Activated via the AMPK/ULK1 Pathway.
Autorzy:
Ma T; First Clinical Medical College, Shandong University of Traditional Chinese Medicine, Jinan 250355, China.
Wang X; Department of Orthopedics, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan 250011, China.
Qu W; First Clinical Medical College, Shandong University of Traditional Chinese Medicine, Jinan 250355, China.
Yang L; Department of Orthopedics, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan 250011, China.
Jing C; Department of Orthopedics, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan 250011, China.
Zhu B; Department of Orthopedics, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan 250011, China.
Zhang Y; Department of Orthopedics, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan 250011, China.
Xie W; Department of Orthopedics, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan 250011, China.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2022 Dec 06; Vol. 27 (23). Date of Electronic Publication: 2022 Dec 06.
Typ publikacji:
Journal Article
MeSH Terms:
Autophagy*
Osteoarthritis, Knee*/metabolism
Rats ; Animals ; Chondrocytes ; Signal Transduction ; Apoptosis ; AMP-Activated Protein Kinases/metabolism ; Autophagy-Related Protein-1 Homolog/metabolism
Czasopismo naukowe
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Tytuł:
Molecular Mechanism of Autophagosome-Lysosome Fusion in Mammalian Cells.
Autorzy:
Ke PY; Department of Biochemistry & Molecular Biology, Graduate Institute of Biomedical Sciences, College of Medicine, Chang Gung University, Taoyuan 33302, Taiwan.; Liver Research Center, Chang Gung Memorial Hospital, Taoyuan 33305, Taiwan.
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Źródło:
Cells [Cells] 2024 Mar 13; Vol. 13 (6). Date of Electronic Publication: 2024 Mar 13.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Autophagosomes*/metabolism
Autophagy*/physiology
Animals ; Macroautophagy ; Homeostasis ; Lysosomes/metabolism ; Mammals
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
GLA Mutations Suppress Autophagy and Stimulate Lysosome Generation in Fabry Disease.
Autorzy:
Li P; Institutes of Biomedical Sciences, The Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education of China, The Key Laboratory of Medical Molecular Cell Biology of Shanxi Province, Shanxi University, Taiyuan 030006, China.
Xi Y; Institutes of Biomedical Sciences, The Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education of China, The Key Laboratory of Medical Molecular Cell Biology of Shanxi Province, Shanxi University, Taiyuan 030006, China.
Zhang Y; Institutes of Biomedical Sciences, The Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education of China, The Key Laboratory of Medical Molecular Cell Biology of Shanxi Province, Shanxi University, Taiyuan 030006, China.
Samad A; Department of Biochemistry, Abdul Wali Khan University Mardan, Mardan 23200, Pakistan.
Lan W; Institutes of Biomedical Sciences, The Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education of China, The Key Laboratory of Medical Molecular Cell Biology of Shanxi Province, Shanxi University, Taiyuan 030006, China.
Wu Y; Institutes of Biomedical Sciences, The Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education of China, The Key Laboratory of Medical Molecular Cell Biology of Shanxi Province, Shanxi University, Taiyuan 030006, China.
Zhao J; Institutes of Biomedical Sciences, The Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education of China, The Key Laboratory of Medical Molecular Cell Biology of Shanxi Province, Shanxi University, Taiyuan 030006, China.
Chen G; Institutes of Biomedical Sciences, The Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education of China, The Key Laboratory of Medical Molecular Cell Biology of Shanxi Province, Shanxi University, Taiyuan 030006, China.
Wu C; Institutes of Biomedical Sciences, The Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education of China, The Key Laboratory of Medical Molecular Cell Biology of Shanxi Province, Shanxi University, Taiyuan 030006, China.
Xiong Q; Institutes of Biomedical Sciences, The Key Laboratory of Chemical Biology and Molecular Engineering of Ministry of Education of China, The Key Laboratory of Medical Molecular Cell Biology of Shanxi Province, Shanxi University, Taiyuan 030006, China.
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Źródło:
Cells [Cells] 2024 Mar 01; Vol. 13 (5). Date of Electronic Publication: 2024 Mar 01.
Typ publikacji:
Journal Article
MeSH Terms:
alpha-Galactosidase*/genetics
Autophagy*/genetics
Fabry Disease*/genetics
Humans ; Lysosomes/metabolism ; Mutation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Autophagy-modulating biomaterials: multifunctional weapons to promote tissue regeneration.
Autorzy:
Wu Y; Heilongjiang Key Laboratory of Tissue Damage and Repair, Mudanjiang Medical University, Mudanjiang, 157000, China.
Li L; Heilongjiang Key Laboratory of Tissue Damage and Repair, Mudanjiang Medical University, Mudanjiang, 157000, China.
Ning Z; Changhai Clinical Research Unit, Shanghai Changhai Hospital, Naval Medical University, Shanghai, 200433, China.
Li C; Heilongjiang Key Laboratory of Tissue Damage and Repair, Mudanjiang Medical University, Mudanjiang, 157000, China.
Yin Y; Heilongjiang Key Laboratory of Tissue Damage and Repair, Mudanjiang Medical University, Mudanjiang, 157000, China.
Chen K; Heilongjiang Key Laboratory of Tissue Damage and Repair, Mudanjiang Medical University, Mudanjiang, 157000, China.
Li L; Department of plastic surgery, Naval Specialty Medical Center of PLA, Shanghai, 200052, China. .
Xu F; Department of plastic surgery, Naval Specialty Medical Center of PLA, Shanghai, 200052, China. fei_.
Gao J; Changhai Clinical Research Unit, Shanghai Changhai Hospital, Naval Medical University, Shanghai, 200433, China. .
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Źródło:
Cell communication and signaling : CCS [Cell Commun Signal] 2024 Feb 15; Vol. 22 (1), pp. 124. Date of Electronic Publication: 2024 Feb 15.
Typ publikacji:
Video-Audio Media; Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Biocompatible Materials*/pharmacology
Biocompatible Materials*/chemistry
Autophagy*
Cell Differentiation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Chaperone-Mediated Autophagy in Pericytes: A Key Target for the Development of New Treatments against Glioblastoma Progression.
Autorzy:
Salinas MD; Cell Therapy and Hematopoietic Transplantation Group, Biochemistry, Molecular Biology B and Immunology Department, University of Murcia (UMU), 30120 Murcia, Spain.; Unit of Autophagy, Immune Response and Tolerance in Pathologic Processes, Biomedical Research Institute of Murcia-Virgen de la Arrixaca (IMIB), 30120 Murcia, Spain.
Valdor R; Cell Therapy and Hematopoietic Transplantation Group, Biochemistry, Molecular Biology B and Immunology Department, University of Murcia (UMU), 30120 Murcia, Spain.; Unit of Autophagy, Immune Response and Tolerance in Pathologic Processes, Biomedical Research Institute of Murcia-Virgen de la Arrixaca (IMIB), 30120 Murcia, Spain.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Aug 10; Vol. 23 (16). Date of Electronic Publication: 2022 Aug 10.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Chaperone-Mediated Autophagy*
Glioblastoma*/pathology
Glioblastoma*/therapy
Autophagy/physiology ; Humans ; Lysosomes/pathology ; Molecular Chaperones/genetics ; Pericytes/pathology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The autophagy inhibitor NSC185058 suppresses mTORC1-mediated protein anabolism in cultured skeletal muscle.
Autorzy:
Ryan PJ; Muscle Biology Laboratory, Department of Kinesiology and Sport Management, Texas A&M University, Gilchrist Building, 2929 Research Parkway, College Station, TX, 77843-4243, USA.
Uranga S; Muscle Biology Laboratory, Department of Kinesiology and Sport Management, Texas A&M University, Gilchrist Building, 2929 Research Parkway, College Station, TX, 77843-4243, USA.
Stanelle ST; Muscle Biology Laboratory, Department of Kinesiology and Sport Management, Texas A&M University, Gilchrist Building, 2929 Research Parkway, College Station, TX, 77843-4243, USA.
Lewis MH; Muscle Biology Laboratory, Department of Kinesiology and Sport Management, Texas A&M University, Gilchrist Building, 2929 Research Parkway, College Station, TX, 77843-4243, USA.
O'Reilly CL; Muscle Biology Laboratory, Department of Kinesiology and Sport Management, Texas A&M University, Gilchrist Building, 2929 Research Parkway, College Station, TX, 77843-4243, USA.
Cardin JM; Muscle Biology Laboratory, Department of Kinesiology and Sport Management, Texas A&M University, Gilchrist Building, 2929 Research Parkway, College Station, TX, 77843-4243, USA.
Deaver JW; Muscle Biology Laboratory, Department of Kinesiology and Sport Management, Texas A&M University, Gilchrist Building, 2929 Research Parkway, College Station, TX, 77843-4243, USA.
Morton AB; Soft Tissue Regeneration and Applied Biomaterials Laboratory, Texas A&M University, Gilchrist Building, 2929 Research Parkway, College Station, TX, 77843-4243, USA.
Fluckey JD; Muscle Biology Laboratory, Department of Kinesiology and Sport Management, Texas A&M University, Gilchrist Building, 2929 Research Parkway, College Station, TX, 77843-4243, USA. .
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Źródło:
Scientific reports [Sci Rep] 2024 Apr 06; Vol. 14 (1), pp. 8094. Date of Electronic Publication: 2024 Apr 06.
Typ publikacji:
Journal Article
MeSH Terms:
TOR Serine-Threonine Kinases*
Autophagy*/physiology
Aminopyridines*
Mechanistic Target of Rapamycin Complex 1/metabolism ; Muscle, Skeletal/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Wyświetlanie 1-20 z 134958

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