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Tytuł:
Chaperone-mediated autophagy degrades Keap1 and promotes Nrf2-mediated antioxidative response.
Autorzy:
Zhu L; Department of Experimental Surgery, Tangdu Hospital, Fourth Military Medical University, Xi'an, China.
He S; Department of Experimental Surgery, Tangdu Hospital, Fourth Military Medical University, Xi'an, China.
Huang L; Department of Experimental Surgery, Tangdu Hospital, Fourth Military Medical University, Xi'an, China.
Ren D; Department of Experimental Surgery, Tangdu Hospital, Fourth Military Medical University, Xi'an, China.
Nie T; Department of Experimental Surgery, Tangdu Hospital, Fourth Military Medical University, Xi'an, China.
Tao K; Department of Experimental Surgery, Tangdu Hospital, Fourth Military Medical University, Xi'an, China.
Xia L; Department of Neurosurgery and Institute for Functional Brain Disorders, Tangdu Hospital, Fourth Military Medical University, Xi'an, China.
Lu F; Department of Experimental Surgery, Tangdu Hospital, Fourth Military Medical University, Xi'an, China.
Mao Z; Departments of Pharmacology and Chemical Biology, Emory University School of Medicine, Atlanta, Georgia, USA.
Yang Q; Department of Experimental Surgery, Tangdu Hospital, Fourth Military Medical University, Xi'an, China.
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Źródło:
Aging cell [Aging Cell] 2022 Jun; Vol. 21 (6), pp. e13616. Date of Electronic Publication: 2022 May 10.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Chaperone-Mediated Autophagy*
NF-E2-Related Factor 2*/genetics
NF-E2-Related Factor 2*/metabolism
Antioxidants/pharmacology ; Autophagy ; Kelch-Like ECH-Associated Protein 1/genetics ; Kelch-Like ECH-Associated Protein 1/metabolism ; Oxidative Stress
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Long non-coding RNA UCA1 promotes autophagy by targeting miR-96-5p in acute myeloid leukaemia.
Autorzy:
Li JJ; Department of Hematology, The First Affiliated Hospital of Bengbu Medical College, Bengbu, China.
Chen XF; Department of Hematology, The First Affiliated Hospital of Bengbu Medical College, Bengbu, China.
Wang M; Department of Hematology, The First Affiliated Hospital of Bengbu Medical College, Bengbu, China.
Zhang PP; Department of Hematology, The First Affiliated Hospital of Bengbu Medical College, Bengbu, China.
Zhang F; Department of Hematology, The First Affiliated Hospital of Bengbu Medical College, Bengbu, China.
Zhang JJ; Department of Oncology, The First Affiliated Hospital of Bengbu Medical College, Bengbu, China.
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Źródło:
Clinical and experimental pharmacology & physiology [Clin Exp Pharmacol Physiol] 2020 May; Vol. 47 (5), pp. 877-885. Date of Electronic Publication: 2020 Feb 13.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autophagy*
Leukemia, Myeloid, Acute/*metabolism
MicroRNAs/*metabolism
RNA, Long Noncoding/*metabolism
Autophagy-Related Protein 7/genetics ; Autophagy-Related Protein 7/metabolism ; Cell Proliferation ; Disease Progression ; Gene Expression Regulation, Neoplastic ; HL-60 Cells ; Humans ; Leukemia, Myeloid, Acute/genetics ; Leukemia, Myeloid, Acute/pathology ; MicroRNAs/genetics ; RNA, Long Noncoding/genetics ; Signal Transduction ; U937 Cells
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
RACK1 promotes autophagy via the PERK signaling pathway to protect against traumatic brain injury in rats.
Autorzy:
Ni H; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, China.; Department of Neurosurgery, The Fourth Affiliated Hospital of Soochow University, Suzhou, China.
Kan X; Department of Neurobiology and Cell Biology, Xuzhou Key Laboratory of Neurobiology, Xuzhou Medical University, Xuzhou, China.
Rui Q; Department of Center of Clinical Laboratory, The Fourth Affiliated Hospital of Soochow University, Suzhou, China.
Zhang Y; Department of Neurobiology and Cell Biology, Xuzhou Key Laboratory of Neurobiology, Xuzhou Medical University, Xuzhou, China.
Zhai W; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, China.
Zhang B; Department of Neurobiology and Cell Biology, Xuzhou Key Laboratory of Neurobiology, Xuzhou Medical University, Xuzhou, China.
Yu Z; Department of Neurosurgery & Brain and Nerve Research Laboratory, The First Affiliated Hospital of Soochow University, Suzhou, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14691.
Typ publikacji:
Journal Article
MeSH Terms:
Signal Transduction*
Brain Injuries, Traumatic*/metabolism
Rats ; Animals ; Rats, Sprague-Dawley ; Neuroprotection ; Apoptosis ; Autophagy ; Receptors for Activated C Kinase
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Activation of transient receptor potential vanilloid 1 ameliorates tau accumulation-induced synaptic damage and cognitive dysfunction via autophagy enhancement.
Autorzy:
Zhang T; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Tian Y; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Zheng X; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Li R; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Hu L; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Shui X; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Mei Y; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Wang Q; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Zhang M; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Zheng X; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Wang L; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Chen D; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Tao W; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.; Key Laboratory of Brain Aging and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
Lee TH; Fujian Key Laboratory of Translational Research in Cancer and Neurodegenerative Diseases, School of Basic Medical Sciences, Fujian Medical University, Fuzhou, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Mar; Vol. 30 (3), pp. e14432. Date of Electronic Publication: 2023 Aug 29.
Typ publikacji:
Journal Article
MeSH Terms:
Antineoplastic Agents*
Cognitive Dysfunction*/drug therapy
Animals ; Humans ; Mice ; AMP-Activated Protein Kinases/metabolism ; Autophagy ; Capsaicin/pharmacology ; Mammals/metabolism ; tau Proteins/metabolism ; TRPV Cation Channels/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Enriched environment ameliorates fear memory impairments induced by sleep deprivation via inhibiting PIEZO1/calpain/autophagy signaling pathway in the basal forebrain.
Autorzy:
Zhang ZQ; The Postgraduate Training Base of Jinzhou Medical University (The PLA Rocket Force Characteristic Medical Center), Beijing, China.; Department of Neurology, The PLA Rocket Force Characteristic Medical Center, Beijing, China.
Lu Y; Department of Neurology, The PLA Rocket Force Characteristic Medical Center, Beijing, China.
Zhang H; Department of Anesthesiology, The PLA Rocket Force Characteristic Medical Center, Beijing, China.
Dong SH; Department of Nuclear Radiation Injury and Monitoring, The PLA Rocket Force Characteristic Medical Center, Beijing, China.
Wu YT; The Postgraduate Training Base of Jinzhou Medical University (The PLA Rocket Force Characteristic Medical Center), Beijing, China.; Department of Neurology, The PLA Rocket Force Characteristic Medical Center, Beijing, China.
Wang SN; Department of Nuclear Radiation Injury and Monitoring, The PLA Rocket Force Characteristic Medical Center, Beijing, China.
Huang AH; Department of Neurology, The PLA Rocket Force Characteristic Medical Center, Beijing, China.
Jiang QS; Department of Nuclear Radiation Injury and Monitoring, The PLA Rocket Force Characteristic Medical Center, Beijing, China.
Yin SM; The Postgraduate Training Base of Jinzhou Medical University (The PLA Rocket Force Characteristic Medical Center), Beijing, China.; Department of Neurology, The PLA Rocket Force Characteristic Medical Center, Beijing, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2024 Feb; Vol. 30 (2), pp. e14365. Date of Electronic Publication: 2023 Jul 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Acrylates*
Basal Forebrain*/metabolism
Calpain*/metabolism
Animals ; Male ; Mice ; Autophagy ; Fear ; Memory Disorders/etiology ; Memory Disorders/therapy ; Signal Transduction ; Sirolimus/pharmacology ; Sirolimus/therapeutic use ; Sleep Deprivation/complications
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Oral Carbon Monoxide Enhances Autophagy Modulation in Prostate, Pancreatic, and Lung Cancers.
Autorzy:
Bi, Jianling (AUTHOR)
Witt, Emily (AUTHOR)
McGovern, Megan K. (AUTHOR)
Cafi, Arielle B. (AUTHOR)
Rosenstock, Lauren L. (AUTHOR)
Pearson, Anna B. (AUTHOR)
Brown, Timothy J. (AUTHOR)
Karasic, Thomas B. (AUTHOR)
Absler, Lucas C. (AUTHOR)
Machkanti, Srija (AUTHOR)
Boyce, Hannah (AUTHOR)
Gallo, David (AUTHOR)
Becker, Sarah L. (AUTHOR)
Ishida, Keiko (AUTHOR)
Jenkins, Joshua (AUTHOR)
Hayward, Alison (AUTHOR)
Scheiflinger, Alexandra (AUTHOR)
Bodeker, Kellie L. (AUTHOR)
Kumar, Ritesh (AUTHOR)
Shaw, Scott K. (AUTHOR)
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Źródło:
Advanced Science. 3/6/2024, Vol. 11 Issue 9, p1-13. 13p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Reversine ameliorates hallmarks of cellular senescence in human skeletal myoblasts via reactivation of autophagy.
Autorzy:
Rajabian N; Department of Chemical and Biological Engineering, University at Buffalo, State University of New York, Amherst, New York, USA.
Choudhury D; Department of Chemical and Biological Engineering, University at Buffalo, State University of New York, Amherst, New York, USA.
Ikhapoh I; Department of Chemical and Biological Engineering, University at Buffalo, State University of New York, Amherst, New York, USA.
Saha S; Department of Biomedical Engineering, University at Buffalo, State University of New York, Amherst, New York, USA.
Kalyankar AS; Department of Biomedical Engineering, University at Buffalo, State University of New York, Amherst, New York, USA.
Mehrotra P; Department of Chemical and Biological Engineering, University at Buffalo, State University of New York, Amherst, New York, USA.
Shahini A; Department of Chemical and Biological Engineering, University at Buffalo, State University of New York, Amherst, New York, USA.
Breed K; Department of Chemical and Biological Engineering, University at Buffalo, State University of New York, Amherst, New York, USA.
Andreadis ST; Department of Chemical and Biological Engineering, University at Buffalo, State University of New York, Amherst, New York, USA.; Department of Biomedical Engineering, University at Buffalo, State University of New York, Amherst, New York, USA.; Center of Excellence in Bioinformatics and Life Sciences, University at Buffalo, State University of New York, Amherst, New York, USA.; Cell, Gene and Tissue Engineering (CGTE) Center, School of Engineering and Applied Sciences, University at Buffalo, State University of New York, Amherst, New York, USA.
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Źródło:
Aging cell [Aging Cell] 2023 Mar; Vol. 22 (3), pp. e13764. Date of Electronic Publication: 2023 Jan 10.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Cellular Senescence*/drug effects
Morpholines*/pharmacology
Purines*/pharmacology
Protein Kinase Inhibitors*/pharmacology
Myoblasts, Skeletal*/drug effects
Myoblasts, Skeletal*/physiology
Autophagy*/drug effects
Muscle Development*/drug effects
Humans ; Insulin/metabolism ; AMP-Activated Protein Kinase Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction/drug effects ; Glycolysis/drug effects ; Citric Acid Cycle/drug effects ; Insulin Resistance ; Cells, Cultured
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Muscle PGC-1α in exercise and fasting-induced regulation of hepatic UPR in mice.
Autorzy:
Kristensen CM; Department of Biology, University of Copenhagen, Copenhagen, Denmark.
Jessen H; Department of Biology, University of Copenhagen, Copenhagen, Denmark.
Ringholm S; Department of Biology, University of Copenhagen, Copenhagen, Denmark.
Pilegaard H; Department of Biology, University of Copenhagen, Copenhagen, Denmark.
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Źródło:
Acta physiologica (Oxford, England) [Acta Physiol (Oxf)] 2018 Dec; Vol. 224 (4), pp. e13158. Date of Electronic Publication: 2018 Jul 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autophagy*/genetics
Muscle Contraction*
Physical Exertion*
Unfolded Protein Response*/genetics
Fasting/*metabolism
Liver/*metabolism
Quadriceps Muscle/*metabolism
Transcription Factors/*metabolism
Animals ; Autophagy-Related Proteins/genetics ; Autophagy-Related Proteins/metabolism ; Female ; Gene Expression Regulation ; Liver/pathology ; Male ; Mice, Transgenic ; Signal Transduction ; Time Factors ; Transcription Factors/genetics ; Up-Regulation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Autophagy-A key pathway for cardiac health and longevity.
Autorzy:
Woodall BP; Skaggs School of Pharmacy and Pharmaceutical Sciences, University of California, San Diego, La Jolla, CA, USA.
Gustafsson ÅB; Skaggs School of Pharmacy and Pharmaceutical Sciences, University of California, San Diego, La Jolla, CA, USA.
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Źródło:
Acta physiologica (Oxford, England) [Acta Physiol (Oxf)] 2018 Aug; Vol. 223 (4), pp. e13074. Date of Electronic Publication: 2018 May 07.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Review
MeSH Terms:
Autophagy*/drug effects
Aging/*pathology
Cardiovascular Diseases/*pathology
Myocytes, Cardiac/*pathology
Age Factors ; Aging/metabolism ; Animals ; Autophagy-Related Proteins/metabolism ; Cardiovascular Agents/therapeutic use ; Cardiovascular Diseases/metabolism ; Cardiovascular Diseases/physiopathology ; Cardiovascular Diseases/prevention & control ; Humans ; Longevity ; Mitochondria, Heart/metabolism ; Mitochondria, Heart/pathology ; Mitophagy ; Myocytes, Cardiac/drug effects ; Myocytes, Cardiac/metabolism ; Risk Factors ; Risk Reduction Behavior ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
IL‐17A is involved in the hyperplasia of blood vessels in local lesions of psoriasis by inhibiting autophagy.
Autorzy:
Wang, Juanjuan (AUTHOR)
Zhou, Ling (AUTHOR)
Hou, Hui (AUTHOR)
Li, Jiao (AUTHOR)
Zhao, Xincheng (AUTHOR)
Li, Jiajie (AUTHOR)
Li, Junqin (AUTHOR)
Niu, Xuping (AUTHOR)
Hou, Ruixia (AUTHOR)
Zhang, Kaiming (AUTHOR)
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Źródło:
Journal of Cosmetic Dermatology. Jan2024, Vol. 23 Issue 1, p326-338. 13p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
(−)‐Epigallocatechin gallate alleviates chronic unpredictable mild stress‐induced depressive symptoms in mice by regulating the mTOR autophagy pathway and inhibiting NLRP3 inflammasome activation.
Autorzy:
Zhang, Yulin (AUTHOR)
Wu, Hongxian (AUTHOR)
Xu, Chaozhi (AUTHOR)
Li, Shanqian (AUTHOR)
Hu, Yue (AUTHOR)
Zhang, Zongyi (AUTHOR)
Wu, Guixian (AUTHOR)
Liu, Yuling (AUTHOR)
Yang, Lin (AUTHOR)
Huang, Yue (AUTHOR)
Lu, Wenjun (AUTHOR)
Hu, Lina (AUTHOR)
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Źródło:
Food Science & Nutrition. Jan2024, Vol. 12 Issue 1, p459-470. 12p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Metabolic reprogramming, autophagy, and ferroptosis: Novel arsenals to overcome immunotherapy resistance in gastrointestinal cancer.
Autorzy:
Wang, Xiangwen (AUTHOR)
Zhou, Liwen (AUTHOR)
Wang, Hongpeng (AUTHOR)
Chen, Wei (AUTHOR)
Jiang, Lei (AUTHOR)
Ming, Guangtao (AUTHOR)
Wang, Jun (AUTHOR)
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Źródło:
Cancer Medicine. Nov2023, Vol. 12 Issue 21, p20573-20589. 17p.
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Aging is associated with a decline in Atg9b-mediated autophagosome formation and appearance of enlarged mitochondria in the heart.
Autorzy:
Liang W; Department of Pharmacology, Skaggs School of Pharmacy and Pharmaceutical Sciences, University of California, San Diego, La Jolla, California, USA.
Moyzis AG; Department of Pharmacology, Skaggs School of Pharmacy and Pharmaceutical Sciences, University of California, San Diego, La Jolla, California, USA.
Lampert MA; Department of Pharmacology, Skaggs School of Pharmacy and Pharmaceutical Sciences, University of California, San Diego, La Jolla, California, USA.
Diao RY; Department of Pharmacology, Skaggs School of Pharmacy and Pharmaceutical Sciences, University of California, San Diego, La Jolla, California, USA.
Najor RH; Department of Pharmacology, Skaggs School of Pharmacy and Pharmaceutical Sciences, University of California, San Diego, La Jolla, California, USA.
Gustafsson ÅB; Department of Pharmacology, Skaggs School of Pharmacy and Pharmaceutical Sciences, University of California, San Diego, La Jolla, California, USA.
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Źródło:
Aging cell [Aging Cell] 2020 Aug; Vol. 19 (8), pp. e13187. Date of Electronic Publication: 2020 Jul 06.
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Aging/*physiology
Autophagy-Related Proteins/*metabolism
Heart/*physiology
Membrane Proteins/*metabolism
Mitochondria, Heart/*physiology
Animals ; Autophagosomes/metabolism ; Autophagosomes/physiology ; Autophagy/physiology ; HeLa Cells ; Humans ; Male ; Mice ; Mitochondria, Heart/metabolism ; Myocardium/cytology ; Myocardium/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Exploration of potential inhibitors for autophagy-related protein 8 as antileishmanial agents.
Autorzy:
Rahman F; Infection and Immunity Lab (414), Department of Biotechnology, Jamia Millia Islamia (A Central University), New Delhi, India.
Ali R; Infection and Immunity Lab (414), Department of Biotechnology, Jamia Millia Islamia (A Central University), New Delhi, India.
Tabrez S; Infection and Immunity Lab (414), Department of Biotechnology, Jamia Millia Islamia (A Central University), New Delhi, India.
Mobeen A; Institute of Genomics and Integrative Biology, New Delhi, India.
Akand SK; Infection and Immunity Lab (414), Department of Biotechnology, Jamia Millia Islamia (A Central University), New Delhi, India.
Arish M; Department of Pulmonary and Critical Care Medicine, Mayo Clinic, Rochester, Minnesota, USA.
AlAsmari AF; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia.
Ali N; Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia.
Rub A; Infection and Immunity Lab (414), Department of Biotechnology, Jamia Millia Islamia (A Central University), New Delhi, India.
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Źródło:
Chemical biology & drug design [Chem Biol Drug Des] 2022 Jun; Vol. 99 (6), pp. 816-827. Date of Electronic Publication: 2022 Feb 22.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Antiprotozoal Agents*/chemistry
Antiprotozoal Agents*/pharmacology
Autophagy-Related Protein 8 Family*/antagonists & inhibitors
Autophagy-Related Protein 8 Family*/chemistry
Autophagy-Related Protein 8 Family*/metabolism
Leishmania donovani*/drug effects
Coumarins/chemistry ; Coumarins/pharmacology ; Humans ; Leishmaniasis, Visceral/drug therapy ; Leishmaniasis, Visceral/metabolism ; Molecular Docking Simulation ; Reactive Oxygen Species/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Arctigenin improves neuropathy via ameliorating apoptosis and modulating autophagy in streptozotocin-induced diabetic mice.
Autorzy:
Medras ZJH; Ministry of Health, Kuwait City, Kuwait.
Mostafa YM; Department of Pharmacology and Toxicology, Faculty of Pharmacy, Suez Canal University, Ismailia, Egypt.; Department of Pharmacology & Toxicology, Faculty of Pharmacy, Badr University in Cairo, Badr, Egypt.
Ahmed AAM; Department of Cytology and Histology, Faculty of Veterinary Medicine, Suez Canal University, Ismailia, Egypt.
El-Sayed NM; Department of Pharmacology and Toxicology, Faculty of Pharmacy, Suez Canal University, Ismailia, Egypt.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2023 Oct; Vol. 29 (10), pp. 3068-3080. Date of Electronic Publication: 2023 May 11.
Typ publikacji:
Journal Article
MeSH Terms:
Diabetes Mellitus, Experimental*/metabolism
Lignans*/pharmacology
Lignans*/therapeutic use
Diabetic Neuropathies*/drug therapy
Mice ; Animals ; Antioxidants/pharmacology ; Antioxidants/therapeutic use ; Streptozocin/toxicity ; Oxidative Stress ; Apoptosis/physiology ; Autophagy/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Degradation of NLRP3 by p62-dependent-autophagy improves cognitive function in Alzheimer's disease by maintaining the phagocytic function of microglia.
Autorzy:
Zhang D; NHC Key Laboratory of Human Disease Comparative Medicine, Beijing Engineering Research Center for Experimental Animal Models of Human Critical Diseases, National center of Technology Innovation for animal model, Changping National laboratory (CPNL), Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences (CAMS) & Peking Union Medical College (PUMC), Beijing, China.
Zhang Y; NHC Key Laboratory of Human Disease Comparative Medicine, Beijing Engineering Research Center for Experimental Animal Models of Human Critical Diseases, National center of Technology Innovation for animal model, Changping National laboratory (CPNL), Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences (CAMS) & Peking Union Medical College (PUMC), Beijing, China.
Pan J; NHC Key Laboratory of Human Disease Comparative Medicine, Beijing Engineering Research Center for Experimental Animal Models of Human Critical Diseases, National center of Technology Innovation for animal model, Changping National laboratory (CPNL), Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences (CAMS) & Peking Union Medical College (PUMC), Beijing, China.
Cao J; NHC Key Laboratory of Human Disease Comparative Medicine, Beijing Engineering Research Center for Experimental Animal Models of Human Critical Diseases, National center of Technology Innovation for animal model, Changping National laboratory (CPNL), Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences (CAMS) & Peking Union Medical College (PUMC), Beijing, China.
Sun X; NHC Key Laboratory of Human Disease Comparative Medicine, Beijing Engineering Research Center for Experimental Animal Models of Human Critical Diseases, National center of Technology Innovation for animal model, Changping National laboratory (CPNL), Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences (CAMS) & Peking Union Medical College (PUMC), Beijing, China.
Li X; NHC Key Laboratory of Human Disease Comparative Medicine, Beijing Engineering Research Center for Experimental Animal Models of Human Critical Diseases, National center of Technology Innovation for animal model, Changping National laboratory (CPNL), Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences (CAMS) & Peking Union Medical College (PUMC), Beijing, China.
Zhang L; NHC Key Laboratory of Human Disease Comparative Medicine, Beijing Engineering Research Center for Experimental Animal Models of Human Critical Diseases, National center of Technology Innovation for animal model, Changping National laboratory (CPNL), Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences (CAMS) & Peking Union Medical College (PUMC), Beijing, China.
Qin C; NHC Key Laboratory of Human Disease Comparative Medicine, Beijing Engineering Research Center for Experimental Animal Models of Human Critical Diseases, National center of Technology Innovation for animal model, Changping National laboratory (CPNL), Institute of Laboratory Animal Sciences, Chinese Academy of Medical Sciences (CAMS) & Peking Union Medical College (PUMC), Beijing, China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2023 Oct; Vol. 29 (10), pp. 2826-2842. Date of Electronic Publication: 2023 Apr 18.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Alzheimer Disease*/metabolism
Animals ; Mice ; Autophagy ; Cognition ; Inflammasomes/metabolism ; Microglia ; NLR Family, Pyrin Domain-Containing 3 Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Acyl coenzyme A binding protein (ACBP): An aging- and disease-relevant "autophagy checkpoint".
Autorzy:
Montégut L; Centre de Recherche des Cordeliers, Equipe labellisée par la Ligue Contre le Cancer, Inserm U1138, Université Paris Cité, Sorbonne Université, Paris, France.; Metabolomics and Cell Biology Platforms, Gustave Roussy Institut, Villejuif, France.; Faculté de Médecine, Université de Paris Saclay, Paris, France.
Abdellatif M; Centre de Recherche des Cordeliers, Equipe labellisée par la Ligue Contre le Cancer, Inserm U1138, Université Paris Cité, Sorbonne Université, Paris, France.; Metabolomics and Cell Biology Platforms, Gustave Roussy Institut, Villejuif, France.; Department of Cardiology, Medical University of Graz, Graz, Austria.; BioTechMed-Graz, Graz, Austria.
Motiño O; Centre de Recherche des Cordeliers, Equipe labellisée par la Ligue Contre le Cancer, Inserm U1138, Université Paris Cité, Sorbonne Université, Paris, France.; Metabolomics and Cell Biology Platforms, Gustave Roussy Institut, Villejuif, France.
Madeo F; BioTechMed-Graz, Graz, Austria.; Institute of Molecular Biosciences, NAWI Graz, University of Graz, Graz, Austria.; Field of Excellence BioHealth, University of Graz, Graz, Austria.
Martins I; Centre de Recherche des Cordeliers, Equipe labellisée par la Ligue Contre le Cancer, Inserm U1138, Université Paris Cité, Sorbonne Université, Paris, France.; Metabolomics and Cell Biology Platforms, Gustave Roussy Institut, Villejuif, France.
Quesada V; Departamento de Bioquímica y Biología Molecular, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), Universidad de Oviedo, Oviedo, Spain.
López-Otín C; Centre de Recherche des Cordeliers, Equipe labellisée par la Ligue Contre le Cancer, Inserm U1138, Université Paris Cité, Sorbonne Université, Paris, France.; Departamento de Bioquímica y Biología Molecular, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), Universidad de Oviedo, Oviedo, Spain.
Kroemer G; Centre de Recherche des Cordeliers, Equipe labellisée par la Ligue Contre le Cancer, Inserm U1138, Université Paris Cité, Sorbonne Université, Paris, France.; Metabolomics and Cell Biology Platforms, Gustave Roussy Institut, Villejuif, France.; Institut du Cancer Paris CARPEM, Department of Biology, Hôpital Européen Georges Pompidou, AP-HP, Paris, France.
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Źródło:
Aging cell [Aging Cell] 2023 Sep; Vol. 22 (9), pp. e13910. Date of Electronic Publication: 2023 Jun 26.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Carrier Proteins*
Diazepam Binding Inhibitor*/metabolism
Animals ; Humans ; Mice ; Acyl Coenzyme A/metabolism ; Aging ; Autophagy ; Mammals/metabolism ; Saccharomyces cerevisiae/metabolism
Czasopismo naukowe
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Tytuł:
KCNQ1OT1 promotes autophagy by regulating miR-200a/FOXO3/ATG7 pathway in cerebral ischemic stroke.
Autorzy:
Yu S; Department of Neurology, Shengjing Hospital of China Medical University, Shenyang, China.
Yu M; Department of Neurosurgery, Shengjing Hospital of China Medical University, Shenyang, China.
He X; Department of Neurology, Shengjing Hospital of China Medical University, Shenyang, China.
Wen L; Department of Neurology, Shengjing Hospital of China Medical University, Shenyang, China.
Bu Z; Department of Neurology, Shengjing Hospital of China Medical University, Shenyang, China.
Feng J; Department of Neurology, Shengjing Hospital of China Medical University, Shenyang, China.
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Źródło:
Aging cell [Aging Cell] 2019 Jun; Vol. 18 (3), pp. e12940. Date of Electronic Publication: 2019 Apr 03.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Autophagy-Related Protein 7/*metabolism
Brain Ischemia/*metabolism
Forkhead Box Protein O3/*metabolism
MicroRNAs/*metabolism
RNA, Long Noncoding/*metabolism
Reperfusion Injury/*metabolism
Stroke/*metabolism
Aged ; Animals ; Autophagosomes/metabolism ; Autophagosomes/ultrastructure ; Autophagy/genetics ; Autophagy-Related Protein 7/genetics ; Brain Ischemia/genetics ; Brain Ischemia/pathology ; Cell Line, Tumor ; Cell Survival/genetics ; Female ; Forkhead Box Protein O3/genetics ; Humans ; Male ; Mice ; Mice, Inbred C57BL ; MicroRNAs/genetics ; Middle Aged ; Potassium Channels, Voltage-Gated/genetics ; Potassium Channels, Voltage-Gated/metabolism ; Reperfusion Injury/genetics ; Reperfusion Injury/pathology ; Signal Transduction/genetics ; Stroke/genetics ; Stroke/pathology ; Vacuoles/genetics ; Vacuoles/metabolism ; Vacuoles/ultrastructure
Czasopismo naukowe
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Wyświetlanie 1-20 z 6258

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