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Tytuł:
STUB1 is acetylated by KAT5 and alleviates myocardial ischemia-reperfusion injury through LATS2-YAP-β-catenin axis.
Autorzy:
Liu C; Department of Cardiovascular Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei, 230022, Anhui Province, P.R. China.
Gui Z; Department of Cardiovascular Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei, 230022, Anhui Province, P.R. China.
An C; Department of Cardiovascular Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei, 230022, Anhui Province, P.R. China.
Sun F; Department of Cardiovascular Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei, 230022, Anhui Province, P.R. China.
Gao X; Department of Cardiovascular Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei, 230022, Anhui Province, P.R. China.
Ge S; Department of Cardiovascular Surgery, The First Affiliated Hospital of Anhui Medical University, Hefei, 230022, Anhui Province, P.R. China. .
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Źródło:
Communications biology [Commun Biol] 2024 Apr 01; Vol. 7 (1), pp. 396. Date of Electronic Publication: 2024 Apr 01.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/genetics
beta Catenin*/metabolism
Myocardial Reperfusion Injury*/metabolism
Humans ; Myocytes, Cardiac/metabolism ; Pyroptosis ; Ubiquitination ; Protein Serine-Threonine Kinases/genetics ; Protein Serine-Threonine Kinases/metabolism ; Tumor Suppressor Proteins/genetics ; Tumor Suppressor Proteins/metabolism ; Ubiquitin-Protein Ligases/metabolism ; Lysine Acetyltransferase 5/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
SFRP2 suppresses trophoblast cell migration by inhibiting the Wnt/β‑catenin pathway.
Autorzy:
Lan R; Department of Obstetrics, Hainan General Hospital, Hainan Affiliated Hospital of Hainan Medical University, Haikou, Hainan 570311, P.R. China.
Yu Y; School of Clinical Medicine, Hainan Medical University, Haikou, Hainan 571199, P.R. China.
Song J; Department of Obstetrics, Hainan General Hospital, Hainan Affiliated Hospital of Hainan Medical University, Haikou, Hainan 570311, P.R. China.
Xue M; School of Clinical Medicine, Hainan Medical University, Haikou, Hainan 571199, P.R. China.
Gong H; Department of Obstetrics, Hainan General Hospital, Hainan Affiliated Hospital of Hainan Medical University, Haikou, Hainan 570311, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2024 Apr; Vol. 29 (4). Date of Electronic Publication: 2024 Mar 01.
Typ publikacji:
Journal Article
MeSH Terms:
Wnt Signaling Pathway*
beta Catenin*/genetics
beta Catenin*/metabolism
Humans ; Female ; Pregnancy ; Cell Line, Tumor ; Glycogen Synthase Kinase 3 beta/genetics ; Glycogen Synthase Kinase 3 beta/metabolism ; Secreted Frizzled-Related Proteins ; Placenta/metabolism ; Wnt Proteins/metabolism ; Trophoblasts/metabolism ; Cell Proliferation ; Cell Movement/genetics ; Membrane Proteins/genetics ; Membrane Proteins/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Knockdown of Kmt2d leads to growth impairment by activating the Akt/β-catenin signaling pathway.
Autorzy:
Shangguan H; Department of Endocrinology, Genetics and Metabolism, Fuzhou Children's Hospital of Fujian Medical University, Fuzhou 350000, China.
Huang X; Department of Endocrinology, Genetics and Metabolism, Fuzhou Children's Hospital of Fujian Medical University, Fuzhou 350000, China.
Lin J; Department of Endocrinology, Genetics and Metabolism, Fuzhou Children's Hospital of Fujian Medical University, Fuzhou 350000, China.
Chen R; Department of Endocrinology, Genetics and Metabolism, Fuzhou Children's Hospital of Fujian Medical University, Fuzhou 350000, China.
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Źródło:
G3 (Bethesda, Md.) [G3 (Bethesda)] 2024 Mar 06; Vol. 14 (3).
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/genetics
beta Catenin*/metabolism
Proto-Oncogene Proteins c-akt*/genetics
Proto-Oncogene Proteins c-akt*/metabolism
Animals ; Cell Proliferation/genetics ; Chondrocytes/metabolism ; Signal Transduction ; Wnt Signaling Pathway/genetics ; Zebrafish/genetics ; Zebrafish/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
β-catenin mediates growth defects induced by centrosome loss in a subset of APC mutant colorectal cancer independently of p53.
Autorzy:
Bourmoum M; Lunenfeld-Tanenbaum Research Institute, Sinai Health System, Toronto, ON, Canada.
Radulovich N; University Health Network, Ontario Cancer Institute/Princess Margaret Cancer Centre, Toronto, ON, Canada.
Sharma A; Lunenfeld-Tanenbaum Research Institute, Sinai Health System, Toronto, ON, Canada.
Tkach JM; Lunenfeld-Tanenbaum Research Institute, Sinai Health System, Toronto, ON, Canada.
Tsao MS; University Health Network, Ontario Cancer Institute/Princess Margaret Cancer Centre, Toronto, ON, Canada.
Pelletier L; Lunenfeld-Tanenbaum Research Institute, Sinai Health System, Toronto, ON, Canada.; Department of Molecular Genetics, University of Toronto, Toronto, ON, Canada.
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Źródło:
PloS one [PLoS One] 2024 Feb 07; Vol. 19 (2), pp. e0295030. Date of Electronic Publication: 2024 Feb 07 (Print Publication: 2024).
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/genetics
beta Catenin*/metabolism
Colorectal Neoplasms*/genetics
Colorectal Neoplasms*/metabolism
Tumor Suppressor Protein p53*/genetics
Tumor Suppressor Protein p53*/metabolism
Adenomatous Polyposis Coli Protein*/genetics
Adenomatous Polyposis Coli Protein*/metabolism
Animals ; Humans ; Centrosome/metabolism ; Pyrimidines ; Sulfones ; Wnt Signaling Pathway
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A WNT mimetic with broad spectrum FZD-specificity decreases fibrosis and improves function in a pulmonary damage model.
Autorzy:
Patel M; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Post Y; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Hill N; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Sura A; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Ye J; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Fisher T; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Suen N; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Zhang M; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Cheng L; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Pribluda A; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Chen H; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Yeh WC; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Li Y; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Baribault H; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA.
Fletcher RB; Surrozen, Inc., 171 Oyster Point Blvd, Suite 400, South San Francisco, CA, 94080, USA. .
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Źródło:
Respiratory research [Respir Res] 2024 Apr 02; Vol. 25 (1), pp. 153. Date of Electronic Publication: 2024 Apr 02.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/metabolism
Idiopathic Pulmonary Fibrosis*/chemically induced
Idiopathic Pulmonary Fibrosis*/drug therapy
Idiopathic Pulmonary Fibrosis*/metabolism
Adult ; Animals ; Humans ; Lung/metabolism ; Wnt Signaling Pathway ; Bleomycin/toxicity
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Wnt5a/β-catenin-mediated epithelial-mesenchymal transition: a key driver of subretinal fibrosis in neovascular age-related macular degeneration.
Autorzy:
Liu D; Department of Ophthalmology of Tongji Hospital and Laboratory of Clinical and Visual Sciences of Tongji Eye Institute, School of Medicine, Tongji University, Shanghai, China.
Du J; Department of Ophthalmology, Shanghai General Hospital (Shanghai First People's Hospital), Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Ocular Fundus Diseases, National Clinical Research Center for Eye Diseases, Shanghai Engineering Center for Visual Science and Photomedicine, Shanghai Engineering Center for Precise Diagnosis and Treatment of Eye Diseases, Shanghai Eye Research Institute, Shanghai, China.
Xie H; Department of Ophthalmology, Shanghai General Hospital (Shanghai First People's Hospital), Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Shanghai Key Laboratory of Ocular Fundus Diseases, National Clinical Research Center for Eye Diseases, Shanghai Engineering Center for Visual Science and Photomedicine, Shanghai Engineering Center for Precise Diagnosis and Treatment of Eye Diseases, Shanghai Eye Research Institute, Shanghai, China.
Tian H; Department of Ophthalmology of Tongji Hospital and Laboratory of Clinical and Visual Sciences of Tongji Eye Institute, School of Medicine, Tongji University, Shanghai, China.
Lu L; Department of Ophthalmology of Tongji Hospital and Laboratory of Clinical and Visual Sciences of Tongji Eye Institute, School of Medicine, Tongji University, Shanghai, China.
Zhang C; Department of Ophthalmology, Shanghai General Hospital (Shanghai First People's Hospital), Shanghai Jiao Tong University School of Medicine, Shanghai, China. .; Shanghai Key Laboratory of Ocular Fundus Diseases, National Clinical Research Center for Eye Diseases, Shanghai Engineering Center for Visual Science and Photomedicine, Shanghai Engineering Center for Precise Diagnosis and Treatment of Eye Diseases, Shanghai Eye Research Institute, Shanghai, China. .
Xu GT; Department of Ophthalmology of Tongji Hospital and Laboratory of Clinical and Visual Sciences of Tongji Eye Institute, School of Medicine, Tongji University, Shanghai, China. .
Zhang J; Department of Ophthalmology, Shanghai General Hospital (Shanghai First People's Hospital), Shanghai Jiao Tong University School of Medicine, Shanghai, China. .; Shanghai Key Laboratory of Ocular Fundus Diseases, National Clinical Research Center for Eye Diseases, Shanghai Engineering Center for Visual Science and Photomedicine, Shanghai Engineering Center for Precise Diagnosis and Treatment of Eye Diseases, Shanghai Eye Research Institute, Shanghai, China. .
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Źródło:
Journal of neuroinflammation [J Neuroinflammation] 2024 Mar 26; Vol. 21 (1), pp. 75. Date of Electronic Publication: 2024 Mar 26.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/metabolism
Macular Degeneration*/metabolism
Sulfonamides*
Humans ; Male ; Animals ; Mice ; Wnt-5a Protein ; Mice, Inbred C57BL ; Retinal Pigment Epithelium/metabolism ; Epithelial-Mesenchymal Transition ; Fibrosis ; RNA, Small Interfering/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
HuR-positive stress granules: Potential targets for age-related osteoporosis.
Autorzy:
Huai Y; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.; Department of Orthopedics, Tangdu Hospital, Air Force Military Medical University, Xi'an, China.
Wang X; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Mao W; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Wang X; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Zhao Y; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Chu X; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Huang Q; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Ru K; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Zhang L; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Li Y; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Chen Z; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
Qian A; Lab for Bone Metabolism, Xi'an Key Laboratory of Special Medicine and Health Engineering, Northwestern Polytechnical University, Xi'an, China.; Key Lab for Space Biosciences and Biotechnology, Research Center for Special Medicine and Health Systems Engineering, Northwestern Polytechnical University, Xi'an, China.; NPU-UAB Joint Laboratory for Bone Metabolism, School of Life Sciences, Northwestern Polytechnical University, Xi'an, China.
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Źródło:
Aging cell [Aging Cell] 2024 Mar; Vol. 23 (3), pp. e14053. Date of Electronic Publication: 2024 Feb 20.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/metabolism
Osteoporosis*/metabolism
Stress Granules*
Humans ; Osteoblasts/metabolism ; Osteogenesis ; RNA-Binding Proteins/metabolism ; ELAV-Like Protein 1/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Lipoteichoic acid restrains macrophage senescence via β-catenin/FOXO1/REDD1 pathway in age-related osteoporosis.
Autorzy:
Cheng W; Department of Orthopaedics, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Guangdong Provincial Key Laboratory of Bone and Cartilage Regenerative Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Fu Y; Department of Orthopaedics, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Guangdong Provincial Key Laboratory of Bone and Cartilage Regenerative Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Lin Z; Department of Orthopaedics, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Guangdong Provincial Key Laboratory of Bone and Cartilage Regenerative Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Huang M; Department of Orthopaedics, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Guangdong Provincial Key Laboratory of Bone and Cartilage Regenerative Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Chen Y; Department of Orthopaedics, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Guangdong Provincial Key Laboratory of Bone and Cartilage Regenerative Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Hu Y; Department of Orthopaedics, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Guangdong Provincial Key Laboratory of Bone and Cartilage Regenerative Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Lin Q; Department of Orthopaedics, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Guangdong Provincial Key Laboratory of Bone and Cartilage Regenerative Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Yu B; Department of Orthopaedics, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Guangdong Provincial Key Laboratory of Bone and Cartilage Regenerative Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Liu G; Department of Orthopaedics, Nanfang Hospital, Southern Medical University, Guangzhou, China.; Guangdong Provincial Key Laboratory of Bone and Cartilage Regenerative Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.
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Źródło:
Aging cell [Aging Cell] 2024 Mar; Vol. 23 (3), pp. e14072. Date of Electronic Publication: 2023 Dec 21.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/metabolism
Osteoporosis*
Lipopolysaccharides*
Teichoic Acids*
Humans ; Aged ; Signal Transduction ; Macrophages/metabolism ; Cellular Senescence ; Wnt Signaling Pathway ; Forkhead Box Protein O1/genetics ; Forkhead Box Protein O1/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
A Protective Role of Canonical Wnt/ β -Catenin Pathway in Pathogenic Bacteria-Induced Inflammatory Responses.
Autorzy:
Jiang Z; Department of Biochemistry and Molecular Biology, Shenyang Medical College, Shenyang 110034, China.; Key Laboratory of Environment Pollution and Microecology of Liaoning Province, Shenyang 110034, China.
Zhou W; Department of Pathogen Biology, Shenyang Medical College, Shenyang 110034, China.
Tian X; Department of Physiology, Shenyang Medical College, Shenyang 110034, China.
Zou P; Department of Biochemistry and Molecular Biology, Shenyang Medical College, Shenyang 110034, China.
Li N; Department of Biochemistry and Molecular Biology, Shenyang Medical College, Shenyang 110034, China.
Zhang C; Department of Pathogen Biology, Shenyang Medical College, Shenyang 110034, China.
Li Y; Department of Pathogen Biology, Shenyang Medical College, Shenyang 110034, China.
Liu G; Key Laboratory of Environment Pollution and Microecology of Liaoning Province, Shenyang 110034, China.; Department of Pathogen Biology, Shenyang Medical College, Shenyang 110034, China.
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Źródło:
Mediators of inflammation [Mediators Inflamm] 2024 Feb 27; Vol. 2024, pp. 8869510. Date of Electronic Publication: 2024 Feb 27 (Print Publication: 2024).
Typ publikacji:
Journal Article; Review
MeSH Terms:
beta Catenin*
Bacterial Infections*
Humans ; Bacteria ; Wnt Signaling Pathway ; Inflammation ; Anti-Inflammatory Agents
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
DPPA2/4 Promote the Pluripotency and Proliferation of Bovine Extended Pluripotent Stem Cells by Upregulating the PI3K/AKT/GSK3β/β-Catenin Signaling Pathway.
Autorzy:
Fang S; The State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, Inner Mongolia University, Hohhot 010070, China.
Wang J; The State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, Inner Mongolia University, Hohhot 010070, China.
Liu G; The State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, Inner Mongolia University, Hohhot 010070, China.
Qu B; The State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, Inner Mongolia University, Hohhot 010070, China.
Chunyu J; The State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, Inner Mongolia University, Hohhot 010070, China.
Xu W; The State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, Inner Mongolia University, Hohhot 010070, China.
Xiang J; The State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, Inner Mongolia University, Hohhot 010070, China.
Li X; The State Key Laboratory of Reproductive Regulation and Breeding of Grassland Livestock, Inner Mongolia University, Hohhot 010070, China.
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Źródło:
Cells [Cells] 2024 Feb 23; Vol. 13 (5). Date of Electronic Publication: 2024 Feb 23.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/metabolism
Pluripotent Stem Cells*/metabolism
Transcription Factors*/metabolism
Nuclear Proteins*/metabolism
Animals ; Cattle ; Cell Proliferation ; Glycogen Synthase Kinase 3 beta/metabolism ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Signal Transduction
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
β -Nicotinamide Mononucleotide Promotes Cell Proliferation and Hair Growth by Reducing Oxidative Stress.
Autorzy:
Xu C; School of Chemistry and Chemical Engineering, Guangxi University, Nanning 530004, China.
Dai J; School of Chemistry and Chemical Engineering, Guangxi University, Nanning 530004, China.
Ai H; Guangdong Institute of Modern Agricultural Equipment, Guangzhou 510145, China.
Du W; Guangdong Homy Genetics Ltd., Foshan 528000, China.
Ji H; School of Chemistry and Chemical Engineering, Guangxi University, Nanning 530004, China.; State Key Laboratory Breeding Base of Green-Chemical Synthesis Technology, Institute of Green Petroleum Processing and Light Hydrocarbon, College of Chemical Engineering, Zhejiang University of Technology, Hangzhou 310014, China.
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Źródło:
Molecules (Basel, Switzerland) [Molecules] 2024 Feb 08; Vol. 29 (4). Date of Electronic Publication: 2024 Feb 08.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/metabolism
Nicotinamide Mononucleotide*
Animals ; Mice ; Humans ; Interleukin-6/metabolism ; Hair ; Hair Follicle/metabolism ; Dihydrotestosterone/metabolism ; Cell Proliferation ; Oxidative Stress
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Wnt/β-catenin signalling activates IMPDH2-mediated purine metabolism to facilitate oxaliplatin resistance by inhibiting caspase-dependent apoptosis in colorectal cancer.
Autorzy:
Huang Y; Department of Interventional Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, China.; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, China.
Chan S; Shenzhen Key Laboratory for Systems Medicine in Inflammatory Diseases, School of Medicine, Shenzhen Campus of Sun Yat-Sen University, Sun Yat-Sen University, Shenzhen, China.
Chen S; Shenzhen Key Laboratory for Systems Medicine in Inflammatory Diseases, School of Medicine, Shenzhen Campus of Sun Yat-Sen University, Sun Yat-Sen University, Shenzhen, China.
Liu X; Shenzhen Key Laboratory for Systems Medicine in Inflammatory Diseases, School of Medicine, Shenzhen Campus of Sun Yat-Sen University, Sun Yat-Sen University, Shenzhen, China.
Li M; Department of Dermatovenereology, The Seventh Affiliated Hospital, Sun Yat-Sen University, Shenzhen, China.
Zheng L; Dongguan Key Laboratory of Medical Bioactive Molecular Developmental and Translational Research, Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.
Dong Z; Shenzhen Key Laboratory for Systems Medicine in Inflammatory Diseases, School of Medicine, Shenzhen Campus of Sun Yat-Sen University, Sun Yat-Sen University, Shenzhen, China.
Yang Z; Shenzhen Key Laboratory for Systems Medicine in Inflammatory Diseases, School of Medicine, Shenzhen Campus of Sun Yat-Sen University, Sun Yat-Sen University, Shenzhen, China.
Liu Z; Shenzhen Key Laboratory for Systems Medicine in Inflammatory Diseases, School of Medicine, Shenzhen Campus of Sun Yat-Sen University, Sun Yat-Sen University, Shenzhen, China.
Zhou D; Shenzhen Key Laboratory for Systems Medicine in Inflammatory Diseases, School of Medicine, Shenzhen Campus of Sun Yat-Sen University, Sun Yat-Sen University, Shenzhen, China.
Zhang X; Shenzhen Key Laboratory for Systems Medicine in Inflammatory Diseases, School of Medicine, Shenzhen Campus of Sun Yat-Sen University, Sun Yat-Sen University, Shenzhen, China. .
Zhang B; Department of Interventional Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, China. .; Biomedical Innovation Center, The Sixth Affiliated Hospital, Sun Yat-Sen University, Guangzhou, China. .
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Źródło:
Journal of translational medicine [J Transl Med] 2024 Feb 03; Vol. 22 (1), pp. 133. Date of Electronic Publication: 2024 Feb 03.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/metabolism
Colorectal Neoplasms*/drug therapy
Colorectal Neoplasms*/genetics
Colorectal Neoplasms*/pathology
Humans ; Apoptosis ; Cell Line, Tumor ; Cell Proliferation ; Drug Resistance, Neoplasm/genetics ; Gene Expression Regulation, Neoplastic ; IMP Dehydrogenase/genetics ; IMP Dehydrogenase/metabolism ; Oxaliplatin/pharmacology ; Oxaliplatin/therapeutic use ; Oxidoreductases/genetics ; Oxidoreductases/metabolism ; Wnt Signaling Pathway
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Identification of the Axis β-Catenin-BTK in the Dynamic Adhesion of Chronic Lymphocytic Leukemia Cells to Their Microenvironment.
Autorzy:
Mihoub I; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
Rharass T; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
Ouriemmi S; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
Oudar A; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
Aubard L; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
Gratio V; INSERM U1149, Université Paris Cité, Hôpital Bichat, 75018 Paris, France.
Lazarian G; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.; AP-HP Hôpital Avicenne, 93000 Bobigny, France.
Ferreira J; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
Dondi E; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
Cymbalista F; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.; AP-HP Hôpital Avicenne, 93000 Bobigny, France.
Levy V; URC, AP-HP Hôpital Avicenne, 93000 Bobigny, France.
Baran-Marszak F; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.; AP-HP Hôpital Avicenne, 93000 Bobigny, France.
Varin-Blank N; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
Ledoux D; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
Le Roy C; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
Gardano L; INSERM, U978, 93000 Bobigny, France.; UFR SMBH, LabEx INFLAMEX, Université Paris 13-«Sorbonne Paris Nord», 93000 Bobigny, France.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Dec 18; Vol. 24 (24). Date of Electronic Publication: 2023 Dec 18.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/genetics
beta Catenin*/metabolism
Leukemia, Lymphocytic, Chronic, B-Cell*/pathology
Agammaglobulinaemia Tyrosine Kinase*/metabolism
Humans ; Cell Communication ; Cell Line, Tumor ; Stromal Cells/metabolism ; Tumor Microenvironment
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Interferon-γ inducible protein 30 promotes the epithelial-mesenchymal transition-like phenotype and chemoresistance by activating EGFR/AKT/GSK3β/β-catenin pathway in glioma.
Autorzy:
Chen Y; Department of Neurosurgery, The Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu, P.R. China.
Xu H; Department of Neurosurgery, The Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu, P.R. China.
Yu P; Department of Neurosurgery, The Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu, P.R. China.
Wang Q; Department of Neurosurgery, The Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu, P.R. China.
Li S; Department of Neurosurgery, The Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu, P.R. China.
Ji F; Department of Neurosurgery, The Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu, P.R. China.
Wu C; Department of Neurosurgery, The Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu, P.R. China.
Lan Q; Department of Neurosurgery, The Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu, P.R. China.
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Źródło:
CNS neuroscience & therapeutics [CNS Neurosci Ther] 2023 Dec; Vol. 29 (12), pp. 4124-4138. Date of Electronic Publication: 2023 Jul 05.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
beta Catenin*/genetics
beta Catenin*/metabolism
Glioma*/genetics
Humans ; Proto-Oncogene Proteins c-akt/metabolism ; Interferon-gamma/metabolism ; Epithelial-Mesenchymal Transition ; Temozolomide/pharmacology ; Temozolomide/therapeutic use ; Glycogen Synthase Kinase 3 beta ; Drug Resistance, Neoplasm ; Cell Line, Tumor ; Cell Movement ; Phenotype ; ErbB Receptors/metabolism ; Cell Proliferation ; Gene Expression Regulation, Neoplastic
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
SOX9 Inhibits the Progression of Osteonecrosis of the Femoral Head via the Activation of the Wnt/Beta-Catenin Pathway.
Autorzy:
Meng X; Trauma Center, Lianyungang City No.1 People's Hospital, Lianyungang, China.
Zhu H; Trauma Center, Lianyungang City No.1 People's Hospital, Lianyungang, China.
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Źródło:
Journal of investigative surgery : the official journal of the Academy of Surgical Research [J Invest Surg] 2023 Dec; Vol. 36 (1), pp. 2197054.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/genetics
beta Catenin*/metabolism
Osteonecrosis*
Humans ; Osteogenesis ; Core Binding Factor Alpha 1 Subunit/genetics ; Core Binding Factor Alpha 1 Subunit/metabolism ; Femur Head ; Cell Differentiation ; Glucocorticoids/pharmacology ; Cells, Cultured ; SOX9 Transcription Factor/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
LMP2 and TAP2 impair tumor growth and metastasis by inhibiting Wnt/β-catenin signaling pathway and EMT in cervical cancer.
Autorzy:
Cheng Z; Department of Pathology, Eastern Hospital, Sichuan Academy of Medical Science & Sichuan Provincial People's Hospital, Chengdu, 610101, China. cheng_.
Wang H; Department of Pathology, Eastern Hospital, Sichuan Academy of Medical Science & Sichuan Provincial People's Hospital, Chengdu, 610101, China.
Yang Z; Department of Pathology, Eastern Hospital, Sichuan Academy of Medical Science & Sichuan Provincial People's Hospital, Chengdu, 610101, China.
Li J; Department of Pathology, Eastern Hospital, Sichuan Academy of Medical Science & Sichuan Provincial People's Hospital, Chengdu, 610101, China.
Chen X; Department of Thoracic Surgery, Eastern Hospital, Sichuan Academy of Medical Science & Sichuan Provincial People's Hospital, Chengdu, 610101, China.
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Źródło:
BMC cancer [BMC Cancer] 2023 Nov 20; Vol. 23 (1), pp. 1128. Date of Electronic Publication: 2023 Nov 20.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*/genetics
beta Catenin*/metabolism
Uterine Cervical Neoplasms*/genetics
Female ; Humans ; Wnt Signaling Pathway ; HeLa Cells ; Epithelial-Mesenchymal Transition/genetics ; Carcinogenesis/genetics ; Cell Proliferation ; Cell Movement ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; ATP Binding Cassette Transporter, Subfamily B, Member 3/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The pathogenic role of succinate-SUCNR1: a critical function that induces renal fibrosis via M2 macrophage.
Autorzy:
Pu M; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.; Department of Ultrasound, Chongqing Key Laboratory of Ultrasound, Molecular Imaging, The Second Affiliated Hospital of Chongqing Medical University, Chongqing, China.
Zhang J; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Hong F; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Wang Y; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Zhang C; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Zeng Y; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Fang Z; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.
Qi W; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.; Guangdong Engineering & Technology Research Center for Gene Manipulation and Biomacromolecular Products, Sun Yat-sen University, Guangzhou, China.
Yang X; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China.; Guangdong Engineering & Technology Research Center for Gene Manipulation and Biomacromolecular Products, Sun Yat-sen University, Guangzhou, China.; China Key Laboratory of Tropical Disease Control (Sun Yat-sen University), Ministry of Education, Guangzhou, China.
Gao G; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China. .; Program of Molecular Medicine, Affiliated Guangzhou Women and Children's Hospital, Zhongshan School of Medicine, Sun Yat-Sen University, Guangzhou, China. .; Guangdong Engineering & Technology Research Center for Gene Manipulation and Biomacromolecular Products, Sun Yat-sen University, Guangzhou, China. .; Guangdong Province Key Laboratory of Brain Function and Disease, Zhongshan School of Medicine, Sun Yat-Sen University, Guangzhou, China. .; Guangdong Provincial Key Laboratory of Diabetology, Guangzhou, Guangdong, China. .
Zhou T; Department of Biochemistry and Molecular Biology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China. .; Guangdong Engineering & Technology Research Center for Gene Manipulation and Biomacromolecular Products, Sun Yat-sen University, Guangzhou, China. .; China Key Laboratory of Tropical Disease Control (Sun Yat-sen University), Ministry of Education, Guangzhou, China. .
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Źródło:
Cell communication and signaling : CCS [Cell Commun Signal] 2024 Jan 30; Vol. 22 (1), pp. 78. Date of Electronic Publication: 2024 Jan 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
beta Catenin*/metabolism
Renal Insufficiency, Chronic*/metabolism
Male ; Mice ; Animals ; Succinic Acid/metabolism ; Liposomes/metabolism ; Clodronic Acid/metabolism ; Glycogen Synthase Kinase 3 beta/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Fibrosis ; Macrophages/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The scaffold protein AXIN1: gene ontology, signal network, and physiological function.
Autorzy:
Qiu L; School of Pharmaceutical Sciences (Shenzhen), Shenzhen Campus of Sun Yat-Sen University, Shenzhen, 518107, China.; School of Life Sciences, Zhengzhou University, Zhengzhou, 450001, China.
Sun Y; School of Pharmaceutical Sciences (Shenzhen), Shenzhen Campus of Sun Yat-Sen University, Shenzhen, 518107, China.
Ning H; School of Pharmaceutical Sciences (Shenzhen), Shenzhen Campus of Sun Yat-Sen University, Shenzhen, 518107, China.
Chen G; School of Pharmaceutical Sciences (Shenzhen), Shenzhen Campus of Sun Yat-Sen University, Shenzhen, 518107, China.
Zhao W; School of Life Sciences, Zhengzhou University, Zhengzhou, 450001, China. .
Gao Y; School of Pharmaceutical Sciences (Shenzhen), Shenzhen Campus of Sun Yat-Sen University, Shenzhen, 518107, China. .
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Źródło:
Cell communication and signaling : CCS [Cell Commun Signal] 2024 Jan 30; Vol. 22 (1), pp. 77. Date of Electronic Publication: 2024 Jan 30.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Wnt Signaling Pathway*/genetics
beta Catenin*/metabolism
Animals ; Gene Ontology ; Axin Protein/genetics ; Axin Protein/metabolism ; Phosphorylation ; Proteolysis ; Mammals/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
KLF15 transcriptionally activates LINC00689 to inhibit colorectal cancer development.
Autorzy:
Cao Y; Department of Nuclear Medicine, Xiangya Third Hospital, Central South University, Changsha, 410013, Hunan Province, PR China.
Li J; Department of Nuclear Medicine, Xiangya Third Hospital, Central South University, Changsha, 410013, Hunan Province, PR China.
Zhang G; Department 2 of Gastrointestinal Surgery, Haikou Hospital Affiliated to Xiangya Medical College of Central South University, Haikou People's Hospital, Haikou, 570208, Hainan Province, PR China.
Fang H; Department of Nuclear Medicine, Xiangya Third Hospital, Central South University, Changsha, 410013, Hunan Province, PR China.
Du Y; Department of Nuclear Medicine, Xiangya Third Hospital, Central South University, Changsha, 410013, Hunan Province, PR China.
Liang Y; Department of Nuclear Medicine, Xiangya Third Hospital, Central South University, Changsha, 410013, Hunan Province, PR China. .
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Źródło:
Communications biology [Commun Biol] 2024 Jan 25; Vol. 7 (1), pp. 130. Date of Electronic Publication: 2024 Jan 25.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
beta Catenin*/metabolism
Colorectal Neoplasms*/metabolism
Humans ; Cell Line, Tumor ; Gene Expression Regulation, Neoplastic ; Transcription Factors/genetics ; Transcription Factors/metabolism ; Protein Serine-Threonine Kinases/metabolism ; Tumor Suppressor Proteins/genetics ; Kruppel-Like Transcription Factors/genetics ; Kruppel-Like Transcription Factors/metabolism ; Heterogeneous-Nuclear Ribonucleoproteins/genetics ; Heterogeneous-Nuclear Ribonucleoproteins/metabolism ; Polypyrimidine Tract-Binding Protein/genetics ; Polypyrimidine Tract-Binding Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Machine learning identifies SLC6A14 as a novel biomarker promoting the proliferation and metastasis of pancreatic cancer via Wnt/β-catenin signaling.
Autorzy:
Dang C; Department of Hepatobiliary Gastrointestinal Surgery, Tianjin Fourth Central Hospital, No.1 Zhongshan Road, Tianjin, China. .
Bian Q; Department of Plastic and Reconstructive Surgery, Tianjin Nankai Hospital, Tianjin, China.
Wang F; Department of Hepatobiliary Gastrointestinal Surgery, Tianjin Fourth Central Hospital, No.1 Zhongshan Road, Tianjin, China.
Wang H; Department of Otorhinolaryngology-Head and Neck Surgery, Tianjin Fourth Central Hospital, Tianjin, China.
Liang Z; Department of Hepatobiliary Gastrointestinal Surgery, Tianjin Fourth Central Hospital, No.1 Zhongshan Road, Tianjin, China.
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Źródło:
Scientific reports [Sci Rep] 2024 Jan 24; Vol. 14 (1), pp. 2116. Date of Electronic Publication: 2024 Jan 24.
Typ publikacji:
Journal Article
MeSH Terms:
beta Catenin*
Pancreatic Neoplasms*/diagnosis
Pancreatic Neoplasms*/genetics
Humans ; Wnt Signaling Pathway/genetics ; Machine Learning ; Biomarkers ; Cell Proliferation/genetics ; Amino Acid Transport Systems
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
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