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Tytuł:
Regulation of human neutrophil IL-1β secretion induced by Escherichia coli O157:H7 responsible for hemolytic uremic syndrome.
Autorzy:
Sabbione F; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Keitelman IA; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Shiromizu CM; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Vereertbrugghen A; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Vera Aguilar D; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Rubatto Birri PN; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Pizzano M; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Ramos MV; Laboratorio de patogénesis e inmunología de procesos infecciosos. Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Fuentes F; Laboratorio de microscopía, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Saposnik L; Instituto de Investigaciones Biotecnológicas, Universidad Nacional de San Martín (UNSAM)-Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Universidad Nacional de San Martín. San Martín, Buenos Aires, Argentina.; Escuela de Bio y Nanotecnologías (EByN), Universidad Nacional de San Martín. San Martín, Buenos Aires, Argentina.
Cernutto A; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Cassataro J; Instituto de Investigaciones Biotecnológicas, Universidad Nacional de San Martín (UNSAM)-Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Universidad Nacional de San Martín. San Martín, Buenos Aires, Argentina.; Escuela de Bio y Nanotecnologías (EByN), Universidad Nacional de San Martín. San Martín, Buenos Aires, Argentina.
Jancic CC; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.; Departamento de Microbiología, Parasitología e Inmunología, Facultad de Medicina, Universidad de Buenos Aires, Buenos Aires, Argentina.
Galletti JG; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Palermo MS; Laboratorio de patogénesis e inmunología de procesos infecciosos. Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.
Trevani AS; Laboratorio de inmunidad innata, Instituto de Medicina Experimental (IMEX)-CONICET, Academia Nacional de Medicina, Buenos Aires, Argentina.; Departamento de Microbiología, Parasitología e Inmunología, Facultad de Medicina, Universidad de Buenos Aires, Buenos Aires, Argentina.
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Źródło:
PLoS pathogens [PLoS Pathog] 2023 Dec 21; Vol. 19 (12), pp. e1011877. Date of Electronic Publication: 2023 Dec 21 (Print Publication: 2023).
Typ publikacji:
Journal Article
MeSH Terms:
Escherichia coli Infections*/metabolism
Escherichia coli Infections*/microbiology
Escherichia coli O157*
Hemolytic-Uremic Syndrome*/metabolism
Hemolytic-Uremic Syndrome*/microbiology
Shiga-Toxigenic Escherichia coli*
Interleukin-1beta*/metabolism
Humans ; Caspases ; Neutrophils
Czasopismo naukowe
Tytuł:
Evasion of host antioxidative response via disruption of NRF2 signaling in fatal Ehrlichia-induced liver injury.
Autorzy:
Sharma AK; Department of Pathology, College of Medicine, University of Illinois at Chicago, Chicago, Illinois, United States of America.
El Andaloussi A; Department of Pathology, College of Medicine, University of Illinois at Chicago, Chicago, Illinois, United States of America.; BioImmune Solutions Inc., 605-1355, Le Corbusier, Laval, Quebec, Canada.
Ismail N; Department of Pathology, College of Medicine, University of Illinois at Chicago, Chicago, Illinois, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2023 Nov 13; Vol. 19 (11), pp. e1011791. Date of Electronic Publication: 2023 Nov 13 (Print Publication: 2023).
Typ publikacji:
Journal Article
MeSH Terms:
Chemical and Drug Induced Liver Injury, Chronic*/pathology
Ehrlichiosis*/microbiology
Mice ; Humans ; Animals ; Ehrlichia ; Antioxidants ; NF-E2-Related Factor 2/metabolism ; Inflammasomes ; Liver/pathology ; Caspases/metabolism ; Signal Transduction ; Inflammation/pathology ; Mice, Inbred C57BL ; Mammals
Czasopismo naukowe
Tytuł:
Helicase-like transcription factor (Hltf)-deletion activates Hmgb1-Rage axis and granzyme A-mediated killing of pancreatic β cells resulting in neonatal lethality.
Autorzy:
Kaur G; Department of Medical Education, Texas Tech University Health Sciences Center, Lubbock, Texas, United States of America.
Helmer RA; Department of Cell Biology and Biochemistry, Texas Tech University Health Sciences Center, Lubbock, Texas, United States of America.
Martinez-Marin D; Department of Cell Biology and Biochemistry, Texas Tech University Health Sciences Center, Lubbock, Texas, United States of America.; Department of Immunology and Molecular Microbiology, Texas Tech University-Health Sciences Center, Lubbock, Texas, United States of America.
Sennoune SR; Department of Cell Biology and Biochemistry, Texas Tech University Health Sciences Center, Lubbock, Texas, United States of America.
Washburn RL; Department of Cell Biology and Biochemistry, Texas Tech University Health Sciences Center, Lubbock, Texas, United States of America.
Martinez-Zaguilan R; Department of Cell Physiology and Molecular Biophysics, Texas Tech University Health Sciences Center, Lubbock, Texas, United States of America.
Dufour JM; Department of Cell Biology and Biochemistry, Texas Tech University Health Sciences Center, Lubbock, Texas, United States of America.
Chilton BS; Department of Cell Biology and Biochemistry, Texas Tech University Health Sciences Center, Lubbock, Texas, United States of America.
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Źródło:
PloS one [PLoS One] 2023 Aug 25; Vol. 18 (8), pp. e0286109. Date of Electronic Publication: 2023 Aug 25 (Print Publication: 2023).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Insulin-Secreting Cells*
Animals ; Mice ; Caspases ; Causality ; Granzymes ; Transcription Factors
Czasopismo naukowe
Tytuł:
Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis.
Autorzy:
Ketelut-Carneiro N; Department of Biochemistry and Immunology, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, SP, Brazil.
Souza COS; Department of Biochemistry and Immunology, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, SP, Brazil.
Benevides L; Department of Biochemistry and Immunology, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, SP, Brazil.
Gardinassi LG; Department of Clinical Analyses, Toxicology and Food Science, School of Pharmaceutical Sciences of Ribeirão Preto, University of São Paulo, Ribeirão Preto, SP, Brazil.
Silva MC; Department of Biochemistry and Immunology, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, SP, Brazil.
Tavares LA; Department of Cell Biology, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, SP, Brazil.
Zamboni DS; Department of Cell Biology, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, SP, Brazil.
Silva JS; Department of Biochemistry and Immunology, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, SP, Brazil.; Fiocruz-Bi-Institutional Translational Medicine Project, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, SP, Brazil.
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Źródło:
PLoS pathogens [PLoS Pathog] 2019 Aug 19; Vol. 15 (8), pp. e1007990. Date of Electronic Publication: 2019 Aug 19 (Print Publication: 2019).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Caspases/*physiology
Immunity, Innate/*immunology
Interleukin-1alpha/*metabolism
Macrophages/*immunology
Paracoccidioides/*immunology
Paracoccidioidomycosis/*immunology
Th17 Cells/*immunology
Animals ; Caspases, Initiator ; Inflammasomes ; Macrophages/metabolism ; Macrophages/microbiology ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Paracoccidioidomycosis/metabolism ; Paracoccidioidomycosis/microbiology ; Th17 Cells/metabolism ; Th17 Cells/microbiology
Czasopismo naukowe
Tytuł:
Guanylate-binding protein 5 licenses caspase-11 for Gasdermin-D mediated host resistance to Brucella abortus infection.
Autorzy:
Cerqueira DM; Departamento de Bioquímica e Imunologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Minas Gerais, Brazil.
Gomes MTR; Departamento de Bioquímica e Imunologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Minas Gerais, Brazil.
Silva ALN; Departamento de Biologia Celular, Universidade de São Paulo-Ribeirão Preto, Brazil.
Rungue M; Departamento de Bioquímica e Imunologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Minas Gerais, Brazil.
Assis NRG; Departamento de Bioquímica e Imunologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Minas Gerais, Brazil.
Guimarães ES; Departamento de Bioquímica e Imunologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Minas Gerais, Brazil.
Morais SB; Departamento de Bioquímica e Imunologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Minas Gerais, Brazil.
Broz P; Department of Biochemistry, University of Lausanne, Epalinges, Switzerland.
Zamboni DS; Departamento de Biologia Celular, Universidade de São Paulo-Ribeirão Preto, Brazil.
Oliveira SC; Departamento de Bioquímica e Imunologia, Instituto de Ciências Biológicas, Universidade Federal de Minas Gerais, Belo Horizonte, Minas Gerais, Brazil.; Instituto Nacional de Ciência e Tecnologia em Doenças Tropicais (INCT-DT), Conselho Nacional de Desenvolvimento Científico e Tecnológico, Ministério de Ciência Tecnologia e Inovação Salvador, Bahia, Brazil.
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Źródło:
PLoS pathogens [PLoS Pathog] 2018 Dec 27; Vol. 14 (12), pp. e1007519. Date of Electronic Publication: 2018 Dec 27 (Print Publication: 2018).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis Regulatory Proteins/*immunology
Brucellosis/*immunology
Caspases/*immunology
GTP-Binding Proteins/*immunology
Immunity, Innate/*immunology
Animals ; Brucella abortus ; Caspases, Initiator ; Intracellular Signaling Peptides and Proteins ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Phosphate-Binding Proteins
Czasopismo naukowe
Tytuł:
Caspase-11-dependent pyroptosis of lung epithelial cells protects from melioidosis while caspase-1 mediates macrophage pyroptosis and production of IL-18.
Autorzy:
Wang J; Department of Microbiology and Immunology, Rosalind Franklin University of Medicine and Science, North Chicago, Illinois, United States of America.
Sahoo M; Department of Microbiology and Immunology, Rosalind Franklin University of Medicine and Science, North Chicago, Illinois, United States of America.
Lantier L; Department of Microbiology and Immunology, Rosalind Franklin University of Medicine and Science, North Chicago, Illinois, United States of America.
Warawa J; Department of Microbiology and Immunology, University of Louisville, Louisville, Kentucky, United States of America.
Cordero H; Department of Microbiology and Immunology, Rosalind Franklin University of Medicine and Science, North Chicago, Illinois, United States of America.
Deobald K; Department of Microbiology and Immunology, Rosalind Franklin University of Medicine and Science, North Chicago, Illinois, United States of America.
Re F; Department of Microbiology and Immunology, Rosalind Franklin University of Medicine and Science, North Chicago, Illinois, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2018 May 23; Vol. 14 (5), pp. e1007105. Date of Electronic Publication: 2018 May 23 (Print Publication: 2018).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Caspase 1/*physiology
Caspases/*physiology
Interleukin-18/*metabolism
Lung/*cytology
Melioidosis/*prevention & control
Pyroptosis/*physiology
Animals ; Burkholderia/physiology ; Caspases, Initiator ; Cell Line ; Disease Models, Animal ; Female ; Interferon-gamma/physiology ; Macrophages/microbiology ; Macrophages/physiology ; Male ; Melioidosis/immunology ; Mice ; Mice, Inbred C57BL ; Reactive Oxygen Species/metabolism ; Respiratory Mucosa/cytology
Czasopismo naukowe
Tytuł:
TNF licenses macrophages to undergo rapid caspase-1, -11, and -8-mediated cell death that restricts Legionella pneumophila infection.
Autorzy:
Pollock TY; Department of Microbiology, University of Pennsylvania Perelman School of Medicine, Philadelphia, Pennsylvania, United States of America.
Vázquez Marrero VR; Department of Microbiology, University of Pennsylvania Perelman School of Medicine, Philadelphia, Pennsylvania, United States of America.
Brodsky IE; Department of Pathobiology, University of Pennsylvania School of Veterinary Medicine, Philadelphia, Pennsylvania, United States of America.
Shin S; Department of Microbiology, University of Pennsylvania Perelman School of Medicine, Philadelphia, Pennsylvania, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2023 Jun 06; Vol. 19 (6), pp. e1010767. Date of Electronic Publication: 2023 Jun 06 (Print Publication: 2023).
Typ publikacji:
Journal Article
MeSH Terms:
Legionnaires' Disease*
Pneumonia*/metabolism
Mice ; Animals ; Humans ; Caspase 1/metabolism ; Caspase 8/metabolism ; Inflammasomes ; Mice, Knockout ; Macrophages ; Caspases/metabolism ; Cell Death ; Tumor Necrosis Factor-alpha/metabolism ; Licensure
Czasopismo naukowe
Tytuł:
HIV-1 release requires Nef-induced caspase activation.
Autorzy:
Segura, Jason (AUTHOR)
Ireland, Joanna (AUTHOR)
Zou, Zhongcheng (AUTHOR)
Roth, Gwynne (AUTHOR)
Buchwald, Julianna (AUTHOR)
Shen, Thomas J. (AUTHOR)
Fischer, Elizabeth (AUTHOR)
Moir, Susan (AUTHOR)
Chun, Tae-Wook (AUTHOR)
Sun, Peter D. (AUTHOR)
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Źródło:
PLoS ONE. 2/13/2023, Vol. 17 Issue 2, p1-34. 34p.
Czasopismo naukowe
Tytuł:
Actin remodeling mediates ROS production and JNK activation to drive apoptosis-induced proliferation.
Autorzy:
Farrell L; University of Birmingham, School of Biosciences, Birmingham, United Kingdom.
Puig-Barbe A; University of Birmingham, School of Biosciences, Birmingham, United Kingdom.
Haque MI; University of Birmingham, School of Biosciences, Birmingham, United Kingdom.; Department of Physiology, Faculty of Veterinary Science, Bangladesh Agricultural University, Mymensingh, Bangladesh.
Amcheslavsky A; University of Massachusetts Medical School, Department of Molecular, Cell and Cancer Biology, Worcester, Massachusetts, United States of America.
Yu M; University of Birmingham, School of Biosciences, Birmingham, United Kingdom.
Bergmann A; University of Massachusetts Medical School, Department of Molecular, Cell and Cancer Biology, Worcester, Massachusetts, United States of America.
Fan Y; University of Birmingham, School of Biosciences, Birmingham, United Kingdom.
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Źródło:
PLoS genetics [PLoS Genet] 2022 Dec 05; Vol. 18 (12), pp. e1010533. Date of Electronic Publication: 2022 Dec 05 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Actins*/genetics
Actins*/metabolism
Drosophila Proteins*/genetics
Drosophila Proteins*/metabolism
Animals ; Reactive Oxygen Species/metabolism ; Drosophila melanogaster/genetics ; Drosophila melanogaster/metabolism ; Apoptosis/genetics ; Caspases/genetics ; Caspases/metabolism ; JNK Mitogen-Activated Protein Kinases/genetics ; JNK Mitogen-Activated Protein Kinases/metabolism ; Cell Proliferation/genetics
Czasopismo naukowe
Tytuł:
Differential activation of NLRP3 inflammasome by Acinetobacter baumannii strains.
Autorzy:
Li FJ; Division of Immunology and Infectious Disease, The John Curtin School of Medical Research, The Australian National University, Canberra, ACT, Australia.
Starrs L; Division of Immunology and Infectious Disease, The John Curtin School of Medical Research, The Australian National University, Canberra, ACT, Australia.
Mathur A; Division of Immunology and Infectious Disease, The John Curtin School of Medical Research, The Australian National University, Canberra, ACT, Australia.
Ishii H; Division of Immunology and Infectious Disease, The John Curtin School of Medical Research, The Australian National University, Canberra, ACT, Australia.
Man SM; Division of Immunology and Infectious Disease, The John Curtin School of Medical Research, The Australian National University, Canberra, ACT, Australia.
Burgio G; Division of Immunology and Infectious Disease, The John Curtin School of Medical Research, The Australian National University, Canberra, ACT, Australia.
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Źródło:
PloS one [PLoS One] 2022 Nov 01; Vol. 17 (11), pp. e0277019. Date of Electronic Publication: 2022 Nov 01 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Inflammasomes*/metabolism
Acinetobacter baumannii*
NLR Family, Pyrin Domain-Containing 3 Protein/metabolism ; Interleukin-1beta/metabolism ; Caspases/metabolism ; Macrophages/metabolism
Czasopismo naukowe
Tytuł:
A caspase-RhoGEF axis contributes to the cell size threshold for apoptotic death in developing Caenorhabditis elegans.
Autorzy:
Sethi A; Faculty of Biology, Center for Integrative Protein Sciences Munich (CIPSM), Ludwig-Maximilians-University Munich, Planegg-Martinsried, Germany.; Department of Cell & Developmental Biology, Division of Biosciences, University College London, London, United Kingdom.
Wei H; Faculty of Biology, Center for Integrative Protein Sciences Munich (CIPSM), Ludwig-Maximilians-University Munich, Planegg-Martinsried, Germany.
Mishra N; Faculty of Biology, Center for Integrative Protein Sciences Munich (CIPSM), Ludwig-Maximilians-University Munich, Planegg-Martinsried, Germany.
Segos I; Department of Cell & Developmental Biology, Division of Biosciences, University College London, London, United Kingdom.
Lambie EJ; Department of Cell & Developmental Biology, Division of Biosciences, University College London, London, United Kingdom.
Zanin E; Faculty of Biology, Center for Integrative Protein Sciences Munich (CIPSM), Ludwig-Maximilians-University Munich, Planegg-Martinsried, Germany.; Department Biology, Friedrich-Alexander-University Erlangen-Nürnberg, Erlangen, Germany.
Conradt B; Department of Cell & Developmental Biology, Division of Biosciences, University College London, London, United Kingdom.
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Źródło:
PLoS biology [PLoS Biol] 2022 Oct 06; Vol. 20 (10), pp. e3001786. Date of Electronic Publication: 2022 Oct 06 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
MeSH Terms:
Caenorhabditis elegans*/metabolism
Caenorhabditis elegans Proteins*/genetics
Caenorhabditis elegans Proteins*/metabolism
Actomyosin/metabolism ; Animals ; Apoptosis/genetics ; Caspases/genetics ; Caspases/metabolism ; Cell Size ; Guanine Nucleotide Exchange Factors/metabolism ; Rho Guanine Nucleotide Exchange Factors/metabolism
Czasopismo naukowe
Tytuł:
Human neutrophil IL1β directs intestinal epithelial cell extrusion during Salmonella infection.
Autorzy:
Lawrence AE; Department of Microbiology and Immunology, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Berger RP; Department of Microbiology and Immunology, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Hill DR; Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Huang S; Department of Cell and Developmental Biology, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Yadagiri VK; Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Bons B; Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Fields C; Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Sule GJ; Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Knight JS; Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Wobus CE; Department of Microbiology and Immunology, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Spence JR; Department of Cell and Developmental Biology, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Young VB; Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
O'Riordan MX; Department of Microbiology and Immunology, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
Abuaita BH; Department of Microbiology and Immunology, University of Michigan Medical School, Ann Arbor, Michigan, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2022 Oct 03; Vol. 18 (10), pp. e1010855. Date of Electronic Publication: 2022 Oct 03 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Neutrophils*
Salmonella Infections*/metabolism
Animals ; Humans ; Mice ; Caspases/metabolism ; Epithelial Cells ; Intestinal Mucosa/microbiology ; Salmonella typhimurium
Czasopismo naukowe
Tytuł:
Metronomic doses and drug schematic combination response tested within chambered coverslips for the treatment of breast cancer cells (JIMT-1).
Autorzy:
Rosero G; National Technological University (UTN) IREN Center, Buenos Aires, Argentina.; Faculty of Medicine, Department of Medicine I, Medical Center-University of Freiburg, University of Freiburg, Freiburg, Germany.
Pattarone G; Faculty of Medicine, Department of Medicine I, Medical Center-University of Freiburg, University of Freiburg, Freiburg, Germany.
Peñaherera A; National Technological University (UTN) IREN Center, Buenos Aires, Argentina.; Faculty of Medicine, Department of Medicine I, Medical Center-University of Freiburg, University of Freiburg, Freiburg, Germany.
Pilz J; Faculty of Medicine, Department of Medicine I, Medical Center-University of Freiburg, University of Freiburg, Freiburg, Germany.
Bödecker J; Faculty of Engineering, Neurorobotics Lab, Computer Science Department, University of Freiburg, Georges-Köhler-Allee, Freiburg, Germany.
Perez M; National Technological University (UTN) IREN Center, Buenos Aires, Argentina.; Faculty of Engineering, University of Buenos Aires (UBA), Institute of Engineering Biomedical, Buenos Aires, Argentina.; Department of Electrical and Computer Engineering, Florida International University, Miami, FL, United States of America.; Collaborative Research Institute Intelligent Oncology, Hermann-Herder-Straße, Freiburg im Breisgau, Germany.
Mertelsmann R; Faculty of Medicine, Department of Medicine I, Medical Center-University of Freiburg, University of Freiburg, Freiburg, Germany.
Lerner B; Faculty of Engineering, University of Buenos Aires (UBA), Institute of Engineering Biomedical, Buenos Aires, Argentina.; Department of Electrical and Computer Engineering, Florida International University, Miami, FL, United States of America.; Collaborative Research Institute Intelligent Oncology, Hermann-Herder-Straße, Freiburg im Breisgau, Germany.
Follo M; Faculty of Medicine, Department of Medicine I, Medical Center-University of Freiburg, University of Freiburg, Freiburg, Germany.
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Źródło:
PloS one [PLoS One] 2022 Sep 29; Vol. 17 (9), pp. e0274911. Date of Electronic Publication: 2022 Sep 29 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Breast Neoplasms*/drug therapy
Apoptosis ; Caspases ; Drug Combinations ; Female ; Humans ; Paclitaxel/pharmacology ; Paclitaxel/therapeutic use
Czasopismo naukowe
Tytuł:
Characterization of the caspase family in zebrafish.
Autorzy:
Spead O; Department of Biological Sciences, University of South Carolina, Columbia, South Carolina, United States of America.
Verreet T; Department of Biological Sciences, University of South Carolina, Columbia, South Carolina, United States of America.
Donelson CJ; Department of Biological Sciences, University of South Carolina, Columbia, South Carolina, United States of America.
Poulain FE; Department of Biological Sciences, University of South Carolina, Columbia, South Carolina, United States of America.
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Źródło:
PloS one [PLoS One] 2018 May 23; Vol. 13 (5), pp. e0197966. Date of Electronic Publication: 2018 May 23 (Print Publication: 2018).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Zebrafish*/genetics
Zebrafish*/growth & development
Caspases/*metabolism
Amino Acid Sequence ; Animals ; Caspases/chemistry ; Caspases/genetics ; Cloning, Molecular ; Gene Expression Regulation, Developmental ; Humans ; Phylogeny
Czasopismo naukowe
Tytuł:
Reactive oxygen species regulate caspase-11 expression and activation of the non-canonical NLRP3 inflammasome during enteric pathogen infection.
Autorzy:
Lupfer CR; Department of Immunology, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America.
Anand PK; Department of Immunology, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America.
Liu Z; Department of Immunology, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America.
Stokes KL; Department of Immunology, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America.
Vogel P; Veterinary Pathology Department, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America.
Lamkanfi M; Department of Medical Protein Research, Vlaams Instituut voor Biotechnologie, Ghent, Belgium; Department of Biochemistry, Ghent University, Ghent, Belgium.
Kanneganti TD; Department of Immunology, St. Jude Children's Research Hospital, Memphis, Tennessee, United States of America.
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Źródło:
PLoS pathogens [PLoS Pathog] 2014 Sep 25; Vol. 10 (9), pp. e1004410. Date of Electronic Publication: 2014 Sep 25 (Print Publication: 2014).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Carrier Proteins/*metabolism
Caspases/*metabolism
Enterobacteriaceae Infections/*immunology
Inflammasomes/*immunology
Nod2 Signaling Adaptor Protein/*physiology
Reactive Oxygen Species/*metabolism
Receptor-Interacting Protein Serine-Threonine Kinases/*physiology
Animals ; Blotting, Western ; Caspases, Initiator ; Citrobacter rodentium/pathogenicity ; Colitis/immunology ; Colitis/metabolism ; Colitis/microbiology ; Colitis/pathology ; Enterobacteriaceae Infections/metabolism ; Enterobacteriaceae Infections/microbiology ; Enterobacteriaceae Infections/pathology ; Female ; Inflammasomes/metabolism ; JNK Mitogen-Activated Protein Kinases/metabolism ; Macrophages/immunology ; Macrophages/metabolism ; Macrophages/microbiology ; Macrophages/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Inbred NOD ; Mice, Knockout ; NLR Family, Pyrin Domain-Containing 3 Protein ; Receptor-Interacting Protein Serine-Threonine Kinase 2 ; Signal Transduction
Czasopismo naukowe
Tytuł:
A role for the transcription factor Mca1 in activating the meiosis-specific copper transporter Mfc1.
Autorzy:
Beaudoin J; Département de Biochimie, Faculté de médecine et des sciences de la santé, Université de Sherbrooke, Sherbrooke, QC, Canada.
Ioannoni R; Département de Biochimie, Faculté de médecine et des sciences de la santé, Université de Sherbrooke, Sherbrooke, QC, Canada.
Normant V; Département de Biochimie, Faculté de médecine et des sciences de la santé, Université de Sherbrooke, Sherbrooke, QC, Canada.
Labbé S; Département de Biochimie, Faculté de médecine et des sciences de la santé, Université de Sherbrooke, Sherbrooke, QC, Canada.
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Źródło:
PloS one [PLoS One] 2018 Aug 07; Vol. 13 (8), pp. e0201861. Date of Electronic Publication: 2018 Aug 07 (Print Publication: 2018).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Caspases/*metabolism
Copper/*metabolism
Membrane Transport Proteins/*metabolism
RNA Polymerase II/*metabolism
Schizosaccharomyces pombe Proteins/*metabolism
Amino Acid Sequence ; Caspases/genetics ; Copper/deficiency ; Gene Expression Regulation, Fungal/physiology ; Genetic Loci ; Meiosis/physiology ; Membrane Transport Proteins/genetics ; Mitosis/physiology ; Promoter Regions, Genetic ; Protein Binding ; Schizosaccharomyces ; Schizosaccharomyces pombe Proteins/genetics
Czasopismo naukowe
Tytuł:
Bcl-2-mediated control of TRAIL-induced apoptotic response in the non-small lung cancer cell line NCI-H460 is effective at late caspase processing steps.
Autorzy:
Danish L; Institute of Cell Biology and Immunology, University of Stuttgart, Stuttgart, Germany.
Imig D; Institute of Systems Theory and Automatic Control, University of Stuttgart, Stuttgart, Germany.
Allgöwer F; Institute of Systems Theory and Automatic Control, University of Stuttgart, Stuttgart, Germany.
Scheurich P; Institute of Cell Biology and Immunology, University of Stuttgart, Stuttgart, Germany.
Pollak N; Institute of Cell Biology and Immunology, University of Stuttgart, Stuttgart, Germany.; Institute of Systems Theory and Automatic Control, University of Stuttgart, Stuttgart, Germany.
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Źródło:
PloS one [PLoS One] 2018 Jun 21; Vol. 13 (6), pp. e0198203. Date of Electronic Publication: 2018 Jun 21 (Print Publication: 2018).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis*
Models, Biological*
Signal Transduction*
Carcinoma, Non-Small-Cell Lung/*metabolism
Caspases/*metabolism
Lung Neoplasms/*metabolism
Proto-Oncogene Proteins c-bcl-2/*metabolism
TNF-Related Apoptosis-Inducing Ligand/*metabolism
Carcinoma, Non-Small-Cell Lung/genetics ; Carcinoma, Non-Small-Cell Lung/pathology ; Caspases/genetics ; HCT116 Cells ; HeLa Cells ; Humans ; Lung Neoplasms/genetics ; Lung Neoplasms/pathology ; Proto-Oncogene Proteins c-bcl-2/genetics ; TNF-Related Apoptosis-Inducing Ligand/genetics ; X-Linked Inhibitor of Apoptosis Protein/genetics ; X-Linked Inhibitor of Apoptosis Protein/metabolism
Czasopismo naukowe
Tytuł:
Macrophage specific caspase-1/11 deficiency protects against cholesterol crystallization and hepatic inflammation in hyperlipidemic mice.
Autorzy:
Hendrikx T; Departments of Molecular Genetics, Electron Microscopy and Internal Medicine, Nutrition and Toxicology Research (NUTRIM) Institute of Maastricht and Maastricht University Medical Centre (MUMC), Maastricht University, Maastricht, The Netherlands.
Bieghs V
Walenbergh SM
van Gorp PJ
Verheyen F
Jeurissen ML
Steinbusch MM
Vaes N
Binder CJ
Koek GH
Stienstra R
Netea MG
Hofker MH
Shiri-Sverdlov R
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Źródło:
PloS one [PLoS One] 2013 Dec 02; Vol. 8 (12), pp. e78792. Date of Electronic Publication: 2013 Dec 02 (Print Publication: 2013).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Caspase 1/*metabolism
Caspases/*metabolism
Hepatitis/*enzymology
Hyperlipidemias/*enzymology
Kupffer Cells/*enzymology
Animals ; Caspase 1/genetics ; Caspases/genetics ; Caspases, Initiator ; Cholesterol/genetics ; Cholesterol/metabolism ; Dietary Fats/adverse effects ; Dietary Fats/pharmacology ; Hepatitis/genetics ; Hepatitis/pathology ; Hyperlipidemias/genetics ; Hyperlipidemias/pathology ; Inflammasomes/genetics ; Inflammasomes/metabolism ; Interleukin-18/genetics ; Interleukin-18/metabolism ; Interleukin-1beta/genetics ; Interleukin-1beta/metabolism ; Kupffer Cells/pathology ; Lipoproteins, LDL/genetics ; Lipoproteins, LDL/metabolism ; Mice ; Mice, Knockout
Czasopismo naukowe
Tytuł:
The colonic pathogen Entamoeba histolytica activates caspase-4/1 that cleaves the pore-forming protein gasdermin D to regulate IL-1β secretion.
Autorzy:
Wang S; Department of Microbiology, Immunology and Infectious Diseases, University of Calgary, Calgary, Alberta, Canada.
Moreau F; Department of Microbiology, Immunology and Infectious Diseases, University of Calgary, Calgary, Alberta, Canada.
Chadee K; Department of Microbiology, Immunology and Infectious Diseases, University of Calgary, Calgary, Alberta, Canada.
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Źródło:
PLoS pathogens [PLoS Pathog] 2022 Mar 18; Vol. 18 (3), pp. e1010415. Date of Electronic Publication: 2022 Mar 18 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Entamoeba histolytica*/metabolism
Caspase 1/genetics ; Caspase 1/metabolism ; Caspases, Initiator/metabolism ; Humans ; Interleukin-1beta ; Intracellular Signaling Peptides and Proteins/genetics ; Intracellular Signaling Peptides and Proteins/metabolism ; Phosphate-Binding Proteins/genetics ; Phosphate-Binding Proteins/metabolism ; Pore Forming Cytotoxic Proteins/metabolism ; Pyroptosis
Czasopismo naukowe

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