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Tytuł:
Tceal7 Regulates Skeletal Muscle Development through Its Interaction with Cdk1.
Autorzy:
Xiong Z; School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China.
Wang M; School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China.
Wu J; School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China.
Shi X; School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2023 Mar 27; Vol. 24 (7). Date of Electronic Publication: 2023 Mar 27.
Typ publikacji:
Journal Article
MeSH Terms:
Cyclin A2*/metabolism
Muscle Development*/genetics
Animals ; Mice ; Amino Acid Sequence ; Muscle, Skeletal/metabolism ; Phosphorylation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Impact of Freeze-Drying on Cord Blood (CB), Serum (S), and Platelet-Rich Plasma (CB-PRP) Preparations on Growth Factor Content and In Vitro Cell Wound Healing.
Autorzy:
Valente S; DIMES, Alma Mater Studiorum University of Bologna, 40138 Bologna, Italy.; Center for Applied Biomedical Research (CRBA), Alma Mater Studiorum University of Bologna, 40138 Bologna, Italy.
Ciavarella C; DIMES, Alma Mater Studiorum University of Bologna, 40138 Bologna, Italy.; Center for Applied Biomedical Research (CRBA), Alma Mater Studiorum University of Bologna, 40138 Bologna, Italy.
Astolfi G; Center for Applied Biomedical Research (CRBA), Alma Mater Studiorum University of Bologna, 40138 Bologna, Italy.; Ophthalmology Unit, DIMES, Alma Mater Studiorum University of Bologna, 40138 Bologna, Italy.
Bergantin E; IRCCS Azienda Ospedaliero Universitaria di Bologna, 40138 Bologna, Italy.
Curti N; eDIMES Lab, DIMES, Alma Mater Studiorum University of Bologna, 40138 Bologna, Italy.
Buzzi M; IRCCS Azienda Ospedaliero Universitaria di Bologna, 40138 Bologna, Italy.
Fontana L; Ophthalmology Unit, DIMES, Alma Mater Studiorum University of Bologna, 40138 Bologna, Italy.; IRCCS Azienda Ospedaliero Universitaria di Bologna, 40138 Bologna, Italy.
Versura P; Center for Applied Biomedical Research (CRBA), Alma Mater Studiorum University of Bologna, 40138 Bologna, Italy.; Ophthalmology Unit, DIMES, Alma Mater Studiorum University of Bologna, 40138 Bologna, Italy.; IRCCS Azienda Ospedaliero Universitaria di Bologna, 40138 Bologna, Italy.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2022 Sep 14; Vol. 23 (18). Date of Electronic Publication: 2022 Sep 14.
Typ publikacji:
Journal Article
MeSH Terms:
Cyclin A2*/metabolism
Platelet-Rich Plasma*/metabolism
Brain-Derived Neurotrophic Factor/metabolism ; Cell Proliferation ; Epidermal Growth Factor/metabolism ; Epidermal Growth Factor/pharmacology ; Fetal Blood ; Humans ; Vimentin/metabolism ; Wound Healing/physiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
BUB1, BUB1B, CCNA2, and CDCA8, along with miR-524-5p, as clinically relevant biomarkers for the diagnosis and treatment of endometrial carcinoma.
Autorzy:
Hao Q; Department of Obstetrics and Gynecology, the Second Hospital of Shanxi Medical University, Taiyuan, 030001, China. .
Wu H; Department of Obstetrics and Gynecology, the Second Hospital of Shanxi Medical University, Taiyuan, 030001, China.
Liu E; Department of Obstetrics and Gynecology, the Second Hospital of Shanxi Medical University, Taiyuan, 030001, China.
Wang L; Department of Obstetrics and Gynecology, the Second Hospital of Shanxi Medical University, Taiyuan, 030001, China.
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Źródło:
BMC cancer [BMC Cancer] 2023 Oct 18; Vol. 23 (1), pp. 995. Date of Electronic Publication: 2023 Oct 18.
Typ publikacji:
Journal Article
MeSH Terms:
MicroRNAs*/genetics
MicroRNAs*/metabolism
Endometrial Neoplasms*/diagnosis
Endometrial Neoplasms*/genetics
Endometrial Neoplasms*/metabolism
Humans ; Female ; Cell Line, Tumor ; Cell Proliferation/genetics ; Biomarkers ; Gene Expression Regulation, Neoplastic ; Cell Movement/genetics ; Cell Cycle Proteins/genetics ; Cell Cycle Proteins/metabolism ; Cyclin A2/genetics ; Protein Serine-Threonine Kinases/genetics ; Protein Serine-Threonine Kinases/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Tanshinone IIA suppresses the progression of lung adenocarcinoma through regulating CCNA2-CDK2 complex and AURKA/PLK1 pathway.
Autorzy:
Li Z; Faculty of Pharmacy, Hubei University of Chinese Medicine, Wuhan, 430065, China.
Zhang Y; Faculty of Pharmacy, Hubei University of Chinese Medicine, Wuhan, 430065, China.
Zhou Y; Faculty of Pharmacy, Hubei University of Chinese Medicine, Wuhan, 430065, China.
Wang F; Faculty of Pharmacy, Hubei University of Chinese Medicine, Wuhan, 430065, China.
Yin C; Faculty of Pharmacy, Hubei University of Chinese Medicine, Wuhan, 430065, China.
Ding L; Faculty of Pharmacy, Hubei University of Chinese Medicine, Wuhan, 430065, China. .
Zhang S; Faculty of Pharmacy, Hubei University of Chinese Medicine, Wuhan, 430065, China. .
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Źródło:
Scientific reports [Sci Rep] 2021 Dec 08; Vol. 11 (1), pp. 23681. Date of Electronic Publication: 2021 Dec 08.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Abietanes/*pharmacology
Adenocarcinoma of Lung/*metabolism
Antineoplastic Agents, Phytogenic/*pharmacology
Aurora Kinase A/*metabolism
Cell Cycle Proteins/*metabolism
Cyclin A2/*metabolism
Cyclin-Dependent Kinase 2/*metabolism
Protein Serine-Threonine Kinases/*metabolism
Proto-Oncogene Proteins/*metabolism
Abietanes/chemistry ; Adenocarcinoma of Lung/drug therapy ; Adenocarcinoma of Lung/etiology ; Adenocarcinoma of Lung/pathology ; Antineoplastic Agents, Phytogenic/chemistry ; Apoptosis/drug effects ; Aurora Kinase A/chemistry ; Biomarkers, Tumor ; Cell Cycle/drug effects ; Cell Cycle Proteins/chemistry ; Cell Line, Tumor ; Computational Biology/methods ; Cyclin A2/chemistry ; Cyclin-Dependent Kinase 2/chemistry ; Disease Susceptibility ; Gene Expression Profiling ; Humans ; Models, Molecular ; Protein Interaction Mapping ; Protein Interaction Maps ; Protein Serine-Threonine Kinases/chemistry ; Proto-Oncogene Proteins/chemistry ; Signal Transduction/drug effects ; Structure-Activity Relationship ; Transcriptome ; Polo-Like Kinase 1
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Long non-coding RNAs HERH-1 and HERH-4 facilitate cyclin A2 expression and accelerate cell cycle progression in advanced hepatocellular carcinoma.
Autorzy:
Liu T; National Health Commission's Key Laboratory of Critical Care Medicine, Tianjin First Central Hospital, School of Medicine, Nankai University, No. 24 Fukang Road, Nankai District, Tianjin, 300192, China. .; Key Laboratory of Transplant Medicine, Chinese Academy of Medical Sciences, Tianjin First Central Hospital, School of Medicine, Nankai University, Tianjin, 300192, China. .
Shi Q; Graduate School, Tianjin University of Traditional Chinese Medicine, Tianjin, 301617, China.
Yang L; Department of Clinical Laboratory, Tianjin First Central Hospital, School of Medicine, Nankai University, Tianjin, 300192, China.
Wang S; National Health Commission's Key Laboratory of Critical Care Medicine, Tianjin First Central Hospital, School of Medicine, Nankai University, No. 24 Fukang Road, Nankai District, Tianjin, 300192, China.; Key Laboratory of Transplant Medicine, Chinese Academy of Medical Sciences, Tianjin First Central Hospital, School of Medicine, Nankai University, Tianjin, 300192, China.; Organ Transplant Center, Tianjin First Central Hospital, School of Medicine, Nankai University, Tianjin, 300192, China.
Song H; Organ Transplant Center, Tianjin First Central Hospital, School of Medicine, Nankai University, Tianjin, 300192, China.
Wang Z; Biological Sample Resource Sharing Center (BSRSC), Tianjin First Central Hospital, School of Medicine, Nankai University, Tianjin, 300192, China.
Xu X; National Health Commission's Key Laboratory of Critical Care Medicine, Tianjin First Central Hospital, School of Medicine, Nankai University, No. 24 Fukang Road, Nankai District, Tianjin, 300192, China.
Liu H; National Health Commission's Key Laboratory of Critical Care Medicine, Tianjin First Central Hospital, School of Medicine, Nankai University, No. 24 Fukang Road, Nankai District, Tianjin, 300192, China.
Zheng H; National Health Commission's Key Laboratory of Critical Care Medicine, Tianjin First Central Hospital, School of Medicine, Nankai University, No. 24 Fukang Road, Nankai District, Tianjin, 300192, China.; Key Laboratory of Transplant Medicine, Chinese Academy of Medical Sciences, Tianjin First Central Hospital, School of Medicine, Nankai University, Tianjin, 300192, China.; Organ Transplant Center, Tianjin First Central Hospital, School of Medicine, Nankai University, Tianjin, 300192, China.
Shen Z; National Health Commission's Key Laboratory of Critical Care Medicine, Tianjin First Central Hospital, School of Medicine, Nankai University, No. 24 Fukang Road, Nankai District, Tianjin, 300192, China. .; Key Laboratory of Transplant Medicine, Chinese Academy of Medical Sciences, Tianjin First Central Hospital, School of Medicine, Nankai University, Tianjin, 300192, China. .; Organ Transplant Center, Tianjin First Central Hospital, School of Medicine, Nankai University, Tianjin, 300192, China. .
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Źródło:
BMC cancer [BMC Cancer] 2021 Aug 26; Vol. 21 (1), pp. 957. Date of Electronic Publication: 2021 Aug 26.
Typ publikacji:
Journal Article
MeSH Terms:
Cell Cycle*
Gene Expression Regulation, Neoplastic*
Biomarkers, Tumor/*metabolism
Carcinoma, Hepatocellular/*pathology
Cyclin A2/*metabolism
Liver Neoplasms/*pathology
RNA, Long Noncoding/*genetics
Apoptosis ; Biomarkers, Tumor/genetics ; Carcinoma, Hepatocellular/genetics ; Carcinoma, Hepatocellular/metabolism ; Cell Proliferation ; Cyclin A2/genetics ; Humans ; Liver Neoplasms/genetics ; Liver Neoplasms/metabolism ; Prognosis ; Survival Rate ; Tumor Cells, Cultured
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
ESRP1 Induces Cervical Cancer Cell G1-Phase Arrest Via Regulating Cyclin A2 mRNA Stability.
Autorzy:
Chen ZH; School of Basic Medicine, Youjiang Medical University for Nationalities, No. 98 Chengxiang Road, Baise 533000, China. .; Heilongjiang Province Key Laboratory of Cancer Prevention and Treatment, Mudanjiang Medical University, No. 3, Tongxiang Street, Mudanjiang 157011, China. .
Jing YJ; Sciences Research Center, Youjiang Medical University for Nationalities, No. 98 Chengxiang Road, Baise 533000, China.
Yu JB; Heilongjiang Province Key Laboratory of Cancer Prevention and Treatment, Mudanjiang Medical University, No. 3, Tongxiang Street, Mudanjiang 157011, China.
Jin ZS; Heilongjiang Province Key Laboratory of Cancer Prevention and Treatment, Mudanjiang Medical University, No. 3, Tongxiang Street, Mudanjiang 157011, China.
Li Z; Heilongjiang Province Key Laboratory of Cancer Prevention and Treatment, Mudanjiang Medical University, No. 3, Tongxiang Street, Mudanjiang 157011, China.
He TT; Sciences Research Center, Youjiang Medical University for Nationalities, No. 98 Chengxiang Road, Baise 533000, China.
Su XZ; Sciences Research Center, Youjiang Medical University for Nationalities, No. 98 Chengxiang Road, Baise 533000, China.
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Źródło:
International journal of molecular sciences [Int J Mol Sci] 2019 Jul 29; Vol. 20 (15). Date of Electronic Publication: 2019 Jul 29.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Expression Regulation, Neoplastic*
RNA Stability*
Cyclin A2/*genetics
G1 Phase Cell Cycle Checkpoints/*genetics
RNA-Binding Proteins/*genetics
Uterine Cervical Neoplasms/*genetics
3' Untranslated Regions ; Cdc20 Proteins/genetics ; Cdc20 Proteins/metabolism ; Cell Line, Tumor ; Cyclin A2/metabolism ; Female ; Humans ; Immunohistochemistry ; Models, Biological ; Protein Binding ; RNA, Messenger/genetics ; RNA-Binding Proteins/metabolism ; Uterine Cervical Neoplasms/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Downregulation of CCNA2 disturbs trophoblast migration, proliferation, and apoptosis during the pathogenesis of recurrent miscarriage.
Autorzy:
Li X; Shanghai Key Laboratory of Embryo Original Diseases, The International Peace Maternity and Child Health Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Ma XL; Shanghai Key Laboratory of Embryo Original Diseases, The International Peace Maternity and Child Health Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Tian FJ; Shanghai Key Laboratory of Embryo Original Diseases, The International Peace Maternity and Child Health Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Wu F; Shanghai Key Laboratory of Embryo Original Diseases, The International Peace Maternity and Child Health Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Zhang J; Shanghai Key Laboratory of Embryo Original Diseases, The International Peace Maternity and Child Health Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Zeng WH; Shanghai Key Laboratory of Embryo Original Diseases, The International Peace Maternity and Child Health Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Lin Y; Shanghai Key Laboratory of Embryo Original Diseases, The International Peace Maternity and Child Health Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Zhang Y; Department of Obstetrics and Gynecology, Renmin Hospital of Wuhan University, Wuhan, China.
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Źródło:
American journal of reproductive immunology (New York, N.Y. : 1989) [Am J Reprod Immunol] 2019 Jul; Vol. 82 (1), pp. e13144. Date of Electronic Publication: 2019 May 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Abortion, Habitual/*metabolism
Cyclin A2/*metabolism
Trophoblasts/*physiology
Abortion, Habitual/genetics ; Adult ; Apoptosis ; Cell Line ; Cell Movement ; Cell Proliferation ; Chorionic Villi/metabolism ; Cyclin A2/genetics ; Down-Regulation ; Female ; Gene Knockdown Techniques ; Humans ; Tumor Suppressor Protein p53/genetics ; Young Adult ; rho-Associated Kinases/genetics ; rho-Associated Kinases/metabolism ; rhoA GTP-Binding Protein/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
The p53/miRNAs/Ccna2 pathway serves as a novel regulator of cellular senescence: Complement of the canonical p53/p21 pathway.
Autorzy:
Xu S; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; The Scientific Research Center of Dongguan, Guangdong Medical University, Dongguan, China.
Wu W; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; The Scientific Research Center of Dongguan, Guangdong Medical University, Dongguan, China.
Huang H; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; The Scientific Research Center of Dongguan, Guangdong Medical University, Dongguan, China.
Huang R; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; The Scientific Research Center of Dongguan, Guangdong Medical University, Dongguan, China.
Xie L; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; The Scientific Research Center of Dongguan, Guangdong Medical University, Dongguan, China.
Su A; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; The Scientific Research Center of Dongguan, Guangdong Medical University, Dongguan, China.
Liu S; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; The Scientific Research Center of Dongguan, Guangdong Medical University, Dongguan, China.
Zheng R; Department of Oncology, Dongguan People's Hospital, Dongguan, China.
Yuan Y; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; The Scientific Research Center of Dongguan, Guangdong Medical University, Dongguan, China.
Zheng HL; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; The Scientific Research Center of Dongguan, Guangdong Medical University, Dongguan, China.
Sun X; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; The Scientific Research Center of Dongguan, Guangdong Medical University, Dongguan, China.
Xiong XD; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; Institute of Biochemistry & Molecular Biology, Guangdong Medical University, Zhanjiang, China.
Liu X; Institute of Aging Research, Guangdong Medical University, Dongguan, China.; Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan, China.; Institute of Biochemistry & Molecular Biology, Guangdong Medical University, Zhanjiang, China.
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Źródło:
Aging cell [Aging Cell] 2019 Jun; Vol. 18 (3), pp. e12918. Date of Electronic Publication: 2019 Mar 07.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cellular Senescence*/genetics
Cyclin A2/*metabolism
Cyclin-Dependent Kinase Inhibitor p21/*metabolism
MicroRNAs/*metabolism
Tumor Suppressor Protein p53/*metabolism
Animals ; Cells, Cultured ; Cyclin A2/deficiency ; Cyclin A2/genetics ; Humans ; Male ; Mice ; Mice, Inbred C57BL ; MicroRNAs/genetics ; NIH 3T3 Cells ; Tumor Suppressor Protein p53/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
ECPPF (E2F1, CCNA2, POLE, PPP2R1A, FBXW7) stratification: Profiling high-risk subtypes of histomorphologically low-risk and treatment-insensitive endometrioid endometrial cancer.
Autorzy:
Gonzalez-Bosquet J; Department of Obstetrics and Gynecology, University of Iowa, Iowa City, Iowa, United States of America.
Weroha SJ; Division of Medical Oncology, Mayo Clinic, Rochester, Minnesota, United States of America.; Division of Clinical Pharmacology, Mayo Clinic, Rochester, Minnesota, United States of America.; Mayo Clinic Cancer Center, Mayo Clinic, Rochester, Minnesota, United States of America.
Bakkum-Gamez JN; Mayo Clinic Cancer Center, Mayo Clinic, Rochester, Minnesota, United States of America.; Department of Obstetrics and Gynecology, Mayo Clinic, Rochester, Minnesota, United States of America.
Weaver AL; Department of Quantitative Health Sciences, Mayo Clinic, Rochester, Minnesota, United States of America.
McGree ME; Department of Quantitative Health Sciences, Mayo Clinic, Rochester, Minnesota, United States of America.
Dowdy SC; Mayo Clinic Cancer Center, Mayo Clinic, Rochester, Minnesota, United States of America.; Department of Obstetrics and Gynecology, Mayo Clinic, Rochester, Minnesota, United States of America.
Famuyide AO; Department of Obstetrics and Gynecology, Mayo Clinic, Rochester, Minnesota, United States of America.
Kipp BR; Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota, United States of America.; Department of Clinical Genomics, Mayo Clinic, Rochester, Minnesota, United States of America.
Halling KC; Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota, United States of America.; Department of Clinical Genomics, Mayo Clinic, Rochester, Minnesota, United States of America.
Yadav S; Division of Medical Oncology, Mayo Clinic, Rochester, Minnesota, United States of America.
Couch FJ; Department of Quantitative Health Sciences, Mayo Clinic, Rochester, Minnesota, United States of America.; Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota, United States of America.; Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, Minnesota, United States of America.
Podratz KC; Department of Obstetrics and Gynecology, Mayo Clinic, Rochester, Minnesota, United States of America.
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Źródło:
PloS one [PLoS One] 2022 Dec 01; Vol. 17 (12), pp. e0278408. Date of Electronic Publication: 2022 Dec 01 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
Carcinoma, Endometrioid*
Endometrial Neoplasms*/genetics
Endometrial Neoplasms*/therapy
Female ; Humans ; Genes, cdc ; F-Box-WD Repeat-Containing Protein 7/genetics ; Genes, Regulator ; Transcription Factors ; E2F1 Transcription Factor ; Cyclin A2 ; Protein Phosphatase 2/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
PDZRN3 protects against apoptosis in myoblasts by maintaining cyclin A2 expression.
Autorzy:
Honda T; Department of Pharmacology, Yamaguchi University Graduate School of Medicine, Ube, Yamaguchi, 755-8505, Japan.
Inui M; Department of Pharmacology, Yamaguchi University Graduate School of Medicine, Ube, Yamaguchi, 755-8505, Japan. .; YIC Rehabilitation College, 4-11-1 Nishiube-Minami, Ube, Yamaguchi, 759-0208, Japan. .
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Źródło:
Scientific reports [Sci Rep] 2020 Jan 24; Vol. 10 (1), pp. 1140. Date of Electronic Publication: 2020 Jan 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Apoptosis/*physiology
Cyclin A2/*biosynthesis
Myoblasts/*metabolism
Ubiquitin-Protein Ligases/*physiology
Animals ; Cell Adhesion ; Cell Cycle ; Cell Division ; Cells, Cultured ; Cyclin A2/genetics ; DNA Damage ; Down-Regulation ; Gene Expression Regulation ; Genomic Instability ; Mesenchymal Stem Cells/metabolism ; Mice ; Myoblasts/cytology ; RNA Interference ; RNA, Messenger/biosynthesis ; RNA, Messenger/genetics ; RNA, Small Interfering/genetics ; RNA, Small Interfering/pharmacology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
MCTS1 promotes laryngeal squamous cell carcinoma cell growth via enhancing LARP7 stability.
Autorzy:
Yang M; Otorhinolaryngology - Head and Neck Surgery, Gansu Provincial Hospital, Lanzhou, China.
Ma B; Otorhinolaryngology - Head and Neck Surgery, Gansu Provincial Hospital, Lanzhou, China.
Liu X; Otorhinolaryngology - Head and Neck Surgery, Affiliated Hospital of Gansu University of Traditional Chinese Medicine, Lanzhou, China.
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Źródło:
Clinical and experimental pharmacology & physiology [Clin Exp Pharmacol Physiol] 2022 Jun; Vol. 49 (6), pp. 652-660. Date of Electronic Publication: 2022 Mar 28.
Typ publikacji:
Journal Article
MeSH Terms:
Head and Neck Neoplasms*
Laryngeal Neoplasms*/genetics
Laryngeal Neoplasms*/pathology
MicroRNAs*/genetics
Cell Cycle Proteins/genetics ; Cell Line, Tumor ; Cell Proliferation/genetics ; Cyclin A2/genetics ; Cyclin A2/metabolism ; Cyclin B1/genetics ; Cyclin B1/metabolism ; Gene Expression Regulation, Neoplastic ; Humans ; Oncogene Proteins/metabolism ; Ribonucleoproteins/genetics ; Ribonucleoproteins/metabolism ; Squamous Cell Carcinoma of Head and Neck/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Microarray analysis reveals Tmub1 as a cell cycle-associated protein in rat hepatocytes.
Autorzy:
Fu H; Department of Hepatobiliary Surgery, Institute of Surgery Research, Daping Hospital, Third Military Medical University, Chongqing 400042, P.R. China.
Xu J; Department of Hepatobiliary Surgery, Institute of Surgery Research, Daping Hospital, Third Military Medical University, Chongqing 400042, P.R. China.
Chen J; Department of Hepatobiliary Surgery, Institute of Surgery Research, Daping Hospital, Third Military Medical University, Chongqing 400042, P.R. China.
Li G; Department of Hepatobiliary Surgery, Institute of Surgery Research, Daping Hospital, Third Military Medical University, Chongqing 400042, P.R. China.
Zhao X; Department of Hepatobiliary Surgery, 187 Military Hospital, Haikou, Hainan 571159, P.R. China.
Chen P; Department of Hepatobiliary Surgery, Institute of Surgery Research, Daping Hospital, Third Military Medical University, Chongqing 400042, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2018 Mar; Vol. 17 (3), pp. 4337-4344. Date of Electronic Publication: 2018 Jan 17.
Typ publikacji:
Journal Article
MeSH Terms:
Carrier Proteins/*genetics
Cell Cycle/*genetics
Cyclin A2/*genetics
Cyclin B1/*genetics
Hepatocytes/*metabolism
Nuclear Proteins/*genetics
RNA, Messenger/*genetics
Animals ; Carrier Proteins/agonists ; Carrier Proteins/antagonists & inhibitors ; Carrier Proteins/metabolism ; Cell Line ; Cell Proliferation ; Cyclin A2/metabolism ; Cyclin B1/metabolism ; Gene Expression Profiling ; Gene Expression Regulation ; Gene Regulatory Networks ; Hepatocytes/cytology ; Microarray Analysis ; Nuclear Proteins/agonists ; Nuclear Proteins/antagonists & inhibitors ; Nuclear Proteins/metabolism ; Protein Interaction Mapping ; RNA, Messenger/metabolism ; Rats ; Signal Transduction ; Transcription, Genetic
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
E2F1 transcriptionally regulates CCNA2 expression to promote triple negative breast cancer tumorigenicity.
Autorzy:
Lu Y; Scientific Development and Planing Department, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.; College of Earth and Environmental Sciences, Lanzhou University, Lanzhou, Gansu, China.; Key Laboratory of Biotherapy and Regenerative Medicine of Gansu Province, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.; Scientific Development and Planing Department, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.
Su F; Department of Oncology, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.; Scientific Development and Planing Department, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.
Yang H; International Medical Department Area B, Gansu Provincial Maternity and Child-care Hospital, Lanzhou, Gansu, China.; Scientific Development and Planing Department, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.
Xiao Y; Breast surgery, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.
Zhang X; Breast surgery, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.
Su H; Department of Radiotherapy, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.
Zhang T; Department of Oncology, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.
Bai Y; College of Earth and Environmental Sciences, Lanzhou University, Lanzhou, Gansu, China.; School of Public Health, Lanzhou University, Lanzhou, Gansu, China.
Ling X; Department of Oncology, The First Hospital of Lanzhou University, Lanzhou, Gansu, China.
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Źródło:
Cancer biomarkers : section A of Disease markers [Cancer Biomark] 2022; Vol. 33 (1), pp. 57-70.
Typ publikacji:
Journal Article
MeSH Terms:
Triple Negative Breast Neoplasms*/pathology
Cell Line, Tumor ; Cell Movement/genetics ; Cell Proliferation/genetics ; Cyclin A2/metabolism ; E2F1 Transcription Factor/genetics ; Gene Expression Regulation, Neoplastic ; Humans ; Up-Regulation
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
CSN1 facilitates proliferation and migration of hepatocellular carcinoma cells by upregulating cyclin A2 expression.
Autorzy:
Fu H; Department of Hepatobiliary Surgery, Daping Hospital, Army Medical University, Chongqing 400042, P.R. China.
Zhang Y; Department of Hepatobiliary Surgery, Daping Hospital, Army Medical University, Chongqing 400042, P.R. China.
Chen Y; Department of Hepatobiliary Surgery, Daping Hospital, Army Medical University, Chongqing 400042, P.R. China.
Chen J; Department of Hepatobiliary Surgery, Daping Hospital, Army Medical University, Chongqing 400042, P.R. China.
Chen P; Department of Hepatobiliary Surgery, Daping Hospital, Army Medical University, Chongqing 400042, P.R. China.
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Źródło:
Molecular medicine reports [Mol Med Rep] 2021 Jan; Vol. 23 (1). Date of Electronic Publication: 2020 Nov 17.
Typ publikacji:
Journal Article
MeSH Terms:
COP9 Signalosome Complex/*genetics
COP9 Signalosome Complex/*metabolism
Carcinoma, Hepatocellular/*genetics
Cyclin A2/*genetics
Cyclin A2/*metabolism
Liver Neoplasms/*genetics
Adult ; Animals ; Apoptosis/genetics ; Carcinoma, Hepatocellular/metabolism ; Carcinoma, Hepatocellular/pathology ; Cell Cycle Checkpoints/genetics ; Cell Line, Tumor ; Cell Movement/genetics ; Cell Proliferation/genetics ; Female ; Gene Expression Regulation, Neoplastic ; Gene Knockdown Techniques ; Humans ; Liver Neoplasms/metabolism ; Liver Neoplasms/pathology ; Male ; Mice, Inbred BALB C ; Mice, Nude ; Middle Aged ; Prognosis ; Transplantation, Heterologous ; Up-Regulation ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Meioc maintains an extended meiotic prophase I in mice.
Autorzy:
Soh YQS; Whitehead Institute, Cambridge, MA, United States of America.; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA, United States of America.
Mikedis MM; Whitehead Institute, Cambridge, MA, United States of America.
Kojima M; Whitehead Institute, Cambridge, MA, United States of America.; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA, United States of America.
Godfrey AK; Whitehead Institute, Cambridge, MA, United States of America.; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA, United States of America.
de Rooij DG; Whitehead Institute, Cambridge, MA, United States of America.
Page DC; Whitehead Institute, Cambridge, MA, United States of America.; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA, United States of America.; Howard Hughes Medical Institute, Whitehead Institute, Cambridge, MA, United States of America.
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Źródło:
PLoS genetics [PLoS Genet] 2017 Apr 05; Vol. 13 (4), pp. e1006704. Date of Electronic Publication: 2017 Apr 05 (Print Publication: 2017).
Typ publikacji:
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Cycle Proteins/*genetics
Cyclin A2/*biosynthesis
Meiosis/*genetics
Prophase/*genetics
RNA-Binding Proteins/*genetics
Animals ; Cell Cycle Proteins/biosynthesis ; Chromosome Pairing/genetics ; Cyclin A2/genetics ; Gene Expression Regulation, Developmental ; Male ; Mice ; Mitosis/genetics ; RNA-Binding Proteins/metabolism ; Spermatocytes ; Spermatogenesis/genetics ; Spermatogonia/growth & development ; Spermatogonia/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Cyclin A2 is an RNA binding protein that controls Mre11 mRNA translation.
Autorzy:
Kanakkanthara A; Department of Pediatric and Adolescent Medicine, Mayo Clinic, Rochester, MN, USA.
Jeganathan KB; Department of Pediatric and Adolescent Medicine, Mayo Clinic, Rochester, MN, USA.
Limzerwala JF; Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN, USA.
Baker DJ; Department of Pediatric and Adolescent Medicine, Mayo Clinic, Rochester, MN, USA.
Hamada M; Department of Pediatric and Adolescent Medicine, Mayo Clinic, Rochester, MN, USA.
Nam HJ; Department of Pediatric and Adolescent Medicine, Mayo Clinic, Rochester, MN, USA.
van Deursen WH; Department of Pediatric and Adolescent Medicine, Mayo Clinic, Rochester, MN, USA.
Hamada N; Department of Pediatric and Adolescent Medicine, Mayo Clinic, Rochester, MN, USA.
Naylor RM; Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN, USA.
Becker NA; Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN, USA.
Davies BA; Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN, USA.
van Ree JH; Department of Pediatric and Adolescent Medicine, Mayo Clinic, Rochester, MN, USA.
Mer G; Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN, USA.
Shapiro VS; Department of Immunology, Mayo Clinic, Rochester, MN, USA.
Maher LJ 3rd; Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN, USA.
Katzmann DJ; Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN, USA.
van Deursen JM; Department of Pediatric and Adolescent Medicine, Mayo Clinic, Rochester, MN, USA. Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN, USA. .
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Źródło:
Science (New York, N.Y.) [Science] 2016 Sep 30; Vol. 353 (6307), pp. 1549-1552.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Chromosomal Instability*
Gene Expression Regulation*
Cyclin A2/*metabolism
DNA Repair Enzymes/*genetics
DNA Replication/*genetics
DNA-Binding Proteins/*genetics
Protein Biosynthesis/*genetics
RNA, Messenger/*metabolism
RNA-Binding Proteins/*metabolism
Animals ; CDC2 Protein Kinase/metabolism ; Centrosome/metabolism ; Cyclin A2/genetics ; DNA Breaks, Double-Stranded ; DNA Repair ; Humans ; Kinesins/metabolism ; MRE11 Homologue Protein ; Meiosis/genetics ; Mice ; Mice, Mutant Strains ; Mitosis/genetics ; RNA, Messenger/genetics ; RNA-Binding Proteins/genetics ; S Phase/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Foci of cyclin A2 interact with actin and RhoA in mitosis.
Autorzy:
Loukil A; Institut de Génétique Moléculaire de Montpellier, CNRS, Université de Montpellier, 1919 route de Mende, 34293 Montpellier, France.
Izard F; Institut de Génétique Moléculaire de Montpellier, CNRS, Université de Montpellier, 1919 route de Mende, 34293 Montpellier, France.
Georgieva M; Institut de Génétique Moléculaire de Montpellier, CNRS, Université de Montpellier, 1919 route de Mende, 34293 Montpellier, France.; Dynamique des Interactions Membranaires Normales et Pathologiques, CNRS, Université de Montpellier, Pl. E. Bataillon, 34095 Montpellier Cedex 5, France.
Mashayekhan S; Dynamique des Interactions Membranaires Normales et Pathologiques, CNRS, Université de Montpellier, Pl. E. Bataillon, 34095 Montpellier Cedex 5, France.
Blanchard JM; Institut de Génétique Moléculaire de Montpellier, CNRS, Université de Montpellier, 1919 route de Mende, 34293 Montpellier, France.
Parmeggiani A; Dynamique des Interactions Membranaires Normales et Pathologiques, CNRS, Université de Montpellier, Pl. E. Bataillon, 34095 Montpellier Cedex 5, France.; Laboratoire Charles Coulomb, CNRS, Université de Montpellier, Pl. E. Bataillon, 34095 Montpellier Cedex 5, France.
Peter M; Institut de Génétique Moléculaire de Montpellier, CNRS, Université de Montpellier, 1919 route de Mende, 34293 Montpellier, France.
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Źródło:
Scientific reports [Sci Rep] 2016 Jun 09; Vol. 6, pp. 27215. Date of Electronic Publication: 2016 Jun 09.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Mitosis*
Actins/*metabolism
Cyclin A2/*metabolism
rhoA GTP-Binding Protein/*metabolism
Autophagy ; Cell Line, Tumor ; Cyclin A2/genetics ; Enzyme Activation ; Humans ; MCF-7 Cells
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Proliferation tracing reveals regional hepatocyte generation in liver homeostasis and repair.
Autorzy:
He L; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
Pu W; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
Liu X; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
Zhang Z; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
Han M; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.; School of Life Science and Technology, ShanghaiTech University, Shanghai, China.
Li Y; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
Huang X; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
Han X; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
Li Y; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
Liu K; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
Shi M; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China.
Lai L; Shanghai Model Organisms Center, Inc., Shanghai, China.
Sun R; Shanghai Model Organisms Center, Inc., Shanghai, China.
Wang QD; Bioscience Cardiovascular, Research and Early Development, Cardiovascular, Renal and Metabolism (CVRM), BioPharmaceuticals R&D, AstraZeneca, Gothenburg, Sweden.
Ji Y; The Collaborative Innovation Center for Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, China.
Tchorz JS; Novartis Institutes for BioMedical Research, Novartis Pharma AG, Basel, Switzerland.
Zhou B; State Key Laboratory of Cell Biology, Shanghai Institute of Biochemistry and Cell Biology, Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China. .; School of Life Science and Technology, ShanghaiTech University, Shanghai, China.; School of Life Science, Hangzhou Institute for Advanced Study, University of Chinese Academy of Sciences, Hangzhou, China.; Institute for Stem Cell and Regeneration, Chinese Academy of Sciences, Beijing, China.
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Źródło:
Science (New York, N.Y.) [Science] 2021 Feb 26; Vol. 371 (6532).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Proliferation*
Liver Regeneration*
Hepatocytes/*physiology
Liver/*physiology
Animals ; Chemical and Drug Induced Liver Injury/pathology ; Chemical and Drug Induced Liver Injury/physiopathology ; Cyclin A2/genetics ; Hepatectomy ; Homeostasis ; Ki-67 Antigen/analysis ; Ki-67 Antigen/genetics ; Liver/cytology ; Mice
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Tytuł:
Identification of LINC00665-miR-let-7b-CCNA2 competing endogenous RNA network associated with prognosis of lung adenocarcinoma.
Autorzy:
Huang Y; Department of Gastroenterology, The First Affiliated Hospital of Guangzhou University of Chinese Medicine, No. 12 Airport Road, Baiyun District, Guangzhou, 510407, China.
Zhong L; Department of Laboratory Medicine, Guangdong Second Provincial General Hospital, Guangzhou, 510317, China.
Nie K; Department of Gastroenterology, The First Affiliated Hospital of Guangzhou University of Chinese Medicine, No. 12 Airport Road, Baiyun District, Guangzhou, 510407, China.
Li L; Department of Laboratory Medicine, Guangdong Second Provincial General Hospital, Guangzhou, 510317, China.
Song S; Department of Laboratory Medicine, Guangdong Second Provincial General Hospital, Guangzhou, 510317, China.
Liu F; Department of Gastroenterology, The First Affiliated Hospital of Guangzhou University of Chinese Medicine, No. 12 Airport Road, Baiyun District, Guangzhou, 510407, China.
Li P; Department of Gastroenterology, The First Affiliated Hospital of Guangzhou University of Chinese Medicine, No. 12 Airport Road, Baiyun District, Guangzhou, 510407, China.
Cao D; Department of Laboratory Medicine, Guangdong Second Provincial General Hospital, Guangzhou, 510317, China. .
Liu Y; Department of Gastroenterology, The First Affiliated Hospital of Guangzhou University of Chinese Medicine, No. 12 Airport Road, Baiyun District, Guangzhou, 510407, China. .; Lingnan Medical Research Center, Guangzhou University of Chinese Medicine, Guangzhou, 510407, China. .
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Źródło:
Scientific reports [Sci Rep] 2021 Feb 24; Vol. 11 (1), pp. 4434. Date of Electronic Publication: 2021 Feb 24.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Adenocarcinoma of Lung/*genetics
Cyclin A2/*genetics
Lung Neoplasms/*genetics
Adenocarcinoma of Lung/pathology ; Aged ; Down-Regulation/genetics ; Female ; Gene Expression Regulation, Neoplastic/genetics ; Gene Regulatory Networks/genetics ; Humans ; Lung/pathology ; Lung Neoplasms/pathology ; Male ; MicroRNAs/genetics ; Middle Aged ; Prognosis ; RNA, Long Noncoding/genetics ; Up-Regulation/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Mmu-miR-125b overexpression suppresses NO production in activated macrophages by targeting eEF2K and CCNA2.
Autorzy:
Xu Z; State Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, 100005, China.
Zhao L; State Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, 100005, China.
Yang X; State Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, 100005, China.
Ma S; State Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, 100005, China.
Ge Y; State Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, 100005, China.
Liu Y; State Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, 100005, China.
Liu S; State Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, 100005, China.
Shi J; State Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, 100005, China. .
Zheng D; State Key Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, 100005, China. .
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Źródło:
BMC cancer [BMC Cancer] 2016 Mar 28; Vol. 16, pp. 252. Date of Electronic Publication: 2016 Mar 28.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cyclin A2/*genetics
Elongation Factor 2 Kinase/*genetics
MicroRNAs/*biosynthesis
Nitric Oxide/*biosynthesis
Cell Line, Tumor ; Cell Proliferation/genetics ; Cyclin A2/biosynthesis ; Elongation Factor 2 Kinase/biosynthesis ; Endotoxins/toxicity ; Gene Expression Regulation ; Humans ; Macrophage Activation/genetics ; Macrophage Activation/immunology ; Macrophages/drug effects ; Macrophages/immunology ; Macrophages/pathology ; MicroRNAs/immunology ; Nitric Oxide/immunology ; Nitric Oxide Synthase Type II/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknieSzczegóły
Wyświetlanie 1-20 z 179

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