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Wyświetlanie 1-20 z 113958
Tytuł:
Ad-hoc modifications of cyclic mimetics of SOCS1 protein: Structural and functional insights.
Autorzy:
La Manna S; Department of Pharmacy, University of Naples 'Federico II', 80131, Naples, Italy.
Fortuna S; CONCEPT Lab, Istituto Italiano di Tecnologia (IIT), Via E. Melen, 83, I-16152, Genova, Italy.
Leone M; Institute of Biostructures and Bioimaging, CNR, 80145, Naples, Italy.
Mercurio FA; Institute of Biostructures and Bioimaging, CNR, 80145, Naples, Italy.
Di Donato I; Department of Pharmacy, University of Naples 'Federico II', 80131, Naples, Italy.
Bellavita R; Department of Pharmacy, University of Naples 'Federico II', 80131, Naples, Italy.
Grieco P; Department of Pharmacy, University of Naples 'Federico II', 80131, Naples, Italy.
Merlino F; Department of Pharmacy, University of Naples 'Federico II', 80131, Naples, Italy.
Marasco D; Department of Pharmacy, University of Naples 'Federico II', 80131, Naples, Italy. Electronic address: .
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Źródło:
European journal of medicinal chemistry [Eur J Med Chem] 2022 Dec 05; Vol. 243, pp. 114781. Date of Electronic Publication: 2022 Sep 19.
Typ publikacji:
Journal Article
MeSH Terms:
Suppressor of Cytokine Signaling Proteins*/chemistry
Suppressor of Cytokine Signaling Proteins*/metabolism
Janus Kinases*/metabolism
Suppressor of Cytokine Signaling 1 Protein/genetics ; Suppressor of Cytokine Signaling 1 Protein/metabolism ; Signal Transduction ; Cytokines/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Single-component lipid nanoparticles for engineering SOCS1 gene-silenced dendritic cells to boost tumor immunotherapy.
Autorzy:
Yu Z; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Drug Discovery for Metabolic Diseases, Center of Advanced Pharmaceuticals and Biomaterials, China Pharmaceutical University, Nanjing 210009, P.R. China. .
Wu M; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Drug Discovery for Metabolic Diseases, Center of Advanced Pharmaceuticals and Biomaterials, China Pharmaceutical University, Nanjing 210009, P.R. China. .
Huang Y; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Drug Discovery for Metabolic Diseases, Center of Advanced Pharmaceuticals and Biomaterials, China Pharmaceutical University, Nanjing 210009, P.R. China. .
Wang Y; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Drug Discovery for Metabolic Diseases, Center of Advanced Pharmaceuticals and Biomaterials, China Pharmaceutical University, Nanjing 210009, P.R. China. .
Chen Y; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Drug Discovery for Metabolic Diseases, Center of Advanced Pharmaceuticals and Biomaterials, China Pharmaceutical University, Nanjing 210009, P.R. China. .
Long Q; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Drug Discovery for Metabolic Diseases, Center of Advanced Pharmaceuticals and Biomaterials, China Pharmaceutical University, Nanjing 210009, P.R. China. .
Lin Z; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Drug Discovery for Metabolic Diseases, Center of Advanced Pharmaceuticals and Biomaterials, China Pharmaceutical University, Nanjing 210009, P.R. China. .
Xue L; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Drug Discovery for Metabolic Diseases, Center of Advanced Pharmaceuticals and Biomaterials, China Pharmaceutical University, Nanjing 210009, P.R. China. .
Ju C; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Drug Discovery for Metabolic Diseases, Center of Advanced Pharmaceuticals and Biomaterials, China Pharmaceutical University, Nanjing 210009, P.R. China. .
Zhang C; State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Drug Discovery for Metabolic Diseases, Center of Advanced Pharmaceuticals and Biomaterials, China Pharmaceutical University, Nanjing 210009, P.R. China. .; Chongqing Innovation Institute of China Pharmaceutical University, Chongqing 401135, China.
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Źródło:
Biomaterials science [Biomater Sci] 2022 Dec 20; Vol. 11 (1), pp. 263-277. Date of Electronic Publication: 2022 Dec 20.
Typ publikacji:
Journal Article
MeSH Terms:
Suppressor of Cytokine Signaling Proteins*/genetics
Suppressor of Cytokine Signaling Proteins*/metabolism
Neoplasms*/metabolism
Animals ; Mice ; Suppressor of Cytokine Signaling 1 Protein/genetics ; Suppressor of Cytokine Signaling 1 Protein/metabolism ; Immunotherapy ; Antigen Presentation ; RNA, Small Interfering/genetics ; RNA, Small Interfering/metabolism ; Dendritic Cells ; Mice, Inbred C57BL ; Tumor Microenvironment
Czasopismo naukowe
Szczegóły
Tytuł:
Structural analysis and conformational dynamics of SOCS1 gene mutations involved in diffuse large B-cell lymphoma.
Autorzy:
Dristy TT; Department of Genetic Engineering and Biotechnology, East West University (EWU), Bangladesh.
Noor AR; Department of Genetic Engineering and Biotechnology, East West University (EWU), Bangladesh.
Dey P; Faculty of Medicine, Shimane University, Japan.
Saha A; Department of Bioinformatics and Biotechnology, Asian University for Women, Bangladesh. Electronic address: .
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Źródło:
Gene [Gene] 2023 May 15; Vol. 864, pp. 147293. Date of Electronic Publication: 2023 Feb 20.
Typ publikacji:
Journal Article
MeSH Terms:
Suppressor of Cytokine Signaling Proteins*/genetics
Lymphoma, Large B-Cell, Diffuse*/genetics
Humans ; Suppressor of Cytokine Signaling 1 Protein/genetics ; Mutation ; Mutation Rate ; Polymorphism, Single Nucleotide
Czasopismo naukowe
Szczegóły
Tytuł:
Expression of mycobacterium tuberculosis induced SOCS3 and STAT3 and the implications on innate immunity in TB patients vs healthy contacts in high TB/HIV endemic setting: A cross-sectional analytical study.
Autorzy:
Lungu P; Department of Internal Medicine, University of Zambia School of Medicine, Lusaka, Zambia.
Mushota K; USAID Sustaining Technical and Analytic Resources (STAR) Project, Lusaka, Zambia.
Njelesani E; Faculty of Medicine, Lusaka Apex Medical University, Lusaka, Zambia.
Sukwa T; Faculty of Medicine, Lusaka Apex Medical University, Lusaka, Zambia.
Lakhi S; Department of Internal Medicine, University of Zambia School of Medicine, Lusaka, Zambia.
Mwaba P; Faculty of Medicine, Lusaka Apex Medical University, Lusaka, Zambia.
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Źródło:
PloS one [PLoS One] 2022 Jul 15; Vol. 17 (7), pp. e0263624. Date of Electronic Publication: 2022 Jul 15 (Print Publication: 2022).
Typ publikacji:
Journal Article
MeSH Terms:
HIV Infections*/complications
Immunity, Innate*/genetics
STAT3 Transcription Factor*/genetics
STAT3 Transcription Factor*/metabolism
Suppressor of Cytokine Signaling 3 Protein*/genetics
Suppressor of Cytokine Signaling 3 Protein*/metabolism
Tuberculosis*/genetics
Tuberculosis*/immunology
Cross-Sectional Studies ; Humans ; Interleukin-6/metabolism ; Mycobacterium tuberculosis/immunology ; Signal Transduction ; Suppressor of Cytokine Signaling Proteins/genetics ; Suppressor of Cytokine Signaling Proteins/metabolism ; Zambia/epidemiology
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
N6-methyladenosine of Socs1 modulates macrophage inflammatory response in different stiffness environments.
Autorzy:
Hu Z; Division of Paediatric Dentistry and Orthodontics, Faculty of Dentistry, The University of Hong Kong, Hong Kong SAR, China.
Li Y; Division of Periodontology and Implant Dentistry, Faculty of Dentistry, The University of Hong Kong, Hong Kong SAR, China.
Yuan W; School of Dentistry, University of California, Los Angeles, Los Angeles, CA 90095, USA.
Jin L; Division of Periodontology and Implant Dentistry, Faculty of Dentistry, The University of Hong Kong, Hong Kong SAR, China.
Leung WK; Division of Periodontology and Implant Dentistry, Faculty of Dentistry, The University of Hong Kong, Hong Kong SAR, China.
Zhang C; Division of Restorative Dental Sciences, Faculty of Dentistry, The University of Hong Kong, Hong Kong SAR, China.
Yang Y; Division of Paediatric Dentistry and Orthodontics, Faculty of Dentistry, The University of Hong Kong, Hong Kong SAR, China.
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Źródło:
International journal of biological sciences [Int J Biol Sci] 2022 Sep 11; Vol. 18 (15), pp. 5753-5769. Date of Electronic Publication: 2022 Sep 11 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Suppressor of Cytokine Signaling Proteins*/genetics
Macrophages*/metabolism
Suppressor of Cytokine Signaling 1 Protein/genetics ; Suppressor of Cytokine Signaling 1 Protein/metabolism ; Adenosine/metabolism ; Cytokines/metabolism ; RNA, Messenger/metabolism ; Chromatin/metabolism ; Hydrogels/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Individual genetic variability mainly of Proinflammatory cytokines, cytokine receptors, and toll-like receptors dictates pathophysiology of COVID-19 disease.
Autorzy:
Vakil MK; Noncommunicable Diseases Research Center, Fasa University of Medical Sciences, Fasa, Iran.
Mansoori Y; Noncommunicable Diseases Research Center, Fasa University of Medical Sciences, Fasa, Iran.
Al-Awsi GRL; University of Al-Qadisiyah, College of Science, Al Diwaniyah, Iraq.; Department of Radiological Techniques, Al-Mustaqbal University College, Babylon, Iraq.
Hosseinipour A; Department of Internal Medicine, Fasa University of Medical Sciences, Fasa, Iran.
Ahsant S; Noncommunicable Diseases Research Center, Fasa University of Medical Sciences, Fasa, Iran.
Ahmadi S; Noncommunicable Diseases Research Center, Fasa University of Medical Sciences, Fasa, Iran.
Ekrahi M; Noncommunicable Diseases Research Center, Fasa University of Medical Sciences, Fasa, Iran.
Montaseri Z; Department of Infectious Diseases, Fasa University of Medical Sciences, Fasa, Iran.
Pezeshki B; Noncommunicable Diseases Research Center, Fasa University of Medical Sciences, Fasa, Iran.
Mohaghegh P; Pediatrics Department, School of Medicine, Fasa University of Medical Sciences, Fasa, Iran.
Sohrabpour M; Noncommunicable Diseases Research Center, Fasa University of Medical Sciences, Fasa, Iran.
Bahmanyar M; Pediatrics Department, School of Medicine, Fasa University of Medical Sciences, Fasa, Iran.
Daraei A; Department of Medical Genetics, School of Medicine, Babol University of Medical Sciences, Babol, Iran.
Dadkhah Jouybari T; Department of Medical Genetics, Faculty of Medical Sciences, Tarbiat Modares University, Tehran, Iran.
Tavassoli A; Department of Pathology, Fasa University of Medical Sciences, Fasa, Iran.
Ghasemian A; Noncommunicable Diseases Research Center, Fasa University of Medical Sciences, Fasa, Iran.
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Źródło:
Journal of medical virology [J Med Virol] 2022 Sep; Vol. 94 (9), pp. 4088-4096. Date of Electronic Publication: 2022 May 31.
Typ publikacji:
Journal Article; Review
MeSH Terms:
COVID-19*/genetics
COVID-19*/physiopathology
Cytokines*/genetics
Receptors, Cytokine*/genetics
Toll-Like Receptors*/genetics
Cytokine Release Syndrome/genetics ; Humans ; SARS-CoV-2
Czasopismo naukowe
Szczegóły
Tytuł:
The Suppressor of Cytokine Signalling family of proteins and their potential impact on COVID-19 disease progression.
Autorzy:
Low ZY; School of Science, Monash University, Bandar Sunway, Selangor, Malaysia.
Wen Yip AJ; School of Science, Monash University, Bandar Sunway, Selangor, Malaysia.
Chow VTK; Infectious Diseases Translational Research Program, Department of Microbiology and Immunology, Yong Loo Lin School of Medicine, National University of Singapore, Singapore.
Lal SK; School of Science, Monash University, Bandar Sunway, Selangor, Malaysia.; Tropical Medicine and Biology Platform, Monash University, Bandar Sunway, Selangor, Malaysia.
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Źródło:
Reviews in medical virology [Rev Med Virol] 2022 May; Vol. 32 (3), pp. e2300. Date of Electronic Publication: 2021 Sep 21.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
COVID-19*
Disease Progression*
Suppressor of Cytokine Signaling Proteins*/genetics
Suppressor of Cytokine Signaling Proteins*/metabolism
Cytokines/metabolism ; Humans ; SARS-CoV-2 ; Suppressor of Cytokine Signaling 3 Protein/genetics ; Suppressor of Cytokine Signaling 3 Protein/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
Analyses of the complex formation of staphylococcal enterotoxin A and the human gp130 cytokine receptor.
Autorzy:
Uzunçayır S; Department of Experimental Medical Science, Lund University, Sweden.
Vera-Rodriguez A; Institute for Protein Innovation, Boston, MA, USA.; Division of Hematology-Oncology, Boston Children's Hospital, Harvard Medical School, Boston, MA, USA.
Regenthal P; Department of Experimental Medical Science, Lund University, Sweden.
Åbacka H; Department of Experimental Medical Science, Lund University, Sweden.
Emanuelsson C; Department of Chemistry, Division for Biochemistry and Structural Biology, Lund University, Sweden.
Bahl CD; Institute for Protein Innovation, Boston, MA, USA.; Division of Hematology-Oncology, Boston Children's Hospital, Harvard Medical School, Boston, MA, USA.
Lindkvist-Petersson K; Department of Experimental Medical Science, Lund University, Sweden.; LINXS - Lund Institute of Advanced Neutron and X-ray Science, Sweden.
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Źródło:
FEBS letters [FEBS Lett] 2022 Apr; Vol. 596 (7), pp. 910-923. Date of Electronic Publication: 2022 Jan 30.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Enterotoxins*/metabolism
Receptors, Cytokine*
Animals ; Cytokine Receptor gp130 ; Glycoproteins ; Humans ; Mice ; Rats ; Superantigens
Czasopismo naukowe
Szczegóły
Tytuł:
Specific cytokines of interleukin-6 family interact with S100 proteins.
Autorzy:
Kazakov AS; Institute for Biological Instrumentation, Pushchino Scientific Center for Biological Research of the Russian Academy of Sciences, Institutskaya str., 7, Pushchino, Moscow region 142290 Russia.
Sokolov AS; Institute for Biological Instrumentation, Pushchino Scientific Center for Biological Research of the Russian Academy of Sciences, Institutskaya str., 7, Pushchino, Moscow region 142290 Russia.
Permyakova ME; Institute for Biological Instrumentation, Pushchino Scientific Center for Biological Research of the Russian Academy of Sciences, Institutskaya str., 7, Pushchino, Moscow region 142290 Russia.
Litus EA; Institute for Biological Instrumentation, Pushchino Scientific Center for Biological Research of the Russian Academy of Sciences, Institutskaya str., 7, Pushchino, Moscow region 142290 Russia.
Uversky VN; Department of Molecular Medicine and Byrd Alzheimer's Research Institute, Morsani College of Medicine, University of South Florida, 12901 Bruce B. Downs Blvd., Tampa, FL MDC07, USA. Electronic address: .
Permyakov EA; Institute for Biological Instrumentation, Pushchino Scientific Center for Biological Research of the Russian Academy of Sciences, Institutskaya str., 7, Pushchino, Moscow region 142290 Russia.
Permyakov SE; Institute for Biological Instrumentation, Pushchino Scientific Center for Biological Research of the Russian Academy of Sciences, Institutskaya str., 7, Pushchino, Moscow region 142290 Russia. Electronic address: .
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Źródło:
Cell calcium [Cell Calcium] 2022 Jan; Vol. 101, pp. 102520. Date of Electronic Publication: 2021 Dec 14.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Interleukin-6*
Receptors, Cytokine*/metabolism
Cytokine Receptor gp130 ; Cytokines/metabolism ; S100 Proteins
Czasopismo naukowe
Szczegóły
Tytuł:
Optimization of Phosphotyrosine Peptides that Target the SH2 Domain of SOCS1 and Block Substrate Ubiquitination.
Autorzy:
Chen H; The Walter and Eliza Hall Institute of Medical Research, Parkville 3052, Australia.; Department of Medical Biology, The University of Melbourne, Parkville 3010, Australia.
Wu Y; The Walter and Eliza Hall Institute of Medical Research, Parkville 3052, Australia.; Department of Medical Biology, The University of Melbourne, Parkville 3010, Australia.
Li K; The Walter and Eliza Hall Institute of Medical Research, Parkville 3052, Australia.; Department of Medical Biology, The University of Melbourne, Parkville 3010, Australia.
Currie I; The Walter and Eliza Hall Institute of Medical Research, Parkville 3052, Australia.; Department of Medical Biology, The University of Melbourne, Parkville 3010, Australia.
Keating N; The Walter and Eliza Hall Institute of Medical Research, Parkville 3052, Australia.; Department of Medical Biology, The University of Melbourne, Parkville 3010, Australia.
Dehkhoda F; The Walter and Eliza Hall Institute of Medical Research, Parkville 3052, Australia.; Department of Medical Biology, The University of Melbourne, Parkville 3010, Australia.
Grohmann C; The Walter and Eliza Hall Institute of Medical Research, Parkville 3052, Australia.; Department of Medical Biology, The University of Melbourne, Parkville 3010, Australia.
Babon JJ; The Walter and Eliza Hall Institute of Medical Research, Parkville 3052, Australia.; Department of Medical Biology, The University of Melbourne, Parkville 3010, Australia.
Nicholson SE; The Walter and Eliza Hall Institute of Medical Research, Parkville 3052, Australia.; Department of Medical Biology, The University of Melbourne, Parkville 3010, Australia.
Sleebs BE; The Walter and Eliza Hall Institute of Medical Research, Parkville 3052, Australia.; Department of Medical Biology, The University of Melbourne, Parkville 3010, Australia.
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Źródło:
ACS chemical biology [ACS Chem Biol] 2022 Feb 18; Vol. 17 (2), pp. 449-462. Date of Electronic Publication: 2022 Jan 06.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Suppressor of Cytokine Signaling Proteins*/chemistry
src Homology Domains*
Phosphotyrosine/metabolism ; Suppressor of Cytokine Signaling 1 Protein/metabolism ; Ubiquitination
Czasopismo naukowe
Szczegóły
Tytuł:
MiR-155-5p modulates inflammatory phenotype of activated oral lichen-planus-associated-fibroblasts by targeting SOCS1.
Autorzy:
Cheng J; Department of Oral Mucosal Diseases, The Affiliated Stomatological Hospital of Nanjing Medical University, Nanjing, China.; Jiangsu Province Key Laboratory of Oral Diseases, Nanjing Medical University, Nanjing, China.; Jiangsu Province Engineering Research Center of Stomatological Translational Medicine, Nanjing, China.
Zhang Y; Department of Oral Mucosal Diseases, The Affiliated Stomatological Hospital of Nanjing Medical University, Nanjing, China.; Jiangsu Province Key Laboratory of Oral Diseases, Nanjing Medical University, Nanjing, China.; Jiangsu Province Engineering Research Center of Stomatological Translational Medicine, Nanjing, China.
Yang J; Department of Oral Mucosal Diseases, The Affiliated Stomatological Hospital of Nanjing Medical University, Nanjing, China.; Jiangsu Province Key Laboratory of Oral Diseases, Nanjing Medical University, Nanjing, China.; Jiangsu Province Engineering Research Center of Stomatological Translational Medicine, Nanjing, China.
Wang Y; Department of Oral Mucosal Diseases, The Affiliated Stomatological Hospital of Nanjing Medical University, Nanjing, China.; Jiangsu Province Key Laboratory of Oral Diseases, Nanjing Medical University, Nanjing, China.; Jiangsu Province Engineering Research Center of Stomatological Translational Medicine, Nanjing, China.
Xu J; Department of Oral Mucosal Diseases, The Affiliated Stomatological Hospital of Nanjing Medical University, Nanjing, China.; Jiangsu Province Key Laboratory of Oral Diseases, Nanjing Medical University, Nanjing, China.; Jiangsu Province Engineering Research Center of Stomatological Translational Medicine, Nanjing, China.
Fan Y; Department of Oral Mucosal Diseases, The Affiliated Stomatological Hospital of Nanjing Medical University, Nanjing, China. .; Jiangsu Province Key Laboratory of Oral Diseases, Nanjing Medical University, Nanjing, China. .; Jiangsu Province Engineering Research Center of Stomatological Translational Medicine, Nanjing, China. .
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Źródło:
Molecular biology reports [Mol Biol Rep] 2022 Aug; Vol. 49 (8), pp. 7783-7792. Date of Electronic Publication: 2022 Jun 22.
Typ publikacji:
Journal Article
MeSH Terms:
Lichen Planus, Oral*/metabolism
Lichen Planus, Oral*/pathology
Inflammation/*metabolism
MicroRNAs/*metabolism
Suppressor of Cytokine Signaling 1 Protein/*metabolism
Fibroblasts/metabolism ; Humans ; Interleukin-6/genetics ; Interleukin-6/metabolism ; Interleukin-8/genetics ; Phenotype ; Suppressor of Cytokine Signaling 1 Protein/antagonists & inhibitors ; Suppressor of Cytokine Signaling Proteins/genetics
Czasopismo naukowe
Szczegóły
Tytuł:
Comparison of cytotoxic potency between freshly cultured and freshly thawed cytokine-induced killer cells from human umbilical cord blood.
Autorzy:
Ngo HT; Stem Cell Institute, University of Science Ho Chi Minh City, Ho Chi Minh City, Viet Nam.; Vietnam National University Ho Chi Minh City, Ho Chi Minh City, Viet Nam.
Dang VT; Stem Cell Institute, University of Science Ho Chi Minh City, Ho Chi Minh City, Viet Nam.; Vietnam National University Ho Chi Minh City, Ho Chi Minh City, Viet Nam.
Nguyen NH; Stem Cell Institute, University of Science Ho Chi Minh City, Ho Chi Minh City, Viet Nam.; Vietnam National University Ho Chi Minh City, Ho Chi Minh City, Viet Nam.
Bui AN; Stem Cell Institute, University of Science Ho Chi Minh City, Ho Chi Minh City, Viet Nam.; Vietnam National University Ho Chi Minh City, Ho Chi Minh City, Viet Nam.
Van Pham P; Stem Cell Institute, University of Science Ho Chi Minh City, Ho Chi Minh City, Viet Nam. .; Vietnam National University Ho Chi Minh City, Ho Chi Minh City, Viet Nam. .; Laboratory of Stem Cell Research and Application, University of Science Ho Chi Minh City, Ho Chi Minh City, Viet Nam. .; Laboratory of Cancer Research, University of Science Ho Chi Minh City, Ho Chi Minh City, Viet Nam. .
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Źródło:
Cell and tissue banking [Cell Tissue Bank] 2023 Mar; Vol. 24 (1), pp. 139-152. Date of Electronic Publication: 2022 Jul 06.
Typ publikacji:
Comparative Study; Journal Article
MeSH Terms:
Cytokine-Induced Killer Cells*
Neoplasms*/pathology
Animals ; Humans ; Mice ; Cell Proliferation ; Cells, Cultured ; Fetal Blood
Czasopismo naukowe
Szczegóły
Tytuł:
Huashibaidu formula attenuates sepsis-induced acute lung injury via suppressing cytokine storm: Implications for treatment of COVID-19.
Autorzy:
Zhang F; Institute of Chinese Materia Medica, China Academy of Chinese Medical Sciences, No. 16 Nanxiaojie, Dongzhimennei Ave, Beijing 100700, China.
Guo F; Institute of Chinese Materia Medica, China Academy of Chinese Medical Sciences, No. 16 Nanxiaojie, Dongzhimennei Ave, Beijing 100700, China.
Zhang Y; Institute of Chinese Materia Medica, China Academy of Chinese Medical Sciences, No. 16 Nanxiaojie, Dongzhimennei Ave, Beijing 100700, China.
Xu H; Institute of Chinese Materia Medica, China Academy of Chinese Medical Sciences, No. 16 Nanxiaojie, Dongzhimennei Ave, Beijing 100700, China.
Liu Y; Institute of Chinese Materia Medica, China Academy of Chinese Medical Sciences, No. 16 Nanxiaojie, Dongzhimennei Ave, Beijing 100700, China.
Lin L; Institute of Chinese Materia Medica, China Academy of Chinese Medical Sciences, No. 16 Nanxiaojie, Dongzhimennei Ave, Beijing 100700, China.
Li H; Institute of Chinese Materia Medica, China Academy of Chinese Medical Sciences, No. 16 Nanxiaojie, Dongzhimennei Ave, Beijing 100700, China.
Yang H; Experimental Research Center, China Academy of Chinese Medical Sciences, No. 16 Nanxiaojie, Dongzhimennei Ave, Beijing 100700, China. Electronic address: .
Huang L; National Resource Center for Chinese Materia Medica, China Academy of Chinese Medical Sciences, No. 16 Nanxiaojie, Dongzhimennei Ave, Beijing 100700, China. Electronic address: .
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Źródło:
Phytomedicine : international journal of phytotherapy and phytopharmacology [Phytomedicine] 2023 Jan; Vol. 109, pp. 154549. Date of Electronic Publication: 2022 Nov 19.
Typ publikacji:
Journal Article
MeSH Terms:
Acute Lung Injury*/drug therapy
Acute Lung Injury*/etiology
Cytokine Release Syndrome*/drug therapy
Cytokine Release Syndrome*/virology
Sepsis*/complications
Sepsis*/drug therapy
Animals ; Rats ; COVID-19 ; Cytokines/metabolism ; Granulocyte-Macrophage Colony-Stimulating Factor/metabolism ; NF-kappa B/metabolism ; Phosphatidylinositol 3-Kinases ; Proto-Oncogene Proteins c-akt/metabolism ; Toll-Like Receptor 4/metabolism ; Tumor Necrosis Factor-alpha/metabolism
Czasopismo naukowe
Szczegóły
Tytuł:
2-Deoxy-d-glucose induces deglycosylation of proinflammatory cytokine receptors and strongly reduces immunological responses in mouse models of inflammation.
Autorzy:
Uehara I; Department of Molecular Oncology, Institute for Advanced Medical Sciences, Nippon Medical School, Tokyo, Japan.
Kajita M; Department of Molecular Oncology, Institute for Advanced Medical Sciences, Nippon Medical School, Tokyo, Japan.
Tanimura A; Department of Molecular Oncology, Institute for Advanced Medical Sciences, Nippon Medical School, Tokyo, Japan.
Hida S; Department of Molecular and Cellular Health Sciences, Graduate School of Pharmaceutical Sciences, Nagoya City University, Nagoya, Japan.
Onda M; Department of Pathology, Integrative Oncological Pathology, Nippon Medical School, Tokyo, Japan.
Naito Z; Department of Pathology, Integrative Oncological Pathology, Nippon Medical School, Tokyo, Japan.
Taki S; Department of Molecular and Cellular Immunology, Shinshu University School of Medicine, Matsumoto, Japan.
Tanaka N; Department of Molecular Oncology, Institute for Advanced Medical Sciences, Nippon Medical School, Tokyo, Japan.
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Źródło:
Pharmacology research & perspectives [Pharmacol Res Perspect] 2022 Apr; Vol. 10 (2), pp. e00940.
Typ publikacji:
Journal Article
MeSH Terms:
Anti-Inflammatory Agents, Non-Steroidal/*pharmacology
Deoxyglucose/*pharmacology
Glycosylation/*drug effects
Inflammation/*prevention & control
Receptors, Cytokine/*drug effects
Animals ; Cells, Cultured ; Cytokine Receptor gp130/antagonists & inhibitors ; Cytokine Receptor gp130/metabolism ; Cytokine Release Syndrome/prevention & control ; Cytokines/metabolism ; Inflammation/chemically induced ; Janus Kinases/drug effects ; Lipopolysaccharides ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Receptors, Cytokine/immunology ; Receptors, Cytokine/metabolism ; Receptors, Interleukin-6/antagonists & inhibitors ; Receptors, Interleukin-6/genetics ; Receptors, Interleukin-6/metabolism
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Adjuvant cytokine-induced killer cell immunotherapy improves long-term survival in patients with stage I-II non-small cell lung cancer after curative surgery.
Autorzy:
Huang J; Biotherapy Center, First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Zhao X; Biotherapy Center, First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Zhang Z; Biotherapy Center, First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Yang S; Biotherapy Center, First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Chen X; Biotherapy Center, First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Shen C; Biotherapy Center, First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Wang L; Cancer Center, First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Qi Y; Department of Thoracic Surgery, First Affiliated Hospital of Zhengzhou University, Zhengzhou, China.
Zhang Y; Biotherapy Center, First Affiliated Hospital of Zhengzhou University, Zhengzhou, China; Cancer Center, First Affiliated Hospital of Zhengzhou University, Zhengzhou, China; School of Life Sciences, Zhengzhou University, Zhengzhou, China; State Key Laboratory of Esophageal Cancer Prevention and Treatment, Zhengzhou, China. Electronic address: .
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Źródło:
Cytotherapy [Cytotherapy] 2023 Feb; Vol. 25 (2), pp. 202-209. Date of Electronic Publication: 2022 Nov 12.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Carcinoma, Non-Small-Cell Lung*/therapy
Cytokine-Induced Killer Cells*
Lung Neoplasms*/therapy
Humans ; Retrospective Studies ; CD8-Positive T-Lymphocytes ; Immunotherapy, Adoptive/adverse effects ; Immunotherapy, Adoptive/methods ; Neoplasm Recurrence, Local/etiology ; Immunotherapy/adverse effects
Czasopismo naukowe
Szczegóły
Tytuł:
Risk factors of cytokine release syndrome: stress, catecholamines, and beyond.
Autorzy:
Xing X; Institute for Immunology and School of Medicine, Tsinghua University, Beijing, 100084, China.
Hu X; Institute for Immunology and School of Medicine, Tsinghua University, Beijing, 100084, China; Beijing Key Laboratory for Immunological Research on Chronic Diseases, Beijing, 100084, China; Tsinghua-Peking Center for Life Sciences, Tsinghua University, Beijing, 100084, China. Electronic address: .
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Źródło:
Trends in immunology [Trends Immunol] 2023 Feb; Vol. 44 (2), pp. 93-100. Date of Electronic Publication: 2022 Dec 30.
Typ publikacji:
Journal Article; Review; Research Support, Non-U.S. Gov't
MeSH Terms:
Catecholamines*/therapeutic use
Cytokine Release Syndrome*
Humans ; Cytokines ; Risk Factors
Czasopismo naukowe
Szczegóły
Tytuł:
Abnormally elevated EZH2-mediated H3K27me3 enhances osteogenesis in aortic valve interstitial cells by inhibiting SOCS3 expression.
Autorzy:
Xie K; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China.
Zeng J; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China.
Wen L; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China.
Peng X; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China; Huazhong University of Science and Technology Union Shenzhen Hospital, 518052, Shenzhen, China.
Lin Z; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China.
Xian G; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China.
Guo Y; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China.
Yang X; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China.
Li P; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China.
Xu D; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China. Electronic address: .
Zeng Q; State Key Laboratory of Organ Failure Research, Department of Cardiology, Nanfang Hospital, Southern Medical University, 510515, Guangzhou, China; Guangdong Provincial Key Laboratory of Shock and Microcirculation, Southern Medical University, 510515, Guangzhou, China; Bioland Laboratory (Guangzhou Regenerative Medicine and Health Guangdong Laboratory), 510005, Guangzhou, China. Electronic address: .
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Źródło:
Atherosclerosis [Atherosclerosis] 2023 Jan; Vol. 364, pp. 1-9. Date of Electronic Publication: 2022 Nov 23.
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Aortic Valve*/metabolism
Aortic Valve Stenosis*/genetics
Aortic Valve Stenosis*/metabolism
Enhancer of Zeste Homolog 2 Protein*/genetics
Enhancer of Zeste Homolog 2 Protein*/metabolism
Enhancer of Zeste Homolog 2 Protein*/pharmacology
Osteogenesis*/genetics
Suppressor of Cytokine Signaling 3 Protein*/genetics
Suppressor of Cytokine Signaling 3 Protein*/metabolism
Animals ; Mice ; Cells, Cultured ; Histones/metabolism ; Epigenesis, Genetic
SCR Disease Name:
Aortic Valve, Calcification of
Czasopismo naukowe
Szczegóły
Tytuł:
Efficacy of adoptively transferred allogeneic CIK cells on colorectal cancer: Augmentative antitumoral effects of GvHD.
Autorzy:
Muhammadnejad S; Gene Therapy Research Center, Digestive Diseases Research Institute, Tehran University of Medical Sciences, Tehran, Iran.
Monzavi SM; Gene Therapy Research Center, Digestive Diseases Research Institute, Tehran University of Medical Sciences, Tehran, Iran; Department of Applied Cell Sciences, School of Advanced Technologies in Medicine, Tehran University of Medical Sciences, Tehran, Iran.
Torabi-Rahvar M; Department of Immunology, School of Medicine, Tehran University of Medical Sciences, Tehran, Iran.
Sotoudeh M; Digestive Oncology Research Center, Digestive Diseases Research Institute, Tehran University of Medical Sciences, Tehran, Iran.
Muhammadnejad A; Cancer Biology Research Center, Cancer Institute of Iran, Tehran University of Medical Sciences, Tehran, Iran.
Tavakoli-Shiraji S; Hematology-Oncology and Stem Cell Transplantation Research Center, Tehran University of Medical Sciences, Tehran, Iran.
Ranjbar A; Gene Therapy Research Center, Digestive Diseases Research Institute, Tehran University of Medical Sciences, Tehran, Iran; SABZ Biomedicals Science-Based Company, Tehran, Iran.
Aghayan SS; Gene Therapy Research Center, Digestive Diseases Research Institute, Tehran University of Medical Sciences, Tehran, Iran; SABZ Biomedicals Science-Based Company, Tehran, Iran.
Khorsand AA; Gene Therapy Research Center, Digestive Diseases Research Institute, Tehran University of Medical Sciences, Tehran, Iran.
Moradzadeh K; Gene Therapy Research Center, Digestive Diseases Research Institute, Tehran University of Medical Sciences, Tehran, Iran; SABZ Biomedicals Science-Based Company, Tehran, Iran.
Janzamin E; Flowcyte Science-Based Company, Tehran, Iran.
Ahmadbeigi N; Gene Therapy Research Center, Digestive Diseases Research Institute, Tehran University of Medical Sciences, Tehran, Iran; SABZ Biomedicals Science-Based Company, Tehran, Iran. Electronic address: .
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Źródło:
International immunopharmacology [Int Immunopharmacol] 2023 Jan; Vol. 114, pp. 109446. Date of Electronic Publication: 2022 Dec 01.
Typ publikacji:
Journal Article
MeSH Terms:
Cytokine-Induced Killer Cells*
Graft vs Host Disease*
Colorectal Neoplasms*/therapy
Colorectal Neoplasms*/pathology
Hematopoietic Stem Cell Transplantation*
Humans ; Mice ; Animals ; Immunotherapy, Adoptive/methods
Czasopismo naukowe
Szczegóły
Tytuł:
Early Use of Corticosteroids following CAR T-Cell Therapy Correlates with Reduced Risk of High-Grade CRS without Negative Impact on Neurotoxicity or Treatment Outcome.
Autorzy:
Lakomy T; Department of Medical Oncology, Inselspital, Bern University Hospital, University of Bern, 3012 Bern, Switzerland.
Akhoundova D; Department of Medical Oncology, Inselspital, Bern University Hospital, University of Bern, 3012 Bern, Switzerland.
Nilius H; University Institute of Clinical Chemistry, Inselspital, Bern University Hospital, University of Bern, 3010 Bern, Switzerland.
Kronig MN; Department of Medical Oncology, Inselspital, Bern University Hospital, University of Bern, 3012 Bern, Switzerland.
Novak U; Department of Medical Oncology, Inselspital, Bern University Hospital, University of Bern, 3012 Bern, Switzerland.
Daskalakis M; Department of Hematology and Central Hematology Laboratory, Inselspital, Bern University Hospital, University of Bern, 3010 Bern, Switzerland.
Bacher U; Department of Hematology and Central Hematology Laboratory, Inselspital, Bern University Hospital, University of Bern, 3010 Bern, Switzerland.
Pabst T; Department of Medical Oncology, Inselspital, Bern University Hospital, University of Bern, 3012 Bern, Switzerland.
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Źródło:
Biomolecules [Biomolecules] 2023 Feb 17; Vol. 13 (2). Date of Electronic Publication: 2023 Feb 17.
Typ publikacji:
Journal Article
MeSH Terms:
Immunotherapy, Adoptive*
Cytokine Release Syndrome*/etiology
Humans ; Receptors, Antigen, T-Cell ; Retrospective Studies ; Adrenal Cortex Hormones/therapeutic use ; Treatment Outcome
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
Tytuł:
Status of TWEAK DNA methylation and mRNA expression in systemic lupus erythematosus.
Autorzy:
Liao L; Department of Rheumatology and Immunology, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
Li S; Department of Rheumatology and Immunology, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
Upreti B; Department of Rheumatology and Immunology, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
Wang X; Department of Rheumatology and Immunology, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
Yang Y; Department of Rheumatology and Immunology, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
Lou X; Department of Rheumatology and Immunology, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
Li L; Department of Rheumatology and Immunology, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
Cui R; Department of Rheumatology and Immunology, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
Liu S; Department of Rheumatology and Immunology, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
Cheng Y; Department of Psychiatry, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
Xu J; Department of Rheumatology and Immunology, 36657First Affiliated Hospital of Kunming Medical University, Kunming, China.
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Źródło:
Lupus [Lupus] 2023 Feb; Vol. 32 (2), pp. 171-179. Date of Electronic Publication: 2022 Nov 23.
Typ publikacji:
Journal Article
MeSH Terms:
Lupus Erythematosus, Systemic*/diagnosis
Cytokine TWEAK*/genetics
Humans ; DNA Methylation ; Enzyme-Linked Immunosorbent Assay ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Tumor Necrosis Factors/genetics
Czasopismo naukowe
Pełny tekst  Link otwiera się w nowym oknie Pełny tekstSzczegóły
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