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Wyszukujesz frazę ""epithelial‐mesenchymal transition"" wg kryterium: Temat


Tytuł:
Cytosolic EpCAM cooperates with H-Ras to regulate epithelial to mesenchymal transition through ZEB1.
Autorzy:
Omar FA; Norton Thoracic Institute, St. Joseph's Hospital and Medical Center, Phoenix, Arizona, United States of America.
Brown TC; Siteman Cancer Center, Washington University School of Medicine, St. Louis, Missouri, United States of America.
Gillanders WE; Siteman Cancer Center, Washington University School of Medicine, St. Louis, Missouri, United States of America.
Fleming TP; Norton Thoracic Institute, St. Joseph's Hospital and Medical Center, Phoenix, Arizona, United States of America.
Smith MA; Norton Thoracic Institute, St. Joseph's Hospital and Medical Center, Phoenix, Arizona, United States of America.
Bremner RM; Norton Thoracic Institute, St. Joseph's Hospital and Medical Center, Phoenix, Arizona, United States of America.
Sankpal NV; Norton Thoracic Institute, St. Joseph's Hospital and Medical Center, Phoenix, Arizona, United States of America.
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Źródło:
PloS one [PLoS One] 2023 May 16; Vol. 18 (5), pp. e0285707. Date of Electronic Publication: 2023 May 16 (Print Publication: 2023).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Epithelial-Mesenchymal Transition*/genetics
Signal Transduction*
Humans ; Cell Line, Tumor ; Cytosol/metabolism ; Epithelial Cell Adhesion Molecule/genetics ; Epithelial Cell Adhesion Molecule/metabolism ; Zinc Finger E-box-Binding Homeobox 1/genetics ; Zinc Finger E-box-Binding Homeobox 1/metabolism
Czasopismo naukowe
Tytuł:
Hydrostatic pressure mediates epithelial-mesenchymal transition of cholangiocytes through RhoA/ROCK and TGF-β/smad pathways.
Autorzy:
Khalifa MO; Department of Stem Cell Biology, Atomic Bomb Disease Institute, Nagasaki University, Nagasaki, Japan.; Department of Molecular Bone Biology, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.; Department of Anatomy and Embryology, Veterinary Medicine, Aswan University, Aswan, Egypt.
Yan C; Department of Stem Cell Biology, Atomic Bomb Disease Institute, Nagasaki University, Nagasaki, Japan.
Chai Y; Department of General Surgery, Jiangxi Provincial Children's Hospital, Nanchang, Jiangxi, China.
Ito K; Department of Molecular Bone Biology, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.
Zhang SH; Department of General Surgery, Jiangxi Provincial Children's Hospital, Nanchang, Jiangxi, China.
Li TS; Department of Stem Cell Biology, Atomic Bomb Disease Institute, Nagasaki University, Nagasaki, Japan.
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Źródło:
PloS one [PLoS One] 2024 Apr 05; Vol. 19 (4), pp. e0300548. Date of Electronic Publication: 2024 Apr 05 (Print Publication: 2024).
Typ publikacji:
Journal Article
MeSH Terms:
Signal Transduction*
Transforming Growth Factor beta*/metabolism
Humans ; Epithelial Cells/metabolism ; Epithelial-Mesenchymal Transition ; Hydrostatic Pressure ; Transforming Growth Factor beta1/metabolism
Czasopismo naukowe
Tytuł:
Melatonin serves as a novel treatment in bladder fibrosis through TGF-β1/Smad and EMT.
Autorzy:
Zhang Y; Department of Urology, Affiliated Hospital of Jiangsu University, Zhenjiang, Jiangsu, China.
Gong S; Department of Urology, Affiliated Hospital of Jiangsu University, Zhenjiang, Jiangsu, China.
He W; Department of Urology, Affiliated Hospital of Jiangsu University, Zhenjiang, Jiangsu, China.
Yuan J; Department of Urology, Affiliated Hospital of Jiangsu University, Zhenjiang, Jiangsu, China.
Dong D; Department of Urology, Affiliated Hospital of Jiangsu University, Zhenjiang, Jiangsu, China.
Zhang J; Department of Urology, Affiliated Hospital of Jiangsu University, Zhenjiang, Jiangsu, China.
Wang H; Department of Urology, Affiliated Hospital of Jiangsu University, Zhenjiang, Jiangsu, China.
Chen B; Department of Urology, Affiliated Hospital of Jiangsu University, Zhenjiang, Jiangsu, China.; Institute of Translational Medicine, Jiangsu University, Zhenjiang, Jiangsu, China.
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Źródło:
PloS one [PLoS One] 2024 Mar 13; Vol. 19 (3), pp. e0295104. Date of Electronic Publication: 2024 Mar 13 (Print Publication: 2024).
Typ publikacji:
Journal Article
MeSH Terms:
Transforming Growth Factor beta1*/metabolism
Melatonin*/pharmacology
Melatonin*/therapeutic use
Rats ; Humans ; Mice ; Animals ; Vimentin/metabolism ; Signal Transduction ; Urinary Bladder/metabolism ; Lipopolysaccharides/pharmacology ; Proteomics ; Fibrosis ; Epithelial-Mesenchymal Transition ; Collagen/pharmacology ; Cadherins/metabolism
Czasopismo naukowe
Tytuł:
Sublethal thermal stress promotes migration and invasion of thyroid cancer cells.
Autorzy:
Kuo CY; Department of Surgery, MacKay Memorial Hospital, Taipei, Taiwan.; Department of Medicine, School of Medicine, MacKay Medical College, New Taipei City, Taiwan.
Tsai CH; Department of Surgery, MacKay Memorial Hospital, Taipei, Taiwan.; Department of Medicine, School of Medicine, MacKay Medical College, New Taipei City, Taiwan.
Wu JK; Department of Medical Research, MacKay Memorial Hospital, Taipei, Taiwan.
Cheng SP; Department of Surgery, MacKay Memorial Hospital, Taipei, Taiwan.; Department of Medicine, School of Medicine, MacKay Medical College, New Taipei City, Taiwan.; Department of Medical Research, MacKay Memorial Hospital, Taipei, Taiwan.; Institute of Biomedical Sciences, MacKay Medical College, New Taipei City, Taiwan.
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Źródło:
PloS one [PLoS One] 2024 Feb 23; Vol. 19 (2), pp. e0298903. Date of Electronic Publication: 2024 Feb 23 (Print Publication: 2024).
Typ publikacji:
Journal Article
MeSH Terms:
Thyroid Neoplasms*
Humans ; Cell Line, Tumor ; Cell Proliferation ; Cell Cycle ; Cell Movement ; Epithelial-Mesenchymal Transition
Czasopismo naukowe
Tytuł:
Human dermal fibroblast subpopulations and epithelial mesenchymal transition signals in hidradenitis suppurativa tunnels are normalized by spleen tyrosine kinase antagonism in vivo.
Autorzy:
Flora, Akshay (AUTHOR)
Jepsen, Rebecca (AUTHOR)
Kozera, Emily K. (AUTHOR)
Woods, Jane A. (AUTHOR)
Cains, Geoffrey D. (AUTHOR)
Radzieta, Michael (AUTHOR)
Jensen, Slade O. (AUTHOR)
Malone, Matthew (AUTHOR)
Frew, John W. (AUTHOR)
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Źródło:
PLoS ONE. 11/3/2023, Vol. 18 Issue 11, p1-13. 13p.
Czasopismo naukowe
Tytuł:
Expression of plant-produced anti-PD-L1 antibody with anoikis sensitizing activity in human lung cancer cells via., suppression on epithelial-mesenchymal transition.
Autorzy:
Phetphoung T; Graduate Program of Pharmaceutical Sciences and Technology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.; Center of Excellence in Plant-produced Pharmaceuticals, Chulalongkorn University, Bangkok, Thailand.; Department of Pharmacognosy and Pharmaceutical Botany, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
Malla A; Baiya Phytopharm Co., Ltd., Bangkok, Thailand.
Rattanapisit K; Baiya Phytopharm Co., Ltd., Bangkok, Thailand.
Pisuttinusart N; Graduate Program of Pharmaceutical Sciences and Technology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.; Center of Excellence in Plant-produced Pharmaceuticals, Chulalongkorn University, Bangkok, Thailand.; Department of Pharmacognosy and Pharmaceutical Botany, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
Damrongyot N; Department of Biochemistry and Microbiology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
Joyjamras K; Pharmacology and Toxicology Unit, Department of Medical Science, Faculty of Science, Rangsit University, Pathum Thani, Thailand.
Chanvorachote P; Department of Pharmacology and Physiology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.; Center of Excellence in Cancer Cell and Molecular Biology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
Phakham T; Center of Excellence in Systems Biology, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand.; Excellence Center for Cancer Immunotherapy, Faculty of Medicine, Chulalongkorn University and King Chulalongkorn Memorial Hospital, Bangkok, Thailand.
Wongtangprasert T; Center of Excellence in Systems Biology, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand.; Excellence Center for Cancer Immunotherapy, Faculty of Medicine, Chulalongkorn University and King Chulalongkorn Memorial Hospital, Bangkok, Thailand.; Excellence Chulalongkorn Comprehensive Cancer Center, King Chulalongkorn Memorial Hospital, Bangkok, Thailand.
Strasser R; Department of Applied Genetics and Cell Biology, University of Natural Resources and Life Sciences, Vienna, Austria.
Chaotham C; Department of Biochemistry and Microbiology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.; Center of Excellence in Cancer Cell and Molecular Biology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
Phoolcharoen W; Center of Excellence in Plant-produced Pharmaceuticals, Chulalongkorn University, Bangkok, Thailand.; Department of Pharmacognosy and Pharmaceutical Botany, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
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Źródło:
PloS one [PLoS One] 2022 Nov 11; Vol. 17 (11), pp. e0274737. Date of Electronic Publication: 2022 Nov 11 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Epithelial-Mesenchymal Transition*
Lung Neoplasms*/drug therapy
Cricetinae ; Animals ; Humans ; CHO Cells ; Anoikis ; Cricetulus ; B7-H1 Antigen/genetics ; B7-H1 Antigen/metabolism
Czasopismo naukowe
Tytuł:
LGR5 promotes invasion and migration by regulating YAP activity in hypopharyngeal squamous cell carcinoma cells under inflammatory condition.
Autorzy:
Zhu Z; Department of Otolaryngology-Head and Neck Surgery, The First Hospital of Jilin University, Changchun, Jilin, China.; Department of Breast Surgery, Jilin University, Changchun, Jilin, China.
Yu S; Department of Otolaryngology-Head and Neck Surgery, The First Hospital of Jilin University, Changchun, Jilin, China.
Niu K; Department of Otolaryngology-Head and Neck Surgery, The First Hospital of Jilin University, Changchun, Jilin, China.
Wang P; Department of Otolaryngology-Head and Neck Surgery, The First Hospital of Jilin University, Changchun, Jilin, China.
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Źródło:
PloS one [PLoS One] 2022 Oct 26; Vol. 17 (10), pp. e0275679. Date of Electronic Publication: 2022 Oct 26 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Epithelial-Mesenchymal Transition*/genetics
Head and Neck Neoplasms*
Humans ; Cell Line, Tumor ; Cell Movement ; Cell Proliferation ; Leucine ; Neoplastic Processes ; Receptors, G-Protein-Coupled/genetics ; Receptors, G-Protein-Coupled/metabolism ; RNA, Small Interfering ; Squamous Cell Carcinoma of Head and Neck ; Verteporfin/pharmacology
Czasopismo naukowe
Tytuł:
Epithelial-mesenchymal transition, regulated by β-catenin and Twist, leads to esophageal wall remodeling in pediatric eosinophilic esophagitis.
Autorzy:
Garcia E; Department of Pediatrics, University of Alberta, Edmonton, Alberta, Canada.
Ladak Z; Department of Pediatrics, University of Alberta, Edmonton, Alberta, Canada.
Landry T; Department of Pediatrics, University of Alberta, Edmonton, Alberta, Canada.
Wollin M; Department of Pediatrics, University of Alberta, Edmonton, Alberta, Canada.
Persad ARL; Department of Pediatrics, University of Alberta, Edmonton, Alberta, Canada.
Sergi CM; Department of Pediatrics, University of Alberta, Edmonton, Alberta, Canada.
Huynh HQ; Department of Pediatrics, University of Alberta, Edmonton, Alberta, Canada.
Persad R; Department of Pediatrics, University of Alberta, Edmonton, Alberta, Canada.
Persad S; Department of Pediatrics, University of Alberta, Edmonton, Alberta, Canada.
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Źródło:
PloS one [PLoS One] 2022 Mar 03; Vol. 17 (3), pp. e0264622. Date of Electronic Publication: 2022 Mar 03 (Print Publication: 2022).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Eosinophilic Esophagitis*/pathology
Epithelial-Mesenchymal Transition*
Nuclear Proteins*/physiology
Twist-Related Protein 1*/physiology
beta Catenin*/physiology
Cadherins/physiology ; Child ; Humans
Czasopismo naukowe
Tytuł:
Correlation of breast cancer microcirculation construction with tumor stem cells (CSCs) and epithelial-mesenchymal transition (EMT) based on contrast-enhanced ultrasound (CEUS).
Autorzy:
Leng X; Department of Ultrasound, the Affiliated Tumor Hospital of Xinjiang Medical University, Urumqi, Xinjiang, China.
Huang G; Department of Hematology and Oncology, the Fifth Affiliated Hospital of Xinjiang Medical University, Urumqi, Xinjiang, China.
Li S; Department of Ultrasound, the Affiliated Tumor Hospital of Xinjiang Medical University, Urumqi, Xinjiang, China.
Yao M; Department of Ultrasound, the Affiliated Tumor Hospital of Xinjiang Medical University, Urumqi, Xinjiang, China.
Ding J; Basic Medical College, Xinjiang Medical University, Urumqi, Xinjiang, China.
Ma F; Department of Ultrasound, the Affiliated Tumor Hospital of Xinjiang Medical University, Urumqi, Xinjiang, China.
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Źródło:
PloS one [PLoS One] 2021 Dec 21; Vol. 16 (12), pp. e0261138. Date of Electronic Publication: 2021 Dec 21 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Epithelial-Mesenchymal Transition*
Microcirculation*
Biomarkers, Tumor/*metabolism
Breast Neoplasms/*pathology
Contrast Media/*metabolism
Neoplastic Stem Cells/*pathology
Ultrasonography, Mammary/*methods
Breast Neoplasms/diagnostic imaging ; Breast Neoplasms/metabolism ; Female ; Humans ; Middle Aged ; Neoplastic Stem Cells/metabolism ; Prognosis
Czasopismo naukowe
Tytuł:
Hyperosmotic stress induces epithelial-mesenchymal transition through rearrangements of focal adhesions in tubular epithelial cells.
Autorzy:
Miyano T; Department of Mechanical Systems Engineering, Graduate School of Systems Design, Tokyo Metropolitan University, Tokyo, Japan.
Suzuki A; Department of Mechanical Systems Engineering, Graduate School of Systems Design, Tokyo Metropolitan University, Tokyo, Japan.
Sakamoto N; Department of Mechanical Systems Engineering, Graduate School of Systems Design, Tokyo Metropolitan University, Tokyo, Japan.
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Źródło:
PloS one [PLoS One] 2021 Dec 21; Vol. 16 (12), pp. e0261345. Date of Electronic Publication: 2021 Dec 21 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Epithelial-Mesenchymal Transition*
Osmotic Pressure*
Stress, Physiological*
Epithelial Cells/*pathology
Fibrosis/*pathology
Focal Adhesions/*pathology
Kidney Tubules/*pathology
Animals ; Epithelial Cells/metabolism ; Fibrosis/metabolism ; Focal Adhesions/metabolism ; Kidney Tubules/metabolism ; Rats
Czasopismo naukowe
Tytuł:
Dermokine mutations contribute to epithelial-mesenchymal transition and advanced melanoma through ERK/MAPK pathways.
Autorzy:
Ma, Wenqiong (AUTHOR)
Wu, Zexiu (AUTHOR)
Maghsoudloo, Mazaher (AUTHOR)
Ijaz, Iqra (AUTHOR)
Dehghan Shasaltaneh, Marzieh (AUTHOR)
Zhang, Yuqin (AUTHOR)
Weng, Qiao (AUTHOR)
Fu, Junjiang (AUTHOR)
Imani, Saber (AUTHOR)
Wen, Qing Lian (AUTHOR)
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Źródło:
PLoS ONE. 7/11/2023, Vol. 18 Issue 7, p1-32. 32p.
Czasopismo naukowe
Tytuł:
Repression of Smad4 by MicroRNA-1285 moderates TGF-β-induced epithelial-mesenchymal transition in proliferative vitreoretinopathy.
Autorzy:
Pao SI; Department of Ophthalmology, Tri-Service General Hospital, National Defense Medical Center, Taipei, Taiwan, Republic of China.
Lin LT; Department of Ophthalmology, Tri-Service General Hospital Songshan Branch, National Defense Medical Center, Taipei, Taiwan, Republic of China.; Graduate Institute of Medical Sciences, National Defense Medical Center, Taipei, Taiwan, Republic of China.
Chen YH; Department of Ophthalmology, Tri-Service General Hospital, National Defense Medical Center, Taipei, Taiwan, Republic of China.; Graduate Institute of Medical Sciences, National Defense Medical Center, Taipei, Taiwan, Republic of China.
Chen CL; Department of Ophthalmology, Tri-Service General Hospital, National Defense Medical Center, Taipei, Taiwan, Republic of China.; Graduate Institute of Medical Sciences, National Defense Medical Center, Taipei, Taiwan, Republic of China.
Chen JT; Department of Ophthalmology, Tri-Service General Hospital, National Defense Medical Center, Taipei, Taiwan, Republic of China.; Graduate Institute of Medical Sciences, National Defense Medical Center, Taipei, Taiwan, Republic of China.
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Źródło:
PloS one [PLoS One] 2021 Aug 12; Vol. 16 (8), pp. e0254873. Date of Electronic Publication: 2021 Aug 12 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Epithelial-Mesenchymal Transition*/drug effects
Epithelial-Mesenchymal Transition*/genetics
MicroRNAs/*metabolism
Smad4 Protein/*metabolism
Transforming Growth Factor beta/*pharmacology
Vitreoretinopathy, Proliferative/*genetics
Vitreoretinopathy, Proliferative/*pathology
Animals ; Biomarkers/metabolism ; Cell Line ; Cell Movement/drug effects ; Cell Movement/genetics ; Cell Transdifferentiation/drug effects ; Cell Transdifferentiation/genetics ; Gene Expression Regulation/drug effects ; Humans ; MicroRNAs/genetics ; Models, Biological ; Rabbits ; Rats ; Retinal Pigment Epithelium/pathology
Czasopismo naukowe
Tytuł:
Norcycloartocarpin targets Akt and suppresses Akt-dependent survival and epithelial-mesenchymal transition in lung cancer cells.
Autorzy:
Nonpanya N; Department of Pharmacology and Physiology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.; Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
Sanookpan K; Department of Pharmacology and Physiology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.; Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
Joyjamras K; Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.; Graduate Program of Pharmaceutical Sciences and Technology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
Wichadakul D; Department of Computer Engineering, Faculty of Engineering, Chulalongkorn University, Bangkok, Thailand.
Sritularak B; Departments of Pharmacognosy and Pharmaceutical Botany, Chulalongkorn University, Bangkok, Thailand.
Chaotham C; Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.; Department of Biochemistry and Microbiology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
Chanvorachote P; Department of Pharmacology and Physiology, Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.; Faculty of Pharmaceutical Sciences, Chulalongkorn University, Bangkok, Thailand.
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Źródło:
PloS one [PLoS One] 2021 Aug 12; Vol. 16 (8), pp. e0254929. Date of Electronic Publication: 2021 Aug 12 (Print Publication: 2021).
Typ publikacji:
Journal Article
MeSH Terms:
Epithelial-Mesenchymal Transition*/drug effects
Flavonoids/*pharmacology
Lung Neoplasms/*pathology
Proto-Oncogene Proteins c-akt/*metabolism
Antineoplastic Agents/pharmacology ; Antineoplastic Agents/therapeutic use ; Apoptosis/drug effects ; Cell Death/drug effects ; Cell Line, Tumor ; Cell Movement/drug effects ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; Flavonoids/chemistry ; Focal Adhesion Protein-Tyrosine Kinases/metabolism ; Humans ; Lung Neoplasms/drug therapy ; Molecular Docking Simulation ; Neoplasm Invasiveness ; Proto-Oncogene Proteins c-akt/chemistry ; Pseudopodia/drug effects ; Pseudopodia/metabolism ; Signal Transduction/drug effects ; Tumor Stem Cell Assay ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
Tytuł:
The possible molecular mechanism underlying the involvement of the variable shear factor QKI in the epithelial-mesenchymal transformation of oesophageal cancer.
Autorzy:
Cui Y; School of Public Health, North China University of Science and Technology, Tangshan, Hebei Province, China.; Department of Hebei Key Laboratory of Medical-Industrial Integration Precision Medicine, Tangshan, Hebei Province, China.
Wu Y; School of Public Health, North China University of Science and Technology, Tangshan, Hebei Province, China.; Department of Hebei Key Laboratory of Medical-Industrial Integration Precision Medicine, Tangshan, Hebei Province, China.
Zhu Y; Department of Hebei Key Laboratory of Medical-Industrial Integration Precision Medicine, Tangshan, Hebei Province, China.; School of Clinical Medicine, North China University of Science and Technology, Tangshan, Hebei Province, China.; Affiliated Hospital, North China University of Science and Technology, Tangshan, Hebei Province, China.
Liu W; Department of Hebei Key Laboratory of Medical-Industrial Integration Precision Medicine, Tangshan, Hebei Province, China.; School of Clinical Medicine, North China University of Science and Technology, Tangshan, Hebei Province, China.; Affiliated Hospital, North China University of Science and Technology, Tangshan, Hebei Province, China.
Huang L; Department of Hebei Key Laboratory of Medical-Industrial Integration Precision Medicine, Tangshan, Hebei Province, China.; School of Clinical Medicine, North China University of Science and Technology, Tangshan, Hebei Province, China.; Affiliated Hospital, North China University of Science and Technology, Tangshan, Hebei Province, China.
Hong Z; School of Public Health, North China University of Science and Technology, Tangshan, Hebei Province, China.; Department of Hebei Key Laboratory of Medical-Industrial Integration Precision Medicine, Tangshan, Hebei Province, China.
Zhang M; School of Public Health, North China University of Science and Technology, Tangshan, Hebei Province, China.; Department of Hebei Key Laboratory of Medical-Industrial Integration Precision Medicine, Tangshan, Hebei Province, China.
Zheng X; School of Public Health, North China University of Science and Technology, Tangshan, Hebei Province, China.; Department of Hebei Key Laboratory of Medical-Industrial Integration Precision Medicine, Tangshan, Hebei Province, China.
Sun G; School of Public Health, North China University of Science and Technology, Tangshan, Hebei Province, China.; Department of Hebei Key Laboratory of Medical-Industrial Integration Precision Medicine, Tangshan, Hebei Province, China.; School of Clinical Medicine, North China University of Science and Technology, Tangshan, Hebei Province, China.; Affiliated Hospital, North China University of Science and Technology, Tangshan, Hebei Province, China.
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Źródło:
PloS one [PLoS One] 2023 Jul 10; Vol. 18 (7), pp. e0288403. Date of Electronic Publication: 2023 Jul 10 (Print Publication: 2023).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
MicroRNAs*/genetics
Esophageal Neoplasms*/genetics
Humans ; RNA, Circular/genetics ; Epithelial-Mesenchymal Transition/genetics ; Matrix Metalloproteinase 1/metabolism ; Matrix Metalloproteinase 10/metabolism ; RNA, Messenger/metabolism ; RNA-Binding Proteins
Czasopismo naukowe
Tytuł:
LINC01117 inhibits invasion and migration of lung adenocarcinoma through influencing EMT process.
Autorzy:
Liu L; Department of Environment and Life Sciences, Beijing University of Technology, Beijing, China.
Ren W; Department of Environment and Life Sciences, Beijing University of Technology, Beijing, China.
Du L; Department of Environment and Life Sciences, Beijing University of Technology, Beijing, China.
Xu K; NHC Key Laboratory of Biosafety, National Institute for Viral Disease Control and Prevention, Beijing, China.
Zhou Y; Department of Environment and Life Sciences, Beijing University of Technology, Beijing, China.
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Źródło:
PloS one [PLoS One] 2023 Jun 29; Vol. 18 (6), pp. e0287926. Date of Electronic Publication: 2023 Jun 29 (Print Publication: 2023).
Typ publikacji:
Journal Article
MeSH Terms:
Adenocarcinoma*/genetics
Adenocarcinoma of Lung*/genetics
Lung Neoplasms*/genetics
Humans ; Cell Nucleus ; Cytoplasm ; Epithelial-Mesenchymal Transition
Czasopismo naukowe
Tytuł:
Cholangiocyte Epithelial to Mesenchymal Transition (EMT) is a potential molecular mechanism driving ischemic cholangiopathy in liver transplantation.
Autorzy:
Wickramaratne N; Department of Surgery, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
Li R; Department of Surgery, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
Tian T; Department of Surgery, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
Khoraki J; Department of Surgery, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
Kang HS; Department of Surgery, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
Chmielewski C; Department of Physiology and Biophysics, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
Maitland J; Department of Pharmacology and Toxicology, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
Liebrecht LK; Department of Surgery, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
Fyffe-Freil R; Department of Physiology and Biophysics, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
Lindell SL; Department of Surgery, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
Mangino MJ; Department of Surgery, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.; Department of Physiology and Biophysics, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.; Department of Emergency Medicine, School of Medicine, Virginia Commonwealth University, Richmond, VA, United States of America.
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Źródło:
PloS one [PLoS One] 2021 Jul 07; Vol. 16 (7), pp. e0246978. Date of Electronic Publication: 2021 Jul 07 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, N.I.H., Extramural
MeSH Terms:
Epithelial-Mesenchymal Transition*
Ischemia/*pathology
Liver Transplantation/*adverse effects
Animals ; Humans ; Male ; Rats
Czasopismo naukowe
Tytuł:
MicroRNA-210-3p is transcriptionally upregulated by hypoxia induction and thus promoting EMT and chemoresistance in glioma cells.
Autorzy:
Liu H; Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Chen C; Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Zeng J; Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Zhao Z; Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Hu Q; Department of Laboratory Medicine, Hospital of Chengdu University of Traditional Chinese Medicine, Chengdu, China.
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Źródło:
PloS one [PLoS One] 2021 Jul 01; Vol. 16 (7), pp. e0253522. Date of Electronic Publication: 2021 Jul 01 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Brain Neoplasms/*genetics
Drug Resistance, Neoplasm/*genetics
Epithelial-Mesenchymal Transition/*genetics
Glioma/*genetics
MicroRNAs/*genetics
Transforming Growth Factor beta1/*biosynthesis
Anaerobiosis/physiology ; Brain Neoplasms/pathology ; Cell Hypoxia/genetics ; Cell Line, Tumor ; Cell Proliferation ; Epithelial-Mesenchymal Transition/physiology ; Gene Expression Regulation, Neoplastic/genetics ; Glioma/pathology ; Humans ; Hypoxia-Inducible Factor 1, alpha Subunit/biosynthesis ; Transcription, Genetic/genetics ; Transcriptional Activation/genetics ; Up-Regulation
Czasopismo naukowe
Tytuł:
Clinicopathological significance of the EMT-related proteins and their interrelationships in prostate cancer. An immunohistochemical study.
Autorzy:
Parol-Kulczyk M; Department of Clinical Pathomorphology, Faculty of Medicine, Collegium Medicum in Bydgoszcz, Nicolaus Copernicus University in Torun, Toruń, Poland.
Gzil A; Department of Clinical Pathomorphology, Faculty of Medicine, Collegium Medicum in Bydgoszcz, Nicolaus Copernicus University in Torun, Toruń, Poland.
Maciejewska J; Department of Clinical Pathomorphology, Faculty of Medicine, Collegium Medicum in Bydgoszcz, Nicolaus Copernicus University in Torun, Toruń, Poland.
Bodnar M; Department of Clinical Pathomorphology, Faculty of Medicine, Collegium Medicum in Bydgoszcz, Nicolaus Copernicus University in Torun, Toruń, Poland.
Grzanka D; Department of Clinical Pathomorphology, Faculty of Medicine, Collegium Medicum in Bydgoszcz, Nicolaus Copernicus University in Torun, Toruń, Poland.
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Źródło:
PloS one [PLoS One] 2021 Jun 22; Vol. 16 (6), pp. e0253112. Date of Electronic Publication: 2021 Jun 22 (Print Publication: 2021).
Typ publikacji:
Journal Article
MeSH Terms:
Epithelial-Mesenchymal Transition*
Antigens, CD/*metabolism
Biomarkers, Tumor/*metabolism
Cadherins/*metabolism
Intramolecular Oxidoreductases/*metabolism
Macrophage Migration-Inhibitory Factors/*metabolism
Prostatic Neoplasms/*pathology
SOXC Transcription Factors/*metabolism
beta Catenin/*metabolism
Aged ; Aged, 80 and over ; Biomarkers, Tumor/analysis ; Humans ; Immunohistochemistry ; Lymphatic Metastasis ; Male ; Middle Aged ; Prognosis ; Prostatic Neoplasms/metabolism ; Tumor Microenvironment
Czasopismo naukowe
Tytuł:
LINC00958 promotes proliferation, migration, invasion, and epithelial-mesenchymal transition of oesophageal squamous cell carcinoma cells.
Autorzy:
Wang B; Beijing International Science and Technology Cooperation Base of Antivirus Drug, Faculty of environment and life, Beijing University of Technology, Beijing, China.
Tang D; Beijing International Science and Technology Cooperation Base of Antivirus Drug, Faculty of environment and life, Beijing University of Technology, Beijing, China.
Liu Z; Beijing International Science and Technology Cooperation Base of Antivirus Drug, Faculty of environment and life, Beijing University of Technology, Beijing, China.
Wang Q; Beijing International Science and Technology Cooperation Base of Antivirus Drug, Faculty of environment and life, Beijing University of Technology, Beijing, China.
Xue S; Beijing International Science and Technology Cooperation Base of Antivirus Drug, Faculty of environment and life, Beijing University of Technology, Beijing, China.
Zhao Z; Beijing International Science and Technology Cooperation Base of Antivirus Drug, Faculty of environment and life, Beijing University of Technology, Beijing, China.
Feng D; Beijing International Science and Technology Cooperation Base of Antivirus Drug, Faculty of environment and life, Beijing University of Technology, Beijing, China.
Sheng C; Beijing International Science and Technology Cooperation Base of Antivirus Drug, Faculty of environment and life, Beijing University of Technology, Beijing, China.
Li J; Beijing International Science and Technology Cooperation Base of Antivirus Drug, Faculty of environment and life, Beijing University of Technology, Beijing, China.
Zhou Z; Beijing International Science and Technology Cooperation Base of Antivirus Drug, Faculty of environment and life, Beijing University of Technology, Beijing, China.
Pokaż więcej
Źródło:
PloS one [PLoS One] 2021 May 18; Vol. 16 (5), pp. e0251797. Date of Electronic Publication: 2021 May 18 (Print Publication: 2021).
Typ publikacji:
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms:
Cell Movement*
Cell Proliferation*
Epithelial-Mesenchymal Transition*
Esophageal Neoplasms/*metabolism
Esophageal Squamous Cell Carcinoma/*metabolism
RNA, Long Noncoding/*metabolism
RNA, Neoplasm/*metabolism
Cell Line, Tumor ; Esophageal Neoplasms/genetics ; Esophageal Neoplasms/pathology ; Esophageal Squamous Cell Carcinoma/genetics ; Esophageal Squamous Cell Carcinoma/pathology ; Humans ; Neoplasm Invasiveness ; RNA, Long Noncoding/genetics ; RNA, Neoplasm/genetics
Czasopismo naukowe

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