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Tytuł:
Alpha-1 antitrypsin expression is upregulated in multidrug-resistant cancer cells.
Autorzy:
Divac Rankov A; Institute of Molecular Genetics and Genetic Engineering, University of Belgrade, Belgrade, Serbia.
Jovanović Stojanov S; Department of Neurobiology, Institute for Biological Research 'Sinisa Stankovic' - National Institute of Republic of Serbia, University of Belgrade, Belgrade, Serbia.
Dragoj M; Department of Neurobiology, Institute for Biological Research 'Sinisa Stankovic' - National Institute of Republic of Serbia, University of Belgrade, Belgrade, Serbia.
Ljujić M; Institute of Molecular Genetics and Genetic Engineering, University of Belgrade, Belgrade, Serbia. .
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Źródło:
Histochemistry and cell biology [Histochem Cell Biol] 2023 May; Vol. 159 (5), pp. 431-437. Date of Electronic Publication: 2022 Dec 19.
Typ publikacji:
Journal Article
MeSH Terms:
Drug Resistance, Multiple*
Glioblastoma*/drug therapy
Glioblastoma*/genetics
Humans ; Interleukin-6 ; Drug Resistance, Neoplasm
Czasopismo naukowe
Tytuł:
CoQ 10 reduces glioblastoma growth and infiltration through proteome remodeling and inhibition of angiogenesis and inflammation.
Autorzy:
Frontiñán-Rubio J; Department of Medical Sciences, Faculty of Medicine, University of Castilla-La Mancha, 13071, Ciudad Real, Spain.; Oxidative Stress and Neurodegeneration Group, Faculty of Medicine, Regional Centre for Biomedical Research, University of Castilla-La Mancha, Ciudad Real, Spain.
Llanos-González E; Department of Medical Sciences, Faculty of Medicine, University of Castilla-La Mancha, 13071, Ciudad Real, Spain.; Oxidative Stress and Neurodegeneration Group, Faculty of Medicine, Regional Centre for Biomedical Research, University of Castilla-La Mancha, Ciudad Real, Spain.
García-Carpintero S; Department of Medical Sciences, Faculty of Medicine, University of Castilla-La Mancha, 13071, Ciudad Real, Spain.; Oxidative Stress and Neurodegeneration Group, Faculty of Medicine, Regional Centre for Biomedical Research, University of Castilla-La Mancha, Ciudad Real, Spain.
Peinado JR; Department of Medical Sciences, Faculty of Medicine, University of Castilla-La Mancha, 13071, Ciudad Real, Spain.; Oxidative Stress and Neurodegeneration Group, Faculty of Medicine, Regional Centre for Biomedical Research, University of Castilla-La Mancha, Ciudad Real, Spain.
Ballesteros-Yáñez I; EMAS Group, Faculty of Medicine, Regional Centre for Biomedical Research, University of Castilla-La Mancha, Ciudad Real, Spain.
Rayo MV; SaBio Research Group, Hunting Resources Research Institute (IREC), Ciudad Real, Spain.
de la Fuente J; SaBio Research Group, Hunting Resources Research Institute (IREC), Ciudad Real, Spain.
Pérez-García VM; Laboratory of Mathematical Oncology, University of Castilla-La Mancha, Ciudad Real, Spain.
Perez-Romasanta LA; Radiology and Medicinal Physics, Institute for Biomedical Research of Salamanca (IBSAL), Salamanca, Spain.
Malumbres M; Cell Division and Cancer Group, Spanish National Cancer Research Centre (CNIO), Madrid, Spain.
Alcaín FJ; Department of Medical Sciences, Faculty of Medicine, University of Castilla-La Mancha, 13071, Ciudad Real, Spain. .; Oxidative Stress and Neurodegeneration Group, Faculty of Medicine, Regional Centre for Biomedical Research, University of Castilla-La Mancha, Ciudad Real, Spain. .
Durán-Prado M; Department of Medical Sciences, Faculty of Medicine, University of Castilla-La Mancha, 13071, Ciudad Real, Spain. .; Oxidative Stress and Neurodegeneration Group, Faculty of Medicine, Regional Centre for Biomedical Research, University of Castilla-La Mancha, Ciudad Real, Spain. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2023 Feb; Vol. 46 (1), pp. 65-77. Date of Electronic Publication: 2022 Nov 02.
Typ publikacji:
Journal Article
MeSH Terms:
Glioblastoma*/pathology
Brain Neoplasms*/pathology
Humans ; Mice ; Animals ; Ubiquinone/pharmacology ; Ubiquinone/therapeutic use ; Proteome ; Antioxidants ; Hypoxia ; Inflammation ; Cell Line, Tumor
Czasopismo naukowe
Tytuł:
Kavain ablates the radio-resistance of IDH-wildtype glioblastoma by targeting LITAF/NF-κB pathway.
Autorzy:
Yu J; Department of Neurosurgery, Children's Hospital of Fudan University, Fudan University, No. 399, Wanyuan Road, Minhang District, Shanghai, 201102, People's Republic of China.
Shi J; Department of Neurosurgery, Center for Clinical Research and Translational Medicine, Yangpu Hospital, School of Medicine, Tongji University, 450 Tengyue Road, Shanghai, 200090, China.
Yuan F; Department of Neurosurgery, Shanghai Sixth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200072, China.
Yin W; Department of Neurosurgery, Center for Clinical Research and Translational Medicine, Yangpu Hospital, School of Medicine, Tongji University, 450 Tengyue Road, Shanghai, 200090, China.
Zeng H; Department of Neurosurgery, Center for Clinical Research and Translational Medicine, Yangpu Hospital, School of Medicine, Tongji University, 450 Tengyue Road, Shanghai, 200090, China.
Ge L; Department of Neurosurgery, Center for Clinical Research and Translational Medicine, Yangpu Hospital, School of Medicine, Tongji University, 450 Tengyue Road, Shanghai, 200090, China.
Li H; Department of Neurosurgery, Children's Hospital of Fudan University, Fudan University, No. 399, Wanyuan Road, Minhang District, Shanghai, 201102, People's Republic of China. .
Wang X; Department of Neurosurgery, Center for Clinical Research and Translational Medicine, Yangpu Hospital, School of Medicine, Tongji University, 450 Tengyue Road, Shanghai, 200090, China. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2023 Feb; Vol. 46 (1), pp. 179-193. Date of Electronic Publication: 2022 Dec 05.
Typ publikacji:
Journal Article
MeSH Terms:
Glioblastoma*/genetics
Brain Neoplasms*/genetics
Humans ; NF-kappa B/metabolism ; Pyrones ; Tumor Necrosis Factor-alpha/metabolism ; Cell Line, Tumor ; Nuclear Proteins/genetics ; Transcription Factors/metabolism
Czasopismo naukowe
Tytuł:
Antitumor Effect of Traditional Drugs for Neurological Disorders: Preliminary Studies in Neural Tumor Cell Lines.
Autorzy:
Doello K; Institute of Biopathology and Regenerative Medicine (IBIMER), Biomedical Research Center (CIBM), 18100, Granada, Spain.; Medical Oncology Service, Virgen de Las Nieves Hospital, 18014, Granada, Spain.
Mesas C; Institute of Biopathology and Regenerative Medicine (IBIMER), Biomedical Research Center (CIBM), 18100, Granada, Spain.; Instituto Biosanitario de Granada (Ibs.Granada), SAS-Universidad de Granada, 18012, Granada, Spain.; Department of Anatomy and Embryology, University of Granada, 18071, Granada, Spain.
Quiñonero F; Institute of Biopathology and Regenerative Medicine (IBIMER), Biomedical Research Center (CIBM), 18100, Granada, Spain.; Instituto Biosanitario de Granada (Ibs.Granada), SAS-Universidad de Granada, 18012, Granada, Spain.; Department of Anatomy and Embryology, University of Granada, 18071, Granada, Spain.
Rama AR; Institute of Biopathology and Regenerative Medicine (IBIMER), Biomedical Research Center (CIBM), 18100, Granada, Spain.; Department of Health Sciences, University Jaén, 23071, Jaén, Spain.
Vélez C; Institute of Biopathology and Regenerative Medicine (IBIMER), Biomedical Research Center (CIBM), 18100, Granada, Spain. .; Instituto Biosanitario de Granada (Ibs.Granada), SAS-Universidad de Granada, 18012, Granada, Spain. .; Department of Anatomy and Embryology, University of Granada, 18071, Granada, Spain. .
Perazzoli G; Institute of Biopathology and Regenerative Medicine (IBIMER), Biomedical Research Center (CIBM), 18100, Granada, Spain.; Department of Medicine, Physiotherapy and Nursing, University of Almería, 04120, Almería, Spain.
Ortiz R; Institute of Biopathology and Regenerative Medicine (IBIMER), Biomedical Research Center (CIBM), 18100, Granada, Spain.; Instituto Biosanitario de Granada (Ibs.Granada), SAS-Universidad de Granada, 18012, Granada, Spain.; Department of Anatomy and Embryology, University of Granada, 18071, Granada, Spain.
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Źródło:
Neurotoxicity research [Neurotox Res] 2022 Dec; Vol. 40 (6), pp. 1645-1652. Date of Electronic Publication: 2022 Nov 30.
Typ publikacji:
Journal Article
MeSH Terms:
Glioblastoma*/drug therapy
Glioblastoma*/pathology
Brain Neoplasms*/drug therapy
Adult ; Humans ; Valproic Acid ; Levetiracetam/pharmacology ; Methotrimeprazine/pharmacology ; Methotrimeprazine/therapeutic use ; Haloperidol ; Biperiden/pharmacology ; Biperiden/therapeutic use ; Dextromethorphan/pharmacology ; Dextromethorphan/therapeutic use ; Fingolimod Hydrochloride ; Cell Line, Tumor ; Apoptosis
Czasopismo naukowe
Tytuł:
Circ_0060055 Promotes the Growth, Invasion, and Radioresistance of Glioblastoma by Targeting MiR-197-3p/API5 Axis.
Autorzy:
Yuan J; Department of Radiotherapy, the Second Affiliated Hospital of Zhengzhou University, Zhengzhou, 450000, Henan, China.
Liu Z; Department of Radiotherapy, the Second Affiliated Hospital of Zhengzhou University, Zhengzhou, 450000, Henan, China.
Liu J; Department of Radiotherapy, the First Affiliated Hospital of Zhengzhou University, No. 1 Jianshe East Road, Erqi District, Zhengzhou, 450000, Henan, China.
Fan R; Department of Radiotherapy, the First Affiliated Hospital of Zhengzhou University, No. 1 Jianshe East Road, Erqi District, Zhengzhou, 450000, Henan, China. .
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Źródło:
Neurotoxicity research [Neurotox Res] 2022 Oct; Vol. 40 (5), pp. 1292-1303. Date of Electronic Publication: 2022 Jul 18.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Neoplasms*/genetics
Brain Neoplasms*/metabolism
Brain Neoplasms*/radiotherapy
Glioblastoma*/genetics
Glioblastoma*/metabolism
Glioblastoma*/radiotherapy
MicroRNAs*/genetics
MicroRNAs*/metabolism
Animals ; Apoptosis Regulatory Proteins/metabolism ; Cell Line, Tumor ; Cell Movement ; Cell Proliferation/genetics ; Gene Expression Regulation, Neoplastic ; Humans ; Nuclear Proteins/genetics ; Nuclear Proteins/metabolism ; RNA, Circular/genetics
Czasopismo naukowe
Tytuł:
MIR99AHG/miR-204-5p/TXNIP/Nrf2/ARE Signaling Pathway Decreases Glioblastoma Temozolomide Sensitivity.
Autorzy:
Zhou L; Department of Neurosurgery, Shanghai Fifth People's Hospital, Fudan University, No.801, Heqing Road, Minhang District, Shanghai, 200240, China.
Ma J; Department of Neurosurgery, Shanghai Fifth People's Hospital, Fudan University, No.801, Heqing Road, Minhang District, Shanghai, 200240, China. .
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Źródło:
Neurotoxicity research [Neurotox Res] 2022 Oct; Vol. 40 (5), pp. 1152-1162. Date of Electronic Publication: 2022 Jul 29.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Neoplasms*/drug therapy
Brain Neoplasms*/genetics
Glioblastoma*/drug therapy
Glioblastoma*/genetics
Glioblastoma*/metabolism
MicroRNAs*/genetics
MicroRNAs*/metabolism
RNA, Long Noncoding*/genetics
Carrier Proteins/genetics ; Carrier Proteins/metabolism ; Carrier Proteins/therapeutic use ; Cell Line, Tumor ; Cell Proliferation ; Gene Expression Regulation, Neoplastic ; Humans ; NF-E2-Related Factor 2/metabolism ; Signal Transduction ; Temozolomide/pharmacology ; Temozolomide/therapeutic use ; Thioredoxins/metabolism
Czasopismo naukowe
Tytuł:
Circular RNA VPS18 Promotes Glioblastoma Progression by Regulating miR-1229-3p/BCAT1 Axis.
Autorzy:
Huang Q; Department of Neurosurgery, Ganzhou People's Hospital of Jiangxi Province, No. 16 Meiguan Avenue, Zhanggong District, Ganzhou, 341000 Jiangxi Province, China.
Li W; Department of Neurosurgery, Ganzhou People's Hospital of Jiangxi Province, No. 16 Meiguan Avenue, Zhanggong District, Ganzhou, 341000 Jiangxi Province, China. .
Huang Y; Department of Neurosurgery, Ganzhou People's Hospital of Jiangxi Province, No. 16 Meiguan Avenue, Zhanggong District, Ganzhou, 341000 Jiangxi Province, China.
Chen Q; Department of Neurosurgery, Ganzhou People's Hospital of Jiangxi Province, No. 16 Meiguan Avenue, Zhanggong District, Ganzhou, 341000 Jiangxi Province, China.
Wei W; Department of Neurosurgery, Ganzhou People's Hospital of Jiangxi Province, No. 16 Meiguan Avenue, Zhanggong District, Ganzhou, 341000 Jiangxi Province, China.
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Źródło:
Neurotoxicity research [Neurotox Res] 2022 Oct; Vol. 40 (5), pp. 1138-1151. Date of Electronic Publication: 2022 Jul 01.
Typ publikacji:
Journal Article
MeSH Terms:
Glioblastoma*/genetics
Glioblastoma*/pathology
MicroRNAs*/genetics
MicroRNAs*/metabolism
Amino Acids, Branched-Chain/metabolism ; Animals ; Cell Line, Tumor ; Cell Proliferation/genetics ; Gene Expression Regulation, Neoplastic ; Humans ; Mice ; RNA, Circular/genetics ; Transaminases/genetics ; Transaminases/metabolism ; Vesicular Transport Proteins/genetics ; Vesicular Transport Proteins/metabolism
Czasopismo naukowe
Tytuł:
Distinct expression and function of breast cancer metastasis suppressor 1 in mutant P53 glioblastoma.
Autorzy:
Babu D; Neuroscience Laboratory, Department of Biotechnology and Bioinformatics, School of Life Sciences, University of Hyderabad, Room No: F-23/F-71, Hyderabad, Telangana State, 500 046, India.
Chintal R; Neuroscience Laboratory, Department of Biotechnology and Bioinformatics, School of Life Sciences, University of Hyderabad, Room No: F-23/F-71, Hyderabad, Telangana State, 500 046, India.
Panigrahi M; Department of Neurosurgery, Krishna Institute of Medical Sciences, 500 003, Secunderabad, Telangana State, India.
Phanithi PB; Neuroscience Laboratory, Department of Biotechnology and Bioinformatics, School of Life Sciences, University of Hyderabad, Room No: F-23/F-71, Hyderabad, Telangana State, 500 046, India. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2022 Dec; Vol. 45 (6), pp. 1451-1465. Date of Electronic Publication: 2022 Oct 26.
Typ publikacji:
Journal Article
MeSH Terms:
Astrocytoma*
Breast Neoplasms*/genetics
Glioblastoma*/genetics
Repressor Proteins*/genetics
Female ; Humans ; Cell Line, Tumor ; ErbB Receptors/metabolism ; Gene Expression Regulation, Neoplastic ; Neoplasm Proteins/metabolism ; NF-kappa B/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/metabolism
Czasopismo naukowe
Tytuł:
A Conceptual Framework for Inducing T Cell-Mediated Immunity Against Glioblastoma.
Autorzy:
Marx S; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, MA, 02215, USA.; Department of Immunology, Harvard Medical School, Boston, MA, 02115, USA.
Godicelj A; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, MA, 02215, USA.; Program in Immunology, Harvard Medical School, Boston, MA, 02115, USA.
Wucherpfennig KW; Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute, Boston, MA, 02215, USA. kai_.; Department of Immunology, Harvard Medical School, Boston, MA, 02115, USA. kai_.; Program in Immunology, Harvard Medical School, Boston, MA, 02115, USA. kai_.; Department of Neurology, Brigham and Women's Hospital, Boston, MA, 02215, USA. kai_.
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Źródło:
Seminars in immunopathology [Semin Immunopathol] 2022 Sep; Vol. 44 (5), pp. 697-707. Date of Electronic Publication: 2022 May 03.
Typ publikacji:
Journal Article; Review; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms:
Glioblastoma*/therapy
Glioma*
Antigens, Neoplasm ; Humans ; Immunity, Cellular ; Immunotherapy ; Ligands
Czasopismo naukowe
Tytuł:
Glioblastoma, from disease understanding towards optimal cell-based in vitro models.
Autorzy:
Boccellato C; Institute of Cell Biology and Immunology, University of Stuttgart, Allmandring 31, 70569, Stuttgart, Germany. .
Rehm M; Institute of Cell Biology and Immunology, University of Stuttgart, Allmandring 31, 70569, Stuttgart, Germany. .; Stuttgart Research Center Systems Biology, University of Stuttgart, 70569, Stuttgart, Germany. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2022 Aug; Vol. 45 (4), pp. 527-541. Date of Electronic Publication: 2022 Jun 28.
Typ publikacji:
Journal Article; Review
MeSH Terms:
Brain Neoplasms*/pathology
Glioblastoma*/pathology
Cell Line, Tumor ; Humans
Czasopismo naukowe
Tytuł:
Thallium Induces Antiproliferative and Cytotoxic Activity in Glioblastoma C6 and U373 Cell Cultures via Apoptosis and Changes in Cell Cycle.
Autorzy:
Rangel-López E; Laboratorio de Aminoácidos Excitadores/Laboratorio de Neurofarmacología Molecular y Nanotecnología, Instituto Nacional de Neurología y Neurocirugía, 14269, Mexico City, Mexico. .
Robles-Bañuelos B; Laboratorio de Aminoácidos Excitadores/Laboratorio de Neurofarmacología Molecular y Nanotecnología, Instituto Nacional de Neurología y Neurocirugía, 14269, Mexico City, Mexico.
Guadiana-Ramírez N; Laboratorio de Aminoácidos Excitadores/Laboratorio de Neurofarmacología Molecular y Nanotecnología, Instituto Nacional de Neurología y Neurocirugía, 14269, Mexico City, Mexico.
Alvarez-Garduño V; Laboratorio de Aminoácidos Excitadores/Laboratorio de Neurofarmacología Molecular y Nanotecnología, Instituto Nacional de Neurología y Neurocirugía, 14269, Mexico City, Mexico.
Galván-Arzate S; Departamento de Neuroquímica, Instituto Nacional de Neurología y Neurocirugía, 14269, Mexico City, Mexico.
Zazueta C; Departamento de Biomedicina Cardiovascular, Instituto Nacional de Cardiología Ignacio Chávez, 14080, Mexico City, Mexico.
Karasu C; Cellular Stress Response and Signal Transduction Research Laboratory, Faculty of Medicine, Department of Medical Pharmacology, Gazi University, Beşevler, 06500, Turkey.
Túnez I; Instituto de Investigaciones Biomédicas Maimonides de Córdoba (IMIBIC), Córdoba, Spain.; Departamento de Bioquímica y Biología Molecular, Facultad de Medicina y Enfermería, Universidad de Córdoba, Córdoba, Spain.; Red Española de Excelencia en Estimulación Cerebral (REDESTIM), Córdoba, Spain.
Tinkov A; IM Sechenov First Moscow State Medical University (Sechenov University), Moscow, Russia.; Yaroslavl State University, Yaroslavl, Russia.
Aschner M; Department of Molecular Pharmacology, Albert Einstein College of Medicine, Bronx, NY, 10461, USA.
Santamaría A; Laboratorio de Aminoácidos Excitadores/Laboratorio de Neurofarmacología Molecular y Nanotecnología, Instituto Nacional de Neurología y Neurocirugía, 14269, Mexico City, Mexico. .
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Źródło:
Neurotoxicity research [Neurotox Res] 2022 Jun; Vol. 40 (3), pp. 814-824. Date of Electronic Publication: 2022 Apr 27.
Typ publikacji:
Journal Article
MeSH Terms:
Antineoplastic Agents*/pharmacology
Glioblastoma*/metabolism
Animals ; Apoptosis ; Cell Culture Techniques ; Cell Cycle ; Rats ; Thallium/toxicity
Czasopismo naukowe
Tytuł:
Cystatin F acts as a mediator of immune suppression in glioblastoma.
Autorzy:
Senjor E; Department of Biotechnology, Jožef Stefan Institute, Jamova cesta 39, SI-1000, Ljubljana, Slovenia.; Faculty of Pharmacy, University of Ljubljana, Aškerčeva cesta 7, SI-1000, Ljubljana, Slovenia.
Perišić Nanut M; Department of Biotechnology, Jožef Stefan Institute, Jamova cesta 39, SI-1000, Ljubljana, Slovenia.
Breznik B; Department of Genetic Toxicology and Cancer Biology, National Institute of Biology, Večna pot 111, SI-1000, Ljubljana, Slovenia.
Mitrović A; Department of Biotechnology, Jožef Stefan Institute, Jamova cesta 39, SI-1000, Ljubljana, Slovenia.
Mlakar J; Institute of Pathology, Medical Faculty, University of Ljubljana, Korytkova 2, SI-1000, Ljubljana, Slovenia.
Rotter A; Department of Genetic Toxicology and Cancer Biology, National Institute of Biology, Večna pot 111, SI-1000, Ljubljana, Slovenia.
Porčnik A; Department of Neurosurgery, University Clinical Centre Ljubljana, SI-1000, Ljubljana, Slovenia.
Lah Turnšek T; Department of Genetic Toxicology and Cancer Biology, National Institute of Biology, Večna pot 111, SI-1000, Ljubljana, Slovenia.
Kos J; Department of Biotechnology, Jožef Stefan Institute, Jamova cesta 39, SI-1000, Ljubljana, Slovenia. .; Faculty of Pharmacy, University of Ljubljana, Aškerčeva cesta 7, SI-1000, Ljubljana, Slovenia. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2021 Oct; Vol. 44 (5), pp. 1051-1063. Date of Electronic Publication: 2021 Jun 29.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Expression Regulation, Neoplastic*
Biomarkers, Tumor/*genetics
Brain Neoplasms/*genetics
Cystatins/*genetics
Glioblastoma/*genetics
Killer Cells, Natural/*metabolism
Neoplastic Stem Cells/*metabolism
Biomarkers, Tumor/metabolism ; Brain Neoplasms/metabolism ; Brain Neoplasms/pathology ; CCAAT-Enhancer-Binding Protein-alpha/genetics ; CCAAT-Enhancer-Binding Protein-alpha/metabolism ; Cell Line, Tumor ; Cells, Cultured ; Cystatins/metabolism ; Cytotoxicity, Immunologic/genetics ; Glioblastoma/metabolism ; Glioblastoma/pathology ; Humans ; Immunohistochemistry ; Microglia/metabolism ; Monocytes/metabolism ; Reverse Transcriptase Polymerase Chain Reaction ; Tumor Cells, Cultured
Czasopismo naukowe
Tytuł:
DNA damage repair in glioblastoma: current perspectives on its role in tumour progression, treatment resistance and PIKKing potential therapeutic targets.
Autorzy:
Lozinski M; School of Biomedical Sciences and Pharmacy, Faculty of Health and Medicine, University of Newcastle, Newcastle, NSW, Australia.; Centre for Drug Repurposing and Medicines Research, University of Newcastle, Newcastle, NSW, Australia.; Hunter Medical Research Institute, Newcastle, NSW, Australia.
Bowden NA; Centre for Drug Repurposing and Medicines Research, University of Newcastle, Newcastle, NSW, Australia.; Hunter Medical Research Institute, Newcastle, NSW, Australia.; School of Medicine and Public Health, Faculty of Health and Medicine, University of Newcastle, Newcastle, NSW, Australia.
Graves MC; Centre for Drug Repurposing and Medicines Research, University of Newcastle, Newcastle, NSW, Australia.; Hunter Medical Research Institute, Newcastle, NSW, Australia.; School of Medicine and Public Health, Faculty of Health and Medicine, University of Newcastle, Newcastle, NSW, Australia.
Fay M; Centre for Drug Repurposing and Medicines Research, University of Newcastle, Newcastle, NSW, Australia.; Hunter Medical Research Institute, Newcastle, NSW, Australia.; Genesis Cancer Care, Gateshead, New South Wales, Australia.
Tooney PA; School of Biomedical Sciences and Pharmacy, Faculty of Health and Medicine, University of Newcastle, Newcastle, NSW, Australia. .; Centre for Drug Repurposing and Medicines Research, University of Newcastle, Newcastle, NSW, Australia. .; Hunter Medical Research Institute, Newcastle, NSW, Australia. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2021 Oct; Vol. 44 (5), pp. 961-981. Date of Electronic Publication: 2021 May 31.
Typ publikacji:
Journal Article; Review
MeSH Terms:
DNA Damage*
Brain Neoplasms/*genetics
DNA Repair/*genetics
Drug Resistance, Neoplasm/*genetics
Glioblastoma/*genetics
Antineoplastic Agents, Alkylating/therapeutic use ; Brain Neoplasms/drug therapy ; Cell Cycle Checkpoints/drug effects ; Cell Cycle Checkpoints/genetics ; DNA Repair/drug effects ; Drug Resistance, Neoplasm/drug effects ; Glioblastoma/drug therapy ; Humans ; Models, Genetic ; Temozolomide/therapeutic use
Czasopismo naukowe
Tytuł:
A comprehensive prognostic signature for glioblastoma patients based on transcriptomics and single cell sequencing.
Autorzy:
Fan F; Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, Hunan, 410008, People's Republic of China.; Center for Medical Genetics & Hunan Provincial Key Laboratory of Medical Genetics, School of Life Sciences, Central South University, Changsha, China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Hunan, China.
Zhang H; Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, Hunan, 410008, People's Republic of China.
Dai Z; Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, Hunan, 410008, People's Republic of China.
Zhang Y; One-third Lab, College of Bioinformatics Science and Technology, Harbin Medical University, Harbin, Hei Longjiang, 150000, People's Republic of China.
Xia Z; Department of Neurology, Hunan Aerospace Hospital, Changsha, China.
Cao H; Department of Psychiatry, The Second People's Hospital of Hunan Province, The Hospital of Hunan University of Chinese Medicine, Changsha, Hunan, China.
Yang K; Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, Hunan, 410008, People's Republic of China.; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Hunan, China.
Hu S; One-third Lab, College of Bioinformatics Science and Technology, Harbin Medical University, Harbin, Hei Longjiang, 150000, People's Republic of China.
Guo Y; Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, Hunan, 410008, People's Republic of China.
Ding F; Department of Clinical Laboratory Medicine, People's Hospital of Ningxia Hui Autonomous Region, First Affiliated Hospital of Northwest Minzu University, Yinchuan, Ningxia, China.
Cheng Q; Department of Neurosurgery, Xiangya Hospital, Central South University, Changsha, Hunan, 410008, People's Republic of China. .; National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Hunan, China. .
Zhang N; One-third Lab, College of Bioinformatics Science and Technology, Harbin Medical University, Harbin, Hei Longjiang, 150000, People's Republic of China. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2021 Aug; Vol. 44 (4), pp. 917-935. Date of Electronic Publication: 2021 Jun 17.
Typ publikacji:
Journal Article
MeSH Terms:
Gene Expression Regulation, Neoplastic*
Brain Neoplasms/*genetics
Gene Expression Profiling/*methods
Glioblastoma/*genetics
Single-Cell Analysis/*methods
Aged ; Brain Neoplasms/diagnosis ; Brain Neoplasms/therapy ; Chi-Square Distribution ; Cohort Studies ; Female ; Glioblastoma/diagnosis ; Glioblastoma/therapy ; Humans ; Kaplan-Meier Estimate ; Male ; Middle Aged ; Multivariate Analysis ; Nomograms ; Prognosis ; ROC Curve
Czasopismo naukowe
Tytuł:
In focus in HCB.
Autorzy:
Taatjes DJ; Department of Pathology and Laboratory Medicine, Larner College of Medicine, University of Vermont, Burlington, VT, 05405, USA. .
Roth J; University of Zurich, CH-8091, Zurich, Switzerland.
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Źródło:
Histochemistry and cell biology [Histochem Cell Biol] 2021 Dec; Vol. 156 (6), pp. 519-522.
Typ publikacji:
Editorial
MeSH Terms:
Paraffin Embedding*
Cytokines/*metabolism
Glioblastoma/*metabolism
Macrophages/*metabolism
Testis/*metabolism
Zika Virus/*metabolism
Electric Impedance ; Formaldehyde ; Glioblastoma/virology ; Humans ; Male
Opinia redakcyjna
Tytuł:
Activation of dopamine receptor D1 inhibits glioblastoma tumorigenicity by regulating autophagic activity.
Autorzy:
Yang K; Dalian Medical University, Dalian, People's Republic of China.
Wei M; Dalian Medical University, Dalian, People's Republic of China.
Yang Z; Liaoning Provincial Center for Clinical Research on Neurological Diseases, The First Affiliated Hospital, Dalian Medical University, Dalian, People's Republic of China.; Liaoning Provincial Key Laboratory for Research on Pathogenic Mechanisms of Neurological Diseases,, The First Affiliated Hospital of Dalian Medical University, Dalian, People's Republic of China.
Fu Z; Liaoning Provincial Center for Clinical Research on Neurological Diseases, The First Affiliated Hospital, Dalian Medical University, Dalian, People's Republic of China.; Liaoning Provincial Key Laboratory for Research on Pathogenic Mechanisms of Neurological Diseases,, The First Affiliated Hospital of Dalian Medical University, Dalian, People's Republic of China.
Xu R; Department of Neurosurgery, The First Affiliated Hospital of Dalian Medical University, Dalian, People's Republic of China.
Cheng C; Liaoning Provincial Center for Clinical Research on Neurological Diseases, The First Affiliated Hospital, Dalian Medical University, Dalian, People's Republic of China.; Liaoning Provincial Key Laboratory for Research on Pathogenic Mechanisms of Neurological Diseases,, The First Affiliated Hospital of Dalian Medical University, Dalian, People's Republic of China.
Chen X; Liaoning Provincial Center for Clinical Research on Neurological Diseases, The First Affiliated Hospital, Dalian Medical University, Dalian, People's Republic of China.; Liaoning Provincial Key Laboratory for Research on Pathogenic Mechanisms of Neurological Diseases,, The First Affiliated Hospital of Dalian Medical University, Dalian, People's Republic of China.
Chen S; Department of Neurology, The Ruijin Hospital of Shanghai Jiao-Tong University School of Medicine, Shanghai, China.
Dammer E; Emory School of Medicine and Center for Neurodegenerative Disease, Atlanta, GA, USA.
Le W; Liaoning Provincial Center for Clinical Research on Neurological Diseases, The First Affiliated Hospital, Dalian Medical University, Dalian, People's Republic of China. .; Liaoning Provincial Key Laboratory for Research on Pathogenic Mechanisms of Neurological Diseases,, The First Affiliated Hospital of Dalian Medical University, Dalian, People's Republic of China. .; 1st Hospital of Dalian Medical University, 193 Lianhe Street, Liaoning Province, Dalian, People's Republic of China. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2020 Dec; Vol. 43 (6), pp. 1175-1190. Date of Electronic Publication: 2020 Aug 06.
Typ publikacji:
Journal Article
MeSH Terms:
Autophagy*/drug effects
Carcinogenesis/*metabolism
Glioblastoma/*pathology
Receptors, Dopamine D1/*metabolism
Animals ; Calcium/metabolism ; Carcinogenesis/drug effects ; Carcinogenesis/pathology ; Cell Line, Tumor ; Cell Proliferation/drug effects ; Glioblastoma/metabolism ; Glioblastoma/ultrastructure ; Humans ; Intracellular Space/metabolism ; Lysosomes/drug effects ; Lysosomes/metabolism ; MAP Kinase Signaling System/drug effects ; Mice, Inbred BALB C ; Mice, Nude ; Models, Biological ; Receptors, Dopamine D1/antagonists & inhibitors ; TOR Serine-Threonine Kinases/metabolism ; Temozolomide/pharmacology ; Treatment Outcome ; Xenograft Model Antitumor Assays
Czasopismo naukowe
Tytuł:
Radiogenomics model for overall survival prediction of glioblastoma.
Autorzy:
Wijethilake N; Department of Biomedical Engineering, National University of Singapore, Singapore, Singapore.; Department of Electronics and Telecommunications, University of Moratuwa, Moratuwa, Sri Lanka.
Islam M; Department of Biomedical Engineering, National University of Singapore, Singapore, Singapore.; NUS Graduate School for Integrative Sciences and Engineering (NGS), National University of Singapore, Singapore, Singapore.
Ren H; Department of Biomedical Engineering, National University of Singapore, Singapore, Singapore. .; Chinese University of Hong Kong, Hong Kong, Hong Kong. .
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Źródło:
Medical & biological engineering & computing [Med Biol Eng Comput] 2020 Aug; Vol. 58 (8), pp. 1767-1777. Date of Electronic Publication: 2020 Jun 03.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Neoplasms/*mortality
Glioblastoma/*mortality
Brain Neoplasms/genetics ; Brain Neoplasms/pathology ; Gene Expression Profiling/methods ; Glioblastoma/genetics ; Glioblastoma/pathology ; Humans ; Magnetic Resonance Imaging/methods ; Prognosis ; Survival Rate ; Transcriptome/genetics
Czasopismo naukowe
Tytuł:
Human bone marrow-derived mesenchymal stem cell-secreted exosomes overexpressing microRNA-34a ameliorate glioblastoma development via down-regulating MYCN.
Autorzy:
Wang B; Department of Neurosurgery, Henan Province People's Hospital, Zhengzhou University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China.; Department of Neurosurgery, People's Hospital Affiliated with Zhengzhou University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China.; Department of Neurosurgery, People's Hospital Affiliated with Medical college of Henan University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China.
Wu ZH; Department of Neurosurgery, Henan Province People's Hospital, Zhengzhou University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China.; Department of Neurosurgery, People's Hospital Affiliated with Zhengzhou University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China.; Department of Neurosurgery, People's Hospital Affiliated with Medical college of Henan University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China.
Lou PY; Department of Neurosurgery, Henan Province People's Hospital, Zhengzhou University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China.; Department of Neurosurgery, People's Hospital Affiliated with Zhengzhou University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China.; Department of Neurosurgery, People's Hospital Affiliated with Medical college of Henan University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China.
Chai C; Department of Ophthalmology, Henan Province People's Hospital, Zhengzhou University, Zhengzhou, 450003, People's Republic of China.
Han SY; Center for Translational Medicine, Henan Province People's Hospital, Zhengzhou University, Zhengzhou, 450003, People's Republic of China.
Ning JF; Department of Neurosurgery, University of Minnesota, Minneapolis, 55455, USA.
Li M; Department of Neurosurgery, Henan Province People's Hospital, Zhengzhou University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China. .; Department of Neurosurgery, People's Hospital Affiliated with Zhengzhou University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China. .; Department of Neurosurgery, People's Hospital Affiliated with Medical college of Henan University, No. 7, Weiwu Road, Zhengzhou, 450003, Henan Province, People's Republic of China. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2019 Dec; Vol. 42 (6), pp. 783-799. Date of Electronic Publication: 2019 Jul 22.
Typ publikacji:
Journal Article; Retracted Publication
MeSH Terms:
Down-Regulation*
Exosomes/*metabolism
Glioblastoma/*therapy
Mesenchymal Stem Cells/*metabolism
MicroRNAs/*metabolism
N-Myc Proto-Oncogene Protein/*genetics
Animals ; Base Sequence ; Cell Differentiation/drug effects ; Cell Line, Tumor ; Cell Movement/drug effects ; Cell Proliferation/drug effects ; Drug Resistance, Neoplasm/drug effects ; Exosomes/drug effects ; Exosomes/ultrastructure ; Gene Expression Regulation, Neoplastic/drug effects ; Glioblastoma/genetics ; Glioblastoma/pathology ; Humans ; Male ; Mesenchymal Stem Cells/drug effects ; Mice, Inbred BALB C ; Mice, Nude ; MicroRNAs/genetics ; Models, Biological ; N-Myc Proto-Oncogene Protein/metabolism ; Neoplasm Invasiveness ; Temozolomide/pharmacology ; Temozolomide/therapeutic use
Czasopismo naukowe
Tytuł:
The tumor suppressor FOXO3a mediates the response to EGFR inhibition in glioblastoma cells.
Autorzy:
Ramis G; Cancer Cell Biology Laboratory, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, and Institut d'Investigació Sanitària Illes Balears (IdISBa), Ctra. Valldemossa Km 7,5, Palma, Illes Balears, Spain.; Cellomics Unit, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Palma, Illes Balears, Spain.
Villalonga-Planells R; Departament de Química, Universitat de les Illes Balears, Palma, Illes Balears, Spain.
Serra-Sitjar M; Cancer Cell Biology Laboratory, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, and Institut d'Investigació Sanitària Illes Balears (IdISBa), Ctra. Valldemossa Km 7,5, Palma, Illes Balears, Spain.
Brell M; Department of Neurosurgery, Hospital Universitari Son Espases, Palma, Illes Balears, Spain.
Fernández de Mattos S; Cancer Cell Biology Laboratory, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, and Institut d'Investigació Sanitària Illes Balears (IdISBa), Ctra. Valldemossa Km 7,5, Palma, Illes Balears, Spain.; Departament de Biologia Fonamental i Ciències de la Salut, Universitat de les Illes Balears, Palma, Illes Balears, Spain.
Villalonga P; Cancer Cell Biology Laboratory, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, and Institut d'Investigació Sanitària Illes Balears (IdISBa), Ctra. Valldemossa Km 7,5, Palma, Illes Balears, Spain. .; Departament de Biologia Fonamental i Ciències de la Salut, Universitat de les Illes Balears, Palma, Illes Balears, Spain. .
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Źródło:
Cellular oncology (Dordrecht) [Cell Oncol (Dordr)] 2019 Aug; Vol. 42 (4), pp. 521-536. Date of Electronic Publication: 2019 Apr 13.
Typ publikacji:
Journal Article
MeSH Terms:
Brain Neoplasms/*metabolism
ErbB Receptors/*antagonists & inhibitors
Forkhead Box Protein O3/*metabolism
Glioblastoma/*metabolism
Brain Neoplasms/drug therapy ; Brain Neoplasms/genetics ; Brain Neoplasms/pathology ; Cell Line, Tumor ; Cell Nucleus/drug effects ; Cell Nucleus/metabolism ; Cell Proliferation/drug effects ; ErbB Receptors/metabolism ; Erlotinib Hydrochloride/pharmacology ; Glioblastoma/drug therapy ; Glioblastoma/genetics ; Glioblastoma/pathology ; Humans ; Phosphorylation/drug effects ; Protein Kinase Inhibitors/pharmacology ; Protein Kinase Inhibitors/therapeutic use ; Transcriptional Activation/genetics ; Trifluoperazine/pharmacology ; Trifluoperazine/therapeutic use
Czasopismo naukowe
Tytuł:
Selenium Enhances the Apoptotic Efficacy of Docetaxel Through Activation of TRPM2 Channel in DBTRG Glioblastoma Cells.
Autorzy:
Ertilav K; Department of Neurosurgery, Faculty of Medicine, Suleyman Demirel University, Isparta, Turkey.
Nazıroğlu M; Neuroscience Research Center, Suleyman Demirel University, Isparta, Turkey.; Drug Discovery and Development Research Group in Neuroscience, BSN Health, Analysis and Innovation, Goller Bolgesi Teknokenti, Suleyman Demirel University, Isparta, Turkey.
Ataizi ZS; Department of Neurosurgery, Yunus Emre State Hospital, Eskişehir, Turkey.
Braidy N; Centre for Healthy Brain Ageing, School of Psychiatry, University of New South Wales, Sydney, Australia. .
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Źródło:
Neurotoxicity research [Neurotox Res] 2019 May; Vol. 35 (4), pp. 797-808. Date of Electronic Publication: 2019 Feb 22.
Typ publikacji:
Journal Article
MeSH Terms:
Antineoplastic Agents/*administration & dosage
Apoptosis/*drug effects
Brain Neoplasms/*drug therapy
Docetaxel/*administration & dosage
Glioblastoma/*drug therapy
Selenium/*administration & dosage
TRPM Cation Channels/*metabolism
Brain Neoplasms/metabolism ; Calcium/metabolism ; Cell Line, Tumor ; Cell Survival/drug effects ; Glioblastoma/metabolism ; Humans ; Membrane Potential, Mitochondrial/drug effects ; Oxidative Stress/drug effects ; Poly (ADP-Ribose) Polymerase-1/metabolism ; Reactive Oxygen Species/metabolism
Czasopismo naukowe

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